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An Epstein-Barr virus-producer line Akata: Establishment of the cell line and analysis of viral DNA
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/ijc.2910330106openalex-snapshot
Cross‐linking of cell surface immunoglobulins induces epstein‐barr virus in burkitt lymphoma lines
inferredcites →https://doi.org/10.1016/0042-6822(90)90458-4openalex-snapshot
Epstein-Barr virus genotypes associated with nasopharyngeal carcinoma in Southern China
inferredcites →https://doi.org/10.1016/0092-8674(88)90028-1openalex-snapshot
Identification and characterization of oriLyt, a lytic origin of DNA replication of Epstein-Barr virus
inferredcites →https://doi.org/10.1038/309078a0openalex-snapshot
Immortalization of monkey epithelial cells by specific fragments of Epstein–Barr virus DNA
inferredcites →https://doi.org/10.1038/310207a0openalex-snapshot
DNA sequence and expression of the B95-8 Epstein—Barr virus genome
inferredcites →https://doi.org/10.1038/340393a0openalex-snapshot
Genetic analysis of immortalizing functions of Epstein–Barr virus in human B lymphocytes
inferredcites →https://doi.org/10.1073/pnas.70.1.190openalex-snapshot
Release of Infectious Epstein-Barr Virus by Transformed Marmoset Leukocytes
inferredcites →https://doi.org/10.1073/pnas.76.2.782openalex-snapshot
Tumor initiators and promoters in the induction of Epstein—Barr virus
inferredcites →https://doi.org/10.1073/pnas.77.5.2999openalex-snapshot
Epstein-Barr virus (B95-8) DNA VII: molecular cloning and detailed mapping.
inferredcites →https://doi.org/10.1073/pnas.77.7.3860openalex-snapshot
Cloning and mapping of BamHi endonuclease fragments of DNA from the transforming B95-8 strain of Epstein-Barr virus.
inferredcites →https://doi.org/10.1073/pnas.86.23.9558openalex-snapshot
Epstein-Barr virus nuclear protein 2 is a key determinant of lymphocyte transformation.
inferredcites →https://doi.org/10.1126/science.2983420openalex-snapshot
A Second Nuclear Protein Is Encoded by Epstein-Barr Virus in Latent Infection
inferredcites →https://doi.org/10.1128/jvi.1.5.1045-1051.1967openalex-snapshot
Immunofluorescence and Herpes-Type Virus Particles in the P3HR-1 Burkitt Lymphoma Cell Line
inferredcites →https://doi.org/10.1128/jvi.15.3.556-559.1975openalex-snapshot
DNA of Epstein-Barr virus. I. Comparative studies of the DNA of Epstein-Barr virus from HR-1 and B95-8 cells: size, structure, and relatedness
inferredcites →https://doi.org/10.1128/jvi.17.3.935-949.1976openalex-snapshot
Proteins of Epstein-Barr virus. I. Analysis of the polypeptides of purified enveloped Epstein-Barr virus
inferredcites →https://doi.org/10.1128/jvi.38.2.632-648.1981openalex-snapshot
Epstein-Barr virus DNA. IX. Variation among viral DNAs from producer and nonproducer infected cells
inferredcites →https://doi.org/10.1128/jvi.43.3.979-986.1982openalex-snapshot
Epstein-Barr virus DNA XII. A variable region of the Epstein-Barr virus genome is included in the P3HR-1 deletion
inferredcites →https://doi.org/10.1128/jvi.57.3.1016-1022.1986openalex-snapshot
trans activation of the latent Epstein-Barr virus (EBV) genome after transfection of the EBV DNA fragment
inferredcites →https://doi.org/10.1128/jvi.63.1.445-449.1989openalex-snapshot
Synchronous and sequential activation of latently infected Epstein-Barr virus genomes
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4448956openalex-extracted
Momordica Anti-Hiv Protein Map30 Abrogates the Persistent Infection of an Oncogenic Human Herpesvirus in Host Cells
inferred← citeshttps://doi.org/10.1002/(sici)1096-9071(199807)55:3<219::aid-jmv7>3.0.co;2-4openalex-snapshot
Upregulation of Epstein-Barr virus-encoded latent membrane protein by human herpesvirus 6 superinfection of EBV-carrying Burkitt lymphoma cells
inferred← citeshttps://doi.org/10.1002/(sici)1096-9071(199901)57:1<36::aid-jmv6>3.0.co;2-1openalex-snapshot
Characterization of natural Epstein-Barr virus infection and replication in smooth muscle cells from a leiomyosarcoma
inferred← citeshttps://doi.org/10.1002/(sici)1096-9071(200006)61:2<241::aid-jmv11>3.0.co;2-2openalex-snapshot
Expression of the Epstein-Barr virus BHRF1 gene, a homologue of Bcl-2, in nasopharyngeal carcinoma tissue
inferred← citeshttps://doi.org/10.1002/1097-0215(20000815)87:4<606::aid-ijc23>3.0.co;2-oopenalex-snapshot
Efficacy of ionizing radiation combined with adenoviral p53 therapy in EBV‐positive nasopharyngeal carcinoma
inferred← citeshttps://doi.org/10.1002/1097-0215(20001101)88:3<439::aid-ijc17>3.0.co;2-#openalex-snapshot
SH2D1A and slam protein expression in human lymphocytes and derived cell lines
inferred← citeshttps://doi.org/10.1002/cam4.1311openalex-snapshot
Antitumor effects of duvelisib on Epstein–Barr virus‐associated lymphoma cells
inferred← citeshttps://doi.org/10.1002/ijc.10498openalex-snapshot
SH2D1A expression in Burkitt lymphoma cells is restricted to EBV positive group I lines and is downregulated in parallel with immunoblastic transformation
inferred← citeshttps://doi.org/10.1002/ijc.2910570623openalex-snapshot
Endogenous TGF‐β contributes to the induction of the EBV lytic cycle in two burkitt lymphoma cell lines
inferred← citeshttps://doi.org/10.1002/jmv.20719openalex-snapshot
Loss of functional transforming growth factor (TGF)-β type II receptor results in insensitivity to TGF-β1-mediated apoptosis and Epstein–Barr virus reactivation
inferred← citeshttps://doi.org/10.1002/jmv.25999openalex-snapshot
Dexamethasone prevents the Epstein‐Barr virus induced epithelial‐mesenchymal transition in A549 cells
