On the public citation graph

Severe combined immunodeficiency due to defective binding of the nuclear factor of activated T cells in T lymphocytes of two male siblings

https://doi.org/10.1002/eji.1830260924
0 signed · 167 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/j.1460-2075.1994.tb06329.xopenalex-snapshot
Calcineurin acts in synergy with PMA to inactivate I kappa B/MAD3, an inhibitor of NF‐kappa B.
inferredcites →https://doi.org/10.1016/0092-8674(92)90598-7openalex-snapshot
ZAP-70: A 70 kd protein-tyrosine kinase that associates with the TCR ζ chain
inferredcites →https://doi.org/10.1016/0092-8674(93)90167-oopenalex-snapshot
Interleukin-2 receptor γ chain mutation results in X-linked severe combined immunodeficiency in humans
inferredcites →https://doi.org/10.1016/0092-8674(94)90056-6openalex-snapshot
JNK is involved in signal integration during costimulation of T lymphocytes
inferredcites →https://doi.org/10.1016/0161-5890(87)90014-9openalex-snapshot
Induction, suppression and superinduction of lymphokine mRNA in T lymphocytes
inferredcites →https://doi.org/10.1016/0167-5699(93)90048-popenalex-snapshot
FK506 and cyclosporin, molecular probes for studying intracellular signal transduction
inferredcites →https://doi.org/10.1016/0167-5699(94)90007-8openalex-snapshot
NF-ATp: a transcription factor required for the co-ordinate induction of several cytokine genes
inferredcites →https://doi.org/10.1016/0952-7915(95)80107-3openalex-snapshot
Transcriptional regulation of the IL-2 gene
inferredcites →https://doi.org/10.1016/1074-7613(95)90027-6openalex-snapshot
Isolation of two new members of the NF-AT gene family and functional characterization of the NF-AT proteins
inferredcites →https://doi.org/10.1016/s0021-9258(18)43833-1openalex-snapshot
The role of NFATp in cyclosporin A-sensitive tumor necrosis factor-alpha gene transcription.
inferredcites →https://doi.org/10.1016/s0021-9258(18)53861-8openalex-snapshot
Characterization of the nuclear and cytoplasmic components of the lymphoid-specific nuclear factor of activated T cells (NF-AT) complex.
inferredcites →https://doi.org/10.1016/s0092-8674(05)80090-xopenalex-snapshot
Complementation cloning of an MHC class II transactivator mutated in hereditary MHC class II deficiency (or bare lymphocyte syndrome)
inferredcites →https://doi.org/10.1038/352803a0openalex-snapshot
Nuclear association of a T-cell transcription factor blocked by FK-506 and cyclosporin A
inferredcites →https://doi.org/10.1038/369497a0openalex-snapshot
NF-AT components define a family of transcription factors targeted in T-cell activation
inferredcites →https://doi.org/10.1038/ng0193-77openalex-snapshot
Independent mutations of the human CD3–ε gene resulting in a T cell receptor/CD3 complex immunodeficiency
inferredcites →https://doi.org/10.1056/nejm199006143222406openalex-snapshot
Severe Combined Immunodeficiency Due to a Specific Defect in the Production of Interleukin-2
inferredcites →https://doi.org/10.1056/nejm199208203270805openalex-snapshot
Primary Immunodeficiency Caused by Mutations in the Gene Encoding the CD3-γ Subunit of the T-Lymphocyte Receptor
inferredcites →https://doi.org/10.1073/pnas.87.24.10033openalex-snapshot
Primary combined immunodeficiency resulting from defective transcription of multiple T-cell lymphokine genes.
inferredcites →https://doi.org/10.1073/pnas.90.10.4728openalex-snapshot
Molecular basis of a multiple lymphokine deficiency in a patient with severe combined immunodeficiency.
inferredcites →https://doi.org/10.1084/jem.171.5.1697openalex-snapshot
Absence of interleukin 2 production in a severe combined immunodeficiency disease syndrome with T cells.
inferredcites →https://doi.org/10.1084/jem.178.5.1483openalex-snapshot
Two essential regulatory elements in the human interferon gamma promoter confer activation specific expression in T cells.
