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miR‐219‐5p inhibits tau phosphorylation by targeting TTBK1 and GSK‐3β in Alzheimer's disease
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2 of the 56 references listed on this page are in the Retraction Watch database as retracted (export 2026-11-04): 10.1002/jcb.26700 (notice 2021-06-30), 10.26355/eurrev_201711_13813 (notice 2020-08-01).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/jcb.26700openalex-snapshot
Retracted: Protective effects of microRNA‐330 on amyloid β‐protein production, oxidative stress, and mitochondrial dysfunction in Alzheimer's disease by targeting VAV1 via the MAPK signaling pathway
inferredcites →https://doi.org/10.1007/s00401-010-0756-0openalex-snapshot
Patterns of microRNA expression in normal and early Alzheimer’s disease human temporal cortex: white matter versus gray matter
inferredcites →https://doi.org/10.1016/j.biopha.2016.04.052openalex-snapshot
MiR-769 promoted cell proliferation in human melanoma by suppressing GSK3B expression
inferredcites →https://doi.org/10.1016/j.biopha.2016.07.010openalex-snapshot
miR-1303 promotes the proliferation of neuroblastoma cell SH-SY5Y by targeting GSK3β and SFRP1
inferredcites →https://doi.org/10.1016/j.biopha.2018.01.086openalex-snapshot
miR-1301-3p promotes prostate cancer stem cell expansion by targeting SFRP1 and GSK3β
inferredcites →https://doi.org/10.1016/j.biopsych.2017.07.023openalex-snapshot
A Novel MicroRNA-124/PTPN1 Signal Pathway Mediates Synaptic and Memory Deficits in Alzheimer’s Disease
inferredcites →https://doi.org/10.1016/j.brainres.2018.08.015openalex-snapshot
Circulating APP, NCAM and Aβ serve as biomarkers for Alzheimer’s disease
inferredcites →https://doi.org/10.1016/j.cell.2009.01.002openalex-snapshot
MicroRNAs: Target Recognition and Regulatory Functions
inferredcites →https://doi.org/10.1016/j.cell.2012.02.005openalex-snapshot
MicroRNAs in Stress Signaling and Human Disease
inferredcites →https://doi.org/10.1016/j.febslet.2012.02.017openalex-snapshot
MiR‐219‐5p inhibits hepatocellular carcinoma cell proliferation by targeting glypican‐3
inferredcites →https://doi.org/10.1016/j.febslet.2015.02.001openalex-snapshot
MicroRNA‐138 promotes tau phosphorylation by targeting retinoic acid receptor alpha
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2018.06.038openalex-snapshot
Plasma lipid peroxidation biomarkers for early and non-invasive Alzheimer Disease detection
inferredcites →https://doi.org/10.1016/j.jnutbio.2017.02.020openalex-snapshot
miR-19 targeting of GSK3β mediates sulforaphane suppression of lung cancer stem cells
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2010.04.034openalex-snapshot
MicroRNA-16 targets amyloid precursor protein to potentially modulate Alzheimer's-associated pathogenesis in SAMP8 mice
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2014.07.035openalex-snapshot
Cross talk between PI3K-AKT-GSK-3β and PP2A pathways determines tau hyperphosphorylation
inferredcites →https://doi.org/10.1016/j.neuropharm.2012.06.050openalex-snapshot
NGF promotes long-term memory formation by activating poly(ADP-ribose)polymerase-1
inferredcites →https://doi.org/10.1016/j.yexcr.2017.02.033openalex-snapshot
MiR-26a enhances invasive capacity by suppressing GSK3β in human lung cancer cells
inferredcites →https://doi.org/10.1038/mtna.2014.47openalex-snapshot
Mir-34: A New Weapon Against Cancer?
inferredcites →https://doi.org/10.1038/nature02621openalex-snapshot
Pathways towards and away from Alzheimer's disease
inferredcites →https://doi.org/10.1038/nrg2290openalex-snapshot
Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight?
