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A method for isolating high quality RNA from mouse cortical and cancellous bone

https://doi.org/10.1016/j.bone.2014.07.022

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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An RNA-seq protocol to identify mRNA expression changes in mouse diaphyseal bone: Applications in mice with bone property alteringLrp5mutations

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Effects of Deletion of ERα in Osteoblast-Lineage Cells on Bone Mass and Adaptation to Mechanical Loading Differ in Female and Male Mice

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Isolation and characterization of human osteoblasts from needle biopsies without in vitro culture

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Adaptation of Tibial Structure and Strength to Axial Compression Depends on Loading History in Both C57BL/6 and BALB/c Mice

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Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor α and disuse

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Tibial compression is anabolic in the adult mouse skeleton despite reduced responsiveness with aging

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The beneficial effect of Icariin on bone is diminished in osteoprotegerin-deficient mice

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Differential load-regulated global gene expression in mouse trabecular osteocytes

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Paradoxical Sost gene expression response to mechanical unloading in metaphyseal bone

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Epidemiology and outcomes of osteoporotic fractures

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Mutation of the mouse klotho gene leads to a syndrome resembling ageing.

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Matrix-embedded cells control osteoclast formation

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The Matricellular Protein Periostin Is Required for Sost Inhibition and the Anabolic Response to Mechanical Loading and Physical Activity

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Wnt/β-Catenin Signaling Is a Component of Osteoblastic Bone Cell Early Responses to Load-bearing and Requires Estrogen Receptor α

inferredcites →https://doi.org/10.1152/japplphysiol.00210.2010openalex-snapshot

Cancellous bone adaptation to tibial compression is not sex dependent in growing mice

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Tibial Loading Increases Osteogenic Gene Expression and Cortical Bone Volume in Mature and Middle-Aged Mice

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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