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Common variants near MC4R are associated with fat mass, weight and risk of obesity
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1016/s0092-8674(00)81865-6openalex-snapshot
Targeted Disruption of the Melanocortin-4 Receptor Results in Obesity in Mice
inferredcites →https://doi.org/10.1038/2404openalex-snapshot
A frameshift mutation in MC4R associated with dominantly inherited human obesity
inferredcites →https://doi.org/10.1038/2407openalex-snapshot
A frameshift mutation in human MC4R is associated with a dominant form of obesity
inferredcites →https://doi.org/10.1038/32911openalex-snapshot
A mutation in the human leptin receptor gene causes obesity and pituitary dysfunction
inferredcites →https://doi.org/10.1038/43185openalex-snapshot
Congenital leptin deficiency is associated with severe early-onset obesity in humans
inferredcites →https://doi.org/10.1038/509openalex-snapshot
Severe early-onset obesity, adrenal insufficiency and red hair pigmentation caused by POMC mutations in humans
inferredcites →https://doi.org/10.1038/nature05911openalex-snapshot
Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
inferredcites →https://doi.org/10.1038/ng.2007.16openalex-snapshot
A survey of genetic human cortical gene expression
inferredcites →https://doi.org/10.1038/ng0797-303openalex-snapshot
Obesity and impaired prohormone processing associated with mutations in the human prohormone convertase 1 gene
inferredcites →https://doi.org/10.1038/ng1999openalex-snapshot
Genome-wide association study identifies a second prostate cancer susceptibility variant at 8q24
inferredcites →https://doi.org/10.1038/ng2048openalex-snapshot
Variation in FTO contributes to childhood obesity and severe adult obesity
inferredcites →https://doi.org/10.1038/ng2088openalex-snapshot
A new multipoint method for genome-wide association studies by imputation of genotypes
inferredcites →https://doi.org/10.1038/ng2109openalex-snapshot
A genome-wide association study of global gene expression
inferredcites →https://doi.org/10.1038/ng2119openalex-snapshot
Discovery of expression QTLs using large-scale transcriptional profiling in human lymphocytes
inferredcites →https://doi.org/10.1038/ng2142openalex-snapshot
Population genomics of human gene expression
inferredcites →https://doi.org/10.1038/scientificamerican0108-42aopenalex-snapshot
The Wellcome Trust Case Control Consortium, U.K.
inferredcites →https://doi.org/10.1038/sj.ijo.0803609openalex-snapshot
The V103I polymorphism of the MC4R gene and obesity: population based studies and meta-analysis of 29 563 individuals
inferredcites →https://doi.org/10.1056/nejmoa022050openalex-snapshot
Clinical Spectrum of Obesity and Mutations in the Melanocortin 4 Receptor Gene
inferredcites →https://doi.org/10.1084/jem.20030613openalex-snapshot
BH3-only Protein Noxa Is a Mediator of Hypoxic Cell Death Induced by Hypoxia-inducible Factor 1α
inferredcites →https://doi.org/10.1086/382490openalex-snapshot
Melanocortin-4 Receptor Gene Variant I103 Is Negatively Associated with Obesity
inferredcites →https://doi.org/10.1086/521580openalex-snapshot
Family-Based Association Tests for Genomewide Association Scans
inferredcites →https://doi.org/10.1093/hmg/ddm132openalex-snapshot
Non-synonymous polymorphisms in melanocortin-4 receptor protect against obesity: the two facets of a Janus obesity gene
inferredcites →https://doi.org/10.1126/science.1141634openalex-snapshot
A Common Variant in the FTO Gene Is Associated with Body Mass Index and Predisposes to Childhood and Adult Obesity
inferredcites →https://doi.org/10.1126/science.1142358openalex-snapshot
Genome-Wide Association Analysis Identifies Loci for Type 2 Diabetes and Triglyceride Levels
inferredcites →https://doi.org/10.1126/science.1142364openalex-snapshot
Replication of Genome-Wide Association Signals in UK Samples Reveals Risk Loci for Type 2 Diabetes
inferredcites →https://doi.org/10.1126/science.1142382openalex-snapshot
A Genome-Wide Association Study of Type 2 Diabetes in Finns Detects Multiple Susceptibility Variants
inferredcites →https://doi.org/10.1159/000353712openalex-snapshot
Impact of Informing Overweight Individuals about the Role of Genetics in Obesity: An Online Experimental Study
inferredcites →https://doi.org/10.1159/000353879openalex-snapshot
Genetics of Food Intake Self-Regulation in Childhood: Literature Review and Research Opportunities
inferredcites →https://doi.org/10.1172/jci9397openalex-snapshot
Dominant and recessive inheritance of morbid obesity associated with melanocortin 4 receptor deficiency
inferredcites →https://doi.org/10.1371/journal.pgen.0030115openalex-snapshot
Genome-Wide Association Scan Shows Genetic Variants in the FTO Gene Are Associated with Obesity-Related Traits
inferredcites →https://doi.org/10.17863/cam.89388openalex-snapshot
EPIC-Norfolk: study design and characteristics of the cohort. European Prospective Investigation of Cancer.
