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Injection of Recombinant Human Type VII Collagen Corrects the Disease Phenotype in a Murine Model of Dystrophic Epidermolysis Bullosa
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archderm.1993.01680270149037openalex-snapshot
Epidermolysis Bullosa: Basic and Clinical Aspects
inferredcites →https://doi.org/10.1001/jama.299.10.1195-bopenalex-snapshot
Fitzpatrick’s Dermatology in General Medicine
inferredcites →https://doi.org/10.1002/(sici)1521-2254(200001/02)2:1<41::aid-jgm79>3.0.co;2-popenalex-snapshot
Long-term reversal of hypercholesterolemia in low density lipoprotein receptor (LDLR)-deficient mice by adenovirus-mediated LDLR gene transfer combined with CD154 blockade
inferredcites →https://doi.org/10.1002/art.1780401002openalex-snapshot
CD40‐CD40 ligand interaction in autoimmune disease
inferredcites →https://doi.org/10.1002/eji.1830250243openalex-snapshot
Studies on the interdependence of gp39 and B7 expression and function during antigen‐specific immune responses
inferredcites →https://doi.org/10.1007/978-3-211-99225-8_6openalex-snapshot
Epidermolysis Bullosa Acquisita
inferredcites →https://doi.org/10.1007/bf00370713openalex-snapshot
Molecular basis for the dystrophic forms of epidermolysis bullosa: mutations in the type VII collagen gene
inferredcites →https://doi.org/10.1016/0076-6879(94)45004-8openalex-snapshot
[2] Fibrillin: Monomers and microfibrils
inferredcites →https://doi.org/10.1016/j.ymthe.2004.05.016openalex-snapshot
Intradermal Injection of Lentiviral Vectors Corrects Regenerated Human Dystrophic Epidermolysis Bullosa Skin Tissue in Vivo
inferredcites →https://doi.org/10.1016/s0021-9258(17)31984-1openalex-snapshot
Cloning of human type VII collagen. Complete primary sequence of the alpha 1(VII) chain and identification of intragenic polymorphisms.
inferredcites →https://doi.org/10.1016/s0021-9258(19)76484-9openalex-snapshot
Anchoring fibrils contain the carboxyl-terminal globular domain of type VII procollagen, but lack the amino-terminal globular domain.
inferredcites →https://doi.org/10.1016/s0945-053x(98)00005-5openalex-snapshot
Mutation analysis and molecular genetics of epidermolysis bullosa
inferredcites →https://doi.org/10.1038/ng1041openalex-snapshot
Restoration of type VII collagen expression and function in dystrophic epidermolysis bullosa
inferredcites →https://doi.org/10.1038/nm1063openalex-snapshot
Injection of recombinant human type VII collagen restores collagen function in dystrophic epidermolysis bullosa
inferredcites →https://doi.org/10.1038/nm766openalex-snapshot
Stable nonviral genetic correction of inherited human skin disease
inferredcites →https://doi.org/10.1038/sj.jid.5700254openalex-snapshot
Induction of Epidermolysis Bullosa Acquisita in Mice by Passive Transfer of Autoantibodies from Patients
inferredcites →https://doi.org/10.1038/sj.mt.6300041openalex-snapshot
Intravenously Injected Human Fibroblasts Home to Skin Wounds, Deliver Type VII Collagen, and Promote Wound Healing
inferredcites →https://doi.org/10.1046/j.1523-1747.2003.12090.xopenalex-snapshot
Suppression of the Immune Response Against Exogenous Desmoglein 3 in Desmoglein 3 Knockout Mice: An Implication for Gene Therapy
inferredcites →https://doi.org/10.1046/j.1523-1747.2003.12571.xopenalex-snapshot
Normal and Gene-Corrected Dystrophic Epidermolysis Bullosa Fibroblasts Alone Can Produce Type VII Collagen at the Basement Membrane Zone
inferredcites →https://doi.org/10.1056/nejm197008272830903openalex-snapshot
Hydroxyprolinemia* an Apparently Harmless Familial Metabolic Disorder
inferredcites →https://doi.org/10.1056/nejm198404193101602openalex-snapshot
Identification of the Skin Basement-Membrane Autoantigen in Epidermolysis Bullosa Acquisita
inferredcites →https://doi.org/10.1073/pnas.88.16.6931openalex-snapshot
Human type VII collagen: cDNA cloning and chromosomal mapping of the gene.
