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Neonatal 6-hydroxydopamine lesioning of rats and dopaminergic neurotoxicity: proposed animal model of Parkinson’s disease

https://doi.org/10.1007/s00702-022-02479-4

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2 of the 190 references listed on this page are in the Retraction Watch database as retracted (export 2026-10-28): 10.1080/21655979.2021.2016046 (notice 2024-02-20), 10.3233/jpd-140368 (notice 2025-04-16).

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Outgoing claims: the papers this work cites, builds on, or argues with.

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Neurochemical Compensation After Nigrostriatal Bundle Injury in an Animal Model of Preclinical Parkinsonism

inferredcites →https://doi.org/10.1002/ana.24066openalex-snapshot

Lewy body extracts from Parkinson disease brains trigger α‐synuclein pathology and neurodegeneration in mice and monkeys

inferredcites →https://doi.org/10.1002/cne.20305openalex-snapshot

Variable effects of chronic subcutaneous administration of rotenone on striatal histology

inferredcites →https://doi.org/10.1002/cne.902450209openalex-snapshot

Sprouting of serotoninergic afferents into striatum after dopamine‐depleting lesions in infant rats: A retrograde transport and immunocytochemical study

inferredcites →https://doi.org/10.1002/jcp.28907openalex-snapshot

MicroRNA‐190 alleviates neuronal damage and inhibits neuroinflammation via Nlrp3 in MPTP‐induced Parkinson's disease mouse model

inferredcites →https://doi.org/10.1006/exnr.2002.8072openalex-snapshot

Subcutaneous Rotenone Exposure Causes Highly Selective Dopaminergic Degeneration and α-Synuclein Aggregation

inferredcites →https://doi.org/10.1006/nbdi.2002.0507openalex-snapshot

Environmental Risk Factors and Parkinson's Disease: Selective Degeneration of Nigral Dopaminergic Neurons Caused by the Herbicide Paraquat

inferredcites →https://doi.org/10.1006/taap.1996.0054openalex-snapshot

Effects of Paraquat on the Substantia Nigra of the Wistar Rats: Neurochemical, Histological, and Behavioral Studies

inferredcites →https://doi.org/10.1007/7854_2015_395openalex-snapshot

Lifelong Rodent Model of Tardive Dyskinesia—Persistence After Antipsychotic Drug Withdrawal

inferredcites →https://doi.org/10.1007/7854_2015_396openalex-snapshot

Perinatal 6-Hydroxydopamine to Produce a Lifelong Model of Severe Parkinson’s Disease

inferredcites →https://doi.org/10.1007/978-1-4684-5191-7_13openalex-snapshot

Neurobiology of D1 Dopamine Receptors after Neonatal-6-OHDA Treatment: Relevance to Lesch-Nyhan Disease

inferredcites →https://doi.org/10.1007/978-3-211-45295-0_43openalex-snapshot

Proposed animal model of severe Parkinson’s disease: neonatal 6-hydroxydopamine lesion of dopaminergic innervation of striatum

inferredcites →https://doi.org/10.1007/bf02245863openalex-snapshot

Enhanced oral activity responses to intrastriatal SKF 38393 andm-CPP are attenuated by intrastriatal mianserin in neonatal 6-OHDA-lesioned rats

inferredcites →https://doi.org/10.1007/bf03033376openalex-snapshot

Dopamine receptor supersensitivity: An outcome and index of neurotoxicity

inferredcites →https://doi.org/10.1007/bf03033391openalex-snapshot

Amphetamine and mCPP Effects on Dopamine and Serotonin Striatalin vivo Microdialysates in an Animal Model of Hyperactivity

inferredcites →https://doi.org/10.1007/bf03033442openalex-snapshot

Serotoninergics attenuate hyperlocomotor activity in rats. Potential new therapeutic strategy for hyperactivity

inferredcites →https://doi.org/10.1007/bf03033481openalex-snapshot

Modeling tardive dyskinesia: Predictive 5-HT2C receptor antagonist treatment

inferredcites →https://doi.org/10.1007/bf03033804openalex-snapshot

Dopamine receptor supersensitivity: Development, mechanisms, presentation, and clinical applicability

inferredcites →https://doi.org/10.1007/s00018-011-0850-zopenalex-snapshot

Transgenic expression and activation of PGC-1α protect dopaminergic neurons in the MPTP mouse model of Parkinson’s disease

inferredcites →https://doi.org/10.1007/s00401-012-1040-2openalex-snapshot

Parkinson’s disease is not associated with gastrointestinal myenteric ganglion neuron loss

inferredcites →https://doi.org/10.1007/s00702-002-0808-2openalex-snapshot

Idiopathic Parkinson's disease: possible routes by which vulnerable neuronal types may be subject to neuroinvasion by an unknown pathogen

inferredcites →https://doi.org/10.1007/s00702-011-0682-xopenalex-snapshot

Biomarker candidates of neurodegeneration in Parkinson’s disease for the evaluation of disease-modifying therapeutics

inferredcites →https://doi.org/10.1007/s00702-019-02025-9openalex-snapshot

α-Synuclein in Parkinson’s disease: causal or bystander?

