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The UPR in atherosclerosis

https://doi.org/10.1007/s00281-013-0372-x

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/biof.17openalex-snapshot

Contributions of hyperhomocysteinemia to atherosclerosis: Causal relationship and potential mechanisms

inferredcites →https://doi.org/10.1002/eji.201141717openalex-snapshot

Regulatory macrophages: Setting the Threshold for Therapy

inferredcites →https://doi.org/10.1016/0092-8674(93)90521-qopenalex-snapshot

A transmembrane protein with a cdc2+CDC28-related kinase activity is required for signaling from the ER to the nucleus

inferredcites →https://doi.org/10.1016/0092-8674(93)90648-aopenalex-snapshot

Transcriptional induction of genes encoding endoplasmic reticulum resident proteins requires a transmembrane protein kinase

inferredcites →https://doi.org/10.1016/j.atherosclerosis.2009.04.019openalex-snapshot

Free cholesterol overloading induced smooth muscle cells death and activated both ER- and mitochondrial-dependent death pathway

inferredcites →https://doi.org/10.1016/j.bcp.2005.03.017openalex-snapshot

Quercetin, but not rutin and quercitrin, prevention of H2O2-induced apoptosis via anti-oxidant activity and heme oxygenase 1 gene expression in macrophages

inferredcites →https://doi.org/10.1016/j.cell.2007.04.027openalex-snapshot

ER Stress Triggers Apoptosis by Activating BH3-Only Protein Bim

inferredcites →https://doi.org/10.1016/j.cell.2009.07.017openalex-snapshot

IRE1α Kinase Activation Modes Control Alternate Endoribonuclease Outputs to Determine Divergent Cell Fates

inferredcites →https://doi.org/10.1016/j.cell.2010.02.034openalex-snapshot

Endoplasmic Reticulum Stress and the Inflammatory Basis of Metabolic Disease

inferredcites →https://doi.org/10.1016/j.cmet.2009.03.003openalex-snapshot

Reduced Apoptosis and Plaque Necrosis in Advanced Atherosclerotic Lesions of Apoe−/− and Ldlr−/− Mice Lacking CHOP

inferredcites →https://doi.org/10.1016/j.cmet.2010.09.010openalex-snapshot

Atherogenic Lipids and Lipoproteins Trigger CD36-TLR2-Dependent Apoptosis in Macrophages Undergoing Endoplasmic Reticulum Stress

inferredcites →https://doi.org/10.1016/j.cmet.2012.07.007openalex-snapshot

IRE1α Induces Thioredoxin-Interacting Protein to Activate the NLRP3 Inflammasome and Promote Programmed Cell Death under Irremediable ER Stress

inferredcites →https://doi.org/10.1016/j.mam.2010.09.002openalex-snapshot

Flavonols and cardiovascular disease

inferredcites →https://doi.org/10.1016/j.mcn.2008.01.014openalex-snapshot

Loss of c/EBP-β activity promotes the adaptive to apoptotic switch in hypoxic cortical neurons

inferredcites →https://doi.org/10.1016/j.molcel.2010.04.001openalex-snapshot

Flavonol Activation Defines an Unanticipated Ligand-Binding Site in the Kinase-RNase Domain of IRE1

inferredcites →https://doi.org/10.1016/j.numecd.2004.04.002openalex-snapshot

Is homocysteine important as risk factor for coronary heart disease?

inferredcites →https://doi.org/10.1016/s0008-6363(98)00212-0openalex-snapshot

Apoptosis of vascular smooth muscle cells in vascular remodelling and atherosclerotic plaque rupture

inferredcites →https://doi.org/10.1016/s0022-2275(20)32363-4openalex-snapshot

Oxidized LDL induces the expression of ALBP/aP2 mRNA and protein in human THP-1 macrophages

inferredcites →https://doi.org/10.1016/s0140-6736(96)07495-8openalex-snapshot

Global mortality, disability, and the contribution of risk factors: Global Burden of Disease Study

inferredcites →https://doi.org/10.1021/jf803390mopenalex-snapshot

Blockade of Oxidized LDL-Triggered Endothelial Apoptosis by Quercetin and Rutin through Differential Signaling Pathways Involving JAK2

inferredcites →https://doi.org/10.1038/89076openalex-snapshot

Lack of macrophage fatty-acid–binding protein aP2 protects mice deficient in apolipoprotein E against atherosclerosis

