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In search of the tumour-suppressor functions of BRCA1 and BRCA2

https://doi.org/10.1038/35044000
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1 of the 41 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-21): 10.1126/science.281.5379.1009 (notice 2003-06-13).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1006/jmbi.1999.2847openalex-snapshot
Analysis of DNA replication forks encountering a pyrimidine dimer in the template to the leading strand 1 1Edited by M. Yaniv
inferredcites →https://doi.org/10.1016/s0092-8674(00)00030-1openalex-snapshot
BRCA1 Is Associated with a Human SWI/SNF-Related Complex
inferredcites →https://doi.org/10.1016/s0092-8674(00)80503-6openalex-snapshot
Dynamic Changes of BRCA1 Subnuclear Location and Phosphorylation State Are Initiated by DNA Damage
inferredcites →https://doi.org/10.1016/s0092-8674(00)81302-1openalex-snapshot
The Tumor Suppressor Gene Brca1 Is Required for Embryonic Cellular Proliferation in the Mouse
inferredcites →https://doi.org/10.1016/s0092-8674(00)81847-4openalex-snapshot
Association of BRCA1 with Rad51 in Mitotic and Meiotic Cells
inferredcites →https://doi.org/10.1016/s0168-9525(99)01930-7openalex-snapshot
Insights into the functions of BRCA1 and BRCA2
inferredcites →https://doi.org/10.1016/s0955-0674(97)80012-9openalex-snapshot
Nuclear organization and gene expression: homologous pairing and long-range interactions
inferredcites →https://doi.org/10.1016/s0960-9822(00)00610-2openalex-snapshot
A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage
inferredcites →https://doi.org/10.1016/s0968-0004(00)01569-3openalex-snapshot
Initiation of genetic recombination and recombination-dependent replication
inferredcites →https://doi.org/10.1016/s1097-2765(00)80035-0openalex-snapshot
Involvement of Brca2 in DNA Repair
inferredcites →https://doi.org/10.1016/s1097-2765(00)80202-6openalex-snapshot
Brca1 Controls Homology-Directed DNA Repair
inferredcites →https://doi.org/10.1016/s1097-2765(00)80238-5openalex-snapshot
Genetic Analysis of BRCA1 Function in a Defined Tumor Cell Line
inferredcites →https://doi.org/10.1016/s1097-2765(00)80276-2openalex-snapshot
Stable Interaction between the Products of the BRCA1 and BRCA2 Tumor Suppressor Genes in Mitotic and Meiotic Cells
inferredcites →https://doi.org/10.1038/15458openalex-snapshot
Diverse and dynamic functions of the Sir silencing complex
inferredcites →https://doi.org/10.1038/304552a0openalex-snapshot
Recombination of parent and daughter strand DNA after UV-irradiation in mammalian cells
inferredcites →https://doi.org/10.1038/35004614openalex-snapshot
hCds1-mediated phosphorylation of BRCA1 regulates the DNA damage response
inferredcites →https://doi.org/10.1038/382678a0openalex-snapshot
Transcriptional activation by BRCA1
inferredcites →https://doi.org/10.1038/386761a0openalex-snapshot
Gatekeepers and caretakers
inferredcites →https://doi.org/10.1038/386772a0openalex-snapshot
Transcriptional activation functions in BRCA2
inferredcites →https://doi.org/10.1038/386804a0openalex-snapshot
Embryonic lethality and radiation hypersensitivity mediated by Rad51 in mice lacking Brca2
inferredcites →https://doi.org/10.1038/ng1296-430openalex-snapshot
Identification of a RING protein that can interact in vivo with the BRCA1 gene product
inferredcites →https://doi.org/10.1038/sj.onc.1203124openalex-snapshot
ATM: A mediator of multiple responses to genotoxic stress
inferredcites →https://doi.org/10.1038/sj.onc.1203269openalex-snapshot
Role of the tumor suppressor gene Brca1 in genetic stability and mammary gland tumor formation
inferredcites →https://doi.org/10.1038/sj.onc.1203334openalex-snapshot
BRCA1 deficient embryonic stem cells display a decreased homologous recombination frequency and an increased frequency of non-homologous recombination that is corrected by expression of a Brca1 transgene
inferredcites →https://doi.org/10.1038/sj.onc.1203971openalex-snapshot
DNA polymerase stalling, sister chromatid recombination and the BRCA genes
inferredcites →https://doi.org/10.1073/pnas.93.24.13595openalex-snapshot
Evidence for a transcriptional activation function of BRCA1 C-terminal region
inferredcites →https://doi.org/10.1073/pnas.96.20.11364openalex-snapshot
RING fingers mediate ubiquitin-conjugating enzyme (E2)-dependent ubiquitination
inferredcites →https://doi.org/10.1093/emboj/17.2.598openalex-snapshot
Rad51‐deficient vertebrate cells accumulate chromosomal breaks prior to cell death
inferredcites →https://doi.org/10.1093/jnci/62.6.1347openalex-snapshot
Malignant Breast Tumors Among Atomic Bomb Survivors, Hiroshima and Nagasaki, 1950–1974
inferredcites →https://doi.org/10.1101/gad.10.19.2423openalex-snapshot
The Atr and Atm protein kinases associate with different sites along meiotically pairing chromosomes.
