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Emerging role of CCN family proteins in fibrosis
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/hep.29418openalex-snapshot
A functional characteristic of cysteine‐rich protein 61: Modulation of myeloid‐derived suppressor cells in liver inflammation
inferredcites →https://doi.org/10.1002/jcp.20251openalex-snapshot
Connective tissue growth factor causes persistent proα2(I) collagen gene expression induced by transforming growth factor‐β in a mouse fibrosis model
inferredcites →https://doi.org/10.1002/jcp.21449openalex-snapshot
Neutralizing monoclonal antibody to human connective tissue growth factor ameliorates transforming growth factor‐β‐induced mouse fibrosis
inferredcites →https://doi.org/10.1002/jcp.27590openalex-snapshot
LncRNA ENST00000453774.1 contributes to oxidative stress defense dependent on autophagy mediation to reduce extracellular matrix and alleviate renal fibrosis
inferredcites →https://doi.org/10.1002/jcp.29386openalex-snapshot
Inhibition of microRNA‐150‐5p alleviates cardiac inflammation and fibrosis via targeting Smad7 in high glucose‐treated cardiac fibroblasts
inferredcites →https://doi.org/10.1002/jor.24337openalex-snapshot
Blocking CCN2 Reduces Progression of Sensorimotor Declines and Fibrosis in a Rat Model of Chronic Repetitive Overuse
inferredcites →https://doi.org/10.1002/jor.24425openalex-snapshot
CCN5 Reduces Ligamentum Flavum Hypertrophy by Modulating the TGF‐β Pathway
inferredcites →https://doi.org/10.1002/path.2277openalex-snapshot
Cellular and molecular mechanisms of fibrosis
inferredcites →https://doi.org/10.1007/978-1-4939-6430-7_37openalex-snapshot
Analysis of Pathological Activities of CCN Proteins in Fibrotic Diseases: Liver Fibrosis
inferredcites →https://doi.org/10.1007/978-981-13-8871-2_17openalex-snapshot
Connective Tissue Growth Factor and Renal Fibrosis
inferredcites →https://doi.org/10.1007/s00018-011-0778-3openalex-snapshot
CCN1/CYR61: the very model of a modern matricellular protein
inferredcites →https://doi.org/10.1007/s10735-018-9796-xopenalex-snapshot
Mechanism of fibrogenesis in submandibular glands in patients with IgG4-RD
inferredcites →https://doi.org/10.1007/s10753-015-0218-xopenalex-snapshot
Functional Effects of WNT1-Inducible Signaling Pathway Protein-1 on Bronchial Smooth Muscle Cell Migration and Proliferation in OVA-Induced Airway Remodeling
inferredcites →https://doi.org/10.1007/s12079-010-0085-zopenalex-snapshot
CCN3/CCN2 regulation and the fibrosis of diabetic renal disease
inferredcites →https://doi.org/10.1007/s12079-011-0141-3openalex-snapshot
CCN3/NOV small interfering RNA enhances fibrogenic gene expression in primary hepatic stellate cells and cirrhotic fat storing cell line CFSC
inferredcites →https://doi.org/10.1007/s12079-012-0180-4openalex-snapshot
Anti-fibrotic effect of CCN3 accompanied by altered gene expression profile of the CCN family
inferredcites →https://doi.org/10.1007/s12079-013-0193-7openalex-snapshot
CCN proteins: A centralized communication network
inferredcites →https://doi.org/10.1007/s12079-015-0281-yopenalex-snapshot
Yin and Yang revisited: CCN3 as an anti-fibrotic therapeutic?
