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Non-Alzheimer’s contributions to dementia and cognitive resilience in The 90+ Study

https://doi.org/10.1007/s00401-018-1872-5

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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DSM-IV: Diagnostic and Statistical Manual of Mental Disorders

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Nigral pathology and parkinsonian signs in elders without Parkinson disease

inferredcites →https://doi.org/10.1007/s00401-007-0266-xopenalex-snapshot

Argyrophilic thorny astrocyte clusters in association with Alzheimer’s disease pathology in possible primary progressive aphasia

inferredcites →https://doi.org/10.1007/s00401-011-0819-xopenalex-snapshot

A peculiar constellation of tau pathology defines a subset of dementia in the elderly

inferredcites →https://doi.org/10.1007/s00401-011-0910-3openalex-snapshot

National Institute on Aging–Alzheimer’s Association guidelines for the neuropathologic assessment of Alzheimer’s disease: a practical approach

inferredcites →https://doi.org/10.1007/s00401-013-1211-9openalex-snapshot

Staging TDP-43 pathology in Alzheimer’s disease

inferredcites →https://doi.org/10.1007/s00401-014-1308-9openalex-snapshot

A comparison of Aβ amyloid pathology staging systems and correlation with clinical diagnosis

inferredcites →https://doi.org/10.1007/s00401-014-1349-0openalex-snapshot

Primary age-related tauopathy (PART): a common pathology associated with human aging

inferredcites →https://doi.org/10.1007/s00401-014-1373-0openalex-snapshot

Differential induction and spread of tau pathology in young PS19 tau transgenic mice following intracerebral injections of pathological tau from Alzheimer’s disease or corticobasal degeneration brains

inferredcites →https://doi.org/10.1007/s00401-015-1509-xopenalex-snapshot

Aging-related tau astrogliopathy (ARTAG): harmonized evaluation strategy

inferredcites →https://doi.org/10.1007/s00401-017-1717-7openalex-snapshot

Impact of multiple pathologies on the threshold for clinically overt dementia

inferredcites →https://doi.org/10.1016/0014-5793(94)00259-2openalex-snapshot

An extensive network of PHFτ‐rich dystrophic neurites permeates neocortex and nearly all neuritic and diffuse amyloid plaques in Alzheimer disease

inferredcites →https://doi.org/10.1016/j.bbadis.2015.08.019openalex-snapshot

Clinical-pathologic correlations in vascular cognitive impairment and dementia

inferredcites →https://doi.org/10.1016/j.dadm.2017.01.005openalex-snapshot

Pathophysiologic relationship between Alzheimer's disease, cerebrovascular disease, and cardiovascular risk: A review and synthesis

inferredcites →https://doi.org/10.1016/j.jalz.2011.10.007openalex-snapshot

National Institute on Aging–Alzheimer's Association guidelines for the neuropathologic assessment of Alzheimer's disease

inferredcites →https://doi.org/10.1016/j.jalz.2016.04.006openalex-snapshot

Microinfarcts are common and strongly related to dementia in the oldest‐old: The 90+ study

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2015.10.009openalex-snapshot

Brain pathologies in extreme old age

inferredcites →https://doi.org/10.1016/s0140-6736(00)03589-3openalex-snapshot

Pathological correlates of late-onset dementia in a multicentre, community-based population in England and Wales

inferredcites →https://doi.org/10.1016/s0197-4580(03)00113-1openalex-snapshot

High prevalence of thorn-shaped astrocytes in the aged human medial temporal lobe

inferredcites →https://doi.org/10.1016/s0895-4356(00)00314-0openalex-snapshot

Adjusting for multiple testing—when and how?

inferredcites →https://doi.org/10.1038/nm.4443openalex-snapshot

Amyloid-β plaques enhance Alzheimer's brain tau-seeded pathologies by facilitating neuritic plaque tau aggregation

inferredcites →https://doi.org/10.1056/nejmoa0806142openalex-snapshot

Age, Neuropathology, and Dementia

inferredcites →https://doi.org/10.1073/pnas.1301175110openalex-snapshot

Brain homogenates from human tauopathies induce tau inclusions in mouse brain

inferredcites →https://doi.org/10.1093/brain/awq141openalex-snapshot

Age, Alzheimer's disease and dementia in the Baltimore Longitudinal Study of Ageing

inferredcites →https://doi.org/10.1093/brain/awr308openalex-snapshot

Neocortical and hippocampal amyloid-β and tau measures associate with dementia in the oldest-old

inferredcites →https://doi.org/10.1093/brain/awu190openalex-snapshot

Perforant path synaptic loss correlates with cognitive impairment and Alzheimer's disease in the oldest-old

inferredcites →https://doi.org/10.1093/jnen/nlw002openalex-snapshot

Altered Proteins in the Aging Brain

inferredcites →https://doi.org/10.1093/jnen/nlw033openalex-snapshot

“New Old Pathologies”: AD, PART, and Cerebral Age-Related TDP-43 With Sclerosis (CARTS)

