On the public citation graph
Variants in MTNR1B influence fasting glucose levels
0 signed · 743 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/bf00280883openalex-snapshot
Homeostasis model assessment: insulin resistance and ?-cell function from fasting plasma glucose and insulin concentrations in man
inferredcites →https://doi.org/10.1007/s10654-007-9199-xopenalex-snapshot
The Rotterdam Study: objectives and design update
inferredcites →https://doi.org/10.1016/s0140-6736(99)01376-8openalex-snapshot
Impact of sleep debt on metabolic and endocrine function
inferredcites →https://doi.org/10.1038/nature04226openalex-snapshot
A haplotype map of the human genome
inferredcites →https://doi.org/10.1038/nature05616openalex-snapshot
A genome-wide association study identifies novel risk loci for type 2 diabetes
inferredcites →https://doi.org/10.1038/ng.120openalex-snapshot
Meta-analysis of genome-wide association data and large-scale replication identifies additional susceptibility loci for type 2 diabetes
inferredcites →https://doi.org/10.1038/ng.2007.16openalex-snapshot
A survey of genetic human cortical gene expression
inferredcites →https://doi.org/10.1038/ng2043openalex-snapshot
A variant in CDKAL1 influences insulin response and risk of type 2 diabetes
inferredcites →https://doi.org/10.1038/ng2109openalex-snapshot
A genome-wide association study of global gene expression
inferredcites →https://doi.org/10.1038/ng2142openalex-snapshot
Population genomics of human gene expression
inferredcites →https://doi.org/10.1055/s-2008-1078730openalex-snapshot
Variants of thePPARG,IGF2BP2,CDKAL1,HHEX, andTCF7L2Genes Confer Risk of Type 2 Diabetes Independently of BMI in the German KORA Studies
inferredcites →https://doi.org/10.1073/pnas.012025199openalex-snapshot
Large-scale analysis of the human and mouse transcriptomes
inferredcites →https://doi.org/10.1073/pnas.92.19.8734openalex-snapshot
Molecular characterization of a second melatonin receptor expressed in human retina and brain: the Mel1b melatonin receptor.
inferredcites →https://doi.org/10.1086/509517openalex-snapshot
A Common Haplotype of the Glucokinase Gene Alters Fasting Glucose and Birth Weight: Association in Six Studies and Population-Genetics Analyses
inferredcites →https://doi.org/10.1111/j.1600-079x.2007.00426.xopenalex-snapshot
Melatonin and type 2 diabetes – a possible link?*
inferredcites →https://doi.org/10.1111/j.1600-079x.2007.00523.xopenalex-snapshot
Function and expression of melatonin receptors on human pancreatic islets
inferredcites →https://doi.org/10.1111/j.1600-079x.2008.00593.xopenalex-snapshot
Involvement of the cGMP pathway in mediating the insulin‐inhibitory effect of melatonin in pancreatic β‐cells
inferredcites →https://doi.org/10.1126/science.1108750openalex-snapshot
Obesity and Metabolic Syndrome in Circadian Clock Mutant Mice
inferredcites →https://doi.org/10.1126/science.1142358openalex-snapshot
Genome-Wide Association Analysis Identifies Loci for Type 2 Diabetes and Triglyceride Levels
inferredcites →https://doi.org/10.1126/science.1142364openalex-snapshot
Replication of Genome-Wide Association Signals in UK Samples Reveals Risk Loci for Type 2 Diabetes
inferredcites →https://doi.org/10.1126/science.1142382openalex-snapshot
A Genome-Wide Association Study of Type 2 Diabetes in Finns Detects Multiple Susceptibility Variants
inferredcites →https://doi.org/10.1126/science.1156849openalex-snapshot
A Polymorphism Within the G6PC2 Gene Is Associated with Fasting Plasma Glucose Levels
inferredcites →https://doi.org/10.1152/ajpendo.1996.271.2.e246openalex-snapshot
Evidence for a circadian rhythm of insulin secretion
inferredcites →https://doi.org/10.1159/000022865openalex-snapshot
Familiality of Quantitative Metabolic Traits in Finnish Families with Non-Insulin-Dependent Diabetes mellitus
inferredcites →https://doi.org/10.1159/000468530openalex-snapshot
Magnesium Replacement Improves the Metabolic Profile in Obese and Pre-Diabetic Patients with Mild-to-Moderate Chronic Kidney Disease: A 3-Month, Randomised, Double-Blind, Placebo-Controlled Study
inferredcites →https://doi.org/10.1172/jci34566openalex-snapshot
Variations in the G6PC2/ABCB11 genomic region are associated with fasting glucose levels
inferredcites →https://doi.org/10.1371/journal.pbio.0060107openalex-snapshot
Mapping the Genetic Architecture of Gene Expression in Human Liver
inferredcites →https://doi.org/10.2337/db07-0053openalex-snapshot
Progression to Type 2 Diabetes Characterized by Moderate Then Rapid Glucose Increases
inferredcites →https://doi.org/10.2337/db07-0615openalex-snapshot
Impact of Common Type 2 Diabetes Risk Polymorphisms in the DESIR Prospective Study