inferredcites →https://doi.org/10.1038/cddis.2011.132openalex-snapshot

ER stress activates the NLRP3 inflammasome via an UPR-independent pathway

inferredcites →https://doi.org/10.1038/nature04399openalex-snapshot

Role of cholesterol and lipid organization in disease

inferredcites →https://doi.org/10.1038/nature06796openalex-snapshot

Translating molecular discoveries into new therapies for atherosclerosis

inferredcites →https://doi.org/10.1038/nature07203openalex-snapshot

From endoplasmic-reticulum stress to the inflammatory response

inferredcites →https://doi.org/10.1038/ncb0311-184openalex-snapshot

Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress

inferredcites →https://doi.org/10.1038/ncb1035openalex-snapshot

The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages

inferredcites →https://doi.org/10.1038/nm.2067openalex-snapshot

Reducing endoplasmic reticulum stress through a macrophage lipid chaperone alleviates atherosclerosis

inferredcites →https://doi.org/10.1038/nrm2199openalex-snapshot

Signal integration in the endoplasmic reticulum unfolded protein response

inferredcites →https://doi.org/10.1038/nrm3270openalex-snapshot

The unfolded protein response: controlling cell fate decisions under ER stress and beyond

inferredcites →https://doi.org/10.1038/sj.embor.7400779openalex-snapshot

Mediators of endoplasmic reticulum stress‐induced apoptosis

inferredcites →https://doi.org/10.1056/nejmra043430openalex-snapshot

Inflammation, Atherosclerosis, and Coronary Artery Disease

inferredcites →https://doi.org/10.1073/pnas.0438011100openalex-snapshot

JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis

inferredcites →https://doi.org/10.1073/pnas.0509487102openalex-snapshot

On the mechanism of sensing unfolded protein in the endoplasmic reticulum

inferredcites →https://doi.org/10.1073/pnas.0605457103openalex-snapshot

Identification of inflammatory gene modules based on variations of human endothelial cell responses to oxidized lipids

inferredcites →https://doi.org/10.1073/pnas.0609671104openalex-snapshot

Combinatorial pattern recognition receptor signaling alters the balance of life and death in macrophages

inferredcites →https://doi.org/10.1073/pnas.0903197106openalex-snapshot

Sustained activation of XBP1 splicing leads to endothelial apoptosis and atherosclerosis development in response to disturbed flow

inferredcites →https://doi.org/10.1074/jbc.m100747200openalex-snapshot

Homocysteine Induces Programmed Cell Death in Human Vascular Endothelial Cells through Activation of the Unfolded Protein Response

inferredcites →https://doi.org/10.1074/jbc.m111.338673openalex-snapshot

Endoplasmic Reticulum Stress Controls M2 Macrophage Differentiation and Foam Cell Formation

inferredcites →https://doi.org/10.1074/jbc.m212897200openalex-snapshot

TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia

inferredcites →https://doi.org/10.1074/jbc.m405195200openalex-snapshot

Enrichment of Endoplasmic Reticulum with Cholesterol Inhibits Sarcoplasmic-Endoplasmic Reticulum Calcium ATPase-2b Activity in Parallel with Increased Order of Membrane Lipids

inferredcites →https://doi.org/10.1074/jbc.m501759200openalex-snapshot

Free Cholesterol-loaded Macrophages Are an Abundant Source of Tumor Necrosis Factor-α and Interleukin-6

inferredcites →https://doi.org/10.1083/jcb.153.5.1011openalex-snapshot

Feedback Inhibition of the Unfolded Protein Response by GADD34-Mediated Dephosphorylation of eIF2α

inferredcites →https://doi.org/10.1083/jcb.200904060openalex-snapshot

Role of ERO1-α–mediated stimulation of inositol 1,4,5-triphosphate receptor activity in endoplasmic reticulum stress–induced apoptosis

inferredcites →https://doi.org/10.1083/jcb.201003138openalex-snapshot

Regulation of basal cellular physiology by the homeostatic unfolded protein response

inferredcites →https://doi.org/10.1091/mbc.e05-12-1107openalex-snapshot

Caspase-2–induced Apoptosis Requires Bid Cleavage: A Physiological Role for Bid in Heat Shock–induced Death

inferredcites →https://doi.org/10.1096/fj.07-105353openalex-snapshot

Hyperhomocysteinemia induced by methionine supplementation does not independently cause atherosclerosis in C57BL/6J mice

inferredcites →https://doi.org/10.1096/fj.10-159319openalex-snapshot

Induction of the unfolded protein response after monocyte to macrophage differentiation augments cell survival in early atherosclerotic lesions

inferredcites →https://doi.org/10.1096/fj.11-182931openalex-snapshot

Peptides derived from the bifunctional kinase/RNase enzyme IRE1α modulate IRE1α activity and protect cells from endoplasmic reticulum stress

inferredcites →https://doi.org/10.1097/00041433-199810000-00012openalex-snapshot

The response‐to‐retention hypothesis of atherogenesis reinforced

inferredcites →https://doi.org/10.1101/gad.1250704openalex-snapshot

CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum

inferredcites →https://doi.org/10.1111/j.1365-2249.2005.02934.xopenalex-snapshot

Macrophage activation switching: an asset for the resolution of inflammation

inferredcites →https://doi.org/10.1111/j.1463-1326.2010.01282.xopenalex-snapshot

Restoring endoplasmic reticulum function by chemical chaperones: an emerging therapeutic approach for metabolic diseases