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Association Between Common Variants Near the Melanocortin 4 Receptor Gene and Severe Antipsychotic Drug–Induced Weight Gain
inferred← citeshttps://doi.org/10.1001/archpediatrics.2011.147openalex-snapshot
The Intergenerational Transmission of Thinness
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Molecular Genetics of Susceptibility to Coronary Heart Disease
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Metabolic Syndrome, Obesity, and Diabetes
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Control of Appetite/Satiety and Energy Balance
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Aetiology of obesity in children
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Genetic Contributions to the Development of Obesity
inferred← citeshttps://doi.org/10.1002/9781444316728.ch22openalex-snapshot
Weight Regulation and Monogenic Obesity
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The serum leptin level and body mass index in Melanesian and Micronesian Solomon Islanders: Focus on genetic factors and urbanization
inferred← citeshttps://doi.org/10.1002/ajhb.22576openalex-snapshot
Common variants in BDNF, FAIM2, FTO, MC4R, NEGR1, and SH2B1 show association with obesity‐related variables in S panish R oma population
inferred← citeshttps://doi.org/10.1002/ajhb.22923openalex-snapshot
A multianalytical approach to evaluate the association of 55 SNPs in 28 genes with obesity risk in N orth I ndian adults
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Distribution of Polymorphisms Associated With Obesity in a Sample of Admixed Mexican Adults
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Are recently identified genetic variants regulating BMI in the general population associated with anorexia nervosa?
inferred← citeshttps://doi.org/10.1002/ajmg.b.31182openalex-snapshot
Absence of association between specific common variants of the obesity‐related FTO gene and psychological and behavioral eating disorder phenotypes
inferred← citeshttps://doi.org/10.1002/dmrr.1221openalex-snapshot
Genome‐wide association studies (GWAS): impact on elucidating the aetiology of diabetes
inferred← citeshttps://doi.org/10.1002/fsn3.70790openalex-snapshot
Association Between Healthful Plant‐Based Dietary Pattern and Adiposity Measures Trajectories and Future Metabolic Diseases Risk: A Prospective Cohort Study
inferred← citeshttps://doi.org/10.1002/gepi.20377openalex-snapshot
Proper analysis of secondary phenotype data in case‐control association studies
inferred← citeshttps://doi.org/10.1002/gepi.20424openalex-snapshot
Genome‐wide association scans for secondary traits using case‐control samples
inferred← citeshttps://doi.org/10.1002/gepi.20552openalex-snapshot
A comparison of approaches to account for uncertainty in analysis of imputed genotypes
inferred← citeshttps://doi.org/10.1002/gepi.22067openalex-snapshot
Improving power of association tests using multiple sets of imputed genotypes from distributed reference panels
inferred← citeshttps://doi.org/10.1002/iub.1596openalex-snapshot
Obesity‐related genetic polymorphisms and adiposity indices in a young Italian population
inferred← citeshttps://doi.org/10.1002/mef2.70057openalex-snapshot
Cilia in Nervous System Development, Function, and Disease
inferred← citeshttps://doi.org/10.1002/mnfr.201000445openalex-snapshot
Evidences on three relevant obesogenes: MC4R , FTO and PPAR γ. Approaches for personalized nutrition
inferred← citeshttps://doi.org/10.1002/mnfr.201400521openalex-snapshot
Can metabolically healthy obesity be explained by diet, genetics, and inflammation?