inferredcites →https://doi.org/10.1007/s12079-016-0324-zopenalex-snapshot
Cell surface receptors for CCN proteins
inferredcites →https://doi.org/10.1007/s12079-016-0346-6openalex-snapshot
CCN family of proteins: critical modulators of the tumor cell microenvironment
inferredcites →https://doi.org/10.1007/s12079-017-0432-4openalex-snapshot
Myeloid deficiency of CCN3 exacerbates liver injury in a mouse model of nonalcoholic fatty liver disease
inferredcites →https://doi.org/10.1007/s12079-019-00542-6openalex-snapshot
Breathe, breathe in the air: the anti-CCN2 antibody pamrevlumab (FG-3019) completes a successful phase II clinical trial for idiopathic pulmonary fibrosis
inferredcites →https://doi.org/10.1007/s12079-019-00543-5openalex-snapshot
CCN proteins are part of a multilayer complex system: a working model
inferredcites →https://doi.org/10.1016/bs.ctdb.2018.02.003openalex-snapshot
Matricellular Proteins: Functional Insights From Non-mammalian Animal Models
inferredcites →https://doi.org/10.1016/j.ajpath.2012.01.031openalex-snapshot
A Novel, Dual Role of CCN3 in Experimental Glomerulonephritis
inferredcites →https://doi.org/10.1016/j.ajpath.2014.07.009openalex-snapshot
Treatment with the Matricellular Protein CCN3 Blocks and/or Reverses Fibrosis Development in Obesity with Diabetic Nephropathy
inferredcites →https://doi.org/10.1016/j.bbamcr.2014.01.023openalex-snapshot
The anti-fibrotic effects of CCN1/CYR61 in primary portal myofibroblasts are mediated through induction of reactive oxygen species resulting in cellular senescence, apoptosis and attenuated TGF-β signaling
inferredcites →https://doi.org/10.1016/j.bbamcr.2016.07.006openalex-snapshot
Adenoviral CCN gene transfers induce in vitro and in vivo endoplasmic reticulum stress and unfolded protein response
inferredcites →https://doi.org/10.1016/j.bbrc.2007.01.195openalex-snapshot
CCN1 protects cardiac myocytes from oxidative stress via β1 integrin–Akt pathway
inferredcites →https://doi.org/10.1016/j.bbrc.2018.09.001openalex-snapshot
CCN1 accelerates re-epithelialization by promoting keratinocyte migration and proliferation during cutaneous wound healing
inferredcites →https://doi.org/10.1016/j.bcp.2004.06.009openalex-snapshot
Nephroblastoma overexpressed gene (NOV) expression in rat hepatic stellate cells
inferredcites →https://doi.org/10.1016/j.biocel.2008.07.025openalex-snapshot
Functions and mechanisms of action of CCN matricellular proteins
inferredcites →https://doi.org/10.1016/j.biocel.2014.06.011openalex-snapshot
miR-92a regulates TGF-β1-induced WISP1 expression in pulmonary fibrosis
inferredcites →https://doi.org/10.1016/j.cell.2008.06.049openalex-snapshot
Senescence of Activated Stellate Cells Limits Liver Fibrosis
inferredcites →https://doi.org/10.1016/j.cellsig.2015.10.013openalex-snapshot
CCN1/CYR61 overexpression in hepatic stellate cells induces ER stress-related apoptosis
inferredcites →https://doi.org/10.1016/j.cellsig.2016.07.009openalex-snapshot
Regulation of the CCN genes by vitamin D: A possible adjuvant therapy in the treatment of cancer and fibrosis
inferredcites →https://doi.org/10.1016/j.cellsig.2018.07.005openalex-snapshot
Portal myofibroblasts are sensitive to CCN-mediated endoplasmic reticulum stress-related apoptosis with potential to attenuate biliary fibrogenesis
inferredcites →https://doi.org/10.1016/j.jacc.2016.01.030openalex-snapshot
Matricellular Protein CCN5 Reverses Established Cardiac Fibrosis
inferredcites →https://doi.org/10.1016/j.jacc.2016.02.047openalex-snapshot
Essential Role for Premature Senescence of Myofibroblasts in Myocardial Fibrosis
inferredcites →https://doi.org/10.1016/j.mam.2018.06.003openalex-snapshot
Organ and tissue fibrosis: Molecular signals, cellular mechanisms and translational implications
inferredcites →https://doi.org/10.1016/j.mam.2018.09.002openalex-snapshot
Liver fibrosis: Pathophysiology, pathogenetic targets and clinical issues
inferredcites →https://doi.org/10.1016/j.matbio.2014.07.005openalex-snapshot
Revisiting the matricellular concept
inferredcites →https://doi.org/10.1016/j.matbio.2016.07.006openalex-snapshot
Degradomic and yeast 2-hybrid inactive catalytic domain substrate trapping identifies new membrane-type 1 matrix metalloproteinase (MMP14) substrates: CCN3 (Nov) and CCN5 (WISP2)