inferredcites →https://doi.org/10.1074/jbc.m108779200openalex-snapshot
The Recombinant Expression of Full-length Type VII Collagen and Characterization of Molecular Mechanisms Underlying Dystrophic Epidermolysis Bullosa
inferredcites →https://doi.org/10.1083/jcb.103.4.1577openalex-snapshot
Type VII collagen is a major structural component of anchoring fibrils.
inferredcites →https://doi.org/10.1083/jcb.104.3.611openalex-snapshot
Type VII collagen forms an extended network of anchoring fibrils.
inferredcites →https://doi.org/10.1084/jem.177.4.925openalex-snapshot
Activated T cells induce expression of B7/BB1 on normal or leukemic B cells through a CD40-dependent signal.
inferredcites →https://doi.org/10.1097/00007890-200201151-00012openalex-snapshot
THE CD154-CD40 COSTIMULATORY PATHWAY IN TRANSPLANTATION
inferredcites →https://doi.org/10.1111/1523-1747.ep12259692openalex-snapshot
Differentiating Anti-Lamina Lucida and Anti-Sublamina Densa Anti-BMZ Antibodies by Indirect Immunofluorescence on 1.0 M Sodium Chloride-Separated Skin
inferredcites →https://doi.org/10.1111/1523-1747.ep12285634openalex-snapshot
Development of an ELISA for Rapid Detection of Anti-Type VII Collagen Autoantibodies in Epidermolysis Bullosa Acquisita
inferredcites →https://doi.org/10.1111/1523-1747.ep12332300openalex-snapshot
Ultraviolet A Irradiation Upregulates Type VII Collagen Expression in Human Dermal Fibroblasts.
inferredcites →https://doi.org/10.1111/1523-1747.ep12349019openalex-snapshot
Cloning of Mouse Type VII Collagen Reveals Evolutionary Conservation of Functional Protein Domains and Genomic Organization
inferredcites →https://doi.org/10.1111/1523-1747.ep12365129openalex-snapshot
Type VII Collagen, Anchoring Fibrils, and Epidermolysis Bullosa
inferredcites →https://doi.org/10.1111/1523-1747.ep12472291openalex-snapshot
Autoantibodies to Type VII Collagen Recognize Epitopes in a Fibronectin-Like Region of the Noncollagenous (NC1) Domain
inferredcites →https://doi.org/10.1111/1523-1747.ep12598050openalex-snapshot
The Epidermal-Dermal Junction
inferredcites →https://doi.org/10.1111/j.0022-202x.2005.23702.xopenalex-snapshot
Evidence that Anti-Type VII Collagen Antibodies Are Pathogenic and Responsible for the Clinical, Histological, and Immunological Features of Epidermolysis Bullosa Acquisita
inferredcites →https://doi.org/10.1126/science.1106209openalex-snapshot
Type VII Collagen Is Required for Ras-Driven Human Epidermal Tumorigenesis
inferredcites →https://doi.org/10.1126/science.7689748openalex-snapshot
Prevention of Collagen-Induced Arthritis with an Antibody to gp39, the Ligand for CD40
inferredcites →https://doi.org/10.1128/jvi.70.9.6370-6377.1996openalex-snapshot
Transient subversion of CD40 ligand function diminishes immune responses to adenovirus vectors in mouse liver and lung tissues
inferredcites →https://doi.org/10.1128/jvi.73.4.3424-3429.1999openalex-snapshot
Systemic and Central Nervous System Correction of Lysosomal Storage in Mucopolysaccharidosis Type VII Mice
inferredcites →https://doi.org/10.1136/jmg.2006.045302openalex-snapshot
Epidermolysis bullosa. II. Type VII collagen mutations and phenotype–genotype correlations in the dystrophic subtypes
inferredcites →https://doi.org/10.1172/jci113373openalex-snapshot
Epidermolysis bullosa acquisita antigen is the globular carboxyl terminus of type VII procollagen.
inferredcites →https://doi.org/10.1172/jci115938openalex-snapshot
Molecular genetics of the cutaneous basement membrane zone. Perspectives on epidermolysis bullosa and other blistering skin diseases.
