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Activation of microglial cells by β-amyloid protein and interferon-γ

https://doi.org/10.1038/374647a0
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Inducible nitric oxide synthase activity of cloned murine microglial cells
inferredcites →https://doi.org/10.1016/0006-8993(92)90380-ropenalex-snapshot
β-Amyloid stimulates glial cells in vitro to produce growth factors that accumulate in senile plaques in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0006-8993(92)91004-xopenalex-snapshot
Microglial-produced nitric oxide and reactive nitrogen oxides mediate neuronal cell death
inferredcites →https://doi.org/10.1016/0012-1606(91)90474-hopenalex-snapshot
Heterotypic and homotypic cellular interactions influencing the growth and differentiation of bipotential oligodendrocyte-type-2 astrocyte progenitors in culture
inferredcites →https://doi.org/10.1016/0092-8674(94)90131-7openalex-snapshot
Hydrogen peroxide mediates amyloid β protein toxicity
inferredcites →https://doi.org/10.1016/0166-2236(93)90007-9openalex-snapshot
Genetics and molecular advances in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0166-2236(93)90070-3openalex-snapshot
A radical hypothesis for neurodegeneration
inferredcites →https://doi.org/10.1016/0166-2236(93)90180-topenalex-snapshot
Macrophages and inflammation in the central nervous system
inferredcites →https://doi.org/10.1016/0361-9230(90)90045-2openalex-snapshot
GABA accumulating neurons are relatively resistant to chronic hypoxia in vitro: An autoradiographic study
inferredcites →https://doi.org/10.1016/0896-6273(94)90159-7openalex-snapshot
Tumor necrosis factors protect neurons against metabolic-excitotoxic insults and promote maintenance of calcium homeostasis
inferredcites →https://doi.org/10.1016/s0021-9258(17)41969-7openalex-snapshot
The role of pyruvate in neuronal calcium homeostasis. Effects on intracellular calcium pools.
inferredcites →https://doi.org/10.1056/nejm199112263252605openalex-snapshot
β-Amyloid and the Pathogenesis of Alzheimer's Disease
inferredcites →https://doi.org/10.1073/pnas.88.16.7247openalex-snapshot
An in vivo model for the neurodegenerative effects of beta amyloid and protection by substance P.
inferredcites →https://doi.org/10.1073/pnas.89.18.8676openalex-snapshot
Interleukin 10 inhibits macrophage microbicidal activity by blocking the endogenous production of tumor necrosis factor alpha required as a costimulatory factor for interferon gamma-induced activation.
inferredcites →https://doi.org/10.1073/pnas.91.8.3270openalex-snapshot
A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease.
inferredcites →https://doi.org/10.1084/jem.178.6.2207openalex-snapshot
Interleukin 10 (IL-10) inhibits the release of proinflammatory cytokines from human polymorphonuclear leukocytes. Evidence for an autocrine role of tumor necrosis factor and IL-1 beta in mediating the production of IL-8 triggered by lipopolysaccharide.
inferredcites →https://doi.org/10.1111/j.1432-1033.1993.tb19893.xopenalex-snapshot
Human and rodent Alzheimer β‐amyloid peptides acquire distinct conformations in membrane‐mimicking solvents
inferredcites →https://doi.org/10.1126/science.2218531openalex-snapshot
Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin Neuropeptides
inferredcites →https://doi.org/10.1126/science.3544219openalex-snapshot
Conservation of Brain Amyloid Proteins in Aged Mammals and Humans with Alzheimer's Disease
inferredcites →https://doi.org/10.1126/science.7901908openalex-snapshot
Oxidative Stress, Glutamate, and Neurodegenerative Disorders
inferredcites →https://doi.org/10.1523/jneurosci.13-04-01676.1993openalex-snapshot
Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state
inferredcites →https://doi.org/10.4049/jimmunol.141.7.2407openalex-snapshot
Release of reactive nitrogen intermediates and reactive oxygen intermediates from mouse peritoneal macrophages. Comparison of activating cytokines and evidence for independent production.
inferredcites →https://doi.org/10.4049/jimmunol.149.8.2736openalex-snapshot
Activated microglia mediate neuronal cell injury via a nitric oxide mechanism
inferredcites →https://doi.org/10.4049/jimmunol.151.3.1473openalex-snapshot
Neuroprotective role of IL-4 against activated microglia.
