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IL‐17 mediates estrogen‐deficient osteoporosis in an Act1‐dependent manner
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/art.10816openalex-snapshot
Interleukin‐17 in rheumatoid arthritis: If T cells were to contribute to inflammation and destruction through synergy
inferredcites →https://doi.org/10.1002/eji.200939670openalex-snapshot
A bone‐protective role for IL‐17 receptor signaling in ovariectomy‐induced bone loss
inferredcites →https://doi.org/10.1002/jbmr.5650101011openalex-snapshot
Inhomogeneity in body fat distribution may result in inaccuracy in the measurement of vertebral bone mass
inferredcites →https://doi.org/10.1016/0169-6009(90)90101-kopenalex-snapshot
Influence of fat on bone measurements with dual-energy absorptiometry
inferredcites →https://doi.org/10.1016/j.bcp.2009.03.008openalex-snapshot
Interleukin-17A inhibits adipocyte differentiation in human mesenchymal stem cells and regulates pro-inflammatory responses in adipocytes
inferredcites →https://doi.org/10.1016/j.bone.2008.02.017openalex-snapshot
Estrogen deficiency increases osteoclastogenesis up-regulating T cells activity: A key mechanism in osteoporosis
inferredcites →https://doi.org/10.1016/j.cyto.2007.12.001openalex-snapshot
Associations of interleukin-6 with interleukin-1β, interleukin-8 and macrophage inflammatory protein-1β in midlife women
inferredcites →https://doi.org/10.1016/j.devcel.2011.08.016openalex-snapshot
Autophagy Proteins Regulate the Secretory Component of Osteoclastic Bone Resorption
inferredcites →https://doi.org/10.1016/s0092-8674(00)00126-4openalex-snapshot
Activation of the IκB Kinase Complex by TRAF6 Requires a Dimeric Ubiquitin-Conjugating Enzyme Complex and a Unique Polyubiquitin Chain
inferredcites →https://doi.org/10.1016/s0968-0004(03)00067-7openalex-snapshot
The STIR-domain superfamily in signal transduction, development and immunity
inferredcites →https://doi.org/10.1038/35085597openalex-snapshot
TAK1 is a ubiquitin-dependent kinase of MKK and IKK
inferredcites →https://doi.org/10.1038/cdd.2009.74openalex-snapshot
IL-17 stimulates the proliferation and differentiation of human mesenchymal stem cells: implications for bone remodeling
inferredcites →https://doi.org/10.1038/ijo.2008.216openalex-snapshot
Increased activity of interleukin-23/interleukin-17 proinflammatory axis in obese women
inferredcites →https://doi.org/10.1038/ni1439openalex-snapshot
The adaptor Act1 is required for interleukin 17–dependent signaling associated with autoimmune and inflammatory disease
inferredcites →https://doi.org/10.1038/nri2586openalex-snapshot
Structure and signalling in the IL-17 receptor family
inferredcites →https://doi.org/10.1073/pnas.0611589104openalex-snapshot
Distinct functional motifs within the IL-17 receptor regulate signal transduction and target gene expression
inferredcites →https://doi.org/10.1073/pnas.251534698openalex-snapshot
Up-regulation of TNF-producing T cells in the bone marrow: A key mechanism by which estrogen deficiency induces bone loss in vivo
inferredcites →https://doi.org/10.1074/jbc.274.12.7724openalex-snapshot
Activation of NF-κB by RANK Requires Tumor Necrosis Factor Receptor-associated Factor (TRAF) 6 and NF-κB-inducing Kinase
inferredcites →https://doi.org/10.1074/jbc.m110.121418openalex-snapshot
SEF/IL-17R (SEFIR) Is Not Enough
inferredcites →https://doi.org/10.1084/jem.20061775openalex-snapshot
Th17 functions as an osteoclastogenic helper T cell subset that links T cell activation and bone destruction
inferredcites →https://doi.org/10.1084/jem.20071133openalex-snapshot
IL-6–dependent spontaneous proliferation is required for the induction of colitogenic IL-17–producing CD8+ T cells
inferredcites →https://doi.org/10.1097/bor.0b013e328277ef2aopenalex-snapshot
Estrogen and CD4+ T cells
inferredcites →https://doi.org/10.1126/scisignal.2000382openalex-snapshot
Act1, a U-box E3 Ubiquitin Ligase for IL-17 Signaling
inferredcites →https://doi.org/10.1126/scisignal.2001843openalex-snapshot
A CC′ Loop Decoy Peptide Blocks the Interaction Between Act1 and IL-17RA to Attenuate IL-17– and IL-25–Induced Inflammation
inferredcites →https://doi.org/10.1126/stke.3982007re4openalex-snapshot
A Role for the Cytoplasmic Adaptor Protein Act1 in Mediating IL-17 Signaling
inferredcites →https://doi.org/10.1172/jci11066openalex-snapshot
Estrogen deficiency induces bone loss by enhancing T-cell production of TNF-α
inferredcites →https://doi.org/10.1172/jci11176openalex-snapshot
TNF-α induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligand
inferredcites →https://doi.org/10.1172/jci119333openalex-snapshot
Transgenic mice expressing soluble tumor necrosis factor-receptor are protected against bone loss caused by estrogen deficiency.
inferredcites →https://doi.org/10.1172/jci200523394openalex-snapshot
IL-1 mediates TNF-induced osteoclastogenesis
inferredcites →https://doi.org/10.1186/ar2936openalex-snapshot
Interleukin-17A upregulates receptor activator of NF-κB on osteoclast precursors
inferredcites →https://doi.org/10.1210/er.2007-0001openalex-snapshot
The Complex Role of Estrogens in Inflammation
inferredcites →https://doi.org/10.1210/jc.2006-1570openalex-snapshot
Timing of Peak Bone Mass: Discrepancies between CT and DXA
inferredcites →https://doi.org/10.1259/bjr.72.859.10624323openalex-snapshot
Anomalies in dual energy X-ray absorptiometry measurements of total-body bone mineral during weight change using Lunar, Hologic and Norland instruments.
