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Adult <i>Camk2a</i> Gene Reinstatement Restores the Learning and Plasticity Deficits of <i>Camk2a</i> Knockout Mice

https://doi.org/10.2139/ssrn.4143785
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27/27 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

5 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 27 checked references that resolve
resolves10.1016/j.biopsych.2014.08.001
Syngap1 Haploinsufficiency Damages a Postnatal Critical Period of Pyramidal Cell Structural Maturation Linked to Cortical Circuit Assembly
resolves10.1523/JNEUROSCI.0640-14.2014
Temporal and Region-Specific Requirements of αCaMKII in Spatial and Contextual Learning
resolves10.1002/acn3.528
<i>De novo</i> variants in <i><scp>CAMK</scp>2A</i> and <i><scp>CAMK</scp>2B</i> cause neurodevelopmental disorders
resolves10.1016/S0169-328X(99)00131-X
Developmental expression of the CaM kinase II isoforms: ubiquitous γ- and δ-CaM kinase II are the early isoforms and most abundant in the developing nervous system
resolves10.1126/science.7939668
Abnormal Fear Response and Aggressive Behavior in Mutant Mice Deficient for α-Calcium-Calmodulin Kinase II
resolves10.1016/j.cell.2012.08.045
Pathogenic SYNGAP1 Mutations Impair Cognitive Development by Disrupting Maturation of Dendritic Spine Synapses
resolves10.1016/S0896-6273(02)01007-3
Inhibitory Autophosphorylation of CaMKII Controls PSD Association, Plasticity, and Learning
resolves10.1016/j.biopsych.2005.07.025
Postnatal Loss of Methyl-CpG Binding Protein 2 in the Forebrain is Sufficient to Mediate Behavioral Aspects of Rett Syndrome in Mice
resolves10.1038/nature13394
Genome sequencing identifies major causes of severe intellectual disability
resolves10.1523/JNEUROSCI.1341-18.2019
CAMK2-Dependent Signaling in Neurons Is Essential for Survival
resolves10.1016/j.conb.2017.10.014
Windows of opportunity: timing in neurodevelopmental disorders
resolves10.1016/j.ajhg.2017.10.003
De Novo Mutations in Protein Kinase Genes CAMK2A and CAMK2B Cause Intellectual Disability
resolves10.1093/hmg/ddt421
Rescue of behavioral and EEG deficits in male and female Mecp2-deficient mice by delayed Mecp2 gene reactivation
resolves10.1016/j.ridd.2010.12.018
Prevalence of intellectual disability: A meta-analysis of population-based studies
resolves10.1007/s40474-016-0085-7
Systematic Review of the Prevalence and Incidence of Intellectual Disabilities: Current Trends and Issues
resolves10.1038/nature16971
Adult restoration of Shank3 expression rescues selective autistic-like phenotypes
resolves10.1016/j.tins.2012.03.005
Sensitive time-windows for susceptibility in neurodevelopmental disorders
resolves10.1016/j.neuron.2014.05.015
Reduced Cognition in Syngap1 Mutants Is Caused by Isolated Damage within Developing Forebrain Excitatory Neurons
resolves10.1523/JNEUROSCI.0083-18.2018
Adult <i>Ube3a</i> Gene Reinstatement Restores the Electrophysiological Deficits of Prefrontal Cortex Layer 5 Neurons in a Mouse Model of Angelman Syndrome
resolves10.1016/S0896-6273(00)80569-3
CaMKIIβ Functions As an F-Actin Targeting Module that Localizes CaMKIIα/β Heterooligomers to Dendritic Spines
resolves10.1172/JCI80554
Ube3a reinstatement identifies distinct developmental windows in a murine Angelman syndrome model
resolves10.1126/science.1321493
Impaired Spatial Learning in α-Calcium-Calmodulin Kinase II Mutant Mice
resolves10.1126/science.1378648
Deficient Hippocampal Long-Term Potentiation in α-Calcium-Calmodulin Kinase II Mutant Mice
resolves10.1186/s13229-018-0231-7
A behavioral test battery for mouse models of Angelman syndrome: a powerful tool for testing drugs and novel Ube3a mutants
resolves10.1186/s13229-019-0277-1
Delayed loss of UBE3A reduces the expression of Angelman syndrome-associated phenotypes
resolves10.1186/s13229-020-00376-9
Assessing the requirements of prenatal UBE3A expression for rescue of behavioral phenotypes in a mouse model for Angelman syndrome
resolves10.1038/nrg3999
Genetic studies in intellectual disability and related disorders
The 5 references without a DOI — listed, not checked
no DOI — not checkedref5
no DOI — not checkedref7
no DOI — not checkedCA1 long-term potentiation is diminished but present in hippocampal slices from ?-CaMKII mutant mice
no DOI — not checkedConsequences of NMDA receptor deficiency can be rescued in the adult brain
no DOI — not checkedPharmacological intervention in young adolescents rescues synaptic physiology and behavioural deficits in Syngap1 +/-mice. Exp
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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