inferredcites →https://doi.org/10.4049/jimmunol.151.8.3999openalex-snapshot
Immune functions of tumor necrosis factor. I. Tumor necrosis factor induces apoptosis of mouse thymocytes and can also stimulate or inhibit IL-6-induced proliferation depending on the concentration of mitogenic costimulation.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Stimulating Neuroprotective and Regenerative Mechanisms in Alzheimer Disease
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T-Cell Interferon Gamma Binding in Patients With Dementia of the Alzheimer Type
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Translational Research on the Way to Effective Therapy for Alzheimer Disease
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Role of Nitric Oxide in the Onset of Facial Nerve Palsy by HSV-1 Infection
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Age-related amyloid ? protein accumulation induces cellular death and macrophage activation in transgenic mice
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?-Amyloid protein enhances macrophage production of oxygen free radicals and glutamate
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Microglial activation induced by factor(s) contained in sera from Alzheimer-related ApoE genotypes
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Nerve growth factor inhibits apoptosis induced by tumor necrosis factor in PC12 cells
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Mannose receptor is present in a functional state in rat microglial cells
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Impairment of hippocampal long-term potentiation by Alzheimer amyloid ?-peptides
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Induction of microglial nitric oxide synthesis by very low density lipoprotein
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Cyclooxygenase-2 expression in rat microglia is induced by adenosine A2a-receptors
inferred← citeshttps://doi.org/10.1002/(sici)1098-1136(199712)21:4<413::aid-glia9>3.0.co;2-3openalex-snapshot
Albumin enhances superoxide production in cultured microglia
inferred← citeshttps://doi.org/10.1002/(sici)1098-1136(199812)24:4<408::aid-glia6>3.0.co;2-dopenalex-snapshot
Expression of Kv1.5 K channels in activated microglia in vivo
inferred← citeshttps://doi.org/10.1002/(sici)1098-1136(20000215)29:4<305::aid-glia2>3.0.co;2-vopenalex-snapshot
Fractalkine modulates TNF-? secretion and neurotoxicity induced by microglial activation
inferred← citeshttps://doi.org/10.1002/(sici)1099-1077(199912)14:8<525::aid-hup140>3.0.co;2-topenalex-snapshot
A unifying hypothesis of Alzheimer's disease. II. Pathophysiological processes
inferred← citeshttps://doi.org/10.1002/0471973122.ch18openalex-snapshot
Redox Proteomics: A New Approach to Investigate Oxidative Stress in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/1097-4547(20001115)62:4<503::aid-jnr4>3.0.co;2-aopenalex-snapshot
Dehydroepiandrosterone inhibits microglial nitric oxide production in a stimulus-specific manner
inferred← citeshttps://doi.org/10.1002/1098-1136(200009)31:3<262::aid-glia70>3.0.co;2-2openalex-snapshot
Phagocytosis of neuronal or glial debris by microglial cells: Upregulation of MHC class II expression and multinuclear giant cell formation in vitro
inferred← citeshttps://doi.org/10.1002/9780470101001.hcn066openalex-snapshot
Pharmacology of Inflammation
inferred← citeshttps://doi.org/10.1002/9781118406557.ch6openalex-snapshot
Neuroinflammation in Alzheimer's, Parkinson's and Huntington's Diseases
inferred← citeshttps://doi.org/10.1002/9781118823408.ch5openalex-snapshot
Nutraceuticals in immunosenescence
inferred← citeshttps://doi.org/10.1002/acn3.50837openalex-snapshot
Translocator protein in late stage Alzheimer’s disease and Dementia with Lewy bodies brains
inferred← citeshttps://doi.org/10.1002/acr.23119openalex-snapshot
Increased Incidence of Amyotrophic Lateral Sclerosis in Polymyositis: A Nationwide Cohort Study
inferred← citeshttps://doi.org/10.1002/agt2.333openalex-snapshot
Protein amyloid aggregate: Structure and function
inferred← citeshttps://doi.org/10.1002/agt2.70023openalex-snapshot
Bridging Mechanical Properties with Atomic Structures of Polymorphic α‐Synuclein Fibrils by Single‐Molecule Analysis and Molecular Dynamics Simulations
inferred← citeshttps://doi.org/10.1002/alz.70472openalex-snapshot
