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Dendritic Cell Amiloride Sensitive Channels Mediate Sodium-induced Inflammation and Hypertension
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Induction, function and regulation of IL‐17‐producing T cells
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Hyperosmotic Stress as a Stimulant for Proinflammatory Cytokine Production
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Protein kinase C phosphorylates a component of NADPH oxidase of neutrophils
inferredcites →https://doi.org/10.1016/0735-1097(93)90743-kopenalex-snapshot
Role of sodium/calcium exchange in the mechanism of myocardial stunning: Protective effect of reperfusion with high sodium solution
inferredcites →https://doi.org/10.1016/j.cmet.2015.02.003openalex-snapshot
Cutaneous Na+ Storage Strengthens the Antimicrobial Barrier Function of the Skin and Boosts Macrophage-Driven Host Defense
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Interleukin-1 Receptor Activation Potentiates Salt Reabsorption in Angiotensin II-Induced Hypertension via the NKCC2 Co-transporter in the Nephron
inferredcites →https://doi.org/10.1016/j.expneurol.2016.03.010openalex-snapshot
High salt drives Th17 responses in experimental autoimmune encephalomyelitis without impacting myeloid dendritic cells
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2004.02.014openalex-snapshot
Localization of isoketal adducts in vivo using a single-chain antibody
inferredcites →https://doi.org/10.1016/j.immuni.2013.08.007openalex-snapshot
Minimal Differentiation of Classical Monocytes as They Survey Steady-State Tissues and Transport Antigen to Lymph Nodes
inferredcites →https://doi.org/10.1016/j.trsl.2014.11.007openalex-snapshot
Effects of dietary salt levels on monocytic cells and immune responses in healthy human subjects: a longitudinal study
inferredcites →https://doi.org/10.1016/j.yjmcc.2011.03.005openalex-snapshot
Vascular smooth muscle Jak2 deletion prevents angiotensin II-mediated neointima formation following injury in mice
inferredcites →https://doi.org/10.1016/s0008-6363(97)00271-xopenalex-snapshot
Contribution of reverse-mode sodium–calcium exchange to contractions in failing human left ventricular myocytes
inferredcites →https://doi.org/10.1016/s0140-6736(05)17741-1openalex-snapshot
Global burden of hypertension: analysis of worldwide data
inferredcites →https://doi.org/10.1038/cr.2015.87openalex-snapshot
High salt primes a specific activation state of macrophages, M(Na)
inferredcites →https://doi.org/10.1038/nature11868openalex-snapshot
Sodium chloride drives autoimmune disease by the induction of pathogenic TH17 cells
inferredcites →https://doi.org/10.1038/nature11984openalex-snapshot
Induction of pathogenic TH17 cells by inducible salt-sensing kinase SGK1
inferredcites →https://doi.org/10.1038/ng0396-248openalex-snapshot
Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1
inferredcites →https://doi.org/10.1038/nm.1960openalex-snapshot
Macrophages regulate salt-dependent volume and blood pressure by a vascular endothelial growth factor-C–dependent buffering mechanism
inferredcites →https://doi.org/10.1042/bj2860687openalex-snapshot
Okadaic acid produces changes in phosphorylation and translocation of proteins and in intracellular calcium in human neutrophils. Relationship with the activation of the NADPH oxidase by different stimuli
inferredcites →https://doi.org/10.1056/nejmra1201534openalex-snapshot
Measuring the Global Burden of Disease
inferredcites →https://doi.org/10.1084/jem.20070657openalex-snapshot
Role of the T cell in the genesis of angiotensin II–induced hypertension and vascular dysfunction
inferredcites →https://doi.org/10.1089/ars.2012.4918openalex-snapshot
Nox2-Induced Production of Mitochondrial Superoxide in Angiotensin II-Mediated Endothelial Oxidative Stress and Hypertension
inferredcites →https://doi.org/10.1093/cvr/cvr059openalex-snapshot
Vascular smooth muscle Jak2 mediates angiotensin II-induced hypertension via increased levels of reactive oxygen species
