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LRP/Amyloid β-Peptide Interaction Mediates Differential Brain Efflux of Aβ Isoforms

https://doi.org/10.1016/j.neuron.2004.07.017
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ana.1009openalex-snapshot
Novel amyloid precursor protein mutation in an Iowa family with dementia and severe cerebral amyloid angiopathy
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Proteolytic processing of the 600 kd low density lipoprotein receptor‐related protein (LRP) occurs in a trans‐Golgi compartment.
inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00623.xopenalex-snapshot
RAP, a specialized chaperone, prevents ligand‐induced ER retention and degradation of LDL receptor‐related endocytic receptors.
inferredcites →https://doi.org/10.1006/exnr.2001.7684openalex-snapshot
Role of Apolipoprotein E Receptors in Regulating the Differential in vivo Neurotrophic Effects of Apolipoprotein E
inferredcites →https://doi.org/10.1007/s004390050980openalex-snapshot
Association between coding variability in the LRP gene and the risk of late-onset Alzheimer's disease
inferredcites →https://doi.org/10.1016/0005-2795(71)90123-1openalex-snapshot
Enzymatic iodination of polypeptides with 125I to high specific activity
inferredcites →https://doi.org/10.1016/0092-8674(95)90320-8openalex-snapshot
LDL receptor-related protein, a multifunctional ApoE receptor, binds secreted β-amyloid precursor protein and mediates its degradation
inferredcites →https://doi.org/10.1016/s0021-9258(18)48529-8openalex-snapshot
Assembly and aggregation properties of synthetic Alzheimer's A4/beta amyloid peptide analogs.
inferredcites →https://doi.org/10.1016/s0074-7696(01)09011-8openalex-snapshot
The roles of receptor-associated protein (RAP) as a molecular chaperone for members of the LDL receptor family
inferredcites →https://doi.org/10.1016/s0092-8674(03)00073-4openalex-snapshot
Coaxing the LDL Receptor Family into the Fold
inferredcites →https://doi.org/10.1016/s0140-6736(05)78749-3openalex-snapshot
Association at LRP gene locus with sporadic late-onset Alzheimer's disease
inferredcites →https://doi.org/10.1016/s0165-0270(03)00206-1openalex-snapshot
A simple method for isolation and characterization of mouse brain microvascular endothelial cells
inferredcites →https://doi.org/10.1016/s0169-409x(02)00149-7openalex-snapshot
Amyloidosis and Alzheimer’s disease
inferredcites →https://doi.org/10.1016/s0197-4580(01)00330-xopenalex-snapshot
FAD mutant PS-1 gene-targeted mice: increased Aβ42 and Aβ deposition without APP overproduction
inferredcites →https://doi.org/10.1016/s0896-6273(01)00234-3openalex-snapshot
The LDL Receptor Gene Family
inferredcites →https://doi.org/10.1016/s0896-6273(01)00475-5openalex-snapshot
Clearing the Brain's Amyloid Cobwebs
inferredcites →https://doi.org/10.1016/s0896-6273(04)00019-4openalex-snapshot
Activated Protein C Prevents Neuronal Apoptosis via Protease Activated Receptors 1 and 3
inferredcites →https://doi.org/10.1016/s1050-1738(02)00154-8openalex-snapshot
Phosphorylation of LRP1 Regulation of Transport and Signal Transduction
inferredcites →https://doi.org/10.1038/416535aopenalex-snapshot
Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo
inferredcites →https://doi.org/10.1038/77397openalex-snapshot
Clearance of amyloid β-peptide from brain: transport or metabolism?