inferred← citeshttps://doi.org/10.1002/jmv.29955openalex-snapshot
EIF4A3 is stabilized by the long noncoding RNA BC200 to regulate gene expression during Epstein–Barr virus infection
inferred← citeshttps://doi.org/10.1002/jmv.70581openalex-snapshot
Model Systems of Gammaherpesvirus Infection, Immunity, and Disease
inferred← citeshttps://doi.org/10.1002/jmv.70883openalex-snapshot
EBV Genome Variations and Association With Diseases
inferred← citeshttps://doi.org/10.1002/jmv.71077openalex-snapshot
Clinical‐Stage Nanatinostat Triggers Productive Epstein‐Barr Virus Lytic Replication
inferred← citeshttps://doi.org/10.1002/path.4198openalex-snapshot
Suppression of the LMP2A target gene, EGR ‐1, protects Hodgkin's lymphoma cells from entry to the EBV lytic cycle
inferred← citeshttps://doi.org/10.1002/path.6040openalex-snapshot
EBV infection mediated BDNF expression is associated with bladder inflammation in interstitial cystitis/bladder pain syndrome with Hunner's lesion
inferred← citeshttps://doi.org/10.1006/prep.2000.1324openalex-snapshot
Bioreactor-Scale Production and One-Step Purification of Epstein–Barr Nuclear Antigen 1 Expressed in Baculovirus-Infected Insect Cells
inferred← citeshttps://doi.org/10.1006/scbi.2001.0413openalex-snapshot
The role of EBERs in oncogenesis
inferred← citeshttps://doi.org/10.1006/viro.1997.8647openalex-snapshot
A Noncanonical Poly(A) Signal, UAUAAA, and Flanking Elements in Epstein–Barr Virus DNA Polymerase mRNA Function in Cleavage and Polyadenylation Assays
inferred← citeshttps://doi.org/10.1006/viro.1998.9411openalex-snapshot
The Truncated Form of the Epstein–Barr Virus LMP-1 Is Dispensable or Complimentable by the Full-Length Form in Virus Infection and Replication
inferred← citeshttps://doi.org/10.1006/viro.1999.0082openalex-snapshot
Patterned Entry and Egress by Epstein–Barr Virus in Polarized CR2-Positive Epithelial Cells
inferred← citeshttps://doi.org/10.1007/978-3-642-56515-1_3openalex-snapshot
Epstein-Barr Virus Nuclear Protein 2-Induced Activation of the EBV-Replicative Cycle in Akata Cells: Analysis by Tetracycline-Regulated Expression
inferred← citeshttps://doi.org/10.1007/978-3-642-56515-1_9openalex-snapshot
Role of Epstein-Barr Virus in Burkitt’s Lymphoma
inferred← citeshttps://doi.org/10.1007/s00281-020-00792-2openalex-snapshot
Epigenetic lifestyle of Epstein-Barr virus
inferred← citeshttps://doi.org/10.1007/s00784-014-1387-yopenalex-snapshot
Prevalence and quantitative analysis of Epstein–Barr virus DNA and Porphyromonas gingivalis associated with Japanese chronic periodontitis patients
inferred← citeshttps://doi.org/10.1007/s11262-006-0010-yopenalex-snapshot
Difference of Epstein–Barr virus isolates from Japanese patients and African Burkitt’s lymphoma cell lines based on the sequence of latent membrane protein 1
inferred← citeshttps://doi.org/10.1007/s11262-008-0323-0openalex-snapshot
The evolution of Epstein-Barr virus inferred from the conservation and mutation of the virus glycoprotein gp350/220 gene
inferred← citeshttps://doi.org/10.1007/s12250-019-00097-1openalex-snapshot
Early Pattern of Epstein-Barr Virus Infection in Gastric Epithelial Cells by “Cell-in-cell”
inferred← citeshttps://doi.org/10.1016/0165-2478(93)90008-popenalex-snapshot
Induction of the lytic viral cycle in Epstein Barr virus carrying Burkitt lymphoma lines is accompanied by increased expression of major histocompatibility complex molecules
inferred← citeshttps://doi.org/10.1016/j.antiviral.2012.09.021openalex-snapshot
Protein kinase inhibitors that inhibit induction of lytic program and replication of Epstein–Barr virus
inferred← citeshttps://doi.org/10.1016/j.antiviral.2014.01.018openalex-snapshot
Small molecule inhibition of Epstein–Barr virus nuclear antigen-1 DNA binding activity interferes with replication and persistence of the viral genome
inferred← citeshttps://doi.org/10.1016/j.antiviral.2016.05.007openalex-snapshot
Luteolin inhibits Epstein-Barr virus lytic reactivation by repressing the promoter activities of immediate-early genes
inferred← citeshttps://doi.org/10.1016/j.antiviral.2019.02.015openalex-snapshot
GAP31 from an ancient medicinal plant exhibits anti-viral activity through targeting to Epstein-Barr virus nuclear antigen 1
inferred← citeshttps://doi.org/10.1016/j.bbrc.2010.02.122openalex-snapshot
KSHV-infected PEL cell lines exhibit a distinct gene expression profile
inferred← citeshttps://doi.org/10.1016/j.bbrc.2012.12.032openalex-snapshot
Modulation of Epstein–Barr Virus Nuclear Antigen 2-dependent transcription by protein arginine methyltransferase 5
inferred← citeshttps://doi.org/10.1016/j.bbrc.2013.03.022openalex-snapshot
The 5′ regulatory sequences of active miR-146a promoters are hypomethylated and associated with euchromatic histone modification marks in B lymphoid cells
inferred← citeshttps://doi.org/10.1016/j.bbrc.2020.01.017openalex-snapshot
Rta is an Epstein-Barr virus tegument protein that improves the stability of capsid protein BORF1
inferred← citeshttps://doi.org/10.1016/j.bbrc.2024.149984openalex-snapshot
Epstein-barr virus infections induce aberrant osteoclastogenesis in immune system-humanized NOD/Shi-scid/IL-2RγCnull mice
inferred← citeshttps://doi.org/10.1016/j.biocel.2016.09.006openalex-snapshot
Concomitant reduction of c-Myc expression and PI3K/AKT/mTOR signaling by quercetin induces a strong cytotoxic effect against Burkitt’s lymphoma
inferred← citeshttps://doi.org/10.1016/j.celrep.2013.09.012openalex-snapshot