inferredcites →https://doi.org/10.1101/gad.11.19.2494openalex-snapshot
Intergenic transcription and transinduction of the human β-globin locus
inferredcites →https://doi.org/10.1101/gad.14.4.397openalex-snapshot
ATR disruption leads to chromosomal fragmentation and early embryonic lethality
inferredcites →https://doi.org/10.1101/gad.14.8.927openalex-snapshot
BASC, a super complex of BRCA1-associated proteins involved in the recognition and repair of aberrant DNA structures
inferredcites →https://doi.org/10.1101/gad.851000openalex-snapshot
Functional interactions between BRCA1 and the checkpoint kinase ATR during genotoxic stress
inferredcites →https://doi.org/10.1111/j.1749-6632.1995.tb12113.xopenalex-snapshot
The Role of Estrogen in Mammary Carcinogenesisa
inferredcites →https://doi.org/10.1126/science.281.5379.1009openalex-snapshot
BRCA1 Required for Transcription-Coupled Repair of Oxidative DNA Damage
inferredcites →https://doi.org/10.1126/science.286.5442.1162openalex-snapshot
Requirement of ATM-Dependent Phosphorylation of Brca1 in the DNA Damage Response to Double-Strand Breaks
inferredcites →https://doi.org/10.1126/science.7545954openalex-snapshot
A Strong Candidate for the Breast and Ovarian Cancer Susceptibility Gene BRCA1
inferredcites →https://doi.org/10.1200/jco.1998.16.2.536openalex-snapshot
Second cancers following pediatric Hodgkin's disease.
inferredcites →https://doi.org/10.1242/dev.125.10.1921openalex-snapshot
An entire functional mammary gland may comprise the progeny from a single cell
inferredcites →https://doi.org/10.1515/bc.1999.019openalex-snapshot
The Functions of Breast Cancer Susceptibility Gene 1 (BRCA1) Product and Its Associated Proteins

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Synthetic Lethality As A Promising Approach For Targeted Cancer Prevention
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Subnuclear organization and trafficking of regulatory proteins: Implications for biological control and cancer
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SQ/TQ cluster domains: concentrated ATM/ATR kinase phosphorylation site regions in DNA-damage-response proteins
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Familial association of specific histologic types of ovarian malignancy with other malignancies
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Critical questions in ovarian cancer research and treatment: Report of an American Association for Cancer Research Special Conference
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Graphene Doped Mn2O3 Nanofibers as a Facile Electroanalytical DNA Point Mutation Detection Platform for Early Diagnosis of Breast/Ovarian Cancer
inferred← citeshttps://doi.org/10.1002/gcc.10105openalex-snapshot
Subcellular localization of the BRCA1 gene product in mitotic cells
inferred← citeshttps://doi.org/10.1002/humu.22150openalex-snapshot
A guide for functional analysis ofBRCA1variants of uncertain significance
inferred← citeshttps://doi.org/10.1002/humu.9049openalex-snapshot
Analysis of breast cancer susceptibility genesBRCA1 andBRCA2 in Thai familial and isolated early-onset breast and ovarian cancer
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Variation in breast cancer risk of heterozygotes for ataxia‐telangiectasia according to environmental factors
inferred← citeshttps://doi.org/10.1002/ijc.10945openalex-snapshot
The product of the candidate prostate cancer susceptibility gene ELAC2 interacts with the γ‐tubulin complex
inferred← citeshttps://doi.org/10.1002/ijc.1329openalex-snapshot
Cancer risk in heterozygotes for ataxia-telangiectasia
inferred← citeshttps://doi.org/10.1002/ijc.20169openalex-snapshot
The variant E233G of the RAD51D gene could be a low‐penetrance allele in high‐risk breast cancer families without BRCA1/2 mutations