Integrating neuroinflammation biomarkers into the ATN(X) framework: Advances in Alzheimer's pathogenesis, diagnosis, and insights from non‐human primate models
inferred← citeshttps://doi.org/10.1002/ana.20502openalex-snapshot
RON‐regulated innate immunity is protective in an animal model of multiple sclerosis
inferred← citeshttps://doi.org/10.1002/cne.10325openalex-snapshot
Glial expression of estrogen and androgen receptors after rat brain injury
inferred← citeshttps://doi.org/10.1002/cne.21252openalex-snapshot
Macrophage depletion in the murine olfactory epithelium leads to increased neuronal death and decreased neurogenesis
inferred← citeshttps://doi.org/10.1002/eji.1830260306openalex-snapshot
Nitric oxide‐mediated suppression of T cell responses during Trypanosoma brucei infection: soluble trypanosome products and interferon‐γ are synergistic inducers of nitric oxide synthase
inferred← citeshttps://doi.org/10.1002/eji.1830271024openalex-snapshot
A Leishmania protein that modulates interleukin (IL)‐12, IL‐10 and tumor necrosis factor‐α production and expression of B7‐1 in human monocyte‐derived antigen‐presenting cells
inferred← citeshttps://doi.org/10.1002/glia.10047openalex-snapshot
Trisialoganglioside GT1b induces in vivo degeneration of nigral dopaminergic neurons: Role of microglia
inferred← citeshttps://doi.org/10.1002/glia.10146openalex-snapshot
Neuroinflammation in Alzheimer's disease and prion disease
inferred← citeshttps://doi.org/10.1002/glia.10149openalex-snapshot
Intracellular signaling in M‐CSF‐induced microglia activation: Role of Iba1
inferred← citeshttps://doi.org/10.1002/glia.10183openalex-snapshot
Hematopoietic prostaglandin D synthase is expressed in microglia in the developing postnatal mouse brain
inferred← citeshttps://doi.org/10.1002/glia.10314openalex-snapshot
Activated glia induce neuron death via MAP kinase signaling pathways involving JNK and p38
inferred← citeshttps://doi.org/10.1002/glia.10316openalex-snapshot
Signaling through JAK2‐STAT5 pathway is essential for IL‐3‐induced activation of microglia
inferred← citeshttps://doi.org/10.1002/glia.10329openalex-snapshot
Glial activation modulates glutamate neurotoxicity in cerebellar granule cell cultures
inferred← citeshttps://doi.org/10.1002/glia.1072openalex-snapshot
Inflammatory repertoire of Alzheimer's disease and nondemented elderly microglia in vitro
inferred← citeshttps://doi.org/10.1002/glia.1094openalex-snapshot
A role for oligodendrocyte‐derived IGF‐1 in trophic support of cortical neurons
inferred← citeshttps://doi.org/10.1002/glia.20034openalex-snapshot
Heterogeneity and potentiation of AMPA type of glutamate receptors in rat cultured microglia
inferred← citeshttps://doi.org/10.1002/glia.20153openalex-snapshot
Retinoic acid inhibits expression of TNF-? and iNOS in activated rat microglia
inferred← citeshttps://doi.org/10.1002/glia.20210openalex-snapshot
Microglia as potential contributors to motor neuron injury in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1002/glia.20681openalex-snapshot
Vasoactive intestinal peptide protects against β‐amyloid‐induced neurodegeneration by inhibiting microglia activation at multiple levels
inferred← citeshttps://doi.org/10.1002/glia.20973openalex-snapshot
The role and therapeutic potential of monocytic cells in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.22754openalex-snapshot
RNA‐sequencing reveals oligodendrocyte and neuronal transcripts in microglia relevant to central nervous system disease
inferred← citeshttps://doi.org/10.1002/glia.23263openalex-snapshot
Sex differences in the phagocytic and migratory activity of microglia and their impairment by palmitic acid
inferred← citeshttps://doi.org/10.1002/glia.23458openalex-snapshot
Microglia heterogeneity along a spatio–temporal axis: More questions than answers
inferred← citeshttps://doi.org/10.1002/glia.23510openalex-snapshot
Microglial activation occurs late during preclinical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.70029openalex-snapshot
Subtle Alterations in Hippocampal Neuronal Activity Coincide With Early Sex‐Specific Differences in Amyloidosis and Microglia in a Pre‐Symptomatic Mouse Model of Alzheimer‐Like Pathology
inferred← citeshttps://doi.org/10.1002/hlca.200390207openalex-snapshot
Novel Anti‐Inflammatory Constituents of Artocarpus rigida
inferred← citeshttps://doi.org/10.1002/jcp.28871openalex-snapshot
Aquaporin 4: A key player in Parkinson's disease
inferred← citeshttps://doi.org/10.1002/jemt.1121openalex-snapshot