inferredcites →https://doi.org/10.2337/db08-0516openalex-snapshot
Common Missense Variant in the Glucokinase Regulatory Protein Gene Is Associated With Increased Plasma Triglyceride and C-Reactive Protein but Lower Fasting Glucose Concentrations
inferredcites →https://doi.org/10.2337/diabetes.55.04.06.db05-1109openalex-snapshot
Coordinate Changes in Plasma Glucose and Pancreatic β-Cell Function in Latino Women at High Risk for Type 2 Diabetes
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1001/jama.2013.2710openalex-snapshot
Melatonin Secretion and the Incidence of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1002/9780470015902.a0021994openalex-snapshot
The Molecular Genetics of Type 2 Diabetes: Past, Present and Future
inferred← citeshttps://doi.org/10.1002/9781118387658.ch26openalex-snapshot
The genetics of type 2 diabetes
inferred← citeshttps://doi.org/10.1002/9781118924853.ch14openalex-snapshot
Genetic Architecture of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1002/9781119564782.ch15openalex-snapshot
G Protein‐Coupled Receptors in Metabolic Disease
inferred← citeshttps://doi.org/10.1002/9781444324808.ch12openalex-snapshot
The Genetics of Type 2 Diabetes: From Candidate Gene Biology to Genome‐Wide Studies
inferred← citeshttps://doi.org/10.1002/9781444355628.ch10openalex-snapshot
Pharmacogenomics and Organ Transplantation
inferred← citeshttps://doi.org/10.1002/dmrr.1221openalex-snapshot
Genome‐wide association studies (GWAS): impact on elucidating the aetiology of diabetes
inferred← citeshttps://doi.org/10.1002/gepi.20450openalex-snapshot
An evaluation of statistical approaches to rare variant analysis in genetic association studies
inferred← citeshttps://doi.org/10.1002/gepi.20525openalex-snapshot
Impact of repeated measures and sample selection on genome‐wide association studies of fasting glucose
inferred← citeshttps://doi.org/10.1002/gepi.20540openalex-snapshot
Meta‐analysis of sex‐specific genome‐wide association studies
inferred← citeshttps://doi.org/10.1002/gepi.20546openalex-snapshot
Meta-analysis of gene-environment interaction: joint estimation of SNP and SNP × environment regression coefficients
inferred← citeshttps://doi.org/10.1002/gepi.21632openalex-snapshot
Fasting Glucose GWAS Candidate Region Analysis Across Ethnic Groups in the Multiethnic Study of Atherosclerosis ( MESA )
inferred← citeshttps://doi.org/10.1002/gepi.21978openalex-snapshot
An Empirical Comparison of Joint and Stratified Frameworks for Studying G × E Interactions: Systolic Blood Pressure and Smoking in the CHARGE Gene‐Lifestyle Interactions Working Group
inferred← citeshttps://doi.org/10.1002/gepi.22123openalex-snapshot
Improved score statistics for meta‐analysis in single‐variant and gene‐level association studies
inferred← citeshttps://doi.org/10.1002/hbm.22494openalex-snapshot
Genetic modifiers of cognitive maintenance among older adults
inferred← citeshttps://doi.org/10.1002/humu.21110openalex-snapshot
Update on mutations in glucokinase ( GCK ), which cause maturity-onset diabetes of the young, permanent neonatal diabetes, and hyperinsulinemic hypoglycemia
inferred← citeshttps://doi.org/10.1002/oby.21030openalex-snapshot
A common variant in the MTNR1b gene is associated with increased risk of impaired fasting glucose (IFG) in youth with obesity
inferred← citeshttps://doi.org/10.1002/stem.2968openalex-snapshot
Melatonin Enhances Proliferation and Modulates Differentiation of Neural Stem Cells Via Autophagy in Hyperglycemia
inferred← citeshttps://doi.org/10.1007/5584_2020_518openalex-snapshot
Diabetes and Genetics: A Relationship Between Genetic Risk Alleles, Clinical Phenotypes and Therapeutic Approaches
inferred← citeshttps://doi.org/10.1007/978-0-387-09488-5_33openalex-snapshot
Genetics of Obesity and Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-4419-6956-9_12openalex-snapshot
DNA Variations, Impaired Insulin Secretion and Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-4419-6956-9_13openalex-snapshot
Genetically Programmed Defects in β-Cell Function
inferred← citeshttps://doi.org/10.1007/978-1-4614-0965-6_10openalex-snapshot
The Genetic Determinants of Common Obesity-Susceptibility
inferred← citeshttps://doi.org/10.1007/978-1-4939-7471-9_18openalex-snapshot
Physiologic Interpretation of GWAS Signals for Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-61737-954-3_2openalex-snapshot
Statistical Issues in Gene Association Studies
inferred← citeshttps://doi.org/10.1007/978-1-62703-779-2_17openalex-snapshot
Exon Sequencing of G Protein-Coupled Receptor Genes and Perspectives for Disease Treatment
inferred← citeshttps://doi.org/10.1007/978-1-84882-120-0openalex-snapshot