Spontaneous Lytic Replication and Epitheliotropism Define an Epstein-Barr Virus Strain Found in Carcinomas
inferred← citeshttps://doi.org/10.1016/j.cyto.2011.12.001openalex-snapshot
Type I interferons directly down-regulate BCL-6 in primary and transformed germinal center B cells: Differential regulation in B cell lines derived from endemic or sporadic Burkitt’s lymphoma
inferred← citeshttps://doi.org/10.1016/j.ebiom.2014.11.007openalex-snapshot
Macroautophagy Proteins Assist Epstein Barr Virus Production and Get Incorporated Into the Virus Particles
inferred← citeshttps://doi.org/10.1016/j.ebiom.2016.05.025openalex-snapshot
Epstein–Barr Virus Infection of Mammary Epithelial Cells Promotes Malignant Transformation
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2009.06.019openalex-snapshot
H2O2 inhibits BCR-dependent immediate early induction of EBV genes in Burkitt's lymphoma cells
inferred← citeshttps://doi.org/10.1016/j.heliyon.2018.e00917openalex-snapshot
Calcium mobilization is responsible for Thapsigargin induced Epstein Barr virus lytic reactivation in in vitro immortalized lymphoblastoid cell lines
inferred← citeshttps://doi.org/10.1016/j.heliyon.2023.e21486openalex-snapshot
Momordica anti-HIV protein MAP30 abrogates the Epstein-Barr virus nuclear antigen 1 dependent functions in host cells
inferred← citeshttps://doi.org/10.1016/j.jviromet.2007.08.012openalex-snapshot
One-step multiplex real-time PCR assay to analyse the latency patterns of Epstein-Barr virus infection
inferred← citeshttps://doi.org/10.1016/j.jviromet.2017.04.012openalex-snapshot
Development of a robust, higher throughput green fluorescent protein (GFP)-based Epstein-Barr Virus (EBV) micro-neutralization assay
inferred← citeshttps://doi.org/10.1016/j.micinf.2003.12.017openalex-snapshot
Characterization of Epstein–Barr virus infection in a human signet ring cell gastric carcinoma cell line, HSC-39
inferred← citeshttps://doi.org/10.1016/j.micinf.2006.11.001openalex-snapshot
Accumulation of Epstein–Barr virus (EBV) BMRF1 protein EA-D during latent EBV activation of Burkitt's lymphoma cell line Raji
inferred← citeshttps://doi.org/10.1016/j.trsl.2013.02.008openalex-snapshot
Concurrent targeting of eicosanoid receptor 1/eicosanoid receptor 4 receptors and COX-2 induces synergistic apoptosis in Kaposi's sarcoma-associated herpesvirus and Epstein-Barr virus associated non-Hodgkin lymphoma cell lines
inferred← citeshttps://doi.org/10.1016/j.virol.2005.09.064openalex-snapshot
Genome-wide transcription program and expression of the Rta responsive gene of Epstein–Barr virus
inferred← citeshttps://doi.org/10.1016/j.virol.2009.01.006openalex-snapshot
The microRNAs of Epstein–Barr Virus are expressed at dramatically differing levels among cell lines
inferred← citeshttps://doi.org/10.1016/j.virol.2009.02.009openalex-snapshot
Activation of the lytic program of the Epstein–Barr virus in Burkitt's lymphoma cells leads to a two steps downregulation of expression of the proapoptotic protein BimEL, one of which is EBV-late-gene expression dependent
inferred← citeshttps://doi.org/10.1016/j.virol.2015.05.006openalex-snapshot
COX-2 induces lytic reactivation of EBV through PGE2 by modulating the EP receptor signaling pathway
inferred← citeshttps://doi.org/10.1016/j.virusres.2013.07.004openalex-snapshot
Downregulation of the polyamine regulator spermidine/spermine N1-acetyltransferase by Epstein–Barr virus in a Burkitt's lymphoma cell line
inferred← citeshttps://doi.org/10.1016/j.ymthe.2004.12.017openalex-snapshot
Eradication of epstein–barr virus episome and associated inhibition of infected tumor cell growth by adenovirus vector-mediated transduction of dominant-negative EBNA1
inferred← citeshttps://doi.org/10.1016/s0042-6822(95)80033-6openalex-snapshot
Characterization of the Epstein-Barr virus Fp promoter
inferred← citeshttps://doi.org/10.1016/s0065-230x(10)07004-1openalex-snapshot
Role of EBERs in the Pathogenesis of EBV Infection
inferred← citeshttps://doi.org/10.1016/s0166-0934(00)00233-0openalex-snapshot
Production monitoring and purification of EBV encoded latent membrane protein 1 expressed and secreted by recombinant baculovirus infected insect cells
inferred← citeshttps://doi.org/10.1016/s0166-0934(02)00233-1openalex-snapshot
Improved subtractive suppression hybridization combined with high density cDNA array screening identifies differentially expressed viral and cellular genes
inferred← citeshttps://doi.org/10.1023/a:1011139530768openalex-snapshot
Human T-cell Leukemia Virus Type-I (HTLV-I) Tax is not the Only One Factor to Enhance Human Immunodeficiency Virus Type-1 (HIV-1) Infection in Culture-supernatants
inferred← citeshttps://doi.org/10.1023/a:1012585709642openalex-snapshot
The Amino Acid Region 248–382 of the Epstein–Barr Virus Nuclear Protein 2 (EBNA2) is Responsible for the EBNA2-Induced EBV Reactivation
inferred← citeshttps://doi.org/10.1023/a:1020113906966openalex-snapshot
Distinct Patterns of Mitogen-Activated Protein Kinase Phosphorylation and Epstein–Barr Virus Gene Expression in Burkitt's Lymphoma Cell Lines versus B Lymphoblastoid Cell Lines
inferred← citeshttps://doi.org/10.1038/mt.2009.78openalex-snapshot
Oncolytic Rat Parvovirus H-1PV, a Candidate for the Treatment of Human Lymphoma: In Vitro and In Vivo Studies
inferred← citeshttps://doi.org/10.1038/onc.2008.75openalex-snapshot
Epstein–Barr virus-encoded small RNA induces IL-10 through RIG-I-mediated IRF-3 signaling
inferred← citeshttps://doi.org/10.1038/s41586-026-10416-8openalex-snapshot
EBV strain interacts with host HLA to drive nasopharyngeal carcinoma risk