inferred← citeshttps://doi.org/10.1002/ijc.20541openalex-snapshot
Mutant BRCA1 alleles transmission: Different approaches and different biases
inferred← citeshttps://doi.org/10.1002/ijc.22915openalex-snapshot
Lymphocyte radiosensitivity in BRCA1 and BRCA2 mutation carriers and implications for breast cancer susceptibility
inferred← citeshttps://doi.org/10.1002/ijc.24684openalex-snapshot
BRCA1 modulates malignant cell behavior, the expression of survivin and chemosensitivity in human breast cancer cells
inferred← citeshttps://doi.org/10.1002/jcb.10081openalex-snapshot
BRCA1 modulates ionizing radiation‐induced nuclear focus formation by the replication protein A p34 subunit
inferred← citeshttps://doi.org/10.1002/jcb.20386openalex-snapshot
BRCA1: A locus‐specific “liaison” in gene expression and genetic integrity
inferred← citeshttps://doi.org/10.1002/jcb.30471openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/jcp.21958openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/jcp.26601openalex-snapshot
Thiostrepton degrades mutant p53 by eliciting an autophagic response in SW480 cells
inferred← citeshttps://doi.org/10.1002/jemt.10178openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/jmr.747openalex-snapshot
Showing your ID: intrinsic disorder as an ID for recognition, regulation and cell signaling
inferred← citeshttps://doi.org/10.1002/mc.10051openalex-snapshot
Comparative analysis of loss of heterozygosity of specific chromosome 3, 13, 17, and X loci and TP53 mutations in human epithelial ovarian cancer
inferred← citeshttps://doi.org/10.1002/mc.10148openalex-snapshot
Novel consensus DNA‐binding sequence for BRCA1 protein complexes
inferred← citeshttps://doi.org/10.1002/mgg3.810openalex-snapshot
Integrative genomic analysis identifies associations of molecular alterations to APOBEC and BRCA1/2 mutational signatures in breast cancer
inferred← citeshttps://doi.org/10.1002/pat.70539openalex-snapshot
Combinatorial Targeted PLGA Nanogels for the Enhanced Delivery of Homologous Recombination Inhibitors to Improve Chemotherapeutic Effectiveness
inferred← citeshttps://doi.org/10.1002/path.1507openalex-snapshot
Expression of BRCA1 protein in benign, borderline, and malignant epithelial ovarian neoplasms and its relationship to methylation and allelic loss of the BRCA1 gene
inferred← citeshttps://doi.org/10.1002/path.1751openalex-snapshot
The GADD45, ZBRK1 and BRCA1 pathway: quantitative analysis of mRNA expression in colon carcinomas
inferred← citeshttps://doi.org/10.1002/path.2500openalex-snapshot
Telomere‐associated proteins: cross‐talk between telomere maintenance and telomere‐lengthening mechanisms
inferred← citeshttps://doi.org/10.1006/excr.2000.5132openalex-snapshot
Interactions between BRCA Proteins and DNA Structure
inferred← citeshttps://doi.org/10.1006/gyno.2002.6646openalex-snapshot
Cell Proliferation and Apoptosis in BRCA-Associated Hereditary Ovarian Cancer
inferred← citeshttps://doi.org/10.1006/gyno.2002.6674openalex-snapshot
Specific Keynote: Hereditary Ovarian Cancer: What We Know
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Modeling chromosomal instability and epithelial carcinogenesis in the telomerase-deficient mouse
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Molecular and pathological characterization of inherited breast cancer
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Mammalian DNA Damage Response Pathway
inferred← citeshttps://doi.org/10.1007/4735_2007_0216openalex-snapshot
BRCA2: safeguarding the genome through homologous recombination
inferred← citeshttps://doi.org/10.1007/7050_2007_035openalex-snapshot
Synaptic and Recombination Nodules in Mammals: Structural Continuity with Shifting Protein Composition
inferred← citeshttps://doi.org/10.1007/978-0-387-33227-7_17openalex-snapshot
Inherited Breast Cancer