Gestational Diabetes During and After Pregnancy
inferred← citeshttps://doi.org/10.1007/978-1-84882-120-0_13openalex-snapshot
Genetics of Gestational Diabetes Mellitus and Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-030-61721-9_22openalex-snapshot
Cytoprotection by Melatonin: The Metabolic Syndrome as an Example
inferred← citeshttps://doi.org/10.1007/978-3-031-22468-3_14openalex-snapshot
Melatonin as a Chronobiotic and Cytoprotector in Healthy Aging
inferred← citeshttps://doi.org/10.1007/978-3-031-66768-8_11openalex-snapshot
Melatonin as a Chronobiotic and Cytoprotector in Non-communicable Diseases: More than an Antioxidant
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_12openalex-snapshot
Gene-Environment Interaction: Methods and Examples in Type 2 Diabetes and Obesity
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_16openalex-snapshot
Genetic and Functional Studies Implicate G6PC2 in the Regulation of Fasting Blood Glucose
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_19openalex-snapshot
From Association to Function: MTNR1B
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_2openalex-snapshot
Genome-Wide Association Studies of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-319-01574-3_3openalex-snapshot
Genome-Wide Association Studies of Quantitative Glycaemic Traits
inferred← citeshttps://doi.org/10.1007/978-3-319-27317-4_6-1openalex-snapshot
Genetics of Diabetes and Diabetic Complications
inferred← citeshttps://doi.org/10.1007/978-3-319-41679-3_12openalex-snapshot
Melatonin as a Medicament for the 24/7 Society: Metabolic Syndrome
inferred← citeshttps://doi.org/10.1007/978-3-319-45015-5_6openalex-snapshot
Genetics of Diabetes and Diabetic Complications
inferred← citeshttps://doi.org/10.1007/978-3-319-55330-6_25openalex-snapshot
Insulin Resistance and Mitochondrial Dysfunction
inferred← citeshttps://doi.org/10.1007/978-3-642-19683-6openalex-snapshot
Sensing the Environment: Regulation of Local and Global Homeostasis by the Skin's Neuroendocrine System
inferred← citeshttps://doi.org/10.1007/978-3-642-19683-6_3openalex-snapshot
Melatoninergic System in the Skin
inferred← citeshttps://doi.org/10.1007/978-3-642-32775-9_28openalex-snapshot
Services Design in a Collaborative Network for Multidisciplinary Research Projects
inferred← citeshttps://doi.org/10.1007/978-3-662-46306-2_12openalex-snapshot
Mapping of Susceptibility Genes for Obesity, Type 2 Diabetes, and the Metabolic Syndrome in Human Populations
inferred← citeshttps://doi.org/10.1007/978-81-322-0825-9_1openalex-snapshot
Melatonin Receptors and Their Role in Human Diseases
inferred← citeshttps://doi.org/10.1007/978-81-322-0825-9_6openalex-snapshot
Melatonin and the Metabolic Syndrome
inferred← citeshttps://doi.org/10.1007/978-94-007-6686-0_43openalex-snapshot
Circadian Control of Islet Function
inferred← citeshttps://doi.org/10.1007/978-94-007-6884-0_43-1openalex-snapshot
Circadian Control of Islet Function
inferred← citeshttps://doi.org/10.1007/978-981-13-1286-1_5openalex-snapshot
Central Circadian Clock Regulates Energy Metabolism
inferred← citeshttps://doi.org/10.1007/978-981-13-8177-5_4openalex-snapshot
Genome-Wide Association Study for Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/bf03401539openalex-snapshot
The rs10830963 variant of melatonin receptor MTNR1B is associated with increased risk for gestational diabetes mellitus in a Greek population
inferred← citeshttps://doi.org/10.1007/s00018-017-2611-0openalex-snapshot
Melatonin, mitochondria, and the metabolic syndrome
inferred← citeshttps://doi.org/10.1007/s00125-009-1297-8openalex-snapshot
A common variant in MTNR1B, encoding melatonin receptor 1B, is associated with type 2 diabetes and fasting plasma glucose in Han Chinese individuals
inferred← citeshttps://doi.org/10.1007/s00125-009-1359-yopenalex-snapshot
Melatonin receptors in pancreatic islets: good morning to a novel type 2 diabetes gene
inferred← citeshttps://doi.org/10.1007/s00125-009-1362-3openalex-snapshot
Combined analysis of 19 common validated type 2 diabetes susceptibility gene variants shows moderate discriminative value and no evidence of gene–gene interaction
inferred← citeshttps://doi.org/10.1007/s00125-009-1392-xopenalex-snapshot
Common genetic variation in the melatonin receptor 1B gene (MTNR1B) is associated with decreased early-phase insulin response
inferred← citeshttps://doi.org/10.1007/s00125-009-1413-9openalex-snapshot
Combined effects of single-nucleotide polymorphisms in GCK, GCKR, G6PC2 and MTNR1B on fasting plasma glucose and type 2 diabetes risk
inferred← citeshttps://doi.org/10.1007/s00125-009-1463-zopenalex-snapshot