inferredcites →https://doi.org/10.1126/science.1090031openalex-snapshot

Bypassing a Kinase Activity with an ATP-Competitive Drug

inferredcites →https://doi.org/10.1126/science.1101902openalex-snapshot

A Selective Inhibitor of eIF2α Dephosphorylation Protects Cells from ER Stress

inferredcites →https://doi.org/10.1126/science.1128294openalex-snapshot

Chemical Chaperones Reduce ER Stress and Restore Glucose Homeostasis in a Mouse Model of Type 2 Diabetes

inferredcites →https://doi.org/10.1126/science.1201396openalex-snapshot

Selective Inhibition of a Regulatory Subunit of Protein Phosphatase 1 Restores Proteostasis

inferredcites →https://doi.org/10.1126/science.1226191openalex-snapshot

IRE1α Cleaves Select microRNAs During ER Stress to Derepress Translation of Proapoptotic Caspase-2

inferredcites →https://doi.org/10.1126/science.287.5453.664openalex-snapshot

Coupling of Stress in the ER to Activation of JNK Protein Kinases by Transmembrane Protein Kinase IRE1

inferredcites →https://doi.org/10.1128/mcb.01507-09openalex-snapshot

Molecular Symbiosis of CHOP and C/EBPβ Isoform LIP Contributes to Endoplasmic Reticulum Stress-Induced Apoptosis

inferredcites →https://doi.org/10.1128/mcb.19.12.8469openalex-snapshot

BCL-2 Is Phosphorylated and Inactivated by an ASK1/Jun N-Terminal Protein Kinase Pathway Normally Activated at G2/M

inferredcites →https://doi.org/10.1128/mcb.21.4.1249-1259.2001openalex-snapshot

Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State

inferredcites →https://doi.org/10.1128/mcb.24.24.10703-10717.2004openalex-snapshot

NAD(P)H Oxidase Nox-4 Mediates 7-Ketocholesterol-Induced Endoplasmic Reticulum Stress and Apoptosis in Human Aortic Smooth Muscle Cells

inferredcites →https://doi.org/10.1155/2012/356487openalex-snapshot

Amelioration of Glucolipotoxicity-Induced Endoplasmic Reticulum Stress by a “Chemical Chaperone” in Human THP-1 Monocytes

inferredcites →https://doi.org/10.1161/01.atv.0000184783.04864.9fopenalex-snapshot

Consequences and Therapeutic Implications of Macrophage Apoptosis in Atherosclerosis

inferredcites →https://doi.org/10.1161/01.atv.0000242903.41158.a1openalex-snapshot

The Unfolded Protein Response Is an Important Regulator of Inflammatory Genes in Endothelial Cells

inferredcites →https://doi.org/10.1161/01.cir.0000134487.51510.97openalex-snapshot

Association of Multiple Cellular Stress Pathways With Accelerated Atherosclerosis in Hyperhomocysteinemic Apolipoprotein E-Deficient Mice

inferredcites →https://doi.org/10.1161/01.cir.0000160864.31351.c1openalex-snapshot

Activation of the Unfolded Protein Response Occurs at All Stages of Atherosclerotic Lesion Development in Apolipoprotein E–Deficient Mice

inferredcites →https://doi.org/10.1161/01.cir.90.4.2126openalex-snapshot

Lewis A. Conner Memorial Lecture. Mechanisms leading to myocardial infarction: insights from studies of vascular biology.

inferredcites →https://doi.org/10.1161/atvbaha.107.159327openalex-snapshot

Role of Smooth Muscle Cells in the Initiation and Early Progression of Atherosclerosis

inferredcites →https://doi.org/10.1161/atvbaha.110.206094openalex-snapshot

The Endoplasmic Reticulum Stress-C/EBP Homologous Protein Pathway-Mediated Apoptosis in Macrophages Contributes to the Instability of Atherosclerotic Plaques

inferredcites →https://doi.org/10.1161/atvbaha.111.232827openalex-snapshot

Paraoxonase-2 Modulates Stress Response of Endothelial Cells to Oxidized Phospholipids and a Bacterial Quorum–Sensing Molecule

inferredcites →https://doi.org/10.1161/circgenetics.110.958926openalex-snapshot

Coronary Artery Endothelial Transcriptome In Vivo

inferredcites →https://doi.org/10.1161/circresaha.108.182568openalex-snapshot

Free Cholesterol Accumulation in Macrophage Membranes Activates Toll-Like Receptors and p38 Mitogen-Activated Protein Kinase and Induces Cathepsin K

inferredcites →https://doi.org/10.1161/circresaha.109.203711openalex-snapshot

Chronic Endoplasmic Reticulum Stress Activates Unfolded Protein Response in Arterial Endothelium in Regions of Susceptibility to Atherosclerosis

inferredcites →https://doi.org/10.1161/circresaha.110.224766openalex-snapshot

The Role of Endoplasmic Reticulum Stress in the Progression of Atherosclerosis

inferredcites →https://doi.org/10.1161/circresaha.110.227819openalex-snapshot

Endoplasmic Reticulum Stress As a Therapeutic Target in Cardiovascular Disease

inferredcites →https://doi.org/10.1161/circulationaha.106.676890openalex-snapshot

