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Axon degeneration: context defines distinct pathways

https://doi.org/10.1016/j.conb.2016.05.002

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2 of the 62 references listed on this page are in the Retraction Watch database as retracted (export 2026-10-21): 10.1038/nature07767 (notice 2023-12-18), 10.1074/jbc.m114.611574 (notice 2016-10-28).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1006/dbio.1994.1234openalex-snapshot

Neurites Can Remain Viable after Destruction of the Neuronal Soma by Programmed Cell Death (Apoptosis)

inferredcites →https://doi.org/10.1007/bf01195555openalex-snapshot

Prolonged survival of transected nerve fibres in C57BL/Ola mice is an intrinsic characteristic of the axon

inferredcites →https://doi.org/10.1016/0006-8993(89)91197-9openalex-snapshot

Death of sensory ganglion neurons after acute withdrawal of nerve growth factor in dissociated cell cultures

inferredcites →https://doi.org/10.1016/j.cell.2014.11.053openalex-snapshot

Pathological Axonal Death through a MAPK Cascade that Triggers a Local Energy Deficit

inferredcites →https://doi.org/10.1016/j.cell.2016.01.032openalex-snapshot

Axon Degeneration Gated by Retrograde Activation of Somatic Pro-apoptotic Signaling

inferredcites →https://doi.org/10.1016/j.celrep.2013.04.013openalex-snapshot

The Phr1 Ubiquitin Ligase Promotes Injury-Induced Axon Self-Destruction

inferredcites →https://doi.org/10.1016/j.celrep.2013.07.015openalex-snapshot

XIAP Regulates Caspase Activity in Degenerating Axons

inferredcites →https://doi.org/10.1016/j.celrep.2015.02.060openalex-snapshot

Absence of SARM1 Rescues Development and Survival of NMNAT2-Deficient Axons

inferredcites →https://doi.org/10.1016/j.celrep.2015.11.032openalex-snapshot

Wallerian Degeneration Is Executed by an NMN-SARM1-Dependent Late Ca2+ Influx but Only Modestly Influenced by Mitochondria

inferredcites →https://doi.org/10.1016/j.conb.2013.06.007openalex-snapshot

Neurite pruning and neuronal cell death: spatial regulation of shared destruction programs

inferredcites →https://doi.org/10.1016/j.nbd.2013.03.009openalex-snapshot

Calpains mediate axonal cytoskeleton disintegration during Wallerian degeneration

inferredcites →https://doi.org/10.1016/j.neuron.2006.07.014openalex-snapshot

Identification of E2/E3 Ubiquitinating Enzymes and Caspase Activity Regulating Drosophila Sensory Neuron Dendrite Pruning

inferredcites →https://doi.org/10.1016/j.neuron.2013.08.034openalex-snapshot

Regulation of Axon Degeneration after Injury and in Development by the Endogenous Calpain Inhibitor Calpastatin

inferredcites →https://doi.org/10.1016/j.neuron.2015.08.029openalex-snapshot

New Views on the Misconstrued: Executioner Caspases and Their Diverse Non-apoptotic Roles

inferredcites →https://doi.org/10.1016/j.neuron.2015.12.023openalex-snapshot

Axon Self-Destruction: New Links among SARM1, MAPKs, and NAD+ Metabolism

inferredcites →https://doi.org/10.1016/j.tcb.2014.04.003openalex-snapshot

Diverse cellular and molecular modes of axon degeneration

inferredcites →https://doi.org/10.1016/j.tibs.2011.08.009openalex-snapshot

BAX unleashed: the biochemical transformation of an inactive cytosolic monomer into a toxic mitochondrial pore

inferredcites →https://doi.org/10.1016/j.tins.2014.07.007openalex-snapshot

Preserve and protect: maintaining axons within functional circuits

inferredcites →https://doi.org/10.1016/s0896-6273(00)80173-7openalex-snapshot

BAX Is Required for Neuronal Death after Trophic Factor Deprivation and during Development

inferredcites →https://doi.org/10.1016/s0896-6273(03)00429-xopenalex-snapshot

Involvement of the Ubiquitin-Proteasome System in the Early Stages of Wallerian Degeneration