inferred← citeshttps://doi.org/10.1038/s41598-025-94737-0openalex-snapshot
Comparison of nanopore with illumina whole genome assemblies of the Epstein-Barr virus in Burkitt lymphoma
inferred← citeshttps://doi.org/10.1038/sj.emboj.7601314openalex-snapshot
EB virus‐encoded RNAs are recognized by RIG‐I and activate signaling to induce type I IFN
inferred← citeshttps://doi.org/10.1038/sj.gt.3302602openalex-snapshot
Epstein–Barr virus vector-mediated gene transfer into human B cells: potential for antitumor vaccination
inferred← citeshttps://doi.org/10.1038/sj.onc.1207648openalex-snapshot
Mcl-1 is required for Akata6 B-lymphoma cell survival and is converted to a cell death molecule by efficient caspase-mediated cleavage
inferred← citeshttps://doi.org/10.1038/sj.onc.1209979openalex-snapshot
Distinct promoters mediate constitutive and inducible Bcl-XL expression in malignant lymphocytes
inferred← citeshttps://doi.org/10.1073/pnas.0305149101openalex-snapshot
Simvastatin induces apoptosis of Epstein-Barr virus (EBV)-transformed lymphoblastoid cell lines and delays development of EBV lymphomas
inferred← citeshttps://doi.org/10.1073/pnas.0609320103openalex-snapshot
Epstein–Barr nuclear antigen leader protein coactivates transcription through interaction with histone deacetylase 4
inferred← citeshttps://doi.org/10.1073/pnas.0730710100openalex-snapshot
T cell leukemia I oncogene expression depends on the presence of Epstein–Barr virus in the virus- carrying Burkitt lymphoma lines
inferred← citeshttps://doi.org/10.1073/pnas.1417891112openalex-snapshot
RETRACTED: Pan-viral-microRNA screening identifies interferon inhibition as a common function of diverse viruses
inferred← citeshttps://doi.org/10.1073/pnas.2336099100openalex-snapshot
Epstein-Barr virus provides a survival factor to Burkitt's lymphomas
inferred← citeshttps://doi.org/10.1073/pnas.92.23.10565openalex-snapshot
Redefining the Epstein-Barr virus-encoded nuclear antigen EBNA-1 gene promoter and transcription initiation site in group I Burkitt lymphoma cell lines.
inferred← citeshttps://doi.org/10.1074/jbc.273.2.932openalex-snapshot
Compensatory Energetic Relationships between Upstream Activators and the RNA Polymerase II General Transcription Machinery
inferred← citeshttps://doi.org/10.1074/jbc.m109.015123openalex-snapshot
Phosphorylation of p27Kip1 by Epstein-Barr Virus Protein Kinase Induces Its Degradation through SCFSkp2 Ubiquitin Ligase Actions during Viral Lytic Replication
inferred← citeshttps://doi.org/10.1074/jbc.m808466200openalex-snapshot
TORC2, a Coactivator of cAMP-response Element-binding Protein, Promotes Epstein-Barr Virus Reactivation from Latency through Interaction with Viral BZLF1 Protein
inferred← citeshttps://doi.org/10.1074/jbc.m809090200openalex-snapshot
Tubacin Kills Epstein-Barr Virus (EBV)-Burkitt Lymphoma Cells by Inducing Reactive Oxygen Species and EBV Lymphoblastoid Cells by Inducing Apoptosis
inferred← citeshttps://doi.org/10.1080/10428194.2016.1213823openalex-snapshot
Ganetespib, an HSP90 inhibitor, kills Epstein–Barr virus (EBV)-infected B and T cells and reduces the percentage of EBV-infected cells in the blood
inferred← citeshttps://doi.org/10.1080/19490976.2025.2519703openalex-snapshot
Gastrointestinal inflammation and cancer: viral and bacterial interplay
inferred← citeshttps://doi.org/10.1084/jem.20161017openalex-snapshot
Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and γδ T cells
inferred← citeshttps://doi.org/10.1086/515290openalex-snapshot
Eradication of Latent Epstein‐Barr Virus by Hydroxyurea Alters the Growth‐Transformed Cell Phenotype
inferred← citeshttps://doi.org/10.1089/153685904322772006openalex-snapshot
Characterization of Monoclonal Antibody to the Epstein-Barr Virus BHRF1 Protein, a Homologue of Bcl-2
inferred← citeshttps://doi.org/10.1089/omi.2016.0158openalex-snapshot
Understanding Epstein-Barr Virus Life Cycle with Proteomics: A Temporal Analysis of Ubiquitination During Virus Reactivation
inferred← citeshttps://doi.org/10.1093/emboj/21.5.954openalex-snapshot
Epstein–Barr virus RNA confers resistance to interferon‐α‐induced apoptosis in Burkitt's lymphoma
inferred← citeshttps://doi.org/10.1093/infdis/jit428openalex-snapshot
Oropharyngeal Group A Streptococcal Colonization Disrupts Latent Epstein-Barr Virus Infection
inferred← citeshttps://doi.org/10.1093/nar/gkn273openalex-snapshot
Conditional gene vectors regulated in cis
inferred← citeshttps://doi.org/10.1096/fj.201701253ropenalex-snapshot
Improving nuclear envelope dynamics by EBV BFRF1 facilitates intranuclear component clearance through autophagy
inferred← citeshttps://doi.org/10.1097/00007890-200109270-00006openalex-snapshot
PATIENTS AT RISK FOR DEVELOPMENT OF POSTTRANSPLANT LYMPHOPROLIFERATIVE DISORDER: PLASMA VERSUS PERIPHERAL BLOOD MONONUCLEAR CELLS AS MATERIAL FOR QUANTIFICATION OF EPSTEIN-BARR VIRAL LOAD BY USING REAL-TIME QUANTITATIVE POLYMERASE CHAIN REACTION1,2
inferred← citeshttps://doi.org/10.1099/0022-1317-73-2-455openalex-snapshot
One-step typing of Epstein-Barr virus by polymerase chain reaction: predominance of type 1 virus in Japan
inferred← citeshttps://doi.org/10.1099/0022-1317-80-11-2879openalex-snapshot
Assembly of the Epstein–Barr virus BBLF4, BSLF1 and BBLF2/3 proteins and their interactive properties
inferred← citeshttps://doi.org/10.1099/0022-1317-80-2-447openalex-snapshot
The major immunogenic epitopes of Epstein-Barr virus (EBV) nuclear antigen 1 are encoded by sequence domains which vary among nasopharyngeal carcinoma biopsies and EBV-associated cell lines.