inferred← citeshttps://doi.org/10.1002/oby.20147openalex-snapshot
The missense variation landscape of FTO, MC4R, and TMEM18 in obese children of African Ancestry
inferred← citeshttps://doi.org/10.1002/oby.20196openalex-snapshot
Interactions of genetic and environmental risk factors with respect to body fat mass in children: Results from the ALSPAC study
inferred← citeshttps://doi.org/10.1002/oby.20268openalex-snapshot
Genetic risk factors for BMI and obesity in an ethnically diverse population: Results from the population architecture using genomics and epidemiology (PAGE) study
inferred← citeshttps://doi.org/10.1002/oby.20459openalex-snapshot
Variation at the melanocortin 4 receptor gene and response to weight‐loss interventions in the diabetes prevention program
inferred← citeshttps://doi.org/10.1002/oby.20540openalex-snapshot
The effect of GWAS identified BMI loci on changes in body weight among middle‐aged danes during a five‐year period
inferred← citeshttps://doi.org/10.1002/oby.20606openalex-snapshot
FTO genetic variants and risk of obesity and type 2 diabetes: A meta‐analysis of 28,394 Indians
inferred← citeshttps://doi.org/10.1002/oby.21402openalex-snapshot
Physical activity attenuates genetic effects on BMI : Results from a study of C hinese adult twins
inferred← citeshttps://doi.org/10.1002/oby.21554openalex-snapshot
Body mass index: Has epidemiology started to break down causal contributions to health and disease?
inferred← citeshttps://doi.org/10.1002/oby.22135openalex-snapshot
A Mathematical Model for Predicting Obesity Transmission with Both Genetic and Nongenetic Heredity
inferred← citeshttps://doi.org/10.1002/oby.23116openalex-snapshot
Genetics of Obesity: What We Have Learned Over Decades of Research
inferred← citeshttps://doi.org/10.1002/sim.4499openalex-snapshot
On the choice of parameterisation and priors for the Bayesian analyses of Mendelian randomisation studies
inferred← citeshttps://doi.org/10.1002/sim.6976openalex-snapshot
Robust analysis of secondary phenotypes in case‐control genetic association studies
inferred← citeshttps://doi.org/10.1007/978-0-387-09488-5_29openalex-snapshot
Candidate Gene and Genome-Wide Association Studies in Behavioral Medicine
inferred← citeshttps://doi.org/10.1007/978-0-387-09488-5_33openalex-snapshot
Genetics of Obesity and Diabetes
inferred← citeshttps://doi.org/10.1007/978-0-387-85960-6_31openalex-snapshot
Genetics in Cardiovascular Behavioral Medicine
inferred← citeshttps://doi.org/10.1007/978-0-387-85960-6_34openalex-snapshot
Insulin, Glucose, and the Metabolic Syndrome in Cardiovascular Behavioral Medicine
inferred← citeshttps://doi.org/10.1007/978-0-387-92271-3_75openalex-snapshot
Genetic Markers, Weight Reduction, and Behavioral Changes in Lifestyle
inferred← citeshttps://doi.org/10.1007/978-0-387-93826-4_28openalex-snapshot
Behavioral Management of Obesity: Enduring Models, Applications to Diabetes Prevention and Management, and Global Dissemination
inferred← citeshttps://doi.org/10.1007/978-1-4419-1607-5_1openalex-snapshot
Principles of Human Energy Metabolism
inferred← citeshttps://doi.org/10.1007/978-1-4419-5515-9_4openalex-snapshot
Genetic Epidemiology of Obesity and Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4419-5800-6_17openalex-snapshot
Genomic Risk Information for Common Health Conditions: Maximizing Kinship-Based Health Promotion
inferred← citeshttps://doi.org/10.1007/978-1-4419-6039-9_15openalex-snapshot
Genetics and Nutrigenomics of Obesity
inferred← citeshttps://doi.org/10.1007/978-1-4419-7034-3_3openalex-snapshot
Monogenic Disorders Within the Energy Balance Pathway
inferred← citeshttps://doi.org/10.1007/978-1-4419-7034-3_5openalex-snapshot
Genome-Wide Association Studies and Human Population Obesity
inferred← citeshttps://doi.org/10.1007/978-1-4614-0965-6_10openalex-snapshot
