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Cocaine-induced oxidative stress precedes cell death in human neuronal progenitor cells
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archopht.1989.01070010853028openalex-snapshot
Orbital Inflammation and Optic Neuropathies Associated With Chronic Sinusitis of Intranasal Cocaine Abuse
inferredcites →https://doi.org/10.1002/(sici)1097-0185(19991215)257:6<208::aid-ar6>3.0.co;2-0openalex-snapshot
Cocaine and apoptosis in myocardial cells
inferredcites →https://doi.org/10.1007/s00268-003-7399-4openalex-snapshot
Redox Processes Underlying the Vascular Repair Reaction
inferredcites →https://doi.org/10.1016/0006-8993(95)00987-5openalex-snapshot
Dopamine release during cocaine self-administration in rats: effect of SCH23390
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Age-associated changes in superoxide dismutase activity, thiobarbituric acid reactivity and reduced glutathione level in the brain and liver in senescence accelerated mice (SAM): a comparison with ddY mice
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Self-administration of the high-affinity cocaine analog 2β-carbomethoxy-3β-(4-fluorophenyl)tropane
inferredcites →https://doi.org/10.1016/0361-9230(91)90284-qopenalex-snapshot
Inhibition of brain MAO-A and animal behaviour induced by p-hydroxyamphetamine
inferredcites →https://doi.org/10.1016/0736-5748(95)00071-2openalex-snapshot
Age‐related changes in antioxidant enzymes, superoxide dismutase, catalase, glutathione peroxidase and glutathione in different regions of mouse brain
inferredcites →https://doi.org/10.1016/j.annepidem.2005.08.003openalex-snapshot
Projecting Drug Use Among Aging Baby Boomers in 2020
inferredcites →https://doi.org/10.1016/j.brainres.2004.05.048openalex-snapshot
Antisense directed at the Aβ region of APP decreases brain oxidative markers in aged senescence accelerated mice
inferredcites →https://doi.org/10.1016/j.cger.2004.02.005openalex-snapshot
Free radicals and brain aging
inferredcites →https://doi.org/10.1016/j.mehy.2005.01.031openalex-snapshot
Unifying mechanism for addiction and toxicity of abused drugs with application to dopamine and glutamate mediators: Electron transfer and reactive oxygen species
inferredcites →https://doi.org/10.1016/j.neubiorev.2003.11.005openalex-snapshot
Aggression and defeat: persistent effects on cocaine self-administration and gene expression in peptidergic and aminergic mesocorticolimbic circuits
inferredcites →https://doi.org/10.1016/j.neuint.2003.11.018openalex-snapshot
Autoxidation and MAO-mediated metabolism of dopamine as a potential cause of oxidative stress: role of ferrous and ferric ions
inferredcites →https://doi.org/10.1016/j.neuropharm.2005.01.018openalex-snapshot
Acute or repeated cocaine administration generates reactive oxygen species and induces antioxidant enzyme activity in dopaminergic rat brain structures
inferredcites →https://doi.org/10.1016/s0006-8993(01)03047-5openalex-snapshot
Cocaine activates redox-regulated transcription factors and induces TNF-α expression in human brain endothelial cells
inferredcites →https://doi.org/10.1016/s0008-6363(03)00499-1openalex-snapshot
Role of reactive oxygen species in cocaine-induced cardiac dysfunction
inferredcites →https://doi.org/10.1016/s0022-3565(25)20684-xopenalex-snapshot
Effects of cocaine and related drugs in nonhuman primates. I. [3H]cocaine binding sites in caudate-putamen.