inferredcites →https://doi.org/10.1093/jnen/nlx007openalex-snapshot

Evaluating the Patterns of Aging-Related Tau Astrogliopathy Unravels Novel Insights Into Brain Aging and Neurodegenerative Diseases

inferredcites →https://doi.org/10.1093/jnen/nlx041openalex-snapshot

Multisite Assessment of Aging-Related Tau Astrogliopathy (ARTAG)

inferredcites →https://doi.org/10.1097/nen.0b013e31815c5efbopenalex-snapshot

Clinicopathologic Correlations in a Large Alzheimer Disease Center Autopsy Cohort

inferredcites →https://doi.org/10.1097/nen.0b013e318232a379openalex-snapshot

Stages of the Pathologic Process in Alzheimer Disease: Age Categories From 1 to 100 Years

inferredcites →https://doi.org/10.1097/nen.0b013e31825018f7openalex-snapshot

Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature

inferredcites →https://doi.org/10.1111/bpa.12536openalex-snapshot

Protein astrogliopathies in human neurodegenerative diseases and aging

inferredcites →https://doi.org/10.1111/bpa.12556openalex-snapshot

Hippocampal sclerosis, hippocampal neuron loss patterns and TDP‐43 in the aged population

inferredcites →https://doi.org/10.1126/sageke.2006.6.re1openalex-snapshot

The Development of Amyloid β Protein Deposits in the Aged Brain

inferredcites →https://doi.org/10.1159/000337230openalex-snapshot

Amyloid Oligomers Exacerbate Tau Pathology in a Mouse Model of Tauopathy

inferredcites →https://doi.org/10.1159/000341581openalex-snapshot

Mesial Temporal Astrocyte Tau Pathology in the MRC-CFAS Ageing Brain Cohort

inferredcites →https://doi.org/10.1186/1745-6215-11-85openalex-snapshot

Assessing and reporting heterogeneity in treatment effects in clinical trials: a proposal

inferredcites →https://doi.org/10.1186/s40478-016-0275-xopenalex-snapshot

Epidemiological pathology of Tau in the ageing brain: application of staging for neuropil threads (BrainNet Europe protocol) to the MRC cognitive function and ageing brain study

inferredcites →https://doi.org/10.1212/wnl.0000000000001831openalex-snapshot

Multiple pathologies are common and related to dementia in the oldest-old

inferredcites →https://doi.org/10.1212/wnl.0000000000004058openalex-snapshot

Diagnosis and management of dementia with Lewy bodies

inferredcites →https://doi.org/10.1212/wnl.0b013e31824e8e7fopenalex-snapshot

Staging and natural history of cerebrovascular pathology in dementia

inferredcites →https://doi.org/10.1523/jneurosci.1230-17.2017openalex-snapshot

Pathological Tau Strains from Human Brains Recapitulate the Diversity of Tauopathies in Nontransgenic Mouse Brain

inferredcites →https://doi.org/10.2174/156720512801322537openalex-snapshot

A Population-Based Clinicopathological Study in the Oldest-Old: The 90+ Study

inferredcites →https://doi.org/10.3233/jad-131880openalex-snapshot

Hippocampal Sclerosis of Aging is a Key Alzheimer's Disease Mimic: Clinical-Pathologic Correlations and Comparisons with both Alzheimer's Disease and Non-Tauopathic Frontotemporal Lobar Degeneration

inferredcites →https://doi.org/10.3233/jad-161208openalex-snapshot

Modifiable Risk Factors for Prevention of Dementia in Midlife, Late Life and the Oldest-Old: Validation of the LIBRA Index

inferredcites →https://doi.org/10.5858/arpa.2016-0469-saopenalex-snapshot

Hippocampal Sclerosis in Older Patients: Practical Examples and Guidance With a Focus on Cerebral Age-Related TDP-43 With Sclerosis

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4068682openalex-extracted

inferred← citeshttps://doi.org/10.1002/acn3.51273openalex-snapshot

Amyloid‐β, cortical thickness, and subsequent cognitive decline in cognitively normal oldest‐old

inferred← citeshttps://doi.org/10.1002/alz.12079openalex-snapshot

Contribution of mixed pathology to medial temporal lobe atrophy in Alzheimer's disease

inferred← citeshttps://doi.org/10.1002/alz.12366openalex-snapshot

Prevalence of cerebral amyloid angiopathy: A systematic review and meta‐analysis

inferred← citeshttps://doi.org/10.1002/alz.13844openalex-snapshot

Sex and gender differences in cognitive resilience to aging and Alzheimer's disease

inferred← citeshttps://doi.org/10.1002/alz.13858openalex-snapshot

A cross‐sectional study of α‐synuclein seed amplification assay in Alzheimer's disease neuroimaging initiative: Prevalence and associations with Alzheimer's disease biomarkers and cognitive function