inferred← citeshttps://doi.org/10.1099/0022-1317-82-10-2373openalex-snapshot
Roles of Epstein–Barr virus glycoproteins gp350 and gp25 in the infection of human epithelial cells
inferred← citeshttps://doi.org/10.1099/0022-1317-82-6-1451openalex-snapshot
Role of Epstein–Barr virus-encoded latent membrane protein 2A on virus-induced immortalization and virus activation
inferred← citeshttps://doi.org/10.1099/jgv.0.000605openalex-snapshot
Epstein–Barr virus exploits host endocytic machinery for cell-to-cell viral transmission rather than a virological synapse
inferred← citeshttps://doi.org/10.1099/jgv.0.000891openalex-snapshot
Molecular diversity of IgG responses to Epstein–Barr virus proteins in asymptomatic Epstein–Barr virus carriers
inferred← citeshttps://doi.org/10.1099/jgv.0.001056openalex-snapshot
Mechanism of activation of the BNLF2a immune evasion gene of Epstein-Barr virus by Zta
inferred← citeshttps://doi.org/10.1099/vir.0.012831-0openalex-snapshot
Quantitative evaluation of the role of Epstein–Barr virus immediate-early protein BZLF1 in B-cell transformation
inferred← citeshttps://doi.org/10.1099/vir.0.81313-0openalex-snapshot
Detection of Epstein–Barr virus BGLF4 protein kinase in virus replication compartments and virus particles
inferred← citeshttps://doi.org/10.1111/cas.12324openalex-snapshot
Enhanced susceptibility of B lymphoma cells to measles virus by E pstein– B arr virus type III latency that upregulates CD 150/signaling lymphocytic activation molecule
inferred← citeshttps://doi.org/10.1111/j.1348-0421.2007.tb03885.xopenalex-snapshot
The Role of p38 Mitogen‐Activated Protein Kinase in Regulating Interleukin‐10 Gene Expression in Burkitt's Lymphoma Cell Lines
inferred← citeshttps://doi.org/10.1111/j.1349-7006.2011.01924.xopenalex-snapshot
Cytokine signatures of transformed B cells with distinct Epstein–Barr virus latencies as a potential diagnostic tool for B cell lymphoma
inferred← citeshttps://doi.org/10.1111/j.1442-200x.1997.tb03574.xopenalex-snapshot
Epstein‐Barr virus infection, Hodgkin's disease, non‐Hodgkin's lymphoma, and reactive follicular hyperplasia in Japanese children: Evaluation of paraffin‐embedded specimens using polymerase chain reaction and immunohistochemistry
inferred← citeshttps://doi.org/10.1128/jvi.00120-07openalex-snapshot
Epstein-Barr Virus BGLF4 Kinase Induces Premature Chromosome Condensation through Activation of Condensin and Topoisomerase II
inferred← citeshttps://doi.org/10.1128/jvi.00336-11openalex-snapshot
cis -Acting Effects on RNA Processing and Drosha Cleavage Prevent Epstein-Barr Virus Latency III BHRF1 Expression
inferred← citeshttps://doi.org/10.1128/jvi.00381-10openalex-snapshot
Mapping the N-Terminal Residues of Epstein-Barr Virus gp42 That Bind gH/gL by Using Fluorescence Polarization and Cell-Based Fusion Assays
inferred← citeshttps://doi.org/10.1128/jvi.00575-07openalex-snapshot
Binding-Site Interactions between Epstein-Barr Virus Fusion Proteins gp42 and gH/gL Reveal a Peptide That Inhibits both Epithelial and B-Cell Membrane Fusion
inferred← citeshttps://doi.org/10.1128/jvi.00626-25openalex-snapshot
Assessing the fitness of Epstein-Barr virus following its reactivation
inferred← citeshttps://doi.org/10.1128/jvi.00712-12openalex-snapshot
Roles of Cell Signaling Pathways in Cell-to-Cell Contact-Mediated Epstein-Barr Virus Transmission
inferred← citeshttps://doi.org/10.1128/jvi.00722-14openalex-snapshot
Activation and Repression of Epstein-Barr Virus and Kaposi's Sarcoma-Associated Herpesvirus Lytic Cycles by Short- and Medium-Chain Fatty Acids
inferred← citeshttps://doi.org/10.1128/jvi.00747-17openalex-snapshot
Drug Modulators of B Cell Signaling Pathways and Epstein-Barr Virus Lytic Activation
inferred← citeshttps://doi.org/10.1128/jvi.00923-10openalex-snapshot
Cellular MicroRNAs 200b and 429 Regulate the Epstein-Barr Virus Switch between Latency and Lytic Replication
inferred← citeshttps://doi.org/10.1128/jvi.00927-19openalex-snapshot
Identifying the Cellular Interactome of Epstein-Barr Virus Lytic Regulator Zta Reveals Cellular Targets Contributing to Viral Replication
inferred← citeshttps://doi.org/10.1128/jvi.00982-07openalex-snapshot
De Novo Protein Synthesis Is Required for Lytic Cycle Reactivation of Epstein-Barr Virus, but Not Kaposi's Sarcoma-Associated Herpesvirus, in Response to Histone Deacetylase Inhibitors and Protein Kinase C Agonists
inferred← citeshttps://doi.org/10.1128/jvi.00998-19openalex-snapshot
Pharmacologic Activation of Lytic Epstein-Barr Virus Gene Expression without Virion Production
inferred← citeshttps://doi.org/10.1128/jvi.01172-09openalex-snapshot
Stimulus Duration and Response Time Independently Influence the Kinetics of Lytic Cycle Reactivation of Epstein-Barr Virus
inferred← citeshttps://doi.org/10.1128/jvi.01197-10openalex-snapshot
Transforming Growth Factor β-Induced Reactivation of Epstein-Barr Virus Involves Multiple Smad-Binding Elements Cooperatively Activating Expression of the Latent-Lytic Switch BZLF1 Gene