inferredcites →https://doi.org/10.1042/bsr20180051openalex-snapshot
miR-15b reduces amyloid-β accumulation in SH-SY5Y cell line through targetting NF-κB signaling and BACE1
inferredcites →https://doi.org/10.1073/pnas.0710263105openalex-snapshot
Loss of microRNA cluster miR-29a/b-1 in sporadic Alzheimer's disease correlates with increased BACE1/β-secretase expression
inferredcites →https://doi.org/10.1074/jbc.m113.518241openalex-snapshot
MicroRNA-339-5p Down-regulates Protein Expression of β-Site Amyloid Precursor Protein-Cleaving Enzyme 1 (BACE1) in Human Primary Brain Cultures and Is Reduced in Brain Tissue Specimens of Alzheimer Disease Subjects
inferredcites →https://doi.org/10.1074/jbc.m206236200openalex-snapshot
Glycogen Synthase Kinase 3β Phosphorylates Tau at Both Primed and Unprimed Sites
inferredcites →https://doi.org/10.1080/1354750x.2018.1468825openalex-snapshot
Comparison of β2-microglobulin serum level between Alzheimer’s patients, cognitive healthy and mild cognitive impaired individuals
inferredcites →https://doi.org/10.1093/emboj/20.1.27openalex-snapshot
Decreased nuclear β‐catenin, tau hyperphosphorylation and neurodegeneration in GSK‐3β conditional transgenic mice
inferredcites →https://doi.org/10.1111/cas.13338openalex-snapshot
micro RNA ‐219‐5p inhibits epithelial‐mesenchymal transition and metastasis of colorectal cancer by targeting lymphoid enhancer‐binding factor 1
inferredcites →https://doi.org/10.1111/j.1471-4159.2006.04059.xopenalex-snapshot
Tau‐tubulin kinase 1 (TTBK1), a neuron‐specific tau kinase candidate, is involved in tau phosphorylation and aggregation
inferredcites →https://doi.org/10.1111/j.1471-4159.2006.04139.xopenalex-snapshot
Chronic lithium administration to FTDP‐17 tau and GSK‐3β overexpressing mice prevents tau hyperphosphorylation and neurofibrillary tangle formation, but pre‐formed neurofibrillary tangles do not revert
inferredcites →https://doi.org/10.1111/jnc.12437openalex-snapshot
Alternative polyadenylation and miR‐34 family members regulate tau expression
inferredcites →https://doi.org/10.1126/science.1141736openalex-snapshot
Reducing Endogenous Tau Ameliorates Amyloid ß-Induced Deficits in an Alzheimer's Disease Mouse Model
inferredcites →https://doi.org/10.1155/2017/5806146openalex-snapshot
miR-135b Plays a Neuroprotective Role by Targeting GSK3β in MPP+-Intoxicated SH-SY5Y Cells
inferredcites →https://doi.org/10.1159/000091044openalex-snapshot
Association between Glycogen Synthase Kinase-3β Genetic Polymorphism and Late-Onset Alzheimer’s Disease
inferredcites →https://doi.org/10.1186/s13287-017-0551-0openalex-snapshot
microRNAs: important regulators of stem cells
inferredcites →https://doi.org/10.1371/journal.pone.0009764openalex-snapshot
Synaptic Plasticity in the Hippocampus of a APP/PS1 Mouse Model of Alzheimer's Disease Is Impaired in Old but Not Young Mice
inferredcites →https://doi.org/10.1371/journal.pone.0063164openalex-snapshot
miR-219-5p Inhibits Receptor Tyrosine Kinase Pathway by Targeting EGFR in Glioblastoma
inferredcites →https://doi.org/10.1515/bmc-2016-0014openalex-snapshot
Alzheimer’s disease: presence and role of microRNAs
inferredcites →https://doi.org/10.1523/jneurosci.1165-14.2014openalex-snapshot
Synaptic and Cognitive Improvements by Inhibition of 2-AG Metabolism Are through Upregulation of MicroRNA-188-3p in a Mouse Model of Alzheimer's Disease
inferredcites →https://doi.org/10.15252/embj.201387576openalex-snapshot
MicroRNA‐125b induces tau hyperphosphorylation and cognitive deficits in Alzheimer's disease
inferredcites →https://doi.org/10.18632/oncotarget.12861openalex-snapshot
MicroRNA-101 reverses temozolomide resistance by inhibition of GSK3β in glioblastoma
inferredcites →https://doi.org/10.18632/oncotarget.14529openalex-snapshot
MiR-410 induces stemness by inhibiting Gsk3β but upregulating β-catenin in non-small cells lung cancer
inferredcites →https://doi.org/10.26355/eurrev_201711_13813openalex-snapshot
MiR-155 affects renal carcinoma cell proliferation, invasion and apoptosis through regulating GSK-3β/β-catenin signaling pathway.
