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Identification of an anabolic selective androgen receptor modulator that actively induces death of androgen-independent prostate cancer cells

https://doi.org/10.1016/j.jsbmb.2014.02.005
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Lower-Dose vs High-Dose Oral Estradiol Therapy of Hormone Receptor–Positive, Aromatase Inhibitor–Resistant Advanced Breast Cancer
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Hormone therapy for patients with prostate carcinoma
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Aberrant expression of Cks1 and Cks2 contributes to prostate tumorigenesis by promoting proliferation and inhibiting programmed cell death
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Adeno‐associated virus–delivered short hairpin‐structured RNA for androgen receptor gene silencing induces tumor eradication of prostate cancer xenografts in nude mice: A preclinical study
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The Role of Hormonal Treatment in Prostate Cancer
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A new human prostate carcinoma cell line, 22Rv1
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Testosterone and the prostate: Implications for the treatment of hypogonadal men
inferredcites →https://doi.org/10.1016/0959-8049(94)90424-3openalex-snapshot
MDA-MB-453, an androgen-responsive human breast carcinoma cell line with high level androgen receptor expression
inferredcites →https://doi.org/10.1016/j.ccr.2009.05.005openalex-snapshot
Development of Androgen Receptor Antagonists with Promising Activity in Castration-Resistant Prostate Cancer
inferredcites →https://doi.org/10.1016/j.cell.2009.04.056openalex-snapshot
Androgen Receptor Regulates a Distinct Transcription Program in Androgen-Independent Prostate Cancer
inferredcites →https://doi.org/10.1016/j.ejca.2007.11.002openalex-snapshot
Tamoxifen: Catalyst for the change to targeted therapy
inferredcites →https://doi.org/10.1016/j.jsbmb.2004.04.009openalex-snapshot
Partial agonist/antagonist properties of androstenedione and 4-androsten-3β,17β-diol
inferredcites →https://doi.org/10.1016/j.jsbmb.2007.10.013openalex-snapshot
Agonist-like SERM effects on ERα-mediated repression of MMP1 promoter activity predict in vivo effects on bone and uterus
inferredcites →https://doi.org/10.1016/j.juro.2008.05.045openalex-snapshot
Alternative Nonsteroidal Antiandrogen Therapy for Advanced Prostate Cancer That Relapsed After Initial Maximum Androgen Blockade
inferredcites →https://doi.org/10.1016/j.mce.2011.03.026openalex-snapshot
Structural features discriminate androgen receptor N/C terminal and coactivator interactions
inferredcites →https://doi.org/10.1016/j.urolonc.2008.03.021openalex-snapshot
Mechanisms mediating androgen receptor reactivation after castration
inferredcites →https://doi.org/10.1016/j.urolonc.2009.03.016openalex-snapshot
Castration-resistant prostate cancer: Targeting androgen metabolic pathways in recurrent disease
inferredcites →https://doi.org/10.1016/s0022-5347(05)00034-0openalex-snapshot
Secondary Hormonal Therapy for Advanced Prostate Cancer
inferredcites →https://doi.org/10.1016/s0039-128x(96)00086-4openalex-snapshot
Androgenic up-regulation of androgen receptor cDNA expression in androgen-independent prostate cancer cells
inferredcites →https://doi.org/10.1016/s0090-4295(02)01593-5openalex-snapshot
Androgen receptor as a target in androgen-independent prostate cancer
inferredcites →https://doi.org/10.1016/s0094-0143(05)70071-9openalex-snapshot
SECONDARY HORMONAL MANIPULATIONS IN HORMONE REFRACTORY PROSTATE CANCER
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Molecular mechanisms of estrogen action: selective ligands and receptor pharmacology
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Synthesis and Characterization of Nonsteroidal-Linked M(CO)3+(M = 99mTc, Re) Compounds Based on the Androgen Receptor Targeting Molecule Flutamide
inferredcites →https://doi.org/10.1021/bi000343+openalex-snapshot
Ets Domain Transcription Factor PE1 Suppresses Human Interstitial Collagenase Promoter Activity by Antagonizing Protein−DNA Interactions at a Critical AP1 Element
inferredcites →https://doi.org/10.1021/cb900043eopenalex-snapshot
Alternative Inhibition of Androgen Receptor Signaling: Peptidomimetic Pyrimidines As Direct Androgen Receptor/Coactivator Disruptors
inferredcites →https://doi.org/10.1021/ja953069topenalex-snapshot
Mechanism-Based Inhibition of Human Steroid 5α-Reductase by Finasteride: Enzyme-Catalyzed Formation of NADP−Dihydrofinasteride, a Potent Bisubstrate Analog Inhibitor
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4-Aza-3-oxo-5.alpha.-androst-1-ene-17.beta.-N-arylcarboxamides as Dual Inhibitors of Human Type 1 and Type 2 Steroid 5.alpha.-Reductases. Dramatic Effect of N-Aryl Substituents on Type 1 and Type 2 5.alpha.-Reductase Inhibitory Potency
inferredcites →https://doi.org/10.1038/ncpuro1296openalex-snapshot
Androgen deprivation therapy: progress in understanding mechanisms of resistance and optimizing androgen depletion
inferredcites →https://doi.org/10.1038/sj.onc.1208677openalex-snapshot