inferredcites →https://doi.org/10.1093/cvr/cvs422openalex-snapshot
Interleukin-17 causes Rho-kinase-mediated endothelial dysfunction and hypertension
inferredcites →https://doi.org/10.1097/mnh.0b013e32833aeb3bopenalex-snapshot
Sodium sensing in the interstitium and relationship to hypertension
inferredcites →https://doi.org/10.1124/pr.56.4.5openalex-snapshot
Pharmacology of Brain Na+/Ca2+ Exchanger: From Molecular Biology to Therapeutic Perspectives
inferredcites →https://doi.org/10.1152/ajpheart.00603.2016openalex-snapshot
Dendritic cells and isolevuglandins in immunity, inflammation, and hypertension
inferredcites →https://doi.org/10.1152/ajpheart.01237.2003openalex-snapshot
Glycosaminoglycan polymerization may enable osmotically inactive Na+storage in the skin
inferredcites →https://doi.org/10.1152/ajpregu.00304.2012openalex-snapshot
Genetic mutation of recombination activating gene 1 in Dahl salt-sensitive rats attenuates hypertension and renal damage
inferredcites →https://doi.org/10.1152/ajprenal.2001.280.4.f675openalex-snapshot
Aldosterone induces rapid apical translocation of ENaC in early portion of renal collecting system: possible role of SGK
inferredcites →https://doi.org/10.1161/01.res.0000187456.06162.cbopenalex-snapshot
Cardiac-Specific Ablation of the Na + -Ca 2+ Exchanger Confers Protection Against Ischemia/Reperfusion Injury
inferredcites →https://doi.org/10.1161/01.res.82.12.1279openalex-snapshot
Phosphorylation of p130Casby Angiotensin II Is Dependent on c-Src, Intracellular Ca2+, and Protein Kinase C
inferredcites →https://doi.org/10.1161/cir.0000000000000485openalex-snapshot
Heart Disease and Stroke Statistics—2017 Update: A Report From the American Heart Association
inferredcites →https://doi.org/10.1161/cir.0000000000000530openalex-snapshot
Correction to: Heart Disease and Stroke Statistics—2017 Update: A Report From the American Heart Association
inferredcites →https://doi.org/10.1161/circresaha.115.308111openalex-snapshot
CD70 Exacerbates Blood Pressure Elevation and Renal Damage in Response to Repeated Hypertensive Stimuli
inferredcites →https://doi.org/10.1161/circresaha.116.303697openalex-snapshot
Inflammation, Immunity, and Hypertensive End-Organ Damage
inferredcites →https://doi.org/10.1161/circulationaha.109.930446openalex-snapshot
Inhibition and Genetic Ablation of the B7/CD28 T-Cell Costimulation Axis Prevents Experimental Hypertension
inferredcites →https://doi.org/10.1161/hc4001.097038openalex-snapshot
Gender Influences [Ca2+]iDuring Metabolic Inhibition in Myocytes Overexpressing the Na+-Ca2+Exchanger
inferredcites →https://doi.org/10.1161/hypertensionaha.109.145094openalex-snapshot
Interleukin 17 Promotes Angiotensin II–Induced Hypertension and Vascular Dysfunction
inferredcites →https://doi.org/10.1161/hypertensionaha.110.162917openalex-snapshot
Fk506 Binding Protein 12 Deficiency in Endothelial and Hematopoietic Cells Decreases Regulatory T Cells and Causes Hypertension
inferredcites →https://doi.org/10.1161/hypertensionaha.111.00566openalex-snapshot
23 Na Magnetic Resonance Imaging-Determined Tissue Sodium in Healthy Subjects and Hypertensive Patients
inferredcites →https://doi.org/10.1161/hypertensionaha.113.00689openalex-snapshot
Immunosenescent CD8 + T Cells and C-X-C Chemokine Receptor Type 3 Chemokines Are Increased in Human Hypertension
inferredcites →https://doi.org/10.1161/hypertensionaha.114.04975openalex-snapshot
Renal Transporter Activation During Angiotensin-II Hypertension is Blunted in Interferon-γ −/− and Interleukin-17A −/− Mice
inferredcites →https://doi.org/10.1172/jci16956openalex-snapshot
Collecting duct–specific gene inactivation of αENaC in the mouse kidney does not impair sodium and potassium balance
inferredcites →https://doi.org/10.1172/jci6821openalex-snapshot
Functional expression of a pseudohypoaldosteronism type I mutated epithelial Na+ channel lacking the pore-forming region of its α subunit
inferredcites →https://doi.org/10.1172/jci74084openalex-snapshot