inferredcites →https://doi.org/10.1016/j.matbio.2018.01.004openalex-snapshot
CCN4/WISP1 controls cutaneous wound healing by modulating proliferation, migration and ECM expression in dermal fibroblasts via α5β1 and TNFα
inferredcites →https://doi.org/10.1016/j.matbio.2018.01.019openalex-snapshot
The big five in fibrosis: Macrophages, myofibroblasts, matrix, mechanics, and miscommunication
inferredcites →https://doi.org/10.1016/j.matbio.2018.03.007openalex-snapshot
Connective tissue growth factor (CTGF) from basics to clinics
inferredcites →https://doi.org/10.1016/j.metabol.2015.11.007openalex-snapshot
Atorvastatin prevents advanced glycation end products (AGEs)-induced cardiac fibrosis via activating peroxisome proliferator-activated receptor gamma (PPAR-γ)
inferredcites →https://doi.org/10.1016/j.metabol.2020.154140openalex-snapshot
Epigenetic silencing of microRNA-125b-5p promotes liver fibrosis in nonalcoholic fatty liver disease via integrin α8-mediated activation of RhoA signaling pathway
inferredcites →https://doi.org/10.1016/j.pharep.2014.08.019openalex-snapshot
Transcriptional and post-transcriptional regulation of CCN genes in failing heart
inferredcites →https://doi.org/10.1016/j.rmed.2019.105821openalex-snapshot
The senescence-associated matricellular protein CCN1 in plasma of human subjects with idiopathic pulmonary fibrosis
inferredcites →https://doi.org/10.1016/j.tibs.2008.07.006openalex-snapshot
The CCN family of proteins: structure–function relationships
inferredcites →https://doi.org/10.1016/j.trsl.2011.01.012openalex-snapshot
MicroRNAs in idiopathic pulmonary fibrosis
inferredcites →https://doi.org/10.1016/j.yjmcc.2010.04.010openalex-snapshot
The opposing effects of CCN2 and CCN5 on the development of cardiac hypertrophy and fibrosis
inferredcites →https://doi.org/10.1016/s1386-6346(03)00241-9openalex-snapshot
Low density lipoprotein receptor-related protein (LRP) is a heparin-dependent adhesion receptor for connective tissue growth factor (CTGF) in rat activated hepatic stellate cells
inferredcites →https://doi.org/10.1016/s2213-2600(19)30262-0openalex-snapshot
Pamrevlumab, an anti-connective tissue growth factor therapy, for idiopathic pulmonary fibrosis (PRAISE): a phase 2, randomised, double-blind, placebo-controlled trial
inferredcites →https://doi.org/10.1021/pr3000927openalex-snapshot
Proteomic analysis of extracellular matrix from the hepatic stellate cell line LX-2 identifies CYR61 and Wnt-5a as novel constituents of fibrotic liver
inferredcites →https://doi.org/10.1038/aps.2017.87openalex-snapshot
CCN3 suppresses TGF-β1-induced extracellular matrix accumulation in human mesangial cells in vitro
inferredcites →https://doi.org/10.1038/jid.2010.15openalex-snapshot
Antagonistic Effect of the Matricellular Signaling Protein CCN3 on TGF-β- and Wnt-Mediated Fibrillinogenesis in Systemic Sclerosis and Marfan Syndrome
inferredcites →https://doi.org/10.1038/ncb2070openalex-snapshot
The matricellular protein CCN1 induces fibroblast senescence and restricts fibrosis in cutaneous wound healing
inferredcites →https://doi.org/10.1038/ncb826openalex-snapshot
Connective-tissue growth factor (CTGF) modulates cell signalling by BMP and TGF-β
inferredcites →https://doi.org/10.1038/ncomms8386openalex-snapshot
The matricellular protein CCN1 mediates neutrophil efferocytosis in cutaneous wound healing
inferredcites →https://doi.org/10.1038/nrgastro.2010.97openalex-snapshot
Evolving challenges in hepatic fibrosis
inferredcites →https://doi.org/10.1038/sj.emboj.7601596openalex-snapshot
Cytotoxicity of TNFα is regulated by integrin‐mediated matrix signaling
inferredcites →https://doi.org/10.1038/srep20547openalex-snapshot
WISP1 mediates IL-6-dependent proliferation in primary human lung fibroblasts
inferredcites →https://doi.org/10.1056/nejmc1504848openalex-snapshot
Fibrosis — A Common Pathway to Organ Injury and Failure
inferredcites →https://doi.org/10.1056/nejmra1300575openalex-snapshot
Fibrosis — A Common Pathway to Organ Injury and Failure
inferredcites →https://doi.org/10.1073/pnas.95.25.14717openalex-snapshot
WISP genes are members of the connective tissue growth factor family that are up-regulated in Wnt-1-transformed cells and aberrantly expressed in human colon tumors
inferredcites →https://doi.org/10.1074/jbc.m107666200openalex-snapshot
