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Expression and localization of GPR91 and GPR99 in murine organs

https://doi.org/10.1007/s00441-015-2318-1

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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The Epididymis

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Knobil and Neill's Physiology of Reproduction

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1H-NMR-based metabonomic analysis of metabolic profiling in diabetic nephropathy rats induced by streptozotocin

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Succinate receptor GPR91 provides a direct link between high glucose levels and renin release in murine and rabbit kidney

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α-Ketoglutarate regulates acid-base balance through an intrarenal paracrine mechanism

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Biologically active polypeptides in submandibular glands.

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What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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GPR91 mediates low shear stress-induced endothelial ferroptosis via EGR1-dependent GPX4 repression

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Discovery of a SUCNR1 antagonist for potential treatment of diabetic nephropathy: In silico and in vitro studies

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Retraction Note: Alpha-ketoglutarate promotes skeletal muscle hypertrophy and protein synthesis through Akt/mTOR signaling pathways

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RETRACTED ARTICLE: Alpha-ketoglutarate promotes skeletal muscle hypertrophy and protein synthesis through Akt/mTOR signaling pathways

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α‐Ketoglutarate prevents skeletal muscle protein degradation and muscle atrophy through PHD3/ADRB2 pathway

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Succinate-SUCNR1 induces renal tubular cell apoptosis

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SUCNR1 regulates insulin secretion and glucose elevates the succinate response in people with prediabetes

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Activation of 2‐oxoglutarate receptor 1 ( OXGR1 ) by α‐ketoglutarate ( αKG ) does not detectably stimulate Pendrin‐mediated anion exchange in Xenopus oocytes

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Exercise‐induced α‐ketoglutaric acid stimulates muscle hypertrophy and fat loss through OXGR1‐dependent adrenal activation

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Succinate induces skeletal muscle fiber remodeling via SUCNR1 signaling

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Alpha-ketoglutaric acid ameliorates hyperglycemia in diabetes by inhibiting hepatic gluconeogenesis via serpina1e signaling

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Additional file 1 of The pathogenic role of succinate-SUCNR1: a critical function that induces renal fibrosis via M2 macrophage

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Additional file 2 of The pathogenic role of succinate-SUCNR1: a critical function that induces renal fibrosis via M2 macrophage

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The role of dietary sugars in intestinal homeostasis and microbiota composition

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