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Omnipresence of inflammasome activities in inflammatory bone diseases

https://doi.org/10.1007/s00281-019-00753-4

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Outgoing claims: the papers this work cites, builds on, or argues with.

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Pathogen blockade of TAK1 triggers caspase-8–dependent cleavage of gasdermin D and cell death

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Porphyromonas gingivalis Infection-Associated Periodontal Bone Resorption Is Dependent on Receptor Activator of NF-κB Ligand

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IL-1 mediates TNF-induced osteoclastogenesis

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TNF regulates transcription of NLRP3 inflammasome components and inflammatory molecules in cryopyrinopathies

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Inflammatory osteolysis: a conspiracy against bone

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Deregulation of the IL-1β axis in chronic recurrent multifocal osteomyelitis

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Mevalonate kinase deficiencies: from mevalonic aciduria to hyperimmunoglobulinemia D syndrome

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Staphylococcus aureus Panton-Valentine leukocidin induces an inflammatory response in human phagocytes via the NLRP3 inflammasome

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Osteoblasts Express NLRP3, a Nucleotide-Binding Domain and Leucine-Rich Repeat Region Containing Receptor Implicated in Bacterially Induced Cell Death

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Prostaglandin E2 Induces Expression of Receptor Activator of Nuclear Factor–κB Ligand/Osteoprotegrin Ligand on Pre-B Cells: Implications for Accelerated Osteoclastogenesis in Estrogen Deficiency

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Fibroblastic Stromal Cells Express Receptor Activator of NF-κB Ligand and Support Osteoclast Differentiation

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Gasdermin D mediates the pathogenesis of neonatal-onset multisystem inflammatory disease in mice

inferredcites →https://doi.org/10.1371/journal.pone.0035979openalex-snapshot

Constitutively Activated NLRP3 Inflammasome Causes Inflammation and Abnormal Skeletal Development in Mice

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Pathogenic Role of Basic Calcium Phosphate Crystals in Destructive Arthropathies

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AIM2 Drives Joint Inflammation in a Self-DNA Triggered Model of Chronic Polyarthritis

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GSDMD membrane pore formation constitutes the mechanism of pyroptotic cell death

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Interleukins in the Control of Osteoclast Differentiation

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Inflammasome-Dependent Release of the Alarmin HMGB1 in Endotoxemia

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Basic Calcium Phosphate Crystals Induce Monocyte/Macrophage IL-1β Secretion through the NLRP3 Inflammasome In Vitro

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Osteoporosis and rheumatic diseases

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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