inferredcites →https://doi.org/10.1172/jci116774openalex-snapshot
Epitope mapping of type VII collagen. Identification of discrete peptide sequences recognized by sera from patients with acquired epidermolysis bullosa.
inferredcites →https://doi.org/10.1172/jci17193openalex-snapshot
Injection of genetically engineered fibroblasts corrects regenerated human epidermolysis bullosa skin tissue
inferredcites →https://doi.org/10.1242/jcs.112.21.3641openalex-snapshot
Targeted inactivation of the type VII collagen gene (Col7a1) in mice results in severe blistering phenotype: a model for recessive dystrophic epidermolysis bullosa
inferredcites →https://doi.org/10.7326/0003-4819-106-6-923_2openalex-snapshot
Dermatology in General Medicine.
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Epidermolysis Bullosa and Kindler Syndrome
inferred← citeshttps://doi.org/10.1002/9781444345384.ch140openalex-snapshot
Skin Gene and Cell Therapy
inferred← citeshttps://doi.org/10.1002/jbm.a.36519openalex-snapshot
A 3D in vitro model of the dermoepidermal junction amenable to mechanical testing
inferred← citeshttps://doi.org/10.1002/path.2973openalex-snapshot
Overexpression of the Flii gene increases dermal–epidermal blistering in an autoimmune ColVII mouse model of epidermolysis bullosa acquisita
inferred← citeshttps://doi.org/10.1002/sctm.17-0182openalex-snapshot
Efficacy of Human Placental-Derived Stem Cells in Collagen VII Knockout (Recessive Dystrophic Epidermolysis Bullosa) Animal Model
inferred← citeshttps://doi.org/10.1007/978-1-4471-2191-6_34openalex-snapshot
Desperate Measures for Desperate Patients: Translational Research in Epidermolysis Bullosa
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Cell- and Protein-Based Therapy Approaches for Epidermolysis Bullosa
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Multisystem Management of Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1007/978-3-662-45698-9_1openalex-snapshot
Biology of the Basement Membrane Zone
inferred← citeshttps://doi.org/10.1007/978-3-662-45698-9_76openalex-snapshot
Future Therapies for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1007/s00105-012-2406-zopenalex-snapshot
Neues über hereditäre, blasenbildende Hauterkrankungen
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Multiple Facetten der genetisch bedingten Hautfragilität
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Basement membranes and human disease
inferred← citeshttps://doi.org/10.1007/s13353-024-00839-2openalex-snapshot
Gene-edited cells: novel allogeneic gene/cell therapy for epidermolysis bullosa
inferred← citeshttps://doi.org/10.1007/s13555-021-00561-5openalex-snapshot
Clinical Perspectives of Gene-Targeted Therapies for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1007/s40257-021-00626-3openalex-snapshot
Investigational Treatments for Epidermolysis Bullosa
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Bullous Disorders of Childhood
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10.1016/b978-0-7020-6275-9.00032-5
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Inherited and Acquired Blistering Diseases
inferred← citeshttps://doi.org/10.1016/j.addr.2021.113842openalex-snapshot
Highly branched poly(β-amino ester)s for gene delivery in hereditary skin diseases
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Molecular organization of the basement membrane zone
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Role of Dermal-Epidermal Basement Membrane Zone in Skin, Cancer, and Developmental Disorders
inferred← citeshttps://doi.org/10.1016/j.det.2009.10.016openalex-snapshot
Animal Models of Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1016/j.det.2009.10.020openalex-snapshot
Dystrophic Epidermolysis Bullosa: Pathogenesis and Clinical Features
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Gene Therapy for Recessive Dystrophic Epidermolysis Bullosa
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New Findings in Genodermatoses
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A phase II randomized vehicle-controlled trial of intradermal allogeneic fibroblasts for recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1016/j.jdermsci.2024.05.004openalex-snapshot
Histological and molecular restoration of type VII collagen in Recessive dystrophic epidermolysis bullosa mouse skin by topical injection of keratinocyte-like cells differentiated from human adipose-derived mesenchymal stromal cells
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Progress toward Treatment and Cure of Epidermolysis Bullosa: Summary of the DEBRA International Research Symposium EB2015
inferred← citeshttps://doi.org/10.1016/j.jid.2016.02.002openalex-snapshot
Collagen VII Half-Life at the Dermal-Epidermal Junction Zone: Implications for Mechanisms and Therapy of Genodermatoses
inferred← citeshttps://doi.org/10.1016/j.jid.2016.02.811openalex-snapshot
Gene-Corrected Fibroblast Therapy for Recessive Dystrophic Epidermolysis Bullosa using a Self-Inactivating COL7A1 Retroviral Vector
inferred← citeshttps://doi.org/10.1016/j.jid.2016.03.005openalex-snapshot
Type VII Collagen Replacement Therapy in Recessive Dystrophic Epidermolysis Bullosa—How Much, How Often?