A variant in the G6PC2/ABCB11 locus is associated with increased fasting plasma glucose, increased basal hepatic glucose production and increased insulin release after oral and intravenous glucose loads
inferred← citeshttps://doi.org/10.1007/s00125-009-1595-1openalex-snapshot
Common variants at the GCK, GCKR, G6PC2–ABCB11 and MTNR1B loci are associated with fasting glucose in two Asian populations
inferred← citeshttps://doi.org/10.1007/s00125-010-1737-5openalex-snapshot
Direct evidence for susceptibility genes for type 2 diabetes on mouse chromosomes 11 and 14
inferred← citeshttps://doi.org/10.1007/s00125-010-1753-5openalex-snapshot
Variants at DGKB/TMEM195, ADRA2A, GLIS3 and C2CD4B loci are associated with reduced glucose-stimulated beta cell function in middle-aged Danish people
inferred← citeshttps://doi.org/10.1007/s00125-010-1790-0openalex-snapshot
Type 2 diabetes risk alleles near ADCY5, CDKAL1 and HHEX-IDE are associated with reduced birthweight
inferred← citeshttps://doi.org/10.1007/s00125-010-1876-8openalex-snapshot
Genetic variants affecting incretin sensitivity and incretin secretion
inferred← citeshttps://doi.org/10.1007/s00125-011-2353-8openalex-snapshot
Common genetic variants differentially influence the transition from clinically defined states of fasting glucose metabolism
inferred← citeshttps://doi.org/10.1007/s00125-011-2355-6openalex-snapshot
Association analysis of 31 common polymorphisms with type 2 diabetes and its related traits in Indian sib pairs
inferred← citeshttps://doi.org/10.1007/s00125-013-2875-3openalex-snapshot
Multiple functional polymorphisms in the G6PC2 gene contribute to the association with higher fasting plasma glucose levels
inferred← citeshttps://doi.org/10.1007/s00125-013-3110-yopenalex-snapshot
Sleep duration does not mediate or modify association of common genetic variants with type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s00125-013-3146-zopenalex-snapshot
Search for genetic determinants of sulfonylurea efficacy in type 2 diabetic patients from China
inferred← citeshttps://doi.org/10.1007/s00125-014-3239-3openalex-snapshot
Genetic variation in MTNR1B is associated with gestational diabetes mellitus and contributes only to the absolute level of beta cell compensation in Mexican Americans
inferred← citeshttps://doi.org/10.1007/s00125-015-3533-8openalex-snapshot
Season-dependent associations of circadian rhythm-regulating loci (CRY1, CRY2 and MTNR1B) and glucose homeostasis: the GLACIER Study
inferred← citeshttps://doi.org/10.1007/s00125-016-3893-8openalex-snapshot
Insomnia does not mediate or modify the association between MTNR1B risk variant rs10830963 and glucose levels
inferred← citeshttps://doi.org/10.1007/s00125-017-4249-8openalex-snapshot
Melatonin signalling and type 2 diabetes risk: too little, too much or just right?
inferred← citeshttps://doi.org/10.1007/s00125-019-05059-6openalex-snapshot
Impact of circadian disruption on glucose metabolism: implications for type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s00125-021-05449-9openalex-snapshot
Multi-omics analysis identifies CpGs near G6PC2 mediating the effects of genetic variants on fasting glucose
inferred← citeshttps://doi.org/10.1007/s00125-021-05635-9openalex-snapshot
Multi-ethnic GWAS and fine-mapping of glycaemic traits identify novel loci in the PAGE Study
inferred← citeshttps://doi.org/10.1007/s00125-022-05747-wopenalex-snapshot
Gut microbiota mediate melatonin signalling in association with type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s00125-025-06381-yopenalex-snapshot
Rare MTNR1B variants causing diminished MT2 signalling associate with elevated HbA1c levels but not with type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s00125-025-06600-6openalex-snapshot
Bridging the variant-to-function gap in type 2 diabetes: advances and challenges
inferred← citeshttps://doi.org/10.1007/s00394-018-1660-yopenalex-snapshot
A circadian rhythm-related MTNR1B genetic variant modulates the effect of weight-loss diets on changes in adiposity and body composition: the POUNDS Lost trial
inferred← citeshttps://doi.org/10.1007/s00406-011-0207-5openalex-snapshot
Neuropsychological endophenotypes in attention-deficit/hyperactivity disorder: a review of genetic association studies
inferred← citeshttps://doi.org/10.1007/s00439-010-0910-8openalex-snapshot
AKT1 polymorphisms are associated with risk for metabolic syndrome
inferred← citeshttps://doi.org/10.1007/s00439-011-1023-8openalex-snapshot
Type 2 diabetes and obesity: genomics and the clinic
inferred← citeshttps://doi.org/10.1007/s00592-009-0129-0openalex-snapshot
Exercise therapy in Type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s00774-023-01480-5openalex-snapshot