inferredcites →https://doi.org/10.1007/s00702-020-02192-0openalex-snapshot

The role of alpha-synuclein as ferrireductase in neurodegeneration associated with Parkinson’s disease

inferredcites →https://doi.org/10.1007/s00702-021-02414-zopenalex-snapshot

Iron as the concert master in the pathogenic orchestra playing in sporadic Parkinson’s disease

inferredcites →https://doi.org/10.1007/s11011-021-00840-2openalex-snapshot

Impaired mitochondrial functions and energy metabolism in MPTP-induced Parkinson’s disease: comparison of mice strains and dose regimens

inferredcites →https://doi.org/10.1007/s11064-017-2373-yopenalex-snapshot

Paraquat-Induced Movement Disorder in Relation to Oxidative Stress-Mediated Neurodegeneration in the Brain of Drosophila melanogaster

inferredcites →https://doi.org/10.1007/s12031-020-01660-2openalex-snapshot

NEAT1 Decreasing Suppresses Parkinson’s Disease Progression via Acting as miR-1301-3p Sponge

inferredcites →https://doi.org/10.1007/s12035-020-02128-5openalex-snapshot

Molecular Regulatory Mechanism and Toxicology of Neurodegenerative Processes in MPTP/Probenecid-Induced Progressive Parkinson’s Disease Mice Model Revealed by Transcriptome

inferredcites →https://doi.org/10.1007/s12640-011-9268-5openalex-snapshot

Neonatal 6-Hydroxydopamine Lesioning Enhances Quinpirole-Induced Vertical Jumping in Rats that were Quinpirole Primed During Postnatal Ontogeny

inferredcites →https://doi.org/10.1007/s12640-013-9401-8openalex-snapshot

Evaluation of Nigrostriatal Neurodegeneration and Neuroinflammation Following Repeated Intranasal 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP) Administration in Mice, an Experimental Model of Parkinson’s Disease

inferredcites →https://doi.org/10.1007/s12640-019-00145-4openalex-snapshot

p-Chloroamphetamine-Enhanced Neostriatal Dopamine Exocytosis in Rats Neonatally Co-lesioned with 6-OHDA and 5,7-DHT: Relevance to Parkinson’s Disease

inferredcites →https://doi.org/10.1007/s13577-017-0187-5openalex-snapshot

MicroRNA-30e regulates neuroinflammation in MPTP model of Parkinson’s disease by targeting Nlrp3

inferredcites →https://doi.org/10.1016/0006-8993(84)90665-6openalex-snapshot

Apparent sprouting of striatal serotonergic terminals after dopamine-depleting brain lesions in neonatal rats

inferredcites →https://doi.org/10.1016/0006-8993(84)90777-7openalex-snapshot

Selective nigral toxicity after systemic administration of 1-methyl-4-phenyl-1,2,5,6-tetrahydropyrine (MPTP) in the squirrel monkey

inferredcites →https://doi.org/10.1016/0006-8993(85)90667-5openalex-snapshot

Hyperinnervation of the striatum by dorsal raphe afferents after dopamine-depleting brain lesions in neonatal rats

inferredcites →https://doi.org/10.1016/0006-8993(90)90036-bopenalex-snapshot

Elevation of dopamine D2 but not D1 receptors in adult rat neostriatum after neonatal 6-hydroxydopamine denervation

inferredcites →https://doi.org/10.1016/0006-8993(90)90474-popenalex-snapshot

MPTP-induced parkinsonism: relative changes in dopamine concentration in subregions of substantia nigra, ventral tegmental area and retrorubral field of symptomatic and asymptomatic vervet monkeys

inferredcites →https://doi.org/10.1016/0006-8993(90)90576-wopenalex-snapshot

Evaluation of a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated C57 black mouse model for parkinsonism

inferredcites →https://doi.org/10.1016/0006-8993(92)90363-eopenalex-snapshot

Ultrastructural analysis of the serotonin hyperinnervation in adult rat neostriatum following neonatal dopamine denervation with 6-hydroxydopamine

inferredcites →https://doi.org/10.1016/0006-8993(93)90995-yopenalex-snapshot

Serotonin 5-HT1 and 5-HT2 receptors in adult rat brain after neonatal destruction of nigrostriatal dopamine neurons: a quantitative autoradiographic study

inferredcites →https://doi.org/10.1016/0006-8993(94)01210-9openalex-snapshot

Quantification of the serotonin hyperinnervation in adult rat neostriatum after neonatal 6-hydroxydopamine lesion of nigral dopamine neurons

inferredcites →https://doi.org/10.1016/0014-2999(77)90256-4openalex-snapshot

Supersensitivity to apomorphine following destruction of the ascending dopamine neurons: Quantification using the rotational model

inferredcites →https://doi.org/10.1016/0014-2999(85)90349-8openalex-snapshot

SCH-23390 antagonism of a D-2 dopamine agonist depends upon catecholaminergic neurons

