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Glutathione and Thioredoxin Antioxidant Pathways Synergize to Drive Cancer Initiation and Progression
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Vitamin E and the Risk of Prostate Cancer
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Thioredoxin and thioredoxin reductase: Current research with special reference to human disease
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A NOVEL MODEL OF CONTINUOUS DEPLETION OF GLUTATHIONE IN MICE TREATED WITH L-BUTHIONINE(S,R)-SULFOXIMINE.
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Incoming claims: where this work appears in other papers’ reference lists and arguments.
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inferred← citeshttps://doi.org/10.1007/s12274-020-3241-7openalex-snapshot
Versatile iron-vitamin K3 derivative-based nanoscale coordination polymer augments tumor ferroptotic therapy
inferred← citeshttps://doi.org/10.1007/s12672-021-00439-0openalex-snapshot
The gold complex auranofin: new perspectives for cancer therapy
inferred← citeshttps://doi.org/10.1007/s13277-016-4873-9openalex-snapshot
The role of oxidative stress on breast cancer development and therapy
inferred← citeshttps://doi.org/10.1007/s13353-023-00797-1openalex-snapshot
Inhibition of the glutamate-cysteine ligase catalytic subunit with buthionine sulfoximine enhances the cytotoxic effect of doxorubicin and cyclophosphamide in Burkitt lymphoma cells
inferred← citeshttps://doi.org/10.1007/s40820-025-02006-6openalex-snapshot
Nature-Inspired Redox Shuttle with Regenerable Antioxidant for Efficient All-Perovskite Tandem Solar Cells
inferred← citeshttps://doi.org/10.1007/s42399-026-02610-6openalex-snapshot
The Redox Imbalance in Cancer: Targeting Glutathione and Thioredoxin Systems to Enhance Chemotherapy Response
inferred← citeshttps://doi.org/10.1007/s43440-026-00857-3openalex-snapshot
Redox rewiring in glioblastoma: the thioredoxin system as a precision therapeutic target
inferred← citeshttps://doi.org/10.1016/b978-0-12-802168-2.00038-5openalex-snapshot
Selenium and Cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-802733-2.00022-0openalex-snapshot
Metabolic Shunt Pathways, Carcinoma, and mTOR
inferred← citeshttps://doi.org/10.1016/b978-0-12-812373-7.00009-7openalex-snapshot
Role of Catechins in Chemosensitization
inferred← citeshttps://doi.org/10.1016/b978-0-12-814625-5.00009-1openalex-snapshot
Bioactive Peptides—Impact in Cancer Therapy
inferred← citeshttps://doi.org/10.1016/b978-0-12-818606-0.00030-4openalex-snapshot
Reactive oxygen species and cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-818606-0.00031-6openalex-snapshot
Perspectives of TrxR1-based cancer therapies
inferred← citeshttps://doi.org/10.1016/b978-0-12-819096-8.00071-9openalex-snapshot
Beneficial and detrimental effects of antioxidants in cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-819547-5.00004-3openalex-snapshot
Endogenous antioxidants in the prognosis and treatment of lung cancer
inferred← citeshttps://doi.org/10.1016/b978-0-323-90219-9.00009-1openalex-snapshot
Thioredoxin and glutathione reductases
inferred← citeshttps://doi.org/10.1016/b978-0-323-90905-1.00003-1openalex-snapshot
Oxidative stress and its biological significance
inferred← citeshttps://doi.org/10.1016/b978-0-443-33459-7.00006-5openalex-snapshot
Oxidative stress and cancer metabolism: From pathogenesis to therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/bs.acr.2017.07.005openalex-snapshot
Targeting the Selenoprotein Thioredoxin Reductase 1 for Anticancer Therapy
inferred← citeshttps://doi.org/10.1016/bs.acr.2018.11.001openalex-snapshot