Causal relationship between glycemic traits and bone mineral density in different age groups and skeletal sites: a Mendelian randomization analysis
inferred← citeshttps://doi.org/10.1007/s10528-017-9827-4openalex-snapshot
Genetic Approaches to the Study of Gene Variants and Their Impact on the Pathophysiology of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/s10528-021-10045-yopenalex-snapshot
Analysis of Evolution and Ethnic Diversity at Glucose-Associated SNPs of Circadian Clock-Related Loci with Cryptochrome 1, Cryptochrome 2, and Melatonin receptor 1B
inferred← citeshttps://doi.org/10.1007/s10549-011-1884-5openalex-snapshot
Melatonin pathway genes and breast cancer risk among Chinese women
inferred← citeshttps://doi.org/10.1007/s10552-011-9760-5openalex-snapshot
Association study of type 2 diabetes genetic susceptibility variants and risk of pancreatic cancer: an analysis of PanScan-I data
inferred← citeshttps://doi.org/10.1007/s10571-023-01324-wopenalex-snapshot
Melatonin and Health: Insights of Melatonin Action, Biological Functions, and Associated Disorders
inferred← citeshttps://doi.org/10.1007/s10620-018-5242-xopenalex-snapshot
Role of the Circadian Clock in the Metabolic Syndrome and Nonalcoholic Fatty Liver Disease
inferred← citeshttps://doi.org/10.1007/s10654-007-9199-xopenalex-snapshot
The Rotterdam Study: objectives and design update
inferred← citeshttps://doi.org/10.1007/s10654-009-9386-zopenalex-snapshot
The Rotterdam Study: 2010 objectives and design update
inferred← citeshttps://doi.org/10.1007/s10654-011-9610-5openalex-snapshot
The Rotterdam Study: 2012 objectives and design update
inferred← citeshttps://doi.org/10.1007/s11033-021-06431-9openalex-snapshot
Type 2 diabetes is associated with the MTNR1B gene, a genetic bridge between circadian rhythm and glucose metabolism, in a Turkish population
inferred← citeshttps://doi.org/10.1007/s11428-009-0500-3openalex-snapshot
Genetische Studien zum Typ-2-Diabetes
inferred← citeshttps://doi.org/10.1007/s11428-011-0779-8openalex-snapshot
Typ-2-Diabetes-assoziierte Gene
inferred← citeshttps://doi.org/10.1007/s11428-013-1020-8openalex-snapshot
Epigenetische Prozesse beim Typ-2-Diabetes
inferred← citeshttps://doi.org/10.1007/s11892-009-0027-4openalex-snapshot
Genome-wide association studies in type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s11892-010-0143-1openalex-snapshot
The Genetics of Insulin Resistance: Where’s Waldo?
inferred← citeshttps://doi.org/10.1007/s11892-010-0154-yopenalex-snapshot
Physiologic Characterization of Type 2 Diabetes–Related Loci
inferred← citeshttps://doi.org/10.1007/s11892-011-0232-9openalex-snapshot
Genetic Determinants of Variability in Glycated Hemoglobin (HbA1c) in Humans: Review of Recent Progress and Prospects for Use in Diabetes Care
inferred← citeshttps://doi.org/10.1007/s11892-011-0245-4openalex-snapshot
Determining Genetic Risk Factors for Pediatric Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/s11892-012-0255-xopenalex-snapshot
Who Should We Target for Diabetes Prevention and Diabetes Risk Reduction?
inferred← citeshttps://doi.org/10.1007/s11892-012-0321-4openalex-snapshot
What Will Diabetes Genomes Tell Us?
inferred← citeshttps://doi.org/10.1007/s11892-012-0327-yopenalex-snapshot
Tired of Diabetes Genetics? Circadian Rhythms and Diabetes: The MTNR1B Story?
inferred← citeshttps://doi.org/10.1007/s11892-012-0354-8openalex-snapshot
Circadian Rhythms, Metabolism, and Insulin Sensitivity: Transcriptional Networks in Animal Models
inferred← citeshttps://doi.org/10.1007/s11892-013-0422-8openalex-snapshot
Moving on from GWAS: Functional Studies on the G6PC2 Gene Implicated in the Regulation of Fasting Blood Glucose
inferred← citeshttps://doi.org/10.1007/s11892-014-0474-4openalex-snapshot
Does Disruption of Circadian Rhythms Contribute to Beta-Cell Failure in Type 2 Diabetes?
inferred← citeshttps://doi.org/10.1007/s11892-014-0477-1openalex-snapshot
Nutrigenetics: Bridging Two Worlds to Understand Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/s11892-014-0550-9openalex-snapshot
Overlap of Genetic Susceptibility to Type 1 Diabetes, Type 2 Diabetes, and Latent Autoimmune Diabetes in Adults
inferred← citeshttps://doi.org/10.1007/s11892-014-0551-8openalex-snapshot
Insights into the Genetic Susceptibility to Type 2 Diabetes from Genome-Wide Association Studies of Glycaemic Traits
inferred← citeshttps://doi.org/10.1007/s11892-016-0737-3openalex-snapshot
Genome-Wide Studies of Type 2 Diabetes and Lipid Traits in Hispanics
inferred← citeshttps://doi.org/10.1007/s11892-017-0852-9openalex-snapshot
How Can Genetic Studies Help Us to Understand Links Between Birth Weight and Type 2 Diabetes?