inferredcites →https://doi.org/10.3233/jad-180364openalex-snapshot
Circulating miR-127-3p as a Potential Biomarker for Differential Diagnosis in Frontotemporal Dementia
inferredcites →https://doi.org/10.3233/jad-2010-091603openalex-snapshot
MiR-107 is Reduced in Alzheimer's Disease Brain Neocortex: Validation Study
inferredcites →https://doi.org/10.3233/jad-2010-101674openalex-snapshot
Homeostatic Disinhibition in the Aging Brain and Alzheimer's Disease
inferredcites →https://doi.org/10.3233/jin-170032openalex-snapshot
2-(2-benzofuranyl)-2-imidazoline (2-BFI) improved the impairments in AD rat models by inhibiting oxidative stress, inflammation and apoptosis
inferredcites →https://doi.org/10.3389/fncel.2014.00037openalex-snapshot
A lentiviral sponge for miR-101 regulates RanBP9 expression and amyloid precursor protein metabolism in hippocampal neurons
inferredcites →https://doi.org/10.3389/fphys.2015.00040openalex-snapshot
The emerging role of microRNAs in Alzheimer's disease
inferredcites →https://doi.org/10.3727/096504016x14768374457986openalex-snapshot
MicroRNA-219-5p Represses the Proliferation, Migration, and Invasion of Gastric Cancer Cells by Targeting the LRH-1/Wnt/β-Catenin Signaling Pathway
inferredcites →https://doi.org/10.3727/096504017x14902261600566openalex-snapshot
MicroRNA-940 Targets INPP4A or GSK3β and Activates the Wnt/β-Catenin Pathway to Regulate the Malignant Behavior of Bladder Cancer Cells
inferredcites →https://doi.org/10.3727/096504018x15213142076069openalex-snapshot
miR-632 Promotes Laryngeal Carcinoma Cell Proliferation, Migration, and Invasion Through Negative Regulation of GSK3β
inferredcites →https://doi.org/10.3892/ijo.2015.3135openalex-snapshot
miRNA-99b-3p functions as a potential tumor suppressor by targeting glycogen synthase kinase-3β in oral squamous cell carcinoma Tca-8113 cells
inferredcites →https://doi.org/10.3892/mmr.2015.3728openalex-snapshot
MicroRNA-29c targets β-site amyloid precursor protein-cleaving enzyme 1 and has a neuroprotective role in vitro and in vivo
inferredcites →https://doi.org/10.3892/ol.2018.8905openalex-snapshot
miRNA‑29a inhibits colon cancer growth by regulation of the PTEN/Akt/GSK3β and Wnt/β‑catenin signaling pathways
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4477094openalex-extracted
Elav-Gal4> Mir-219 Induces Changes in Developmental Survival and Behavior in Drosophila Growth
inferred← citeshttps://doi.org/10.1002/jcp.30779openalex-snapshot
A novel approach to type 3 diabetes mechanism: The interplay between noncoding RNAs and insulin signaling pathway in Alzheimer's disease
inferred← citeshttps://doi.org/10.1007/978-3-030-71612-7_2openalex-snapshot
RNA Modifications in Neurodegenerations
inferred← citeshttps://doi.org/10.1007/s11011-024-01520-7openalex-snapshot
Exosomes and non-coding RNAs: bridging the gap in Alzheimer’s pathogenesis and therapeutics
inferred← citeshttps://doi.org/10.1007/s12035-021-02632-2openalex-snapshot
Long Non-coding RNA RMST Worsens Ischemic Stroke via MicroRNA-221-3p/PIK3R1/TGF-β Signaling Pathway
inferred← citeshttps://doi.org/10.1007/s12035-022-02727-4openalex-snapshot
MicroRNAs in the pathophysiology of Alzheimer’s disease and Parkinson's disease: an overview
inferred← citeshttps://doi.org/10.1007/s12035-023-03908-5openalex-snapshot
Alterations of mRNAs and Non-coding RNAs Associated with Neuroinflammation in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-024-04364-5openalex-snapshot
Integrative mRNA and miRNA Expression Profiles from Developing Zebrafish Head Highlight Brain-Preference Genes and Regulatory Networks
inferred← citeshttps://doi.org/10.1016/b978-0-443-44173-8.00007-3openalex-snapshot
Noncoding RNAs regulating serine/threonine kinases in neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.arr.2023.102003openalex-snapshot
Targeting epigenetics: A novel promise for Alzheimer’s disease treatment
inferred← citeshttps://doi.org/10.1016/j.arr.2024.102483openalex-snapshot
Deciphering the role of miRNAs in Alzheimer's disease: Predictive targeting and pathway modulation – A systematic review
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103211openalex-snapshot
Cell-specific MicroRNA networks orchestrate the pathogenesis of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2020.118696openalex-snapshot