DC isoketal-modified proteins activate T cells and promote hypertension
inferredcites →https://doi.org/10.1172/jci80919openalex-snapshot
High salt reduces the activation of IL-4– and IL-13–stimulated macrophages
inferredcites →https://doi.org/10.3109/10715762.2010.540242openalex-snapshot
EPR detection of cellular and mitochondrial superoxide using cyclic hydroxylamines
inferredcites →https://doi.org/10.5049/ebp.2014.12.1.7openalex-snapshot
Dietary Salt Intake and Hypertension
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Expression Characteristics of Immune Cells and Related Molecules in Hypertension Patients
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Animal models of hypertension and concurrent organs injury
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Innate Immune Cells and Hypertension: Neutrophils and Neutrophil Extracellular Traps (NETs)
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Actions of Dendritic Cells in the Kidney during Hypertension
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Epithelial Na+Channels Function as Extracellular Sensors
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Innate Immune Cells and Hypertension: Neutrophils and Neutrophil Extracellular Traps (NETs)
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Actions of Dendritic Cells in the Kidney during Hypertension
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Epithelial Na + Channels Function as Extracellular Sensors
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Epithelial Sodium Channels (ENaCs)
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Endoplasmic Reticulum Stress in Hypertension and Salt Sensitivity of Blood Pressure
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Inflammation in Chronic Kidney Disease
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Na+i/K+i imbalance contributes to gene expression in endothelial cells exposed to elevated NaCl
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Immune and inflammatory mechanisms in hypertension
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Renal NOXA1/NOX1 Signaling Regulates Epithelial Sodium Channel and Sodium Retention in Angiotensin II-induced Hypertension
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inferred← citeshttps://doi.org/10.1093/ajh/hpaa145openalex-snapshot
Innate Immune Mechanisms of Arterial Hypertension and Autoimmune Disease
inferred← citeshttps://doi.org/10.1093/ajh/hpac077openalex-snapshot
Update on Immune Mechanisms in Hypertension
inferred← citeshttps://doi.org/10.1093/ajh/hpae006openalex-snapshot
The Pathogenesis and Impact of Arterial Stiffening in Hypertension: The 2023 John H. Laragh Research Award
inferred← citeshttps://doi.org/10.1093/ajh/hpaf019openalex-snapshot
Plasmacytoid Dendritic Cell Content Is Associated With Plasma Aldosterone Concentration in Patients With Primary Aldosteronism
inferred← citeshttps://doi.org/10.1093/ajh/hpz139openalex-snapshot
Augmenting Renal Lymphatic Density Prevents Angiotensin II-Induced Hypertension in Male and Female Mice
inferred← citeshttps://doi.org/10.1093/braincomms/fcae147openalex-snapshot
High-salt diet induces microbiome dysregulation, neuroinflammation and anxiety in the chronic period after mild repetitive closed head injury in adolescent mice
inferred← citeshttps://doi.org/10.1093/ckj/sfad059openalex-snapshot
A new immune disease: systemic hypertension
inferred← citeshttps://doi.org/10.1093/cvr/cvaa207openalex-snapshot
Sodium activates human monocytes via the NADPH oxidase and isolevuglandin formation
inferred← citeshttps://doi.org/10.1093/cvr/cvaa326openalex-snapshot
Role of the vascular endothelial sodium channel activation in the genesis of pathologically increased cardiovascular stiffness
inferred← citeshttps://doi.org/10.1093/cvr/cvac028openalex-snapshot
Hypersensitive or hypertensive? IgE–FcɛR1 signalling in mast cells adds a new piece to the immunity and hypertension puzzle
inferred← citeshttps://doi.org/10.1093/cvr/cvae229openalex-snapshot
Dietary salt, vascular dysfunction, and cognitive impairment
