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Synergistic interaction between amyloid and tau predicts the progression to dementia
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archneur.62.2.192openalex-snapshot
Defining Molecular Targets to Prevent Alzheimer Disease
inferredcites →https://doi.org/10.1001/archneurol.2011.3354openalex-snapshot
Amyloid-β–Associated Clinical Decline Occurs Only in the Presence of Elevated P-tau
inferredcites →https://doi.org/10.1001/jamaneurol.2015.1633openalex-snapshot
Comparison of Visual and Quantitative Florbetapir F 18 Positron Emission Tomography Analysis in Predicting Mild Cognitive Impairment Outcomes
inferredcites →https://doi.org/10.1001/jamaneurol.2015.2323openalex-snapshot
Role of β-Amyloidosis and Neurodegeneration in Subsequent Imaging Changes in Mild Cognitive Impairment
inferredcites →https://doi.org/10.1002/ana.21610openalex-snapshot
Cerebrospinal fluid biomarker signature in Alzheimer's disease neuroimaging initiative subjects
inferredcites →https://doi.org/10.1002/ana.22248openalex-snapshot
Longitudinal assessment of Aβ and cognition in aging and Alzheimer disease
inferredcites →https://doi.org/10.1002/ana.23650openalex-snapshot
Amyloid deposition, hypometabolism, and longitudinal cognitive decline
inferredcites →https://doi.org/10.1002/ana.23931openalex-snapshot
Mild cognitive impairment due to Alzheimer disease in the community
inferredcites →https://doi.org/10.1002/ana.24186openalex-snapshot
Cerebrospinal fluid β‐amyloid and phospho‐tau biomarker interactions affecting brain structure in preclinical Alzheimer disease
inferredcites →https://doi.org/10.1007/bf00308809openalex-snapshot
Neuropathological stageing of Alzheimer-related changes
inferredcites →https://doi.org/10.1007/s00259-013-2562-0openalex-snapshot
Five-year follow-up of 11C-PIB uptake in Alzheimer’s disease and MCI
inferredcites →https://doi.org/10.1007/s00401-010-0794-7openalex-snapshot
Tau pathology in children and young adults: can you still be unconditionally baptist?
inferredcites →https://doi.org/10.1007/s00401-013-1151-4openalex-snapshot
Longitudinal change in CSF Tau and Aβ biomarkers for up to 48 months in ADNI
inferredcites →https://doi.org/10.1016/b978-012264841-0/50047-0openalex-snapshot
Developments in Random Field Theory
inferredcites →https://doi.org/10.1016/j.jalz.2011.03.005openalex-snapshot
The diagnosis of dementia due to Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jalz.2011.03.008openalex-snapshot
The diagnosis of mild cognitive impairment due to Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jalz.2015.05.004openalex-snapshot
The Alzheimer's Disease Neuroimaging Initiative phase 2: Increasing the length, breadth, and depth of our understanding
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2015.08.029openalex-snapshot
Suspected non-AD pathology in mild cognitive impairment
inferredcites →https://doi.org/10.1016/j.neuroimage.2009.05.037openalex-snapshot
Neural correlates of Alzheimer's disease and mild cognitive impairment: A systematic and quantitative meta-analysis involving 1351 patients
inferredcites →https://doi.org/10.1016/j.neuron.2014.10.038openalex-snapshot
The Evolution of Preclinical Alzheimer’s Disease: Implications for Prevention Trials
inferredcites →https://doi.org/10.1016/s1474-4422(12)70291-0openalex-snapshot
Tracking pathophysiological processes in Alzheimer's disease: an updated hypothetical model of dynamic biomarkers
inferredcites →https://doi.org/10.1038/mp.2016.37openalex-snapshot
Amyloid-β and hyperphosphorylated tau synergy drives metabolic decline in preclinical Alzheimer’s disease
inferredcites →https://doi.org/10.1038/nm.3810openalex-snapshot
Understanding Biomarkers of Neurodegeneration: Ultrasensitive detection techniques pave the way for mechanistic understanding