inferred← citeshttps://doi.org/10.1002/alz.14037openalex-snapshot

Prevalence and clinicopathological features of primary age‐related tauopathy (PART): A large forensic autopsy study

inferred← citeshttps://doi.org/10.1002/alz.14138openalex-snapshot

The Bayesian approach for real‐world implementation of plasma p‐tau217 in tertiary care memory clinics in Thailand

inferred← citeshttps://doi.org/10.1002/alz.70037openalex-snapshot

Driving research on successful aging and neuroprotection in Latin America: Insights from the inaugural symposium on brain resilience and healthy longevity

inferred← citeshttps://doi.org/10.1002/alz.70151openalex-snapshot

Resistance and resilience to Alzheimer's disease in Down syndrome

inferred← citeshttps://doi.org/10.1002/alz.70575openalex-snapshot

Cognitive and neuropsychological trajectories in patients with mixed neurodegenerative pathologies

inferred← citeshttps://doi.org/10.1002/alz.70613openalex-snapshot

Hippocampal tau distribution in primary age‐related tauopathy: The Vantaa 85+ Study

inferred← citeshttps://doi.org/10.1002/alz.70683openalex-snapshot

Compression of cognitive decline and cognitive resilience in extreme longevity

inferred← citeshttps://doi.org/10.1002/alz.70988openalex-snapshot

Reframing TDP‐43 in Alzheimer's disease: From co‐pathology to neurovascular culprit

inferred← citeshttps://doi.org/10.1002/pmic.202400298openalex-snapshot

Proteomic Insight Into Alzheimer's Disease Pathogenesis Pathways

inferred← citeshttps://doi.org/10.1007/s00018-019-03059-8openalex-snapshot

Phosphorylated and aggregated TDP-43 with seeding properties are induced upon mutant Huntingtin (mHtt) polyglutamine expression in human cellular models

inferred← citeshttps://doi.org/10.1007/s00401-022-02432-5openalex-snapshot

Neuropathological associations of limbic-predominant age-related TDP-43 encephalopathy neuropathological change (LATE-NC) differ between the oldest-old and younger-old

inferred← citeshttps://doi.org/10.1007/s00401-022-02444-1openalex-snapshot

Frequency of LATE neuropathologic change across the spectrum of Alzheimer’s disease neuropathology: combined data from 13 community-based or population-based autopsy cohorts

inferred← citeshttps://doi.org/10.1007/s00401-022-02524-2openalex-snapshot

LATE-NC staging in routine neuropathologic diagnosis: an update

inferred← citeshttps://doi.org/10.1007/s00401-023-02606-9openalex-snapshot

Characterization of hippocampal sclerosis of aging and its association with other neuropathologic changes and cognitive deficits in the oldest-old

inferred← citeshttps://doi.org/10.1007/s00401-024-02716-yopenalex-snapshot

Disentangling and quantifying the relative cognitive impact of concurrent mixed neurodegenerative pathologies

inferred← citeshttps://doi.org/10.1007/s00401-024-02789-9openalex-snapshot

Association of quantitative histopathology measurements with antemortem medial temporal lobe cortical thickness in the Alzheimer’s disease continuum

inferred← citeshttps://doi.org/10.1007/s00401-025-02929-9openalex-snapshot

Differences and overlaps in TDP-43 pathology of ‘pure’ LATE-NC compared to LATE-NC coexisting with Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s00401-025-02943-xopenalex-snapshot

Primary age-related tauopathy

inferred← citeshttps://doi.org/10.1007/s00401-025-02971-7openalex-snapshot

Associations of pathologic Parkinson’s disease (PD) and co-pathologies with cognitive decline and progression of parkinsonian signs in decedents with subclinical disease

inferred← citeshttps://doi.org/10.1007/s00401-026-03010-9openalex-snapshot

Contribution of health history and neuropathologic changes to the likelihood of dementia in those with intermediate/high Alzheimer’s pathology: findings from The 90 + Study

inferred← citeshttps://doi.org/10.1007/s00401-026-03027-0openalex-snapshot

Molecular signatures and biomarker development for limbic-predominant age-related TDP-43 encephalopathy (LATE)

inferred← citeshttps://doi.org/10.1007/s00401-026-03031-4openalex-snapshot

Neocortical tau burden determines the degree of cognitive impairment in individuals with Braak stage V neurofibrillary degeneration

inferred← citeshttps://doi.org/10.1007/s00415-020-09718-2openalex-snapshot

TDP-43 is associated with a reduced likelihood of rendering a clinical diagnosis of dementia with Lewy bodies in autopsy-confirmed cases of transitional/diffuse Lewy body disease

inferred← citeshttps://doi.org/10.1007/s00428-024-03781-0openalex-snapshot

Flanagan’s condensed protocol for neurodegenerative diseases. Implementation in a clinical autopsy setting with partial supervision of a neuropathologist

inferred← citeshttps://doi.org/10.1007/s10354-021-00848-4openalex-snapshot

Neuropathologie der Demenzen

inferred← citeshttps://doi.org/10.1007/s11357-021-00407-0openalex-snapshot

Tau and TDP-43 synergy: a novel therapeutic target for sporadic late-onset Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s13311-022-01185-zopenalex-snapshot

