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Contribution of non‐reference alleles in mt DNA of Alzheimer's disease patients
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/1531-8249(200008)48:2<148::aid-ana3>3.0.co;2-7openalex-snapshot
Alzheimer's disease cybrids replicate ?-amyloid abnormalities through cell death pathways
inferredcites →https://doi.org/10.1016/j.bbagen.2011.08.008openalex-snapshot
A mitochondrial etiology of Alzheimer and Parkinson disease
inferredcites →https://doi.org/10.1016/j.cell.2012.09.004openalex-snapshot
Heteroplasmy of Mouse mtDNA Is Genetically Unstable and Results in Altered Behavior and Cognition
inferredcites →https://doi.org/10.1016/j.mito.2006.07.002openalex-snapshot
Quantitation of heteroplasmy of mtDNA sequence variants identified in a population of AD patients and controls by array-based resequencing
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2010.04.031openalex-snapshot
Association between mitochondrial DNA variations and Alzheimer's disease in the ADNI cohort
inferredcites →https://doi.org/10.1038/nrg3275openalex-snapshot
Human mitochondrial DNA: roles of inherited and somatic mutations
inferredcites →https://doi.org/10.1073/pnas.0403649101openalex-snapshot
Alzheimer's brains harbor somatic mtDNA control-region mutations that suppress mitochondrial transcription and replication
inferredcites →https://doi.org/10.1089/rej.2005.8.172openalex-snapshot
Mitochondrial Microheteroplasmy and a Theory of Aging and Age-Related Disease
inferredcites →https://doi.org/10.1093/hmg/11.2.133openalex-snapshot
High aggregate burden of somatic mtDNA point mutations in aging and Alzheimer's disease brain
inferredcites →https://doi.org/10.1093/hmg/dds435openalex-snapshot
Universal heteroplasmy of human mitochondrial DNA
inferredcites →https://doi.org/10.1093/jnen/60.8.759openalex-snapshot
Oxidative Damage Is the Earliest Event in Alzheimer Disease
inferredcites →https://doi.org/10.1093/nar/30.1.207openalex-snapshot
Gene Expression Omnibus: NCBI gene expression and hybridization array data repository
inferredcites →https://doi.org/10.1097/01.gim.0000237782.94878.05openalex-snapshot
Chip-based mtDNA mutation screening enables fast and reliable genetic diagnosis of OXPHOS patients
inferredcites →https://doi.org/10.1101/gr.122226.111openalex-snapshot
Analysis of alternative splicing associated with aging and neurodegeneration in the human brain
inferredcites →https://doi.org/10.1101/gr.2228504openalex-snapshot
The Human MitoChip: A High-Throughput Sequencing Microarray for Mitochondrial Mutation Detection
inferredcites →https://doi.org/10.1111/1467-9868.00346openalex-snapshot
A Direct Approach to False Discovery Rates
inferredcites →https://doi.org/10.1186/1471-2105-12-402openalex-snapshot
Mitochondrial genome sequence analysis: A custom bioinformatics pipeline substantially improves Affymetrix MitoChip v2.0 call rate and accuracy
inferredcites →https://doi.org/10.1212/wnl.0b013e31824e8f1dopenalex-snapshot
No consistent evidence for association between mtDNA variants and Alzheimer disease
inferredcites →https://doi.org/10.1523/jneurosci.21-12-04183.2001openalex-snapshot
Increased Lipid Peroxidation Precedes Amyloid Plaque Formation in an Animal Model of Alzheimer Amyloidosis
inferredcites →https://doi.org/10.3233/jad-2006-9210openalex-snapshot
Mitochondrial genomic contribution to mitochondrial dysfunction in Alzheimer's disease
inferredcites →https://doi.org/10.3233/jad-2010-100351openalex-snapshot
Systemic Mitochondrial Dysfunction and the Etiology of Alzheimer's Disease and Down Syndrome Dementia
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4498411openalex-extracted
The Effect of Frailty and Somatic Mutation Burden on the Association between Mitochondrial DNA Copy Number and Dementia Risk
inferred← citeshttps://doi.org/10.1002/alz.12812openalex-snapshot
The role of mitochondrial genome abundance in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70839openalex-snapshot
Mitochondrial haplogroups, but not heteroplasmy, are associated with midlife cognitive function
inferred← citeshttps://doi.org/10.1007/s11357-024-01355-1openalex-snapshot
Mitochondrial DNA copy number associated dementia risk by somatic mutations and frailty
inferred← citeshttps://doi.org/10.1016/bs.pmbts.2016.12.017openalex-snapshot
Mitochondria, Cybrids, Aging, and Alzheimer's Disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2018.09.035openalex-snapshot
Cause or casualty: The role of mitochondrial DNA in aging and age-associated disease
inferred← citeshttps://doi.org/10.1016/j.expneurol.2020.113321openalex-snapshot
Mitochondria in Alzheimer's disease and their potential role in Alzheimer's proteostasis
inferred← citeshttps://doi.org/10.1016/j.mad.2024.112001openalex-snapshot
Convergence between brain aging and Alzheimer’s disease: Focus on mitochondria
inferred← citeshttps://doi.org/10.1021/acschemneuro.2c00512openalex-snapshot
Could Amyloid-β 1–42 or α-Synuclein Interact Directly with Mitochondrial DNA? A Hypothesis
inferred← citeshttps://doi.org/10.1111/ene.70014openalex-snapshot
Mitochondrial DNA ( mtDNA ) as fluid biomarker in neurodegenerative disorders: A systematic review
inferred← citeshttps://doi.org/10.1186/s12883-026-04857-xopenalex-snapshot
Blood mitochondrial heteroplasmic variants and cognitive performance in late midlife: REGARDS study
inferred← citeshttps://doi.org/10.1186/s40246-023-00516-1openalex-snapshot
Mitochondrial genome study in blood of maternally inherited ALS cases
inferred← citeshttps://doi.org/10.14336/ad.2016.0629openalex-snapshot
Biocomplexity and Fractality in the Search of Biomarkers of Aging and Pathology: Focus on Mitochondrial DNA and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.14336/ad.2023.1123openalex-snapshot
Association of Inflammatory Mediators with Mitochondrial DNA Variants in Geriatric COVID-19 Patients
inferred← citeshttps://doi.org/10.3233/jad-221286openalex-snapshot
The Alzheimer’s Disease Mitochondrial Cascade Hypothesis: A Current Overview
inferred← citeshttps://doi.org/10.3233/jad-240092openalex-snapshot
Recent Advances of Mitochondrial Alterations in Alzheimer’s Disease: A Perspective of Mitochondrial Basic Events
inferred← citeshttps://doi.org/10.3389/fnagi.2015.00142openalex-snapshot
Role of diffuse low-level heteroplasmy of mitochondrial DNA in Alzheimer’s disease neurodegeneration
inferred← citeshttps://doi.org/10.3390/fractalfract6040219openalex-snapshot
Mitochondrial DNA Profiling by Fractal Lacunarity to Characterize the Senescent Phenotype as Normal Aging or Pathological Aging
inferred← citeshttps://doi.org/10.6084/m9.figshare.23803160openalex-snapshot
Additional file 1 of Mitochondrial genome study in blood of maternally inherited ALS cases
inferred← citeshttps://doi.org/10.6084/m9.figshare.23803163openalex-snapshot
Additional file 2 of Mitochondrial genome study in blood of maternally inherited ALS cases
inferred← citeshttps://doi.org/10.6084/m9.figshare.23803166openalex-snapshot
Additional file 3 of Mitochondrial genome study in blood of maternally inherited ALS cases
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