inferred← citeshttps://doi.org/10.1007/978-0-387-68969-2_8openalex-snapshot
BRCA1-Induced Ovarian Oncogenesis
inferred← citeshttps://doi.org/10.1007/978-0-387-68969-2_9openalex-snapshot
Role of p53 and Rb in Ovarian Cancer
inferred← citeshttps://doi.org/10.1007/978-0-387-78818-0_11openalex-snapshot
Hormonal Heterogeneity of Endometrial Cancer
inferred← citeshttps://doi.org/10.1007/978-0-387-87366-4_3openalex-snapshot
Inherited Breast Cancer
inferred← citeshttps://doi.org/10.1007/978-0-387-87374-9_17openalex-snapshot
Inherited Breast Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4020-5867-7_3openalex-snapshot
BRCA1 in initiation, invasion, and metastasis of breast cancer: a perspective from the tumor microenvironment
inferred← citeshttps://doi.org/10.1007/978-1-4471-2375-0_6openalex-snapshot
Reproductive System
inferred← citeshttps://doi.org/10.1007/978-1-4614-8633-6_1openalex-snapshot
Epidemiology of Epithelial Ovarian Carcinoma
inferred← citeshttps://doi.org/10.1007/978-1-59259-791-8_17openalex-snapshot
DNA Repair Defects in Cancer
inferred← citeshttps://doi.org/10.1007/978-1-59745-035-5_30openalex-snapshot
Molecular Profiling in Breast Cancer
inferred← citeshttps://doi.org/10.1007/978-1-59745-458-2_33openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/978-1-60327-303-9_8openalex-snapshot
Biological and Molecular Effects of Vitamin D on Bone
inferred← citeshttps://doi.org/10.1007/978-3-030-44743-4_15openalex-snapshot
Long Noncoding RNAs as Players in Breast Tumorigenesis
inferred← citeshttps://doi.org/10.1007/978-3-030-56051-5_5openalex-snapshot
Hereditary Cancers and Genetics
inferred← citeshttps://doi.org/10.1007/978-3-031-09169-8_4openalex-snapshot
Hereditary Ovarian Cancer
inferred← citeshttps://doi.org/10.1007/978-3-319-16304-8_2openalex-snapshot
Molecular Biology Basics in the “Omics” Era: Cancer Pathology
inferred← citeshttps://doi.org/10.1007/978-3-319-21683-6_18openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/978-3-319-59198-8_9openalex-snapshot
Radiation Oncology Considerations in the Management of Mutation Carriers with Breast Cancer
inferred← citeshttps://doi.org/10.1007/978-3-319-64096-9_23openalex-snapshot
DNA Damage Repair
inferred← citeshttps://doi.org/10.1007/978-3-540-71021-9_13openalex-snapshot
BRCA2: safeguarding the genome through homologous recombination
inferred← citeshttps://doi.org/10.1007/978-981-10-6020-5_13openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/bf02982016openalex-snapshot
Molecular Pathogenesis of Fanconi Anemia
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Current Knowledge on the Pathophysiology of Fanconi Anemia: From Genes to Phenotypes
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Cancer Pharmacogenomics
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Zinc and human health: an update
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Molecular biosensing system based on intrinsically disordered proteins
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Genetic and epigenetic features in radiation sensitivity
inferred← citeshttps://doi.org/10.1007/s00259-004-1731-6openalex-snapshot
Genetic and epigenetic features in radiation sensitivity
inferred← citeshttps://doi.org/10.1007/s00280-014-2581-yopenalex-snapshot
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inferred← citeshttps://doi.org/10.1007/s00412-008-0154-8openalex-snapshot
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network
inferred← citeshttps://doi.org/10.1007/s00438-011-0612-5openalex-snapshot
EMSY overexpression disrupts the BRCA2/RAD51 pathway in the DNA-damage response: implications for chromosomal instability/recombination syndromes as checkpoint diseases
inferred← citeshttps://doi.org/10.1007/s10269-005-0283-0openalex-snapshot
Quelle place tiennent les gènes BRCA1 et BRCA2 dans les risques héréditaires de cancer de l’ovaire?