inferredcites →https://doi.org/10.1038/nm0402-403openalex-snapshot
Hematopoietic stem cells differentiate into vascular cells that participate in the pathogenesis of atherosclerosis
inferredcites →https://doi.org/10.1038/nm826openalex-snapshot
Activated protein C blocks p53-mediated apoptosis in ischemic human brain endothelium and is neuroprotective
inferredcites →https://doi.org/10.1038/nm890openalex-snapshot
RAGE mediates amyloid-β peptide transport across the blood-brain barrier and accumulation in brain
inferredcites →https://doi.org/10.1038/nm926openalex-snapshot
Lipoprotein receptor–mediated induction of matrix metalloproteinase by tissue plasminogen activator
inferredcites →https://doi.org/10.1046/j.1471-4159.1997.69051904.xopenalex-snapshot
α2‐Macroglobulin Complexes with and Mediates the Endocytosis of β‐Amyloid Peptide via Cell Surface Low‐Density Lipoprotein Receptor‐Related Protein
inferredcites →https://doi.org/10.1046/j.1471-4159.1999.0731393.xopenalex-snapshot
α2‐Macroglobulin Enhances the Clearance of Endogenous Soluble β‐Amyloid Peptide via Low‐Density Lipoprotein Receptor‐Related Protein in Cortical Neuron
inferredcites →https://doi.org/10.1046/j.1471-4159.2002.01009.xopenalex-snapshot
Low‐density lipoprotein receptor‐related protein levels and endocytic function are reduced by overexpression of the FE65 adaptor protein, FE65L1
inferredcites →https://doi.org/10.1073/pnas.0305803101openalex-snapshot
Increased soluble amyloid-β peptide and memory deficits in amyloid model mice overexpressing the low-density lipoprotein receptor-related protein
inferredcites →https://doi.org/10.1073/pnas.162228299openalex-snapshot
Clusterin promotes amyloid plaque formation and is critical for neuritic toxicity in a mouse model of Alzheimer's disease
inferredcites →https://doi.org/10.1073/pnas.1834302100openalex-snapshot
Alzheimer's disease-affected brain: Presence of oligomeric Aβ ligands (ADDLs) suggests a molecular basis for reversible memory loss
inferredcites →https://doi.org/10.1073/pnas.93.9.4229openalex-snapshot
Glycoprotein 330/megalin: probable role in receptor-mediated transport of apolipoprotein J alone and in a complex with Alzheimer disease amyloid beta at the blood-brain and blood-cerebrospinal fluid barriers.
inferredcites →https://doi.org/10.1074/jbc.271.18.10602openalex-snapshot
The Interaction between Apolipoprotein E and Alzheimer's Amyloid β-Peptide Is Dependent on β-Peptide Conformation
inferredcites →https://doi.org/10.1074/jbc.275.10.7410openalex-snapshot
Modulation of β-Amyloid Precursor Protein Processing by the Low Density Lipoprotein Receptor-related Protein (LRP)
inferredcites →https://doi.org/10.1074/jbc.m101589200openalex-snapshot
Differential Functions of Members of the Low Density Lipoprotein Receptor Family Suggested by Their Distinct Endocytosis Rates
inferredcites →https://doi.org/10.1074/jbc.m102293200openalex-snapshot
The α-Chains of C4b-binding Protein Mediate Complex Formation with Low Density Lipoprotein Receptor-related Protein
inferredcites →https://doi.org/10.1074/jbc.m104135200openalex-snapshot
Pathogenic Effects of D23N Iowa Mutant Amyloid β-Protein
inferredcites →https://doi.org/10.1074/jbc.m201750200openalex-snapshot
Oligomeric and Fibrillar Species of Amyloid-β Peptides Differentially Affect Neuronal Viability
inferredcites →https://doi.org/10.1074/jbc.m312946200openalex-snapshot
Early-onset and Robust Cerebral Microvascular Accumulation of Amyloid β-Protein in Transgenic Mice Expressing Low Levels of a Vasculotropic Dutch/Iowa Mutant Form of Amyloid β-Protein Precursor
inferredcites →https://doi.org/10.1091/mbc.e02-03-0152openalex-snapshot
Proteasome Regulates the Delivery of LDL Receptor-related Protein into the Degradation Pathway
inferredcites →https://doi.org/10.1093/emboj/cdf568openalex-snapshot
The cytoplasmic domain of the LDL receptor‐related protein regulates multiple steps in APP processing
inferredcites →https://doi.org/10.1097/00041433-200210000-00013openalex-snapshot
Genetics and molecular biology: phosphorylation of low-density lipoprotein receptor-related protein by platelet-derived growth factor
inferredcites →https://doi.org/10.1126/science.1067568openalex-snapshot
Brain to Plasma Amyloid-β Efflux: a Measure of Brain Amyloid Burden in a Mouse Model of Alzheimer's Disease
inferredcites →https://doi.org/10.1126/science.1072994openalex-snapshot
The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics
inferredcites →https://doi.org/10.1126/science.1079469openalex-snapshot
Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis
inferredcites →https://doi.org/10.1126/science.2111584openalex-snapshot
Mutation of the Alzheimer's Disease Amyloid Gene in Hereditary Cerebral Hemorrhage, Dutch Type
inferredcites →https://doi.org/10.1126/science.274.5284.99openalex-snapshot
Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic Mice
inferredcites →https://doi.org/10.1172/jci10498openalex-snapshot
Clearance of Alzheimer’s amyloid-β1-40 peptide from brain by LDL receptor–related protein-1 at the blood-brain barrier
inferredcites →https://doi.org/10.1172/jci11013openalex-snapshot
Modulation of amyloid β-protein clearance and Alzheimer’s disease susceptibility by the LDL receptor–related protein pathway
inferredcites →https://doi.org/10.1172/jci13992openalex-snapshot
LRP: a multifunctional scavenger and signaling receptor
inferredcites →https://doi.org/10.1172/jci2029openalex-snapshot
Human blood-brain barrier receptors for Alzheimer's amyloid-beta 1- 40. Asymmetrical binding, endocytosis, and transcytosis at the apical side of brain microvascular endothelial cell monolayer.
