Reference health

Air Pollution and Alzheimer’s Disease: A Systematic Review and Meta-Analysis

https://doi.org/10.3233/jad-200483
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60/60 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

3 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 60 checked references that resolve
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Ozone, Particulate Matter, and Newly Diagnosed Alzheimer's Disease: A Population-Based Cohort Study in Taiwan
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Influence of PM2.5 Exposure Level on the Association between Alzheimer’s Disease and Allergic Rhinitis: A National Population-Based Cohort Study
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PM2.5, SO2 and NO2 co-exposure impairs neurobehavior and induces mitochondrial injuries in the mouse brain
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Particulate matter (PM10) exposure induces endothelial dysfunction and inflammation in rat brain
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Oxidative stress and inflammation are associated with physical frailty in patients with Alzheimer's disease
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resolves10.1016/j.envres.2019.108638
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resolves10.1097/MD.0000000000002418
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Oxidative Stress, Synaptic Dysfunction, and Alzheimer’s Disease
resolves10.1016/j.ynstr.2018.05.003
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resolves10.1515/revneuro-2012-0084
The effects of ozone exposure and associated injury mechanisms on the central nervous system
resolves10.3233/JAD-170857
Ozone Atmospheric Pollution and Alzheimer’s Disease: From Epidemiological Facts to Molecular Mechanisms
resolves10.1016/j.nrl.2017.04.001
La respuesta sistémica Th17/IL-17A aparece antes del proceso neurodegenerativo en el hipocampo de ratas expuestas a bajas dosis de ozono
resolves10.1016/j.neuroscience.2015.07.011
Oxidative stress caused by ozone exposure induces β-amyloid 1–42 overproduction and mitochondrial accumulation by activating the amyloidogenic pathway
resolves10.1155/2016/2125643
Syntaxin 5 Overexpression and<i>β</i>-Amyloid 1–42 Accumulation in Endoplasmic Reticulum of Hippocampal Cells in Rat Brain Induced by Ozone Exposure
resolves10.1093/toxsci/kfv124
Cyclic Ozone Exposure Induces Gender-Dependent Neuropathology and Memory Decline in an Animal Model of Alzheimer’s Disease
resolves10.1016/j.jhazmat.2012.04.063
In vivo screening to determine neurological hazards of nitrogen dioxide (NO2) using Wistar rats
resolves10.1016/j.envres.2015.02.022
Acute nitrogen dioxide inhalation induces mitochondrial dysfunction in rat brain
resolves10.1038/srep22429
NO2 inhalation promotes Alzheimer’s disease-like progression: cyclooxygenase-2-derived prostaglandin E2 modulation and monoacylglycerol lipase inhibition-targeted medication
resolves10.1164/rccm.201606-1275CI
Carbon Monoxide Poisoning: Pathogenesis, Management, and Future Directions of Therapy
resolves10.3109/08958378.2014.970786
Proteins as biomarkers of carbon monoxide neurotoxicity
resolves10.1371/journal.pmed.1002668
Association between ambient air pollution and daily hospital admissions for ischemic stroke: A nationwide time-series analysis
resolves10.1080/15376516.2017.1337256
The role of pro-/anti-inflammation imbalance in Aβ42 accumulation of rat brain co-exposed to fine particle matter and sulfur dioxide
resolves10.1080/01926230490520232
Brain Inflammation and Alzheimer's-Like Pathology in Individuals Exposed to Severe Air Pollution
The 3 references without a DOI — listed, not checked
no DOI — not checkedHealth Effects Institute (2019) State of Global Air 2019. Special Report. Health Effects Institute, Boston, MA.
no DOI — not checkedWorld Health Organization (2006) WHO Air quality guidelines for particulate matter, ozone, nitrogen dioxide and sulfur dioxide.
no DOI — not checkedInflammatory effects of SO2 inhalation at low concentration in rat cortex
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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