Heterogeneity in Alzheimer's Disease Diagnosis and Progression Rates: Implications for Therapeutic Trials

inferred← citeshttps://doi.org/10.1016/b978-0-12-804766-8.00013-3openalex-snapshot

Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-818000-6.00010-xopenalex-snapshot

The aging brain and brain banking

inferred← citeshttps://doi.org/10.1016/bs.pmbts.2019.10.002openalex-snapshot

Neuroimmune interactions in Alzheimer's disease—New frontier with old challenges?

inferred← citeshttps://doi.org/10.1016/j.arr.2019.101002openalex-snapshot

A third of community-dwelling elderly with intermediate and high level of Alzheimer’s neuropathologic changes are not demented: A meta-analysis

inferred← citeshttps://doi.org/10.1016/j.arr.2023.101916openalex-snapshot

The unique neuropathological vulnerability of the human brain to aging

inferred← citeshttps://doi.org/10.1016/j.cger.2022.07.005openalex-snapshot

Neuropathology of the Common Forms of Dementia

inferred← citeshttps://doi.org/10.1016/j.cortex.2019.10.009openalex-snapshot

Amount and delay insensitivity during intertemporal choice in three neurodegenerative diseases reflects dorsomedial prefrontal atrophy

inferred← citeshttps://doi.org/10.1016/j.cpha.2021.02.007openalex-snapshot

Seasons and Seasonings of the Older Years

inferred← citeshttps://doi.org/10.1016/j.jpha.2023.05.008openalex-snapshot

The role of signaling crosstalk of microglia in hippocampus on progression of ageing and Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/j.nbd.2020.105138openalex-snapshot

The relation between tau pathology and granulovacuolar degeneration of neurons

inferred← citeshttps://doi.org/10.1016/j.nbd.2024.106412openalex-snapshot

Pathologic correlates of aging-related tau astrogliopathy: ARTAG is associated with LATE-NC and cerebrovascular pathologies, but not with ADNC

inferred← citeshttps://doi.org/10.1016/j.nbd.2026.107538openalex-snapshot

Ageing-related tau astrogliopathy in a population-based study of the oldest old (Vantaa 85+)

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2020.04.001openalex-snapshot

Association between transactive response DNA-binding protein of 43 kDa type and cognitive resilience to Alzheimer’s disease: a case-control study

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2020.10.002openalex-snapshot

Cognitive and neuroimaging profiles of older adults with dual decline in memory and gait speed

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2022.03.009openalex-snapshot

Cognitive resilience to three dementia-related neuropathologies in an oldest-old man: A case report from The 90+ Study

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2022.07.010openalex-snapshot

Association of glial tau pathology and LATE-NC in the ageing brain

inferred← citeshttps://doi.org/10.1016/j.neuroimage.2020.117269openalex-snapshot

The effects of voluntary running on cerebrovascular morphology and spatial short-term memory in a mouse model of amyloidosis

inferred← citeshttps://doi.org/10.1016/j.neurol.2020.01.355openalex-snapshot

Cognitive deficit, and neuropathological correlates, in the oldest-old

inferred← citeshttps://doi.org/10.1016/j.neuron.2020.09.042openalex-snapshot

High-Contrast In Vivo Imaging of Tau Pathologies in Alzheimer’s and Non-Alzheimer’s Disease Tauopathies

inferred← citeshttps://doi.org/10.1016/j.parkreldis.2021.10.010openalex-snapshot

Low soluble amyloid-β 42 is associated with smaller brain volume in Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.tjpad.2024.100009openalex-snapshot

Usefulness of Cerebrospinal Fluid Alzheimer's disease biomarkers in older patients: Evidence from a national multicenter prospective study

inferred← citeshttps://doi.org/10.1016/j.tjpad.2024.100011openalex-snapshot

Informing etiological heterogeneity of mild cognitive impairment and risk for progression to dementia with plasma p-tau217

inferred← citeshttps://doi.org/10.1017/9781108888202openalex-snapshot

Brain Fables

inferred← citeshttps://doi.org/10.1017/9781108888202.001openalex-snapshot

Preface 1 – The Question

inferred← citeshttps://doi.org/10.1017/9781108888202.002openalex-snapshot

Preface 2 – Enter Patient

inferred← citeshttps://doi.org/10.1017/9781108888202.003openalex-snapshot

The Shaky Six and the “Second Reality”

inferred← citeshttps://doi.org/10.1017/9781108888202.004openalex-snapshot

Pieces of a Puzzle?