inferredcites →https://doi.org/10.1212/wnl.49.1.56openalex-snapshot
Genetic association of the low-density lipoprotein receptor-related protein gene (LRP), and apolipoprotein E receptor, with late-onset Alzheimer's disease
inferredcites →https://doi.org/10.1212/wnl.50.6.1905openalex-snapshot
Confirmation of an association between a polymorphism in exon 3 of the low‐density lipoprotein receptor‐related protein gene and Alzheimer's disease
inferredcites →https://doi.org/10.1523/jneurosci.18-01-00195.1998openalex-snapshot
Isoform-specific effect of apolipoprotein E on cell survival and beta-amyloid-induced toxicity in rat hippocampal pyramidal neuronal cultures.
inferredcites →https://doi.org/10.1523/jneurosci.21-02-00372.2001openalex-snapshot
Age-Dependent Changes in Brain, CSF, and Plasma Amyloid β Protein in the Tg2576 Transgenic Mouse Model of Alzheimer's Disease
inferredcites →https://doi.org/10.1523/jneurosci.22-21-09298.2002openalex-snapshot
Increased Extracellular Amyloid Deposition and Neurodegeneration in Human Amyloid Precursor Protein Transgenic Mice Deficient in Receptor-Associated Protein
inferredcites →https://doi.org/10.1523/jneurosci.23-01-00029.2003openalex-snapshot
Novel Therapeutic Approach for the Treatment of Alzheimer's Disease by Peripheral Administration of Agents with an Affinity to β-Amyloid
inferredcites →https://doi.org/10.1523/jneurosci.23-26-08844.2003openalex-snapshot
In vivo assessment of brain interstitial fluid with microdialysis reveals plaque-associated changes in amyloid-beta metabolism and half-life.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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The case for blood–brain barrier dysfunction in the pathogenesis of Alzheimer's disease
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From the liver to the blood–brain barrier: An interconnected system regulating brain amyloid‐β levels
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Immunohistochemical study of semicarbazide‐sensitive amine oxidase/vascular adhesion protein‐1 in the hippocampal vasculature: Pathological synergy of Alzheimer's disease and diabetes mellitus
inferred← citeshttps://doi.org/10.1002/jnr.23964openalex-snapshot
Limitations of the hCMEC/D3 cell line as a model for Aβ clearance by the human blood‐brain barrier
inferred← citeshttps://doi.org/10.1002/pmic.201600439openalex-snapshot
Plasma Membrane Protein Profiling in Beta‐Amyloid‐Treated Microglia Cell Line
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Quantitative systems pharmacology model of the amyloid pathway in Alzheimer's disease: Insights into the therapeutic mechanisms of clinical candidates
inferred← citeshttps://doi.org/10.1002/syn.20644openalex-snapshot
Upregulation of RAGE at the blood‐brain barrier in streptozotocin‐induced diabetic mice
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Apolipoprotein E receptor pathways in Alzheimer disease
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Metabolite Clearance During Wakefulness and Sleep
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The Blood Brain Barrier
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CNS Drug Delivery: Opioid Peptides and the Blood-Brain Barrier
inferred← citeshttps://doi.org/10.1007/978-1-0716-2289-6_20openalex-snapshot
An In Vitro Model of the Blood–Brain Barrier to Study Alzheimer’s Disease: The Role of β-Amyloid and Its Influence on PBMC Infiltration
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inferred← citeshttps://doi.org/10.1007/978-1-4614-8313-7_10openalex-snapshot
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Disease Influence on BBB Transport in Inflammatory Disorders
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Targeted Chemotherapy in Drug-Resistant Tumors, Noninvasive Imaging of P-Glycoprotein-Mediated Functional Transport in Cancer, and Emerging Role of Pgp in Neurodegenerative Diseases
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The Blood–Brain Barriers
inferred← citeshttps://doi.org/10.1007/978-3-031-80849-4_12openalex-snapshot
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The Blood-Brain Barriers
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The Relationship Between the Continuum of Elevated Adiposity, Hyperinsulinemia, and Type 2 Diabetes and Late-onset Alzheimer’s Disease: An Epidemiological Perspective