inferredcites →https://doi.org/10.1038/nrn2967openalex-snapshot
Amyloid-β and tau — a toxic pas de deux in Alzheimer's disease
inferredcites →https://doi.org/10.1083/jcb.201407065openalex-snapshot
Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β
inferredcites →https://doi.org/10.1093/oso/9780195123425.001.0001openalex-snapshot
Mild Cognitive Impairment
inferredcites →https://doi.org/10.1126/science.1194653openalex-snapshot
Tau Reduction Prevents Aβ-Induced Defects in Axonal Transport
inferredcites →https://doi.org/10.1159/000334295openalex-snapshot
Relationship between Memory Performance and β-Amyloid Deposition at Different Stages of Alzheimer’s Disease
inferredcites →https://doi.org/10.1177/0969141315597959openalex-snapshot
Correction factors for self-selection when evaluating screening programmes
inferredcites →https://doi.org/10.1212/01.wnl.0000183284.92920.f2openalex-snapshot
Phosphorylated tau predicts rate of cognitive decline in MCI subjects: A comparative CSF study
inferredcites →https://doi.org/10.1212/01.wnl.0000244749.20056.d4openalex-snapshot
Amyloid, hypometabolism, and cognition in Alzheimer disease
inferredcites →https://doi.org/10.1212/wnl.0000000000000432openalex-snapshot
Long-term effects of amyloid, hypometabolism, and atrophy on neuropsychological functions
inferredcites →https://doi.org/10.1212/wnl.0000000000001209openalex-snapshot
Mild cognitive impairment with suspected nonamyloid pathology (SNAP)
inferredcites →https://doi.org/10.1212/wnl.0b013e318212015eopenalex-snapshot
Amyloid PET imaging in patients with mild cognitive impairment
inferredcites →https://doi.org/10.1373/clinchem.2004.039347openalex-snapshot
Simultaneous Measurement of β-Amyloid(1–42), Total Tau, and Phosphorylated Tau (Thr181) in Cerebrospinal Fluid by the xMAP Technology
inferredcites →https://doi.org/10.1385/jmn:17:2:101openalex-snapshot
Pathologic Correlates of Nondemented Aging, Mild Cognitive Impairment, and Early-Stage Alzheimer's Disease
inferredcites →https://doi.org/10.3389/fninf.2016.00020openalex-snapshot
VoxelStats: A MATLAB Package for Multi-Modal Voxel-Wise Brain Image Analysis
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4588112openalex-extracted
Neuronal Expression of Human Amyloid-β and Tau Drives Global Phenotypic and Multi-Omic Changes in C. elegans
inferred← citeshttps://doi.org/10.1002/14651858.cd012216.pub2openalex-snapshot
18F PET with florbetapir for the early diagnosis of Alzheimer’s disease dementia and other dementias in people with mild cognitive impairment (MCI)
inferred← citeshttps://doi.org/10.1002/acn3.51457openalex-snapshot
Amyloid‐dependent and amyloid‐independent effects of Tau in individuals without dementia
inferred← citeshttps://doi.org/10.1002/alz.71742openalex-snapshot
Unique transcriptomic alterations in 5XFAD;PS19 mouse model identify glial lipid dysregulation and coordinated microglial–oligodendrocyte responses
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Association of Presynaptic Loss with Alzheimer's Disease and Cognitive Decline
inferred← citeshttps://doi.org/10.1002/cbic.202100383openalex-snapshot
Thiophene‐Based Dual Modulators of Aβ and Tau Aggregation
inferred← citeshttps://doi.org/10.1002/dad2.70496openalex-snapshot
APOE and amyloid‐tau pathology in cognitively unimpaired older adults
inferred← citeshttps://doi.org/10.1002/hbm.25133openalex-snapshot
Nonlinear biomarker interactions in conversion from mild cognitive impairment to Alzheimer's disease
inferred← citeshttps://doi.org/10.1007/978-3-030-93080-6_5openalex-snapshot
Neuro-Symbolic Neurodegenerative Disease Modeling as Probabilistic Programmed Deep Kernels
inferred← citeshttps://doi.org/10.1007/s00259-018-3933-3openalex-snapshot
Amyloid and tau signatures of brain metabolic decline in preclinical Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00285-023-01985-7openalex-snapshot