The Genetic Determinants of Common Obesity-Susceptibility
inferred← citeshttps://doi.org/10.1007/978-1-4614-3492-4_6openalex-snapshot
The Neurogenetics of Energy Balance
inferred← citeshttps://doi.org/10.1007/978-1-4614-8642-8_3openalex-snapshot
Genome-Wide Association Studies of Obesity
inferred← citeshttps://doi.org/10.1007/978-1-4614-8642-8_5openalex-snapshot
Genetics of Childhood Obesity
inferred← citeshttps://doi.org/10.1007/978-1-4614-8642-8_6openalex-snapshot
Genetic Pleiotropies of Obesity
inferred← citeshttps://doi.org/10.1007/978-1-4939-0823-3_3openalex-snapshot
The Role of the Ghrelin Receptor in Appetite and Energy Metabolism
inferred← citeshttps://doi.org/10.1007/978-1-4939-1197-4_20openalex-snapshot
Genomic and Clinical Predictors Associated with Long-Term Success After Bariatric Surgery
inferred← citeshttps://doi.org/10.1007/978-1-4939-1206-3_2openalex-snapshot
The Pathophysiology of Obesity and Obesity-Related Diseases
inferred← citeshttps://doi.org/10.1007/978-1-4939-7471-9_18openalex-snapshot
Physiologic Interpretation of GWAS Signals for Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-60327-256-8_2openalex-snapshot
Fetal Origins of Obesity and Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-60327-416-6_17openalex-snapshot
The Genetic Epidemiology of Obesity: A Case Study
inferred← citeshttps://doi.org/10.1007/978-1-60761-355-8_1openalex-snapshot
Fundamental Concepts in Exercise Genomics
inferred← citeshttps://doi.org/10.1007/978-1-60761-355-8_3openalex-snapshot
Can You Be Born a Couch Potato? The Genomic Regulation of Physical Activity
inferred← citeshttps://doi.org/10.1007/978-1-60761-355-8_9openalex-snapshot
A Synopsis of Exercise Genomics Research and a Vision for its Future Translation into Practice
inferred← citeshttps://doi.org/10.1007/978-1-60761-934-5_8openalex-snapshot
Animal Models of Eating Disorders
inferred← citeshttps://doi.org/10.1007/978-1-61737-954-3_2openalex-snapshot
Statistical Issues in Gene Association Studies
inferred← citeshttps://doi.org/10.1007/978-1-61779-530-5_5openalex-snapshot
Genome-Wide Association Studies in Disease Risk Calculation: The Role of Bioinformatics in Patient Care
inferred← citeshttps://doi.org/10.1007/978-1-62703-779-2_17openalex-snapshot
Exon Sequencing of G Protein-Coupled Receptor Genes and Perspectives for Disease Treatment
inferred← citeshttps://doi.org/10.1007/978-1-84882-120-0openalex-snapshot
Gestational Diabetes During and After Pregnancy
inferred← citeshttps://doi.org/10.1007/978-1-84882-120-0_13openalex-snapshot
Genetics of Gestational Diabetes Mellitus and Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-2-8178-0332-6_24openalex-snapshot
Génétique des obésités humaines
inferred← citeshttps://doi.org/10.1007/978-2-8178-0343-2_24openalex-snapshot
Genetics of the Human Obesities
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The Pathophysiology of Obesity and Obesity-Related Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-62187-2_12openalex-snapshot
Hypothalamic Obesity and Wasting Syndromes
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Detecting Changepoint in Gene Expressions over Time: An Application to Childhood Obesity
inferred← citeshttps://doi.org/10.1007/978-3-030-72999-8_3openalex-snapshot
Pathophysiology of Obesity and Cancer: Drugs and Signaling Targets
inferred← citeshttps://doi.org/10.1007/978-3-031-40116-9_11openalex-snapshot
Genetics of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-031-40116-9_48openalex-snapshot
Principles of Energy Homeostasis
inferred← citeshttps://doi.org/10.1007/978-3-031-62491-9_17openalex-snapshot
Genetic Variants Connected to the Obesity
inferred← citeshttps://doi.org/10.1007/978-3-032-24278-5_6openalex-snapshot
The Pathophysiology of Obesity and Obesity-Related Diseases
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_2openalex-snapshot