inferredcites →https://doi.org/10.1093/hmg/2.8.1099openalex-snapshot
The interleukin-2 receptor γ chain maps to Xq13.1 and is mutated in X-linked severe combined immunodeficiency, SCIDX1
inferredcites →https://doi.org/10.1093/nar/17.15.6419openalex-snapshot
Rapid detection of octamer binding proteins with ‘mini extracts’, prepared from a small number of cells
inferredcites →https://doi.org/10.1101/gad.7.2.188openalex-snapshot
Jun family members are controlled by a calcium-regulated, cyclosporin A-sensitive signaling pathway in activated T lymphocytes.
inferredcites →https://doi.org/10.1126/science.1846244openalex-snapshot
Regulation of Interleukin-2 Gene Enhancer Activity by the T Cell Accessory Molecule CD28
inferredcites →https://doi.org/10.1126/science.2540528openalex-snapshot
Regulation of Lymphokine Messenger RNA Stability by a Surface-Mediated T Cell Activation Pathway
inferredcites →https://doi.org/10.1126/science.272.5263.892openalex-snapshot
An Enhanced Immune Response in Mice Lacking the Transcription Factor NFAT1
inferredcites →https://doi.org/10.1126/science.8202712openalex-snapshot
Human Severe Combined Immunodeficiency Due to a Defect in ZAP-70, a T Cell Tyrosine Kinase
inferredcites →https://doi.org/10.1126/science.8235597openalex-snapshot
Isolation of the Cyclosporin-Sensitive T Cell Transcription Factor NFATp
inferredcites →https://doi.org/10.1128/mcb.12.7.3149openalex-snapshot
Induction of the POU Domain Transcription Factor Oct-2 during T-Cell Activation by Cognate Antigen
inferredcites →https://doi.org/10.1128/mcb.15.11.6299openalex-snapshot
Novel NFAT Sites That Mediate Activation of the Interleukin-2 Promoter in Response to T-Cell Receptor Stimulation
inferredcites →https://doi.org/10.1128/mcb.15.5.2697openalex-snapshot
NFATx, a Novel Member of the Nuclear Factor of Activated T Cells Family That Is Expressed Predominantly in the Thymus
inferredcites →https://doi.org/10.1146/annurev.immunol.4.1.593openalex-snapshot
The Role of the T3/Antigen Receptor Complex in T-Cell Activation
inferredcites →https://doi.org/10.1146/annurev.iy.10.040192.002511openalex-snapshot
Cyclosporin A, FK-506, and Rapamycin: Pharmacologic Probes of Lymphocyte Signal Transduction
inferredcites →https://doi.org/10.4049/jimmunol.133.6.3054openalex-snapshot
Regulation of interleukin 2 receptor expression: effects of phorbol diester, phospholipase C, and reexposure to lectin or antigen.
inferredcites →https://doi.org/10.4049/jimmunol.134.3.1599openalex-snapshot
Activation of a human T cell line: a two-stimulus requirement in the pretranslational events involved in the coordinate expression of interleukin 2 and gamma-interferon genes.
inferredcites →https://doi.org/10.4049/jimmunol.136.12.4503openalex-snapshot
Expression of interleukin 2, interferon-gamma, and the IL 2 receptor by human peripheral blood lymphocytes.
inferredcites →https://doi.org/10.4049/jimmunol.142.1.274openalex-snapshot
Complete nucleotide sequence of the chromosomal gene for human IL-4 and its expression.
inferredcites →https://doi.org/10.4049/jimmunol.146.12.4105openalex-snapshot
CD69 in resting and activated T lymphocytes. Its association with a GTP binding protein and biochemical requirements for its expression
inferredcites →https://doi.org/10.4049/jimmunol.150.8.3284openalex-snapshot
A protein of the AP-1 family is a component of nuclear factor of activated T cells
inferredcites →https://doi.org/10.4049/jimmunol.152.2.429openalex-snapshot
Monocytes provide a novel costimulatory signal to T cells that is not mediated by the CD28/B7 interaction.