inferred← citeshttps://doi.org/10.1093/cvr/cvy112openalex-snapshot
Hypertension and increased endothelial mechanical stretch promote monocyte differentiation and activation: roles of STAT3, interleukin 6 and hydrogen peroxide
inferred← citeshttps://doi.org/10.1093/eurheartj/ehae670openalex-snapshot
Dendritic cell mineralocorticoid receptor controls blood pressure by regulating T helper 17 differentiation: role of the Plcβ1/4–Stat5–NF-κB pathway
inferred← citeshttps://doi.org/10.1093/ibd/izac058openalex-snapshot
Is Salt at Fault? Dietary Salt Consumption and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1093/jb/mvad009openalex-snapshot
Hyperosmotic stress response regulates interstitial homeostasis and pathogenic inflammation
inferred← citeshttps://doi.org/10.1093/ndt/gfy362openalex-snapshot
Emerging evidence of an effect of salt on innate and adaptive immunity
inferred← citeshttps://doi.org/10.1097/cm9.0000000000004098openalex-snapshot
High-salt diet in macrophage-associated metabolic disorders: Mechanisms and therapeutic implications
inferred← citeshttps://doi.org/10.1097/crd.0000000000000834openalex-snapshot
Salt Sensitivity of Blood Pressure and the Role of the Immune System in Hypertension
inferred← citeshttps://doi.org/10.1097/hjh.0000000000003829openalex-snapshot
Recent updates on the influence of iron and magnesium on vascular, renal, and adipose inflammation and possible consequences for hypertension
inferred← citeshttps://doi.org/10.1097/hjh.0000000000004440openalex-snapshot
The role of the immune system in hypertension
inferred← citeshttps://doi.org/10.1097/mnh.0000000000000479openalex-snapshot
New insights regarding epithelial Na+ channel regulation and its role in the kidney, immune system and vasculature
inferred← citeshttps://doi.org/10.1097/mnh.0000000000000635openalex-snapshot
Actions of immune cells in the hypertensive kidney
inferred← citeshttps://doi.org/10.1097/mnh.0000000000000862openalex-snapshot
Targeting inflammation in hypertension
inferred← citeshttps://doi.org/10.1097/mnh.0000000000000963openalex-snapshot
Dendritic cell epithelial sodium channel induced inflammation and salt-sensitive hypertension
inferred← citeshttps://doi.org/10.1097/mnh.0000000000000966openalex-snapshot
Interferon gamma in the pathogenesis of hypertension − recent insights
inferred← citeshttps://doi.org/10.1101/2024.06.10.598374openalex-snapshot
Myeloid Cell Glucocorticoid, Not Mineralocorticoid Receptor Signaling, Contributes to Salt-Sensitive Hypertension in Humans via Cortisol
inferred← citeshttps://doi.org/10.1101/cshperspect.a041173openalex-snapshot
Lymphatics in Cardiovascular Physiology
inferred← citeshttps://doi.org/10.1111/apha.70308openalex-snapshot
Renal Myeloid Mechanisms, Cross‐Talk in Hypertension
inferred← citeshttps://doi.org/10.1111/bph.14359openalex-snapshot
Salt, inflammation, IL‐17 and hypertension
inferred← citeshttps://doi.org/10.1111/bph.14427openalex-snapshot
Role of immune cells in hypertension
inferred← citeshttps://doi.org/10.1111/bph.14659openalex-snapshot
Immunity and hypertension: New targets to lighten the pressure
inferred← citeshttps://doi.org/10.1111/cpr.13250openalex-snapshot
The modulatory effect of high salt on immune cells and related diseases
inferred← citeshttps://doi.org/10.1111/dom.14980openalex-snapshot
The role of dietary salt in metabolism and energy balance: Insights beyond cardiovascular disease
inferred← citeshttps://doi.org/10.1111/febs.16155openalex-snapshot
Mechanisms of sodium‐mediated injury in cardiovascular disease: old play, new scripts
inferred← citeshttps://doi.org/10.1111/imr.70093openalex-snapshot
Hypertension as an Immune Disorder: From Cellular Drivers to Molecular Mediators
inferred← citeshttps://doi.org/10.1111/jch.13566openalex-snapshot
Mineralocorticoid antagonists and ENaC inhibitors in hyperaldosteronism
inferred← citeshttps://doi.org/10.1111/jch.13786openalex-snapshot
Measurement of sodium intake or measurement of the detrimental effects of sodium on health in individual subjects?
