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Tetracyclines and Chemically Modified Tetracycline-3 (CMT-3) Modulate Cytokine Secretion by Lipopolysaccharide-Stimulated Whole Blood

https://doi.org/10.1007/s10753-009-9111-9
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Inhibition of cell proliferation, invasion, tumor growth and metastasis by an oral non‐antimicrobial tetracycline analog (COL‐3) in a metastatic prostate cancer model
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Actinobacillus actinomycetemcomitans lipopolysaccharide regulates matrix metalloproteinase, tissue inhibitors of matrix metalloproteinase, and plasminogen activator production by human gingival fibroblasts: A potential role in connective tissue destruction
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Tetracyclines: Nonantibiotic properties and their clinical implications
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Interleukin-6 and interleukin-11 support human osteoclast formation by a RANKL-independent mechanism
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In Vitro Sensitivity of the Three Mammalian Collagenases to Tetracycline Inhibition: Relationship to Bone and Cartilage Degradation
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Concentration of 3 tetracyclines in plasma, gingival crevice fluid and saliva
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Relationship of changes in interleukin‐8 levels and granulocyte elastase activity in gingival crevicular fluid to subgingival periodontopathogens followingnon‐surgical periodontal therapy in subjects with chronic periodontitis
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Characterization of cellular infiltrate, detection of chemokine receptor CCR5 and interleukin‐8 and RANTES chemokines in adult periodontitis
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Matrix Metalloproteinase-8 Is Expressed in Rheumatoid Synovial Fibroblasts and Endothelial Cells
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Matrix metalloproteinases: Contribution to pathogenesis, diagnosis and treatment of periodontal inflammation
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Porphyromonas gingivalis-induced inflammatory mediator profile in anex vivohuman whole blood model
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Tetracyclines in the management of periodontal diseases
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Microbial complexes in subgingival plaque
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Cranberry components inhibit interleukin‐6, interleukin‐8, and prostaglandin E 2 production by lipopolysaccharide‐activated gingival fibroblasts
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The protective nature of host responses in periodontal diseases
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The host response to the microbial challenge in periodontitis: assembling the players
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Suppression of human neutrophil functions by tetracyclines
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Role of cytokines and inflammatory mediators in tissue destruction
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Matrix metalloproteinases (MMPs) in oral diseases
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Procedure for isolation of bacterial lipopolysaccharides from both smooth and rough Pseudomonas aeruginosa and Salmonella typhimurium strains
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Tetracyclines Inhibit Connective Tissue Breakdown by Multiple Non-Antimicrobial Mechanisms
inferredcites →https://doi.org/10.1177/10454411910020030201openalex-snapshot
Tetracyclines Inhibit Connective Tissue Breakdown: New Therapeutic Implications for an Old Family of Drugs
inferredcites →https://doi.org/10.1177/10454411980090030101openalex-snapshot
Cytokine Expression in Periodontal Health and Disease
inferredcites →https://doi.org/10.1177/154405910608500306openalex-snapshot
Anti-inflammatory Activity of a High-molecular-weight Cranberry Fraction on Macrophages Stimulated by Lipopolysaccharides from Periodontopathogens
inferredcites →https://doi.org/10.1177/154411130301400605openalex-snapshot
G enes and G ene P olymorphisms A ssociated with P eriodontal D isease
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Regulation of Matrix Metalloproteinases (MMP-2, -3, -9, and -13) by Interleukin-1 and Interleukin-6 in Mouse Calvaria: Association of MMP Induction with Bone Resorption
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Assessment of Interleukin‐6 in the Pathogenesis of Periodontal Disease
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Long‐Term Use of Subantimicrobial Dose Doxycycline Does Not Lead to Changes in Antimicrobial Susceptibility
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The Contribution of Interleukin‐1 and Tumor Necrosis Factor to Periodontal Tissue Destruction
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Doxycycline Reduces Lipopolysaccharide‐Induced Inflammatory Mediator Secretion in Macrophage and Ex Vivo Human Whole Blood Models
inferredcites →https://doi.org/10.4049/jimmunol.160.1.403openalex-snapshot
IL-1 and TNF Antagonists Inhibit the Inflammatory Response and Bone Loss in Experimental Periodontitis
inferredcites →https://doi.org/10.4049/jimmunol.163.6.3459openalex-snapshot
A Novel Mechanism of Action of Chemically Modified Tetracyclines: Inhibition of COX-2-Mediated Prostaglandin E2 Production
inferredcites →https://doi.org/10.4049/jimmunol.176.1.567openalex-snapshot
Doxycycline Modulates Nitric Oxide Production in Murine Lung Epithelial Cells

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Chemotactic and Immunoregulatory Properties of Bone Cells are Modulated by Endotoxin-Stimulated Lymphocytes
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Anti-inflammatory properties of Doxycycline and Minocycline in experimental models: an in vivo and in vitro comparative study
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A Tetracycline Analog Improves Acute Respiratory Distress Syndrome Survival in an Ovine Model
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Tetracyclines Modify Translation By Targeting Key Human rRNA Substructures
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Zoledronic acid affects the cytotoxic effects of Chlamydia pneumoniae and the modulation of cytokine production in human osteosarcoma cells
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Levels of gingival crevicular metalloproteinases-8 and -9 in periodontitis
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COL-3, a Chemically Modified Tetracycline, Inhibits Lipopolysaccharide-Induced Microglia Activation and Cytokine Expression in the Brain
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Tetracyclines Diminish In Vitro IFN-γ and IL-17-Producing Adaptive and Innate Immune Cells in Multiple Sclerosis
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Advantages of Dynamic Navigation in Prosthetic Implant Treatment in Terms of the Clinical Evaluation and Salivary Pro-Inflammatory Biomarkers: A Clinical Study
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The Chemically-Modified Tetracycline COL-3 and Its Parent Compound Doxycycline Prevent Microglial Inflammatory Responses by Reducing Glucose-Mediated Oxidative Stress
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Association of Cytokines, High Sensitive C-Reactive Protein, VEGF and Beta-Defensin-1 Gene Polymorphisms and Their Protein Expressions with Chronic Periodontitis in the Chinese Population
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Burn-induced distant organ ınjury in rats and the effect of minocycline
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Therapeutic application of natural inhibitors against snake venom phospholipase A2
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