Genome-Wide Association Studies of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_21openalex-snapshot
The Value of Genetic Variation in the Prediction of Obesity
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_4openalex-snapshot
Genome-Wide Association Studies (GWAS) of Adiposity
inferred← citeshttps://doi.org/10.1007/978-3-319-11251-0_11openalex-snapshot
Genetics of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-319-11251-0_48openalex-snapshot
Principles of Energy Homeostasis
inferred← citeshttps://doi.org/10.1007/978-3-319-12125-3_11-1openalex-snapshot
Genetics of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-319-12125-3_11-2openalex-snapshot
Genetics of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-319-12125-3_48-1openalex-snapshot
Principles of Energy Homeostasis
inferred← citeshttps://doi.org/10.1007/978-3-319-12125-3_48-2openalex-snapshot
Principles of Energy Homeostasis
inferred← citeshttps://doi.org/10.1007/978-3-319-12766-8_4openalex-snapshot
Genetic Contribution: Common Forms of Obesity
inferred← citeshttps://doi.org/10.1007/978-3-319-12766-8_5openalex-snapshot
The Role of the GWAS Identified FTO Locus in Regulating Body Size and Composition
inferred← citeshttps://doi.org/10.1007/978-3-319-19821-7_7openalex-snapshot
Genetics of Human Obesity
inferred← citeshttps://doi.org/10.1007/978-3-319-27449-2_8openalex-snapshot
The “Toxic” Effects of a Perinatal Obesogenic Environment: Maternal Obesity and Impacts on Future Generations
inferred← citeshttps://doi.org/10.1007/978-3-319-47143-3_3openalex-snapshot
Preconception and Prenatal Factors and Metabolic Risk
inferred← citeshttps://doi.org/10.1007/978-3-319-52031-5_11openalex-snapshot
The Genetic Determinants of Common Obesity-Susceptibility
inferred← citeshttps://doi.org/10.1007/978-3-540-37654-5_10openalex-snapshot
Lessons from the Genome-Wide Association Studies for Complex Multifactorial Disorders and Traits
inferred← citeshttps://doi.org/10.1007/978-3-642-24716-3_3openalex-snapshot
The Neuroendocrine Circuitry Controlled by POMC, MSH, and AGRP
inferred← citeshttps://doi.org/10.1007/978-3-642-38364-9_6openalex-snapshot
Suchtaspekte bei Adipositas
inferred← citeshttps://doi.org/10.1007/978-3-662-46306-2_12openalex-snapshot
Mapping of Susceptibility Genes for Obesity, Type 2 Diabetes, and the Metabolic Syndrome in Human Populations
inferred← citeshttps://doi.org/10.1007/978-90-481-3261-4_7openalex-snapshot
Molecular Diagnosis in the Post Genomic and Proteomic Era
inferred← citeshttps://doi.org/10.1007/978-94-007-7772-9_7openalex-snapshot
Gamete/Embryo-Fetal Origins of Obesity
inferred← citeshttps://doi.org/10.1007/978-981-13-6260-6_8openalex-snapshot
Aldehyde Dehydrogenases Genetic Polymorphism and Obesity: From Genomics to Behavior and Health
inferred← citeshttps://doi.org/10.1007/978-981-19-5082-7_5openalex-snapshot
Precision Medicine in Endocrinology Practice
inferred← citeshttps://doi.org/10.1007/bf03195727openalex-snapshot
High-fat diet leads to a decreased methylation of theMc4r gene in the obese BFMI and the lean B6 mouse lines
inferred← citeshttps://doi.org/10.1007/s00103-010-1078-zopenalex-snapshot
Genetische Ursachen der Adipositas
inferred← citeshttps://doi.org/10.1007/s00103-012-1490-7openalex-snapshot
Die Heinz Nixdorf Recall Studie
inferred← citeshttps://doi.org/10.1007/s00109-009-0451-6openalex-snapshot
Combined effects of MC4R and FTO common genetic variants on obesity in European general populations
inferred← citeshttps://doi.org/10.1007/s00125-008-1151-4openalex-snapshot
The search for putative unifying genetic factors for components of the metabolic syndrome
inferred← citeshttps://doi.org/10.1007/s00125-009-1419-3openalex-snapshot
Is the thrifty genotype hypothesis supported by evidence based on confirmed type 2 diabetes- and obesity-susceptibility variants?