Subendothelial Lipoprotein Retention as the Initiating Process in Atherosclerosis

inferredcites →https://doi.org/10.1161/circulationaha.106.682054openalex-snapshot

Increased Endoplasmic Reticulum Stress in Atherosclerotic Plaques Associated With Acute Coronary Syndrome

inferredcites →https://doi.org/10.1161/circulationaha.109.900928openalex-snapshot

Reduction of AMP-Activated Protein Kinase α2 Increases Endoplasmic Reticulum Stress and Atherosclerosis In Vivo

inferredcites →https://doi.org/10.1161/circulationaha.110.014050openalex-snapshot

Involvement of Endoplasmic Stress Protein C/EBP Homologous Protein in Arteriosclerosis Acceleration With Augmented Biological Stress Responses

inferredcites →https://doi.org/10.1161/hypertensionaha.109.129924openalex-snapshot

High Dietary Taurine Reduces Apoptosis and Atherosclerosis in the Left Main Coronary Artery

inferredcites →https://doi.org/10.1172/jci37262openalex-snapshot

Macrophage deficiency of p38α MAPK promotes apoptosis and plaque necrosis in advanced atherosclerotic lesions in mice

inferredcites →https://doi.org/10.1172/jci38857openalex-snapshot

Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways

inferredcites →https://doi.org/10.1186/1476-511x-10-229openalex-snapshot

M2 macrophages exhibit higher sensitivity to oxLDL-induced lipotoxicity than other monocyte/macrophage subtypes

inferredcites →https://doi.org/10.1258/ebm.2012.012027openalex-snapshot

Quercetin protects macrophages from oxidized low-density lipoprotein-induced apoptosis by inhibiting the endoplasmic reticulum stress-C/EBP homologous protein pathway

inferredcites →https://doi.org/10.1371/journal.pone.0008852openalex-snapshot

Macrophage Plasticity in Experimental Atherosclerosis

inferredcites →https://doi.org/10.1371/journal.pone.0039586openalex-snapshot

CHOP Potentially Co-Operates with FOXO3a in Neuronal Cells to Regulate PUMA and BIM Expression in Response to ER Stress

inferredcites →https://doi.org/10.2337/db09-1637openalex-snapshot

Activation of AMP-Activated Protein Kinase Inhibits Oxidized LDL-Triggered Endoplasmic Reticulum Stress In Vivo

inferredcites →https://doi.org/10.3410/f.717959717.793462943openalex-snapshot

Faculty Opinions recommendation of IRE1α cleaves select microRNAs during ER stress to derepress translation of proapoptotic Caspase-2.

inferredcites →https://doi.org/10.7326/0003-4819-131-5-199909070-00008openalex-snapshot

Homocyst(e)ine and Cardiovascular Disease: A Critical Review of the Epidemiologic Evidence

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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XBP1: An Adaptor in the Pathogenesis of Atherosclerosis

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Protein-Rich Foods, Sea Foods, and Gut Microbiota Amplify Immune Responses in Chronic Diseases and Cancers – Targeting Perk as a Novel Therapeutic Strategy for Cancer

inferred← citeshttps://doi.org/10.1002/biof.1635openalex-snapshot

High‐throughput profiling reveals perturbation of endoplasmic reticulum stress‐related genes in atherosclerosis induced by high‐cholesterol diet and the protective role of vitamin E

inferred← citeshttps://doi.org/10.1002/jbt.70881openalex-snapshot

Inflammation and Stress: The Resemblance and Variance Between Atherosclerosis and Carcinogenesis—A Pilot Study

inferred← citeshttps://doi.org/10.1002/jcp.28446openalex-snapshot

Scutellarin exerts protective effects against atherosclerosis in rats by regulating the Hippo–FOXO3A and PI3K/AKT signaling pathways

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Dietary compound α‐asarone alleviates ER stress‐mediated apoptosis in 7β‐hydroxycholesterol‐challenged macrophages

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The Unfolded Protein Response in the Immune Cell Development: Putting the Caretaker in the Driving Seat

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Coordinating Organismal Metabolism During Protein Misfolding in the ER Through the Unfolded Protein Response