inferredcites →https://doi.org/10.1016/0014-2999(85)90528-xopenalex-snapshot

Mesolimbic dopaminergic neurons are not spared by MPTP neurotoxicity in mice

inferredcites →https://doi.org/10.1016/0014-2999(86)90482-6openalex-snapshot

1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration to C57-black mice leads to parallel decrements in neostriatal dopamine content and tyrosine hydroxylase activity

inferredcites →https://doi.org/10.1016/0014-2999(90)94542-6openalex-snapshot

Production of prolonged supersensitization of dopamine D2 receptors

inferredcites →https://doi.org/10.1016/0014-2999(94)90316-6openalex-snapshot

Hypersensitivity to serotonin and its agonists in serotonin-hyperinnervated neostriatum after neonatal dopamine denervation

inferredcites →https://doi.org/10.1016/0014-2999(94)90771-4openalex-snapshot

Enhanced oral activity response to A77636 in neonatal 6-hydroxydopamine-lesioned rats

inferredcites →https://doi.org/10.1016/0014-2999(94)90803-6openalex-snapshot

Persistent oral dyskinesias in haloperidol-withdrawn neonatal 6-hydroxydopamine-lesioned rats

inferredcites →https://doi.org/10.1016/0016-5085(84)90079-9openalex-snapshot

Esophageal Lewy bodies associated with ganglion cell loss in achalasia

inferredcites →https://doi.org/10.1016/0024-3205(85)90146-8openalex-snapshot

Inhibition of NADH-linked oxidation in brain mitochondria by 1-methyl-4-phenyl-pyridine, a metabolite of the neurotoxin, 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridine

inferredcites →https://doi.org/10.1016/0024-3205(87)90588-1openalex-snapshot

Behavioral and biochemical changes following acute administration of MPTP and MPP+

inferredcites →https://doi.org/10.1016/0091-3057(91)90146-sopenalex-snapshot

Supersensitized D1 receptors mediate enhanced oral activity after neonatal 6-OHDA

inferredcites →https://doi.org/10.1016/0091-3057(91)90614-8openalex-snapshot

Potentiation of spiperone-induced oral activity in rats after neonatal 6-hydroxydopamine

inferredcites →https://doi.org/10.1016/0091-3057(92)90383-qopenalex-snapshot

Supersensitized oral responses to a serotonin agonist in neonatal 6-OHDA-treated rats

inferredcites →https://doi.org/10.1016/0091-3057(93)90534-zopenalex-snapshot

Enhanced pilocarpine-induced oral activity responses in neonatal 6-OHDA treated rats

inferredcites →https://doi.org/10.1016/0091-3057(94)00328-9openalex-snapshot

Enhanced quinpirole response in rats lesioned neonatally with 5,7-dihydroxytryptamine

inferredcites →https://doi.org/10.1016/0149-7634(94)00019-wopenalex-snapshot

Dopamine receptor supersensitivity

inferredcites →https://doi.org/10.1016/0165-3806(93)90126-uopenalex-snapshot

Age-dependence of a 6-hydroxydopamine lesion on SKF 38393- and m-chlorophenylpiperazine-induced oral activity responses of rats

inferredcites →https://doi.org/10.1016/0165-3806(93)90211-ropenalex-snapshot

Dose-related effects of a neonatal 6-OHDA lesion on SKF 38393- and m-chlorophenylpiperazine-induced oral activity responses of rats

inferredcites →https://doi.org/10.1016/0304-3940(85)90074-6openalex-snapshot

Effects of N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine and its metabolite, N-methyl-4-phenylpyridinium ion, on dopaminergic nigrostriatal neurons in the mouse

inferredcites →https://doi.org/10.1016/0304-3940(85)90580-4openalex-snapshot

Dopaminergic toxicity of rotenone and the 1-methyl-4-phenylpyridinium ion after their stereotaxic administration to rats: Implication for the mechanism of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine toxicity

inferredcites →https://doi.org/10.1016/0304-3940(87)90366-1openalex-snapshot

Inhibition of ATP synthesis by 1-methyl-4-phenylpyridinium ion (MPP+) in isolated mitochondria from mouse brains

inferredcites →https://doi.org/10.1016/0304-3940(87)90369-7openalex-snapshot

Difference in monoamine oxidase B activity between C57 black and albino NMRI mouse strains may explain differential effects of the neurotoxin MPTP

inferredcites →https://doi.org/10.1016/0304-3940(89)90176-6openalex-snapshot

Comparison of the effects of intracerebrally administered MPP+ (1-methyl-4-phenylpyridinium) in three species: microdialysis of dopamine and metabolites in mouse, rat and monkey striatum

inferredcites →https://doi.org/10.1016/0306-4522(93)90011-4openalex-snapshot

Changes of D1 and D2 receptors in adult rat neostriatum after neonatal dopamine denervation: Quantitative data from ligand binding,in situ hybridization and iontophoresis

inferredcites →https://doi.org/10.1016/0306-4522(94)90100-7openalex-snapshot

Dopamine neoinnervation in the substantia nigra and hyperinnervation in the interpeduncular nucleus of adult rat following neonatal cerebroventricular administration of 6-hydroxydopamine

inferredcites →https://doi.org/10.1016/0361-9230(94)90013-2openalex-snapshot

Proposed animal model of attention deficit hyperactivity disorder

inferredcites →https://doi.org/10.1016/b978-0-444-63945-5.00017-9openalex-snapshot