inferred← citeshttps://doi.org/10.1017/9781108888202.005openalex-snapshot

Disease “Redefinition”: A Tough Pill to Swallow

inferred← citeshttps://doi.org/10.1017/9781108888202.006openalex-snapshot

Disease Subtypes: The Promise and the Fallacy

inferred← citeshttps://doi.org/10.1017/9781108888202.007openalex-snapshot

Protein Paradox

inferred← citeshttps://doi.org/10.1017/9781108888202.008openalex-snapshot

The Fault in Our Models

inferred← citeshttps://doi.org/10.1017/9781108888202.009openalex-snapshot

Biomarkers: The Promise and the Fallacy

inferred← citeshttps://doi.org/10.1017/9781108888202.010openalex-snapshot

Lessons from Oncology

inferred← citeshttps://doi.org/10.1017/9781108888202.011openalex-snapshot

Symptomatic vs. Disease-Modifying Therapies

inferred← citeshttps://doi.org/10.1017/9781108888202.012openalex-snapshot

The Hypothesis That Refuses to Die

inferred← citeshttps://doi.org/10.1017/9781108888202.013openalex-snapshot

Our Living Dissonance

inferred← citeshttps://doi.org/10.1017/9781108888202.014openalex-snapshot

The Scientific and Lay Narratives

inferred← citeshttps://doi.org/10.1017/9781108888202.015openalex-snapshot

Challenges Viewed from Afar

inferred← citeshttps://doi.org/10.1017/9781108888202.016openalex-snapshot

The Moonshot: Population-Based Studies of Aging

inferred← citeshttps://doi.org/10.1017/9781108888202.017openalex-snapshot

Predictions for the 2020s and Beyond

inferred← citeshttps://doi.org/10.1017/9781108888202.018openalex-snapshot

Epilogue

inferred← citeshttps://doi.org/10.1017/9781108888202.019openalex-snapshot

Note Added at Press Time – Reviving LOF

inferred← citeshttps://doi.org/10.1017/9781108888202.020openalex-snapshot

References

inferred← citeshttps://doi.org/10.1017/9781108888202.021openalex-snapshot

Index

inferred← citeshttps://doi.org/10.1017/9781108985055.015openalex-snapshot

Cognitive Disorders of the Very Old: From Clinic to Pathology

inferred← citeshttps://doi.org/10.1017/cjn.2022.34openalex-snapshot

Current Concepts of Mixed Pathologies in Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1021/acsnano.3c09311openalex-snapshot

High-Performance Plasma Biomarker Panel for Alzheimer’s Disease Screening Using a Femtomolar-Level Label-Free Biosensing System

inferred← citeshttps://doi.org/10.1021/jacs.5c02811openalex-snapshot

Strain-Distinct α-Synuclein and Tau Cross-Seeding Uncovered by Correlative Approach with Optical Photothermal Infrared Sub-Micron Imaging

inferred← citeshttps://doi.org/10.1038/s41467-019-13787-xopenalex-snapshot

Transmission of tauopathy strains is independent of their isoform composition

inferred← citeshttps://doi.org/10.1038/s41467-025-56469-7openalex-snapshot

Early Alzheimer’s Disease with frequent neuritic plaques harbors neocortical tau seeds distinct from primary age-related tauopathy

inferred← citeshttps://doi.org/10.1038/s41582-022-00642-9openalex-snapshot

Lesions without symptoms: understanding resilience to Alzheimer disease neuropathological changes

inferred← citeshttps://doi.org/10.1038/s41582-023-00846-7openalex-snapshot

Limbic-predominant age-related TDP43 encephalopathy (LATE) neuropathological change in neurodegenerative diseases

inferred← citeshttps://doi.org/10.1055/b-0034-22397openalex-snapshot

1.4 Neuropathologie der Demenzen

inferred← citeshttps://doi.org/10.1055/s-0040-1713628openalex-snapshot

Chronic Traumatic Encephalopathy and Neuropathological Comorbidities

inferred← citeshttps://doi.org/10.1080/13854046.2025.2454047openalex-snapshot

Chronic traumatic encephalopathy: State-of-the-science update and narrative review

inferred← citeshttps://doi.org/10.1080/13854046.2025.2482083openalex-snapshot

Integrating home-based video teleneuropsychology into neurology clinical practice: Utility in patients with suspected typical or atypical Alzheimer’s disease presentations

inferred← citeshttps://doi.org/10.1093/brain/awaa219openalex-snapshot

Limbic-predominant age-related TDP-43 encephalopathy differs from frontotemporal lobar degeneration

inferred← citeshttps://doi.org/10.1093/brain/awab092openalex-snapshot

To what degree is late life cognitive decline driven by age-related neuropathologies?

