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Linking a genetic defect to its cellular phenotype in a cardiac arrhythmia
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1016/s0006-3495(90)82560-1openalex-snapshot
Global parameter optimization for cardiac potassium channel gating models
inferredcites →https://doi.org/10.1016/s0006-3495(95)80271-7openalex-snapshot
Early afterdepolarizations in cardiac myocytes: mechanism and rate dependence
inferredcites →https://doi.org/10.1016/s0006-3495(95)80358-9openalex-snapshot
A mutation in the pore of the sodium channel alters gating
inferredcites →https://doi.org/10.1038/376683a0openalex-snapshot
Molecular mechanism for an inherited cardiac arrhythmia
inferredcites →https://doi.org/10.1073/pnas.93.23.13200openalex-snapshot
Characterization of human cardiac Na + channel mutations in the congenital long QT syndrome
inferredcites →https://doi.org/10.1113/jphysiol.1952.sp004764openalex-snapshot
A quantitative description of membrane current and its application to conduction and excitation in nerve
inferredcites →https://doi.org/10.1113/jphysiol.1995.sp020754openalex-snapshot
Inactivation of the cardiac Na+ channels in guinea‐pig ventricular cells through the open state.
inferredcites →https://doi.org/10.1126/science.2540529openalex-snapshot
Two Molecular Transitions Influence Cardiac Sodium Channel Gating
inferredcites →https://doi.org/10.1126/science.272.5262.681openalex-snapshot
Molecular Genetic Insights into Cardiovascular Disease
inferredcites →https://doi.org/10.1126/science.281.5373.32openalex-snapshot
Tracking Down Mutations That Can Stop the Heart
inferredcites →https://doi.org/10.1152/ajpheart.1998.274.5.h1643openalex-snapshot
Multiple effects of KPQ deletion mutation on gating of human cardiac Na+channels expressed in mammalian cells
inferredcites →https://doi.org/10.1161/01.cir.92.12.3381openalex-snapshot
Long QT Syndrome Patients With Mutations of the SCN5A and HERG Genes Have Differential Responses to Na + Channel Blockade and to Increases in Heart Rate
inferredcites →https://doi.org/10.1161/01.res.74.6.1071openalex-snapshot
A dynamic model of the cardiac ventricular action potential. I. Simulations of ionic currents and concentration changes.
inferredcites →https://doi.org/10.1161/01.res.74.6.1097openalex-snapshot
A dynamic model of the cardiac ventricular action potential. II. Afterdepolarizations, triggered activity, and potentiation.
inferredcites →https://doi.org/10.1161/01.res.77.1.140openalex-snapshot
Two Components of the Delayed Rectifier K+Current in Ventricular Myocytes of the Guinea Pig Type
inferredcites →https://doi.org/10.1161/01.res.78.5.916openalex-snapshot
Multiple Mechanisms of Na + Channel– Linked Long-QT Syndrome
inferredcites →https://doi.org/10.1172/jci119335openalex-snapshot
Pharmacological targeting of long QT mutant sodium channels.
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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A guide to modelling cardiac electrical activity in anatomically detailed ventricles
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Contributions of HERG K+ current to repolarization of the human ventricular action potential
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Changes in action potentials and intracellular ionic homeostasis in a ventricular cell model related to a persistent sodium current in SCN5A mutations underlying LQT3
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SCN5A channelopathies – An update on mutations and mechanisms
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Multistability property in cardiac ionic models of mammalian and human ventricular cells
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Multi-scale modeling in biology: How to bridge the gaps between scales?
