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Upregulation of complement C1q reflects mucosal regeneration in a mouse model of colitis

https://doi.org/10.1007/s00795-020-00266-2

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/aja.1001410407openalex-snapshot

Origin, differentiation and renewal of the four main epithelial cell types in the mouse small intestine V. Unitarian theory of the origin of the four epithelial cell types

inferredcites →https://doi.org/10.1016/0016-5085(89)91409-1openalex-snapshot

Persistent complement activation in submucosal blood vessels of active inflammatory bowel disease: Immunohistochemical evidence

inferredcites →https://doi.org/10.1016/j.bbadis.2012.06.011openalex-snapshot

Alteration of colonic stem cell gene signatures during the regenerative response to injury

inferredcites →https://doi.org/10.1016/j.cell.2012.03.047openalex-snapshot

Complement C1q Activates Canonical Wnt Signaling and Promotes Aging-Related Phenotypes

inferredcites →https://doi.org/10.1038/ajg.2015.120openalex-snapshot

C-Reactive Protein, Fecal Calprotectin, and Stool Lactoferrin for Detection of Endoscopic Activity in Symptomatic Inflammatory Bowel Disease Patients: A Systematic Review and Meta-Analysis

inferredcites →https://doi.org/10.1038/mi.2013.73openalex-snapshot

New targets for mucosal healing and therapy in inflammatory bowel diseases

inferredcites →https://doi.org/10.1038/mi.2015.63openalex-snapshot

Wound repair: role of immune–epithelial interactions

inferredcites →https://doi.org/10.1038/ncomms7241openalex-snapshot

Complement C1q-induced activation of β-catenin signalling causes hypertensive arterial remodelling

inferredcites →https://doi.org/10.1038/nrgastro.2009.203openalex-snapshot

Clinical implications of mucosal healing for the management of IBD

inferredcites →https://doi.org/10.1038/nrm1203openalex-snapshot

Trefoil factors: initiators of mucosal healing

inferredcites →https://doi.org/10.1053/gast.2002.32994openalex-snapshot

Immunostimulatory DNA ameliorates experimental and spontaneous murine colitis

inferredcites →https://doi.org/10.1053/j.gastro.2007.10.029openalex-snapshot

Tracking Down the Stem Cells of the Intestine: Strategies to Identify Adult Stem Cells

inferredcites →https://doi.org/10.1053/j.gastro.2015.07.003openalex-snapshot

Biomarkers of Inflammation in Inflammatory Bowel Disease

inferredcites →https://doi.org/10.1056/nejm199005103221903openalex-snapshot

Enhanced Local Production of Complement Components in the Small Intestines of Patients with Crohn's Disease

inferredcites →https://doi.org/10.1093/ecco-jcc/jjw132openalex-snapshot

Leucine-rich Alpha-2 Glycoprotein is a Serum Biomarker of Mucosal Healing in Ulcerative Colitis

inferredcites →https://doi.org/10.1097/mpg.0000000000001444openalex-snapshot

Correlation of Erythrocyte Sedimentation Rate and C‐Reactive Protein With Pediatric Inflammatory Bowel Disease Activity

inferredcites →https://doi.org/10.1111/cei.12464openalex-snapshot

Regulation of the alternative pathway of complement modulates injury and immunity in a chronic model of dextran sulphate sodium-induced colitis

inferredcites →https://doi.org/10.1126/science.1180794openalex-snapshot

Coexistence of Quiescent and Active Adult Stem Cells in Mammals

inferredcites →https://doi.org/10.1126/science.1248012openalex-snapshot

An integral program for tissue renewal and regeneration: Wnt signaling and stem cell control

inferredcites →https://doi.org/10.1136/gut.30.3.361openalex-snapshot

Deposits of terminal complement complex (TCC) in muscularis mucosae and submucosal vessels in ulcerative colitis and Crohn's disease of the colon.

inferredcites →https://doi.org/10.1136/gut.33.7.902openalex-snapshot

Surface epithelium related activation of complement differs in Crohn's disease and ulcerative colitis.

inferredcites →https://doi.org/10.1136/gutjnl-2012-302830openalex-snapshot

Mucosal healing in inflammatory bowel diseases: a systematic review

inferredcites →https://doi.org/10.1172/jci22996openalex-snapshot

Toll-like receptor 9–induced type I IFN protects mice from experimental colitis

inferredcites →https://doi.org/10.3748/wjg.14.348openalex-snapshot

Epithelial restitution and wound healing in inflammatory bowel disease

inferredcites →https://doi.org/10.4049/jimmunol.1200553openalex-snapshot

Complement-Dependent Injury and Protection in a Murine Model of Acute Dextran Sulfate Sodium-Induced Colitis

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4122922openalex-extracted

inferred← citeshttps://doi.org/10.1016/j.phrs.2022.106309openalex-snapshot

Paeoniflorin Prevents Aberrant Proliferation and Differentiation of Intestinal Stem Cells by Controlling C1q Release from Macrophages in Chronic Colitis

inferred← citeshttps://doi.org/10.1111/sji.13329openalex-snapshot

Revisiting the relationship between complement and ulcerative colitis

inferred← citeshttps://doi.org/10.1172/jci188348openalex-snapshot

The complement system in intestinal inflammation and cancer

inferred← citeshttps://doi.org/10.1186/s40001-025-03400-xopenalex-snapshot

Dual roles of complement in ulcerative colitis: insights from clinical studies and animal research

inferred← citeshttps://doi.org/10.3389/fimmu.2021.688036openalex-snapshot

Interleukin-22 Deficiency Contributes to Dextran Sulfate Sodium-Induced Inflammation in Japanese Medaka, Oryzias latipes

inferred← citeshttps://doi.org/10.64898/2026.09.24.753972openalex-snapshot

Gut complement C1q and C3 interact with Clostridioides difficile spores via CdeM and contribute to pathogenesis

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