Prion-like propagation of pathology in Parkinson disease

inferredcites →https://doi.org/10.1016/j.bbapap.2015.01.007openalex-snapshot

Proteomic profiling in MPTP monkey model for early Parkinson disease biomarker discovery

inferredcites →https://doi.org/10.1016/j.bbrc.2016.11.144openalex-snapshot

Injury-stimulated Sonic hedgehog expression in microglia contributes to neuroinflammatory response in the MPTP model of Parkinson's disease

inferredcites →https://doi.org/10.1016/j.bj.2018.02.005openalex-snapshot

Nrf2 activation attenuates the early suppression of mitochondrial respiration due to the α-synuclein overexpression

inferredcites →https://doi.org/10.1016/j.brainres.2014.09.046openalex-snapshot

α-Synuclein amino terminus regulates mitochondrial membrane permeability

inferredcites →https://doi.org/10.1016/j.brainres.2017.12.002openalex-snapshot

Characterization of early-onset motor deficits in the Pink1−/− mouse model of Parkinson disease

inferredcites →https://doi.org/10.1016/j.brainres.2018.10.033openalex-snapshot

In search of early neuroradiological biomarkers for Parkinson’s Disease: Alterations in resting state functional connectivity and gray matter microarchitecture in PINK1 −/− rats

inferredcites →https://doi.org/10.1016/j.ejphar.2006.08.092openalex-snapshot

Histamine H3 receptor agonist- and antagonist-evoked vacuous chewing movements in 6-OHDA-lesioned rats occurs in an absence of change in microdialysate dopamine levels

inferredcites →https://doi.org/10.1016/j.ejphar.2017.06.029openalex-snapshot

A novel GLP-1/GIP dual agonist is more effective than liraglutide in reducing inflammation and enhancing GDNF release in the MPTP mouse model of Parkinson's disease

inferredcites →https://doi.org/10.1016/j.etp.2012.09.001openalex-snapshot

Toxicity of MPTP on neurotransmission in three mouse models of Parkinson's disease

inferredcites →https://doi.org/10.1016/j.expneurol.2007.05.010openalex-snapshot

Loss of enteric dopaminergic neurons and associated changes in colon motility in an MPTP mouse model of Parkinson's disease

inferredcites →https://doi.org/10.1016/j.expneurol.2012.07.021openalex-snapshot

Non-human primate FOG develops with advanced parkinsonism induced by MPTP Treatment

inferredcites →https://doi.org/10.1016/j.expneurol.2015.10.010openalex-snapshot

JNK-mediated activation of ATF2 contributes to dopaminergic neurodegeneration in the MPTP mouse model of Parkinson's disease

inferredcites →https://doi.org/10.1016/j.expneurol.2021.113904openalex-snapshot

The long noncoding RNA GAS5 potentiates neuronal injury in Parkinson's disease by binding to microRNA-150 to regulate Fosl1 expression

inferredcites →https://doi.org/10.1016/j.freeradbiomed.2013.01.003openalex-snapshot

Oxidative damage to macromolecules in human Parkinson disease and the rotenone model

inferredcites →https://doi.org/10.1016/j.ijbiomac.2016.10.021openalex-snapshot

α-synuclein aggregation and its modulation

inferredcites →https://doi.org/10.1016/j.intimp.2020.106441openalex-snapshot

NLRP3 inflammasome and glia maturation factor coordinately regulate neuroinflammation and neuronal loss in MPTP mouse model of Parkinson’s disease

inferredcites →https://doi.org/10.1016/j.jmb.2011.06.026openalex-snapshot

Structured Regions of α-Synuclein Fibrils Include the Early-Onset Parkinson's Disease Mutation Sites

inferredcites →https://doi.org/10.1016/j.jneumeth.2018.10.037openalex-snapshot

A non-human primate model for stable chronic Parkinson’s disease induced by MPTP administration based on individual behavioral quantification

inferredcites →https://doi.org/10.1016/j.molbrainres.2004.11.005openalex-snapshot

Redox cycling of the herbicide paraquat in microglial cultures

inferredcites →https://doi.org/10.1016/j.nbd.2009.06.017openalex-snapshot

Chronic rotenone exposure reproduces Parkinson's disease gastrointestinal neuropathology

inferredcites →https://doi.org/10.1016/j.nbd.2015.06.003openalex-snapshot

Intrastriatal injection of pre-formed mouse α-synuclein fibrils into rats triggers α-synuclein pathology and bilateral nigrostriatal degeneration

inferredcites →https://doi.org/10.1016/j.neulet.2021.136177openalex-snapshot

GSK-3 mediates nuclear translocation of p62/SQSTM1 in MPTP-induced mouse model of Parkinson’s disease

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2017.05.022openalex-snapshot

Homozygous alpha-synuclein p.A53V in familial Parkinson's disease

inferredcites →https://doi.org/10.1016/j.neuron.2005.05.010openalex-snapshot

Effects of α-Synuclein Immunization in a Mouse Model of Parkinson’s Disease

inferredcites →https://doi.org/10.1016/j.neuroscience.2006.05.039openalex-snapshot