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Conformational changes of an ion-channel during gating and emerging electrophysiologic properties: Application of a computational approach to cardiac Kv7.1
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Alternative strategies in arrhythmia therapy: Evaluation of Na/Ca exchange as an anti-arrhythmic target
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Molecular mechanism matters: Benefits of mechanistic computational models for drug development
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Antiarrhythmic mechanisms of beta blocker therapy
inferred← citeshttps://doi.org/10.1016/j.physrep.2014.05.002openalex-snapshot
Nonlinear and stochastic dynamics in the heart
inferred← citeshttps://doi.org/10.1016/j.physrep.2022.06.003openalex-snapshot
The physics of heart rhythm disorders
inferred← citeshttps://doi.org/10.1016/j.premed.2025.100007openalex-snapshot
Harnessing machine learning to transform precision cardiology
inferred← citeshttps://doi.org/10.1016/j.procs.2010.04.178openalex-snapshot
Stochastic models and simulation of ion channel dynamics
inferred← citeshttps://doi.org/10.1016/j.stemcr.2022.06.005openalex-snapshot
Characterizing arrhythmia using machine learning analysis of Ca2+ cycling in human cardiomyocytes
inferred← citeshttps://doi.org/10.1016/j.tcm.2003.10.001openalex-snapshot
Inherited Sodium Channelopathies A Continuum of Channel Dysfunction
inferred← citeshttps://doi.org/10.1016/j.toxicon.2008.10.015openalex-snapshot
Actions of ATX-II and other gating-modifiers on Na+ currents in HEK-293 cells expressing WT and ΔKPQ hNaV 1.5 Na+ channels
inferred← citeshttps://doi.org/10.1016/j.vascn.2016.05.005openalex-snapshot
A temperature-dependent in silico model of the human ether-à-go-go-related (hERG) gene channel
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2007.11.015openalex-snapshot
Cardiac potassium channel dysfunction in sudden infant death syndrome
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2008.04.001openalex-snapshot
Another calcium paradox in heart failure
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2009.04.003openalex-snapshot
Diastolic transient inward current in long QT syndrome type 3 is caused by Ca2+ overload and inhibited by ranolazine
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2011.12.003openalex-snapshot
Pathophysiology of the cardiac late Na current and its potential as a drug target
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2013.02.009openalex-snapshot
β-adrenergic stimulation activates early afterdepolarizations transiently via kinetic mismatch of PKA targets
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2019.12.014openalex-snapshot
The cardiac CaMKII-Nav1.5 relationship: From physiology to pathology
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2020.12.008openalex-snapshot
Mechanisms underlying age-associated manifestation of cardiac sodium channel gain-of-function
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2024.11.014openalex-snapshot
The sodium/glucose cotransporter 2 inhibitor Empagliflozin inhibits long QT 3 late sodium currents in a mutation specific manner
inferred← citeshttps://doi.org/10.1016/j.ymeth.2020.01.010openalex-snapshot
Multi-scale approaches for the simulation of cardiac electrophysiology: II – Tissue-level structure and function
inferred← citeshttps://doi.org/10.1016/s0002-9343(00)00714-2openalex-snapshot
Molecular biology of sodium channels and their role in cardiac arrhythmias
inferred← citeshttps://doi.org/10.1016/s0006-3495(00)76783-xopenalex-snapshot
Action Potential and Contractility Changes in [Na+]i Overloaded Cardiac Myocytes: A Simulation Study
inferred← citeshttps://doi.org/10.1016/s0006-3495(04)74315-5openalex-snapshot
Theoretical Investigation of the Neuronal Na+ Channel SCN1A: Abnormal Gating and Epilepsy
inferred← citeshttps://doi.org/10.1016/s0008-6363(00)00293-5openalex-snapshot
Cellular consequences of HERG mutations in the long QT syndrome: precursors to sudden cardiac death
inferred← citeshttps://doi.org/10.1016/s0008-6363(01)00332-7openalex-snapshot
Multiple interactions determine cellular electrical processes in the multicellular tissue
inferred← citeshttps://doi.org/10.1016/s0008-6363(01)00465-5openalex-snapshot
New approaches to antiarrhythmic therapy: emerging therapeutic applications of the cell biology of cardiac arrhythmias
inferred← citeshttps://doi.org/10.1016/s0008-6363(02)00265-1openalex-snapshot
Rate-dependent QT shortening mechanism for the LQT3 ΔKPQ mutant
inferred← citeshttps://doi.org/10.1016/s0008-6363(02)00486-8openalex-snapshot
Pharmacological rescue of mutant ion channels
inferred← citeshttps://doi.org/10.1016/s0008-6363(02)00714-9openalex-snapshot
Genetic control of sodium channel function
inferred← citeshttps://doi.org/10.1016/s0033-0620(03)00073-2openalex-snapshot
Mechanisms of genetic arrhythmias: from DNA to ECG
inferred← citeshttps://doi.org/10.1016/s0065-7743(02)37028-3openalex-snapshot
Chapter 27. Biosimulation: Dynamic modeling of biological systems
inferred← citeshttps://doi.org/10.1016/s0167-7799(03)00162-8openalex-snapshot
Will genomics revolutionise pharmaceutical R&D?