Microglial interaction with β‐amyloid: Implications for the pathogenesis of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/jlb.3mr1021-531ropenalex-snapshot
Microglial activation in Alzheimer's disease: The role of flavonoids and microRNAs
inferred← citeshttps://doi.org/10.1002/jlb.63.6.740openalex-snapshot
Melanocortin peptides inhibit production of proinflammatory cytokines and nitric oxide by activated microglia
inferred← citeshttps://doi.org/10.1002/jlb.65.4.416openalex-snapshot
Insights into the neurodegenerative process of Alzheimer's disease: a role for mononuclear phagocyte-associated inflammation and neurotoxicity
inferred← citeshttps://doi.org/10.1002/jnr.10304openalex-snapshot
Fractalkine and fractalkine receptors in human neurons and glial cells
inferred← citeshttps://doi.org/10.1002/jnr.10469openalex-snapshot
Effects of tumor necrosis factor‐alpha central administration on hippocampal damage in rat induced by amyloid beta‐peptide (25–35)
inferred← citeshttps://doi.org/10.1002/jnr.10706openalex-snapshot
Synergistic expression of inducible nitric oxide synthase by phorbol ester and interferon‐γ is mediated through NF‐κB and ERK in microglial cells
inferred← citeshttps://doi.org/10.1002/jnr.1097openalex-snapshot
Age‐ and concentration‐dependent neuroprotection and toxicity by TNF in cortical neurons from β‐amyloid
inferred← citeshttps://doi.org/10.1002/jnr.1208openalex-snapshot
Vitamin E suppression of microglial activation is neuroprotective
inferred← citeshttps://doi.org/10.1002/jnr.20093openalex-snapshot
Microglia and Alzheimer's disease pathogenesis
inferred← citeshttps://doi.org/10.1002/jnr.20478openalex-snapshot
Immortalized human microglial cell line: Phenotypic expression
inferred← citeshttps://doi.org/10.1002/jnr.20752openalex-snapshot
Lipopolysaccharide-induced microglial activation induces learning and memory deficits without neuronal cell deathin rats
inferred← citeshttps://doi.org/10.1002/jnr.20876openalex-snapshot
Protective effect of D609 against amyloid-beta1–42-induced oxidative modification of neuronal proteins: Redox proteomics study
inferred← citeshttps://doi.org/10.1002/jnr.21011openalex-snapshot
IL‐1β, an immediate early protein secreted by activated microglia, induces iNOS/NO in C6 astrocytoma cells through p38 MAPK and NF‐κB pathways
inferred← citeshttps://doi.org/10.1002/jnr.21663openalex-snapshot
Age‐related changes to tumor necrosis factor receptors affect neuron survival in the presence of beta‐amyloid
inferred← citeshttps://doi.org/10.1002/jnr.22088openalex-snapshot
Desacyl‐ghrelin and synthetic GH‐secretagogues modulate the production of inflammatory cytokines in mouse microglia cells stimulated by β‐amyloid fibrils
inferred← citeshttps://doi.org/10.1002/jnr.22582openalex-snapshot
Involvement of interleukin‐1 in lipopolysaccaride‐induced microglial activation and learning and memory deficits
inferred← citeshttps://doi.org/10.1002/neu.20099openalex-snapshot
Increased microglial activation and astrogliosis after intranasal administration of kainic acid in C57BL/6 mice
inferred← citeshttps://doi.org/10.1002/pmic.200700695openalex-snapshot
Retracted: Mitochondria, the missing link between body and soul: Proteomic prospective evidence
inferred← citeshttps://doi.org/10.1002/ptr.2872openalex-snapshot
Glabridin inhibits lipopolysaccharide‐induced activation of a microglial cell line, BV‐2, by blocking NF‐κB and AP‐1
inferred← citeshttps://doi.org/10.1002/ptr.4810openalex-snapshot
Methylalpinumisoflavone Inhibits Lipopolysaccharide‐Induced Inflammation in Microglial Cells by the NF‐kappaB and MAPK Signaling Pathway
inferred← citeshttps://doi.org/10.1002/smll.202101743openalex-snapshot
Ultrasensitive Multiparameter Phenotyping of Rare Cells Using an Integrated Digital‐Molecular‐Counting Microfluidic Well Plate
inferred← citeshttps://doi.org/10.1002/stem.1125openalex-snapshot
Soluble CCL5 Derived from Bone Marrow-Derived Mesenchymal Stem Cells and Activated by Amyloid β Ameliorates Alzheimer's Disease in Mice by Recruiting Bone Marrow-Induced Microglia Immune Responses
inferred← citeshttps://doi.org/10.1002/stem.277openalex-snapshot
Intracerebral Transplantation of Bone Marrow-Derived Mesenchymal Stem Cells Reduces Amyloid-Beta Deposition and Rescues Memory Deficits in Alzheimer's Disease Mice by Modulation of Immune Responses
inferred← citeshttps://doi.org/10.1006/abbi.1998.0658openalex-snapshot