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BRCA1- und BRCA2-Genmutationen: Klinische Konsequenzen beim vererbbaren Brust- und Eierstockkrebs
inferred← citeshttps://doi.org/10.1007/s10549-006-9189-9openalex-snapshot
Telomerase immortalization of human mammary epithelial cells derived from a BRCA2 mutation carrier
inferred← citeshttps://doi.org/10.1007/s10549-006-9493-4openalex-snapshot
BRCA1 and BRCA2 germline mutation analysis in the Indonesian population
inferred← citeshttps://doi.org/10.1007/s10549-008-0089-zopenalex-snapshot
Genetic variation in DNA repair pathway genes and premenopausal breast cancer risk
inferred← citeshttps://doi.org/10.1007/s10549-010-0947-3openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/s10549-013-2729-1openalex-snapshot
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Brief Report: Towards a dietary prevention of hereditary breast cancer
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inferred← citeshttps://doi.org/10.1007/s12032-016-0849-2openalex-snapshot
rs15869 at miRNA binding site in BRCA2 is associated with breast cancer susceptibility
inferred← citeshttps://doi.org/10.1007/s12254-009-0093-yopenalex-snapshot
Clinical implications of DNA repair genetic alterations in cancer
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Pathology of hereditary breast cancer
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Ionizing Radiation as a Carcinogen
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Functional evidence (II) protein and enzyme function
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Assessing Pelvic Epithelial Cancer Risk and Intercepting Early Malignancy
inferred← citeshttps://doi.org/10.1016/b978-0-323-95488-4.00106-6openalex-snapshot
Ionizing Radiation as a Carcinogen
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Current trends in breast cancer: pathology, breast cancer microscopic analysis, breast cancer stages, and classification
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Gene Therapy of Ovarian Cancer—State-of-the-Art and Future Perspectives
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Involvement of Nuclear Architecture in Regulating Gene Expression in Bone Cells
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Assessing Pelvic Cancer Risk and Intercepting Early Malignancy
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Current and emerging biomarkers in ovarian cancer diagnosis; CA125 and beyond
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An architectural perspective of vitamin D responsiveness
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Combinatorial organization of the transcriptional regulatory machinery in biological control and cancer
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Concomitant BRCA1 and BRCA2 gene mutations in an Ashkenazi Jewish Woman with primary breast and ovarian cancer
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Protein phosphatase 5 is necessary for ATR-mediated DNA repair
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Molecular analysis of DNA repair gene methylation and protein expression during chemical-induced rat lung carcinogenesis
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Fine tuning chemotherapy to match BRCA1 status
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Functional assays for BRCA1 and BRCA2
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Counseling for male BRCA mutation carriers – a review
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DNA damaging agents and DNA repair: From carcinogenesis to cancer therapy
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Novel BRCA1/2 mutations in Serbian breast and breast–ovarian cancer patients with hereditary predisposition
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The cell-type-specificity of inherited predispositions to tumours: review and hypothesis
inferred← citeshttps://doi.org/10.1016/j.canlet.2004.08.009openalex-snapshot
RAD51, genomic stability, and tumorigenesis
inferred← citeshttps://doi.org/10.1016/j.canlet.2004.11.001openalex-snapshot
Loss of heterozygosity at the BRCA1 locus in Tunisian women with sporadic breast cancer
inferred← citeshttps://doi.org/10.1016/j.canlet.2008.04.046openalex-snapshot
RAP80 and RNF8, key players in the recruitment of repair proteins to DNA damage sites
inferred← citeshttps://doi.org/10.1016/j.canlet.2013.12.026openalex-snapshot
BRCA1 deficiency induces protective autophagy to mitigate stress and provides a mechanism for BRCA1 haploinsufficiency in tumorigenesis
inferred← citeshttps://doi.org/10.1016/j.canrad.2004.11.003openalex-snapshot
Bases génétiques de la radiosensibilité des cancers du sein
inferred← citeshttps://doi.org/10.1016/j.ccr.2005.08.004openalex-snapshot
BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ
inferred← citeshttps://doi.org/10.1016/j.ccr.2006.01.013openalex-snapshot
X chromosomal abnormalities in basal-like human breast cancer
inferred← citeshttps://doi.org/10.1016/j.ccr.2013.03.025openalex-snapshot
Function of BRCA1 in the DNA Damage Response Is Mediated by ADP-Ribosylation
inferred← citeshttps://doi.org/10.1016/j.cell.2005.06.022openalex-snapshot
A Role for Proapoptotic BID in the DNA-Damage Response
inferred← citeshttps://doi.org/10.1016/j.cellsig.2009.09.007openalex-snapshot
A comparison of BRCA1 nuclear localization with 14 DNA damage response proteins and domains: Identification of specific differences between BRCA1 and 53BP1 at DNA damage-induced foci
inferred← citeshttps://doi.org/10.1016/j.celrep.2015.09.040openalex-snapshot
Poly(ADP-Ribose) Mediates the BRCA2-Dependent Early DNA Damage Response
inferred← citeshttps://doi.org/10.1016/j.critrevonc.2005.05.003openalex-snapshot
BRCA1 and BRCA2: The genetic testing and the current management options for mutation carriers
inferred← citeshttps://doi.org/10.1016/j.critrevonc.2006.12.004openalex-snapshot
Genetic susceptibility for breast cancer: How many more genes to be found?