The Angiogenic Factor Cyr61 Activates a Genetic Program for Wound Healing in Human Skin Fibroblasts
inferredcites →https://doi.org/10.1074/jbc.m108339200openalex-snapshot
WISP-1 Binds to Decorin and Biglycan
inferredcites →https://doi.org/10.1074/jbc.m110.137273openalex-snapshot
Mechanical Stretch Induces Epithelial-Mesenchymal Transition in Alveolar Epithelia via Hyaluronan Activation of Innate Immunity
inferredcites →https://doi.org/10.1074/jbc.m203727200openalex-snapshot
The Nephroblastoma Overexpressed Gene (NOV/ccn3) Protein Associates with Notch1 Extracellular Domain and Inhibits Myoblast Differentiation via Notch Signaling Pathway
inferredcites →https://doi.org/10.1074/jbc.m809757200openalex-snapshot
WISP1, a Pro-mitogenic, Pro-survival Factor, Mediates Tumor Necrosis Factor-α (TNF-α)-stimulated Cardiac Fibroblast Proliferation but Inhibits TNF-α-induced Cardiomyocyte Death
inferredcites →https://doi.org/10.1080/08977190802025602openalex-snapshot
Connective tissue growth factor: Structure–function relationships of a mosaic, multifunctional protein
inferredcites →https://doi.org/10.1083/jcb.114.6.1285openalex-snapshot
Connective tissue growth factor: a cysteine-rich mitogen secreted by human vascular endothelial cells is related to the SRC-induced immediate early gene product CEF-10.
inferredcites →https://doi.org/10.1083/jcb.130.3.503openalex-snapshot
Diversity of function is inherent in matricellular proteins: an appraisal of thrombospondin 1.
inferredcites →https://doi.org/10.1084/jem.20110551openalex-snapshot
Integrating mechanisms of pulmonary fibrosis
inferredcites →https://doi.org/10.1096/fj.201500173openalex-snapshot
The matricellular protein CCN1 enhances TGF‐β1/SMAD3‐dependent profibrotic signaling in fibroblasts and contributes to fibrogenic responses to lung injury
inferredcites →https://doi.org/10.1096/fj.201600433ropenalex-snapshot
Mesenchymal stromal cells and liver fibrosis: a complicated relationship
inferredcites →https://doi.org/10.1096/fj.201902063ropenalex-snapshot
SHIP‐1, a target of miR‐155, regulates endothelial cell responses in lung fibrosis
inferredcites →https://doi.org/10.1096/fj.202000240rropenalex-snapshot
Blocking CTGF/CCN2 reduces established skeletal muscle fibrosis in a rat model of overuse injury
inferredcites →https://doi.org/10.1097/00004630-199711000-00002openalex-snapshot
THE 1997 MOYER AWARD Cytokine Production in Patients with Hypertrophic Burn Scars
inferredcites →https://doi.org/10.1097/hco.0000000000000158openalex-snapshot
Inflammation in cardiac injury, repair and regeneration
inferredcites →https://doi.org/10.1097/paf.0b013e31828d69b5openalex-snapshot
Correlations Between CCN1 Immunoexpression and Myocardial Histologic Lesions in Sudden Cardiac Death
inferredcites →https://doi.org/10.1111/1440-1681.12470openalex-snapshot
CCN 2 and CCN 5 exerts opposing effect on fibroblast proliferation and transdifferentiation induced by TGF ‐β
inferredcites →https://doi.org/10.1111/exd.13806openalex-snapshot
CCN proteins as potential actionable targets in scleroderma
inferredcites →https://doi.org/10.1111/j.1440-169x.2009.01077.xopenalex-snapshot
Role of CCN, a vertebrate specific gene family, in development
inferredcites →https://doi.org/10.1111/j.1478-3231.2012.02837.xopenalex-snapshot
Adenoviral CCN 3/ NOV gene transfer fails to mitigate liver fibrosis in an experimental bile duct ligation model because of hepatocyte apoptosis
inferredcites →https://doi.org/10.1111/tri.12639openalex-snapshot
Systemic overexpression of matricellular protein CCN1 exacerbates obliterative bronchiolitis in mouse tracheal allografts
inferredcites →https://doi.org/10.1126/science.aaa5612openalex-snapshot
The DNA damage response induces inflammation and senescence by inhibiting autophagy of GATA4
inferredcites →https://doi.org/10.1128/mcb.00049-13openalex-snapshot
Matricellular Protein CCN1 Promotes Regression of Liver Fibrosis through Induction of Cellular Senescence in Hepatic Myofibroblasts
inferredcites →https://doi.org/10.1128/mcb.10.7.3569openalex-snapshot
Expression of cyr61, a Growth Factor-Inducible Immediate-Early Gene
inferredcites →https://doi.org/10.1128/mcb.12.1.10openalex-snapshot
Proviral rearrangements and overexpression of a new cellular gene (nov) in myeloblastosis-associated virus type 1-induced nephroblastomas.