inferred← citeshttps://doi.org/10.1113/ep089553openalex-snapshot
Ovariectomy and high salt increase blood pressure and alter sodium transport proteins in peripheral blood mononuclear cells of adult Wistar rats
inferred← citeshttps://doi.org/10.1113/jp286756openalex-snapshot
Tumour necrosis factor‐alpha at the intersection of renal epithelial and immune cell function
inferred← citeshttps://doi.org/10.1142/s0192415x2550051xopenalex-snapshot
Plant-Derived Natural Products Ameliorating Hypertension via Signaling Pathways: A Review
inferred← citeshttps://doi.org/10.1146/annurev-physiol-022724-105050openalex-snapshot
Epithelial Na+ Channels, Immune Cells, and Salt
inferred← citeshttps://doi.org/10.1152/ajpheart.00096.2019openalex-snapshot
Role of T-cell activation in salt-sensitive hypertension
inferred← citeshttps://doi.org/10.1152/ajpheart.00100.2019openalex-snapshot
Adaptive immune cells in calcific aortic valve disease
inferred← citeshttps://doi.org/10.1152/ajpheart.00504.2025openalex-snapshot
Epithelial sodium channels: contributions to the regulation of vascular function and blood pressure
inferred← citeshttps://doi.org/10.1152/ajpregu.00207.2022openalex-snapshot
Epithelial sodium channels in macrophage migration and polarization: role of proinflammatory cytokines TNFα and IFNγ
inferred← citeshttps://doi.org/10.1152/ajprenal.00268.2023openalex-snapshot
Amiloride lowers plasma TNF and interleukin-6 but not interleukin-17A in patients with hypertension and type 2 diabetes
inferred← citeshttps://doi.org/10.1152/ajprenal.00309.2024openalex-snapshot
Recent advances on immunity and hypertension: the new cells on the kidney block
inferred← citeshttps://doi.org/10.1152/ajprenal.00369.2021openalex-snapshot
Splenocyte transfer from hypertensive donors eliminates premenopausal female protection from ANG II-induced hypertension
inferred← citeshttps://doi.org/10.1155/2021/3178796openalex-snapshot
The Potential of Mesenchymal Stem Cells for the Treatment of Cytokine Storm due to COVID‐19
inferred← citeshttps://doi.org/10.1159/000502011openalex-snapshot
Salt and Water Retention Is Associated with Microinflammation and Endothelial Injury in Chronic Kidney Disease
inferred← citeshttps://doi.org/10.1161/circresaha.119.315343openalex-snapshot
A20 in Myeloid Cells Protects Against Hypertension by Inhibiting Dendritic Cell-Mediated T-Cell Activation
inferred← citeshttps://doi.org/10.1161/circresaha.121.318063openalex-snapshot
Oxidative Stress and Hypertension
inferred← citeshttps://doi.org/10.1161/circresaha.121.318082openalex-snapshot
Pathophysiology of Hypertension
inferred← citeshttps://doi.org/10.1161/circresaha.121.319643openalex-snapshot
Growth Arrest Specific-6 and Axl Coordinate Inflammation and Hypertension
inferred← citeshttps://doi.org/10.1161/circresaha.122.320818openalex-snapshot
DC ENaC-Dependent Inflammasome Activation Contributes to Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/circresaha.123.322982openalex-snapshot
Salt Sensitivity of Blood Pressure
inferred← citeshttps://doi.org/10.1161/circresaha.124.323595openalex-snapshot
Myeloid-Specific JAK2 Contributes to Inflammation and Salt Sensitivity of Blood Pressure
inferred← citeshttps://doi.org/10.1161/circresaha.124.323979openalex-snapshot
HIV-Associated Hypertension: Risks, Mechanisms, and Knowledge Gaps
inferred← citeshttps://doi.org/10.1161/circresaha.124.324890openalex-snapshot
ET-3/ETBR Mediates Na + -Activated Immune Signaling and Kidney Lymphatic Dynamics
inferred← citeshttps://doi.org/10.1161/circresaha.124.325439openalex-snapshot
Epigenetic Regulation of Innate and Adaptive Immune Cells in Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/circresaha.125.327976openalex-snapshot
Myeloid Activator Protein-1 Complex Contributes to Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/hyp.0000000000000247openalex-snapshot
Hypertension and the Gut Microbiome: A Science Advisory From the American Heart Association
inferred← citeshttps://doi.org/10.1161/hypertensionaha.118.10211openalex-snapshot
Sodium Handling by the Blood Vessel Wall
inferred← citeshttps://doi.org/10.1161/hypertensionaha.118.11128openalex-snapshot
Salt Intake and Immunity
inferred← citeshttps://doi.org/10.1161/hypertensionaha.118.11339openalex-snapshot
Epithelial Sodium Channel in Aldosterone-Induced Endothelium Stiffness and Aortic Dysfunction
inferred← citeshttps://doi.org/10.1161/hypertensionaha.118.12330openalex-snapshot
Multifaceted Functions of Epithelial Na + Channel in Modulating Blood Pressure