inferred← citeshttps://doi.org/10.1007/978-4-431-55763-0_19openalex-snapshot
Catabolism and Anabolism of Amyloid-β
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Modes of Aβ toxicity in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00018-017-2463-7openalex-snapshot
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PCSK9 acts as a key regulator of Aβ clearance across the blood–brain barrier
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The role of lipoprotein receptors on the physiological function of APP
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Relative paucity of tau accumulation in the small areas with abundant Aβ42-positive capillary amyloid angiopathy within a given cortical region in the brain of patients with Alzheimer pathology
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Neurovascular mechanisms and blood–brain barrier disorder in Alzheimer’s disease
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The overlap between neurodegenerative and vascular factors in the pathogenesis of dementia
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inferred← citeshttps://doi.org/10.1007/s11357-025-01657-yopenalex-snapshot
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inferred← citeshttps://doi.org/10.1007/s12017-008-8035-0openalex-snapshot
Maximizing the Potential of Plasma Amyloid-Beta as a Diagnostic Biomarker for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12017-017-8446-xopenalex-snapshot
Low-Density Lipoprotein Receptor-Related Protein-1 (LRP1) C4408R Mutant Promotes Amyloid Precursor Protein (APP) α-Cleavage in Vitro
inferred← citeshttps://doi.org/10.1007/s12031-025-02339-2openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/s12035-007-0029-7openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/s12035-009-8092-xopenalex-snapshot
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inferred← citeshttps://doi.org/10.1007/s12035-014-8749-yopenalex-snapshot
The Role of Cholesterol Metabolism in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-015-9319-7openalex-snapshot
Dissecting the Contribution of Vascular Alterations and Aging to Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-017-0806-xopenalex-snapshot
Trafficking in Alzheimer’s Disease: Modulation of APP Transport and Processing by the Transmembrane Proteins LRP1, SorLA, SorCS1c, Sortilin, and Calsyntenin
inferred← citeshttps://doi.org/10.1007/s12035-018-0896-0openalex-snapshot
Magnesium Reduces Blood-Brain Barrier Permeability and Regulates Amyloid-β Transcytosis
inferred← citeshttps://doi.org/10.1007/s12035-020-01982-7openalex-snapshot
Silencing of LRP1 Exacerbates Inflammatory Response Via TLR4/NF-κB/MAPKs Signaling Pathways in APP/PS1 Transgenic Mice
inferred← citeshttps://doi.org/10.1007/s12035-021-02379-wopenalex-snapshot
Biphasic Effects of Ethanol Exposure on Waste Metabolites Clearance in the CNS
inferred← citeshttps://doi.org/10.1007/s12035-026-06066-6openalex-snapshot
The Clearance-Centered Bottleneck in Alzheimer’s Disease: From Coupled Glymphatic–Lymphatic Circuits to Therapeutic Opportunities
inferred← citeshttps://doi.org/10.1007/s12192-014-0566-8openalex-snapshot
Evidence against a role of P-glycoprotein in the clearance of the Alzheimer’s disease Aβ1–42 peptides
inferred← citeshttps://doi.org/10.1007/s12264-013-1425-9openalex-snapshot
Environmental factors in the development and progression of late-onset Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s12264-020-00548-wopenalex-snapshot
Quantitative Determination of Glymphatic Flow Using Spectrophotofluorometry
inferred← citeshttps://doi.org/10.1007/s12640-017-9784-zopenalex-snapshot
RAGE-NF-κB-PPARγ Signaling is Involved in AGEs-Induced Upregulation of Amyloid-β Influx Transport in an In Vitro BBB Model
inferred← citeshttps://doi.org/10.1007/s12975-011-0125-xopenalex-snapshot
Blood–Brain Barrier Pathophysiology in Traumatic Brain Injury
inferred← citeshttps://doi.org/10.1007/s12975-012-0185-6openalex-snapshot
Mixed Cerebrovascular Disease and the Future of Stroke Prevention
inferred← citeshttps://doi.org/10.1007/s12975-013-0309-7openalex-snapshot
Alzheimer’s Silent Partner: Cerebral Amyloid Angiopathy
inferred← citeshttps://doi.org/10.1007/s13760-024-02706-7openalex-snapshot