Hypoxia-inducible factors promote breast cancer stem cell specification and maintenance in response to hypoxia or cytotoxic chemotherapy
inferred← citeshttps://doi.org/10.1016/bs.acr.2021.01.002openalex-snapshot
Autophagy and senescence in cancer therapy
inferred← citeshttps://doi.org/10.1016/bs.acr.2024.04.001openalex-snapshot
Unresolved questions regarding cellular cysteine sources and their possible relationships to ferroptosis
inferred← citeshttps://doi.org/10.1016/bs.ircmb.2019.05.001openalex-snapshot
Sulfur metabolism and its contribution to malignancy
inferred← citeshttps://doi.org/10.1016/j.abb.2020.108438openalex-snapshot
Role of amino acids in regulation of ROS balance in cancer
inferred← citeshttps://doi.org/10.1016/j.aca.2026.345120openalex-snapshot
A peptide-functionalized quantum Dots/MOF nanosheets fluorescence biosensor for glutathione sensing and cellular imaging
inferred← citeshttps://doi.org/10.1016/j.addr.2016.02.002openalex-snapshot
Cancer stem cells: Radioresistance, prediction of radiotherapy outcome and specific targets for combined treatments
inferred← citeshttps://doi.org/10.1016/j.addr.2026.115884openalex-snapshot
Nanoscale metal-organic frameworks regulate redox homeostasis for Cancer therapy
inferred← citeshttps://doi.org/10.1016/j.addr.2026.115942openalex-snapshot
Redox-responsive nanomaterials for mRNA delivery: From rational design to therapeutic applications
inferred← citeshttps://doi.org/10.1016/j.addr.2026.115956openalex-snapshot
Coupling redox regulation with Cancer immunotherapy: The challenge of polyphenol Nanocarriers
inferred← citeshttps://doi.org/10.1016/j.arabjc.2023.105373openalex-snapshot
Geraniol inhibits cell growth and promotes caspase-dependent apoptosis in nasopharyngeal cancer C666-1 cells via inhibiting PI3K/Akt/mTOR signaling pathway
inferred← citeshttps://doi.org/10.1016/j.archger.2025.105877openalex-snapshot
Exploring the association between aging, ferroptosis, and common age-related diseases
inferred← citeshttps://doi.org/10.1016/j.asjsur.2022.02.005openalex-snapshot
Glutamate-cysteine ligase catalytic and its modifier function as novel immunotargets in gastric adenocarcinoma
inferred← citeshttps://doi.org/10.1016/j.bbadis.2020.165721openalex-snapshot
Metabolic reprogramming and disease progression in cancer patients
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2016.04.026openalex-snapshot
Targeting succinate:ubiquinone reductase potentiates the efficacy of anticancer therapy
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2025.120100openalex-snapshot
Cationic liposome-based silencing of thioredoxin reductase-1 for sensitization of MDA-MB 231 cells to gamma radiation
inferred← citeshttps://doi.org/10.1016/j.bbcan.2016.02.006openalex-snapshot
Mitochondrial dysfunction in DDR-related cancer predisposition syndromes
inferred← citeshttps://doi.org/10.1016/j.bbcan.2021.188559openalex-snapshot
Metabolic reprogramming in renal cancer: Events of a metabolic disease
inferred← citeshttps://doi.org/10.1016/j.bbcan.2023.189023openalex-snapshot
Metabolic reprogramming in nasopharyngeal carcinoma: Mechanisms and therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/j.bbrc.2019.11.078openalex-snapshot
Efficacy of glutathione inhibitors for the treatment of ARID1A-deficient diffuse-type gastric cancers
inferred← citeshttps://doi.org/10.1016/j.bcp.2021.114818openalex-snapshot
Molecular alterations that precede the establishment of the hallmarks of cancer: An approach on the prevention of hepatocarcinogenesis
inferred← citeshttps://doi.org/10.1016/j.bcp.2025.116763openalex-snapshot