inferredcites →https://doi.org/10.4049/jimmunol.153.12.5572openalex-snapshot
Octamer factors exert a dual effect on the IL-2 and IL-4 promoters.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4000894openalex-extracted
Evidence That a Pathogenic Allele in the L-Type Calcium Channel Gene Underlies a Novel X-Linked Primary Immunodeficient Channelopathy
inferred← citeshttps://doi.org/10.1002/eji.201243255openalex-snapshot
Interference with C a2+ release activated C a2+ ( CRAC ) channel function delays T ‐cell arrest in vivo
inferred← citeshttps://doi.org/10.1002/jbmr.3022openalex-snapshot
DLX3-Dependent Regulation of Ion Transporters and Carbonic Anhydrases is Crucial for Enamel Mineralization
inferred← citeshttps://doi.org/10.1002/jcb.29243openalex-snapshot
The critical role of calcineurin/NFAT (C/N) pathways and effective antitumor prospect for colorectal cancers
inferred← citeshttps://doi.org/10.1007/978-3-540-33395-1_22openalex-snapshot
Primary Immunodeficiencies
inferred← citeshttps://doi.org/10.1007/978-3-540-78936-9_2openalex-snapshot
Combined T and B Cell Immunodeficiencies
inferred← citeshttps://doi.org/10.1007/978-3-642-31785-9_2openalex-snapshot
Combined T and B Cell Immunodeficiencies
inferred← citeshttps://doi.org/10.1007/978-3-662-52909-6_2openalex-snapshot
Combined T- and B-Cell Immunodeficiencies
inferred← citeshttps://doi.org/10.1007/978-3-7091-0962-5_19openalex-snapshot
Immune System
inferred← citeshttps://doi.org/10.1007/978-4-431-56068-5_40openalex-snapshot
Elucidation of the Exacerbation Mechanism of Autoimmune Diseases Caused by Disruption of the Ion Homeostasis
inferred← citeshttps://doi.org/10.1007/978-94-007-2888-2_15openalex-snapshot
Store-Operated Ca2+ Entry
inferred← citeshttps://doi.org/10.1007/s00424-009-0777-5openalex-snapshot
CRAC channelopathies
inferred← citeshttps://doi.org/10.1007/s12016-017-8607-6openalex-snapshot
Calcium Signaling: From Normal B Cell Development to Tolerance Breakdown and Autoimmunity.
inferred← citeshttps://doi.org/10.1007/s12026-015-8728-zopenalex-snapshot
Intracellular Ca2+ and related proteins in patients with oral lichen planus
inferred← citeshttps://doi.org/10.1016/b978-0-12-383834-6.00084-7openalex-snapshot
Immunodeficiency Disorders
inferred← citeshttps://doi.org/10.1016/b978-0-12-405546-9.00040-6openalex-snapshot
Mucocutaneous Candidiasis
inferred← citeshttps://doi.org/10.1016/b978-0-12-407870-3.00001-9openalex-snapshot
Store-Operated Orai Channels
inferred← citeshttps://doi.org/10.1016/b978-0-12-800100-4.00004-0openalex-snapshot
Combined Immunodeficiencies with Nonfunctional T Lymphocytes
inferred← citeshttps://doi.org/10.1016/b978-0-323-05659-5.00046-2openalex-snapshot
Primary Immunodeficiency Diseases
inferred← citeshttps://doi.org/10.1016/b978-0-323-08593-9.00073-5openalex-snapshot
Primary Immunodeficiency Diseases
inferred← citeshttps://doi.org/10.1016/b978-0-323-95061-9.00028-xopenalex-snapshot
Infections in primary immunodeficiency
inferred← citeshttps://doi.org/10.1016/b978-3-437-23256-5.16001-1openalex-snapshot
Kapitel E1 Literaturverzeichnis zu Peter, Pichler, Müller-Ladner (Hrsg.): Klinische Immunologie
inferred← citeshttps://doi.org/10.1016/b978-343721311-3.50051-3openalex-snapshot
Störungen der zellularen Immunfunktion
inferred← citeshttps://doi.org/10.1016/bs.ai.2018.02.003openalex-snapshot
Molecular Classification of Primary Immunodeficiencies of T Lymphocytes
inferred← citeshttps://doi.org/10.1016/j.bbrc.2018.04.083openalex-snapshot
Orai1 downregulation impairs lymphocyte function in type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.ceca.2007.03.007openalex-snapshot
Signalling to transcription: Store-operated Ca2+ entry and NFAT activation in lymphocytes
inferred← citeshttps://doi.org/10.1016/j.ceca.2016.12.006openalex-snapshot
Pore opening mechanism of CRAC channels
inferred← citeshttps://doi.org/10.1016/j.ceca.2019.03.004openalex-snapshot