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ER Protein Quality Control and the Unfolded Protein Response in the Heart

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Atherosclerosis

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NR4A1 deficiency promotes carotid plaque vulnerability by activating integrated stress response via targeting Bcat1

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Possible role of fibroblast growth factor 21 on atherosclerosis via amelioration of endoplasmic reticulum stress-mediated apoptosis in apoE−/− mice

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Overexpression of CREB protein protects from tunicamycin-induced apoptosis in various rat cell types

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Catalpol Inhibits Homocysteine-induced Oxidation and Inflammation via Inhibiting Nox4/NF-κB and GRP78/PERK Pathways in Human Aorta Endothelial Cells

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Endoplasmic reticulum stress-induced hepatic stellate cell apoptosis through calcium-mediated JNK/P38 MAPK and Calpain/Caspase-12 pathways

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Apocynin protects endothelial cells from endoplasmic reticulum stress-induced apoptosis via IRE1α engagement

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IRE1α: from the function to the potential therapeutic target in atherosclerosis

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Pollen Typhae Total Flavone Inhibits Endoplasmic Reticulum Stress-Induced Apoptosis in Human Aortic-Vascular Smooth Muscle Cells through Down-Regulating PERK-eIF2α-ATF4-CHOP Pathway

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Endoplasmic reticulum Metallo protease 1, a triggering factor for unfolded protein response and promising target in colorectal cancer

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AIP1-Mediated Stress Signaling in Atherosclerosis and Arteriosclerosis

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Contribution of Endoplasmic Reticulum Stress to the Clinical Instability of Carotid Plaques in Human Carotid Stenosis

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10.1016/b978-0-323-63600-1.00003-x

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Promoting athero-protective immunity by vaccination with low density lipoprotein-derived antigens

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Unfolded protein response in cardiovascular disease

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Macrophage Apoptosis and Necrotic Core Development in Atherosclerosis: A Rapidly Advancing Field with Clinical Relevance to Imaging and Therapy

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ER Stress and Angiogenesis

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Catestatin improves insulin sensitivity by attenuating endoplasmic reticulum stress: In vivo and in silico validation

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The UPR Activator ATF6 Responds to Proteotoxic and Lipotoxic Stress by Distinct Mechanisms

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Small molecules as modulators of the proteostasis machinery: Implication in cardiovascular diseases

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Astragaloside IV protects against C/EBP homologous protein-mediated apoptosis in oxidized low-density lipoprotein-treated macrophages by promoting autophagy

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Hypoxia triggers endothelial endoplasmic reticulum stress and apoptosis via induction of VLDL receptor

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Basic mechanisms in endoplasmic reticulum stress and relation to cardiovascular diseases

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Trimethylamine N-oxide promotes PERK-mediated endothelial-mesenchymal transition and apoptosis thereby aggravates atherosclerosis

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Intercepting the Lipid-Induced Integrated Stress Response Reduces Atherosclerosis

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PPARβ/δ activation protects against corticosterone-induced ER stress in astrocytes by inhibiting the CpG hypermethylation of microRNA-181a

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Cholesterol enriched diet suppresses ATF6 and PERK and upregulates the IRE1 pathways of the unfolded protein response in spontaneously hypertensive rats: Relevance to pathophysiology of atherosclerosis in the setting of hypertension

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Protein-rich foods, sea foods, and gut microbiota amplify immune responses in chronic diseases and cancers – Targeting PERK as a novel therapeutic strategy for chronic inflammatory diseases, neurodegenerative disorders, and cancer

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Natural products targeting ER stress pathway for the treatment of cardiovascular diseases

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Impact of high cholesterol and endoplasmic reticulum stress on metabolic diseases: An updated mini-review

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8-Chloroadenosine induces apoptosis in human coronary artery endothelial cells through the activation of the unfolded protein response

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Polycyclic aromatic hydrocarbons-enriched diesel exhaust particles induced hypertrophy in rat primary neonatal cardiomyocytes and apoptotic cell death in H9c2 cardiomyocytes

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Pharmacological evidence for a role of the transient receptor potential canonical 3 (TRPC3) channel in endoplasmic reticulum stress-induced apoptosis of human coronary artery endothelial cells