Measurement and Characterization of Superoxide Generation in Microglial Cells: Evidence for an NADPH Oxidase-Dependent Pathway
inferred← citeshttps://doi.org/10.1006/abio.2000.4928openalex-snapshot
A Cell-Based Method for the Detection of Nanomolar Concentrations of Bioactive Amyloid
inferred← citeshttps://doi.org/10.1006/bbrc.1997.7408openalex-snapshot
Modulation of Nitric Oxide Production in Human Macrophages by Apolipoprotein-E and Amyloid-Beta Peptide
inferred← citeshttps://doi.org/10.1006/bbrc.1998.9237openalex-snapshot
Cholesterol Oxides Induce Programmed Cell Death in Microglial Cells
inferred← citeshttps://doi.org/10.1006/bbrc.1999.1276openalex-snapshot
α-MSH Peptides Inhibit Production of Nitric Oxide and Tumor Necrosis Factor-α by Microglial Cells Activated with β-Amyloid and Interferon γ
inferred← citeshttps://doi.org/10.1006/bbrc.2001.4299openalex-snapshot
BiP/GRP78-Induced Production of Cytokines and Uptake of Amyloid-β(1-42) Peptide in Microglia
inferred← citeshttps://doi.org/10.1006/brbi.2000.0606openalex-snapshot
Age-Dependent Cytokine Responses: Trimethyltin Hippocampal Injury in Wild-Type, APOE Knockout, and APOE4 Mice
inferred← citeshttps://doi.org/10.1006/brbi.2001.0647openalex-snapshot
T-helper-1 and T-helper-2 Responses in Psychiatric Disorders
inferred← citeshttps://doi.org/10.1006/excr.1997.3606openalex-snapshot
Amyloid β-Protein25-35Increases Cellular APP and Inhibits the Secretion of APPs in Human Extraneuronal Cells
inferred← citeshttps://doi.org/10.1006/excr.2002.5655openalex-snapshot
The Physiological Functions of Prion Protein
inferred← citeshttps://doi.org/10.1006/exnr.1996.6373openalex-snapshot
Nitric Oxide Synthase in Reactive Astrocytes Adjacent to β-Amyloid Plaques
inferred← citeshttps://doi.org/10.1006/exnr.1997.6678openalex-snapshot
Rat Microglia Exhibit Increased Density on Alzheimer's Plaquesin Vitro
inferred← citeshttps://doi.org/10.1006/exnr.1998.6920openalex-snapshot
Inflammation and Alzheimer's Disease: Relationships between Pathogenic Mechanisms and Clinical Expression
inferred← citeshttps://doi.org/10.1006/exnr.1998.6977openalex-snapshot
Myeloperoxidase Polymorphism Is Associated with Gender Specific Risk for Alzheimer's Disease
inferred← citeshttps://doi.org/10.1006/exnr.1999.7260openalex-snapshot
Complement Component C1q Modulates the Phagocytosis of Aβ by Microglia
inferred← citeshttps://doi.org/10.1006/exnr.2000.7316openalex-snapshot
A Possible Model of Senile Plaques Using Synthetic Amyloid β-Protein and Rat Glial Culture
inferred← citeshttps://doi.org/10.1006/exnr.2000.7334openalex-snapshot
A Serum Factor Enhances Production of Nitric Oxide and Tumor Necrosis Factor-α From Cultured Microglia
inferred← citeshttps://doi.org/10.1006/exnr.2002.7991openalex-snapshot
Acute Neuroinflammation Exacerbates Excitotoxicity in Rat Hippocampus in Vivo
inferred← citeshttps://doi.org/10.1006/nbdi.2001.0437openalex-snapshot
Generation and Characterization of Immortalized Human Microglial Cell Lines: Expression of Cytokines and Chemokines
inferred← citeshttps://doi.org/10.1006/nbdi.2002.0492openalex-snapshot
β-Amyloid Peptides Decrease Soluble Guanylyl Cyclase Expression in Astroglial Cells
inferred← citeshttps://doi.org/10.1006/nbdi.2002.0519openalex-snapshot
Interactions of Aβ with Endogenous Anti-Inflammatory Agents: A Basis for Chronic Neuroinflammation in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1006/niox.1996.0105openalex-snapshot
Control of Nitric Oxide Production by Transforming Growth Factor-β1: Mechanistic Insights and Potential Relevance to Human Disease
inferred← citeshttps://doi.org/10.1006/niox.2000.0280openalex-snapshot
Phagocytosis is Regulated by Nitric Oxide in Murine Microglia
inferred← citeshttps://doi.org/10.1007/978-0-387-30377-2_6openalex-snapshot
Neuroprotective Mechanisms: Oxidative Stress as a Target for Neuroprotective Therapies in Alzheimer's and Parkinson's Disease
inferred← citeshttps://doi.org/10.1007/978-0-387-70830-0_11openalex-snapshot
Aging and Cognitive Decline: Neuroprotective Strategies
inferred← citeshttps://doi.org/10.1007/978-0-387-72573-4_15openalex-snapshot
Cytokines and Chemokines
inferred← citeshttps://doi.org/10.1007/978-1-4419-7104-3_17openalex-snapshot
Brain Protein Oxidation and Modification for Good or for Bad in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4419-8959-8_18openalex-snapshot
Markers of Excitotoxicity in Patients: Tool for Diagnosis or Therapy?
