On the public citation graph
Meta-analysis of genome-wide association data identifies a risk locus for major mood disorders on 3p21.1
0 signed · 202 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/gepi.20297openalex-snapshot
Estimation of significance thresholds for genomewide association scans
inferredcites →https://doi.org/10.1016/b978-008055232-3.60649-xopenalex-snapshot
Manic-Depressive Illness
inferredcites →https://doi.org/10.1016/j.ajhg.2009.01.013openalex-snapshot
Genotype-Imputation Accuracy across Worldwide Human Populations
inferredcites →https://doi.org/10.1016/j.biochi.2008.10.019openalex-snapshot
Polybromo-1: The chromatin targeting subunit of the PBAF complex
inferredcites →https://doi.org/10.1016/s0140-6736(84)92810-1openalex-snapshot
MANIC DEPRESSIVE ILLNESS
inferredcites →https://doi.org/10.1038/mp.2008.126openalex-snapshot
A framework for interpreting genome-wide association studies of psychiatric disorders
inferredcites →https://doi.org/10.1038/mp.2008.13openalex-snapshot
Impairment in role functioning in mental and chronic medical disorders in the United States: results from the National Comorbidity Survey Replication
inferredcites →https://doi.org/10.1038/mp.2008.131openalex-snapshot
Genome-wide association study of recurrent major depressive disorder in two European case–control cohorts
inferredcites →https://doi.org/10.1038/mp.2008.133openalex-snapshot
Gene-wide analyses of genome-wide association data sets: evidence for multiple common risk alleles for schizophrenia and bipolar disorder and for overlap in genetic risk
inferredcites →https://doi.org/10.1038/mp.2009.43openalex-snapshot
Genome-wide association study of bipolar disorder in European American and African American individuals
inferredcites →https://doi.org/10.1038/nature05911openalex-snapshot
Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
inferredcites →https://doi.org/10.1038/nature08185openalex-snapshot
Common polygenic variation contributes to risk of schizophrenia and bipolar disorder
inferredcites →https://doi.org/10.1038/ng.2007.16openalex-snapshot
A survey of genetic human cortical gene expression
inferredcites →https://doi.org/10.1038/sj.ejhg.5201979openalex-snapshot
Genome-wide association of major depression: description of samples for the GAIN Major Depressive Disorder Study: NTR and NESDA biobank projects
inferredcites →https://doi.org/10.1038/sj.mp.4002012openalex-snapshot
A genome-wide association study implicates diacylglycerol kinase eta (DGKH) and several other genes in the etiology of bipolar disorder
inferredcites →https://doi.org/10.1038/sj.mp.4002151openalex-snapshot
Whole-genome association study of bipolar disorder
inferredcites →https://doi.org/10.1073/pnas.0813386106openalex-snapshot
Genome-wide association and meta-analysis of bipolar disorder in individuals of European ancestry
inferredcites →https://doi.org/10.1086/519795openalex-snapshot
PLINK: A Tool Set for Whole-Genome Association and Population-Based Linkage Analyses
inferredcites →https://doi.org/10.1093/acprof:oso/9780195326543.001.0001openalex-snapshot
Systematic Reviews and Meta-Analysis
inferredcites →https://doi.org/10.1093/aje/kwn235openalex-snapshot
Seven Lipoprotein Lipase Gene Polymorphisms, Lipid Fractions, and Coronary Disease: A HuGE Association Review and Meta-Analysis
inferredcites →https://doi.org/10.1093/bioinformatics/19.1.149openalex-snapshot
Genetic Power Calculator: design of linkage and association genetic mapping studies of complex traits
inferredcites →https://doi.org/10.1093/nar/29.9.e45openalex-snapshot
A new mathematical model for relative quantification in real-time RT-PCR
inferredcites →https://doi.org/10.1101/gad.55671openalex-snapshot
A nucleolar mechanism controlling cell proliferation in stem cells and cancer cells
inferredcites →https://doi.org/10.1101/gr.3216905openalex-snapshot
Cis -acting expression quantitative trait loci in mice
inferredcites →https://doi.org/10.1111/j.0006-341x.1999.00997.xopenalex-snapshot
Genomic Control for Association Studies
inferredcites →https://doi.org/10.1111/j.1365-2753.2008.00986.xopenalex-snapshot
Interpretation of tests of heterogeneity and bias in meta‐analysis
inferredcites →https://doi.org/10.1172/jci34772openalex-snapshot
A HapMap harvest of insights into the genetics of common disease
inferredcites →https://doi.org/10.1192/bjp.184.5.386openalex-snapshot
Global burden of depressive disorders in the year 2000
inferredcites →https://doi.org/10.1371/journal.pbio.1000001openalex-snapshot
Tissue-Specific Genetic Control of Splicing: Implications for the Study of Complex Traits
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citesno DOI in the source recordopenalex-snapshot
Staphylococcal nasal carriage of health care workers.
