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Interplay of SMARCAD1 and BRCA1 at Replication Forks to Maintain Genome Integrity

https://doi.org/10.2139/ssrn.3581365
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30/30 checkable references clean · checked 2026-08-19

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

The 30 checked references that resolve
resolves10.26508/lsa.201900433
Chromatin remodeler Fft3 plays a dual role at blocked DNA replication forks
resolves10.1080/14728222.2016.1195059
SMARCAD1 knockdown uncovers its role in breast cancer cell migration, invasion, and metastasis
resolves10.1016/j.gde.2012.11.001
To spread or not to spread—chromatin modifications in response to DNA damage
resolves10.1159/000494163
SMARCAD1 in Breast Cancer Progression
resolves10.1074/jbc.m109.082149
The Snf2 Homolog Fun30 Acts as a Homodimeric ATP-dependent Chromatin-remodeling Enzyme
resolves10.7554/elife.21687
Targeting of the Fun30 nucleosome remodeller by the Dpb11 scaffold facilitates cell cycle-regulated DNA end resection
resolves10.3389/fmolb.2019.00078
Nucleosome Remodeling by Fun30SMARCAD1 in the DNA Damage Response
resolves10.1158/0008-5472.can-18-2077
Radiosensitivity Is an Acquired Vulnerability of PARPi-Resistant BRCA1-Deficient Tumors
resolves10.1016/j.celrep.2018.06.061
RADX Modulates RAD51 Activity to Control Replication Fork Protection
resolves10.1038/nbt.3519
Near-optimal probabilistic RNA-seq quantification
resolves10.1074/jbc.m113.471979
The ATP-dependent Chromatin Remodeling Enzyme Fun30 Represses Transcription by Sliding Promoter-proximal Nucleosomes
resolves10.1038/nature10166
Integrated genomic analyses of ovarian carcinoma
resolves10.1016/j.isci.2018.03.016
SMARCAD1 Phosphorylation and Ubiquitination Are Required for Resection during DNA Double-Strand Break Repair
resolves10.1038/nature19826
Erratum: Replication fork stability confers chemoresistance in BRCA-deficient cells
resolves10.1093/bioinformatics/bty560
fastp: an ultra-fast all-in-one FASTQ preprocessor
resolves10.1016/j.molcel.2016.12.020
Forging Ahead through Darkness: PCNA, Still the Principal Conductor at the Replication Fork
resolves10.1038/emboj.2012.214
Mouse Rif1 is a key regulator of the replication‐timing programme in mammalian cells
resolves10.1038/nature11353
The yeast Fun30 and human SMARCAD1 chromatin remodellers promote DNA end resection
resolves10.1038/nbt.1511
MaxQuant enables high peptide identification rates, individualized p.p.b.-range mass accuracies and proteome-wide protein quantification
resolves10.1038/nsmb.3236
Human BRCA1–BARD1 ubiquitin ligase activity counteracts chromatin barriers to DNA resection
resolves10.1074/jbc.ra117.000959
The CUE1 domain of the SNF2-like chromatin remodeler SMARCAD1 mediates its association with KRAB-associated protein 1 (KAP1) and KAP1 target genes
resolves10.1038/ncomms12425
Synthetic viability by BRCA2 and PARP1/ARTD1 deficiencies
resolves10.1038/nmeth.4535
BRCA-deficient mouse mammary tumor organoids to study cancer-drug resistance
resolves10.1016/j.molcel.2017.06.023
RADX Promotes Genome Stability and Modulates Chemosensitivity by Regulating RAD51 at Replication Forks
resolves10.1158/2159-8290.cd-12-0049
Loss of 53BP1 Causes PARP Inhibitor Resistance in <i>Brca1</i> -Mutated Mouse Mammary Tumors
resolves10.1101/gad.13.20.2633
XRCC3 promotes homology-directed repair of DNA damage in mammalian cells
resolves10.1101/gad.12.24.3831
Double-strand break repair by interchromosomal recombination: suppression of chromosomal translocations
resolves10.1038/nmeth.3047
Improved vectors and genome-wide libraries for CRISPR screening
resolves10.1038/nmeth.2089
NIH Image to ImageJ: 25 years of image analysis
resolves10.1016/j.nbd.2006.08.004
Role of the transcription factor E2F1 in CXCR4-mediated neurotoxicity and HIV neuropathology
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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