GSK3 and miRNA in neural tissue: From brain development to neurodegenerative diseases
inferred← citeshttps://doi.org/10.1016/j.bbr.2021.113481openalex-snapshot
Role of RhoA/ROCK signaling in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.116880openalex-snapshot
The role of exosome-shuttled miRNAs in heavy metal-induced peripheral tissues and neuroinflammation in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.exger.2021.111336openalex-snapshot
A network pharmacology-based study on key pharmacological pathways and targets of Qi Fu Yin acting on Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.intimp.2021.107578openalex-snapshot
RETRACTED: Inhibition of interleukin-1β plays a protective role in Alzheimer’s disease by promoting microRNA-9-5p and downregulating targeting protein for xenopus kinesin-like protein 2
inferred← citeshttps://doi.org/10.1016/j.jchemneu.2023.102236openalex-snapshot
Non-coding RNAs as key players in the neurodegenerative diseases: Multi-platform strategies and approaches for exploring the Genome’s dark matter
inferred← citeshttps://doi.org/10.1016/j.mcn.2025.104005openalex-snapshot
GSK-3β dysregulation in aging: Implications for tau pathology and Alzheimer's disease progression
inferred← citeshttps://doi.org/10.1016/j.omtn.2024.102439openalex-snapshot
Therapeutic potential and microRNA regulating properties of phytochemicals in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1017/cjn.2025.10361openalex-snapshot
MicroRNA Dysregulation in Neurodegeneration: Role, Biomarker Potential and Therapeutics
inferred← citeshttps://doi.org/10.1051/bioconf/20236001001openalex-snapshot
MicroRNAs as Possible Biomarkers for Diagnosis and Therapy of Alzheimer’s Disease by Regulating the Abnormal Expression of Genes Related to Tau
inferred← citeshttps://doi.org/10.1093/ejo/cjad002openalex-snapshot
Biological characteristics of microRNAs secreted by exosomes of periodontal ligament stem cells due to mechanical force
inferred← citeshttps://doi.org/10.1111/cns.14688openalex-snapshot
Recognizing Alzheimer's disease from perspective of oligodendrocytes: Phenomena or pathogenesis?
inferred← citeshttps://doi.org/10.1111/cns.14818openalex-snapshot
GSK3: A potential target and pending issues for treatment of Alzheimer's disease
inferred← citeshttps://doi.org/10.1155/2021/9912686openalex-snapshot
Epigenetic Modulation of Microglia Function and Phenotypes in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1177/03000605241279190openalex-snapshot
Impact of microRNAs on cardiovascular diseases and aging
inferred← citeshttps://doi.org/10.1186/s11671-026-04763-8openalex-snapshot
Targeted nanomedicine strategies for Alzheimer’s disease therapy
inferred← citeshttps://doi.org/10.1186/s13195-023-01264-zopenalex-snapshot
Pathogenesis of miR-155 on nonmodifiable and modifiable risk factors in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1186/s40035-022-00302-4openalex-snapshot
Melatonin ameliorates tau-related pathology via the miR-504-3p and CDK5 axis in Alzheimer’s disease
inferred← citeshttps://doi.org/10.14715/cmb/2023.69.9.17openalex-snapshot
MiR-669b-5p inhibits the Alzheimer's disease development via regulation of CHEK2 in Neuro2a APPSwe/Δ9 cells
inferred← citeshttps://doi.org/10.1515/revneuro-2025-0013openalex-snapshot
Exercise-induced modulation of miRNAs and gut microbiome: a holistic approach to neuroprotection in Alzheimer’s disease
inferred← citeshttps://doi.org/10.21203/rs.3.rs-3410080/v1openalex-snapshot
Integrative mRNA and miRNA expression profiles from developing zebrafish head highlight nervous system-preference genes and regulatory networks
inferred← citeshttps://doi.org/10.21203/rs.3.rs-784797/v1openalex-snapshot
Exploring Biomarkers Related to Autophagy in Alzheimer's Disease Based on Pathway Crosstalk Analysis
inferred← citeshttps://doi.org/10.2147/dddt.s289473openalex-snapshot
Epigallocatechin-3-Gallate Provides Protection Against Alzheimer’s Disease-Induced Learning and Memory Impairments in Rats
inferred← citeshttps://doi.org/10.2147/jir.s304828openalex-snapshot
Exogenous Plant gma-miR-159a, Identified by miRNA Library Functional Screening, Ameliorated Hepatic Stellate Cell Activation and Inflammation via Inhibiting GSK-3β-Mediated Pathways
inferred← citeshttps://doi.org/10.2174/0929867327666200121122910openalex-snapshot