inferredcites →https://doi.org/10.1038/35037739openalex-snapshot

Apoptosis in the nervous system

inferredcites →https://doi.org/10.1038/cdd.2014.164openalex-snapshot

A rise in NAD precursor nicotinamide mononucleotide (NMN) after injury promotes axon degeneration

inferredcites →https://doi.org/10.1038/cdd.2014.47openalex-snapshot

Programmed cell death during neuronal development: the sympathetic neuron model

inferredcites →https://doi.org/10.1038/cr.2009.11openalex-snapshot

A retrograde apoptotic signal originating in NGF-deprived distal axons of rat sympathetic neurons in compartmented cultures

inferredcites →https://doi.org/10.1038/nature07767openalex-snapshot

RETRACTED ARTICLE: APP binds DR6 to trigger axon pruning and neuron death via distinct caspases

inferredcites →https://doi.org/10.1038/ncomms2910openalex-snapshot

Distinct pathways mediate axon degeneration during apoptosis and axon-specific pruning

inferredcites →https://doi.org/10.1038/nmeth777openalex-snapshot

A microfluidic culture platform for CNS axonal injury, regeneration and transport

inferredcites →https://doi.org/10.1038/nn.2290openalex-snapshot

A dual leucine kinase–dependent axon self-destruction program promotes Wallerian degeneration

inferredcites →https://doi.org/10.1038/nn770openalex-snapshot

Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric gene

inferredcites →https://doi.org/10.1038/nrn3680openalex-snapshot

Wallerian degeneration: an emerging axon death pathway linking injury and disease

inferredcites →https://doi.org/10.1038/sj.cdd.4401147openalex-snapshot

The proapoptotic proteins Bax and Bak are not involved in Wallerian degeneration

inferredcites →https://doi.org/10.1038/sj.cdd.4402024openalex-snapshot

Apoptosome dependent caspase-3 activation pathway is non-redundant and necessary for apoptosis in sympathetic neurons

inferredcites →https://doi.org/10.1073/pnas.1211074110openalex-snapshot

DLK initiates a transcriptional program that couples apoptotic and regenerative responses to axonal injury

inferredcites →https://doi.org/10.1073/pnas.1216204109openalex-snapshot

SCG10 is a JNK target in the axonal degeneration pathway

inferredcites →https://doi.org/10.1073/pnas.1508337112openalex-snapshot

Local axonal protection by WldS as revealed by conditional regulation of protein stability

inferredcites →https://doi.org/10.1073/pnas.74.10.4516openalex-snapshot

Local control of neurite development by nerve growth factor

inferredcites →https://doi.org/10.1074/jbc.m114.611574openalex-snapshot

Spy1 Protein Mediates Phosphorylation and Degradation of SCG10 Protein in Axonal Degeneration

inferredcites →https://doi.org/10.1083/jcb.200307130openalex-snapshot

Critical function of endogenous XIAP in regulating caspase activation during sympathetic neuronal apoptosis

inferredcites →https://doi.org/10.1083/jcb.200504028openalex-snapshot

A local mechanism mediates NAD-dependent protection of axon degeneration

inferredcites →https://doi.org/10.1083/jcb.200708086openalex-snapshot

Chromatin modification of Apaf-1 restricts the apoptotic pathway in mature neurons

inferredcites →https://doi.org/10.1083/jcb.201103153openalex-snapshot

DLK induces developmental neuronal degeneration via selective regulation of proapoptotic JNK activity

inferredcites →https://doi.org/10.1083/jcb.201108111openalex-snapshot

Axon degeneration: Molecular mechanisms of a self-destruction pathway

inferredcites →https://doi.org/10.1111/j.1471-4159.2005.03676.xopenalex-snapshot

The BH3‐only protein Puma is both necessary and sufficient for neuronal apoptosis induced by DNA damage in sympathetic neurons

inferredcites →https://doi.org/10.1126/science.1098014openalex-snapshot

Increased Nuclear NAD Biosynthesis and SIRT1 Activation Prevent Axonal Degeneration

inferredcites →https://doi.org/10.1126/science.1223899openalex-snapshot

dSarm/Sarm1 Is Required for Activation of an Injury-Induced Axon Death Pathway

inferredcites →https://doi.org/10.1126/science.1258366openalex-snapshot

SARM1 activation triggers axon degeneration locally via NAD + destruction

inferredcites →https://doi.org/10.1128/mcb.21.14.4713-4724.2001openalex-snapshot

The MLK Family Mediates c-Jun N-Terminal Kinase Activation in Neuronal Apoptosis

inferredcites →https://doi.org/10.1146/annurev-cellbio-100913-013038openalex-snapshot