Brain insulin resistance mediated cognitive impairment and neurodegeneration: Type-3 diabetes or Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-394596-9.00002-0openalex-snapshot
Genetic Animal Models of Cerebral Vasculopathies
inferred← citeshttps://doi.org/10.1016/b978-0-12-417042-1.00002-4openalex-snapshot
Signaling Pathways Involved in Cognitive Enhancement
inferred← citeshttps://doi.org/10.1016/b978-0-12-800101-1.00013-2openalex-snapshot
Impaired Insulin Signaling and Mechanisms of Memory Loss
inferred← citeshttps://doi.org/10.1016/b978-0-12-802173-6.00009-5openalex-snapshot
Blood–Brain Barrier Transport of Alzheimer’s Amyloid β-Peptide
inferred← citeshttps://doi.org/10.1016/b978-0-12-809937-7.00001-xopenalex-snapshot
Neurochemical Aspects of β-Amyloid Cascade Hypothesis for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-809937-7.00002-1openalex-snapshot
Risk Factors for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-813866-3.00001-1openalex-snapshot
Genomics of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-816412-9.00005-7openalex-snapshot
Blood-Brain Barrier–Targeted Nanotechnological Advances
inferred← citeshttps://doi.org/10.1016/b978-0-323-89835-5.00035-1openalex-snapshot
Linking copper and neurotoxic activities to the CNS
inferred← citeshttps://doi.org/10.1016/b978-0-323-95735-9.00031-0openalex-snapshot
An overview on pathophysiology and therapeutic approaches of Alzheimer's disease and Parkinson's disease
inferred← citeshttps://doi.org/10.1016/b978-0-443-19176-3.00035-2openalex-snapshot
Genomics of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/b978-0-443-28979-8.00008-5openalex-snapshot
Metabolism of Aβ: Catabolic and glymphatic systems
inferred← citeshttps://doi.org/10.1016/b978-008045046-9.00491-5openalex-snapshot
Blood–Brain Barrier and Neurovascular Mechanisms of Neurodegeneration and Injury
inferred← citeshttps://doi.org/10.1016/bs.apha.2014.06.004openalex-snapshot
Delivery of Therapeutic Peptides and Proteins to the CNS
inferred← citeshttps://doi.org/10.1016/bs.ircmb.2017.07.002openalex-snapshot
New Insights Into Autophagy Dysfunction Related to Amyloid Beta Toxicity and Neuropathology in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1016/bs.irn.2021.06.009openalex-snapshot
Alcohol, inflammation, and blood-brain barrier function in health and disease across development
inferred← citeshttps://doi.org/10.1016/bs.vh.2024.04.004openalex-snapshot
A circadian clock regulates the blood-brain barrier across phylogeny
inferred← citeshttps://doi.org/10.1016/j.addr.2011.12.005openalex-snapshot
Drug delivery to the brain in Alzheimer's disease: Consideration of the blood–brain barrier
inferred← citeshttps://doi.org/10.1016/j.ajpath.2013.01.021openalex-snapshot
The Bradykinin B1 Receptor Regulates Aβ Deposition and Neuroinflammation in Tg-SwDI Mice
inferred← citeshttps://doi.org/10.1016/j.apsb.2020.11.023openalex-snapshot
Evolution of blood–brain barrier in brain diseases and related systemic nanoscale brain-targeting drug delivery strategies
inferred← citeshttps://doi.org/10.1016/j.apsb.2024.05.017openalex-snapshot
Counteracting Alzheimer's disease via normalizing neurovascular unit with a self-regulated multi-functional nano-modulator
inferred← citeshttps://doi.org/10.1016/j.apsb.2024.06.013openalex-snapshot
Amylovis-201 is a new dual-target ligand, acting as an anti-amyloidogenic compound and a potent agonist of the σ1 chaperone protein
inferred← citeshttps://doi.org/10.1016/j.arr.2016.11.003openalex-snapshot
Disordered APP metabolism and neurovasculature in trauma and aging: Combined risks for chronic neurodegenerative disorders
inferred← citeshttps://doi.org/10.1016/j.arr.2021.101451openalex-snapshot
Abnormal amyloid beta metabolism in systemic abnormalities and Alzheimer’s pathology: Insights and therapeutic approaches from periphery
inferred← citeshttps://doi.org/10.1016/j.arr.2024.102446openalex-snapshot
Senescence-associated microvascular endothelial dysfunction: A focus on the blood-brain and blood-retinal barriers
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103392openalex-snapshot
A silent RAGE: The inflammatory pathway to dementia in Parkinson’s disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2009.10.011openalex-snapshot
Mouse models of neurological disorders: A view from the blood–brain barrier