The role of A$$\beta $$ and Tau proteins in Alzheimer’s disease: a mathematical model on graphs
inferred← citeshttps://doi.org/10.1007/s00401-018-1932-xopenalex-snapshot
Current state of Alzheimer’s fluid biomarkers
inferred← citeshttps://doi.org/10.1007/s00401-019-02054-4openalex-snapshot
The basis of cellular and regional vulnerability in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00702-020-02232-9openalex-snapshot
Neuropathological assessment of the Alzheimer spectrum
inferred← citeshttps://doi.org/10.1007/s10072-025-08623-8openalex-snapshot
Sex and gender-related differences in neurological diseases: current challenges and recommendations for clinical practice
inferred← citeshttps://doi.org/10.1007/s11064-020-03115-xopenalex-snapshot
Dimethyl Fumarate Mitigates Tauopathy in Aβ-Induced Neuroblastoma SH-SY5Y Cells
inferred← citeshttps://doi.org/10.1007/s12035-023-03217-xopenalex-snapshot
TREM2 Inhibits Tau Hyperphosphorylation and Neuronal Apoptosis via the PI3K/Akt/GSK-3β Signaling Pathway In vivo and In vitro
inferred← citeshttps://doi.org/10.1007/s13365-025-01265-8openalex-snapshot
Herpesvirus initiation of dementias and autoimmune diseases
inferred← citeshttps://doi.org/10.1016/j.bbadis.2021.166162openalex-snapshot
Phosphorylated tau targeted small-molecule PROTACs for the treatment of Alzheimer's disease and tauopathies
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2021.121360openalex-snapshot
Precise gene delivery systems with detachable albumin shell remodeling dysfunctional microglia by TREM2 for treatment of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.biopsych.2020.06.029openalex-snapshot
Longitudinal Cognitive and Biomarker Measurements Support a Unidirectional Pathway in Alzheimer’s Disease Pathophysiology
inferred← citeshttps://doi.org/10.1016/j.bios.2024.116405openalex-snapshot
Strategies for measuring concentrations and forms of amyloid-β peptides
inferred← citeshttps://doi.org/10.1016/j.brain.2020.100020openalex-snapshot
A sensitivity analysis of a mathematical model for the synergistic interplay of amyloid beta and tau on the dynamics of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.cis.2022.102824openalex-snapshot
Detection and modulation of neurodegenerative processes using graphene-based nanomaterials: Nanoarchitectonics and applications
inferred← citeshttps://doi.org/10.1016/j.dadm.2017.08.001openalex-snapshot
Characterizing biomarker features of cognitively normal individuals with ventriculomegaly
inferred← citeshttps://doi.org/10.1016/j.dadm.2018.08.005openalex-snapshot
In vivo coupling of tau pathology and cortical thinning in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.dadm.2019.08.002openalex-snapshot
Plasma neurofilament light associates with Alzheimer's disease metabolic decline in amyloid‐positive individuals
inferred← citeshttps://doi.org/10.1016/j.drudis.2018.11.005openalex-snapshot
Pharmacophore-based models for therapeutic drugs against phosphorylated tau in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2020.07.327openalex-snapshot
Role of tau protein in Alzheimer's disease: The prime pathological player
inferred← citeshttps://doi.org/10.1016/j.jalz.2017.06.002openalex-snapshot
The pitfalls of biomarker‐based classification schemes
inferred← citeshttps://doi.org/10.1016/j.jalz.2018.08.005openalex-snapshot
Understanding disease progression and improving Alzheimer's disease clinical trials: Recent highlights from the Alzheimer's Disease Neuroimaging Initiative
inferred← citeshttps://doi.org/10.1016/j.nbd.2020.104811openalex-snapshot
Alzheimer's disease pathology in APOE transgenic mouse models: The Who, What, When, Where, Why, and How
inferred← citeshttps://doi.org/10.1016/j.nbd.2024.106473openalex-snapshot
