On the public citation graph
Periostin promotes immunosuppressive premetastatic niche formation to facilitate breast tumour metastasis
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2 of the 47 references listed on this page are in the Retraction Watch database as retracted (export 2026-10-28): 10.1038/nature04695 (notice 2020-03-18), 10.1038/nature14492 (notice 2023-04-17).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/path.4467openalex-snapshotCancer‐associated fibroblasts predict poor outcome and promote periostin‐dependent invasion in oesophageal adenocarcinoma
inferredcites →https://doi.org/10.1038/ncb3090openalex-snapshotPeriostin secreted by glioblastoma stem cells recruits M2 tumour-associated macrophages and promotes malignant growth
inferredcites →https://doi.org/10.1038/nm.3497openalex-snapshotTissue mechanics modulate microRNA-dependent PTEN expression to regulate malignant progression
inferredcites →https://doi.org/10.1038/onc.2011.181openalex-snapshotTwist2 contributes to breast cancer progression by promoting an epithelial–mesenchymal transition and cancer stem-like cell self-renewal
inferredcites →https://doi.org/10.1158/0008-5472.can-11-1792openalex-snapshotMonocytic CCR2+ Myeloid-Derived Suppressor Cells Promote Immune Escape by Limiting Activated CD8 T-cell Infiltration into the Tumor Microenvironment
inferredcites →https://doi.org/10.1172/jci40483openalex-snapshotMembrane-associated Hsp72 from tumor-derived exosomes mediates STAT3-dependent immunosuppressive function of mouse and human myeloid-derived suppressor cells
inferredcites →https://doi.org/10.4049/jimmunol.1103505openalex-snapshotMicroRNA-494 Is Required for the Accumulation and Functions of Tumor-Expanded Myeloid-Derived Suppressor Cells via Targeting of PTEN
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/cnr2.70044openalex-snapshotThe Recruitment and Immune Suppression Mechanisms of Myeloid‐Derived Suppressor Cells and Their Impact on Bone Metastatic Cancer
inferred← citeshttps://doi.org/10.1002/hed.25952openalex-snapshotTranscripto‐based network analysis reveals a model of gene activation in tongue squamous cell carcinomas
inferred← citeshttps://doi.org/10.1002/path.5756openalex-snapshotPeriostin deficiency reduces diethylnitrosamine ‐induced liver cancer in mice by decreasing hepatic stellate cell activation and cancer cell proliferation
inferred← citeshttps://doi.org/10.1007/s10585-025-10377-8openalex-snapshotDefining the Tumor-Bone axis: the role of extracellular vesicles in a crucial exchange route for breast cancer development and progression
inferred← citeshttps://doi.org/10.1016/j.bbcan.2025.189360openalex-snapshotSpatiotemporal dynamics of the pre-metastatic niche: remodeling of lung microenvironment before breast cancer metastasis and targeted intervention strategies
inferred← citeshttps://doi.org/10.1016/j.jcmgh.2022.10.001openalex-snapshotSingle-cell Transcriptomics Reveals Early Molecular and Immune Alterations Underlying the Serrated Neoplasia Pathway Toward Colorectal Cancer
inferred← citeshttps://doi.org/10.1016/j.lfs.2017.05.030openalex-snapshotRETRACTED: Effects of lentivirus-mediated silencing of Periostin on tumor microenvironment and bone metastasis via the integrin-signaling pathway in lung cancer
inferred← citeshttps://doi.org/10.1016/j.phymed.2023.155208openalex-snapshotOleanolic acid inhibits hypoxic tumor-derived exosomes-induced premetastatic niche formation in hepatocellular carcinoma by targeting ERK1/2-NFκB signaling
inferred← citeshttps://doi.org/10.1038/s41598-020-67643-wopenalex-snapshotSystems biology comprehensive analysis on breast cancer for identification of key gene modules and genes associated with TNM-based clinical stages
inferred← citeshttps://doi.org/10.1101/2023.06.08.544265openalex-snapshotConcerted epithelial and stromal changes during progression of Barrett’s Esophagus to invasive adenocarcinoma exposed by multi-scale, multi-omics analysis
inferred← citeshttps://doi.org/10.1111/cas.13567openalex-snapshotCross‐talk between ovarian cancer cells and macrophages through periostin promotes macrophage recruitment
inferred← citeshttps://doi.org/10.1111/cas.13712openalex-snapshotPeriostin and CA242 as potential diagnostic serum biomarkers complementing CA19.9 in detecting pancreatic cancer
inferred← citeshttps://doi.org/10.3389/fonc.2020.540484openalex-snapshotCD62Ldim Neutrophils Specifically Migrate to the Lung and Participate in the Formation of the Pre-Metastatic Niche of Breast Cancer
inferred← citeshttps://doi.org/10.3390/cancers13205072openalex-snapshotPeriostin Exon-21 Antibody Neutralization of Triple-Negative Breast Cancer Cell-Derived Periostin Regulates Tumor-Associated Macrophage Polarization and Angiogenesis
inferred← citeshttps://doi.org/10.3390/ijms22031227openalex-snapshotiPSC-Derived Hereditary Breast Cancer Model Reveals the BRCA1-Deleted Tumor Niche as a New Culprit in Disease Progression
inferred← citeshttps://doi.org/10.3390/ijms24076353openalex-snapshotDental Follicle Stem Cells Promote Periodontal Regeneration through Periostin-Mediated Macrophage Infiltration and Reprogramming in an Inflammatory Microenvironment
inferred← citeshttps://doi.org/10.7150/thno.94207openalex-snapshotCancer-associated fibroblast-derived periostin promotes papillary thyroid tumor growth through integrin-FAK-STAT3 signaling
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