inferred← citeshttps://doi.org/10.1002/9780470015902.a0025048openalex-snapshot
Genetics of Depression
inferred← citeshttps://doi.org/10.1002/ajmg.b.31172openalex-snapshot
Genome‐wide association analysis of age at onset and psychotic symptoms in bipolar disorder
inferred← citeshttps://doi.org/10.1002/ajmg.b.32069openalex-snapshot
Genetic heterogeneity according to age at onset in bipolar disorder: A combined positional cloning and candidate gene approach
inferred← citeshttps://doi.org/10.1002/ajmg.b.32127openalex-snapshot
Refinement of chromosome 3p22.3 region and identification of a susceptibility gene for bipolar affective disorder
inferred← citeshttps://doi.org/10.1002/ajmg.b.32132openalex-snapshot
Selected rapporteur summaries from the XX world congress of psychiatric genetics, Hamburg, Germany, october 14–18, 2012
inferred← citeshttps://doi.org/10.1002/ajmg.b.32174openalex-snapshot
Disorders and borders: Psychiatric genetics and nosology
inferred← citeshttps://doi.org/10.1002/ajmg.b.32455openalex-snapshot
The role of protein intrinsic disorder in major psychiatric disorders
inferred← citeshttps://doi.org/10.1007/7854_2010_110openalex-snapshot
Drosophila as a Model Organism for the Study of Neuropsychiatric Disorders
inferred← citeshttps://doi.org/10.1007/7854_2011_185openalex-snapshot
Behavioral Genetics of Affective and Anxiety Disorders
inferred← citeshttps://doi.org/10.1007/7854_2012_225openalex-snapshot
The Genetic Basis of Depression
inferred← citeshttps://doi.org/10.1007/978-1-4614-0514-6_13openalex-snapshot
New Frontiers in Nucleolar Research: Nucleostemin and Related Proteins
inferred← citeshttps://doi.org/10.1007/978-3-642-17426-1_3openalex-snapshot
Possible Roles of DNA Methylation in Bipolar Disorder
inferred← citeshttps://doi.org/10.1007/978-94-007-5558-1_5openalex-snapshot
QTL Mapping of Molecular Traits for Studies of Human Complex Diseases
inferred← citeshttps://doi.org/10.1007/978-981-13-1071-3_6openalex-snapshot
QTL Mapping of Molecular Traits for Studies of Human Complex Diseases
inferred← citeshttps://doi.org/10.1007/s00115-010-3053-1openalex-snapshot
Die Zukunft der Depressionsforschung
inferred← citeshttps://doi.org/10.1007/s11019-012-9408-6openalex-snapshot
Genetics and psychiatry: a proposal for the application of the precautionary principle
inferred← citeshttps://doi.org/10.1007/s11920-010-0150-6openalex-snapshot
Overview of the Genetics of Major Depressive Disorder
inferred← citeshttps://doi.org/10.1007/s11920-011-0226-yopenalex-snapshot
Genome-Wide Searches for Bipolar Disorder Genes
inferred← citeshttps://doi.org/10.1007/s11920-012-0256-0openalex-snapshot
Animal Models of Stress Vulnerability and Resilience in Translational Research
inferred← citeshttps://doi.org/10.1007/s12035-016-0041-xopenalex-snapshot
Identification of a Bipolar Disorder Vulnerable Gene CHDH at 3p21.1
inferred← citeshttps://doi.org/10.1007/s12035-016-9990-3openalex-snapshot
Evaluation of European Schizophrenia GWAS Loci in Asian Populations via Comprehensive Meta-Analyses
inferred← citeshttps://doi.org/10.1007/s40142-014-0030-1openalex-snapshot
Genetics of Psychosis in Alzheimer Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-382227-7.00088-4openalex-snapshot
Schizophrenia and Bipolar Disorder
inferred← citeshttps://doi.org/10.1016/b978-0-12-383834-6.00117-8openalex-snapshot
Schizophrenia and Affective Disorders
inferred← citeshttps://doi.org/10.1016/b978-0-12-800105-9.00002-0openalex-snapshot
Contribution of Genetic Epidemiology to Our Understanding of Psychiatric Disorders
inferred← citeshttps://doi.org/10.1016/b978-0-12-819182-8.00011-9openalex-snapshot
Genetic factors in the etiology of bipolar disorder
inferred← citeshttps://doi.org/10.1016/j.ajhg.2011.04.014openalex-snapshot
Random-Effects Model Aimed at Discovering Associations in Meta-Analysis of Genome-wide Association Studies
inferred← citeshttps://doi.org/10.1016/j.biopsych.2010.09.042openalex-snapshot
Gene Expression and Genetic Variation Data Implicate PCLO in Bipolar Disorder
inferred← citeshttps://doi.org/10.1016/j.biopsych.2012.02.040openalex-snapshot
Replication Study and Meta-Analysis in European Samples Supports Association of the 3p21.1 Locus with Bipolar Disorder
inferred← citeshttps://doi.org/10.1016/j.biopsych.2013.03.033openalex-snapshot
A Genome-wide Association Analysis of a Broad Psychosis Phenotype Identifies Three Loci for Further Investigation
inferred← citeshttps://doi.org/10.1016/j.coph.2011.08.003openalex-snapshot
From genes to therapeutic targets for psychiatric disorders – what to expect?