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.08.024openalex-snapshot
Deposition of amyloid β in the walls of human leptomeningeal arteries in relation to perivascular drainage pathways in cerebral amyloid angiopathy
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.10.020openalex-snapshot
The influence of the amyloid ß-protein and its precursor in modulating cerebral hemostasis
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.11.010openalex-snapshot
Vascular mTOR-dependent mechanisms linking the control of aging to Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.12.016openalex-snapshot
Neurovascular dysfunction and neurodegeneration in dementia and Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2016.01.013openalex-snapshot
Role of P-glycoprotein in mediating rivastigmine effect on amyloid-β brain load and related pathology in Alzheimer's disease mouse model
inferred← citeshttps://doi.org/10.1016/j.bbadis.2019.04.016openalex-snapshot
Blood-brain barrier dysfunction underlying Alzheimer's disease is induced by an SSAO/VAP-1-dependent cerebrovascular activation with enhanced Aβ deposition
inferred← citeshttps://doi.org/10.1016/j.bbadis.2019.04.019openalex-snapshot
Astaxanthin exerts protective effects similar to bexarotene in Alzheimer's disease by modulating amyloid-beta and cholesterol homeostasis in blood-brain barrier endothelial cells
inferred← citeshttps://doi.org/10.1016/j.bbadis.2023.166729openalex-snapshot
Sleep deprivation modulates APOE and LDL receptor-related protein 1 through thyroid hormone T4 and impairs Aβ clearance in hippocampus of rats
inferred← citeshttps://doi.org/10.1016/j.bbadis.2023.166967openalex-snapshot
Blood-brain barrier transporters: An overview of function, dysfunction in Alzheimer's disease and strategies for treatment
inferred← citeshttps://doi.org/10.1016/j.bbalip.2010.02.007openalex-snapshot
Formation and function of apolipoprotein E-containing lipoproteins in the nervous system
inferred← citeshttps://doi.org/10.1016/j.bbalip.2010.03.011openalex-snapshot
Brain cholesterol in normal and pathological aging
inferred← citeshttps://doi.org/10.1016/j.bbalip.2022.159123openalex-snapshot
Emerging role of HDL in brain cholesterol metabolism and neurodegenerative disorders
inferred← citeshttps://doi.org/10.1016/j.bbamem.2008.10.022openalex-snapshot
The blood-brain barrier in brain homeostasis and neurological diseases
inferred← citeshttps://doi.org/10.1016/j.bbamem.2017.07.001openalex-snapshot
Fatty acid-based lipidomics and membrane remodeling induced by apoE3 and apoE4 in human neuroblastoma cells
inferred← citeshttps://doi.org/10.1016/j.bbi.2009.01.017openalex-snapshot
Lipopolysaccharide alters the blood–brain barrier transport of amyloid β protein: A mechanism for inflammation in the progression of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.bbi.2014.08.007openalex-snapshot
The blood-brain barrier in neuroimmunology: Tales of separation and assimilation
inferred← citeshttps://doi.org/10.1016/j.bbi.2018.03.007openalex-snapshot
Cognitive benefits of lithium chloride in APP/PS1 mice are associated with enhanced brain clearance of β-amyloid
inferred← citeshttps://doi.org/10.1016/j.bbi.2018.08.016openalex-snapshot
Commentary on the 2018 Named Series on blood-brain interfaces: Roles of neuroimmunomodulation in health and disease
inferred← citeshttps://doi.org/10.1016/j.bbrc.2008.05.045openalex-snapshot
Clearance and deposition of extracellular α-synuclein aggregates in microglia
inferred← citeshttps://doi.org/10.1016/j.bbrc.2009.08.020openalex-snapshot
MicroRNA-205 inhibits tumor cell migration through down-regulating the expression of the LDL receptor-related protein 1
inferred← citeshttps://doi.org/10.1016/j.bcp.2024.116643openalex-snapshot
The LDL Receptor-Related Protein 1: Mechanisms and roles in promoting Aβ efflux transporter in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.biocel.2014.04.021openalex-snapshot
LRP1-dependent pepsin clearance induced by 2′-hydroxycinnamaldehyde attenuates breast cancer cell invasion
inferred← citeshttps://doi.org/10.1016/j.biomag.2012.03.006openalex-snapshot
Aβ1-40 clearance from the brain by regulation of LRP-1: A study on effects of insulin, rosiglitazone or the combination in type 2 diabetic GK rats
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2014.03.082openalex-snapshot