inferredcites →https://doi.org/10.1136/mp.56.2.127openalex-snapshot
Proposal for a unified CCN nomenclature: Table 1
inferredcites →https://doi.org/10.1152/ajpcell.00028.2020openalex-snapshot
Conjunction junction, what’s the function? CCN proteins as targets in fibrosis and cancers
inferredcites →https://doi.org/10.1152/ajpheart.00428.2007openalex-snapshot
Wnt-induced secreted protein-1 is a prohypertrophic and profibrotic growth factor
inferredcites →https://doi.org/10.1152/ajplung.00190.2014openalex-snapshot
CYR61 (CCN1) overexpression induces lung injury in mice
inferredcites →https://doi.org/10.1155/2015/764641openalex-snapshot
Fibrosis Related Inflammatory Mediators: Role of the IL‐10 Cytokine Family
inferredcites →https://doi.org/10.1159/000491984openalex-snapshot
Prognostic Significance of Serum Cysteine-Rich Protein 61 in Patients with Acute Heart Failure
inferredcites →https://doi.org/10.1161/01.cir.0000127952.90508.9dopenalex-snapshot
Regulation of Proangiogenic Factor CCN1 in Cardiac Muscle
inferredcites →https://doi.org/10.1161/jaha.118.009111openalex-snapshot
Postinfarction Hearts Are Protected by Premature Senescent Cardiomyocytes Via GATA4‐Dependent CCN1 Secretion
inferredcites →https://doi.org/10.1164/rccm.200907-1009ocopenalex-snapshot
A Role for Wnt Signaling Genes in the Pathogenesis of Impaired Lung Function in Asthma
inferredcites →https://doi.org/10.1165/rcmb.2012-0127ocopenalex-snapshot
WNT1-Inducible Signaling Pathway Protein 1 Contributes to Ventilator-Induced Lung Injury
inferredcites →https://doi.org/10.1172/jci33288openalex-snapshot
Increased local expression of coagulation factor X contributes to the fibrotic response in human and murine lung injury
inferredcites →https://doi.org/10.1172/jci33950openalex-snapshot
WNT1-inducible signaling protein–1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosis
inferredcites →https://doi.org/10.1172/jci79327openalex-snapshot
CCN1 induces hepatic ductular reaction through integrin αvβ5–mediated activation of NF-κB
inferredcites →https://doi.org/10.1182/blood-2006-07-036202openalex-snapshot
The angiogenic factor CCN1 promotes adhesion and migration of circulating CD34+ progenitor cells: potential role in angiogenesis and endothelial regeneration
inferredcites →https://doi.org/10.1210/edrv.20.2.0360openalex-snapshot
The Connective Tissue Growth Factor/Cysteine- Rich 61/Nephroblastoma Overexpressed (CCN) Family
inferredcites →https://doi.org/10.1210/en.2011-0234openalex-snapshot
Effect of CCN2 on FGF2-Induced Proliferation and MMP9 and MMP13 Productions by Chondrocytes
inferredcites →https://doi.org/10.1242/jcs.03270openalex-snapshot
All in the CCN family: essential matricellular signaling modulators emerge from the bunker
inferredcites →https://doi.org/10.1371/journal.pone.0137876openalex-snapshot
Reduced NOV/CCN3 Expression Limits Inflammation and Interstitial Renal Fibrosis after Obstructive Nephropathy in Mice
inferredcites →https://doi.org/10.1371/journal.pone.0186740openalex-snapshot
Antioxidants and NOX1/NOX4 inhibition blocks TGFβ1-induced CCN2 and α-SMA expression in dermal and gingival fibroblasts
inferredcites →https://doi.org/10.1371/journal.pone.0207228openalex-snapshot
CCN5 knockout mice exhibit lipotoxic cardiomyopathy with mild obesity and diabetes
inferredcites →https://doi.org/10.1371/journal.pone.0208897openalex-snapshot
The matricellular protein CCN5 inhibits fibrotic deformation of retinal pigment epithelium