Cotargeting of thioredoxin 1 and glutamate-cysteine ligase in both imatinib-sensitive and imatinib-resistant CML cells
inferred← citeshttps://doi.org/10.1016/j.bcp.2026.118376openalex-snapshot
PHGDH: metabolic enzyme or signaling hub? From broad-spectrum inhibition toward context-adapted therapeutic targeting
inferred← citeshttps://doi.org/10.1016/j.biocel.2022.106346openalex-snapshot
Free radicals: Relationship to Human Diseases and Potential Therapeutic applications
inferred← citeshttps://doi.org/10.1016/j.biochi.2022.01.017openalex-snapshot
The unfolded protein response and the biology of uveal melanoma
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2019.119500openalex-snapshot
Nanotherapeutics interfere with cellular redox homeostasis for highly improved photodynamic therapy
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2020.120554openalex-snapshot
Effect of Cellular and ECM Aging on Human iPSC-derived Cardiomyocyte Performance, Maturity and Senescence
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2021.121110openalex-snapshot
Application of glutathione depletion in cancer therapy: Enhanced ROS-based therapy, ferroptosis, and chemotherapy
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2022.121565openalex-snapshot
Advanced nanomedicines for the regulation of cancer metabolism
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2024.122673openalex-snapshot
Hydroxyethyl starch-based self-reinforced nanomedicine inhibits both glutathione and thioredoxin antioxidant pathways to boost reactive oxygen species-powered immunotherapy
inferred← citeshttps://doi.org/10.1016/j.bioorg.2022.105711openalex-snapshot
Rheumatoid arthritis drug sinomenine induces apoptosis of cervical tumor cells by targeting thioredoxin reductase in vitro and in vivo
inferred← citeshttps://doi.org/10.1016/j.bioorg.2025.109147openalex-snapshot
Disruption of cellular redox homeostasis by bigelovin triggers oxidative stress-mediated apoptosis in Fibrosarcoma
inferred← citeshttps://doi.org/10.1016/j.biopha.2019.109532openalex-snapshot
Dual roles of glutathione S-transferase mu 1 in the development and metastasis of hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1016/j.biopha.2020.110037openalex-snapshot
The function of miR-200 family in oxidative stress response evoked in cancer chemotherapy and radiotherapy
inferred← citeshttps://doi.org/10.1016/j.biopha.2020.110710openalex-snapshot
Emerging mechanisms and applications of ferroptosis in the treatment of resistant cancers
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.116711openalex-snapshot
NADPH and NAC synergistically inhibits chronic ocular hypertension-induced neurodegeneration and neuroinflammation through regulating p38/MAPK pathway and peroxidation
inferred← citeshttps://doi.org/10.1016/j.biopha.2026.119327openalex-snapshot
A metabolomics and lipidomics atlas of pulmonary large cell neuroendocrine carcinoma
inferred← citeshttps://doi.org/10.1016/j.biotechadv.2018.01.007openalex-snapshot
Targeting of stress response pathways in the prevention and treatment of cancer
inferred← citeshttps://doi.org/10.1016/j.bj.2015.10.002openalex-snapshot
Metabolic, autophagic, and mitophagic activities in cancer initiation and progression
inferred← citeshttps://doi.org/10.1016/j.bj.2017.06.002openalex-snapshot
Thioredoxin promotes survival signaling events under nitrosative/oxidative stress associated with cancer development
inferred← citeshttps://doi.org/10.1016/j.canlet.2015.07.004openalex-snapshot
Polyphenols as mitochondria-targeted anticancer drugs
inferred← citeshttps://doi.org/10.1016/j.canlet.2016.02.058openalex-snapshot