Degeneration of dopaminergic mesocortical neurons and activation of compensatory processes induced by a long-term paraquat administration in rats: Implications for Parkinson’s disease

inferredcites →https://doi.org/10.1016/j.neuroscience.2020.06.001openalex-snapshot

Down-regulation of DJ-1 Augments Neuroinflammation via Nrf2/Trx1/NLRP3 Axis in MPTP-induced Parkinson’s Disease Mouse Model

inferredcites →https://doi.org/10.1016/j.tins.2010.09.004openalex-snapshot

α-Synuclein and dopamine at the crossroads of Parkinson's disease

inferredcites →https://doi.org/10.1016/s0006-291x(86)80483-1openalex-snapshot

Uptake of the neurotoxin 1-methyl-4-phenylpyridine (MPP+) by mitochondria and its relation to the inhibition of the mitochondrial oxidation of NAD+-linked substrates by MPP+

inferredcites →https://doi.org/10.1016/s0006-8993(00)02496-3openalex-snapshot

Potentiated and preferential effects of combined paraquat and maneb on nigrostriatal dopamine systems: environmental risk factors for Parkinson’s disease?

inferredcites →https://doi.org/10.1016/s0006-8993(98)01192-5openalex-snapshot

Paraquat elicited neurobehavioral syndrome caused by dopaminergic neuron loss

inferredcites →https://doi.org/10.1016/s0021-9258(19)42751-8openalex-snapshot

The Generation of Hydrogen Peroxide, Superoxide Radical, and Hydroxyl Radical by 6-Hydroxydopamine, Dialuric Acid, and Related Cytotoxic Agents

inferredcites →https://doi.org/10.1016/s0021-9258(19)57434-8openalex-snapshot

Energy-dependent uptake of N-methyl-4-phenylpyridinium, the neurotoxic metabolite of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine, by mitochondria.

inferredcites →https://doi.org/10.1016/s0022-3565(24)36400-6openalex-snapshot

Persistent Spontaneous Oral Dyskinesias in Haloperidol-Withdrawn Rats Neonatally Lesioned with 6-Hydroxydopamine: Absence of an Association with the Bmax for [3H]Raclopride Binding to Neostriatal Homogenates

inferredcites →https://doi.org/10.1016/s0022-3565(25)11111-7openalex-snapshot

Supersensitization of the oral response to SKF 38393 in neonatal 6-OHDA-lesioned rats is mediated through a serotonin system.

inferredcites →https://doi.org/10.1016/s0022-3565(25)21630-5openalex-snapshot

Behavioral differences between neonatal and adult 6-hydroxydopamine-treated rats to dopamine agonists: relevance to neurological symptoms in clinical syndromes with reduced brain dopamine.

inferredcites →https://doi.org/10.1016/s0022-3565(25)22430-2openalex-snapshot

Altered dopamine and serotonin metabolism in the dopamine-denervated and serotonin-hyperinnervated neostriatum of adult rat after neonatal 6-hydroxydopamine.

inferredcites →https://doi.org/10.1016/s0022-3565(25)22697-0openalex-snapshot

6-hydroxydopamine treatments enhance behavioral responses to intracerebral microinjection of D1- and D2-dopamine agonists into nucleus accumbens and striatum without changing dopamine antagonist binding.

inferredcites →https://doi.org/10.1016/s0022-3565(25)23467-xopenalex-snapshot

Dopamine agonist-induced locomotor activity in rats treated with 6-hydroxydopamine at differing ages: functional supersensitivity of D-1 dopamine receptors in neonatally lesioned rats.

inferredcites →https://doi.org/10.1016/s0022-3565(25)23963-5openalex-snapshot

Evidence that D-1 dopamine receptors contribute to the supersensitive behavioral responses induced by L-dihydroxyphenylalanine in rats treated neonatally with 6-hydroxydopamine.

inferredcites →https://doi.org/10.1016/s0022-3565(25)38471-5openalex-snapshot

Supersensitization of the oral response to SKF 38393 in neonatal 6-hydroxydopamine-lesioned rats is eliminated by neonatal 5,7-dihydroxytryptamine treatment.

inferredcites →https://doi.org/10.1016/s0091-3057(84)80110-0openalex-snapshot

Neonatal-6-hydroxydopamine treatment: Model of susceptibility for self-mutilation in the lesch-nyhan syndrome

inferredcites →https://doi.org/10.1016/s0166-4328(02)00180-8openalex-snapshot

Rotenone destroys dopaminergic neurons and induces parkinsonian symptoms in rats

inferredcites →https://doi.org/10.1016/s1734-1140(09)70037-4openalex-snapshot

Neonatal co-lesion by DSP-4 and 5,7-DHT produces adulthood behavioral sensitization to dopamine D2 receptor agonists