inferred← citeshttps://doi.org/10.1016/s1050-1738(01)00116-5openalex-snapshot
Inherited Sodium Channelopathies Novel Therapeutic and Proarrhythmic Molecular Mechanisms
inferred← citeshttps://doi.org/10.1016/s1050-1738(02)00211-6openalex-snapshot
Leucine/Isoleucine Zipper Coordination of Ion Channel Macromolecular Signaling Complexes in the Heart Roles in Inherited Arrhythmias
inferred← citeshttps://doi.org/10.1017/s0033583506004227openalex-snapshot
Computational biology in the study of cardiac ion channels and cell electrophysiology
inferred← citeshttps://doi.org/10.1021/cr300360kopenalex-snapshot
Ion Channel Associated Diseases: Overview of Molecular Mechanisms
inferred← citeshttps://doi.org/10.1038/msb.2009.51openalex-snapshot
Systems biology and the virtual physiological human
inferred← citeshttps://doi.org/10.1038/nm0901-1021openalex-snapshot
Abrupt rate accelerations or premature beats cause life-threatening arrhythmias in mice with long-QT3 syndrome
inferred← citeshttps://doi.org/10.1038/nrd1131openalex-snapshot
Making Better Drugs: Decision Gates in Non-Clinical Drug Development
inferred← citeshttps://doi.org/10.1038/nrg2548openalex-snapshot
Quantitative approaches in developmental biology
inferred← citeshttps://doi.org/10.1038/s41371-021-00505-8openalex-snapshot
Certain beta blockers (e.g., bisoprolol) may be reevaluated in hypertension guidelines for patients with left ventricular hypertrophy to diminish the ventricular arrhythmic risk
inferred← citeshttps://doi.org/10.1038/s41569-018-0104-yopenalex-snapshot
Computational models in cardiology
inferred← citeshttps://doi.org/10.1038/s41598-017-11210-3openalex-snapshot
The effects of ageing and adrenergic challenge on electrocardiographic phenotypes in a murine model of long QT syndrome type 3
inferred← citeshttps://doi.org/10.1038/s41598-018-30577-5openalex-snapshot
Predicting changes to INa from missense mutations in human SCN5A
inferred← citeshttps://doi.org/10.1038/sj.bjp.0705379openalex-snapshot
Action potential changes associated with a slowed inactivation of cardiac voltage‐gated sodium channels by KB130015
inferred← citeshttps://doi.org/10.1038/sj.bjp.0706709openalex-snapshot
Molecular basis of ranolazine block of LQT‐3 mutant sodium channels: evidence for site of action
inferred← citeshttps://doi.org/10.1038/srep13287openalex-snapshot
Purkinje Cells as Sources of Arrhythmias in Long QT Syndrome Type 3
inferred← citeshttps://doi.org/10.1053/euhj.2001.3036openalex-snapshot
New approaches to antiarrhythmic therapy; emerging therapeutic applications of the cell biology of cardiac arrhythmias
inferred← citeshttps://doi.org/10.1063/1.1500496openalex-snapshot
Enhanced self-termination of re-entrant arrhythmias as a pharmacological strategy for antiarrhythmic action
inferred← citeshttps://doi.org/10.1063/1.1636272openalex-snapshot
The role of M cells and the long QT syndrome in cardiac arrhythmias: Simulation studies of reentrant excitations using a detailed electrophysiological model
inferred← citeshttps://doi.org/10.1063/1.2404634openalex-snapshot
Virtual cell and tissue dynamics of ectopic activation of the ventricles
inferred← citeshttps://doi.org/10.1063/1.4999351openalex-snapshot
Memory in a fractional-order cardiomyocyte model alters properties of alternans and spontaneous activity
inferred← citeshttps://doi.org/10.1063/5.0127713openalex-snapshot
Emerging methods to model cardiac ion channel and myocyte electrophysiology
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