inferred← citeshttps://doi.org/10.1016/j.jid.2016.05.115openalex-snapshot
miR-29 Regulates Type VII Collagen in Recessive Dystrophic Epidermolysis Bullosa
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The Molecular Revolution in Cutaneous Biology: Emerging Landscape in Genomic Dermatology: New Mechanistic Ideas, Gene Editing, and Therapeutic Breakthroughs
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Gene Therapy for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1016/j.jid.2019.05.031openalex-snapshot
Human Mesenchymal Stromal Cells Engineered to Express Collagen VII Can Restore Anchoring Fibrils in Recessive Dystrophic Epidermolysis Bullosa Skin Graft Chimeras
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Disorders of the cutaneous basement membrane zone—The paradigm of epidermolysis bullosa
inferred← citeshttps://doi.org/10.1016/j.matbio.2017.12.010openalex-snapshot
Therapies for genetic extracellular matrix diseases of the skin
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Around the collagen triple helix: an introduction to studying associated genetic and acquired diseases
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Molecular therapies for heritable blistering diseases
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Novel eRF3a degrader enhances gentamicin-induced premature termination codon readthrough in epidermolysis bullosa
inferred← citeshttps://doi.org/10.1016/j.trsl.2015.05.008openalex-snapshot
Gene editing toward the use of autologous therapies in recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1016/j.xjidi.2023.100191openalex-snapshot
Cytosine Deaminase Base Editing to Restore COL7A1 in Dystrophic Epidermolysis Bullosa Human: Murine Skin Model
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Progress in skin gene therapy: From the inside and out
inferred← citeshttps://doi.org/10.1016/s0140-6736(13)61897-8openalex-snapshot
Allogeneic blood and bone marrow cells for the treatment of severe epidermolysis bullosa: repair of the extracellular matrix
inferred← citeshttps://doi.org/10.1038/gt.2010.36openalex-snapshot
Immune reactivity to type VII collagen: implications for gene therapy of recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1038/gt.2011.135openalex-snapshot
Early intra-amniotic gene transfer using lentiviral vector improves skin blistering phenotype in a murine model of Herlitz junctional epidermolysis bullosa
inferred← citeshttps://doi.org/10.1038/jid.2010.75openalex-snapshot
Animal Models of Epidermolysis Bullosa: Update 2010
inferred← citeshttps://doi.org/10.1038/jid.2010.90openalex-snapshot
Progress in Epidermolysis Bullosa Research: Toward Treatment and Cure
inferred← citeshttps://doi.org/10.1038/jid.2011.172openalex-snapshot
Correction of Dog Dystrophic Epidermolysis Bullosa by Transplantation of Genetically Modified Epidermal Autografts
inferred← citeshttps://doi.org/10.1038/jid.2012.457openalex-snapshot
Topically Applied Flightless I Neutralizing Antibodies Improve Healing of Blistered Skin in a Murine Model of Epidermolysis Bullosa Acquisita
inferred← citeshttps://doi.org/10.1038/jid.2013.10openalex-snapshot
Intravenously Injected Recombinant Human Type VII Collagen Homes to Skin Wounds and Restores Skin Integrity of Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1038/jid.2013.127openalex-snapshot
Progress in Epidermolysis Bullosa Research: Summary of DEBRA International Research Conference 2012
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Systemic Protein Therapy for Recessive Dystrophic Epidermolysis Bullosa: How Far Are We from Clinical Translation?