A nonlinear meccano for Alzheimer's emergence by amyloid β-mediated glutamatergic hyperactivity
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2019.03.016openalex-snapshot
Relevance of the interplay between amyloid and tau for cognitive impairment in early Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2021.06.004openalex-snapshot
Apolipoprotein E4, amyloid, and cognition in Alzheimer's and Lewy body disease
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2021.09.019openalex-snapshot
Abnormal tau in amyloid PET negative individuals
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2022.03.005openalex-snapshot
Rates of β-amyloid deposition indicate widespread simultaneous accumulation throughout the brain
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2022.07.003openalex-snapshot
Amyloid- β and tau deposition influences cognitive and functional decline in Down syndrome
inferred← citeshttps://doi.org/10.1016/j.neurol.2025.07.016openalex-snapshot
The challenging concept of preclinical Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.nicl.2017.10.034openalex-snapshot
Interactive versus additive relationships between regional cortical thinning and amyloid burden in predicting clinical decline in mild AD and MCI individuals
inferred← citeshttps://doi.org/10.1021/acschemneuro.1c00045openalex-snapshot
A Mechanism for the Inhibition of Tau Neurotoxicity: Studies with Artificial Membranes, Isolated Mitochondria, and Intact Cells
inferred← citeshttps://doi.org/10.1021/acschemneuro.3c00014openalex-snapshot
Differential Binding and Conformational Dynamics of Tau Microtubule-Binding Repeats with a Preformed Amyloid-β Fibril Seed
inferred← citeshttps://doi.org/10.1038/s41380-020-0688-6openalex-snapshot
APOEε4 potentiates the relationship between amyloid-β and tau pathologies
inferred← citeshttps://doi.org/10.1038/s41380-022-01436-7openalex-snapshot
Age, sex and APOE-ε4 modify the balance between soluble and fibrillar β-amyloid in non-demented individuals: topographical patterns across two independent cohorts
inferred← citeshttps://doi.org/10.1038/s41398-025-03589-9openalex-snapshot
Salivary mitochondrial DNA is associated with biomarkers of Alzheimer’s disease in cognitively normal older adults
inferred← citeshttps://doi.org/10.1038/s41467-019-10217-wopenalex-snapshot
Aβ-induced vulnerability propagates via the brain’s default mode network
inferred← citeshttps://doi.org/10.1038/s41541-022-00544-3openalex-snapshot
Immunogenicity of MultiTEP platform technology-based Tau vaccine in non-human primates
inferred← citeshttps://doi.org/10.1038/s41598-024-60843-8openalex-snapshot
Exploration of neuroanatomical characteristics to differentiate prodromal Alzheimer’s disease from cognitively unimpaired amyloid-positive individuals
inferred← citeshttps://doi.org/10.1038/s43587-021-00138-zopenalex-snapshot
Endophenotype-based in silico network medicine discovery combined with insurance record data mining identifies sildenafil as a candidate drug for Alzheimer’s disease
inferred← citeshttps://doi.org/10.1038/s43856-023-00324-7openalex-snapshot
Cortical microstructural changes predict tau accumulation and episodic memory decline in older adults harboring amyloid
inferred← citeshttps://doi.org/10.1063/5.0152605openalex-snapshot
Stability and bifurcation analysis of Alzheimer’s disease model with diffusion and three delays
inferred← citeshttps://doi.org/10.1080/13803395.2022.2036327openalex-snapshot
Using the ATN system as a guide for the neuropsychological assessment of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/brain/awab248openalex-snapshot
Longitudinal 18F-MK-6240 tau tangles accumulation follows Braak stages
inferred← citeshttps://doi.org/10.1093/brain/awad397openalex-snapshot
Sex-specific modulation of amyloid-β on tau phosphorylation underlies faster tangle accumulation in females