inferred← citeshttps://doi.org/10.1007/s00125-011-2044-5openalex-snapshot
Genetic predisposition to obesity leads to increased risk of type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s00125-011-2086-8openalex-snapshot
Association of genetic variants for susceptibility to obesity with type 2 diabetes in Japanese individuals
inferred← citeshttps://doi.org/10.1007/s00125-011-2320-4openalex-snapshot
Association studies of novel obesity-related gene variants with quantitative metabolic phenotypes in a population-based sample of 6,039 Danish individuals
inferred← citeshttps://doi.org/10.1007/s00125-011-2370-7openalex-snapshot
Association of genetic variation in FTO with risk of obesity and type 2 diabetes with data from 96,551 East and South Asians
inferred← citeshttps://doi.org/10.1007/s00125-012-2638-6openalex-snapshot
Impact of FTO genotypes on BMI and weight in polycystic ovary syndrome: a systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1007/s00125-012-2655-5openalex-snapshot
Common polymorphism near the MC4R gene is associated with type 2 diabetes: data from a meta-analysis of 123,373 individuals
inferred← citeshttps://doi.org/10.1007/s00125-013-2985-yopenalex-snapshot
Pleiotropic effects of obesity-susceptibility loci on metabolic traits: a meta-analysis of up to 37,874 individuals
inferred← citeshttps://doi.org/10.1007/s00125-013-2999-5openalex-snapshot
The biology of FTO: from nucleic acid demethylase to amino acid sensor
inferred← citeshttps://doi.org/10.1007/s00125-014-3270-4openalex-snapshot
Genetic susceptibility to type 2 diabetes and obesity: from genome-wide association studies to rare variants and beyond
inferred← citeshttps://doi.org/10.1007/s00213-023-06331-9openalex-snapshot
Genetic association of the rs17782313 polymorphism with antipsychotic-induced weight gain
inferred← citeshttps://doi.org/10.1007/s00335-009-9211-8openalex-snapshot
Identification of genetic loci involved in diabetes using a rat model of depression
inferred← citeshttps://doi.org/10.1007/s00335-010-9247-9openalex-snapshot
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice
inferred← citeshttps://doi.org/10.1007/s00335-014-9541-zopenalex-snapshot
Genetic strategies to understand physiological pathways regulating body weight
inferred← citeshttps://doi.org/10.1007/s00394-019-01955-0openalex-snapshot
The MC4R genetic variants are associated with lower visceral fat accumulation and higher postprandial relative increase in carbohydrate utilization in humans
inferred← citeshttps://doi.org/10.1007/s00394-025-03713-xopenalex-snapshot
Impact of recognition of genetic information related to BMI on changes in physical activity, dietary intake, and blood cholesterol level: a randomized controlled trial
inferred← citeshttps://doi.org/10.1007/s00403-012-1296-3openalex-snapshot
Selected variants of the melanocortin 4 receptor gene (MC4R) do not confer susceptibility to female pattern hair loss
inferred← citeshttps://doi.org/10.1007/s00438-015-1015-9openalex-snapshot
Current review of genetics of human obesity: from molecular mechanisms to an evolutionary perspective
inferred← citeshttps://doi.org/10.1007/s00438-021-01793-yopenalex-snapshot
Effect of interaction between obesity-promoting genetic variants and behavioral factors on the risk of obese phenotypes
inferred← citeshttps://doi.org/10.1007/s00439-010-0917-1openalex-snapshot
Genetic risk sum score comprised of common polygenic variation is associated with body mass index
inferred← citeshttps://doi.org/10.1007/s00439-011-0960-6openalex-snapshot
A dynamic model for genome-wide association studies
inferred← citeshttps://doi.org/10.1007/s00439-011-0964-2openalex-snapshot
Revisiting Mendelian disorders through exome sequencing
inferred← citeshttps://doi.org/10.1007/s00439-011-0969-xopenalex-snapshot
Recent progress in the study of the genetics of height
inferred← citeshttps://doi.org/10.1007/s00439-011-1023-8openalex-snapshot
Type 2 diabetes and obesity: genomics and the clinic
inferred← citeshttps://doi.org/10.1007/s00439-012-1246-3openalex-snapshot
Gene-environment interactions and obesity traits among postmenopausal African-American and Hispanic women in the Women’s Health Initiative SHARe Study
inferred← citeshttps://doi.org/10.1007/s00439-013-1293-4openalex-snapshot
Obesity-related genetic variants, human pigmentation, and risk of melanoma