inferred← citeshttps://doi.org/10.1007/s11892-017-0957-1openalex-snapshot
Disentangling the Role of Melatonin and its Receptor MTNR1B in Type 2 Diabetes: Still a Long Way to Go?
inferred← citeshttps://doi.org/10.1007/s11892-020-01355-3openalex-snapshot
Genetic Studies of Gestational Diabetes and Glucose Metabolism in Pregnancy
inferred← citeshttps://doi.org/10.1007/s11906-011-0242-7openalex-snapshot
Mendelian Randomization: Application to Cardiovascular Disease
inferred← citeshttps://doi.org/10.1007/s12170-010-0129-1openalex-snapshot
Type 2 Diabetes and Genetics, 2010: Translating Knowledge into Understanding
inferred← citeshttps://doi.org/10.1007/s12170-012-0281-xopenalex-snapshot
Recent Developments in the Genetic and Genomic Basis of Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/s12265-012-9385-4openalex-snapshot
Does Familial Clustering of Risk Factors for Long-Term Diabetic Complications Leave Any Place for Genes That Act independently?
inferred← citeshttps://doi.org/10.1007/s13258-011-0097-yopenalex-snapshot
Effects of MTNR1B variants on fasting glucose levels in a Korean population
inferred← citeshttps://doi.org/10.1007/s13340-015-0202-6openalex-snapshot
Replication study of the association of rs7578597 in THADA, rs10886471 in GRK5, and rs7403531 in RASGRP1 with susceptibility to type 2 diabetes among a Japanese population
inferred← citeshttps://doi.org/10.1007/s13340-016-0279-6openalex-snapshot
Metabolic disturbances: role of the circadian timing system and sleep
inferred← citeshttps://doi.org/10.1007/s40263-019-00680-wopenalex-snapshot
Adverse Events Associated with Melatonin for the Treatment of Primary or Secondary Sleep Disorders: A Systematic Review
inferred← citeshttps://doi.org/10.1007/s40618-023-02014-6openalex-snapshot
A novel combination of sitagliptin and melatonin ameliorates T2D manifestations: studies on experimental diabetic models
inferred← citeshttps://doi.org/10.1007/s43440-024-00683-5openalex-snapshot
Practical application of melatonin for pancreas disorders: protective roles against inflammation, malignancy, and dysfunctions
inferred← citeshttps://doi.org/10.1016/b978-0-12-374934-5.00004-0openalex-snapshot
Genome-Wide Association Studies and Genotyping Technologies
inferred← citeshttps://doi.org/10.1016/b978-0-12-799961-6.00001-9openalex-snapshot
Metabolomics and Cardiovascular Medicine
inferred← citeshttps://doi.org/10.1016/b978-0-12-802928-2.00029-1openalex-snapshot
Genetics and Diabetes
inferred← citeshttps://doi.org/10.1016/b978-0-12-815373-4.00018-6openalex-snapshot
Social jetlag, circadian disruption, and cardiometabolic disease risk
inferred← citeshttps://doi.org/10.1016/b978-0-12-849886-6.00009-4openalex-snapshot
Nutrigenomics and personalized nutrition for the prevention of hyperglycemia and type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/b978-0-323-24288-2.00020-9openalex-snapshot
Endocrine Physiology in Relation to Sleep and Sleep Disturbances
inferred← citeshttps://doi.org/10.1016/b978-0-323-24288-2.00039-8openalex-snapshot
Circadian Dysregulation in Mental and Physical Health
inferred← citeshttps://doi.org/10.1016/b978-0-323-87862-3.00002-1openalex-snapshot
Insulin Resistance: Pathophysiology, Molecular Mechanisms, and Genetic Insights
inferred← citeshttps://doi.org/10.1016/b978-0-443-13954-3.00040-1openalex-snapshot
Social jetlag, circadian disruption, and cardiometabolic disease risk
inferred← citeshttps://doi.org/10.1016/b978-0-443-33590-7.00012-5openalex-snapshot
Genetic and epigenetic landscape of type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/b978-1-4160-6645-3.00026-8openalex-snapshot
Endocrine Physiology in Relation to Sleep and Sleep Disturbances
inferred← citeshttps://doi.org/10.1016/bs.adgen.2024.03.001openalex-snapshot
Host genetics and nutrition
inferred← citeshttps://doi.org/10.1016/j.ajhg.2010.01.025openalex-snapshot
A Weighted False Discovery Rate Control Procedure Reveals Alleles at FOXA2 that Influence Fasting Glucose Levels
inferred← citeshttps://doi.org/10.1016/j.ajhg.2011.11.029openalex-snapshot
Five Years of GWAS Discovery
inferred← citeshttps://doi.org/10.1016/j.ajhg.2012.08.002openalex-snapshot
Genetic Risk Factors for Type 2 Diabetes: A Trans-Regulatory Genetic Architecture?