inferred← citeshttps://doi.org/10.1007/978-1-4612-4116-4_49openalex-snapshot
Therapeutic Strategies in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4614-3804-5_6openalex-snapshot
Beneficial Effects of Curcumin on Neurological Disorders
inferred← citeshttps://doi.org/10.1007/978-1-4614-3903-5_21openalex-snapshot
Adenosine and Multiple Sclerosis
inferred← citeshttps://doi.org/10.1007/978-1-4614-8313-7_4openalex-snapshot
Interactions Between Neurons and Microglia During Neuroinflammation
inferred← citeshttps://doi.org/10.1007/978-1-4614-9123-1_17openalex-snapshot
White Matter Damage in Multiple Sclerosis
inferred← citeshttps://doi.org/10.1007/978-1-4615-0323-1_17openalex-snapshot
Cytokine Regulation in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-1171-7_23openalex-snapshot
Inflammatory Mechanisms in Alzheimer’s Disease: β-Amyloid-Stimulated Proinflammatory Responses are Blocked by PPARγ Agonists
inferred← citeshttps://doi.org/10.1007/978-1-4615-4685-6_13openalex-snapshot
Axotomy-Induced Glial Reactions in Normal and Cytokine Transgenic Mice
inferred← citeshttps://doi.org/10.1007/978-1-4615-4685-6_20openalex-snapshot
Transgenic Models of Tnf Induced Demyelination
inferred← citeshttps://doi.org/10.1007/978-1-4615-4685-6_9openalex-snapshot
Possible Role of Microglial Prostanoids and Free Radicals in Neuroprotection and Neurodegeneration
inferred← citeshttps://doi.org/10.1007/978-1-4615-4709-9_64openalex-snapshot
Ifn-γ Activated Indoleamine 2,3-Dioxygenase Activity in Human Cells is an Antiparasitic and an Antibacterial Effector Mechanism
inferred← citeshttps://doi.org/10.1007/978-1-4615-5405-9_15openalex-snapshot
Homeostatic Effects of Adenosine on Potentially Neurotoxic Glial Cell Activation
inferred← citeshttps://doi.org/10.1007/978-1-4615-5405-9_27openalex-snapshot
Neuroprotective Signal Transduction Pathways of sAPP and TNF
inferred← citeshttps://doi.org/10.1007/978-1-4615-5405-9_3openalex-snapshot
Neuroinflammation and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-6001-2_2openalex-snapshot
Pathological Changes in the Elderly Human Brain
inferred← citeshttps://doi.org/10.1007/978-1-4757-4139-1_1openalex-snapshot
Physiology and Pathophysiology of Microglial Cell Function
inferred← citeshttps://doi.org/10.1007/978-1-4757-4139-1_12openalex-snapshot
Beta Amyloid Protein Clearance and Microglial Activation
inferred← citeshttps://doi.org/10.1007/978-1-4757-4139-1_9openalex-snapshot
Neuroprotective Roles of Microglia in the Central Nervous System
inferred← citeshttps://doi.org/10.1007/978-1-4899-0133-0_13openalex-snapshot
Role of Apolipoprotein E in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4899-0197-2_11openalex-snapshot
Nitric Oxide and Oxidative Damage in the CNS
inferred← citeshttps://doi.org/10.1007/978-1-4939-1429-6_6openalex-snapshot
Neuroprotection Versus Neurotoxicity
inferred← citeshttps://doi.org/10.1007/978-1-4939-3777-6_7openalex-snapshot
Genetic Models of Alzheimer’s Disease: The Influence of Apolipoprotein E allele Isoforms on Behaviour in Laboratory Animals
inferred← citeshttps://doi.org/10.1007/978-1-59259-105-3_20openalex-snapshot
Activated Neuroglia in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-108-4_14openalex-snapshot
Microglia in the CNS
inferred← citeshttps://doi.org/10.1007/978-1-59259-297-5_12openalex-snapshot
Neuroinflammatory Environments Promote Amyloid-ß Deposition and Posttranslational Modification
inferred← citeshttps://doi.org/10.1007/978-1-59259-297-5_5openalex-snapshot
Neurotoxic Mechanisms of Nitric Oxide
inferred← citeshttps://doi.org/10.1007/978-1-59259-315-6_10openalex-snapshot
Protective Effect of Cyclosporin A on Glial Activation and White Matter Alterations Induced by Chronic Cerebral Hypoperfusion
inferred← citeshttps://doi.org/10.1007/978-1-59259-315-6_7openalex-snapshot
Immunosuppressants and Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-471-9_5openalex-snapshot
Apoptosis and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-471-9_8openalex-snapshot
Calcium Homeostasis and Free Radical Metabolism as Convergence Points in the Pathophysiology of Dementia
inferred← citeshttps://doi.org/10.1007/978-1-59259-473-3_1openalex-snapshot
Roles of CNS Macrophages in Neurodegeneration
inferred← citeshttps://doi.org/10.1007/978-1-59259-473-3_10openalex-snapshot
Reactive Oxygen Involvement in Neurodegenerative Pathways
inferred← citeshttps://doi.org/10.1007/978-1-59259-473-3_13openalex-snapshot
The Role of Glial Nitric Oxide in Neurotoxicity and Central Nervous System Diseases
inferred← citeshttps://doi.org/10.1007/978-1-59259-473-3_2openalex-snapshot
Inflammatory Markers in Chronic Neurodegenerative Disorders with Emphasis on Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-473-3_6openalex-snapshot
Inflammatory Mechanisms of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-691-1_5openalex-snapshot
An Intracerebral Tumor Necrosis Factor-α Infusion Model for Inflammation in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-888-5_7openalex-snapshot