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Activation of the unfolded protein response downregulates cardiac ion channels in human induced pluripotent stem cell-derived cardiomyocytes

inferred← citeshttps://doi.org/10.1038/aps.2017.81openalex-snapshot

Activation of aldehyde dehydrogenase 2 slows down the progression of atherosclerosis via attenuation of ER stress and apoptosis in smooth muscle cells

inferred← citeshttps://doi.org/10.1038/cddis.2017.277openalex-snapshot

Dickkopf1 destabilizes atherosclerotic plaques and promotes plaque formation by inducing apoptosis of endothelial cells through activation of ER stress

inferred← citeshttps://doi.org/10.1038/nrrheum.2016.192openalex-snapshot

Causes and consequences of endoplasmic reticulum stress in rheumatic disease

inferred← citeshttps://doi.org/10.1038/s41580-024-00794-0openalex-snapshot

Homeostasis control in health and disease by the unfolded protein response

inferred← citeshttps://doi.org/10.1073/pnas.1621188114openalex-snapshot

Targeting IRE1 with small molecules counteracts progression of atherosclerosis

inferred← citeshttps://doi.org/10.1074/jbc.m113.524512openalex-snapshot

Endoplasmic Reticulum Stress Promotes Macrophage-derived Foam Cell Formation by Up-regulating Cluster of Differentiation 36 (CD36) Expression

inferred← citeshttps://doi.org/10.1074/jbc.m115.641415openalex-snapshot

Dickkopf Homolog 3 Induces Stem Cell Differentiation into Smooth Muscle Lineage via ATF6 Signalling

inferred← citeshttps://doi.org/10.1080/10799893.2021.1918167openalex-snapshot

Methylglyoxal stimulates endoplasmic reticulum stress in vascular smooth muscle cells

inferred← citeshttps://doi.org/10.1080/15376516.2016.1273429openalex-snapshot

The effects of endoplasmic reticulum stress inducer thapsigargin on the toxicity of ZnO or TiO 2 nanoparticles to human endothelial cells

inferred← citeshttps://doi.org/10.1089/ars.2016.6697openalex-snapshot

Protein Thiol Redox Signaling in Monocytes and Macrophages

inferred← citeshttps://doi.org/10.1093/toxsci/kfu233openalex-snapshot

Molecular Mechanisms of Acrolein Toxicity: Relevance to Human Disease

inferred← citeshttps://doi.org/10.1097/xce.0000000000000068openalex-snapshot

The role of lipoxins in cardiometabolic physiology and disease

inferred← citeshttps://doi.org/10.1101/615997openalex-snapshot

Catestatin improves insulin sensitivity in diet-induced obese mice: in vivo and in silico validation

inferred← citeshttps://doi.org/10.1101/cshperspect.a041400openalex-snapshot

Endoplasmic Reticulum Membrane Homeostasis and the Unfolded Protein Response

inferred← citeshttps://doi.org/10.1111/joim.12562openalex-snapshot

Calcium signalling and ER stress in insulin resistance and atherosclerosis

inferred← citeshttps://doi.org/10.1126/science.aau6499openalex-snapshot

IRE1α–XBP1 signaling in leukocytes controls prostaglandin biosynthesis and pain

inferred← citeshttps://doi.org/10.1126/scitranslmed.aaf9087openalex-snapshot

Prevention of atherosclerosis by bioactive palmitoleate through suppression of organelle stress and inflammasome activation

inferred← citeshttps://doi.org/10.1155/2017/2049217openalex-snapshot

The Role of Endoplasmic Reticulum Stress in Cardiovascular Disease and Exercise

inferred← citeshttps://doi.org/10.1155/2019/3462530openalex-snapshot

ER Stress Activates the NLRP3 Inflammasome: A Novel Mechanism of Atherosclerosis

inferred← citeshttps://doi.org/10.1155/2023/7006749openalex-snapshot

RNA Sequencing Reveals the Differentially Expressed circRNAs between Stable and Unstable Carotid Atherosclerotic Plaques

inferred← citeshttps://doi.org/10.1159/000363008openalex-snapshot

The Impact of Intermittent and Repetitive Cold Stress Exposure on Endoplasmic Reticulum Stress and Instability of Atherosclerotic Plaques

inferred← citeshttps://doi.org/10.1161/hypertensionaha.115.05377openalex-snapshot

Brain Endoplasmic Reticulum Stress Mechanistically Distinguishes the Saline-Intake and Hypertensive Response to Deoxycorticosterone Acetate–Salt

inferred← citeshttps://doi.org/10.1161/jaha.114.000949openalex-snapshot

Endoplasmic Reticulum Stress Effector CCAAT/Enhancer‐binding Protein Homologous Protein (CHOP) Regulates Chronic Kidney Disease–Induced Vascular Calcification

inferred← citeshttps://doi.org/10.1161/jaha.120.020441openalex-snapshot

Aging and Hypercholesterolemia Differentially Affect the Unfolded Protein Response in the Vasculature of ApoE −/− Mice

inferred← citeshttps://doi.org/10.1186/s11658-016-0017-xopenalex-snapshot

Posttranscriptional and transcriptional regulation of endothelial nitric-oxide synthase during hypoxia: the role of microRNAs

inferred← citeshttps://doi.org/10.1186/s12859-023-05545-0openalex-snapshot

Ab initio protein structure prediction: the necessary presence of external force field as it is delivered by Hsp40 chaperone

