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Ascorbic Acid Reprograms Epigenome and Epitranscriptome by Reducing Fe(III) in the Catalytic Cycle of Dioxygenases

https://doi.org/10.1021/acschembio.3c00567

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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A DFT Study of Nucleobase Dealkylation by the DNA Repair Enzyme AlkB

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N6-methyladenosine-dependent regulation of messenger RNA stability

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Structure of a Naegleria Tet-like dioxygenase in complex with 5-methylcytosine DNA

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Structural insight into substrate preference for TET-mediated oxidation

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N6-Methyladenosine in nuclear RNA is a major substrate of the obesity-associated FTO

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Molecular mechanism for vitamin C-derived C5-glyceryl-methylcytosine DNA modification catalyzed by algal TET homologue CMD1

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m6A RNA modifications are measured at single-base resolution across the mammalian transcriptome

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Absolute quantification of single-base m6A methylation in the mammalian transcriptome using GLORI

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Ascorbic acid induces global epigenetic reprogramming to promote meiotic maturation and developmental competence of porcine oocytes

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Human ABH3 structure and key residues for oxidative demethylation to reverse DNA/RNA damage

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STAR: ultrafast universal RNA-seq aligner

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featureCounts: an efficient general purpose program for assigning sequence reads to genomic features

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Trimmomatic: a flexible trimmer for Illumina sequence data

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Conversion of 5-Methylcytosine to 5-Hydroxymethylcytosine in Mammalian DNA by MLL Partner TET1

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Tet Proteins Can Convert 5-Methylcytosine to 5-Formylcytosine and 5-Carboxylcytosine

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Tet-Mediated Formation of 5-Carboxylcytosine and Its Excision by TDG in Mammalian DNA

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RNA modifications modulate gene expression during development

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Regulation of the Epigenome by Vitamin C

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Vitamin C Sensitizes Melanoma to BET Inhibitors

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What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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