Functional Imaging and Psychopathological Consequences of Inflammation in Alzheimer’s Dementia
inferred← citeshttps://doi.org/10.1007/978-1-59745-422-3_6openalex-snapshot
Impact of A β and Tau on Cognition in Mouse Models of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-60761-898-0_7openalex-snapshot
Pathological Validation of Animal Models of Dementia
inferred← citeshttps://doi.org/10.1007/978-1-84628-440-3_2openalex-snapshot
The Aβcentric Pathway of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-84628-440-3_7openalex-snapshot
Aβ Variants and Their Impact on Amyloid Formation and Alzheimer’s Disease Progression
inferred← citeshttps://doi.org/10.1007/978-3-030-18323-3_13openalex-snapshot
Biological Psychiatry and Psychopharmacology
inferred← citeshttps://doi.org/10.1007/978-3-031-68237-7_22openalex-snapshot
Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-032-04239-2_4openalex-snapshot
The Neuroprotective Effect of Garlic
inferred← citeshttps://doi.org/10.1007/978-3-0348-8350-4_11openalex-snapshot
Microglia
inferred← citeshttps://doi.org/10.1007/978-3-0348-8350-4_8openalex-snapshot
Proinflammatory actions of derivatives of the β amyloid precursor protein
inferred← citeshttps://doi.org/10.1007/978-3-319-40764-7_10openalex-snapshot
Age-Dependent Changes in the Activation and Regulation of Microglia
inferred← citeshttps://doi.org/10.1007/978-3-319-44022-4_12openalex-snapshot
Macrophages, Microglia and Dendritic Cell Function
inferred← citeshttps://doi.org/10.1007/978-3-319-44022-4_17openalex-snapshot
Cytokines and Chemokines
inferred← citeshttps://doi.org/10.1007/978-3-319-44022-4_30openalex-snapshot
Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-540-76330-7_12openalex-snapshot
Long-Term Potentiation and Aβ: Targeting Aβ Species, Cellular Mechanisms and Putative Receptors
inferred← citeshttps://doi.org/10.1007/978-3-642-37922-2_69openalex-snapshot
Serum miR-124 and TNF-α are Biomarkers of Ischemic Cerebrovascular Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-59643-8_4openalex-snapshot
Cytokine Communication Between Neurons and Glia and the Pathogenesis of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-59643-8_6openalex-snapshot
Mechanistic and Metaphorical Connections Between NF-KB and the Secreted Alzheimer’s β-Amyloid Precursor Protein
inferred← citeshttps://doi.org/10.1007/978-3-662-03248-0_2openalex-snapshot
The Vascular Dementias and Cerebrovascular Involvement in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-662-03248-0_8openalex-snapshot
Cytokines in Alzheimer’s and Other Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/978-3-662-05073-6_3openalex-snapshot
Elucidating Molecular Mechanisms of Alzheimer’s Disease in Microglial Cultures
inferred← citeshttps://doi.org/10.1007/978-3-662-05073-6_6openalex-snapshot
BSE, Scrapie, and vCJD: Infectious Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/978-3-662-06274-6_3openalex-snapshot
Reducing Neuroinflammation
inferred← citeshttps://doi.org/10.1007/978-3-662-12579-3_7openalex-snapshot
Role for TNF in CNS Inflammation, Demyelination and Neurodegeneration Studied in Transgenic Mice
inferred← citeshttps://doi.org/10.1007/978-3-662-22516-5_5openalex-snapshot
Oxidative Stress in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-7091-6467-9_23openalex-snapshot
Perspectives in clinical Alzheimer’s disease research and the development of antidementia drugs
inferred← citeshttps://doi.org/10.1007/978-3-7091-6781-6_11openalex-snapshot
Cytokines in CNS disorders: neurotoxicity versus neuroprotection
inferred← citeshttps://doi.org/10.1007/978-3-7091-6781-6_30openalex-snapshot
Cerebrolysin® reduces microglial activation in vivo and in vitro: a potential mechanism of neuroprotection
inferred← citeshttps://doi.org/10.1007/978-3-7091-6842-4_19openalex-snapshot
Cell death in prion disease
inferred← citeshttps://doi.org/10.1007/978-3-7091-7508-8_16openalex-snapshot
Pathological immuno-reactions of glial cells in Alzheimer’s disease and possible sites of interference
inferred← citeshttps://doi.org/10.1007/978-4-431-54502-6_5openalex-snapshot
Roles of Microglia in Spinal Cord Injury
inferred← citeshttps://doi.org/10.1007/978-81-322-2035-0_11openalex-snapshot
Free Radicals and Oxidative Stress in Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1007/978-94-024-1079-2_12openalex-snapshot
Taurine Chloramine Suppresses LPS-Induced Neuroinflammatory Responses through Nrf2-Mediated Heme Oxygenase-1 Expression in Mouse BV2 Microglial Cells
inferred← citeshttps://doi.org/10.1007/978-94-024-1088-4_7openalex-snapshot
TRPC Channels and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-13-9636-6_13openalex-snapshot
Neurobiological Mechanisms Involved in the Pathogenesis of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-15-6883-1_2openalex-snapshot
Neurons and Plasticity: What Do Glial Cells Have to Do with This?