inferred← citeshttps://doi.org/10.1186/s12906-015-0759-4openalex-snapshot

Ethanol extract of propolis protects macrophages from oxidized low density lipoprotein-induced apoptosis by inhibiting CD36 expression and endoplasmic reticulum stress-C/EBP homologous protein pathway

inferred← citeshttps://doi.org/10.1186/s12944-025-02554-1openalex-snapshot

High-fat stimulation induces atrial structural remodeling via the TPM1/P53/SHISA5 Axis

inferred← citeshttps://doi.org/10.1186/s12967-018-1481-zopenalex-snapshot

Endoplasmic reticulum stress in perivascular adipose tissue promotes destabilization of atherosclerotic plaque by regulating GM-CSF paracrine

inferred← citeshttps://doi.org/10.1186/s12967-025-07402-5openalex-snapshot

Foam cells: tracing the diverse cellular origins and their linked signaling pathways

inferred← citeshttps://doi.org/10.1371/journal.pgen.1011014openalex-snapshot

Genome-wide census of ATF4 binding sites and functional profiling of trait-associated genetic variants overlapping ATF4 binding motifs

inferred← citeshttps://doi.org/10.1371/journal.pone.0075249openalex-snapshot

A Deficiency of Herp, an Endoplasmic Reticulum Stress Protein, Suppresses Atherosclerosis in ApoE Knockout Mice by Attenuating Inflammatory Responses

inferred← citeshttps://doi.org/10.1371/journal.pone.0087787openalex-snapshot

The Modulation of Endoplasmic Reticulum Stress by Chemical Chaperone Upregulates Immune Negative Cytokine IL-35 in Apolipoprotein E-Deficient Mice

inferred← citeshttps://doi.org/10.1371/journal.pone.0110581openalex-snapshot

Purple Perilla Extracts Allay ER Stress in Lipid-Laden Macrophages

inferred← citeshttps://doi.org/10.14336/ad.2022.0824openalex-snapshot

X-box Binding Protein 1: An Adaptor in the Pathogenesis of Atherosclerosis

inferred← citeshttps://doi.org/10.1515/labmed-2017-0138openalex-snapshot

ATF3 and EGR2 gene expression levels in sdLDL-treated macrophages of patients with coronary artery stenosis

inferred← citeshttps://doi.org/10.15252/emmm.202115344openalex-snapshot

Intercepting IRE1 kinase‐FMRP signaling prevents atherosclerosis progression

inferred← citeshttps://doi.org/10.15407/fz64.02.054openalex-snapshot

The study of the initial stages of atherogenesis against high-fat diet

inferred← citeshttps://doi.org/10.1615/critrevimmunol.2014010267openalex-snapshot

Scavenger Receptor-A (CD204): A Two-Edged Sword in Health and Disease

inferred← citeshttps://doi.org/10.17802/2306-1278-2024-13-3s-191-207openalex-snapshot

CURRENT APPROACHES TO THE IDENTIFICATION OF CELLULAR MARKERS OF ENDOTHELIAL DYSFUNCTION

inferred← citeshttps://doi.org/10.18295/2075-0528.1676openalex-snapshot

The Role of Damage-Associated Molecular Patterns (DAMPs) in Human Diseases : Part II: DAMPs as diagnostics, prognostics and therapeutics in clinical medicine

inferred← citeshttps://doi.org/10.18632/oncotarget.10025openalex-snapshot

Methamphetamine-mediated endoplasmic reticulum (ER) stress induces type-1 programmed cell death in astrocytes via ATF6, IRE1α and PERK pathways

inferred← citeshttps://doi.org/10.18632/oncotarget.19849openalex-snapshot

Unfolded protein response signaling impacts macrophage polarity to modulate breast cancer cell clearance and melanoma immune checkpoint therapy responsiveness

inferred← citeshttps://doi.org/10.18632/oncotarget.20431openalex-snapshot

Salubrinal attenuated retinal neovascularization by inhibiting CHOP-HIF1α-VEGF pathways

inferred← citeshttps://doi.org/10.2147/dddt.s512752openalex-snapshot

Oleanolic Acid@SPIONs Alleviates Lipid-Oxidative Stress Injury of Zebrafish Blood Vessels via Regulating the Expression of JNK and MAPK Signaling Pathways in Vascular Endothelial Cells

inferred← citeshttps://doi.org/10.2174/2666796703666220602155421openalex-snapshot

In Silico Modeling of COVID-19 Pandemic Course Differentiation Using the FOD Model

inferred← citeshttps://doi.org/10.22074/cellj.2021.7849openalex-snapshot

Toll-Like Receptor 4: A Macrophage Cell Surface Receptor Is Activated By Trimethylamine-N-Oxide.