CRAC channels and disease – From human CRAC channelopathies and animal models to novel drugs
inferred← citeshttps://doi.org/10.1016/j.clim.2010.01.011openalex-snapshot
Immunodeficiency due to mutations in ORAI1 and STIM1
inferred← citeshttps://doi.org/10.1016/j.coi.2008.04.004openalex-snapshot
Calcium signaling in lymphocytes
inferred← citeshttps://doi.org/10.1016/j.cub.2006.06.036openalex-snapshot
Calcium Influx: Beyond ‘Current’ Biology
inferred← citeshttps://doi.org/10.1016/j.immuni.2007.11.012openalex-snapshot
Human Primary Immunodeficiency Diseases
inferred← citeshttps://doi.org/10.1016/j.immuni.2017.09.003openalex-snapshot
Store-Operated Ca2+ Entry Controls Clonal Expansion of T Cells through Metabolic Reprogramming
inferred← citeshttps://doi.org/10.1016/j.it.2020.06.012openalex-snapshot
CRAC Channels and Calcium Signaling in T Cell-Mediated Immunity
inferred← citeshttps://doi.org/10.1016/j.jaci.2009.10.007openalex-snapshot
ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia
inferred← citeshttps://doi.org/10.1016/j.jaci.2013.01.020openalex-snapshot
Partial defects of T-cell development associated with poor T-cell function
inferred← citeshttps://doi.org/10.1016/j.jaci.2017.10.031openalex-snapshot
ORAI1 mutations abolishing store-operated Ca2+ entry cause anhidrotic ectodermal dysplasia with immunodeficiency
inferred← citeshttps://doi.org/10.1016/j.jphyss.2025.100013openalex-snapshot
Calcium response via CRAC channels in human synovial cells induced by shear stress in rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.micinf.2009.04.008openalex-snapshot
The calcium/NFAT pathway: role in development and function of regulatory T cells
inferred← citeshttps://doi.org/10.1016/j.molmed.2007.01.004openalex-snapshot
New insights into the molecular mechanisms of store-operated Ca2+ signaling in T cells
inferred← citeshttps://doi.org/10.1016/j.tips.2008.11.005openalex-snapshot
STIM and Orai: the long-awaited constituents of store-operated calcium entry
inferred← citeshttps://doi.org/10.1016/s0171-2985(00)80060-1openalex-snapshot
Impaired NFAT Regulation and its Role in a Severe Combined Immunodeficiency
inferred← citeshttps://doi.org/10.1016/s1016-8478(23)17383-3openalex-snapshot
Regulation of NFAT Activation: a Potential Therapeutic Target for Immunosuppression
inferred← citeshttps://doi.org/10.1016/s1074-7613(00)80188-0openalex-snapshot
Manipulating Immune Responses with Immunosuppressive Agents that Target NFAT
inferred← citeshttps://doi.org/10.1016/s1074-7613(00)80403-3openalex-snapshot
Down-Regulation of IL-4 Gene Transcription and Control of Th2 Cell Differentiation by a Mechanism Involving NFAT1
inferred← citeshttps://doi.org/10.1016/s1155-1984(15)40482-0openalex-snapshot
10.1016/s1155-1984(15)40482-0
inferred← citeshttps://doi.org/10.1023/b:joci.0000010425.30967.0fopenalex-snapshot
Deficient IFN- Expression in Umbilical Cord Blood (UCB) T Cells Can Be Rescued by IFN- -Mediated Increase in NFATc2 Expression
inferred← citeshttps://doi.org/10.1038/86318openalex-snapshot
Gene regulation mediated by calcium signals in T lymphocytes
inferred← citeshttps://doi.org/10.1038/nature04702openalex-snapshot
A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function
inferred← citeshttps://doi.org/10.1038/ncomms5814openalex-snapshot
Loss of IP3R-dependent Ca2+ signalling in thymocytes leads to aberrant development and acute lymphoblastic leukemia
inferred← citeshttps://doi.org/10.1038/ni.f.220openalex-snapshot
Calcium signaling in immune cells
inferred← citeshttps://doi.org/10.1038/ni1550openalex-snapshot
Defective mast cell effector functions in mice lacking the CRACM1 pore subunit of store-operated calcium release–activated calcium channels
inferred← citeshttps://doi.org/10.1038/ni1574openalex-snapshot
Dual functions for the endoplasmic reticulum calcium sensors STIM1 and STIM2 in T cell activation and tolerance
inferred← citeshttps://doi.org/10.1038/nrd3136openalex-snapshot