inferred← citeshttps://doi.org/10.1093/brain/awae372openalex-snapshot
Patterns of tau, amyloid and synuclein pathology in ageing, Alzheimer’s disease and synucleinopathies
inferred← citeshttps://doi.org/10.1093/brain/awaf033openalex-snapshot
Plasma phosphorylated tau217 strongly associates with memory deficits in the Alzheimer’s disease spectrum
inferred← citeshttps://doi.org/10.1093/brain/awy188openalex-snapshot
Microglial activation correlates in vivo with both tau and amyloid in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/braincomms/fcac026openalex-snapshot
Grey matter network markers identify individuals with prodromal Alzheimer’s disease who will show rapid clinical decline
inferred← citeshttps://doi.org/10.1093/braincomms/fcad216openalex-snapshot
Association of the fibronectin type III domain–containing protein 5 rs1746661 single nucleotide polymorphism with reduced brain glucose metabolism in elderly humans
inferred← citeshttps://doi.org/10.1096/fj.202000051rrropenalex-snapshot
Virulent Pseudomonas aeruginosa infection converts antimicrobial amyloids into cytotoxic prions
inferred← citeshttps://doi.org/10.1101/2021.03.16.435674openalex-snapshot
Stable brain PET metabolic networks using a multiple sampling scheme
inferred← citeshttps://doi.org/10.1101/2023.06.01.542377openalex-snapshot
Neuronal expression of human amyloid-β and Tau drives global phenotypic and multi-omic changes in C. elegans
inferred← citeshttps://doi.org/10.1101/2023.09.27.559737openalex-snapshot
Spatial association between distributed β-amyloid and tau varies with cognition
inferred← citeshttps://doi.org/10.1101/2023.10.09.561541openalex-snapshot
The nonlinear meccano of hyperactivity in Alzheimer
inferred← citeshttps://doi.org/10.1109/jbhi.2019.2929264openalex-snapshot
Dynamic Prediction in Clinical Survival Analysis Using Temporal Convolutional Networks
inferred← citeshttps://doi.org/10.1109/sipaim67325.2025.11283325openalex-snapshot
White Matter Tract Vulnerability to Amyloid Pathology on the Alzheimer’s Disease Continuum
inferred← citeshttps://doi.org/10.1111/pcn.13704openalex-snapshot
Synergism of ApoE4 and systemic infectious burden is mediated by the APOE ‐ NLRP3 axis in Alzheimer's disease
inferred← citeshttps://doi.org/10.1159/000508625openalex-snapshot
Pathobiological Subtypes of Alzheimer Disease
inferred← citeshttps://doi.org/10.1159/000530907openalex-snapshot
CSF Aβ40 Levels Do Not Correlate with the Clinical Manifestations of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1177/13872877251322196openalex-snapshot
Synergistic associations of amyloid-β and phosphorylated tau with tau aggregation and cognitive decline in Alzheimer's disease
inferred← citeshttps://doi.org/10.1186/s13195-018-0402-yopenalex-snapshot
In vivo quantification of neurofibrillary tangles with [18F]MK-6240
inferred← citeshttps://doi.org/10.1186/s13195-019-0541-9openalex-snapshot
Amyloid β-protein oligomers promote the uptake of tau fibril seeds potentiating intracellular tau aggregation
inferred← citeshttps://doi.org/10.1186/s13195-019-0556-2openalex-snapshot
Testing a MultiTEP-based combination vaccine to reduce Aβ and tau pathology in Tau22/5xFAD bigenic mice
inferred← citeshttps://doi.org/10.1186/s13578-021-00712-3openalex-snapshot
Soluble amyloid-beta isoforms predict downstream Alzheimer’s disease pathology
inferred← citeshttps://doi.org/10.1186/s40478-017-0505-xopenalex-snapshot
Pyroglutamate and Isoaspartate modified Amyloid-Beta in ageing and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1212/wnl.0000000000008361openalex-snapshot
Applying the ATN scheme in a memory clinic population
inferred← citeshttps://doi.org/10.1503/jpn.170109openalex-snapshot
Neuroimaging tests for clinical psychiatry: Are we there yet?