Sculpting Neural Circuits by Axon and Dendrite Pruning

inferredcites →https://doi.org/10.1146/annurev.neuro.28.061604.135632openalex-snapshot

AXON RETRACTION AND DEGENERATION IN DEVELOPMENT AND DISEASE

inferredcites →https://doi.org/10.1146/annurev.neuro.28.061604.135659openalex-snapshot

GROWTH AND SURVIVAL SIGNALS CONTROLLING SYMPATHETIC NERVOUS SYSTEM DEVELOPMENT

inferredcites →https://doi.org/10.1146/annurev.neuro.29.051605.112800openalex-snapshot

ADAPTIVE ROLES OF PROGRAMMED CELL DEATH DURING NERVOUS SYSTEM DEVELOPMENT

inferredcites →https://doi.org/10.1371/journal.pbio.1000300openalex-snapshot

Endogenous Nmnat2 Is an Essential Survival Factor for Maintenance of Healthy Axons

inferredcites →https://doi.org/10.1371/journal.pbio.1001440openalex-snapshot

The Highwire Ubiquitin Ligase Promotes Axonal Degeneration by Tuning Levels of Nmnat Protein

inferredcites →https://doi.org/10.1523/jneurosci.0922-10.2010openalex-snapshot

Axonal Degeneration Is Regulated by the Apoptotic Machinery or a NAD + -Sensitive Pathway in Insects and Mammals

inferredcites →https://doi.org/10.1523/jneurosci.1197-13.2013openalex-snapshot

Sarm1-Mediated Axon Degeneration Requires Both SAM and TIR Interactions

inferredcites →https://doi.org/10.1523/jneurosci.2039-12.2012openalex-snapshot

Spatially Coordinated Kinase Signaling Regulates Local Axon Degeneration

inferredcites →https://doi.org/10.1523/jneurosci.2320-06.2006openalex-snapshot

Stimulation of Nicotinamide Adenine Dinucleotide Biosynthetic Pathways Delays Axonal Degeneration after Axotomy

inferredcites →https://doi.org/10.1523/jneurosci.2484-15.2015openalex-snapshot

Live Imaging of Calcium Dynamics during Axon Degeneration Reveals Two Functionally Distinct Phases of Calcium Influx

inferredcites →https://doi.org/10.1523/jneurosci.2939-10.2010openalex-snapshot

Amyloid Precursor Protein Cleavage-Dependent and -Independent Axonal Degeneration Programs Share a Common Nicotinamide Mononucleotide Adenylyltransferase 1-Sensitive Pathway

inferredcites →https://doi.org/10.1523/jneurosci.3012-12.2012openalex-snapshot

A Caspase Cascade Regulating Developmental Axon Degeneration

inferredcites →https://doi.org/10.1523/jneurosci.3347-10.2011openalex-snapshot

Sensory Neuropathy Attributable to Loss of Bcl-w

inferredcites →https://doi.org/10.1523/jneurosci.3522-13.2014openalex-snapshot

Genetic Analysis Reveals that Amyloid Precursor Protein and Death Receptor 6 Function in the Same Pathway to Control Axonal Pruning Independent of β-Secretase

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Intravitreal application of AAV-BDNF or mutant AAV-CRMP2 protects retinal ganglion cells and stabilizes axons and myelin after partial optic nerve injury

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Presymptomatic macrophage targeting has a long-lasting therapeutic effect on treatment termination in a mouse model of Charcot-Marie-Tooth 1

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Axonal spheroids are regulated by Schwann cells after peripheral nerve injury

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FK506-binding protein-like and FK506-binding protein 8 regulate dual leucine zipper kinase degradation and neuronal responses to axon injury

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Structural Evidence for an Octameric Ring Arrangement of SARM1

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