Store-operated CRAC channels: function in health and disease
inferred← citeshttps://doi.org/10.1038/nri2152openalex-snapshot
Calcium signalling in lymphocyte activation and disease
inferred← citeshttps://doi.org/10.1038/nri2195openalex-snapshot
Immunological and genetic bases of new primary immunodeficiencies
inferred← citeshttps://doi.org/10.1038/nri3233openalex-snapshot
Ion channels and transporters in lymphocyte function and immunity
inferred← citeshttps://doi.org/10.1038/s41392-021-00823-wopenalex-snapshot
T cell receptor (TCR) signaling in health and disease
inferred← citeshttps://doi.org/10.1038/s41423-022-00928-4openalex-snapshot
Disrupted Ca2+ homeostasis and immunodeficiency in patients with functional IP3 receptor subtype 3 defects
inferred← citeshttps://doi.org/10.1038/s41577-018-0110-7openalex-snapshot
Calcium signalling in T cells
inferred← citeshttps://doi.org/10.1051/medsci/2011278016openalex-snapshot
Immunodéficiences et pathologies associées aux mutations dans STIM/ORAI
inferred← citeshttps://doi.org/10.1073/pnas.0707798104openalex-snapshot
Mitochondria control calcium entry at the immunological synapse
inferred← citeshttps://doi.org/10.1073/pnas.1013285108openalex-snapshot
ORAI1-mediated calcium influx is required for human cytotoxic lymphocyte degranulation and target cell lysis
inferred← citeshttps://doi.org/10.1074/jbc.m113.450254openalex-snapshot
Differential Roles of the C and N Termini of Orai1 Protein in Interacting with Stromal Interaction Molecule 1 (STIM1) for Ca2+ Release-activated Ca2+ (CRAC) Channel Activation
inferred← citeshttps://doi.org/10.1074/jbc.m806726200openalex-snapshot
STIM1- and Orai1-dependent Store-operated Calcium Entry Regulates Human Myoblast Differentiation
inferred← citeshttps://doi.org/10.1074/jbc.m808346200openalex-snapshot
The Orai1 Severe Combined Immune Deficiency Mutation and Calcium Release-activated Ca2+ Channel Function in the Heterozygous Condition
inferred← citeshttps://doi.org/10.1080/17402520600800705openalex-snapshot
Current Perspectives on Primary Immunodeficiency Diseases
inferred← citeshttps://doi.org/10.1084/jem.20011758openalex-snapshot
Transient Receptor Potential 1 Regulates Capacitative Ca2+ Entry and Ca2+ Release from Endoplasmic Reticulum in B Lymphocytes
inferred← citeshttps://doi.org/10.1084/jem.20050687openalex-snapshot
A severe defect in CRAC Ca2+ channel activation and altered K+ channel gating in T cells from immunodeficient patients
inferred← citeshttps://doi.org/10.1101/2021.05.29.21257775openalex-snapshot
Disrupted Ca 2+ homeostasis and immunodeficiency in patients with functional Inositol 1,4,5-trisphosphate receptor subtype 3 defects
inferred← citeshttps://doi.org/10.1101/864280openalex-snapshot
Mutation of an L-Type Calcium Channel Gene Leads to a Novel Human Primary Cellular Immunodeficiency
inferred← citeshttps://doi.org/10.1111/cei.13488openalex-snapshot
Emerging patterns of regulatory T cell function in tuberculosis
inferred← citeshttps://doi.org/10.1111/j.1582-4934.2010.01097.xopenalex-snapshot
STIM proteins: integrators of signalling pathways in development, differentiation and disease
inferred← citeshttps://doi.org/10.1111/j.1582-4934.2011.01395.xopenalex-snapshot
Orais and STIMs: physiological mechanisms and disease
inferred← citeshttps://doi.org/10.1111/j.1600-065x.2009.00818.xopenalex-snapshot
ORAI1 and STIM1 deficiency in human and mice: roles of store‐operated Ca 2+ entry in the immune system and beyond
inferred← citeshttps://doi.org/10.1111/j.1600-065x.2009.00819.xopenalex-snapshot
Calcium signaling in the development and function of T‐lineage cells
inferred← citeshttps://doi.org/10.1111/j.1749-6632.2011.06240.xopenalex-snapshot
Immunodeficiency due to defects in store‐operated calcium entry
inferred← citeshttps://doi.org/10.1111/nyas.12938openalex-snapshot
Diseases caused by mutations in ORAI1 and STIM1
