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Fibronectin contributes to pathological cardiac hypertrophy but not physiological growth

https://doi.org/10.1007/s00395-013-0375-8
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/emmm.201200237openalex-snapshot
Plasma fibronectin deficiency impedes atherosclerosis progression and fibrous cap formation
inferredcites →https://doi.org/10.1002/path.3010openalex-snapshot
Recipient‐derived EDA fibronectin promotes cardiac allograft fibrosis
inferredcites →https://doi.org/10.1006/jmcc.2000.1119openalex-snapshot
Outside-in Signalling of Fibronectin Stimulates Cardiomyocyte Hypertrophy in Cultured Neonatal Rat Ventricular Myocytes
inferredcites →https://doi.org/10.1007/s00395-011-0213-9openalex-snapshot
Mitochondrial biogenesis and PGC-1α deacetylation by chronic treadmill exercise: differential response in cardiac and skeletal muscle
inferredcites →https://doi.org/10.1007/s00395-012-0326-9openalex-snapshot
Interferon regulatory factor 3 is a negative regulator of pathological cardiac hypertrophy
inferredcites →https://doi.org/10.1007/s00395-013-0332-6openalex-snapshot
Neuronal nitric oxide synthase is indispensable for the cardiac adaptive effects of exercise
inferredcites →https://doi.org/10.1016/j.ceb.2008.06.001openalex-snapshot
The role of integrin binding sites in fibronectin matrix assembly in vivo
inferredcites →https://doi.org/10.1016/j.cell.2010.11.036openalex-snapshot
C/EBPβ Controls Exercise-Induced Cardiac Growth and Protects against Pathological Cardiac Remodeling
inferredcites →https://doi.org/10.1016/j.devcel.2008.12.007openalex-snapshot
Cardiac Fibroblasts Regulate Myocardial Proliferation through β1 Integrin Signaling
inferredcites →https://doi.org/10.1016/j.jash.2008.04.012openalex-snapshot
Reduced isoproterenol-induced renin-angiotensin changes and extracellular matrix deposition in hearts of TGR(A1–7)3292 rats
inferredcites →https://doi.org/10.1016/j.yjmcc.2009.07.015openalex-snapshot
The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction
inferredcites →https://doi.org/10.1038/85471openalex-snapshot
Plasma fibronectin supports neuronal survival and reduces brain injury following transient focal cerebral ischemia but is not essential for skin-wound healing and hemostasis.
inferredcites →https://doi.org/10.1038/labinvest.3700345openalex-snapshot
Role of the integrin-linked kinase/PINCH1/alpha-parvin complex in cardiac myocyte hypertrophy
inferredcites →https://doi.org/10.1038/nature12229openalex-snapshot
An siRNA screen for NFAT activation identifies septins as coordinators of store-operated Ca2+ entry
inferredcites →https://doi.org/10.1038/ncb1399openalex-snapshot
Syndecan-4 regulates non-canonical Wnt signalling and is essential for convergent and extension movements in Xenopus embryos
inferredcites →https://doi.org/10.1038/nm1671openalex-snapshot
Pim-1 regulates cardiomyocyte survival downstream of Akt
inferredcites →https://doi.org/10.1038/nrm3495openalex-snapshot
Molecular basis of physiological heart growth: fundamental concepts and new players
inferredcites →https://doi.org/10.1073/pnas.0606663104openalex-snapshot
Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy
inferredcites →https://doi.org/10.1074/jbc.m109.098236openalex-snapshot
The CCAAT/Enhancer Binding Protein β (C/EBPβ) Cooperates with NFAT to Control Expression of the Calcineurin Regulatory Protein RCAN1–4
inferredcites →https://doi.org/10.1074/jbc.m206532200openalex-snapshot
Requirement of Nuclear Factor of Activated T-cells in Calcineurin-mediated Cardiomyocyte Hypertrophy
inferredcites →https://doi.org/10.1074/jbc.m909099199openalex-snapshot
A Role for Focal Adhesion Kinase in Phenylephrine-induced Hypertrophy of Rat Ventricular Cardiomyocytes
inferredcites →https://doi.org/10.1093/cvr/cvn063openalex-snapshot
Imatinib mesylate attenuates fibrosis in coxsackievirus b3-induced chronic myocarditis
inferredcites →https://doi.org/10.1093/eurjhf/hfr017openalex-snapshot
Exercise Training in Heart Failure: From Theory to Practice. A Consensus Document of the Heart Failure Association and the European Association for Cardiovascular Prevention and Rehabilitation
inferredcites →https://doi.org/10.1111/1523-1747.ep12667776openalex-snapshot
Fibronectin Gene Transcription Is Enhanced in Abnormal Wound Healing
inferredcites →https://doi.org/10.1136/jcp.36.12.1377openalex-snapshot
Fibronectin in blood products--an in vitro and in vivo study.
inferredcites →https://doi.org/10.1146/annurev.physiol.65.092101.142243openalex-snapshot
Cardiac Hypertrophy: The Good, the Bad, and the Ugly
inferredcites →https://doi.org/10.1152/physrev.00008.2011openalex-snapshot
Matricellular Proteins in Cardiac Adaptation and Disease
inferredcites →https://doi.org/10.1152/physrev.00012.2007openalex-snapshot
Myocardial Matrix Remodeling and the Matrix Metalloproteinases: Influence on Cardiac Form and Function
inferredcites →https://doi.org/10.1161/01.cir.0000120390.68287.bbopenalex-snapshot
Hypertrophy of the Heart
inferredcites →https://doi.org/10.1161/01.cir.0000141733.55236.9dopenalex-snapshot
Effectiveness of β-Blockade in Experimental Chronic Aortic Regurgitation
inferredcites →https://doi.org/10.1161/01.cir.88.6.2849openalex-snapshot
Identification of functional angiotensin II receptors on rat cardiac fibroblasts.
