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Clinicopathological and prognostic significance of C/EBP homologous protein (CHOP) in advanced gastric cancer
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1 of the 27 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-28): 10.1007/s00418-012-0967-7 (notice 2021-07-01).
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dataset doi:10.7910/DVN/WT8VOA (CC BY 4.0).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s10549-014-2967-xopenalex-snapshotThe endoplasmic reticulum stress markers GRP78 and CHOP predict disease-free survival and responsiveness to chemotherapy in breast cancer
inferredcites →https://doi.org/10.1016/j.clinre.2014.11.010openalex-snapshotDown-regulation of C/EBP homologous protein (CHOP) expression in gastric cardia adenocarcinoma: Their relationship with clinicopathological parameters and prognostic significance
inferredcites →https://doi.org/10.1016/j.ygyno.2012.11.024openalex-snapshotThe endoplasmic reticulum stress marker, glucose-regulated protein-78 (GRP78) in visceral adipocytes predicts endometrial cancer progression and patient survival
inferredcites →https://doi.org/10.1038/srep03219openalex-snapshotZebularine inhibits tumorigenesis and stemness of colorectal cancer via p53-dependent endoplasmic reticulum stress
inferredcites →https://doi.org/10.1038/srep38369openalex-snapshotrpL3 promotes the apoptosis of p53 mutated lung cancer cells by down-regulating CBS and NFκB upon 5-FU treatment
inferredcites →https://doi.org/10.1128/aac.00070-14openalex-snapshotColistin-Induced Nephrotoxicity in Mice Involves the Mitochondrial, Death Receptor, and Endoplasmic Reticulum Pathways
inferredcites →https://doi.org/10.18632/oncotarget.11321openalex-snapshotThe mitochondrial and endoplasmic reticulum pathways involved in the apoptosis of bursa of Fabricius cells in broilers exposed to dietary aflatoxin B1
inferredcites →https://doi.org/10.18632/oncotarget.2727openalex-snapshotCarnosic acid sensitized TRAIL-mediated apoptosis through down-regulation of c-FLIP and Bcl-2 expression at the post translational levels and CHOP-dependent up-regulation of DR5, Bim, and PUMA expression in human carcinoma caki cells
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