inferred← citeshttps://doi.org/10.1016/j.dscb.2025.100304openalex-snapshot
CRISPR-Cas9 gene editing in neuropsychiatric disorders: Challenges and promising avenues
inferred← citeshttps://doi.org/10.1016/j.ebiom.2024.105530openalex-snapshot
Shared genetic architecture and bidirectional clinical risks within the psycho-metabolic nexus
inferred← citeshttps://doi.org/10.1016/j.gene.2017.12.025openalex-snapshot
Exome sequencing of a large family identifies potential candidate genes contributing risk to bipolar disorder
inferred← citeshttps://doi.org/10.1016/j.jad.2012.08.020openalex-snapshot
Aberrant executive attention in unaffected youth at familial risk for mood disorders
inferred← citeshttps://doi.org/10.1016/j.jad.2013.10.027openalex-snapshot
Investigating the genetic variation underlying episodicity in major depressive disorder: Suggestive evidence for a bipolar contribution
inferred← citeshttps://doi.org/10.1016/j.jad.2018.07.046openalex-snapshot
Multivariate analysis of genome-wide data to identify potential pleiotropic genes for five major psychiatric disorders using MetaCCA
inferred← citeshttps://doi.org/10.1016/j.jpsychires.2012.04.017openalex-snapshot
ANK3 and CACNA1C – Missing genetic link for bipolar disorder and major depressive disorder in two German case-control samples
inferred← citeshttps://doi.org/10.1016/j.jpsychires.2015.06.004openalex-snapshot
Examining gene–environment interactions in comorbid depressive and disruptive behavior disorders using a Bayesian approach
inferred← citeshttps://doi.org/10.1016/j.jpsychires.2019.04.007openalex-snapshot
Unravelling the genetic basis of schizophrenia and bipolar disorder with GWAS: A systematic review
inferred← citeshttps://doi.org/10.1016/j.mehy.2018.02.009openalex-snapshot
Molecular hypotheses to explain the shared pathways and underlying pathobiological causes in catatonia and in catatonic presentations in neuropsychiatric disorders
inferred← citeshttps://doi.org/10.1016/j.nbd.2010.08.005openalex-snapshot
Does gene deletion of AMPA GluA1 phenocopy features of schizoaffective disorder?
inferred← citeshttps://doi.org/10.1016/j.neubiorev.2011.09.001openalex-snapshot
Genome wide association studies (GWAS) and copy number variation (CNV) studies of the major psychoses: What have we learnt?