inferredcites →https://doi.org/10.1017/s0317167100037860openalex-snapshot

The Use of the MPTP-Treated Mouse as an Animal Model of Parkinsonism

inferredcites →https://doi.org/10.1038/12647openalex-snapshot

Intragenic deletion in the gene encoding ubiquitin carboxy-terminal hydrolase in gad mice

inferredcites →https://doi.org/10.1038/81834openalex-snapshot

Chronic systemic pesticide exposure reproduces features of Parkinson's disease

inferredcites →https://doi.org/10.1038/emboj.2010.223openalex-snapshot

Inhibition of mitochondrial fusion by α‐synuclein is rescued by PINK1, Parkin and DJ‐1

inferredcites →https://doi.org/10.1038/nn.2349openalex-snapshot

Mutant LRRK2R1441G BAC transgenic mice recapitulate cardinal features of Parkinson's disease

inferredcites →https://doi.org/10.1038/s41467-018-06111-6openalex-snapshot

Mutant FUS causes DNA ligation defects to inhibit oxidative damage repair in Amyotrophic Lateral Sclerosis

inferredcites →https://doi.org/10.1038/s41598-019-47227-zopenalex-snapshot

Alpha-synuclein is a DNA binding protein that modulates DNA repair with implications for Lewy body disorders

inferredcites →https://doi.org/10.1046/j.1460-9568.2003.02781.xopenalex-snapshot

Age‐related irreversible progressive nigrostriatal dopaminergic neurotoxicity in the paraquat and maneb model of the Parkinson's disease phenotype

inferredcites →https://doi.org/10.1046/j.1471-4159.1994.63041282.xopenalex-snapshot

Neonatal 6‐Hydroxydopamine and Adult SKF 38393 Treatments Alter Dopamine D1 Receptor mRNA Levels: Absence of Other Neurochemical Associations with the Enhanced Behavioral Responses of Lesioned Rats

inferredcites →https://doi.org/10.1046/j.1471-4159.1998.70030889.xopenalex-snapshot

Serotonin Neural Adaptations to Ontogenetic Loss of Dopamine Neurons in Rat Brain

inferredcites →https://doi.org/10.1073/pnas.0409598102openalex-snapshot

Parkin-deficient mice are not a robust model of parkinsonism

inferredcites →https://doi.org/10.1073/pnas.0409713102openalex-snapshot

Parkinson-like syndrome induced by continuous MPTP infusion: Convergent roles of the ubiquitin-proteasome system and α-synuclein

inferredcites →https://doi.org/10.1073/pnas.0807581105openalex-snapshot

Mitochondrial complex I inhibition is not required for dopaminergic neuron death induced by rotenone, MPP + , or paraquat

inferredcites →https://doi.org/10.1073/pnas.1604645113openalex-snapshot

Mutations associated with familial Parkinson’s disease alter the initiation and amplification steps of α-synuclein aggregation

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Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects

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Bach1 derepression is neuroprotective in a mouse model of Parkinson’s disease

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A primate model of parkinsonism: selective destruction of dopaminergic neurons in the pars compacta of the substantia nigra by N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine.

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The Herbicide Paraquat Causes Up-regulation and Aggregation of α-Synuclein in Mice

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Lewy Body-like α-Synuclein Aggregates Resist Degradation and Impair Macroautophagy

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Parkin-deficient Mice Exhibit Nigrostriatal Deficits but Not Loss of Dopaminergic Neurons

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The Herbicide Paraquat Induces Dopaminergic Nigral Apoptosis through Sustained Activation of the JNK Pathway

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Mitochondria Are a Major Source of Paraquat-induced Reactive Oxygen Species Production in the Brain

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Paraquat Neurotoxicity Is Mediated by a Bak-dependent Mechanism

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The Bipyridyl Herbicide Paraquat Produces Oxidative Stress-Mediated Toxicity in Human Neuroblastoma SH-SY5Y Cells: Relevance to the Dopaminergic Pathogenesis

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RETRACTED ARTICLE: Knockdown of forkhead box protein P1 alleviates hypoxia reoxygenation injury in H9c2 cells through regulating Pik3ip1/Akt/eNOS and ROS/mPTP pathway

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Toxicity of Redox Cycling Pesticides in Primary Mesencephalic Cultures

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Mitochondrial Permeability Transition Pore Component Cyclophilin D Distinguishes Nigrostriatal Dopaminergic Death Paradigms in the MPTP Mouse Model of Parkinson's Disease

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Targeting Nrf2-Mediated Gene Transcription by Extremely Potent Synthetic Triterpenoids Attenuate Dopaminergic Neurotoxicity in the MPTP Mouse Model of Parkinson's Disease

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LRRK2BAC transgenic rats develop progressive, L-DOPA-responsive motor impairment, and deficits in dopamine circuit function

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Modeling Parkinson's Disease in Primates: The MPTP Model

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Partial depletion and repopulation of microglia have different effects in the acute MPTP mouse model of Parkinson’s disease

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Adipsia and Aphagia after 6‐Hydroxydopamine Induced Degeneration of the Nigro‐striatal Dopamine System

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Mice overexpressing wild‐type human alpha‐synuclein display alterations in colonic myenteric ganglia and defecation

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Parkinson's disease: a dual‐hit hypothesis