inferred← citeshttps://doi.org/10.1128/jvi.01518-06openalex-snapshot
Characterization of the Uracil-DNA Glycosylase Activity of Epstein-Barr Virus BKRF3 and Its Role in Lytic Viral DNA Replication
inferred← citeshttps://doi.org/10.1128/jvi.01593-18openalex-snapshot
A Genome-Wide Epstein-Barr Virus Polyadenylation Map and Its Antisense RNA to EBNA
inferred← citeshttps://doi.org/10.1128/jvi.01822-06openalex-snapshot
EBNA2 Interferes with the Germinal Center Phenotype by Downregulating BCL6 and TCL1 in Non-Hodgkin's Lymphoma Cells
inferred← citeshttps://doi.org/10.1128/jvi.01899-23openalex-snapshot
BGLF4 kinase regulates the formation of the EBV cytoplasmic assembly compartment and the recruitment of cellular IQGAP1 for virion release
inferred← citeshttps://doi.org/10.1128/jvi.01979-08openalex-snapshot
Distinctive Effects of the Epstein-Barr Virus Family of Repeats on Viral Latent Gene Promoter Activity and B-Lymphocyte Transformation
inferred← citeshttps://doi.org/10.1128/jvi.02028-19openalex-snapshot
B Cell-Specific Transcription Activator PAX5 Recruits p300 To Support EBNA1-Driven Transcription
inferred← citeshttps://doi.org/10.1128/jvi.02114-16openalex-snapshot
The SWI/SNF Chromatin Regulator BRG1 Modulates the Transcriptional Regulatory Activity of the Epstein-Barr Virus DNA Polymerase Processivity Factor BMRF1
inferred← citeshttps://doi.org/10.1128/jvi.02241-06openalex-snapshot
Bortezomib Induces Apoptosis of Epstein-Barr Virus (EBV)-Transformed B Cells and Prolongs Survival of Mice Inoculated with EBV-Transformed B Cells
inferred← citeshttps://doi.org/10.1128/jvi.02244-06openalex-snapshot
Epstein-Barr Virus BHRF1 Micro- and Stable RNAs during Latency III and after Induction of Replication
inferred← citeshttps://doi.org/10.1128/jvi.02706-09openalex-snapshot
Either ZEB1 or ZEB2/SIP1 Can Play a Central Role in Regulating the Epstein-Barr Virus Latent-Lytic Switch in a Cell-Type-Specific Manner
inferred← citeshttps://doi.org/10.1128/jvi.02880-14openalex-snapshot
BGLF4 Kinase Modulates the Structure and Transport Preference of the Nuclear Pore Complex To Facilitate Nuclear Import of Epstein-Barr Virus Lytic Proteins
inferred← citeshttps://doi.org/10.1128/jvi.03189-14openalex-snapshot
Clustered MicroRNAs of the Epstein-Barr Virus Cooperatively Downregulate an Epithelial Cell-Specific Metastasis Suppressor
inferred← citeshttps://doi.org/10.1128/jvi.06829-11openalex-snapshot
Latency Type-Dependent Modulation of Epstein-Barr Virus-Encoded Latent Membrane Protein 1 Expression by Type I Interferons in B Cells
inferred← citeshttps://doi.org/10.1128/jvi.66.9.5590-5593.1992openalex-snapshot
Partial elimination of Epstein-Barr virus plasmids from Burkitt's lymphoma cells by transfecting the BZLF1 gene
inferred← citeshttps://doi.org/10.1128/jvi.67.6.3240-3245.1993openalex-snapshot
Analysis of the BZLF1 promoter of Epstein-Barr virus: identification of an anti-immunoglobulin response sequence
inferred← citeshttps://doi.org/10.1128/jvi.67.8.4651-4658.1993openalex-snapshot
Functional expression and characterization of the Epstein-Barr virus DNA polymerase catalytic subunit
inferred← citeshttps://doi.org/10.1128/jvi.68.9.6069-6073.1994openalex-snapshot
Isolation of Epstein-Barr virus (EBV)-negative cell clones from the EBV-positive Burkitt's lymphoma (BL) line Akata: malignant phenotypes of BL cells are dependent on EBV
inferred← citeshttps://doi.org/10.1128/jvi.69.8.5039-5047.1995openalex-snapshot
The Epstein-Barr virus BamHI F promoter is an early lytic promoter: lack of correlation with EBNA 1 gene transcription in group 1 Burkitt's lymphoma cell lines
inferred← citeshttps://doi.org/10.1128/jvi.70.10.7260-7263.1996openalex-snapshot
Clonal propagation of Epstein-Barr virus (EBV) recombinants in EBV-negative Akata cells
inferred← citeshttps://doi.org/10.1128/jvi.70.6.3423-3431.1996openalex-snapshot
Plasmid-like replicative intermediates of the Epstein-Barr virus lytic origin of DNA replication
inferred← citeshttps://doi.org/10.1128/jvi.70.9.5758-5768.1996openalex-snapshot
oriP is essential for EBNA gene promoter activity in Epstein-Barr virus-immortalized lymphoblastoid cell lines
inferred← citeshttps://doi.org/10.1128/jvi.72.1.158-163.1998openalex-snapshot
Epstein-Barr Virus Lacking Glycoprotein gp42 Can Bind to B Cells but Is Not Able To Infect
inferred← citeshttps://doi.org/10.1128/jvi.72.10.8043-8051.1998openalex-snapshot
The Epstein-Barr Virus Immediate-Early Gene Product, BRLF1, Interacts with the Retinoblastoma Protein during the Viral Lytic Cycle
inferred← citeshttps://doi.org/10.1128/jvi.72.11.8485-8492.1998openalex-snapshot
The Epstein-Barr Virus (EBV) SM Protein Enhances Pre-mRNA Processing of the EBV DNA Polymerase Transcript
inferred← citeshttps://doi.org/10.1128/jvi.72.11.9150-9156.1998openalex-snapshot
Epstein-Barr Virus Contributes to the Malignant Phenotype and to Apoptosis Resistance in Burkitt’s Lymphoma Cell Line Akata
inferred← citeshttps://doi.org/10.1128/jvi.73.12.9827-9831.1999openalex-snapshot