inferred← citeshttps://doi.org/10.1016/j.neubiorev.2016.05.031openalex-snapshot
BDNF Val66Met polymorphism and bipolar disorder in European populations: A risk association in case-control, family-based and GWAS studies
inferred← citeshttps://doi.org/10.1016/j.neuron.2014.05.042openalex-snapshot
MicroRNA 135 Is Essential for Chronic Stress Resiliency, Antidepressant Efficacy, and Intact Serotonergic Activity
inferred← citeshttps://doi.org/10.1016/j.pnpbp.2025.111396openalex-snapshot
Identifying novel protein biomarkers with cross-psychiatric disorders effects and potential intervention targets: Evidence from proteomic-Mendelian randomization
inferred← citeshttps://doi.org/10.1016/j.psychres.2010.06.008openalex-snapshot
Depression symptom severity and error-related brain activity
inferred← citeshttps://doi.org/10.1016/j.schres.2014.10.055openalex-snapshot
Innate immune response is differentially dysregulated between bipolar disease and schizophrenia
inferred← citeshttps://doi.org/10.1016/j.tips.2011.02.013openalex-snapshot
Personalized medicine can pave the way for the safe use of CB1 receptor antagonists
inferred← citeshttps://doi.org/10.1016/j.yebeh.2015.07.003openalex-snapshot
Epilepsy and bipolar disorder
inferred← citeshttps://doi.org/10.1016/s0140-6736(12)62129-1openalex-snapshot
Identification of risk loci with shared effects on five major psychiatric disorders: a genome-wide analysis
inferred← citeshttps://doi.org/10.1016/s0140-6736(13)60855-7openalex-snapshot
Genetics of bipolar disorder
inferred← citeshttps://doi.org/10.1017/cbo9781107477278.014openalex-snapshot
Genetic factors in the etiology of bipolar disorder
inferred← citeshttps://doi.org/10.1037/a0036295openalex-snapshot
Response inhibition and psychopathology: A meta-analysis of go/no-go task performance.
inferred← citeshttps://doi.org/10.1038/mp.2010.109openalex-snapshot
Genome-wide association study of major depressive disorder: new results, meta-analysis, and lessons learned
inferred← citeshttps://doi.org/10.1038/mp.2011.125openalex-snapshot
Genome-wide association study of Alzheimer's disease with psychotic symptoms
inferred← citeshttps://doi.org/10.1038/mp.2011.157openalex-snapshot
Genome-wide association study meta-analysis of European and Asian-ancestry samples identifies three novel loci associated with bipolar disorder
inferred← citeshttps://doi.org/10.1038/mp.2011.4openalex-snapshot
A genome-wide association study of attempted suicide
inferred← citeshttps://doi.org/10.1038/mp.2012.13openalex-snapshot
Prediction of susceptibility to major depression by a model of interactions of multiple functional genetic variants and environmental factors
inferred← citeshttps://doi.org/10.1038/mp.2012.157openalex-snapshot
Common variant at 16p11.2 conferring risk of psychosis
inferred← citeshttps://doi.org/10.1038/mp.2012.21openalex-snapshot
A mega-analysis of genome-wide association studies for major depressive disorder
inferred← citeshttps://doi.org/10.1038/mp.2012.48openalex-snapshot
Association at SYNE1 in both bipolar disorder and recurrent major depression
inferred← citeshttps://doi.org/10.1038/mp.2012.67openalex-snapshot
Genome-wide significant associations in schizophrenia to ITIH3/4, CACNA1C and SDCCAG8, and extensive replication of associations reported by the Schizophrenia PGC
inferred← citeshttps://doi.org/10.1038/mp.2016.231openalex-snapshot
The protocadherin 17 gene affects cognition, personality, amygdala structure and function, synapse development and risk of major mood disorders
inferred← citeshttps://doi.org/10.1038/mp.2017.243openalex-snapshot
The cAMP responsive element-binding (CREB)-1 gene increases risk of major psychiatric disorders
inferred← citeshttps://doi.org/10.1038/nature09856openalex-snapshot
Post-traumatic stress disorder is associated with PACAP and the PAC1 receptor
inferred← citeshttps://doi.org/10.1038/ng.3062openalex-snapshot
Data use under the NIH GWAS Data Sharing Policy and future directions
inferred← citeshttps://doi.org/10.1038/ng.3801openalex-snapshot
Diversity in non-repetitive human sequences not found in the reference genome
inferred← citeshttps://doi.org/10.1038/ng0111-3openalex-snapshot
Replication of association of 3p21.1 with susceptibility to bipolar disorder but not major depression
inferred← citeshttps://doi.org/10.1038/ng0111-5openalex-snapshot