inferredcites →https://doi.org/10.1016/s0891-5849(96)00629-6openalex-snapshot
Oxidative Stress Hypothesis in Alzheimer's Disease
inferredcites →https://doi.org/10.1016/s1567-5769(03)00050-xopenalex-snapshot
Immunosuppression and oxidative stress induced by acute and chronic exposure to cocaine in rat
inferredcites →https://doi.org/10.1037/e497012006-003openalex-snapshot
Neural Mechanisms of the Reinforcing Action of Cocaine
inferredcites →https://doi.org/10.1038/35040009openalex-snapshot
Apoptosis in neurodegenerative disorders
inferredcites →https://doi.org/10.1046/j.1471-4159.1997.68062442.xopenalex-snapshot
Cocaine Induces Apoptosis in Cortical Neurons of Fetal Mice
inferredcites →https://doi.org/10.1093/gerona/59.5.m478openalex-snapshot
Free Radicals: Key to Brain Aging and Heme Oxygenase as a Cellular Response to Oxidative Stress
inferredcites →https://doi.org/10.1097/00001756-200508010-00017openalex-snapshot
Memory impairments and oxidative stress in the hippocampus of in-utero cocaine-exposed rats
inferredcites →https://doi.org/10.1097/01.wco.0000162851.44897.8fopenalex-snapshot
Effect of inflammation on central nervous system development and vulnerability: review
inferredcites →https://doi.org/10.1111/j.1471-4159.2006.03760.xopenalex-snapshot
Cocaine induced inflammatory response in human neuronal progenitor cells
inferredcites →https://doi.org/10.1126/science.694530openalex-snapshot
Cocaine Plasma Concentration: Relation to Physiological and Subjective Effects in Humans
inferredcites →https://doi.org/10.1385/ct:3:2:141openalex-snapshot
Cocaine, Not Morphine, Causes the Generation of Reactive Oxygen Species and Activation of NF-κB in Transiently Cotransfected Heart Cells
inferredcites →https://doi.org/10.1523/jneurosci.16-23-07416.1996openalex-snapshot
Differential Reinforcing Effects of Cocaine and GBR-12909: Biochemical Evidence for Divergent Neuroadaptive Changes in the Mesolimbic Dopaminergic System
inferredcites →https://doi.org/10.3109/08923979909007140openalex-snapshot
Effect of Short Term Cocaine Administration On the Immune System of Yg and Old C57BL/6 Female Mice
inferredcites →https://doi.org/10.3109/10715769709065765openalex-snapshot
Oxidative Modification of Glutamine Synthetase by Amyloid Beta Peptide
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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The Neurocognitive Control and Vagal Regulation Impaired and Recovered in Males with Substance Use Disorders of Different Abstinence Phases
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Sex‐specific behavioral and neurogenic responses to cocaine in mice lacking and blocking dopamine D1 or dopamine D2 receptors
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Total antioxidant capacity is significantly lower in cocaine‐dependent and methamphetamine‐dependent patients relative to normal controls: results from a preliminary study
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Neuroproteomics and its applications in research on nicotine and other drugs of abuse
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Effects of addictive drugs on adult neural stem/progenitor cells
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Dimethyltryptamine and harmine, components of ayahuasca, prevented cocaine-induced apoptosis in SH-SY5Y human neuroblastoma cells
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Cocaine Mediated Neuroinflammation: Role of Dysregulated Autophagy in Pericytes
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Oxidative Stress Biomarkers in Some Rat Brain Structures and Peripheral Organs Underwent Cocaine
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A Comprehensive View of the Neurotoxicity Mechanisms of Cocaine and Ethanol
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Ketamine-Ethanol Combination Decreases Reduced Glutathione Levels and Activates both Intrinsic and Extrinsic Apoptotic Pathways Prior to Neuronal Death in SH-SY5Y Cells
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Additive cortical gray matter deficits in people living with HIV who use cocaine
inferred← citeshttps://doi.org/10.1016/b978-0-12-803750-8.00021-xopenalex-snapshot
Role of Mitochondria on the Neurological Effects of Cocaine
inferred← citeshttps://doi.org/10.1016/b978-0-12-803750-8.00036-1openalex-snapshot
Long-Lasting Changes Following Repeated Cocaine Use
inferred← citeshttps://doi.org/10.1016/bs.pbr.2015.07.004openalex-snapshot
Drug-induced neurotoxicity in addiction medicine
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Is tau pathology a relevant factor in neuronal damage induced by alcohol and other drugs?