Oncogenic Role of Epstein-Barr Virus-Encoded RNAs in Burkitt’s Lymphoma Cell Line Akata
inferred← citeshttps://doi.org/10.1128/jvi.73.4.3154-3161.1999openalex-snapshot
Expression of EBNA-1 mRNA Is Regulated by Cell Cycle during Epstein-Barr Virus Type I Latency
inferred← citeshttps://doi.org/10.1128/jvi.73.6.4678-4688.1999openalex-snapshot
Control of Cell Cycle Entry and Apoptosis in B Lymphocytes Infected by Epstein-Barr Virus
inferred← citeshttps://doi.org/10.1128/jvi.73.6.5214-5219.1999openalex-snapshot
Epstein-Barr Virus (EBV) Nuclear Protein 2-Induced Disruption of EBV Latency in the Burkitt’s Lymphoma Cell Line Akata: Analysis by Tetracycline-Regulated Expression
inferred← citeshttps://doi.org/10.1128/jvi.74.22.10838-10845.2000openalex-snapshot
Latent Membrane Protein 2A-Mediated Effects on the Phosphatidylinositol 3-Kinase/Akt Pathway
inferred← citeshttps://doi.org/10.1128/jvi.74.23.11162-11172.2000openalex-snapshot
Epstein-Barr Virus That Lacks Glycoprotein gN Is Impaired in Assembly and Infection
inferred← citeshttps://doi.org/10.1128/jvi.75.20.9977-9982.2001openalex-snapshot
Replacement of the Epstein-Barr Virus Plasmid with the EBER Plasmid in Burkitt's Lymphoma Cells
inferred← citeshttps://doi.org/10.1128/jvi.75.3.1561-1564.2001openalex-snapshot
Role of bcl-2 in Epstein-Barr Virus-Induced Malignant Conversion of Burkitt's Lymphoma Cell Line Akata
inferred← citeshttps://doi.org/10.1128/jvi.78.13.7004-7015.2004openalex-snapshot
Production of High-Titer Epstein-Barr Virus Recombinants Derived from Akata Cells by Using a Bacterial Artificial Chromosome System
inferred← citeshttps://doi.org/10.1128/jvi.78.17.9412-9422.2004openalex-snapshot
Epstein-Barr Virus (EBV) SM Protein Induces and Recruits Cellular Sp110b To Stabilize mRNAs and Enhance EBV Lytic Gene Expression
inferred← citeshttps://doi.org/10.1128/jvi.79.19.12280-12285.2005openalex-snapshot
Epstein-Barr Virus-Encoded Poly(A) − RNA Confers Resistance to Apoptosis Mediated through Fas by Blocking the PKR Pathway in Human Epithelial Intestine 407 Cells
inferred← citeshttps://doi.org/10.1128/jvi.79.7.4415-4424.2005openalex-snapshot
Epstein-Barr Virus Transforming Protein LMP1 Plays a Critical Role in Virus Production
inferred← citeshttps://doi.org/10.1128/jvi.79.9.5875-5879.2005openalex-snapshot
Induction of Lytic Epstein-Barr Virus (EBV) Infection by Synergistic Action of Rituximab and Dexamethasone Renders EBV-Positive Lymphoma Cells More Susceptible to Ganciclovir Cytotoxicity In Vitro and In Vivo
inferred← citeshttps://doi.org/10.1128/jvi.80.5.2548-2565.2006openalex-snapshot
Virus and Cell RNAs Expressed during Epstein-Barr Virus Replication
inferred← citeshttps://doi.org/10.1128/mbio.02338-23openalex-snapshot
Characterization of target gene regulation by the two Epstein-Barr virus oncogene LMP1 domains essential for B-cell transformation
inferred← citeshttps://doi.org/10.1128/mcb.17.1.364openalex-snapshot
Host-Cell-Determined Methylation of Specific Epstein-Barr Virus Promoters Regulates the Choice between Distinct Viral Latency Programs
inferred← citeshttps://doi.org/10.1128/mcb.17.2.873openalex-snapshot
Constitutive Activation of Epstein-Barr Virus (EBV) Nuclear Antigen 1 Gene Transcription by IRF1 and IRF2 during Restricted EBV Latency
inferred← citeshttps://doi.org/10.1128/mcb.22.7.2136-2146.2002openalex-snapshot
Protein Kinase A Associates with HA95 and Affects Transcriptional Coactivation by Epstein-Barr Virus Nuclear Proteins
inferred← citeshttps://doi.org/10.1158/0008-5472.can-03-1562openalex-snapshot
Epstein-Barr Virus Infection of Human Natural Killer Cell Lines and Peripheral Blood Natural Killer Cells
inferred← citeshttps://doi.org/10.1158/1078-0432.ccr-24-1142openalex-snapshot
The Epstein–Barr Virus Nuclear Antigen 1 Variant Associated with Nasopharyngeal Carcinoma Defines the Sequence Criteria for Serologic Risk Prediction
inferred← citeshttps://doi.org/10.1172/jci158978openalex-snapshot
An allosteric inhibitor of sirtuin 2 deacetylase activity exhibits broad-spectrum antiviral activity
inferred← citeshttps://doi.org/10.1182/blood-2005-06-2569openalex-snapshot
IL-10 can induce the expression of EBV-encoded latent membrane protein-1 (LMP-1) in the absence of EBNA-2 in B lymphocytes and in Burkitt lymphoma- and NK lymphoma-derived cell lines
inferred← citeshttps://doi.org/10.1182/blood-2006-08-040634openalex-snapshot
Hsp72 up-regulates Epstein-Barr virus EBNALP coactivation with EBNA2
inferred← citeshttps://doi.org/10.1182/blood.v89.5.1665openalex-snapshot
Lytic Replication of Epstein-Barr Virus in the Peripheral Blood: Analysis of Viral Gene Expression in B Lymphocytes During Infectious Mononucleosis and in the Normal Carrier State
inferred← citeshttps://doi.org/10.1182/blood.v90.3.1200openalex-snapshot
Human Herpesvirus 6 (HHV-6)–Positive Burkitt's Lymphoma: Establishment of a Novel Cell Line Infected With HHV-6
inferred← citeshttps://doi.org/10.1186/1465-9921-7-114openalex-snapshot
Microarray identifies ADAM family members as key responders to TGF-β1 in alveolar epithelial cells