Reply to “Replication of association of 3p21.1 with susceptibility to bipolar disorder but not major depression”
inferred← citeshttps://doi.org/10.1038/nrg3240openalex-snapshot
Genetic architectures of psychiatric disorders: the emerging picture and its implications
inferred← citeshttps://doi.org/10.1038/s10038-024-01281-2openalex-snapshot
Contribution of rare variants to heritability of a disease is much greater than conventionally estimated: modification of allele distribution model
inferred← citeshttps://doi.org/10.1038/s41380-019-0592-0openalex-snapshot
The genome-wide risk alleles for psychiatric disorders at 3p21.1 show convergent effects on mRNA expression, cognitive function, and mushroom dendritic spine
inferred← citeshttps://doi.org/10.1038/s41380-019-0634-7openalex-snapshot
The genetics of bipolar disorder
inferred← citeshttps://doi.org/10.1038/s41380-020-00866-5openalex-snapshot
Integrative analyses prioritize GNL3 as a risk gene for bipolar disorder
inferred← citeshttps://doi.org/10.1038/s41380-021-01151-9openalex-snapshot
CADPS functional mutations in patients with bipolar disorder increase the sensitivity to stress
inferred← citeshttps://doi.org/10.1038/s41380-025-02933-1openalex-snapshot
Human mood disorder risk gene Synaptotagmin-14 contributes to mania-like behaviors in mice
inferred← citeshttps://doi.org/10.1038/s41398-017-0019-0openalex-snapshot
Common variants at 2q11.2, 8q21.3, and 11q13.2 are associated with major mood disorders
inferred← citeshttps://doi.org/10.1038/s41398-018-0337-xopenalex-snapshot
Replicated associations of FADS1, MAD1L1, and a rare variant at 10q26.13 with bipolar disorder in Chinese population
inferred← citeshttps://doi.org/10.1038/s41398-020-0708-yopenalex-snapshot
Novel genetic susceptibility loci identified by family based whole exome sequencing in Han Chinese schizophrenia patients
inferred← citeshttps://doi.org/10.1038/s41398-021-01500-wopenalex-snapshot
Assessment of suicide attempt and death in bipolar affective disorder: a combined clinical and genetic approach
inferred← citeshttps://doi.org/10.1038/s41398-021-01608-zopenalex-snapshot
Genome-wide differentially methylated genes associated with posttraumatic stress disorder and longitudinal change in methylation in rape survivors
inferred← citeshttps://doi.org/10.1038/s41467-024-53585-8openalex-snapshot
NEK4 modulates circadian fluctuations of emotional behaviors and synaptogenesis in male mice
inferred← citeshttps://doi.org/10.1038/s41598-018-26042-yopenalex-snapshot
Genetic variant for behavioral regulation factor of executive function and its possible brain mechanism in attention deficit hyperactivity disorder
inferred← citeshttps://doi.org/10.1038/s42003-021-02356-yopenalex-snapshot
New and sex-specific migraine susceptibility loci identified from a multiethnic genome-wide meta-analysis
inferred← citeshttps://doi.org/10.1038/s42003-024-07236-9openalex-snapshot
Accounting for heterogeneity due to environmental sources in meta-analysis of genome-wide association studies
inferred← citeshttps://doi.org/10.1038/srep17156openalex-snapshot
Copy number variations play important roles in heredity of common diseases: a novel method to calculate heritability of a polymorphism
inferred← citeshttps://doi.org/10.1038/tp.2011.42openalex-snapshot
Meta-analysis of genome-wide association studies identifies common variants in CTNNA2 associated with excitement-seeking
inferred← citeshttps://doi.org/10.1038/tp.2014.71openalex-snapshot
Investigation of manic and euthymic episodes identifies state- and trait-specific gene expression and STAB1 as a new candidate gene for bipolar disorder
inferred← citeshttps://doi.org/10.1038/tp.2015.42openalex-snapshot
Genome-wide association study of behavioural and psychiatric features in human prion disease
inferred← citeshttps://doi.org/10.1038/tpj.2011.9openalex-snapshot
Interaction networks of lithium and valproate molecular targets reveal a striking enrichment of apoptosis functional clusters and neurotrophin signaling
inferred← citeshttps://doi.org/10.1080/13546805.2012.690938openalex-snapshot
Genetic and neurocognitive foundations of emotion abnormalities in bipolar disorder
inferred← citeshttps://doi.org/10.1080/14737175.2019.1550361openalex-snapshot