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2‐Deoxyglucose Enhances 1‐Methyl‐4‐Phenyl‐1, 2, 3, 6‐Tetrahydropyridine‐Induced ATP Loss in the Mouse Brain

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Advances in Our Understanding of the Mechanisms of the Neurotoxicity of MPTP and Related Compounds

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Role of oxidative stress in paraquat‐induced dopaminergic cell degeneration

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Denervation, Hyperinnervation, and Interactive Regulation of Dopamine and Serotonin Receptorsa

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Parkinson's Disease

inferredcites →https://doi.org/10.1111/jnc.13042openalex-snapshot

Rotenone decreases intracellular aldehyde dehydrogenase activity: implications for the pathogenesis of Parkinson's disease

inferredcites →https://doi.org/10.1111/nmo.12176openalex-snapshot

Alpha‐synuclein expression patterns in the colonic submucosal plexus of the aging Fischer 344 rat: implications for biopsies in aging and neurodegenerative disorders?

inferredcites →https://doi.org/10.1124/jpet.108.141861openalex-snapshot

Paraquat Exposure Reduces Nicotinic Receptor-Evoked Dopamine Release in Monkey Striatum

inferredcites →https://doi.org/10.1124/jpet.115.228510openalex-snapshot

Enteric Dysfunctions in Experimental Parkinson’s Disease: Alterations of Excitatory Cholinergic Neurotransmission Regulating Colonic Motility in Rats

inferredcites →https://doi.org/10.1124/mol.113.087965openalex-snapshot

The Iron-Binding Protein Lactoferrin Protects Vulnerable Dopamine Neurons from Degeneration by Preserving Mitochondrial Calcium Homeostasis

inferredcites →https://doi.org/10.1126/science.1195227openalex-snapshot

α-Synuclein Promotes SNARE-Complex Assembly in Vivo and in Vitro

inferredcites →https://doi.org/10.1126/science.181.4098.456openalex-snapshot

6-Hydroxydopamine: Evidence for Superoxide Radical as an Oxidative Intermediate

inferredcites →https://doi.org/10.1126/science.287.5456.1265openalex-snapshot

Dopaminergic Loss and Inclusion Body Formation in α-Synuclein Mice: Implications for Neurodegenerative Disorders

inferredcites →https://doi.org/10.1126/science.6823561openalex-snapshot

Chronic Parkinsonism in Humans Due to a Product of Meperidine-Analog Synthesis

inferredcites →https://doi.org/10.1155/2019/7935820openalex-snapshot

Atrophic Myenteric and Submucosal Neurons Are Observed in Parkinson’s Disease

inferredcites →https://doi.org/10.1186/1471-2202-10-88openalex-snapshot

Paraquat induces oxidative stress, neuronal loss in substantia nigra region and Parkinsonism in adult rats: Neuroprotection and amelioration of symptoms by water-soluble formulation of Coenzyme Q10

inferredcites →https://doi.org/10.1186/s13041-016-0237-zopenalex-snapshot

Altered DNA base excision repair profile in brain tissue and blood in Alzheimer’s disease

inferredcites →https://doi.org/10.1186/s40478-017-0416-xopenalex-snapshot

AAV1/2-induced overexpression of A53T-α-synuclein in the substantia nigra results in degeneration of the nigrostriatal system with Lewy-like pathology and motor impairment: a new mouse model for Parkinson’s disease

inferredcites →https://doi.org/10.1212/01.wnl.0000271080.53272.c7openalex-snapshot

α-Synuclein gene duplication is present in sporadic Parkinson disease

inferredcites →https://doi.org/10.1212/wnl.43.6.1150openalex-snapshot

Environmental antecedents of young‐onset Parkinson's disease

inferredcites →https://doi.org/10.1242/dmm.049082openalex-snapshot

The rat rotenone model reproduces the abnormal pattern of central catecholamine metabolism found in Parkinson's disease

inferredcites →https://doi.org/10.1371/journal.pone.0005777openalex-snapshot

Parkinson Phenotype in Aged PINK1-Deficient Mice Is Accompanied by Progressive Mitochondrial Dysfunction in Absence of Neurodegeneration

inferredcites →https://doi.org/10.1371/journal.pone.0030745openalex-snapshot

Paraquat, but Not Maneb, Induces Synucleinopathy and Tauopathy in Striata of Mice through Inhibition of Proteasomal and Autophagic Pathways

inferredcites →https://doi.org/10.1371/journal.pone.0034713openalex-snapshot

Differential Effects of UCHL1 Modulation on Alpha-Synuclein in PD-Like Models of Alpha-Synucleinopathy

inferredcites →https://doi.org/10.1385/mn:31:1-3:081openalex-snapshot

Oxidative Stress in Mitochondria: Decision to Survival and Death of Neurons in Neurodegenerative Disorders

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Distinct Nrf2 Signaling Mechanisms of Fumaric Acid Esters and Their Role in Neuroprotection against 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Induced Experimental Parkinson's-Like Disease

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-Synuclein Phosphorylation Enhances Eosinophilic Cytoplasmic Inclusion Formation in SH-SY5Y Cells