inferred← citeshttps://doi.org/10.1016/j.cub.2003.11.029openalex-snapshot
BRCA1/BARD1 Orthologs Required for DNA Repair in Caenorhabditis elegans
inferred← citeshttps://doi.org/10.1016/j.cub.2009.02.018openalex-snapshot
PALB2 Links BRCA1 and BRCA2 in the DNA-Damage Response
inferred← citeshttps://doi.org/10.1016/j.devcel.2011.10.010openalex-snapshot
Oncogenic Ras Regulates BRIP1 Expression to Induce Dissociation of BRCA1 from Chromatin, Inhibit DNA Repair, and Promote Senescence
inferred← citeshttps://doi.org/10.1016/j.dnarep.2004.05.007openalex-snapshot
The role of the DNA double-strand break response network in meiosis
inferred← citeshttps://doi.org/10.1016/j.dnarep.2004.08.010openalex-snapshot
Molecular analysis of sister chromatid recombination in mammalian cells
inferred← citeshttps://doi.org/10.1016/j.dnarep.2006.08.009openalex-snapshot
BRCA1 ubiquitylation of CtIP: Just the tIP of the iceberg?
inferred← citeshttps://doi.org/10.1016/j.dnarep.2007.02.020openalex-snapshot
Minding the gap: The underground functions of BRCA1 and BRCA2 at stalled replication forks
inferred← citeshttps://doi.org/10.1016/j.dnarep.2008.08.001openalex-snapshot
Mitotic delay in lymphocytes from BRCA1 heterozygotes unable to reduce the radiation-induced chromosomal damage
inferred← citeshttps://doi.org/10.1016/j.dnarep.2009.10.010openalex-snapshot
Linking up and interacting with BRCT domains
inferred← citeshttps://doi.org/10.1016/j.dnarep.2019.102668openalex-snapshot
How do mutations affecting the breast cancer genes BRCA1 and BRCA2 cause cancer susceptibility?
inferred← citeshttps://doi.org/10.1016/j.dnarep.2021.103181openalex-snapshot
The dark side of homology-directed repair
inferred← citeshttps://doi.org/10.1016/j.ejca.2008.07.033openalex-snapshot
HMGA1 protein is a novel target of the ATM kinase
inferred← citeshttps://doi.org/10.1016/j.ejca.2013.04.028openalex-snapshot
Contralateral breast cancer after radiotherapy among BRCA1 and BRCA2 mutation carriers: A WECARE Study Report
inferred← citeshttps://doi.org/10.1016/j.etap.2010.09.006openalex-snapshot
Altered BRCA1 and BRCA2 responses and mutation of BRCA1 gene in mice exposed chronically and transgenerationally to aqueous extract of betel nut (AEBN)
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2005.02.037openalex-snapshot
Upregulation of the BRCA1 gene in human germ cells and in preimplantation embryos
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2008.12.052openalex-snapshot
Fertility in women with BRCA mutations: a case-control study
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2014.08.014openalex-snapshot
BRCA1 germline mutations may be associated with reduced ovarian reserve
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2015.07.1162openalex-snapshot
BRCA mutation carriers show normal ovarian response in in vitro fertilization cycles
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2017.12.004openalex-snapshot
BRCA mutations and reproduction
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2021.07.033openalex-snapshot
CometChip analysis of human primary lymphocytes enables quantification of inter-individual differences in the kinetics of repair of oxidative DNA damage
inferred← citeshttps://doi.org/10.1016/j.humpath.2005.06.006openalex-snapshot
Hereditary ovarian cancer
inferred← citeshttps://doi.org/10.1016/j.ijrobp.2009.10.020openalex-snapshot
Increased Chromosomal Radiosensitivity in Women Carrying BRCA1/BRCA2 Mutations Assessed With the G2 Assay
inferred← citeshttps://doi.org/10.1016/j.jbiosc.2016.09.006openalex-snapshot
Lengthening of high-yield production levels of monoclonal antibody-producing Chinese hamster ovary cells by downregulation of breast cancer 1
inferred← citeshttps://doi.org/10.1016/j.jchromb.2005.05.006openalex-snapshot
Quantification by affinity perfusion chromatography of phosphorylated BRCAl and BRCA2 proteins from tumor cells after lycopene treatment
inferred← citeshttps://doi.org/10.1016/j.jmb.2004.10.045openalex-snapshot
Characterization of Segments from the Central Region of BRCA1: An Intrinsically Disordered Scaffold for Multiple Protein–Protein and Protein–DNA Interactions?