inferred← citeshttps://doi.org/10.1093/brain/awad059openalex-snapshot

Pathological combinations in neurodegenerative disease are heterogeneous and disease-associated

inferred← citeshttps://doi.org/10.1093/brain/awae212openalex-snapshot

Limbic-predominant age-related TDP-43 encephalopathy in the oldest old: a population-based study

inferred← citeshttps://doi.org/10.1093/brain/awae250openalex-snapshot

Grey matter ageing-related tau astrogliopathy: associations with brain pathologies and cognitive decline

inferred← citeshttps://doi.org/10.1093/brain/awaf073openalex-snapshot

Traumatic brain injury or head impacts from contact sports are associated with tau astrogliopathy

inferred← citeshttps://doi.org/10.1093/brain/awz099openalex-snapshot

Limbic-predominant age-related TDP-43 encephalopathy (LATE): consensus working group report

inferred← citeshttps://doi.org/10.1093/braincomms/fcaa210openalex-snapshot

Astroglial tau pathology alone preferentially concentrates at sulcal depths in chronic traumatic encephalopathy neuropathologic change

inferred← citeshttps://doi.org/10.1093/jnen/nlab050openalex-snapshot

LATE Neuropathologic Changes with Little or No Alzheimer Disease is Common and is Associated with Cognitive Impairment but Not Frontotemporal Dementia

inferred← citeshttps://doi.org/10.1093/jnen/nlac115openalex-snapshot

Cognitive resistance to and resilience against multiple comorbid neurodegenerative pathologies and the impact of APOE status

inferred← citeshttps://doi.org/10.1093/jnen/nlae032openalex-snapshot

Limbic-predominant age-related TDP-43 encephalopathy (LATE-NC): Co-pathologies and genetic risk factors provide clues about pathogenesis

inferred← citeshttps://doi.org/10.1093/jnen/nlag032openalex-snapshot

Cortical subpial aging-related tau astrogliopathy in older adult men who participated in youth amateur American-style football

inferred← citeshttps://doi.org/10.1093/jnen/nlz017openalex-snapshot

Chronic Traumatic Encephalopathy (CTE) Is Absent From a European Community-Based Aging Cohort While Cortical Aging-Related Tau Astrogliopathy (ARTAG) Is Highly Prevalent

inferred← citeshttps://doi.org/10.1093/jnen/nlz094openalex-snapshot

Astrocytic Tau Deposition Is Frequent in Typical and Atypical Alzheimer Disease Presentations

inferred← citeshttps://doi.org/10.1093/jnen/nlz126openalex-snapshot

Limbic Predominant Age-Related TDP-43 Encephalopathy (LATE): Clinical and Neuropathological Associations

inferred← citeshttps://doi.org/10.1097/wco.0000000000000664openalex-snapshot

Are comorbidities compatible with a molecular pathological classification of neurodegenerative diseases?

inferred← citeshttps://doi.org/10.1097/wco.0000000000000910openalex-snapshot

What have we learned from cognition in the oldest-old

inferred← citeshttps://doi.org/10.1101/2024.01.28.577642openalex-snapshot

Astrocytic tau inclusions lead to microglial abnormalities, but leave neuronal networks intact

inferred← citeshttps://doi.org/10.1111/bpa.12719openalex-snapshot

Neuropathological lesions in the very old: results from a large Brazilian autopsy study

inferred← citeshttps://doi.org/10.1111/bpa.13058openalex-snapshot

Enhanced expression of autophagy‐related p62 without increased deposits of neurodegeneration‐associated proteins in glioblastoma and surrounding tissue – An autopsy‐based study

inferred← citeshttps://doi.org/10.1111/nan.12788openalex-snapshot

Primary age‐related tauopathy in a Finnish population‐based study of the oldest old (Vantaa 85+)

inferred← citeshttps://doi.org/10.1111/nan.12923openalex-snapshot

Insights into the pathological basis of dementia from population‐based neuropathology studies

inferred← citeshttps://doi.org/10.1111/nan.70081openalex-snapshot

VCING Scoring and White Matter Arteriolosclerosis Show Relationships to Dementia Status and White Matter Damage in an Unselected Ageing Brain Cohort

inferred← citeshttps://doi.org/10.1111/neup.12708openalex-snapshot

Multiple system aging‐related tau astrogliopathy with complex proteinopathy in an oligosymptomatic octogenarian

inferred← citeshttps://doi.org/10.1159/000547067openalex-snapshot

Falls in the Oldest Old: Role of Gender, Living Situation, and Assistive Devices

inferred← citeshttps://doi.org/10.1177/13872877251365560openalex-snapshot

Autoimmune antibodies and systemic inflammatory markers are prevalent and associated with cognition in individuals aged 90+

inferred← citeshttps://doi.org/10.1186/s12877-021-02327-2openalex-snapshot

Pre-stroke cognitive impairment is associated with vascular imaging pathology: a prospective observational study