Neuronal Development-Related miRNAs as Biomarkers for Alzheimer's Disease, Depression, Schizophrenia and Ionizing Radiation Exposure
inferred← citeshttps://doi.org/10.2174/1567202617666200128141938openalex-snapshot
MiR-335-5p Inhibits β-Amyloid (Aβ) Accumulation to Attenuate Cognitive Deficits Through Targeting c-jun-N-terminal Kinase 3 in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/jad-215349openalex-snapshot
Putative Factors Interfering Cell Cycle Re-Entry in Alzheimer’s Disease: An Omics Study with Differential Expression Meta-Analytics and Co-Expression Profiling
inferred← citeshttps://doi.org/10.3233/jad-215408openalex-snapshot
Zeaxanthin Attenuates the Vicious Circle Between Endoplasmic Reticulum Stress and Tau Phosphorylation: Involvement of GSK-3β Activation
inferred← citeshttps://doi.org/10.3233/jad-215434openalex-snapshot
Sporadic Alzheimer’s Disease- and Neurotoxicity-Related microRNAs Affecting Key Events of Tau-Driven Adverse Outcome Pathway Toward Memory Loss
inferred← citeshttps://doi.org/10.3349/ymj.2022.63.3.282openalex-snapshot
MiR-322-5p Alleviates Cell Injury and Impairment of Cognitive Function in Vascular Dementia by Targeting TSPAN5
inferred← citeshttps://doi.org/10.3389/fneur.2020.538301openalex-snapshot
Epigenetics: Recent Advances and Its Role in the Treatment of Alzheimer's Disease
inferred← citeshttps://doi.org/10.3389/fneur.2020.578720openalex-snapshot
Advances and Challenges in Understanding MicroRNA Function in Tauopathies: A Case Study of miR-132/212
inferred← citeshttps://doi.org/10.3389/fnmol.2020.00160openalex-snapshot
MicroRNAs in Alzheimer’s Disease: Function and Potential Applications as Diagnostic Biomarkers
inferred← citeshttps://doi.org/10.3389/fnmol.2024.1386735openalex-snapshot
MicroRNA biomarkers as next-generation diagnostic tools for neurodegenerative diseases: a comprehensive review
inferred← citeshttps://doi.org/10.3390/antiox9111095openalex-snapshot
Non-Coding RNAs as Sensors of Oxidative Stress in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.3390/cells10123479openalex-snapshot
Co-Expression Network Analysis of Micro-RNAs and Proteins in the Alzheimer’s Brain: A Systematic Review of Studies in the Last 10 Years
inferred← citeshttps://doi.org/10.3390/genes11090983openalex-snapshot
MicroRNAs Modulate the Pathogenesis of Alzheimer’s Disease: An In Silico Analysis in the Human Brain
inferred← citeshttps://doi.org/10.3390/ijms20236055openalex-snapshot
Interplay between MicroRNAs and Oxidative Stress in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.3390/ijms21144977openalex-snapshot
Modulation of MicroRNAs as a Potential Molecular Mechanism Involved in the Beneficial Actions of Physical Exercise in Alzheimer Disease
inferred← citeshttps://doi.org/10.3390/ijms241511942openalex-snapshot
Dysregulated miRNA and mRNA Expression Affect Overlapping Pathways in a Huntington’s Disease Model
inferred← citeshttps://doi.org/10.3390/ijms242216259openalex-snapshot
Potential Implications of miRNAs in the Pathogenesis, Diagnosis, and Therapeutics of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/ijms25189936openalex-snapshot
Insights into the Role of microRNAs as Clinical Tools for Diagnosis, Prognosis, and as Therapeutic Targets in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/ijms26189109openalex-snapshot
Exploring Molecular and Clinical Dimensions of Glaucoma as a Neurodegenerative Disease
inferred← citeshttps://doi.org/10.3390/medicina61081462openalex-snapshot
From Better Diagnostics to Earlier Treatment: The Rapidly Evolving Alzheimer’s Disease Landscape
inferred← citeshttps://doi.org/10.4103/nrr.nrr-d-25-00190openalex-snapshot
Interplay between brain-specific microRNAs and Alzheimer’s disease
inferred← citeshttps://doi.org/10.4103/rps.rps_163_23openalex-snapshot
MicroRNA-219 in the central nervous system: a potential theranostic approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.19731764openalex-snapshot
Additional file 1 of Melatonin ameliorates tau-related pathology via the miR-504-3p and CDK5 axis in Alzheimer’s disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.19731767openalex-snapshot
Additional file 2 of Melatonin ameliorates tau-related pathology via the miR-504-3p and CDK5 axis in Alzheimer’s disease
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