inferred← citeshttps://doi.org/10.1007/s00439-014-1477-6openalex-snapshot
Common genetic variation in and near the melanocortin 4 receptor gene (MC4R) is associated with body mass index in American Indian adults and children
inferred← citeshttps://doi.org/10.1007/s00439-015-1533-xopenalex-snapshot
Obesity and genomics: role of technology in unraveling the complex genetic architecture of obesity
inferred← citeshttps://doi.org/10.1007/s00787-010-0096-6openalex-snapshot
From monogenic to polygenic obesity: recent advances
inferred← citeshttps://doi.org/10.1007/s10059-010-0015-1openalex-snapshot
Podophyllotoxin Induces CREB Phosphorylation and CRE-Driven Gene Expression via PKA but Not MAPKs
inferred← citeshttps://doi.org/10.1007/s10528-015-9700-2openalex-snapshot
Obesity, More than a ‘Cosmetic’ Problem. Current Knowledge and Future Prospects of Human Obesity Genetics
inferred← citeshttps://doi.org/10.1007/s10654-011-9583-4openalex-snapshot
Influence of common variants in FTO and near INSIG2 and MC4R on growth curves for adiposity in African– and European–American youth
inferred← citeshttps://doi.org/10.1007/s10654-017-0290-7openalex-snapshot
Epidemiology in Germany—general development and personal experience
inferred← citeshttps://doi.org/10.1007/s10742-011-0071-9openalex-snapshot
Genes as instruments for studying risk behavior effects: an application to maternal smoking and orofacial clefts
inferred← citeshttps://doi.org/10.1007/s10815-014-0411-0openalex-snapshot
Clinically relevant known and candidate genes for obesity and their overlap with human infertility and reproduction
inferred← citeshttps://doi.org/10.1007/s11033-011-1053-2openalex-snapshot
Influence of genomic variation in FTO at 16q12.2, MC4R at 18q22 and NRXN3 at 14q31 genes on breast cancer risk
inferred← citeshttps://doi.org/10.1007/s11033-012-1476-4openalex-snapshot
A Common variant near the melanocortin 4 receptor is associated with low-density lipoprotein cholesterol and total cholesterol in the Chinese Han population
inferred← citeshttps://doi.org/10.1007/s11033-012-2089-7openalex-snapshot
The joint effect of cigarette smoking and polymorphisms on LRP5, LEPR, near MC4R and SH2B1 genes on metabolic syndrome susceptibility in Taiwan
inferred← citeshttps://doi.org/10.1007/s11033-013-2780-3openalex-snapshot
Interaction between obesity-related genes, FTO and MC4R, associated to an increase of breast cancer risk
inferred← citeshttps://doi.org/10.1007/s11033-013-2994-4openalex-snapshot
The role of common and rare MC4R variants and FTO polymorphisms in extreme form of obesity
inferred← citeshttps://doi.org/10.1007/s11033-014-3627-2openalex-snapshot
Association of obesity with rs1421085 and rs9939609 polymorphisms of FTO gene
inferred← citeshttps://doi.org/10.1007/s11033-022-07254-yopenalex-snapshot
Association of gene polymorphisms with body weight changes in prediabetic patients
inferred← citeshttps://doi.org/10.1007/s11055-017-0426-yopenalex-snapshot
The Melanocortin Signal System of the Hypothalamus and Its Functional State in Type 2 Diabetes Mellitus and Metabolic Syndrome
inferred← citeshttps://doi.org/10.1007/s11154-019-09505-zopenalex-snapshot
Genetics, adaptation to environmental changes and archaic admixture in the pathogenesis of diabetes mellitus in Indigenous Australians
inferred← citeshttps://doi.org/10.1007/s11154-023-09804-6openalex-snapshot
Genetics and epigenetics in the obesity phenotyping scenario
inferred← citeshttps://doi.org/10.1007/s11427-018-9489-xopenalex-snapshot
Testing the role of genetic variation of the MC4R gene in Chinese population in antipsychotic-induced metabolic disturbance
inferred← citeshttps://doi.org/10.1007/s11684-013-0263-5openalex-snapshot
Leptin signaling and leptin resistance
inferred← citeshttps://doi.org/10.1007/s11684-014-0373-8openalex-snapshot
Advancement in genetic variants conferring obesity susceptibility from genome-wide association studies
inferred← citeshttps://doi.org/10.1007/s11690-013-0357-xopenalex-snapshot
Genetics of the human obesities
inferred← citeshttps://doi.org/10.1007/s11695-013-1080-2openalex-snapshot
Genetic Risk Score Does Not Predict the Outcome of Obesity Surgery
inferred← citeshttps://doi.org/10.1007/s11695-019-04200-zopenalex-snapshot
Updates on Monogenic Obesity in a Multifactorial Disease
inferred← citeshttps://doi.org/10.1007/s11825-011-0295-7openalex-snapshot
Wie groß sind die kleinen genetischen Risiken?