inferred← citeshttps://doi.org/10.1038/jid.2013.475openalex-snapshot
De Novo Anti-Type VII Collagen Antibodies in Patients with Recessive Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1038/jid.2015.149openalex-snapshot
Recessive Dystrophic Epidermolysis Bullosa: Advances in the Laboratory Leading to New Therapies
inferred← citeshttps://doi.org/10.1038/jid.2015.291openalex-snapshot
Intravenously Administered Recombinant Human Type VII Collagen Derived from Chinese Hamster Ovary Cells Reverses the Disease Phenotype in Recessive Dystrophic Epidermolysis Bullosa Mice
inferred← citeshttps://doi.org/10.1038/jid.2015.364openalex-snapshot
Lentiviral Engineered Fibroblasts Expressing Codon-Optimized COL7A1 Restore Anchoring Fibrils in RDEB
inferred← citeshttps://doi.org/10.1038/mt.2008.262openalex-snapshot
Can Type VII Collagen Injections Cure Dystrophic Epidermolysis Bullosa?
inferred← citeshttps://doi.org/10.1038/mt.2009.144openalex-snapshot
Mechanisms of Fibroblast Cell Therapy for Dystrophic Epidermolysis Bullosa: High Stability of Collagen VII Favors Long-term Skin Integrity
inferred← citeshttps://doi.org/10.1038/mt.2013.87openalex-snapshot
Topical Application of Recombinant Type VII Collagen Incorporates Into the Dermal–Epidermal Junction and Promotes Wound Closure
inferred← citeshttps://doi.org/10.1038/mt.2014.140openalex-snapshot
Aminoglycosides Restore Full-length Type VII Collagen by Overcoming Premature Termination Codons: Therapeutic Implications for Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1038/mt.2015.47openalex-snapshot
From Marrow to Matrix: Novel Gene and Cell Therapies for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1038/mt.2016.92openalex-snapshot
Analysis of the functional consequences of targeted exon deletion in COL7A1 reveals prospects for dystrophic epidermolysis bullosa therapy
inferred← citeshttps://doi.org/10.1038/mtna.2016.3openalex-snapshot
A Gene Gun-mediated Nonviral RNA trans-splicing Strategy for Col7a1 Repair
inferred← citeshttps://doi.org/10.1038/mtna.2016.87openalex-snapshot
Antisense Oligonucleotide-mediated Exon Skipping as a Systemic Therapeutic Approach for Recessive Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1038/s41591-022-01737-yopenalex-snapshot
In vivo topical gene therapy for recessive dystrophic epidermolysis bullosa: a phase 1 and 2 trial
inferred← citeshttps://doi.org/10.1038/skinbio.2012.2openalex-snapshot
Molecular Heterogeneity of Blistering Disorders: The Paradigm of Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1038/skinbio.2012.9openalex-snapshot
Molecular Therapeutics for Heritable Skin Diseases
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A knot polymer mediated non-viral gene transfection for skin cells
inferred← citeshttps://doi.org/10.1042/ebc20180071openalex-snapshot
Basement membrane collagens and disease mechanisms
inferred← citeshttps://doi.org/10.1056/nejmoa0910501openalex-snapshot
Bone Marrow Transplantation for Recessive Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1073/pnas.1614775114openalex-snapshot
Efficient in vivo gene editing using ribonucleoproteins in skin stem cells of recessive dystrophic epidermolysis bullosa mouse model
inferred← citeshttps://doi.org/10.1080/13543776.2019.1608181openalex-snapshot
Patent landscape of molecular and cellular targeted therapies for recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1080/14728214.2020.1839049openalex-snapshot
Emerging drugs for the treatment of epidermolysis bullosa
inferred← citeshttps://doi.org/10.1089/hum.2010.023openalex-snapshot
Long-Term Type VII Collagen Restoration to Human Epidermolysis Bullosa Skin Tissue
inferred← citeshttps://doi.org/10.1097/01.ewx.0000437657.38804.96openalex-snapshot
Inherited epidermolysis bullosa
inferred← citeshttps://doi.org/10.1097/mop.0b013e32833bb74fopenalex-snapshot
Inherited epidermolysis bullosa: recent basic and clinical advances
inferred← citeshttps://doi.org/10.1101/cshperspect.a015156openalex-snapshot
Cell Therapy in Dermatology
inferred← citeshttps://doi.org/10.1101/cshperspect.a035667openalex-snapshot