inferred← citeshttps://doi.org/10.1016/j.bbagen.2012.12.013openalex-snapshot
Multiple roles of glucose-6-phosphatases in pathophysiology
inferred← citeshttps://doi.org/10.1016/j.beem.2010.09.001openalex-snapshot
New evidence for a role of melatonin in glucose regulation
inferred← citeshttps://doi.org/10.1016/j.beem.2011.12.002openalex-snapshot
Genetic determinants of glucose homeostasis
inferred← citeshttps://doi.org/10.1016/j.bmcl.2015.12.102openalex-snapshot
Carbazole-containing sulfonamides and sulfamides: Discovery of cryptochrome modulators as antidiabetic agents
inferred← citeshttps://doi.org/10.1016/j.bmcl.2017.12.051openalex-snapshot
Carbazole-containing amides and ureas: Discovery of cryptochrome modulators as antihyperglycemic agents
inferred← citeshttps://doi.org/10.1016/j.cbpa.2009.04.622openalex-snapshot
Emerging therapies for metabolic diseases—the focus is on diabetes and obesity
inferred← citeshttps://doi.org/10.1016/j.clnu.2017.04.003openalex-snapshot
Late dinner impairs glucose tolerance in MTNR1B risk allele carriers: A randomized, cross-over study
inferred← citeshttps://doi.org/10.1016/j.clnu.2017.08.015openalex-snapshot
Association of the rs10830963 polymorphism in melatonin receptor type 1B (MTNR1B) with metabolic response after weight loss secondary to a hypocaloric diet based in Mediterranean style
inferred← citeshttps://doi.org/10.1016/j.cmet.2010.09.012openalex-snapshot
Global Epigenomic Analysis of Primary Human Pancreatic Islets Provides Insights into Type 2 Diabetes Susceptibility Loci
inferred← citeshttps://doi.org/10.1016/j.cmet.2014.12.020openalex-snapshot
Rare and Common Genetic Events in Type 2 Diabetes: What Should Biologists Know?
inferred← citeshttps://doi.org/10.1016/j.cmet.2016.04.009openalex-snapshot
Increased Melatonin Signaling Is a Risk Factor for Type 2 Diabetes
inferred← citeshttps://doi.org/10.1016/j.coemr.2020.11.008openalex-snapshot
Pharmacogenomics of GPCR genes in type 2 diabetes and obesity
inferred← citeshttps://doi.org/10.1016/j.coi.2009.09.003openalex-snapshot
Novel genetic etiologies of severe congenital neutropenia
inferred← citeshttps://doi.org/10.1016/j.comcom.2020.09.005openalex-snapshot
Designing of fog based FBCMI2E Model using machine learning approaches for intelligent communication systems
inferred← citeshttps://doi.org/10.1016/j.cub.2013.01.048openalex-snapshot
Circadian Disruption Leads to Insulin Resistance and Obesity
inferred← citeshttps://doi.org/10.1016/j.earlhumdev.2020.105219openalex-snapshot
Gestational diabetes, environmental temperature and climate factors – From epidemiological evidence to physiological mechanisms
inferred← citeshttps://doi.org/10.1016/j.endien.2019.12.004openalex-snapshot
Dietary-fat effect of the rs10830963 polymorphism in MTNR1B on insulin resistance in response to 3 months weight-loss diets
inferred← citeshttps://doi.org/10.1016/j.endinu.2019.02.007openalex-snapshot
Efecto del polimorfismo rs10830963 MTNR1B y la composición de grasa de la dieta en la resistencia a la insulina tras la pérdida de peso durante 3 meses
inferred← citeshttps://doi.org/10.1016/j.febslet.2011.03.021openalex-snapshot
Circadian disruption and SCN control of energy metabolism
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2010.01.059openalex-snapshot
Common genetic variation in MTNR1B is associated with serum testosterone, glucose tolerance, and insulin secretion in polycystic ovary syndrome patients
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2018.02.022openalex-snapshot
Untimely oxidative stress in β-cells leads to diabetes – Role of circadian clock in β-cell function
inferred← citeshttps://doi.org/10.1016/j.gene.2015.04.064openalex-snapshot
MTNR1B gene polymorphisms and susceptibility to Type 2 Diabetes: A pilot study in South Indians
inferred← citeshttps://doi.org/10.1016/j.gene.2018.07.066openalex-snapshot
Association of Native American ancestry and common variants in ACE, ADIPOR2, MTNR1B, GCK, TCF7L2 and FTO genes with glycemic traits in Colombian population
inferred← citeshttps://doi.org/10.1016/j.gene.2020.145256openalex-snapshot
Molecular evolution of melatonin receptor genes (mtnr) in vertebrates and its shedding light on mtnr1c
inferred← citeshttps://doi.org/10.1016/j.gene.2022.146378openalex-snapshot
Association of glucokinase gene and glucokinase regulatory protein gene polymorphisms with gestational diabetes mellitus: A case–control study