inferred← citeshttps://doi.org/10.1007/s11825-017-0156-0openalex-snapshot
Polygene Varianten und Epigenetik bei Adipositas
inferred← citeshttps://doi.org/10.1007/s11886-016-0755-4openalex-snapshot
Genetics of Insulin Resistance and the Metabolic Syndrome
inferred← citeshttps://doi.org/10.1007/s11886-017-0938-7openalex-snapshot
The Genetics of Physical Activity
inferred← citeshttps://doi.org/10.1007/s11886-020-01422-xopenalex-snapshot
Obesity Genomics and Metabolomics: a Nexus of Cardiometabolic Risk
inferred← citeshttps://doi.org/10.1007/s11892-008-0033-yopenalex-snapshot
Genes and type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1007/s11892-009-0027-4openalex-snapshot
Genome-wide association studies in type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s11892-010-0143-1openalex-snapshot
The Genetics of Insulin Resistance: Where’s Waldo?
inferred← citeshttps://doi.org/10.1007/s11892-012-0255-xopenalex-snapshot
Who Should We Target for Diabetes Prevention and Diabetes Risk Reduction?
inferred← citeshttps://doi.org/10.1007/s11892-015-0648-8openalex-snapshot
Insights into the Genetic Susceptibility to Type 2 Diabetes from Genome-Wide Association Studies of Obesity-Related Traits
inferred← citeshttps://doi.org/10.1007/s11894-010-0116-1openalex-snapshot
Childhood Obesity: Update on Predisposing Factors and Prevention Strategies
inferred← citeshttps://doi.org/10.1007/s11906-011-0242-7openalex-snapshot
Mendelian Randomization: Application to Cardiovascular Disease
inferred← citeshttps://doi.org/10.1007/s12020-010-9413-6openalex-snapshot
Combined effects of FTO rs9939609 and MC4R rs17782313 on obesity and BMI in Chinese Han populations
inferred← citeshttps://doi.org/10.1007/s12020-012-9869-7openalex-snapshot
Genetic association study of WNT10B polymorphisms with BMD and adiposity parameters in Danish and Belgian males
inferred← citeshttps://doi.org/10.1007/s12020-013-9909-yopenalex-snapshot
Replication of genetic effects of MC4R polymorphisms on body mass index in a Korean population
inferred← citeshttps://doi.org/10.1007/s12020-014-0217-yopenalex-snapshot
An obesity genetic risk score predicts risk of insulin resistance among Chinese children
inferred← citeshttps://doi.org/10.1007/s12020-014-0306-yopenalex-snapshot
A polymorphism near MC4R gene (rs17782313) is associated with serum triglyceride levels in the general Japanese population: the J-MICC Study
inferred← citeshttps://doi.org/10.1007/s12020-015-0616-8openalex-snapshot
Associations between a common variant near the MC4R gene and serum triglyceride levels in an obese pediatric cohort
inferred← citeshttps://doi.org/10.1007/s12020-018-1587-3openalex-snapshot
Association between LEPR, FTO, MC4R, and PPARG-2 polymorphisms with obesity traits and metabolic phenotypes in school-aged children
inferred← citeshttps://doi.org/10.1007/s12020-023-03642-xopenalex-snapshot
Mechanisms and risk factors of metabolic syndrome in children and adolescents
inferred← citeshttps://doi.org/10.1007/s12020-026-04696-3openalex-snapshot
Genetic determinants of obesity: mechanisms, clinical implications, and targeted therapies
inferred← citeshttps://doi.org/10.1007/s12035-017-0414-9openalex-snapshot
Melanocortin Receptor-4 Gene Polymorphisms in Glioblastoma Patients Treated with Concomitant Radio-Chemotherapy
inferred← citeshttps://doi.org/10.1007/s12041-010-0066-2openalex-snapshot
Common variants in the gene for the serotonin receptor 6 (HTR6) do not contribute to obesity
inferred← citeshttps://doi.org/10.1007/s12041-018-0987-8openalex-snapshot
Genetics of obesity and its measures in India
inferred← citeshttps://doi.org/10.1007/s12160-009-9116-5openalex-snapshot
Appetite is a Heritable Phenotype Associated with Adiposity
inferred← citeshttps://doi.org/10.1007/s12170-009-0003-1openalex-snapshot
Genomewide association studies and lipid risk factors
inferred← citeshttps://doi.org/10.1007/s12170-009-0061-4openalex-snapshot
The genetic determinants of common human obesity
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