inferred← citeshttps://doi.org/10.1523/jneurosci.0485-18.2018openalex-snapshot
Subthreshold Amyloid Predicts Tau Deposition in Aging
inferred← citeshttps://doi.org/10.17879/freeneuropathology-2020-3050openalex-snapshot
Neuropathology of the Alzheimer's continuum: an update
inferred← citeshttps://doi.org/10.20944/preprints202211.0032.v1openalex-snapshot
Nano-Theranostics Approaches and Mitochondrial Targeted Drug Delivery: Alzheimer ’s Disease Therapeutic Interventions
inferred← citeshttps://doi.org/10.21203/rs.3.rs-1032345/v1openalex-snapshot
Soluble Amyloid-Beta Isoforms Predict Downstream Alzheimer’s Disease Pathology
inferred← citeshttps://doi.org/10.2174/1874120701913010055openalex-snapshot
Amyloid-Negative Dementia in the Elderly is Associated with High Accumulation of Tau in the Temporal Lobes
inferred← citeshttps://doi.org/10.3233/adr-200247openalex-snapshot
Borrelidin from Saltern-Derived Halophilic Nocardiopsis sp. Dissociates Amyloid- β and Tau Fibrils
inferred← citeshttps://doi.org/10.3233/jad-179924openalex-snapshot
Targeting Alzheimer’s Disease at the Right Time and the Right Place: Validation of a Personalized Approach to Diagnosis and Treatment
inferred← citeshttps://doi.org/10.3233/jad-215465openalex-snapshot
The Outcome of Patients with Amyloid-Negative Amnestic Mild Cognitive Impairment
inferred← citeshttps://doi.org/10.3389/fnins.2021.606600openalex-snapshot
Effects of Alzheimer’s and Vascular Pathologies on Structural Connectivity in Early- and Late-Onset Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fnmol.2019.00268openalex-snapshot
RETRACTED: Different Amyloid-β Self-Assemblies Have Distinct Effects on Intracellular Tau Aggregation
inferred← citeshttps://doi.org/10.3390/biom12111676openalex-snapshot
Linking the Amyloid, Tau, and Mitochondrial Hypotheses of Alzheimer’s Disease and Identifying Promising Drug Targets
inferred← citeshttps://doi.org/10.3390/biom15010128openalex-snapshot
Deciphering the Role of Adrenergic Receptors in Alzheimer’s Disease: Paving the Way for Innovative Therapies
inferred← citeshttps://doi.org/10.3390/ijms20081871openalex-snapshot
Centella asiatica Protects d-Galactose/AlCl3 Mediated Alzheimer’s Disease-Like Rats via PP2A/GSK-3β Signaling Pathway in Their Hippocampus
inferred← citeshttps://doi.org/10.3390/ijms21030703openalex-snapshot
Computer-Aided Drug Design of β-Secretase, γ-Secretase and Anti-Tau Inhibitors for the Discovery of Novel Alzheimer’s Therapeutics
inferred← citeshttps://doi.org/10.3390/ijms22010461openalex-snapshot
Intermittent Hypoxic Conditioning Rescues Cognition and Mitochondrial Bioenergetic Profile in the Triple Transgenic Mouse Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/ijms24065285openalex-snapshot
A Polyaminobiaryl-Based β-secretase Modulator Alleviates Cognitive Impairments, Amyloid Load, Astrogliosis, and Neuroinflammation in APPSwe/PSEN1ΔE9 Mice Model of Amyloid Pathology
inferred← citeshttps://doi.org/10.3390/ijms25179690openalex-snapshot
Alzheimer’s Disease Pathology and Assistive Nanotheranostic Approaches for Its Therapeutic Interventions
inferred← citeshttps://doi.org/10.3390/molecules29030722openalex-snapshot
Early Diagnosis of Neurodegenerative Diseases: What Has Been Undertaken to Promote the Transition from PET to Fluorescence Tracers
inferred← citeshttps://doi.org/10.3390/pharmaceutics15102369openalex-snapshot
New Hybrid Compounds Incorporating Natural Products as Multifunctional Agents against Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3988/jcn.2020.16.1.131openalex-snapshot
Clinical Usefulness of 18F-FC119S Positron-Emission Tomography as an Auxiliary Diagnostic Method for Dementia: An Open-Label, Single-Dose, Evaluator-Blind Clinical Trial
inferred← citeshttps://doi.org/10.48550/arxiv.1811.10746openalex-snapshot
MATCH-Net: Dynamic Prediction in Survival Analysis using Convolutional Neural Networks
inferred← citeshttps://doi.org/10.48550/arxiv.2001.08345openalex-snapshot
Target-Embedding Autoencoders for Supervised Representation Learning
inferred← citeshttps://doi.org/10.70675/873ca377z9a97z43f6z8051zdab4082686baopenalex-snapshot
Regulation of Alzheimer pathology by amyloid seeds : from toxic effects to therapeutic opportunities, in mouse model of amyloidosis
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