Toward Combined Cell and Gene Therapy for Genodermatoses
inferred← citeshttps://doi.org/10.1101/cshperspect.a041229openalex-snapshot
Stairways to Advanced Therapies for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1111/bjd.12715openalex-snapshot
High levels of type VII collagen expression in recessive dystrophic epidermolysis bullosa cutaneous squamous cell carcinoma keratinocytes increases PI3K and MAPK signalling, cell migration and invasion
inferred← citeshttps://doi.org/10.1111/bjd.14168openalex-snapshot
Ventricular dysfunction and aortic dilation in patients with recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1111/bjd.20910openalex-snapshot
The potential of gene therapy for recessive dystrophic epidermolysis bullosa*
inferred← citeshttps://doi.org/10.1111/exd.12084openalex-snapshot
One goal, different strategies – molecular and cellular approaches for the treatment of inherited skin fragility disorders
inferred← citeshttps://doi.org/10.1111/exd.12246openalex-snapshot
Gene therapy: pursuing restoration of dermal adhesion in recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1111/exd.12666openalex-snapshot
Therapies for inherited skin fragility disorders
inferred← citeshttps://doi.org/10.1111/j.1365-4632.2011.05130.xopenalex-snapshot
Progress in epidermolysis bullosa: summary of a workshop in CILAD‐2010*
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Autoimmunity against type VII collagen in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1111/j.1749-6632.2010.05463.xopenalex-snapshot
Inherited epidermolysis bullosa: past, present, and future
inferred← citeshttps://doi.org/10.1111/pde.12157openalex-snapshot
Junctional Epidermolysis Bullosa Incidence and Survival: 5‐Year Experience of the Dystrophic Epidermolysis Bullosa Research Association of America (DebRA) Nurse Educator, 2007 to 2011
inferred← citeshttps://doi.org/10.1146/annurev-genom-090413-025540openalex-snapshot
The Genetics of Skin Fragility
inferred← citeshttps://doi.org/10.11606/d.87.2019.tde-05012021-135737openalex-snapshot
Produção em escala laboratorial e caracterização de variantes do colágeno tipo VII humano recombinante resistentes à clivagem por metaloproteinases de matriz
inferred← citeshttps://doi.org/10.1172/jci.insight.126243openalex-snapshot
Safety and early efficacy outcomes for lentiviral fibroblast gene therapy in recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1172/jci92707openalex-snapshot
Gentamicin induces functional type VII collagen in recessive dystrophic epidermolysis bullosa patients
inferred← citeshttps://doi.org/10.1182/blood-2008-12-189712openalex-snapshot
Bone marrow cells can manipulate healing
inferred← citeshttps://doi.org/10.1186/1750-1172-5-12openalex-snapshot
Inherited epidermolysis bullosa
inferred← citeshttps://doi.org/10.1186/1750-1172-6-49openalex-snapshot
The potential investment impact of improved access to accelerated approval on the development of treatments for low prevalence rare diseases
inferred← citeshttps://doi.org/10.1186/scrt511openalex-snapshot
Preconditioning of mesenchymal stem cells for improved transplantation efficacy in recessive dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1201/b18002-52openalex-snapshot
Cutaneous Cell- and Gene-Based Therapies for Inherited and Acquired Skin Disorders
inferred← citeshttps://doi.org/10.12703/p6-35openalex-snapshot
Advances in understanding and treating dystrophic epidermolysis bullosa
inferred← citeshttps://doi.org/10.1371/journal.pone.0137639openalex-snapshot
Lysyl Hydroxylase 3 Localizes to Epidermal Basement Membrane and Is Reduced in Patients with Recessive Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1517/17460441.2013.761202openalex-snapshot
Animal models of skin disease for drug discovery
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Management of epidermolysis bullosa
inferred← citeshttps://doi.org/10.15252/emmm.201404353openalex-snapshot
A single epidermal stem cell strategy for safe ex vivo gene therapy
inferred← citeshttps://doi.org/10.1590/s0365-05962013000200001openalex-snapshot