inferred← citeshttps://doi.org/10.3345/kjp.2018.06856openalex-snapshot

Changes of Bax, Bcl-2, CCR-2, MCP-1, and TGF-β1 genes in the left ventricle of spontaneously hypertensive rat after losartan treatment

inferred← citeshttps://doi.org/10.3389/fcell.2021.674103openalex-snapshot

Pathological Crosstalk Between Oxidized LDL and ER Stress in Human Diseases: A Comprehensive Review

inferred← citeshttps://doi.org/10.3389/fcvm.2022.988266openalex-snapshot

NF-κB and its crosstalk with endoplasmic reticulum stress in atherosclerosis

inferred← citeshttps://doi.org/10.3389/fgene.2014.00242openalex-snapshot

Endoplasmic reticulum stress in hepatic steatosis and inflammatory bowel diseases

inferred← citeshttps://doi.org/10.3389/fncel.2014.00213openalex-snapshot

A Molecular Web: Endoplasmic Reticulum Stress, Inflammation, and Oxidative Stress

inferred← citeshttps://doi.org/10.3389/fphar.2018.00565openalex-snapshot

Berberine Attenuates Macrophages Infiltration in Intracranial Aneurysms Potentially Through FAK/Grp78/UPR Axis

inferred← citeshttps://doi.org/10.3389/fphys.2024.1347888openalex-snapshot

Developmental endothelial locus 1: the present and future of an endogenous factor in vessels

inferred← citeshttps://doi.org/10.3390/antiox13020232openalex-snapshot

Reactive Carbonyl Species and Protein Lipoxidation in Atherogenesis

inferred← citeshttps://doi.org/10.3390/biom12050622openalex-snapshot

How Similar Are Proteins and Origami?

inferred← citeshttps://doi.org/10.3390/ijms18122721openalex-snapshot

Sphingosine-1-Phosphate Receptor 1, Expressed in Myeloid Cells, Slows Diet-Induced Atherosclerosis and Protects against Macrophage Apoptosis in Ldlr KO Mice

inferred← citeshttps://doi.org/10.3390/ijms21010169openalex-snapshot

Endoplasmic Reticulum Stress Pathway, the Unfolded Protein Response, Modulates Immune Function in the Tumor Microenvironment to Impact Tumor Progression and Therapeutic Response

inferred← citeshttps://doi.org/10.3390/ijms23052504openalex-snapshot

Atherosclerosis in HIV Patients: What Do We Know so Far?

inferred← citeshttps://doi.org/10.3390/ijms23137373openalex-snapshot

GANAB and N-Glycans Substrates Are Relevant in Human Physiology, Polycystic Pathology and Multiple Sclerosis: A Review

inferred← citeshttps://doi.org/10.3390/jcdd13020085openalex-snapshot

Atherosclerosis: A Pathologist’s Perspective

inferred← citeshttps://doi.org/10.3390/jcdd13090435openalex-snapshot

Atherosclerotic Cell Fates: A Single-Cell View of ER Stress

inferred← citeshttps://doi.org/10.3390/jcm8122047openalex-snapshot

Ca2+ Flux: Searching for a Role in Efferocytosis of Apoptotic Cells in Atherosclerosis

inferred← citeshttps://doi.org/10.3390/life13020496openalex-snapshot

The Joint Link of the rs1051730 and rs1902341 Polymorphisms and Cigarette Smoking to Peripheral Artery Disease and Atherosclerotic Lesions of Different Arterial Beds

inferred← citeshttps://doi.org/10.3892/etm.2020.8733openalex-snapshot

Rosuvastatin protects against endothelial cell apoptosis in vitro and alleviates atherosclerosis in ApoE‑/‑ mice by suppressing endoplasmic reticulum stress

inferred← citeshttps://doi.org/10.3892/mmr.2016.4975openalex-snapshot

Atorvastatin attenuates atherosclerotic plaque destabilization by inhibiting endoplasmic reticulum stress in hyperhomocysteinemic mice

inferred← citeshttps://doi.org/10.70675/9406b143zf2d7z40b8zbdc2z2c90188b8eb1openalex-snapshot

Implication des phagocytes mononuclées dans l'évolution de la plaque d'athérosclérose et relation avec l'homéostasie du cholestérol et des lipoprotéines

inferred← citeshttps://doi.org/10.70675/be4af298z55cbz4f73z8215zc8e08ac3608eopenalex-snapshot

Régulations intrinsèques et extrinsèques du métabolisme de la glutamine sur les fonctions effectrices des macrophages et conséquences sur le développement des plaques d'athéromes

inferred← citeshttps://doi.org/10.71548/15747openalex-snapshot

The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death

inferred← citeshttps://doi.org/10.7554/elife.20390openalex-snapshot

Experimental reconstitution of chronic ER stress in the liver reveals feedback suppression of BiP mRNA expression

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