Biomarkers for bipolar disorder: current status and challenges ahead
inferred← citeshttps://doi.org/10.1093/bioinformatics/btu783openalex-snapshot
MGAS: a powerful tool for multivariate gene-based genome-wide association analysis
inferred← citeshttps://doi.org/10.1093/biomtc/ujag035openalex-snapshot
Ultra-high-dimensional threshold selection for quantile feature screening with false discovery rate error rate control: a case study on high blood pressure analysis
inferred← citeshttps://doi.org/10.1093/hmg/ddq368openalex-snapshot
Synthetic associations in the context of genome-wide association scan signals
inferred← citeshttps://doi.org/10.1093/hmg/ddq452openalex-snapshot
The Machado–Joseph disease-associated mutant form of ataxin-3 regulates parkin ubiquitination and stability
inferred← citeshttps://doi.org/10.1093/hmg/ddq471openalex-snapshot
Most genome-wide significant susceptibility loci for schizophrenia and bipolar disorder reported to date cross-traditional diagnostic boundaries
inferred← citeshttps://doi.org/10.1093/ijnp/pyu019openalex-snapshot
Proteomic Enrichment Analysis of Psychotic and Affective Disorders Reveals Common Signatures in Presynaptic Glutamatergic Signaling and Energy Metabolism
inferred← citeshttps://doi.org/10.1093/med/9780199797608.003.0089openalex-snapshot
Genetics of Bipolar Disorder and Schizophrenia
inferred← citeshttps://doi.org/10.1097/jcma.0000000000001245openalex-snapshot
Association of PBRM1 rs2251219 with major depressive disorder and biochemical variables in a Taiwanese cohort
inferred← citeshttps://doi.org/10.1097/ypg.0b013e32833d17c3openalex-snapshot
Selected summaries from the XVII World Congress of Psychiatric Genetics, San Diego, California, USA, 4–8 November 2009
inferred← citeshttps://doi.org/10.1097/ypg.0b013e328341a346openalex-snapshot
Database of genetic studies of bipolar disorder
inferred← citeshttps://doi.org/10.1097/ypg.0b013e32835d7028openalex-snapshot
A large-scale candidate gene analysis of mood disorders
inferred← citeshttps://doi.org/10.1101/116624openalex-snapshot
Genotype-phenotype association mining in bipolar disorder: market research meets complex genetics
inferred← citeshttps://doi.org/10.1101/2025.06.23.25330155openalex-snapshot
Genomic dimensions deconstruct the clinical heterogeneity of bipolar disorder
inferred← citeshttps://doi.org/10.1111/bdi.12489openalex-snapshot
Longitudinal differences in white matter integrity in youth at high familial risk for bipolar disorder
inferred← citeshttps://doi.org/10.1111/j.1369-1600.2011.00365.xopenalex-snapshot
Protracted abstinence from distinct drugs of abuse shows regulation of a common gene network
inferred← citeshttps://doi.org/10.1111/j.1399-0004.2011.01773.xopenalex-snapshot
Recent genomic advances in schizophrenia
inferred← citeshttps://doi.org/10.1111/j.1440-1819.2011.02193.xopenalex-snapshot
Nominal association between a polymorphism in DGKH and bipolar disorder detected in a meta‐analysis of East Asian case–control samples
inferred← citeshttps://doi.org/10.1111/j.1530-0277.2011.01564.xopenalex-snapshot
Measures of Current Alcohol Consumption and Problems: Two Independent Twin Studies Suggest a Complex Genetic Architecture
inferred← citeshttps://doi.org/10.1111/j.1601-183x.2011.00721.xopenalex-snapshot
Genome-wide association study on bipolar disorder in the Bulgarian population
inferred← citeshttps://doi.org/10.1111/j.1755-5949.2010.00187.xopenalex-snapshot
Genomics and Pharmacogenomics of Schizophrenia
inferred← citeshttps://doi.org/10.1111/jnu.12006openalex-snapshot
The Implications of Genomics on the Nursing Care of Adults With Neuropsychiatric Conditions
inferred← citeshttps://doi.org/10.1111/pcn.12528openalex-snapshot
Application of induced pluripotent stem cells to understand neurobiological basis of bipolar disorder and schizophrenia
inferred← citeshttps://doi.org/10.1155/2017/7014146openalex-snapshot
Physical Activity Modulates Common Neuroplasticity Substrates in Major Depressive and Bipolar Disorder
inferred← citeshttps://doi.org/10.1159/000327158openalex-snapshot
Modifiers and Subtype-Specific Analyses in Whole-Genome Association Studies: A Likelihood Framework
inferred← citeshttps://doi.org/10.1159/000437323openalex-snapshot
A Case Study of Fixed-Effects and Random-Effects Meta-Analysis Models for Genome-Wide Association Studies in Celiac Disease
