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Phosphoinositide-3-kinase regulatory subunit 4 participates in the occurrence and development of amyotrophic lateral sclerosis by regulating autophagy
54/54 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
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The 54 checked references that resolve
resolves10.1007/s12035-013-8623-3
Oxidative Stress and Autophagic Alteration in Brainstem of SOD1-G93A Mouse Model of ALS
resolves10.7554/eLife.05115
Architecture and dynamics of the autophagic phosphatidylinositol 3-kinase complex
resolves10.1186/s13024-019-0313-9
Network approach identifies Pacer as an autophagy protein involved in ALS pathogenesis
resolves10.4103/1673-5374.293142
The important functional role of TDP-43 plays in amyotrophic lateral sclerosis-frontotemporal dementia
resolves10.1001/archneurol.2011.250
<emph type="ital">SQSTM1</emph> Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis
resolves10.1128/MCB.00079-13
Role of Membrane Association and Atg14-Dependent Phosphorylation in Beclin-1-Mediated Autophagy
resolves10.1074/jbc.M608787200
p62 Accumulates and Enhances Aggregate Formation in Model Systems of Familial Amyotrophic Lateral Sclerosis
resolves10.1016/j.devcel.2016.04.018
The Autophagy Machinery Controls Cell Death Switching between Apoptosis and Necroptosis
resolves10.1038/s41593-018-0170-9
Publisher Correction: Mutations in Vps15 perturb neuronal migration in mice and are associated with neurodevelopmental disease in humans
resolves10.1038/nature04724
Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice
resolves10.1371/journal.pone.0015445
Characterization of a Novel SOD-1(G93A) Transgenic Mouse Line with Very Decelerated Disease Development
resolves10.1007/s00401-005-1019-3
Redox system expression in the motor neurons in amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS, superoxide dismutase 1 (SOD1)-mutated familial ALS, and SOD1-mutated ALS animal models
resolves10.1038/nature04723
Loss of autophagy in the central nervous system causes neurodegeneration in mice
resolves10.3390/cells8121502
Cytoplasmic Restriction of Mutated SOD1 Impairs the DNA Repair Process in Spinal Cord Neurons
resolves10.7150/ijbs.16158
Changes in the Expression of FUS/TLS in Spinal Cords of SOD1 G93A Transgenic Mice and Correlation with Motor-Neuron Degeneration
resolves10.7150/ijbs.19150
Aldehyde Dehydrogenases 1A2 Expression and Distribution are Potentially Associated with Neuron Death in Spinal Cord of Tg(SOD1*G93A)1Gur Mice
resolves10.7150/ijbs.15938
The Overexpression of TDP-43 Protein in the Neuron and Oligodendrocyte Cells Causes the Progressive Motor Neuron Degeneration in the SOD1 G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis
resolves10.1038/ncb1846
Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages
resolves10.1186/s13041-018-0373-8
Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model
resolves10.4161/auto.28784
Pathogenic role of BECN1/Beclin 1 in the development of amyotrophic lateral sclerosis
resolves10.1002/emmm.201202057
Defects of Vps15 in skeletal muscles lead to autophagic vacuolar myopathy and lysosomal disease
resolves10.1080/15548627.2016.1226736
Characterization of Atg38 and NRBF2, a fifth subunit of the autophagic Vps34/PIK3C3 complex
resolves10.1074/jbc.272.4.2477
Characterization of p150, an Adaptor Protein for the Human Phosphatidylinositol (PtdIns) 3-Kinase
resolves10.1371/journal.pbio.3000410
The ARRIVE guidelines 2.0: Updated guidelines for reporting animal research
resolves10.1038/362059a0
Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis
resolves10.1073/pnas.1704294114
Distinct roles for motor neuron autophagy early and late in the SOD1
<sup>G93A</sup>
mouse model of ALS
resolves10.1080/15548627.2019.1659612
SQSTM1/p62 and PPARGC1A/PGC-1alpha at the interface of autophagy and vascular senescence
resolves10.1097/NEN.0b013e3182160690
Autophagy in Spinal Cord Motor Neurons in Sporadic Amyotrophic Lateral Sclerosis
resolves10.1016/j.neuron.2013.07.027
Neuroprotection through Excitability and mTOR Required in ALS Motoneurons to Delay Disease and Extend Survival
resolves10.1016/j.yexcr.2010.07.008
A phosphatidylinositol 3-kinase class III sub-complex containing VPS15, VPS34, Beclin 1, UVRAG and BIF-1 regulates cytokinesis and degradative endocytic traffic
resolves10.1021/cn500147v
Proteasome Activation is a Mechanism for Pyrazolone Small Molecules Displaying Therapeutic Potential in Amyotrophic Lateral Sclerosis
resolves10.1007/s10571-010-9507-y
The Evolutionarily Conserved Interaction Between LC3 and p62 Selectively Mediates Autophagy-Dependent Degradation of Mutant Huntingtin
resolves10.4103/1673-5374.306065
Considerations on the concept, definition, and diagnosis of amyotrophic lateral sclerosis
resolves10.4103/1673-5374.285004
An appropriate level of autophagy reduces emulsified isoflurane-induced apoptosis in fetal neural stem cells
resolves10.1007/s12035-017-0406-9
Expression and Distribution of Arylsulfatase B are Closely Associated with Neuron Death in SOD1 G93A Transgenic Mice
resolves10.7150/ijbs.26829
Preliminary Observation about Alteration of Proteins and Their Potential Functions in Spinal Cord of SOD1 G93A Transgenic Mice
resolves10.1016/j.bmc.2011.11.039
Cyclohexane 1,3-diones and their inhibition of mutant SOD1-dependent protein aggregation and toxicity in PC12 cells
resolves10.1007/s00401-014-1336-5
Aggregation-prone c9FTD/ALS poly(GA) RAN-translated proteins cause neurotoxicity by inducing ER stress
resolves10.1159/000369466
An Astrocyte Regenerative Response from Vimentin-Containing Cells in the Spinal Cord of Amyotrophic Lateral Sclerosis's Disease-Like Transgenic (G93A SOD1) Mice
The 1 reference without a DOI — listed, not checked
no DOI — not checkedCai B, Fan DS (2013) Germline degradation of a mouse model of familial amyotrophic lateral sclerosis when breeding. Zhongguo Zuzhi Gongcheng Yanjiu 17:4521-4528.
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