inferred← citeshttps://doi.org/10.1113/jphysiol.2012.233221openalex-snapshot
Regulation of lymphocyte function by ORAI and STIM proteins in infection and autoimmunity
inferred← citeshttps://doi.org/10.1124/mi.10.4.4openalex-snapshot
Pharmacology of Store-operated Calcium Channels
inferred← citeshttps://doi.org/10.1128/mcb.00360-08openalex-snapshot
Hair Loss and Defective T- and B-Cell Function in Mice Lacking ORAI1
inferred← citeshttps://doi.org/10.1146/annurev-immunol-032414-112212openalex-snapshot
Ion Channels in Innate and Adaptive Immunity
inferred← citeshttps://doi.org/10.1146/annurev-immunol-032712-095927openalex-snapshot
Functional T Cell Immunodeficiencies (with T Cells Present)
inferred← citeshttps://doi.org/10.1146/annurev-physiol-052521-013426openalex-snapshot
Physiological Functions of CRAC Channels
inferred← citeshttps://doi.org/10.1146/annurev.immunol.15.1.93openalex-snapshot
NATURALLY OCCURRING PRIMARY DEFICIENCIES OF THE IMMUNE SYSTEM
inferred← citeshttps://doi.org/10.1159/000289196openalex-snapshot
Transcriptional Control of the TNF Gene
inferred← citeshttps://doi.org/10.1172/jci131935openalex-snapshot
Orai1: CRACing the Th17 response in AKI
inferred← citeshttps://doi.org/10.1172/jci76602openalex-snapshot
CD4+ and CD8+ T cell–dependent antiviral immunity requires STIM1 and STIM2
inferred← citeshttps://doi.org/10.1172/jci80273openalex-snapshot
STIM1 controls T cell–mediated immune regulation and inflammation in chronic infection
inferred← citeshttps://doi.org/10.1177/0009922815574079openalex-snapshot
Primary Immunodeficiency Diseases: An Opportunity in Pediatrics for Improving Patient Outcomes.
inferred← citeshttps://doi.org/10.1177/1756285613487782openalex-snapshot
Targeting ion channels for the treatment of autoimmune neuroinflammation
inferred← citeshttps://doi.org/10.1177/25152564241291374openalex-snapshot
Store-Operated Ca 2+ Entry in Fibrosis and Tissue Remodeling
inferred← citeshttps://doi.org/10.1182/blood-2015-03-631374openalex-snapshot
Omenn syndrome associated with a functional reversion due to a somatic second-site mutation in CARD11 deficiency
inferred← citeshttps://doi.org/10.1182/blood.v98.5.1480openalex-snapshot
Regulation of interferon-γ gene expression by nuclear factor of activated T cells
inferred← citeshttps://doi.org/10.1189/jlb.0404259openalex-snapshot
Expression and regulation of NFAT (nuclear factors of activated T cells) in human CD34+ cells: down-regulation upon myeloid differentiation
inferred← citeshttps://doi.org/10.1201/9781315152592-6openalex-snapshot
Function of Orai/Stim Proteins Studied in Transgenic Animal Models
inferred← citeshttps://doi.org/10.1242/jcs.264681openalex-snapshot
DHHC21 is a STIM1 protein S-acyltransferase that modulates immune function in vivo
inferred← citeshttps://doi.org/10.1371/journal.pone.0015090openalex-snapshot
Calpain 2 Controls Turnover of LFA-1 Adhesions on Migrating T Lymphocytes
inferred← citeshttps://doi.org/10.1371/journal.pone.0082944openalex-snapshot
Antibody-Mediated Targeting of the Orai1 Calcium Channel Inhibits T Cell Function
inferred← citeshttps://doi.org/10.1517/14728222.12.4.391openalex-snapshot
New therapeutic targets in immune disorders: ItpkB, Orai1 and UNC93B
inferred← citeshttps://doi.org/10.15252/emmm.201911592openalex-snapshot
STIM1‐mediated calcium influx controls antifungal immunity and the metabolic function of non‐pathogenic Th17 cells
inferred← citeshttps://doi.org/10.17638/00001482openalex-snapshot
The regulation of STIM1 translocation to the plasma membrane
inferred← citeshttps://doi.org/10.2741/540openalex-snapshot
Physiological and pathophysiological functions of SOCE in the immune system
inferred← citeshttps://doi.org/10.2741/e540openalex-snapshot
Physiological and pathophysiological functions of SOCE in the immune system
inferred← citeshttps://doi.org/10.3389/fimmu.2013.00099openalex-snapshot
Ca2+ Influx in T Cells: How Many Ca2+ Channels?