inferredcites →https://doi.org/10.1359/jbmr.060726openalex-snapshot
T Lymphocyte–Deficient Mice Lose Trabecular Bone Mass With Ovariectomy
inferredcites →https://doi.org/10.1359/jbmr.1997.12.11.1908openalex-snapshot
Anomalies in the Measurement of Changes in Total-Body Bone Mineral by Dual-Energy X-Ray Absorptiometry During Weight Change
inferredcites →https://doi.org/10.1359/jbmr.1997.12.6.935openalex-snapshot
The Functional Block of TNF but Not of IL-6 Prevents Bone Loss in Ovariectomized Mice
inferredcites →https://doi.org/10.4049/jimmunol.167.2.1004openalex-snapshot
IL-1-Independent Role of IL-17 in Synovial Inflammation and Joint Destruction During Collagen-Induced Arthritis
inferredcites →https://doi.org/10.4049/jimmunol.171.11.6173openalex-snapshot
Suppression of Immune Induction of Collagen-Induced Arthritis in IL-17-Deficient Mice
inferredcites →https://doi.org/10.4049/jimmunol.171.12.6936openalex-snapshot
Estrogen Receptor α Mediates Estrogen’s Immune Protection in Autoimmune Disease
inferredcites →https://doi.org/10.4049/jimmunol.173.8.4838openalex-snapshot
Marrow Stromal Cells and Osteoclast Precursors Differentially Contribute to TNF-α-Induced Osteoclastogenesis In Vivo
inferredcites →https://doi.org/10.4049/jimmunol.176.10.6262openalex-snapshot
Interleukin-17 Acts Independently of TNF-α under Arthritic Conditions
inferredcites →https://doi.org/10.4049/jimmunol.182.3.1631openalex-snapshot
The Critical Role of Epithelial-Derived Act1 in IL-17- and IL-25-Mediated Pulmonary Inflammation
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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IL-17A modulates osteoclast precursors' apoptosis through autophagy-TRAF3 signaling during osteoclastogenesis
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Recent genetic advances in innate immunity of psoriatic arthritis
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IL-17A Is Increased in Humans with Primary Hyperparathyroidism and Mediates PTH-Induced Bone Loss in Mice
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Elucidating common biomarkers and pathways of osteoporosis and aortic valve calcification: insights into new therapeutic targets
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Chronic skin inflammation leads to bone loss by IL-17–mediated inhibition of Wnt signaling in osteoblasts
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Ovariectomy induces bone loss via microbial-dependent trafficking of intestinal TNF+ T cells and Th17 cells
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Callus γδ T cells and microbe-induced intestinal Th17 cells improve fracture healing in mice
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IL-17A deficiency promotes periosteal bone formation in a model of inflammatory arthritis
inferred← citeshttps://doi.org/10.1186/s43166-021-00083-0openalex-snapshot
Serum interleukin-17 and estradiol levels in postmenopausal women in relation to osteoporosis
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A Study to Explore the Correlation between Immune Levels and Osteoporosis in Postmenopausal Women
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Functional roles of immune cells in osteoporosis
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Rewiring osteoimmune homeostasis in osteoporosis: T-cell subsets as key regulators and emerging therapeutic targets
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Estrogen Receptor Downregulates Expression of PD-1/PD-L1 and Infiltration of CD8+ T Cells by Inhibiting IL-17 Signaling Transduction in Breast Cancer
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Gut microbiota-immune-bone axis in osteoporosis: mechanisms and intervention strategies
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Mechanistic Insights and Therapeutic Strategies in Osteoporosis: A Comprehensive Review
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Immunological Aspects of the Tumor Microenvironment and Epithelial-Mesenchymal Transition in Gastric Carcinogenesis
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DKK-1 Is Underexpressed in Mesenchymal Stem Cells from Patients with Ankylosing Spondylitis and Further Downregulated by IL-17
inferred← citeshttps://doi.org/10.3390/metabo16080556openalex-snapshot
Immunometabolic Remodeling in Osteosarcopenia: Inflammaging, Mitochondrial Dysfunction, Gut-Derived Metabolites and Therapeutic Opportunities
inferred← citeshttps://doi.org/10.4274/tod.65982openalex-snapshot
The Relationship Between Interleukin-17 and Osteoporosis in Patients with Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.55453/rjmm.2026.129.5.5openalex-snapshot
Dental Implants in Postmenopausal Women: Biological Considerations, Clinical Outcomes, and Treatment Guidelines
inferred← citeshttps://doi.org/10.5772/intechopen.100904openalex-snapshot
Vitamin D Deficiency - New Insights
inferred← citeshttps://doi.org/10.5772/intechopen.106716openalex-snapshot
Hypovitaminosis D in Postmenopause
inferred← citeshttps://doi.org/10.5772/intechopen.97549openalex-snapshot
Osteoporosis: A Multifactorial Disease
inferred← citeshttps://doi.org/10.7150/jca.10695openalex-snapshot
Clinical Significance of Female-hormones and Cytokines in Breast Cancer Patients Complicated with Aromatase Inhibitor-related Osteoarthropathy - Efficacy of Vitamin E
inferred← citeshttps://doi.org/10.7936/k7kp809gopenalex-snapshot
Single-chain RANKL Provides Insights into RANK Receptor Oligomerization and a Novel Approach to Osteoclast Inhibition
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