Molecular cloning and characterization of STAMP2, an androgen-regulated six transmembrane protein that is overexpressed in prostate cancer
inferredcites →https://doi.org/10.1056/nejmoa030660openalex-snapshot
The Influence of Finasteride on the Development of Prostate Cancer
inferredcites →https://doi.org/10.1074/jbc.c500324200openalex-snapshot
Interaction between the Androgen Receptor and RNase L Mediates a Cross-talk between the Interferon and Androgen Signaling Pathways
inferredcites →https://doi.org/10.1074/jbc.m109.049734openalex-snapshot
Identification of Anabolic Selective Androgen Receptor Modulators with Reduced Activities in Reproductive Tissues and Sebaceous Glands
inferredcites →https://doi.org/10.1074/jbc.m109.099002openalex-snapshot
Discovery of the Selective Androgen Receptor Modulator MK-0773 Using a Rational Development Strategy Based on Differential Transcriptional Requirements for Androgenic Anabolism Versus Reproductive Physiology
inferredcites →https://doi.org/10.1074/jbc.m310206200openalex-snapshot
Identification of Genetic Pathways Activated by the Androgen Receptor during the Induction of Proliferation in the Ventral Prostate Gland
inferredcites →https://doi.org/10.1093/med/9780190655341.003.0002openalex-snapshot
The Influence of Finasteride on the Development of Prostate Cancer
inferredcites →https://doi.org/10.1097/00001813-200511000-00001openalex-snapshot
Targeting CK2 for cancer therapy
inferredcites →https://doi.org/10.1097/01.ju.0000106190.32540.6copenalex-snapshot
Alternative Antiandrogens to Treat Prostate Cancer Relapse After Initial Hormone Therapy
inferredcites →https://doi.org/10.1097/mco.0b013e32832a3d79openalex-snapshot
Selective androgen receptor modulators as function promoting therapies
inferredcites →https://doi.org/10.1097/mou.0b013e328329b73aopenalex-snapshot
Novel secondary hormonal therapy in advanced prostate cancer: an update
inferredcites →https://doi.org/10.1097/mou.0b013e328329eb29openalex-snapshot
The utility of 5-α reductase inhibitors in the prevention and diagnosis of prostate cancer
inferredcites →https://doi.org/10.1126/science.1168175openalex-snapshot
Development of a Second-Generation Antiandrogen for Treatment of Advanced Prostate Cancer
inferredcites →https://doi.org/10.1158/0008-5472.can-09-0029openalex-snapshot
A Century of Deciphering the Control Mechanisms of Sex Steroid Action in Breast and Prostate Cancer: The Origins of Targeted Therapy and Chemoprevention
inferredcites →https://doi.org/10.1158/0008-5472.can-09-1111openalex-snapshot
Discovery of BMS-641988, a Novel and Potent Inhibitor of Androgen Receptor Signaling for the Treatment of Prostate Cancer
inferredcites →https://doi.org/10.1158/1535-7163.mct-08-0230openalex-snapshot
Androgen receptor inactivation contributes to antitumor efficacy of 17α-hydroxylase/17,20-lyase inhibitor 3β-hydroxy-17-(1H-benzimidazole-1-yl)androsta-5,16-diene in prostate cancer
inferredcites →https://doi.org/10.1186/bcr2255openalex-snapshot
Estrogen regulation of apoptosis: how can one hormone stimulate and inhibit?
inferredcites →https://doi.org/10.1210/en.139.3.1411openalex-snapshot
The Integrin Ligand Echistatin Prevents Bone Loss in Ovariectomized Mice and Rats
inferredcites →https://doi.org/10.1210/en.2004-0627openalex-snapshot
Comparison of the Pharmacological Effects of a Novel Selective Androgen Receptor Modulator, the 5α-Reductase Inhibitor Finasteride, and the Antiandrogen Hydroxyflutamide in Intact Rats: New Approach for Benign Prostate Hyperplasia
inferredcites →https://doi.org/10.1210/en.2004-1132openalex-snapshot
Androgenic Induction of Growth and Differentiation in the Rodent Uterus Involves the Modulation of Estrogen-Regulated Genetic Pathways
inferredcites →https://doi.org/10.1210/me.2005-0190openalex-snapshot
Antagonist-Induced, Activation Function-2-Independent Estrogen Receptor α Phosphorylation
inferredcites →https://doi.org/10.1210/me.8.1.21openalex-snapshot
Human estrogen receptor transactivational capacity is determined by both cellular and promoter context and mediated by two functionally distinct intramolecular regions
inferredcites →https://doi.org/10.1308/003588407x179125openalex-snapshot
A Review of Phase III Clinical Trials of Prostate Cancer Chemoprevention
inferredcites →https://doi.org/10.1517/14728220903544507openalex-snapshot
TGF-β signalling and immunity in prostate tumourigenesis
inferredcites →https://doi.org/10.2174/138161206776056056openalex-snapshot
Controversies of Androgen Ablation Therapy for Metastatic Prostate Cancer
inferredcites →https://doi.org/10.2174/1389201043376616openalex-snapshot
Targeting Steroid Hormone Receptor Pathways in the Treatment of Hormone Dependent Cancers
inferredcites →https://doi.org/10.2174/157489207782497172openalex-snapshot
The Androgen Receptor as Putative Therapeutic Target in Hormone-Refractory Prostate Cancer

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Combination of curcumin and bicalutamide enhanced the growth inhibition of androgen-independent prostate cancer cells through SAPK/JNK and MEK/ERK1/2-mediated targeting NF-κB/p65 and MUC1-C
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Selective Androgen Receptor Modulator S42 Suppresses Prostate Cancer Cell Proliferation
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