inferred← citeshttps://doi.org/10.1016/j.bcp.2017.05.003openalex-snapshot
The impact of cocaine on adult hippocampal neurogenesis: Potential neurobiological mechanisms and contributions to maladaptive cognition in cocaine addiction disorder
inferred← citeshttps://doi.org/10.1016/j.biopha.2026.119423openalex-snapshot
Multilevel impact of cocaine on honeybees: From neurochemistry and mitochondrial dysfunction to superorganism
inferred← citeshttps://doi.org/10.1016/j.biosystems.2008.02.002openalex-snapshot
Phosphene phenomenon: A new concept
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Cellular and molecular mechanisms involved in the neurotoxicity of opioid and psychostimulant drugs
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Tempol attenuates cocaine-induced death of PC12 cells through decreased oxidative damage
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Protective effects of SA-31 in a psychostimulant-induced neurotoxicity model using SH-SY5Y cells
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Exposure and coexposure to cocaine and benzoylecgonine at environmentally relevant levels cause long-term oxidative effects in zebrafish
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Relationship between DNA damage measured by the comet-assay and cognitive function
inferred← citeshttps://doi.org/10.1016/j.neubiorev.2008.01.004openalex-snapshot
Neurotoxic profiles of HIV, psychostimulant drugs of abuse, and their concerted effect on the brain: Current status of dopamine system vulnerability in NeuroAIDS
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The role of the habenula in the transition from reward to misery in substance use and mood disorders
inferred← citeshttps://doi.org/10.1016/j.neuint.2009.10.001openalex-snapshot
Cocaine-induced status epilepticus and death generate oxidative stress in prefrontal cortex and striatum of mice
inferred← citeshttps://doi.org/10.1016/j.neuint.2015.12.006openalex-snapshot
Cocaine and mitochondria-related signaling in the brain: A mechanistic view and future directions
inferred← citeshttps://doi.org/10.1016/j.neulet.2018.09.021openalex-snapshot
Association between serum malondialdehyde levels and depression during early methamphetamine withdrawal
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2013.06.034openalex-snapshot
Neurodegeneration of lateral habenula efferent fibers after intermittent cocaine administration: Implications for deep brain stimulation
inferred← citeshttps://doi.org/10.1016/j.physbeh.2025.115012openalex-snapshot
Impairments and recovery of neurocognitive control and cardiac vagal regulation in males with substance use disorders
inferred← citeshttps://doi.org/10.1016/j.pscychresns.2014.01.005openalex-snapshot
Chronic cocaine administration causes extensive white matter damage in brain: Diffusion tensor imaging and immunohistochemistry studies
inferred← citeshttps://doi.org/10.1016/j.reprotox.2007.04.004openalex-snapshot
Embryonic oxidative stress as a mechanism of teratogenesis with special emphasis on diabetic embryopathy
inferred← citeshttps://doi.org/10.1016/j.taap.2015.04.013openalex-snapshot
Enhancement of endocannabinoid signaling protects against cocaine-induced neurotoxicity
inferred← citeshttps://doi.org/10.1016/j.toxlet.2016.06.002openalex-snapshot
The role of the NADPH oxidase derived brain oxidative stress in the cocaine-related death associated with excited delirium: A literature review
inferred← citeshttps://doi.org/10.1016/j.toxrep.2019.11.001openalex-snapshot
Anhydroecgonine methyl ester (AEME), a cocaine pyrolysis product, impairs glutathione-related enzymes response and increases lipid peroxidation in the hippocampal cell culture
inferred← citeshttps://doi.org/10.1017/s1355617712001634openalex-snapshot
Isolating Cognitive and Neurologic HIV Effects in Substance-Dependent, Confounded Cohorts: A Pilot Study
inferred← citeshttps://doi.org/10.1038/s41598-017-11511-7openalex-snapshot
Chronic treatment with Tempol during acquisition or withdrawal from CPP abolishes the expression of cocaine reward and diminishes oxidative damage
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Identification of biochemical and cytotoxic markers in cocaine treated PC12 cells
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Glucagon-like peptide 1 receptor activation regulates cocaine actions and dopamine homeostasis in the lateral septum by decreasing arachidonic acid levels
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The role of mitochondria in cocaine addiction
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Gulf War agents trigger discrete transcriptional changes in human neuronal cells
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Cocaine-mediated microglial activation involves the ER stress-autophagy axis
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Ramelteon protects against human pulmonary microvascular endothelial cell injury induced by lipopolysaccharide (LPS) via activating nuclear factor erythroid 2-related factor 2 (Nrf2)/heme oxygenase-1 (HO-1) pathway
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