inferred← citeshttps://doi.org/10.1016/j.genrep.2020.100695openalex-snapshot
MTNR1B common genetic variant is associated with type 2 diabetes mellitus risk
inferred← citeshttps://doi.org/10.1016/j.heliyon.2022.e11786openalex-snapshot
Melatonin receptor 1B gene rs10830963 C/G polymorphism associated with type 2 diabetes mellitus: An updated meta-analysis of 13,752 participants
inferred← citeshttps://doi.org/10.1016/j.heliyon.2024.e33638openalex-snapshot
Establishing causal relationships between insomnia and gestational diabetes mellitus using Mendelian randomization
inferred← citeshttps://doi.org/10.1016/j.hoc.2012.11.004openalex-snapshot
Genetics and Pathophysiology of Severe Congenital Neutropenia Syndromes Unrelated to Neutrophil Elastase
inferred← citeshttps://doi.org/10.1016/j.humimm.2014.01.008openalex-snapshot
Association of KCNQ1 and KLF14 polymorphisms and risk of type 2 diabetes mellitus: A global meta-analysis
inferred← citeshttps://doi.org/10.1016/j.humimm.2015.03.005openalex-snapshot
Association of LRP5, TCF7L2, and GCG variants and type 2 diabetes mellitus as well as fasting plasma glucose and lipid metabolism indexes
inferred← citeshttps://doi.org/10.1016/j.jacc.2012.02.043openalex-snapshot
Nonfasting Glucose, Ischemic Heart Disease, and Myocardial Infarction
inferred← citeshttps://doi.org/10.1016/j.jdiacomp.2020.107534openalex-snapshot
A circadian rhythm-related MTNR1B genetic variant (rs10830963) modulate body weight change and insulin resistance after 9 months of a high protein/low carbohydrate vs a standard hypocaloric diet
inferred← citeshttps://doi.org/10.1016/j.jmb.2019.12.038openalex-snapshot
Human Physiology of Genetic Defects Causing Beta-cell Dysfunction
inferred← citeshttps://doi.org/10.1016/j.lfs.2024.122993openalex-snapshot
Role of melatonin in mitigation of insulin resistance and ensuing diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.mam.2023.101213openalex-snapshot
WITHDRAWN: Gene-environment interactions that influence CVD, lipid traits, obesity, diabetes, and hypertension appear to be able to influence gene therapy
inferred← citeshttps://doi.org/10.1016/j.mce.2013.04.002openalex-snapshot
Genetics of diabetes – Are we missing the genes or the disease?
inferred← citeshttps://doi.org/10.1016/j.mce.2015.01.024openalex-snapshot
Circadian rhythms in glucose and lipid metabolism in nocturnal and diurnal mammals
inferred← citeshttps://doi.org/10.1016/j.mce.2024.112439openalex-snapshot
Melatonin induces white-to-beige adipocyte transdifferentiation through melatonin receptor 1-mediated direct browning and indirect M2 polarization
inferred← citeshttps://doi.org/10.1016/j.metabol.2011.11.013openalex-snapshot
Regulation of core clock genes in human islets
inferred← citeshttps://doi.org/10.1016/j.metabol.2015.08.003openalex-snapshot
Common type 2 diabetes risk variant in MTNR1B worsens the deleterious effect of melatonin on glucose tolerance in humans
inferred← citeshttps://doi.org/10.1016/j.metabol.2017.10.008openalex-snapshot
Sleep devices: wearables and nearables, informational and interventional, consumer and clinical
inferred← citeshttps://doi.org/10.1016/j.mito.2018.04.004openalex-snapshot
Etiology of type 2 diabetes and Alzheimer's disease: Exploring the mitochondria
inferred← citeshttps://doi.org/10.1016/j.mmm.2025.04.009openalex-snapshot
Diabètes monogéniques et oligogéniques : mieux les reconnaître pour une médecine de précision
inferred← citeshttps://doi.org/10.1016/j.molcel.2011.07.019openalex-snapshot
SIRT3 Deficiency and Mitochondrial Protein Hyperacetylation Accelerate the Development of the Metabolic Syndrome
inferred← citeshttps://doi.org/10.1016/j.molmed.2010.06.004openalex-snapshot
The emerging genetics of type 2 diabetes
inferred← citeshttps://doi.org/10.1016/j.molmed.2023.08.009openalex-snapshot
Monoamines' role in islet cell function and type 2 diabetes risk
inferred← citeshttps://doi.org/10.1016/j.molmet.2014.03.002openalex-snapshot
Circadian control of glucose metabolism
inferred← citeshttps://doi.org/10.1016/j.ncl.2019.05.002openalex-snapshot
Genetics of Circadian Rhythms
inferred← citeshttps://doi.org/10.1016/j.neuint.2016.09.006openalex-snapshot
Melatonin attenuates the high-fat diet and streptozotocin-induced reduction in rat hippocampal neurogenesis
showing the first 200 of 712 · page through all 712
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.