inferred← citeshttps://doi.org/10.1186/1742-4690-11-11openalex-snapshot
Cyclophilin A promotes HIV-1 reverse transcription but its effect on transduction correlates best with its effect on nuclear entry of viral cDNA
inferred← citeshttps://doi.org/10.1186/1743-422x-10-314openalex-snapshot
Comprehensive profiling of Epstein-Barr virus-encoded miRNA species associated with specific latency types in tumor cells
inferred← citeshttps://doi.org/10.1186/s12903-017-0438-6openalex-snapshot
Prevalence of Epstein-Barr virus DNA and Porphyromonas gingivalis in Japanese peri-implantitis patients
inferred← citeshttps://doi.org/10.1186/s40880-016-0079-1openalex-snapshot
Co-infection of Epstein-Barr virus and human papillomavirus in human tumorigenesis
inferred← citeshttps://doi.org/10.1369/jhc.5a6646.2005openalex-snapshot
Genetic Immunization: A New Monoclonal Antibody for the Detection of BCL-6 Protein in Paraffin Sections
inferred← citeshttps://doi.org/10.1371/journal.pbio.0030404openalex-snapshot
Epstein-Barr Virus Provides a New Paradigm: A Requirement for the Immediate Inhibition of Apoptosis
inferred← citeshttps://doi.org/10.1371/journal.pone.0021176openalex-snapshot
Id1 Interacts and Stabilizes the Epstein-Barr Virus Latent Membrane Protein 1 (LMP1) in Nasopharyngeal Epithelial Cells
inferred← citeshttps://doi.org/10.1371/journal.pone.0027758openalex-snapshot
Unexpected Instability of Family of Repeats (FR), the Critical cis-Acting Sequence Required for EBV Latent Infection, in EBV-BAC Systems
inferred← citeshttps://doi.org/10.1371/journal.pone.0028506openalex-snapshot
Latent Epstein-Barr Virus Can Inhibit Apoptosis in B Cells by Blocking the Induction of NOXA Expression
inferred← citeshttps://doi.org/10.1371/journal.pone.0051306openalex-snapshot
Resveratrol Prevents EBV Transformation and Inhibits the Outgrowth of EBV-Immortalized Human B Cells
inferred← citeshttps://doi.org/10.1371/journal.pone.0106788openalex-snapshot
Gene Expression Profiling Identifies IRF4-Associated Molecular Signatures in Hematological Malignancies
inferred← citeshttps://doi.org/10.1371/journal.pone.0112564openalex-snapshot
CD137 Expression Is Induced by Epstein-Barr Virus Infection through LMP1 in T or NK Cells and Mediates Survival Promoting Signals
inferred← citeshttps://doi.org/10.1371/journal.ppat.1001294openalex-snapshot
A Viral microRNA Cluster Strongly Potentiates the Transforming Properties of a Human Herpesvirus
inferred← citeshttps://doi.org/10.1371/journal.ppat.1003084openalex-snapshot
The Nuclear Chaperone Nucleophosmin Escorts an Epstein-Barr Virus Nuclear Antigen to Establish Transcriptional Cascades for Latent Infection in Human B Cells
inferred← citeshttps://doi.org/10.1371/journal.ppat.1005414openalex-snapshot
BS69/ZMYND11 C-Terminal Domains Bind and Inhibit EBNA2
inferred← citeshttps://doi.org/10.1371/journal.ppat.1008365openalex-snapshot
B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection
inferred← citeshttps://doi.org/10.1371/journal.ppat.1010453openalex-snapshot
Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells
inferred← citeshttps://doi.org/10.1371/journal.ppat.1011040openalex-snapshot
New insight into Epstein-Barr virus infection using models of stratified epithelium
inferred← citeshttps://doi.org/10.1371/journal.ppat.1011477openalex-snapshot
Changes in SUMO-modified proteins in Epstein-Barr virus infection identifies reciprocal regulation of TRIM24/28/33 complexes and the lytic switch BZLF1
inferred← citeshttps://doi.org/10.1371/journal.ppat.1012132openalex-snapshot
Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice
inferred← citeshttps://doi.org/10.1371/journal.ppat.1012934openalex-snapshot
Inhibition of the Integrated stress response by Epstein-Barr virus oncoprotein LMP1 attenuates epithelial cell differentiation and lytic viral reactivation
inferred← citeshttps://doi.org/10.1385/1-59259-227-9:243openalex-snapshot
Biosynthetic Radiolabeling of Virus Glycoproteins for Immunoprecipitation and Electrophoretic Analysis
inferred← citeshttps://doi.org/10.1620/tjem.170.71openalex-snapshot
High Epstein-Barr Virus(EBV) Susceptibility of Both Lymphoblastoid and Lymphoma Cell Lines Derived from a Japanese Patient with EBV Genome-Positive Burkitt's Lymphoma.
inferred← citeshttps://doi.org/10.18632/oncotarget.2098openalex-snapshot
The translation inhibitor silvestrol exhibits direct anti-tumor activity while preserving innate and adaptive immunity against EBV-driven lymphoproliferative disease
inferred← citeshttps://doi.org/10.21203/rs.2.11335/v1openalex-snapshot
Effects of initial periodontal therapy on the prevalence of Epstein-Barr virus DNA and Porphyromonas gingivalis in Japanese chronic periodontitis patients
inferred← citeshttps://doi.org/10.21203/rs.2.16315/v1openalex-snapshot
Effects of initial periodontal therapy on the prevalence of Epstein-Barr virus DNA and Porphyromonas gingivalis in Japanese chronic periodontitis patients
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