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The Nigrostriatal Dopaminergic System as a Preferential Target of Repeated Exposures to Combined Paraquat and Maneb: Implications for Parkinson's Disease

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α-Synuclein Is Localized to Mitochondria-Associated ER Membranes

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Early and Progressive Sensorimotor Anomalies in Mice Overexpressing Wild-Type Human α-Synuclein

inferredcites →https://doi.org/10.1523/jneurosci.5314-13.2014openalex-snapshot

Reducing C-Terminal-Truncated Alpha-Synuclein by Immunotherapy Attenuates Neurodegeneration and Propagation in Parkinson's Disease-Like Models

inferredcites →https://doi.org/10.18154/rwth-conv-084442openalex-snapshot

Frequency Dependence of Signal Power and Spatial Reach of the Local Field Potential

inferredcites →https://doi.org/10.18632/aging.203545openalex-snapshot

Deficiency of miR-29b2/c leads to accelerated aging and neuroprotection in MPTP-induced Parkinson’s disease mice

inferredcites →https://doi.org/10.2147/tacg.s11639openalex-snapshot

Genetic basis of Parkinson's disease: inheritance, penetrance, and expression

inferredcites →https://doi.org/10.3233/jpd-140368openalex-snapshot

RETRACTED: Genetic Susceptibility Model of Parkinson’s Disease Resulting from Exposure of DJ-1 Deficient Mice to MPTP: Evaluation of Neuroprotection by Ubisol-Q10

inferredcites →https://doi.org/10.3233/jpd-160779openalex-snapshot

Implication of Alpha-Synuclein Phosphorylation at S129 in Synucleinopathies: What Have We Learned in the Last Decade?

inferredcites →https://doi.org/10.3233/rnn-110222openalex-snapshot

Modeling early Parkinson's disease pathology with chronic low dose MPTP treatment

inferredcites →https://doi.org/10.3390/ijms17020151openalex-snapshot

Microglia-Mediated Neuroinflammation and Neurotrophic Factor-Induced Protection in the MPTP Mouse Model of Parkinson’s Disease-Lessons from Transgenic Mice

inferredcites →https://doi.org/10.3390/ijms21093254openalex-snapshot

Chronic Systemic Exposure to Low-Dose Rotenone Induced Central and Peripheral Neuropathology and Motor Deficits in Mice: Reproducible Animal Model of Parkinson’s Disease

inferredcites →https://doi.org/10.3390/ijms22094643openalex-snapshot

Genes Implicated in Familial Parkinson’s Disease Provide a Dual Picture of Nigral Dopaminergic Neurodegeneration with Mitochondria Taking Center Stage

inferredcites →https://doi.org/10.3390/ijms222312616openalex-snapshot

Decrease in ITGA7 Levels Is Associated with an Increase in α-Synuclein Levels in an MPTP-Induced Parkinson’s Disease Mouse Model and SH-SY5Y Cells

inferredcites →https://doi.org/10.5607/en.2019.28.3.414openalex-snapshot

Abnormal Mitochondria in a Non-human Primate Model of MPTP-induced Parkinson's Disease: Drp1 and CDK5/p25 Signaling

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Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Facile Biological Oxidation of Dopamine to 6-Hydroxydopamine p -Quinone in a Sequential Two-Step Process: Implications for Parkinson’s Disease

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PT109B, a Multikinase Inhibitor, Converts Astrocytes into Dopaminergic Neurons and Alleviates Parkinson's Disease in Mice

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Behavior Changes in Quinpirole Obsessive-Compulsive Disorder Rats Treated with 6-Hydroxydopamine and the Corresponding Dopaminergic Compulsive Loop Mechanism

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Overexpression of Lmx1a/NeuroD1 Mediates the Differentiation of Pulmonary Mesenchymal Stem Cells into Dopaminergic Neurons and Repairs Motor Dysfunction in Parkinson’s Disease Rats

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Chiisanoside Mediates the Parkin/ZNF746/PGC-1α Axis by Downregulating MiR-181a to Improve Mitochondrial Biogenesis in 6-OHDA-Caused Neurotoxicity Models In Vitro and In Vivo: Suggestions for Prevention of Parkinson’s Disease

inferred← citeshttps://doi.org/10.3390/brainsci13040538openalex-snapshot

Dopaminergic System in Promoting Recovery from General Anesthesia

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Syringin Prevents 6-Hydroxydopamine Neurotoxicity by Mediating the MiR-34a/SIRT1/Beclin-1 Pathway and Activating Autophagy in SH-SY5Y Cells and the Caenorhabditis elegans Model

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Ginkgolide B Attenuates 6-Hydroxydopamine-Induced Cellular Damage and Modulates Oxidant-Evoked Ca2+ Signalling in SH-SY5Y Cells

inferred← citeshttps://doi.org/10.4103/1673-5374.380878openalex-snapshot

Single-neuron neurodegeneration as a degenerative model for Parkinson’s disease

inferred← citeshttps://doi.org/10.61845/agrimedical.1471854openalex-snapshot

Experimental Models in Parkinson’s Disease: Advantages and Disadvantages

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