Reference health
Lipopolysaccharide Sensitizes Neonatal Hypoxic-Ischemic Brain Injury in a MyD88-Dependent Manner
not checkable · checked 2026-07-22
This work could not be checked — none of the deposited references carry a DOI. No claim is made either way about its reference list.
53 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 53 references without a DOI — listed, not checked
no DOI — not checkedInflammatory brain damage in preterm newborns: dry numbers, wet lab, and causal inferences.
no DOI — not checkedWhite matter injury after repeated endotoxin exposure in the preterm ovine fetus.
no DOI — not checkedMaturational effects of lipopolysaccharide on white-matter injury in fetal sheep.
no DOI — not checkedEffect of inflammation on central nervous system development and vulnerability.
no DOI — not checkedLipopolysaccharide induces both a primary and a secondary phase of sensitization in the developing rat brain.
no DOI — not checkedBacterial endotoxin sensitizes the immature brain to hypoxic-ischaemic injury.
no DOI — not checkedN-Acetylcysteine reduces lipopolysaccharide-sensitized hypoxic-ischemic brain injury.
no DOI — not checkedDual role of intrauterine immune challenge on neonatal and adult brain vulnerability to hypoxia-ischemia.
no DOI — not checkedToll-like receptor signalling.
no DOI — not checkedThe family of five: TIR-domain-containing adaptors in Toll-like receptor signalling.
no DOI — not checkedActivation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway.
no DOI — not checkedChromatin-IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors.
no DOI — not checkedDisruption of interleukin-18, but not interleukin-1, increases vulnerability to preterm delivery and fetal mortality after intrauterine inflammation.
no DOI — not checkedThe influence of immaturity on hypoxic-ischemic brain damage in the rat.
no DOI — not checkedActivation of ERK1/2 after neonatal rat cerebral hypoxia-ischaemia.
no DOI — not checkedX-linked inhibitor of apoptosis (XIAP) protein protects against caspase activation and tissue loss after neonatal hypoxia-ischemia.
no DOI — not checkedImmediate early gene induction after neonatal hypoxia-ischemia.
no DOI — not checkedDevelopmental changes in the sensitivity of the neonatal rat brain to hypoxic/ischemic injury.
no DOI — not checkedThe nonerythropoietic asialoerythropoietin protects against neonatal hypoxia-ischemia as potently as erythropoietin.
no DOI — not checkedInterleukin-18 involvement in hypoxic-ischemic brain injury.
no DOI — not checkedWhite matter injury in the immature brain: role of interleukin-18.
no DOI — not checkedCaspase-3 activation after neonatal rat cerebral hypoxia-ischemia.
no DOI — not checkedMyD88-dependent signals are essential for the host immune response in experimental brain abscess.
no DOI — not checkedAchieving stability of lipopolysaccharide-induced NF-κB activation.
no DOI — not checkedThe IκB-NF-κB signaling module: temporal control and selective gene activation.
no DOI — not checkedMarked age-dependent neuroprotection by brain-derived neurotrophic factor against neonatal hypoxic-ischemic brain injury.
no DOI — not checkedInvolvement of caspase-3 in cell death after hypoxia-ischemia declines during brain maturation.
no DOI — not checkedThe influence of age on apoptotic and other mechanisms of cell death after cerebral hypoxia-ischemia.
no DOI — not checkedCytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade.
no DOI — not checkedA vicious cycle involving release of heat shock protein 60 from injured cells and activation of Toll-like receptor 4 mediates neurodegeneration in the CNS.
no DOI — not checkedPivotal role for neuronal Toll-like receptors in ischemic brain injury and functional deficits.
no DOI — not checkedAcute brain injury triggers MyD88-dependent, TLR2/4-independent inflammatory responses.
no DOI — not checkedInnate immunity mediated by TLR9 modulates pathogenicity in an animal model of multiple sclerosis.
no DOI — not checkedEffect of lipopolysaccharide on global gene expression in the immature rat brain.
no DOI — not checkedUnresponsiveness of MyD88-deficient mice to endotoxin.
no DOI — not checkedCentral role for MyD88 in the responses of microglia to pathogen-associated molecular patterns.
no DOI — not checkedBrain injury induced by intracerebral injection of interleukin-1β and tumor necrosis factor-α in the neonatal rat.
no DOI — not checkedCyclooxygenase-2 mediates the sensitizing effects of systemic IL-1-β on excitotoxic brain lesions in newborn mice.
no DOI — not checkedAttenuation of hypoxia-ischemia-induced monocyte chemoattractant protein-1 expression in brain of neonatal mice deficient in interleukin-1 converting enzyme.
no DOI — not checkedNeuroinflammation and both cytotoxic and vasogenic edema are reduced in interleukin-1 type 1 receptor-deficient mice conferring neuroprotection.
no DOI — not checkedStrong neuroprotection by inhibition of NF-κB after neonatal hypoxia-ischemia involves apoptotic mechanisms but is independent of cytokines.
no DOI — not checkedA dual role of the NF-κB pathway in neonatal hypoxic-ischemic brain damage.
no DOI — not checkedThe expression and function of chemokines involved in CNS inflammation.
no DOI — not checkedMicroglia: active sensor and versatile effector cells in the normal and pathologic brain.
no DOI — not checkedMicroglia as a source and target of cytokines.
no DOI — not checkedThe protein tyrosine kinase inhibitor AG126 prevents the massive microglial cytokine induction by pneumococcal cell walls.
no DOI — not checkedSelective ablation of proliferating microglial cells exacerbates ischemic injury in the brain.
no DOI — not checkedNeuroprotective effects of resident microglia following acute brain injury.
no DOI — not checkedCaspase inhibitor affords neuroprotection with delayed administration in a rat model of neonatal hypoxic-ischemic brain injury.
no DOI — not checkedHypoxic preconditioning confers long-term reduction of brain injury and improvement of neurological ability in immature rats.
no DOI — not checkedGlobal gene expression in the developing rat brain after hypoxic preconditioning: involvement of apoptotic mechanisms?.
no DOI — not checkedInflammation-induced preconditioning in the immature brain.
no DOI — not checkedCaspase 3 activation is essential for neuroprotection in preconditioning.
What this badge says. CiteStamped means the CHECKABLE references of this
work were clean at the dated check: each resolved to a known work in a public registry, and none
carried a retraction notice at that time. It says nothing about the quality, findings,
or importance of the work itself, and nothing about references deposited without a DOI.
checked 2026-07-22 — re-checked daily as this page is visited; titles and statuses come from Crossref and DataCite and are not part of the signed record
Embed this badge
Both snippets point at the live badge image and link back to this page. The badge re-renders from the daily check, so an embed never goes stale by more than a day of visits.
<a href="https://citestamp.com/citestamped/10.4049/jimmunol.0900762"><img src="https://citestamp.com/citestamped/10.4049/jimmunol.0900762/badge.svg" alt="CiteStamped reference-health badge" width="460" height="64"></a>
[](https://citestamp.com/citestamped/10.4049/jimmunol.0900762)