Piperlongumine as a direct TrxR1 inhibitor with suppressive activity against gastric cancer
inferred← citeshttps://doi.org/10.1016/j.canlet.2017.10.014openalex-snapshot
Survival of the fittest: Cancer challenges T cell metabolism
inferred← citeshttps://doi.org/10.1016/j.canlet.2019.11.037openalex-snapshot
Harnessing altered oxidative metabolism in cancer by augmented prooxidant therapy
inferred← citeshttps://doi.org/10.1016/j.canlet.2021.03.030openalex-snapshot
Jolkinolide B targets thioredoxin and glutathione systems to induce ROS-mediated paraptosis and apoptosis in bladder cancer cells
inferred← citeshttps://doi.org/10.1016/j.canlet.2024.217429openalex-snapshot
Sulforaphane potentiates the efficacy of chemoradiotherapy in glioblastoma by selectively targeting thioredoxin reductase 1
inferred← citeshttps://doi.org/10.1016/j.cbi.2017.05.007openalex-snapshot
Superoxide-hydrogen peroxide genetic imbalance modulates differentially the oxidative metabolism on human peripheral blood mononuclear cells exposed to seleno-L-methionine
inferred← citeshttps://doi.org/10.1016/j.cca.2018.10.004openalex-snapshot
Challenges facing nanotoxicology and nanomedicine due to cellular diversity
inferred← citeshttps://doi.org/10.1016/j.ccell.2015.01.007openalex-snapshot
Reactive Oxygen Species in Cancer: A Dance with the Devil
inferred← citeshttps://doi.org/10.1016/j.ccell.2018.05.001openalex-snapshot
Cancer Cells Co-opt the Neuronal Redox-Sensing Channel TRPA1 to Promote Oxidative-Stress Tolerance
inferred← citeshttps://doi.org/10.1016/j.ccell.2018.12.009openalex-snapshot
Targeting the Vulnerability of Glutathione Metabolism in ARID1A-Deficient Cancers
inferred← citeshttps://doi.org/10.1016/j.ccell.2019.01.017openalex-snapshot
Glutathione Metabolism: An Achilles’ Heel of ARID1A-Deficient Tumors
inferred← citeshttps://doi.org/10.1016/j.ccell.2019.04.002openalex-snapshot
Targeting Ferroptosis to Iron Out Cancer
inferred← citeshttps://doi.org/10.1016/j.ccell.2020.06.001openalex-snapshot
Oxidative Stress in Cancer
inferred← citeshttps://doi.org/10.1016/j.ccr.2025.217519openalex-snapshot
Cuproptosis and its cooperative role in cancer therapy: exploring new treatment strategies
inferred← citeshttps://doi.org/10.1016/j.ceca.2017.01.007openalex-snapshot
Crosstalk between calcium and reactive oxygen species signaling in cancer
inferred← citeshttps://doi.org/10.1016/j.cej.2021.128880openalex-snapshot
Glutathione-mediated nanomedicines for cancer diagnosis and therapy
inferred← citeshttps://doi.org/10.1016/j.cej.2022.137110openalex-snapshot
The composites of triple-helix glucan nanotubes/selenium nanoparticles target hepatocellular carcinoma to enhance ferroptosis by depleting glutathione and augmenting redox imbalance
inferred← citeshttps://doi.org/10.1016/j.cej.2026.177768openalex-snapshot
A sequential cascaded cGAS–STING amplifier based on hybrid multienzymes for efficient catalytic immunotherapy of hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1016/j.cell.2016.12.039openalex-snapshot
Understanding the Intersections between Metabolism and Cancer Biology
inferred← citeshttps://doi.org/10.1016/j.cell.2019.07.031openalex-snapshot
The Oncogenic Action of NRF2 Depends on De-glycation by Fructosamine-3-Kinase
inferred← citeshttps://doi.org/10.1016/j.celrep.2016.12.061openalex-snapshot
Targeted Inhibition of EGFR and Glutaminase Induces Metabolic Crisis in EGFR Mutant Lung Cancer
inferred← citeshttps://doi.org/10.1016/j.celrep.2017.06.066openalex-snapshot
A Positive Feedback Loop between Sestrin2 and mTORC2 Is Required for the Survival of Glutamine-Depleted Lung Cancer Cells
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