inferredcites →https://doi.org/10.1161/01.res.0000109415.17511.18openalex-snapshot
Calcineurin/NFAT Coupling Participates in Pathological, but not Physiological, Cardiac Hypertrophy
inferredcites →https://doi.org/10.1161/01.res.74.4.727openalex-snapshot
Angiotensin II induces fibronectin expression associated with cardiac fibrosis in the rat.
inferredcites →https://doi.org/10.1161/circresaha.109.194035openalex-snapshot
CaMKII Negatively Regulates Calcineurin–NFAT Signaling in Cardiac Myocytes
inferredcites →https://doi.org/10.1161/circresaha.109.198416openalex-snapshot
Avoidance of Transient Cardiomyopathy in Cardiomyocyte-Targeted Tamoxifen-Induced MerCreMer Gene Deletion Models
inferredcites →https://doi.org/10.1161/circresaha.109.214353openalex-snapshot
Erythropoietin Activates Mitochondrial Biogenesis and Couples Red Cell Mass to Mitochondrial Mass in the Heart
inferredcites →https://doi.org/10.1161/circresaha.110.224428openalex-snapshot
Lack of Fibronectin-EDA Promotes Survival and Prevents Adverse Remodeling and Heart Function Deterioration After Myocardial Infarction
inferredcites →https://doi.org/10.1161/circresaha.113.301152openalex-snapshot
Fibronectin Is Essential for Reparative Cardiac Progenitor Cell Response After Myocardial Infarction
inferredcites →https://doi.org/10.1172/jci115492openalex-snapshot
Accumulation of fetal fibronectin mRNAs during the development of rat cardiac hypertrophy induced by pressure overload.
inferredcites →https://doi.org/10.1172/jci115685openalex-snapshot
Rapid expression of fibronectin in the rabbit heart after myocardial infarction with and without reperfusion.
inferredcites →https://doi.org/10.1172/jci60329openalex-snapshot
Metabolic stress–induced activation of FoxO1 triggers diabetic cardiomyopathy in mice
inferredcites →https://doi.org/10.1172/jci62839openalex-snapshot
Signaling effectors underlying pathologic growth and remodeling of the heart
inferredcites →https://doi.org/10.1371/journal.pone.0028302openalex-snapshot
Syndecan-4 Is Essential for Development of Concentric Myocardial Hypertrophy via Stretch-Induced Activation of the Calcineurin-NFAT Pathway

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Gene expression analysis to identify mechanisms underlying heart failure susceptibility in mice and humans
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AAV-mediated expression of NFAT decoy oligonucleotides protects from cardiac hypertrophy and heart failure
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Macrophages enhance sodium channel expression in cardiomyocytes
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Quantitative proteomics study reveals differential proteomic signature in dilated, restrictive, and hypertrophic cardiomyopathies
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Pregestational type 2 diabetes mellitus induces cardiac hypertrophy in the murine embryo through cardiac remodeling and fibrosis
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Comparative proteomic analysis of mouse models of pathological and physiological cardiac hypertrophy, with selection of biomarkers of pathological hypertrophy by integrative proteogenomics
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Fibronectin and transforming growth factor beta contribute to erythropoietin resistance and maladaptive cardiac hypertrophy
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Extracellular matrix-derived materials for tissue engineering and regenerative medicine: A journey from isolation to characterization and application
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Myocardial Interstitial Fibrosis in Nonischemic Heart Disease, Part 3/4
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Sex-specific cytotoxicity of ostarine in cardiomyocytes
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Systems-biology dissection of mechanisms and chemical basis of herbal formula in treating chronic myocardial ischemia
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C-MORE: A high-content single-cell morphology recognition methodology for liquid biopsies toward personalized cardiovascular medicine
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Creld1 regulates myocardial development and function
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Recombinant spider silk protein eADF4(C16)-RGD coatings are suitable for cardiac tissue engineering
inferred← citeshttps://doi.org/10.1038/s41598-020-71264-8openalex-snapshot
Myocardial proteomic profile in pulmonary arterial hypertension
inferred← citeshttps://doi.org/10.1038/s41598-023-30514-1openalex-snapshot
A Kinase Interacting Protein 1 (AKIP1) promotes cardiomyocyte elongation and physiological cardiac remodelling
inferred← citeshttps://doi.org/10.1038/s41598-024-65612-1openalex-snapshot
The extent of postpartum cardiac reverse remodeling is reflected in urine proteome
inferred← citeshttps://doi.org/10.1093/lifemeta/load021openalex-snapshot
Extracellular matrix remodelling in obesity and metabolic disorders
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RBFOX2 is required for establishing RNA regulatory networks essential for heart development
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Cardiomyocytes Deficiency of SENP1 Promotes Myocardial Fibrosis via Paracrine Action
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Novel insights into the function and dynamics of extracellular matrix in liver fibrosis
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Fibroblasts under pressure: cardiac fibroblast responses to hypertension and antihypertensive therapies
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From bump to pump: extracellular matrix remodeling, dynamics, and biomechanics in the maternal heart
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Novel Multiplexed Plasma Biomarker Panel Has Diagnostic and Prognostic Potential in Children With Hypertrophic Cardiomyopathy
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Metabolic Dysfunction Consistent With Premature Aging Results From Deletion of Pim Kinases
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Mechanical Regulation of Cardiac Aging in Model Systems
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Animal Models of Exercise From Rodents to Pythons
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Inhibition of Hypertrophy, Per Se, May Not Be a Good Therapeutic Strategy in Ventricular Pressure Overload: Other Approaches Could Be More Beneficial
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Inhibiting Fibronectin Attenuates Fibrosis and Improves Cardiac Function in a Model of Heart Failure
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