Inherited epidermolysis bullosa: clinical and therapeutic aspects
inferred← citeshttps://doi.org/10.17116/klinderma202524061822openalex-snapshot
First experience of the application of beremagene geperpavec genetically engineered drug for treatment of congenital dystrophic epidermolysis bullosa in the Russian Federation
inferred← citeshttps://doi.org/10.21236/ada590831openalex-snapshot
Stem-cell Based Therapies for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.2217/rme.13.33openalex-snapshot
The Long and Winding Road That Leads to a Cure for Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.2302/kjm.2014-0013-reopenalex-snapshot
Advances in Gene/Cell Therapy in Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.2336/nishinihonhifu.76.230openalex-snapshot
The Understanding of Genodermatores Based on the Biochemical Pathogenesis (2)
inferred← citeshttps://doi.org/10.2492/inflammregen.31.282openalex-snapshot
Regenerative medicine for severe congenital skin disorders: restoration of deficient skin component proteins by stem cell therapy
inferred← citeshttps://doi.org/10.25208/vdv551-2020-96-1-10-17openalex-snapshot
Congenital epidermolysis bullosa: modern methods of diagnosis and therapy. Prospects for regenerative medicine
inferred← citeshttps://doi.org/10.3109/07853890.2013.866441openalex-snapshot
Inherited blistering skin diseases: underlying molecular mechanisms and emerging therapies
inferred← citeshttps://doi.org/10.3109/15419061.2013.854334openalex-snapshot
A Skin-depth Analysis of Integrins: Role of the Integrin Network in Health and Disease
inferred← citeshttps://doi.org/10.3389/fbioe.2026.1901459openalex-snapshot
Gene therapy for genodermatoses at the crossroads of innovation and clinical translation
inferred← citeshttps://doi.org/10.3389/fgene.2021.658295openalex-snapshot
Gene Replacement Therapies for Genodermatoses: A Status Quo
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1789537openalex-snapshot
MesenSistem-EB: systemic haploidentical mesenchymal stem cell therapy in recessive dystrophic epidermolysis bullosa associated with clinical benefits and correlated with MCP1 and sCD40L dynamics
inferred← citeshttps://doi.org/10.3389/fmed.2026.1912442openalex-snapshot
Recombinant humanized type VII collagen for skin repair and regeneration: prospects for reconstructing the dermal-epidermal junction
inferred← citeshttps://doi.org/10.3389/fnmol.2017.00298openalex-snapshot
Design and Characterization of a Human Monoclonal Antibody that Modulates Mutant Connexin 26 Hemichannels Implicated in Deafness and Skin Disorders
inferred← citeshttps://doi.org/10.3390/bioengineering7040155openalex-snapshot
Three Decades of Research on Recombinant Collagens: Reinventing the Wheel or Developing New Biomedical Products?
inferred← citeshttps://doi.org/10.3390/ijms19030762openalex-snapshot
RNA Trans-Splicing Modulation via Antisense Molecule Interference
inferred← citeshttps://doi.org/10.3390/ijms20225707openalex-snapshot
Epidermolysis Bullosa-Associated Squamous Cell Carcinoma: From Pathogenesis to Therapeutic Perspectives
inferred← citeshttps://doi.org/10.4103/idoj.idoj_287_18openalex-snapshot
Newer treatment modalities in epidermolysis bullosa
inferred← citeshttps://doi.org/10.4103/ijpd.ijpd_16_17openalex-snapshot
Inherited epidermolysis bullosa: A multisystem disease of skin and mucosae fragility
inferred← citeshttps://doi.org/10.47196/da.v29i1.2312openalex-snapshot
Nuevos tratamientos para el manejo de la epidermólisis ampollar
inferred← citeshttps://doi.org/10.54097/h6mszb30openalex-snapshot
Developing an Enhanced Computational Framework for Simulating Myocyte Membrane Mechanics: Catch-Bond Dynamics and Algorithmic Optimization
inferred← citeshttps://doi.org/10.58830/ozgur.pub1358.c5474openalex-snapshot
Nadir Görülen Kalıtsal Epitel Doku Hastalıkları
inferred← citeshttps://doi.org/10.70675/154c0828z26c5z465dz9d51zb8270ed8438dopenalex-snapshot
Identification of extracellular matrix biomarkers and targets in Dystrophic epidermolysis bullosa
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