Protection of SK-N-MC cells against β-amyloid peptide-induced degeneration using neuron growth factor-loaded liposomes with surface lactoferrin
inferred← citeshttps://doi.org/10.1016/j.biopha.2020.110887openalex-snapshot
Melatonin regulates Aβ production/clearance balance and Aβ neurotoxicity: A potential therapeutic molecule for Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.117575openalex-snapshot
The potential role of transcription factor sterol regulatory element binding proteins (SREBPs) in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.brainres.2006.07.112openalex-snapshot
Aluminum complexing enhances amyloid β protein penetration of blood–brain barrier
inferred← citeshttps://doi.org/10.1016/j.brainres.2006.08.064openalex-snapshot
A rat homologue of CED-6 is expressed in neurons and interacts with clathrin
inferred← citeshttps://doi.org/10.1016/j.brainres.2014.10.047openalex-snapshot
In vitro discrimination of the role of LRP1 at the BBB cellular level: Focus on brain capillary endothelial cells and brain pericytes
inferred← citeshttps://doi.org/10.1016/j.brainres.2015.04.056openalex-snapshot
Glio-vascular changes during ageing in wild-type and Alzheimer׳s disease-like APP/PS1 mice
inferred← citeshttps://doi.org/10.1016/j.brainres.2019.146379openalex-snapshot
Chronic cerebral hypoperfusion alters amyloid-β transport related proteins in the cortical blood vessels of Alzheimer’s disease model mouse
inferred← citeshttps://doi.org/10.1016/j.brainres.2023.148681openalex-snapshot
Effects of DHA (omega-3 fatty acid) and estradiol on amyloid β-peptide regulation in the brain
inferred← citeshttps://doi.org/10.1016/j.brainres.2024.149205openalex-snapshot
Recent updates on immunotherapy in neurodegenerative diseases
inferred← citeshttps://doi.org/10.1016/j.brainresbull.2016.04.019openalex-snapshot
Physiological and pathological roles of the γ-secretase complex
inferred← citeshttps://doi.org/10.1016/j.brainresbull.2020.03.018openalex-snapshot
Revisiting the blood-brain barrier: A hard nut to crack in the transportation of drug molecules
inferred← citeshttps://doi.org/10.1016/j.cccb.2022.100133openalex-snapshot
rTg-D: A novel transgenic rat model of cerebral amyloid angiopathy Type-2
inferred← citeshttps://doi.org/10.1016/j.cell.2005.02.008openalex-snapshot
Twenty Years of the Alzheimer’s Disease Amyloid Hypothesis: A Genetic Perspective
inferred← citeshttps://doi.org/10.1016/j.cell.2015.10.067openalex-snapshot
Establishment and Dysfunction of the Blood-Brain Barrier
inferred← citeshttps://doi.org/10.1016/j.celrep.2020.108447openalex-snapshot
Reduction of mNAT1/hNAT2 Contributes to Cerebral Endothelial Necroptosis and Aβ Accumulation in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/j.chembiol.2014.09.008openalex-snapshot
The Membrane-Lytic Peptides K8L9 and Melittin Enter Cancer Cells via Receptor Endocytosis following Subcytotoxic Exposure
inferred← citeshttps://doi.org/10.1016/j.conb.2018.12.014openalex-snapshot
Transcytosis at the blood–brain barrier
inferred← citeshttps://doi.org/10.1016/j.cyto.2008.08.013openalex-snapshot
Diagnostic utility of APOE, soluble CD40, CD40L, and Aβ1–40 levels in plasma in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.ddtec.2016.07.002openalex-snapshot
Altered blood–brain barrier transport in neuro-inflammatory disorders
inferred← citeshttps://doi.org/10.1016/j.drudis.2006.08.004openalex-snapshot
Clearance of amyloid-beta in Alzheimer's disease: progress, problems and perspectives
inferred← citeshttps://doi.org/10.1016/j.drudis.2008.12.004openalex-snapshot
Monitoring the amyloid beta-peptide in vivo – caveat emptor
inferred← citeshttps://doi.org/10.1016/j.ejmech.2013.03.014openalex-snapshot
Analysis of the antioxidant properties of differently substituted 2- and 3-indolyl carbohydrazides and related derivatives
inferred← citeshttps://doi.org/10.1016/j.endend.2013.12.017openalex-snapshot
10.1016/j.endend.2013.12.017
inferred← citeshttps://doi.org/10.1016/j.endend.2014.01.021openalex-snapshot
10.1016/j.endend.2014.01.021
inferred← citeshttps://doi.org/10.1016/j.endend.2014.01.022openalex-snapshot
10.1016/j.endend.2014.01.022
inferred← citeshttps://doi.org/10.1016/j.exger.2009.09.008openalex-snapshot
Biomarkers for Alzheimer’s disease and other forms of dementia: Clinical needs, limitations and future aspects
