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Targeting the r(CGG) Repeats That Cause FXTAS with Modularly Assembled Small Molecules and Oligonucleotides
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Outgoing claims: the papers this work cites, builds on, or argues with.
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Sam68 sequestration and partial loss of function are associated with splicing alterations in FXTAS patients
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Design and Analysis of Effects of Triplet Repeat Oligonucleotides in Cell Models for Myotonic Dystrophy
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Disruption of Splicing Regulated by a CUG-Binding Protein in Myotonic Dystrophy
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RNA-MEDIATED NEUROMUSCULAR DISORDERS
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The Medicinal Chemistry of Oligonucleotides
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Intention tremor, parkinsonism, and generalized brain atrophy in male carriers of fragile X
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What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Advances on the Mechanisms and Therapeutic Strategies in Non-coding CGG Repeat Expansion Diseases
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Molecular Pathophysiology of Fragile X-Associated Tremor/Ataxia Syndrome and Perspectives for Drug Development
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