inferred← citeshttps://doi.org/10.3389/fncel.2025.1627305openalex-snapshot
Neuromodulation influences T lymphocyte calcium signaling and alpha synuclein clearance: implications for Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fphys.2020.604941openalex-snapshot
STIM1/ORAI1 Loss-of-Function and Gain-of-Function Mutations Inversely Impact on SOCE and Calcium Homeostasis and Cause Multi-Systemic Mirror Diseases
inferred← citeshttps://doi.org/10.3389/fphys.2023.1130175openalex-snapshot
Amelogenesis imperfecta: Next-generation sequencing sheds light on Witkop’s classification
inferred← citeshttps://doi.org/10.3389/fphys.2023.1330259openalex-snapshot
Regulatory mechanisms controlling store-operated calcium entry
inferred← citeshttps://doi.org/10.3390/cells11111849openalex-snapshot
Science CommuniCa2+tion Developing Scientific Literacy on Calcium: The Involvement of CRAC Currents in Human Health and Disease
inferred← citeshttps://doi.org/10.3390/cells13060468openalex-snapshot
Synthetic Biology Meets Ca2+ Release-Activated Ca2+ Channel-Dependent Immunomodulation
inferred← citeshttps://doi.org/10.3390/ijms26010383openalex-snapshot
The Molecular Biology of Placental Transport of Calcium to the Human Foetus
inferred← citeshttps://doi.org/10.4049/jimmunol.0802888openalex-snapshot
STIM1-Independent T Cell Development and Effector Function In Vivo
inferred← citeshttps://doi.org/10.4049/jimmunol.1001796openalex-snapshot
Store-Operated Ca2+ Entry through ORAI1 Is Critical for T Cell-Mediated Autoimmunity and Allograft Rejection
inferred← citeshttps://doi.org/10.4049/jimmunol.1102507openalex-snapshot
Antiviral and Regulatory T Cell Immunity in a Patient with Stromal Interaction Molecule 1 Deficiency
inferred← citeshttps://doi.org/10.4049/jimmunol.1400285openalex-snapshot
6-Methoxyflavone Inhibits NFAT Translocation into the Nucleus and Suppresses T Cell Activation
inferred← citeshttps://doi.org/10.4049/jimmunol.165.1.297openalex-snapshot
The Duration of Nuclear Residence of NFAT Determines the Pattern of Cytokine Expression in Human SCID T Cells
inferred← citeshttps://doi.org/10.4049/jimmunol.1700192openalex-snapshot
Efficiency and Safety of CRAC Inhibitors in Human Rheumatoid Arthritis Xenograft Models
inferred← citeshttps://doi.org/10.4049/jimmunol.1800837openalex-snapshot
What’s Bred in the Bone: Calcium Channels in Lymphocytes
inferred← citeshttps://doi.org/10.4049/jimmunol.1901065openalex-snapshot
Ca2+–Calcineurin Axis–Controlled NFAT Nuclear Translocation Is Crucial for Optimal T Cell Immunity in an Early Vertebrate
inferred← citeshttps://doi.org/10.4161/chan.24302openalex-snapshot
Emerging roles of store-operated Ca2+entry through STIM and ORAI proteins in immunity, hemostasis and cancer
inferred← citeshttps://doi.org/10.4161/chan.25292openalex-snapshot
Molecular pharmacology of store-operated CRAC channels
inferred← citeshttps://doi.org/10.4161/chan.26156openalex-snapshot
Exploring the unique features of the ARC channel, a Store-independent Orai channel
inferred← citeshttps://doi.org/10.4172/2155-9899.s12-005openalex-snapshot
T Cell Co-inhibitory Receptors-Functions and Signalling Mechanisms
inferred← citeshttps://doi.org/10.70675/b096c0a2zef1bz46bfz9e9cz962e8f4cf345openalex-snapshot
Impact de la delphinidine sur les fonctions de lymphocytes T chez les sujets sains et les patients atteints de syndrome métabolique
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.