inferredcites →https://doi.org/10.1371/journal.pone.0218178openalex-snapshot
Yin/Yang expression of CCN family members: Transforming growth factor beta 1, via ALK5/FAK/MEK, induces CCN1 and CCN2, yet suppresses CCN3, expression in human dermal fibroblasts
inferredcites →https://doi.org/10.14670/hh-25.795openalex-snapshot
The CCN proteins: important signaling mediators in stem cell differentiation and tumorigenesis.
inferredcites →https://doi.org/10.14715/cmb/2018.64.3.2openalex-snapshot
CCN3, a key matricellular protein, distinctly inhibits TGFβ1-mediated Smad1/5/8 signalling in human podocyte culture
inferredcites →https://doi.org/10.2152/jmi.58.188openalex-snapshot
CCN6 as a profibrotic mediator that stimulates the proliferation of lung fibroblasts via the integrin .BETA.1/focal adhesion kinase pathway
inferredcites →https://doi.org/10.2174/1381612043382567openalex-snapshot
Structural Basis and Therapeutic Implication of the Interaction of CCN Proteins with Glycoconjugates
inferredcites →https://doi.org/10.2353/ajpath.2009.080241openalex-snapshot
CCN3 (NOV) Is a Negative Regulator of CCN2 (CTGF) and a Novel Endogenous Inhibitor of the Fibrotic Pathway in an in Vitro Model of Renal Disease
inferredcites →https://doi.org/10.3109/1354750x.2012.722229openalex-snapshot
Urinary mRNA expression of CCN2/CCN3 as a noninvasive marker for monitoring glomerular structure changes in nondiabetic chronic kidney disease
inferredcites →https://doi.org/10.3389/fphar.2020.01235openalex-snapshot
Endogenous CCN5 Participates in Angiotensin II/TGF-β1 Networking of Cardiac Fibrosis in High Angiotensin II-Induced Hypertensive Heart Failure
inferredcites →https://doi.org/10.3892/ijmm.2013.1565openalex-snapshot
CCN5 overexpression inhibits profibrotic phenotypes via the PI3K/Akt signaling pathway in lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis and in an in vivo model of lung fibrosis
inferredcites →https://doi.org/10.3892/ijmm.2014.1735openalex-snapshot
Effects of CCN3 on fibroblast proliferation, apoptosis and extracellular matrix production
inferredcites →https://doi.org/10.3892/ijmm.2015.2190openalex-snapshot
CCN5 attenuates profibrotic phenotypes of fibroblasts through the Smad6-CCN2 pathway: Potential role in epidural fibrosis
inferredcites →https://doi.org/10.3892/ijmm.2017.3356openalex-snapshot
CCN1/Cyr61 enhances the function of hepatic stellate cells in promoting the progression of hepatocellular carcinoma
inferredcites →https://doi.org/10.4049/jimmunol.0902792openalex-snapshot
Matricellular Protein CCN1 Activates a Proinflammatory Genetic Program in Murine Macrophages
inferredcites →https://doi.org/10.5114/aoms.2015.52371openalex-snapshot
Experimental research Blockade of CCN4 attenuates CCl 4 -induced liver fibrosis
inferredcites →https://doi.org/10.5603/fhc.2012.0014openalex-snapshot
Atrial expression of the CCN1 and CCN2 proteins in chronic heart failure
inferredcites →https://doi.org/10.5603/fhc.2013.0012openalex-snapshot
CCN1 expression in interleukin-6 deficient mouse kidney in experimental model of heart failure
inferredcites →https://doi.org/10.7754/clin.lab.2013.121035openalex-snapshot
Wnt-Induced Secreted Protein 1/CCN4 in Liver Fibrosis Both in vitro and in vivo
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4637594openalex-extracted
Role of Curcumin in Mediating BSA Fibrillation in a Cu 2 System: A Mechanistic Insight
inferred← citeshttps://doi.org/10.1002/ccs3.12027openalex-snapshot