inferred← citeshttps://doi.org/10.1016/j.jmb.2023.168277openalex-snapshot
BRCA1/2 Haploinsufficiency: Exploring the Impact of Losing one Allele
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2010.02.013openalex-snapshot
Recruitment and subnuclear distribution of the regulatory machinery during 1α,25-dihydroxy vitamin D3-mediated transcriptional upregulation in osteoblasts
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2013.09.009openalex-snapshot
Novel roles of 1α,25(OH)2D3 on DNA repair provide new strategies for breast cancer treatment
inferred← citeshttps://doi.org/10.1016/j.jtbi.2010.12.033openalex-snapshot
Evolutionary dynamics of BRCA1 alterations in breast tumorigenesis
inferred← citeshttps://doi.org/10.1016/j.jtcvs.2013.09.060openalex-snapshot
BRCA1 shields vascular smooth muscle cells from oxidative stress
inferred← citeshttps://doi.org/10.1016/j.micpath.2010.11.007openalex-snapshot
Peptide aptamer mimicking RAD51-binding domain of BRCA2 inhibits DNA damage repair and survival in Trypanosoma brucei
inferred← citeshttps://doi.org/10.1016/j.molcel.2004.12.007openalex-snapshot
Control of Sister Chromatid Recombination by Histone H2AX
inferred← citeshttps://doi.org/10.1016/j.molmed.2006.09.004openalex-snapshot
Deconstructing the molecular portrait of basal-like breast cancer
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2004.04.001openalex-snapshot
Basal, oxidative and alkylative DNA damage, DNA repair efficacy and mutagen sensitivity in breast cancer
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2004.05.011openalex-snapshot
DNA damage and repair in type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2005.05.010openalex-snapshot
Genomic instability and cancer: Networks involved in response to DNA damage
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2005.06.020openalex-snapshot
Ionizing radiation and genetic risks
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2005.12.016openalex-snapshot
Loss of Ubr2, an E3 ubiquitin ligase, leads to chromosome fragility and impaired homologous recombinational repair
inferred← citeshttps://doi.org/10.1016/j.mrrev.2003.06.011openalex-snapshot
Enhanced sensitivity of peripheral blood lymphocytes from women carrying a BRCA1 mutation towards the mutagenic effects of various cytostatics
inferred← citeshttps://doi.org/10.1016/j.procbio.2023.12.020openalex-snapshot
Revamping precision treatment with nanoparticles envisaging effective drug delivery systems for ovarian cancer
inferred← citeshttps://doi.org/10.1016/j.prp.2010.06.006openalex-snapshot
Importance of DNA damage checkpoints in the pathogenesis of human cancers
inferred← citeshttps://doi.org/10.1016/j.radonc.2011.05.043openalex-snapshot
Elevated radiation-induced γH2AX foci in G2 phase heterozygous BRCA2 fibroblasts
inferred← citeshttps://doi.org/10.1016/j.radonc.2011.07.007openalex-snapshot
Adjuvant chemotherapy and radiotherapy in triple-negative breast carcinoma: A prospective randomized controlled multi-center trial
inferred← citeshttps://doi.org/10.1016/j.rvsc.2015.10.003openalex-snapshot
Promoter mutation and reduced expression of BRCA1 in canine mammary tumors
inferred← citeshttps://doi.org/10.1016/j.semcancer.2005.06.002openalex-snapshot
Selective evolutionary pressure from the tissue microenvironment drives tumor progression
inferred← citeshttps://doi.org/10.1016/j.semcdb.2003.09.011openalex-snapshot