inferred← citeshttps://doi.org/10.1159/000488590openalex-snapshot
Further Evidence of an Association between NCAN rs1064395 and Bipolar Disorder
inferred← citeshttps://doi.org/10.1159/000493941openalex-snapshot
VRK2, a Candidate Gene for Psychiatric and Neurological Disorders
inferred← citeshttps://doi.org/10.1176/appi.ajp.2010.10040541openalex-snapshot
Genome-Wide Association Study of Suicide Attempts in Mood Disorder Patients
inferred← citeshttps://doi.org/10.1176/appi.ajp.2011.10091319openalex-snapshot
A 3p26-3p25 Genetic Linkage Finding for DSM-IV Major Depression in Heavy Smoking Families
inferred← citeshttps://doi.org/10.1176/appi.neuropsych.14060132openalex-snapshot
A Novel Anxiety and Affective Spectrum Disorder of Mind and Body—The ALPIM (Anxiety-Laxity-Pain-Immune-Mood) Syndrome: A Preliminary Report
inferred← citeshttps://doi.org/10.1177/02698811211032609openalex-snapshot
Association study of BDNF Val66Met gene polymorphism with bipolar disorder and lithium treatment response in Indian population
inferred← citeshttps://doi.org/10.1177/1073858410381533openalex-snapshot
Genome-wide Association: From Confounded to Confident
inferred← citeshttps://doi.org/10.1186/1471-2350-15-2openalex-snapshot
Genome-wide association study of bipolar disorder in Canadian and UK populations corroborates disease loci including SYNE1 and CSMD1
inferred← citeshttps://doi.org/10.1186/1471-244x-10-59openalex-snapshot
The DAOA/G30 locus and affective disorders: haplotype based association study in a polydiagnostic approach
inferred← citeshttps://doi.org/10.1186/2193-9616-1-15openalex-snapshot
Comparison of gene expression profiles in the blood, hippocampus and prefrontal cortex of rats
inferred← citeshttps://doi.org/10.1186/s12863-015-0274-0openalex-snapshot
Comparative genome-wide association studies of a depressive symptom phenotype in a repeated measures setting by race/ethnicity in the multi-ethnic study of atherosclerosis
inferred← citeshttps://doi.org/10.1186/s12916-023-02931-6openalex-snapshot
Identification of a psychiatric risk gene NISCH at 3p21.1 GWAS locus mediating dendritic spine morphogenesis and cognitive function
inferred← citeshttps://doi.org/10.1186/s13073-016-0308-xopenalex-snapshot
Identification of the BRD1 interaction network and its impact on mental disorder risk
inferred← citeshttps://doi.org/10.1186/s40345-018-0132-xopenalex-snapshot
Detecting significant genotype–phenotype association rules in bipolar disorder: market research meets complex genetics
inferred← citeshttps://doi.org/10.1186/s40345-021-00228-2openalex-snapshot
Predicting the risk and timing of major mood disorder in offspring of bipolar parents: exploring the utility of a neural network approach
inferred← citeshttps://doi.org/10.1186/s41065-019-0095-7openalex-snapshot
Association of SYNE1 locus with bipolar disorder in Chinese population
inferred← citeshttps://doi.org/10.1192/bjp.bp.110.079665openalex-snapshot
From predisposition to illness: genetically sensitive intermediate pathways to mood disorders
inferred← citeshttps://doi.org/10.1192/bjp.bp.114.156976openalex-snapshot
Impact of a cis-associated gene expression SNP on chromosome 20q11.22 on bipolar disorder susceptibility, hippocampal structure and cognitive performance
inferred← citeshttps://doi.org/10.1192/j.eurpsy.2019.6openalex-snapshot
An atlas of genetic correlations between psychiatric disorders and human blood plasma proteome
inferred← citeshttps://doi.org/10.1371/journal.pgen.1002134openalex-snapshot
Genome-Wide Association of Bipolar Disorder Suggests an Enrichment of Replicable Associations in Regions near Genes
inferred← citeshttps://doi.org/10.1371/journal.pgen.1004229openalex-snapshot
Genomic View of Bipolar Disorder Revealed by Whole Genome Sequencing in a Genetic Isolate
inferred← citeshttps://doi.org/10.1371/journal.pone.0009697openalex-snapshot
A New Statistic to Evaluate Imputation Reliability
inferred← citeshttps://doi.org/10.1371/journal.pone.0015463openalex-snapshot
Duplication of the SLIT3 Locus on 5q35.1 Predisposes to Major Depressive Disorder
inferred← citeshttps://doi.org/10.1371/journal.pone.0051504openalex-snapshot
Molecular Sex Differences in Human Serum
inferred← citeshttps://doi.org/10.1371/journal.pone.0064947openalex-snapshot