inferred← citeshttps://doi.org/10.1161/hypertensionaha.119.12761openalex-snapshot
High Salt Activates CD11c + Antigen-Presenting Cells via SGK (Serum Glucocorticoid Kinase) 1 to Promote Renal Inflammation and Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/hypertensionaha.119.13667openalex-snapshot
Classical Dendritic Cells Mediate Hypertension by Promoting Renal Oxidative Stress and Fluid Retention
inferred← citeshttps://doi.org/10.1161/hypertensionaha.119.13850openalex-snapshot
Central EP3 (E Prostanoid 3) Receptors Mediate Salt-Sensitive Hypertension and Immune Activation
inferred← citeshttps://doi.org/10.1161/hypertensionaha.119.13861openalex-snapshot
Crosstalk Between Vascular Redox and Calcium Signaling in Hypertension Involves TRPM2 (Transient Receptor Potential Melastatin 2) Cation Channel
inferred← citeshttps://doi.org/10.1161/hypertensionaha.120.14481openalex-snapshot
Epithelial Sodium Channel and Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/hypertensionaha.120.16518openalex-snapshot
EP3 (E-Prostanoid 3) Receptor Mediates Impaired Vasodilation in a Mouse Model of Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/hypertensionaha.121.16495openalex-snapshot
Immune Mechanisms of Dietary Salt-Induced Hypertension and Kidney Disease: Harry Goldblatt Award for Early Career Investigators 2020
inferred← citeshttps://doi.org/10.1161/hypertensionaha.121.17955openalex-snapshot
Immune System and Microvascular Remodeling in Humans
inferred← citeshttps://doi.org/10.1161/hypertensionaha.121.18513openalex-snapshot
Rare Variants in Genes Encoding Subunits of the Epithelial Na + Channel Are Associated With Blood Pressure and Kidney Function
inferred← citeshttps://doi.org/10.1161/hypertensionaha.121.18734openalex-snapshot
Type 1 Angiotensin Receptors on CD11c-Expressing Cells Protect Against Hypertension by Regulating Dendritic Cell–Mediated T Cell Activation
inferred← citeshttps://doi.org/10.1161/hypertensionaha.122.18554openalex-snapshot
The Immune System in Hypertension: a Lost Shaker of Salt 2021 Lewis K. Dahl Memorial Lecture
inferred← citeshttps://doi.org/10.1161/hypertensionaha.123.17959openalex-snapshot
Salt Sensitivity: Causes, Consequences, and Recent Advances
inferred← citeshttps://doi.org/10.1161/hypertensionaha.123.19489openalex-snapshot
Sodium as an Important Regulator of Immunometabolism
inferred← citeshttps://doi.org/10.1161/hypertensionaha.123.21285openalex-snapshot
Eicosanoid-Regulated Myeloid ENaC and Isolevuglandin Formation in Human Salt-Sensitive Hypertension
inferred← citeshttps://doi.org/10.1161/hypertensionaha.123.21287openalex-snapshot
Aldosterone Antagonism Is More Effective at Reducing Blood Pressure and Excessive Renal ENaC Activity in AngII-Infused Female Rats Than in Males
inferred← citeshttps://doi.org/10.1161/hypertensionaha.123.22031openalex-snapshot
Recent Advances in Understanding Peripheral and Gut Immune Cell-Mediated Salt-Sensitive Hypertension and Nephropathy
inferred← citeshttps://doi.org/10.1161/hypertensionaha.124.21355openalex-snapshot
Immune Mechanisms in Hypertension
inferred← citeshttps://doi.org/10.1161/hypertensionaha.125.17950openalex-snapshot
Food as Medicine for Hypertension: Microbiota as Mediators
inferred← citeshttps://doi.org/10.1161/jaha.118.011450openalex-snapshot
Elevated Eosinophils as a Feature of Inflammation Associated With Hypertension in Virally Suppressed People Living With HIV
inferred← citeshttps://doi.org/10.1161/jaha.125.045304openalex-snapshot
High‐Salt Diet–Induced Endothelial Dysfunction Is Mediated by Cellular Senescence
inferred← citeshttps://doi.org/10.1172/jci.insight.126241openalex-snapshot
High dietary salt–induced DC activation underlies microbial dysbiosis-associated hypertension
inferred← citeshttps://doi.org/10.1172/jci.insight.136678openalex-snapshot
Isolevuglandins disrupt PU.1-mediated C1q expression and promote autoimmunity and hypertension in systemic lupus erythematosus
inferred← citeshttps://doi.org/10.1172/jci.insight.156437openalex-snapshot
Dissociation of sodium-chloride cotransporter expression and blood pressure during chronic high dietary potassium supplementation
inferred← citeshttps://doi.org/10.1172/jci.insight.161878openalex-snapshot
Dicarbonyl-modified lipoproteins contribute to proteinuric kidney injury
inferred← citeshttps://doi.org/10.1172/jci.insight.163757openalex-snapshot
Isolevuglandins disrupt PU.1-mediated C1q expression and promote autoimmunity and hypertension in systemic lupus erythematosus
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