Matricellular proteins: Potential biomarkers in head and neck cancer
inferred← citeshttps://doi.org/10.1002/ccs3.70051openalex-snapshot
Interaction between nuclear‐translocated cellular communication network factor 2 and purine‐rich box 1 regulates the expression of fibrosis‐related genes
inferred← citeshttps://doi.org/10.1002/jcp.31390openalex-snapshot
The novel thromboxane prostanoid receptor mediates CTGF production to drive human nasal fibroblast self‐migration through NF‐κB and PKCδ‐CREB signaling pathways
inferred← citeshttps://doi.org/10.1007/s00210-024-03170-zopenalex-snapshot
Zerumbone alleviated bleomycin-induced pulmonary fibrosis in mice via SIRT1/Nrf2 pathway
inferred← citeshttps://doi.org/10.1007/s12079-023-00762-xopenalex-snapshot
Significance of CCNs in liver regeneration
inferred← citeshttps://doi.org/10.1007/s12079-023-00765-8openalex-snapshot
Recent progress on the role of cellular communication network factors (CCN) 3, 4 and 6 in regulating adiposity, liver fibrosis and pancreatic islets
inferred← citeshttps://doi.org/10.1007/s12079-023-00778-3openalex-snapshot
The advance of CCN3 in fibrosis
inferred← citeshttps://doi.org/10.1016/j.bcp.2023.115873openalex-snapshot
Emerging role of m6A modification in fibrotic diseases and its potential therapeutic effect
inferred← citeshttps://doi.org/10.1016/j.bioadv.2024.214119openalex-snapshot
Piezoelectric nanocomposite electrospun dressings: Tailoring mechanics for scar-free wound recovery
inferred← citeshttps://doi.org/10.1016/j.enceco.2025.12.021openalex-snapshot
Polystyrene microplastics modulation of hexavalent chromium toxicity in quails: transcriptomic and toxicological insights
inferred← citeshttps://doi.org/10.1016/j.identj.2024.06.011openalex-snapshot
Effects and Significance of Dicliptera chinensis Polysaccharide on the Expression of Transforming Growth Factor β1/Connective Tissue Growth Factor Pathway in the Masseter and Head and Neck Skin of Rats With Radiation-Induced Fibrosis
inferred← citeshttps://doi.org/10.1016/j.isci.2025.113993openalex-snapshot
Mechanobiology and the extracellular matrix in pulmonary fibrosis
inferred← citeshttps://doi.org/10.1016/j.jdsct.2025.100129openalex-snapshot
CCN1 as an emerging key regulator in sensitive skin and skin aging: Mechanisms and therapeutic potential
inferred← citeshttps://doi.org/10.1016/j.stem.2022.01.012openalex-snapshot
MBNL1 drives dynamic transitions between fibroblasts and myofibroblasts in cardiac wound healing
inferred← citeshttps://doi.org/10.1038/s12276-021-00636-5openalex-snapshot
WISP-1 induced by mechanical stress contributes to fibrosis and hypertrophy of the ligamentum flavum through Hedgehog-Gli1 signaling
inferred← citeshttps://doi.org/10.1038/s41419-025-07547-7openalex-snapshot
A novel clinically relevant antagonistic interplay between prolactin and oncogenic YAP-CCN2 pathways as a differentiation therapeutic target in breast cancer
inferred← citeshttps://doi.org/10.1071/rd24199openalex-snapshot
The influence of CCN family proteins on ovarian physiology and pathology
inferred← citeshttps://doi.org/10.1080/10985549.2023.2253130openalex-snapshot
Module 4-Deficient CCN2/Connective Tissue Growth Factor Attenuates the Progression of Renal Fibrosis via Suppression of Focal Adhesion Kinase Phosphorylation in Tubular Epithelial Cells
inferred← citeshttps://doi.org/10.1093/brain/awae414openalex-snapshot
Distinct transcriptional changes distinguish efficient and poor remyelination in multiple sclerosis