Possible Associations of NTRK2 Polymorphisms with Antidepressant Treatment Outcome: Findings from an Extended Tag SNP Approach
inferred← citeshttps://doi.org/10.1371/journal.pone.0070964openalex-snapshot
Genetic Variants on 3q21 and in the Sp8 Transcription Factor Gene (SP8) as Susceptibility Loci for Psychotic Disorders: A Genetic Association Study
inferred← citeshttps://doi.org/10.1371/journal.pone.0128605openalex-snapshot
Explore the Features of Brain-Derived Neurotrophic Factor in Mood Disorders
inferred← citeshttps://doi.org/10.1371/journal.pone.0157086openalex-snapshot
Functional Characterization of Schizophrenia-Associated Variation in CACNA1C
inferred← citeshttps://doi.org/10.1586/ecp.10.35openalex-snapshot
Imaging genetics: implications for research on variable antidepressant drug response
inferred← citeshttps://doi.org/10.2217/pgs-2020-0157openalex-snapshot
Therapy Response Prediction in Major Depressive Disorder: Current and Novel Genomic Markers Influencing Pharmacokinetics and Pharmacodynamics
inferred← citeshttps://doi.org/10.2217/pme.12.67openalex-snapshot
Research Highlights: Highlights from the Latest Articles in Psychiatric Genetics
inferred← citeshttps://doi.org/10.3109/15622975.2011.596220openalex-snapshot
The genetic blueprint of major depressive disorder: Contributions of imaging genetics studies
inferred← citeshttps://doi.org/10.3389/fgene.2013.00087openalex-snapshot
Converging evidence for epistasis between ANK3 and potassium channel gene KCNQ2 in bipolar disorder
inferred← citeshttps://doi.org/10.3389/fpsyt.2014.00098openalex-snapshot
Integrated Neurobiology of Bipolar Disorder
inferred← citeshttps://doi.org/10.3390/futurepharmacol4030032openalex-snapshot
Pharmacogenetics and the Blood–Brain Barrier: A Whirlwind Tour of Potential Clinical Utility
inferred← citeshttps://doi.org/10.37099/mtu.dc.etdr/436openalex-snapshot
Heterologous Expression and Purification of Full-Length Human Polybromo-1 Protein
inferred← citeshttps://doi.org/10.4018/978-1-4666-8559-8.ch009openalex-snapshot
Genomic Technologies and the Emergence of BRICS (Brazil, Russia, India, China, and South Africa)
inferred← citeshttps://doi.org/10.4018/978-1-4666-8559-8.ch012openalex-snapshot
Wealth Creation from the Commercialization of Genomics Science
inferred← citeshttps://doi.org/10.4061/2010/159761openalex-snapshot
Pharmacogenomics of Mood Stabilizers in the Treatment of Bipolar Disorder
inferred← citeshttps://doi.org/10.4236/ojpsych.2013.31008openalex-snapshot
Genomics of schizophrenia and pharmacogenomics of antipsychotic drugs
inferred← citeshttps://doi.org/10.47749/t/unicamp.2011.784055openalex-snapshot
Aplicação de modelos estatísticos e desenvolvimento de algoritmos para estudo genético de doenças neuro-psiquiátricas
inferred← citeshttps://doi.org/10.48496/9v1q-rb59openalex-snapshot
Genome-Scale Genetics: Lessons from Founder Populations
inferred← citeshttps://doi.org/10.5124/jkma.25.0143openalex-snapshot
Recent advances in the genetic architecture of bipolar disorder and its clinical implications: a narrative review
inferred← citeshttps://doi.org/10.5706/nph201212009openalex-snapshot
Beyond structural equation modeling: model properties and effect size from a Bayesian viewpoint. An example of complex phenotype-genotype associations in depression.
inferred← citeshttps://doi.org/10.6084/m9.figshare.14889673openalex-snapshot
Additional file 1 of Predicting the risk and timing of major mood disorder in offspring of bipolar parents: exploring the utility of a neural network approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26604079openalex-snapshot
Additional file 1 of Identification of a psychiatric risk gene NISCH at 3p21.1 GWAS locus mediating dendritic spine morphogenesis and cognitive function
inferred← citeshttps://doi.org/10.6084/m9.figshare.26604082openalex-snapshot
Additional file 2 of Identification of a psychiatric risk gene NISCH at 3p21.1 GWAS locus mediating dendritic spine morphogenesis and cognitive function
inferred← citeshttps://doi.org/10.7936/k7vt1qgmopenalex-snapshot
Gene Association Mapping in the Era of Next-Generation Sequencing and Systems Biology
inferred← citeshttps://doi.org/10.7936/k7wd3xkcopenalex-snapshot
New methods for discovering common and rare genetic variants in human disease
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.