inferred← citeshttps://doi.org/10.1186/s13024-024-00719-7openalex-snapshot

The concept of resilience to Alzheimer’s Disease: current definitions and cellular and molecular mechanisms

inferred← citeshttps://doi.org/10.1186/s13064-026-00243-yopenalex-snapshot

Molecular mechanisms of aging in the pathogenesis of Alzheimer’s disease

inferred← citeshttps://doi.org/10.1186/s13195-023-01329-zopenalex-snapshot

Quantitative estimate of cognitive resilience and its medical and genetic associations

inferred← citeshttps://doi.org/10.1186/s13195-024-01510-yopenalex-snapshot

Identifying longitudinal cognitive resilience from cross-sectional amyloid, tau, and neurodegeneration

inferred← citeshttps://doi.org/10.1186/s13195-025-01904-6openalex-snapshot

Longitudinal assessment of cognitive decline and resilience in high-level Alzheimer disease neuropathologic change

inferred← citeshttps://doi.org/10.1186/s40478-019-0743-1openalex-snapshot

Resistance and resilience to Alzheimer’s disease pathology are associated with reduced cortical pTau and absence of limbic-predominant age-related TDP-43 encephalopathy in a community-based cohort

inferred← citeshttps://doi.org/10.1186/s40478-021-01152-3openalex-snapshot

Space-occupying brain lesions, trauma-related tau astrogliopathy, and ARTAG: a report of two cases and a literature review

inferred← citeshttps://doi.org/10.1186/s40478-021-01171-0openalex-snapshot

Mixed pathologies in pancreatic β cells from subjects with neurodegenerative diseases and their interaction with prion protein

inferred← citeshttps://doi.org/10.1186/s40478-021-01233-3openalex-snapshot

Predictors of cognitive impairment in primary age-related tauopathy: an autopsy study

inferred← citeshttps://doi.org/10.1186/s40478-022-01324-9openalex-snapshot

Differential protein expression in the hippocampi of resilient individuals identified by digital spatial profiling

inferred← citeshttps://doi.org/10.1186/s40478-022-01370-3openalex-snapshot

Changes in glial cell phenotypes precede overt neurofibrillary tangle formation, correlate with markers of cortical cell damage, and predict cognitive status of individuals at Braak III-IV stages

inferred← citeshttps://doi.org/10.1186/s40478-024-01760-9openalex-snapshot

Gene-expression profiling of individuals resilient to Alzheimer's disease reveals higher expression of genes related to metallothionein and mitochondrial processes and no changes in the unfolded protein response

inferred← citeshttps://doi.org/10.1212/con.0000000000001137openalex-snapshot

Neuropathology of Dementia Disorders

inferred← citeshttps://doi.org/10.1212/wn9.0000000000000165openalex-snapshot

Association Between Aging-Related Tau Astrogliopathy and Financial and Health Literacy in Community-Dwelling Older Adults

inferred← citeshttps://doi.org/10.1212/wnl.0000000000008937openalex-snapshot

Language and spatial dysfunction in Alzheimer disease with white matter thorn-shaped astrocytes

inferred← citeshttps://doi.org/10.1212/wnl.0000000000200832openalex-snapshot

Association of Cognition and Dementia With Neuropathologic Changes of Alzheimer Disease and Other Conditions in the Oldest Old

inferred← citeshttps://doi.org/10.1212/wnl.0000000000207159openalex-snapshot

Cognitive and Clinical Characteristics of Patients With Limbic-Predominant Age-Related TDP-43 Encephalopathy

inferred← citeshttps://doi.org/10.1371/journal.pone.0359109openalex-snapshot

Age at dementia diagnosis, subtype associations, and neuroimaging differentiation in Bangladesh: The CARED study

inferred← citeshttps://doi.org/10.1373/jalm.2019.029587openalex-snapshot

Detection of Alzheimer Disease Pathology in Patients Using Biochemical Biomarkers: Prospects and Challenges for Use in Clinical Practice

inferred← citeshttps://doi.org/10.21926/obm.geriatr.1903063openalex-snapshot

Curing Alzheimer’s Disease: Myriad Causes and Myriad Cures Await

inferred← citeshttps://doi.org/10.31234/osf.io/4t5qaopenalex-snapshot

From genes to network models of Alzheimer's disease: Biological framework for multi-scale brain simulation with The Virtual Brain

inferred← citeshttps://doi.org/10.3233/jad-201166openalex-snapshot

TAR DNA-Binding Protein 43 Is Associated with Rate of Memory, Functional and Global Cognitive Decline in the Decade Prior to Death

inferred← citeshttps://doi.org/10.3233/jad-201213openalex-snapshot

Alzheimer’s Disease Neuropathological Comorbidities are Common in the Younger-Old

inferred← citeshttps://doi.org/10.3233/jad-215444openalex-snapshot

Assessment of Cognitive Symptoms in Brain Bank-Registered Control Subjects: Feasibility and Utility of a Telephone-Based Screening