inferred← citeshttps://doi.org/10.1101/2021.01.26.428279openalex-snapshot
Fibroblast State Reversal By MBNL1-Dependent Transcriptome Modification Regulates Cardiac Repair
inferred← citeshttps://doi.org/10.1172/jci190635openalex-snapshot
Adipocyte death promotes hepatic infiltration of S100A8+ macrophages and steatotic liver disease progression in mice
inferred← citeshttps://doi.org/10.1186/s12967-024-05364-8openalex-snapshot
An overview of CCN4 (WISP1) role in human diseases
inferred← citeshttps://doi.org/10.21037/atm-22-2496openalex-snapshot
Rhamnocitrin decreases fibrosis of ovarian granulosa cells by regulating the activation of the PPARγ/NF-κB/TGF-β1/Smad2/3 signaling pathway mediated by Wisp2
inferred← citeshttps://doi.org/10.3389/fcell.2022.1073320openalex-snapshot
Matricellular proteins in cutaneous wound healing
inferred← citeshttps://doi.org/10.3390/antiox12081541openalex-snapshot
CCN2 Activates RIPK3, NLRP3 Inflammasome, and NRF2/Oxidative Pathways Linked to Kidney Inflammation
inferred← citeshttps://doi.org/10.3390/biom14101277openalex-snapshot
Cellular and Molecular Mechanisms of Hypertrophy of Ligamentum Flavum
inferred← citeshttps://doi.org/10.3390/genes12111659openalex-snapshot
NOV/CCN3 Promotes Cell Migration and Invasion in Intrahepatic Cholangiocarcinoma via miR-92a-3p
inferred← citeshttps://doi.org/10.3390/ijms22094340openalex-snapshot
Targeting CCN Proteins in Rheumatoid Arthritis and Osteoarthritis
inferred← citeshttps://doi.org/10.3390/ijms22105234openalex-snapshot
Role of Matricellular CCN Proteins in Skeletal Muscle: Focus on CCN2/CTGF and Its Regulation by Vasoactive Peptides
inferred← citeshttps://doi.org/10.3390/ijms222413418openalex-snapshot
Metabolic Effects of CCN5/WISP2 Gene Deficiency and Transgenic Overexpression in Mice
inferred← citeshttps://doi.org/10.3390/ijms241813866openalex-snapshot
Blocking CCN2 Reduces Established Palmar Neuromuscular Fibrosis and Improves Function Following Repetitive Overuse Injury
inferred← citeshttps://doi.org/10.3390/ijms25010290openalex-snapshot
Balance between Pro- and Antifibrotic Proteins in Mesenchymal Stromal Cell Secretome Fractions Revealed by Proteome and Cell Subpopulation Analysis
inferred← citeshttps://doi.org/10.3390/ijms25094692openalex-snapshot
Connective Tissue Growth Factor: Regulation, Diseases, and Drug Discovery
inferred← citeshttps://doi.org/10.3390/ijms27020612openalex-snapshot
The Adipokine Axis in Heart Failure: Linking Obesity, Sarcopenia and Cardiac Dysfunction in HFpEF
inferred← citeshttps://doi.org/10.3390/ijms27073047openalex-snapshot
Novel Molecular Insights into the Anti-Inflammatory and Antifibrotic Effects of Dexamethasone on Human Ligamentum Flavum-Derived Cells
inferred← citeshttps://doi.org/10.3390/jcdd10120483openalex-snapshot
Hippo Signaling Mediates TGFβ-Dependent Transcriptional Inputs in Cardiac Cushion Mesenchymal Cells to Regulate Extracellular Matrix Remodeling
inferred← citeshttps://doi.org/10.34133/research.0762openalex-snapshot
CCN5 Drives Leydig Cell Aging and Testicular Dysfunction: Insights into Fibrosis, Lipid Dysregulation, and Therapeutic Potential
inferred← citeshttps://doi.org/10.64898/2026.01.15.699691openalex-snapshot
Uncovering the function of Wisp1 in whole-body glucose homeostasis: insights from Wisp1 knockout mice
inferred← citeshttps://doi.org/10.7150/thno.78876openalex-snapshot
Targeting cullin neddylation for cancer and fibrotic diseases
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