inferred← citeshttps://doi.org/10.1016/j.expneurol.2006.05.019openalex-snapshot
Blood-borne factors inhibit Alzheimer's β-amyloid fibril formation in vitro
inferred← citeshttps://doi.org/10.1016/j.expneurol.2009.04.025openalex-snapshot
Transport of prion protein across the blood–brain barrier
inferred← citeshttps://doi.org/10.1016/j.expneurol.2010.05.017openalex-snapshot
The role of low-density receptor-related protein 1 (LRP1) as a competitive substrate of the amyloid precursor protein (APP) for BACE1
inferred← citeshttps://doi.org/10.1016/j.expneurol.2020.113257openalex-snapshot
Brain injury-induced dysfunction of the blood brain barrier as a risk for dementia
inferred← citeshttps://doi.org/10.1016/j.expneurol.2025.115149openalex-snapshot
Cellular expression of low-density lipoprotein receptor-related protein 1 and amyloid beta deposition in human and rat epileptogenic brain
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2019.07.029openalex-snapshot
Alcohol promotes waste clearance in the CNS via brain vascular reactivity
inferred← citeshttps://doi.org/10.1016/j.gene.2016.10.022openalex-snapshot
Integrated late onset Alzheimer's disease (LOAD) susceptibility genes: Cholesterol metabolism and trafficking perspectives
inferred← citeshttps://doi.org/10.1016/j.ijpharm.2023.122614openalex-snapshot
Strategic nanocarriers to control neurodegenerative disorders: Concept, challenges, and future perspective
inferred← citeshttps://doi.org/10.1016/j.jagp.2012.08.017openalex-snapshot
Angiotensin Converting Enzyme Inhibitors and Alzheimer Disease in the Presence of the Apolipoprotein E4 Allele
inferred← citeshttps://doi.org/10.1016/j.jagp.2019.06.011openalex-snapshot
Apolipoprotein E ε4 Allele is Associated With Plasma Amyloid Beta and Amyloid Beta Transporter Levels: A Cross-sectional Study in a Rural Area of Xi'an, China
inferred← citeshttps://doi.org/10.1016/j.jalz.2006.09.002openalex-snapshot
The enigma of mixed dementia
inferred← citeshttps://doi.org/10.1016/j.jalz.2010.06.004openalex-snapshot
Biomarkers in Alzheimer's disease drug development
inferred← citeshttps://doi.org/10.1016/j.jalz.2010.12.005openalex-snapshot
Butyrylcholinesterase genotype and gender influence Alzheimer's disease phenotype
inferred← citeshttps://doi.org/10.1016/j.jalz.2014.10.008openalex-snapshot
Vascular contributions to cognitive impairment and dementia including Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.jalz.2017.06.2266openalex-snapshot
Amyloid β concentrations and stable isotope labeling kinetics of human plasma specific to central nervous system amyloidosis
inferred← citeshttps://doi.org/10.1016/j.jalz.2018.10.006openalex-snapshot
Quantitative proteomics reveals distinct composition of amyloid plaques in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.jbc.2021.100370openalex-snapshot
Hepatic LDL receptor-related protein-1 deficiency alters mitochondrial dynamics through phosphatidylinositol 4,5-bisphosphate reduction
inferred← citeshttps://doi.org/10.1016/j.jconrel.2018.10.034openalex-snapshot
Boosting RNAi therapy for orthotopic glioblastoma with nontoxic brain-targeting chimaeric polymersomes
inferred← citeshttps://doi.org/10.1016/j.jconrel.2022.02.015openalex-snapshot
Recent advances in Bio-conjugated nanocarriers for crossing the Blood-Brain Barrier in (pre-)clinical studies with an emphasis on vesicles
inferred← citeshttps://doi.org/10.1016/j.jconrel.2024.01.047openalex-snapshot
Alzheimer's disease current therapies, novel drug delivery systems and future directions for better disease management
inferred← citeshttps://doi.org/10.1016/j.jconrel.2025.114255openalex-snapshot
ApoE-Corona oncolytic adenovirus nanoparticles enable blood–brain barrier penetration for glioblastoma immunotherapy
inferred← citeshttps://doi.org/10.1016/j.jddst.2023.104174openalex-snapshot
Blood brain barrier-on-a-chip to model neurological diseases
inferred← citeshttps://doi.org/10.1016/j.jff.2021.104644openalex-snapshot
Lycopene can modulate the LRP1 and RAGE transporters expression at the choroid plexus in Alzheimer’s disease rat
inferred← citeshttps://doi.org/10.1016/j.jinorgbio.2007.04.013openalex-snapshot
Metalloprobes: Synthesis, characterization, and potency of a novel gallium(III) complex in human epidermal carcinoma cells
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