The cell biology of a novel chromosomal RNA: chromosome painting by XIST/Xist RNA initiates a remodeling cascade
inferred← citeshttps://doi.org/10.1016/j.tcb.2005.05.005openalex-snapshot
Nuclear tumor suppressors in space and time
inferred← citeshttps://doi.org/10.1016/j.tibs.2004.04.009openalex-snapshot
Emerging functions of BRCA2 in DNA recombination
inferred← citeshttps://doi.org/10.1016/j.tibs.2012.06.007openalex-snapshot
Links between genome integrity and BRCA1 tumor suppression
inferred← citeshttps://doi.org/10.1016/j.yexcr.2003.09.027openalex-snapshot
Cytoplasmic mislocalization of BRCA1 caused by cancer-associated mutations in the BRCT domain
inferred← citeshttps://doi.org/10.1016/j.yexcr.2004.05.004openalex-snapshot
BARD1 regulates BRCA1 apoptotic function by a mechanism involving nuclear retention
inferred← citeshttps://doi.org/10.1016/j.ygeno.2004.08.019openalex-snapshot
Structural evolution of the BRCA1 genomic region in primates
inferred← citeshttps://doi.org/10.1016/s0002-9440(10)62317-6openalex-snapshot
Mechanogenomic Control of DNA Exposure and Sequestration
inferred← citeshttps://doi.org/10.1016/s0006-291x(02)00624-1openalex-snapshot
Cloning a cDNA encoding an alternatively spliced protein of BRCA2-associated factor 35
inferred← citeshttps://doi.org/10.1016/s0006-291x(02)00625-3openalex-snapshot
Immunohistological detection of BRAF25 in human prostate tumor and cancer specimens
inferred← citeshttps://doi.org/10.1016/s0006-291x(02)02751-1openalex-snapshot
Identification of genes induced by BRCA1 in breast cancer cells
inferred← citeshttps://doi.org/10.1016/s0009-8981(01)00671-4openalex-snapshot
Prevalence of breast cancer predisposition gene mutations in Chinese women and guidelines for genetic testing
inferred← citeshttps://doi.org/10.1016/s0014-4827(02)00019-8openalex-snapshot
Regulation of tumor suppressors by nuclear-cytoplasmic shuttling
inferred← citeshttps://doi.org/10.1016/s0014-4827(03)00368-9openalex-snapshot
Unique tissue-specific level of DNA nucleotide excision repair in primary human mammary epithelial cultures
inferred← citeshttps://doi.org/10.1016/s0014-5793(03)00978-5openalex-snapshot
BRCA1 interacts with FHL2 and enhances FHL2 transactivation function
inferred← citeshttps://doi.org/10.1016/s0027-5107(00)00106-8openalex-snapshot
Ionizing radiation and genetic risks
inferred← citeshttps://doi.org/10.1016/s0027-5107(01)00300-1openalex-snapshot
Mutagen sensitivity of peripheral blood from women carrying a BRCA1 or BRCA2 mutation
inferred← citeshttps://doi.org/10.1016/s0027-5107(02)00224-5openalex-snapshot
Recombinational DNA repair and human disease
inferred← citeshttps://doi.org/10.1016/s0065-230x(01)83007-4openalex-snapshot
ATM: Genome stability, neuronal development, and cancer cross paths
inferred← citeshttps://doi.org/10.1016/s0065-230x(02)84001-5openalex-snapshot
Gene expression in inherited breast cancer
inferred← citeshttps://doi.org/10.1016/s0065-2660(06)58003-4openalex-snapshot
Involvement of Homologous Recombination in Carcinogenesis
inferred← citeshttps://doi.org/10.1016/s0092-8674(02)00615-3openalex-snapshot
Cancer Susceptibility and the Functions of BRCA1 and BRCA2
inferred← citeshttps://doi.org/10.1016/s0092-8674(02)01052-8openalex-snapshot
BRCA1 Supports XIST RNA Concentration on the Inactive X Chromosome
inferred← citeshttps://doi.org/10.1016/s0092-8674(03)01075-4openalex-snapshot
Principles of Tumor Suppression
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