inferred← citeshttps://doi.org/10.1007/978-981-32-9717-3_9openalex-snapshot
Co-signaling Molecules in Neurological Diseases
inferred← citeshttps://doi.org/10.1007/978-981-96-4926-6_5openalex-snapshot
Metal Ion Neurotoxicity—Role of Pro-inflammatory Mediators
inferred← citeshttps://doi.org/10.1007/bf02436085openalex-snapshot
Free radical theory of aging: Alzheimer’s disease pathogenesis
inferred← citeshttps://doi.org/10.1007/bf02740619openalex-snapshot
The effects of interferon-γ on the central nervous system
inferred← citeshttps://doi.org/10.1007/bf03033140openalex-snapshot
In vitro regulation of rat derived microglia
inferred← citeshttps://doi.org/10.1007/bf03033792openalex-snapshot
Oxidative neuropathology and putative chemical entities for alzheimer’s disease: Neuroprotective effects of salen-manganese catalytic anti-oxidants
inferred← citeshttps://doi.org/10.1007/bf03033906openalex-snapshot
Glial cell dysregulation: a new perspective on Alzheimer disease
inferred← citeshttps://doi.org/10.1007/bf03033980openalex-snapshot
Cytokine production by a human microglial cell line: Effects of ßamyloid and α-melanocyte-stimulating hormone
inferred← citeshttps://doi.org/10.1007/s00011-007-6088-4openalex-snapshot
Nitric oxide and pro-inflammatory cytokines correlate with pain intensity in chronic pain patients
inferred← citeshttps://doi.org/10.1007/s00018-012-1015-4openalex-snapshot
Targeting microglia-mediated neurotoxicity: the potential of NOX2 inhibitors
inferred← citeshttps://doi.org/10.1007/s00018-014-1670-8openalex-snapshot
Microglia and inflammation: conspiracy, controversy or control?
inferred← citeshttps://doi.org/10.1007/s000180050428openalex-snapshot
Molecular pathogenesis of apolipoprotein E-mediated amyloidosis in late-onset Alzheimer's disease
inferred← citeshttps://doi.org/10.1007/s00108-002-0636-3openalex-snapshot
Ätiologie, Pathologie und Diagnostik der Prionkrankheiten des Menschen
inferred← citeshttps://doi.org/10.1007/s001090050097openalex-snapshot
Activation of microglia in the brains of humans with heart disease and hypercholesterolemic rabbits
inferred← citeshttps://doi.org/10.1007/s001090050119openalex-snapshot
Clinical implications of the involvement of tPA in neuronal cell death
inferred← citeshttps://doi.org/10.1007/s001140100237openalex-snapshot
Alzheimer's disease: molecular concepts and therapeutic targets
inferred← citeshttps://doi.org/10.1007/s00204-017-1939-4openalex-snapshot
High bioavailability curcumin: an anti-inflammatory and neurosupportive bioactive nutrient for neurodegenerative diseases characterized by chronic neuroinflammation
inferred← citeshttps://doi.org/10.1007/s00213-011-2386-1openalex-snapshot
Xanthoceraside attenuates amyloid β peptide25–35-induced learning and memory impairments in mice
inferred← citeshttps://doi.org/10.1007/s00216-013-6904-3openalex-snapshot
Advances in electrochemical detection for study of neurodegenerative disorders
inferred← citeshttps://doi.org/10.1007/s00221-003-1389-5openalex-snapshot
Microglial activation induces cell death, inhibits neurite outgrowth and causes neurite retraction of differentiated neuroblastoma cells
inferred← citeshttps://doi.org/10.1007/s00281-016-0615-8openalex-snapshot
Potential importance of B cells in aging and aging-associated neurodegenerative diseases
inferred← citeshttps://doi.org/10.1007/s00401-002-0624-7openalex-snapshot
Amyloid β plaque-associated proteins C1q and SAP enhance the Aβ1–42 peptide-induced cytokine secretion by adult human microglia in vitro
inferred← citeshttps://doi.org/10.1007/s00401-009-0556-6openalex-snapshot
Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease
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