inferred← citeshttps://doi.org/10.3233/jad-220655openalex-snapshot

Mixed Pathologies in a Subject with a Novel PSEN1 G206R Mutation

inferred← citeshttps://doi.org/10.3233/jad-221062openalex-snapshot

Superior Global Cognition in Oldest-Old Is Associated with Resistance to Neurodegenerative Pathologies: Results from The 90+ Study

inferred← citeshttps://doi.org/10.3233/jad-230022openalex-snapshot

Cognitive and Neuropsychological Profiles in Alzheimer’s Disease and Primary Age-Related Tauopathy and the Influence of Comorbid Neuropathologies

inferred← citeshttps://doi.org/10.33552/ijnc.2019.02.000526openalex-snapshot

Aging as Planned Obsolescence: The Case of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.3389/fimmu.2026.1933760openalex-snapshot

An analysis of cerebral amyloid angiopathy based on samples from human brain bank

inferred← citeshttps://doi.org/10.3389/fnagi.2020.00096openalex-snapshot

Astroglia and Tau: New Perspectives

inferred← citeshttps://doi.org/10.3389/fncom.2019.00054openalex-snapshot

Linking Molecular Pathways and Large-Scale Computational Modeling to Assess Candidate Disease Mechanisms and Pharmacodynamics in Alzheimer's Disease

inferred← citeshttps://doi.org/10.3389/fninf.2021.630172openalex-snapshot

Bridging Scales in Alzheimer's Disease: Biological Framework for Brain Simulation With The Virtual Brain

inferred← citeshttps://doi.org/10.3390/biom15101367openalex-snapshot

Association of Plasma Placental Growth Factor with White Matter Hyperintensities in Alzheimer’s Disease

inferred← citeshttps://doi.org/10.3390/brainsci11020278openalex-snapshot

Multiple Subtypes of Alzheimer’s Disease Base on Brain Atrophy Pattern

inferred← citeshttps://doi.org/10.3390/brainsci11070927openalex-snapshot

Aging-Related Tau Astrogliopathy in Aging and Neurodegeneration

inferred← citeshttps://doi.org/10.3390/diagnostics12102458openalex-snapshot

The Impact of Cerebral Amyloid Angiopathy on Functional Outcome of Patients Affected by Spontaneous Intracerebral Hemorrhage Discharged from Intensive Inpatient Rehabilitation: A Cohort Study

inferred← citeshttps://doi.org/10.3390/ijms27156964openalex-snapshot

Senescence Markers and Associated Transcriptomic Changes Are Expressed at Early Stages of Alzheimer’s Neuropathology but Are Not Independently Related to Dementia

inferred← citeshttps://doi.org/10.3390/jcm13226755openalex-snapshot

Neuropathological Heterogeneity of Dementia Due to Combined Pathology in Aged Patients: Clinicopathological Findings in the Vallecas Alzheimer’s Reina Sofía Cohort

inferred← citeshttps://doi.org/10.3390/neuroglia1020023openalex-snapshot

Understanding the Relevance of Aging-Related Tau Astrogliopathy (ARTAG)

inferred← citeshttps://doi.org/10.4132/jptm.2019.03.14openalex-snapshot

Primary Age-Related Tauopathy: An Elderly Brain Pathology Frequently Encountered during Autopsy

inferred← citeshttps://doi.org/10.6084/m9.figshare.14391481openalex-snapshot

Additional file 1 of Mixed pathologies in pancreatic β cells from subjects with neurodegenerative diseases and their interaction with prion protein

inferred← citeshttps://doi.org/10.6084/m9.figshare.14782749openalex-snapshot

Additional file 1 of Pre-stroke cognitive impairment is associated with vascular imaging pathology: a prospective observational study

inferred← citeshttps://doi.org/10.6084/m9.figshare.14782752openalex-snapshot

Additional file 2 of Pre-stroke cognitive impairment is associated with vascular imaging pathology: a prospective observational study

inferred← citeshttps://doi.org/10.6084/m9.figshare.14782755openalex-snapshot

Additional file 3 of Pre-stroke cognitive impairment is associated with vascular imaging pathology: a prospective observational study

inferred← citeshttps://doi.org/10.6084/m9.figshare.14782758openalex-snapshot

Additional file 4 of Pre-stroke cognitive impairment is associated with vascular imaging pathology: a prospective observational study

inferred← citeshttps://doi.org/10.6084/m9.figshare.26742605openalex-snapshot

Additional file 1 of Identifying longitudinal cognitive resilience from cross-sectional amyloid, tau, and neurodegeneration

inferred← citeshttps://doi.org/10.70675/44d51667ze0fez4d16z9334zc48e6ea9d190openalex-snapshot

Diversité clinico-radiologique des microangiopathies cérébrales : Etude phénotypique et génétique de l’angiopathie amyloïde cérébrale et des calcifications cérébrales primaires

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.