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Ozonized carbon black induces mitochondrial dysfunction and DNA damage

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Physicochemical characteristics and toxic effects of ozone-oxidized black carbon particles

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Oxidative stress and inflammation generated DNA damage by exposure to air pollution particles

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PM 2.5 in China: Measurements, sources, visibility and health effects, and mitigation

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Effects of diesel particles on the control of intracellular mycobacterial growth by human macrophagesin vitro

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Clean Air for Megacities

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Carbon black and titanium dioxide nanoparticles elicit distinct apoptotic pathways in bronchial epithelial cells

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What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Systematic review on primary and secondary genotoxicity of carbon black nanoparticles in mammalian cells and animals

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Oxidized carbon black nanoparticles induce endothelial damage through C-X-C chemokine receptor 3-mediated pathway

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Black carbon particles and ozone-oxidized black carbon particles induced lung damage in mice through an interleukin-33 dependent pathway

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Redox cell signalling triggered by black carbon and/or radiofrequency electromagnetic fields: Influence on cell death

inferred← citeshttps://doi.org/10.1016/j.tiv.2018.09.008openalex-snapshot

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Ozone and particulate matter co-exposure at the air-liquid interface: Establishing an approach to assess pollutant interactions in vitro

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Fresh and ozonized black carbon promoted DNA damage and repair responses in A549 cells

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Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

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Degenerative and Regenerative Actin Cytoskeleton Rearrangements, Cell Death, and Paradoxical Proliferation in the Gills of Pearl Gourami (Trichogaster leerii) Exposed to Suspended Soot Microparticles

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4. Le stress radicalaire : facteur d’adaptation ou de désadaptation ?

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Additional file 3 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

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Additional file 4 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

inferred← citeshttps://doi.org/10.6084/m9.figshare.17210265openalex-snapshot

Additional file 5 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

inferred← citeshttps://doi.org/10.6084/m9.figshare.17210268openalex-snapshot

Additional file 6 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

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Additional file 7 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

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Additional file 8 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

inferred← citeshttps://doi.org/10.6084/m9.figshare.17210283openalex-snapshot

Additional file 9 of Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction

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