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High levels of GDF15 in thalassemia suppress expression of the iron regulatory protein hepcidin
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/pbc.20616openalex-snapshot
Urinary hepcidin in congenital chronic anemias
inferredcites →https://doi.org/10.1007/s00114-004-0575-zopenalex-snapshot
Regulation of bone morphogenetic proteins in early embryonic development
inferredcites →https://doi.org/10.1016/j.beha.2004.08.020openalex-snapshot
Hepcidin—a regulator of intestinal iron absorption and iron recycling by macrophages
inferredcites →https://doi.org/10.1016/j.cmet.2005.10.010openalex-snapshot
A role of SMAD4 in iron metabolism through the positive regulation of hepcidin expression
inferredcites →https://doi.org/10.1016/j.cytogfr.2003.10.004openalex-snapshot
The transforming growth factor-β superfamily of receptors
inferredcites →https://doi.org/10.1016/j.hepres.2005.08.005openalex-snapshot
Upregulation of hepcidin by interleukin-1β in human hepatoma cell lines
inferredcites →https://doi.org/10.1016/s0167-4781(97)00122-xopenalex-snapshot
PLAB, a novel placental bone morphogenetic protein
inferredcites →https://doi.org/10.1038/ng1777openalex-snapshot
Bone morphogenetic protein signaling by hemojuvelin regulates hepcidin expression
inferredcites →https://doi.org/10.1053/plac.2002.0881openalex-snapshot
Macrophage Inhibitory Cytokine-1 in Gestational Tissues and Maternal Serum in Normal and Pre-eclamptic Pregnancy
inferredcites →https://doi.org/10.1073/pnas.0530278100openalex-snapshot
Large-scale delineation of secreted protein biomarkers overexpressed in cancer tissue and serum
inferredcites →https://doi.org/10.1073/pnas.0603124103openalex-snapshot
Bone morphogenetic proteins 2, 4, and 9 stimulate murine hepcidin 1 expression independently of Hfe, transferrin receptor 2 (Tfr2), and IL-6
inferredcites →https://doi.org/10.1097/00005792-196411000-00021openalex-snapshot
THALASSEMIA AS A PROLIFERATIVE DISORDER
inferredcites →https://doi.org/10.1111/j.1365-2141.2006.06258.xopenalex-snapshot
Downregulation of hepcidin and haemojuvelin expression in the hepatocyte cell‐line HepG2 induced by thalassaemic sera
inferredcites →https://doi.org/10.1128/.20.10.3742-3751.2000openalex-snapshot
Characterization of Growth-Differentiation Factor 15, a Transforming Growth Factor β Superfamily Member Induced following Liver Injury
inferredcites →https://doi.org/10.1136/gut.2003.031153openalex-snapshot
Changes in the expression of intestinal iron transport and hepatic regulatory molecules explain the enhanced iron absorption associated with pregnancy in the rat
inferredcites →https://doi.org/10.1146/annurev.biochem.67.1.753openalex-snapshot
TGF-β SIGNAL TRANSDUCTION
inferredcites →https://doi.org/10.1146/annurev.nutr.26.061505.111303openalex-snapshot
Regulation of Iron Metabolism by Hepcidin
inferredcites →https://doi.org/10.1152/physiol.00052.2005openalex-snapshot
The Ins and Outs of Iron Homeostasis
inferredcites →https://doi.org/10.1158/1078-0432.ccr-05-0564openalex-snapshot
Serum Markers in Patients with Resectable Pancreatic Adenocarcinoma: Macrophage Inhibitory Cytokine 1 versus CA19-9
inferredcites →https://doi.org/10.1161/01.res.0000202804.84885.d0openalex-snapshot
GDF15/MIC-1 Functions As a Protective and Antihypertrophic Factor Released From the Myocardium in Association With SMAD Protein Activation
inferredcites →https://doi.org/10.1182/blood-2004-12-4844openalex-snapshot
Hepcidin in iron overload disorders
inferredcites →https://doi.org/10.1182/blood.v96.7.2606openalex-snapshot
A correlation of erythrokinetics, ineffective erythropoiesis, and erythroid precursor apoptosis in Thai patients with thalassemia
inferredcites →https://doi.org/10.1182/blood.v99.8.3005openalex-snapshot
Fetal and adult hemoglobin production during adult erythropoiesis: coordinate expression correlates with cell proliferation
inferredcites →https://doi.org/10.5483/bmbrep.2006.39.6.649openalex-snapshot
NSAID Activated Gene (NAG-1), a Modulator of Tumorigenesis
inferredcites →https://doi.org/10.5694/j.1326-5377.1973.tb111034.xopenalex-snapshot
The Thalassæmia Syndromes.
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/9781118853771.ch6openalex-snapshot
Haemoglobin and the Inherited Disorders of Globin Synthesis
inferred← citeshttps://doi.org/10.1002/9781118925645.ch10openalex-snapshot
Cellular and Systemic Iron Homeostasis
inferred← citeshttps://doi.org/10.1002/9781118925645.ch11openalex-snapshot
Iron Deficiency, Iron Overload and Therapy
inferred← citeshttps://doi.org/10.1002/9781119251316.ch30openalex-snapshot
Hemochromatosis and Iron Storage Disorders
inferred← citeshttps://doi.org/10.1002/9781119951438.eibc2131openalex-snapshot
Iron Homeostasis: Hepcidin Regulation
inferred← citeshttps://doi.org/10.1002/9781119951438.eibc2137openalex-snapshot
Iron: Molecular Basis of Hemochromatosis
inferred← citeshttps://doi.org/10.1002/9781444323160.ch3openalex-snapshot
Iron Metabolism, Iron Deficiency and Disorders of Haem Synthesis
inferred← citeshttps://doi.org/10.1002/9781444323160.ch6openalex-snapshot
Haemoglobin and the Inherited Disorders of Globin Synthesis
inferred← citeshttps://doi.org/10.1002/ajh.22118openalex-snapshot
Erythrocyte pyruvate kinase deficiency in an old‐order Amish cohort: Longitudinal risk and disease management
inferred← citeshttps://doi.org/10.1002/ajh.23110openalex-snapshot
Targeting the hepcidin–ferroportin axis to develop new treatment strategies for anemia of chronic disease and anemia of inflammation
inferred← citeshttps://doi.org/10.1002/ajh.23405openalex-snapshot
Treating iron overload in patients with non‐transfusion‐dependent thalassemia
inferred← citeshttps://doi.org/10.1002/ajh.23406openalex-snapshot
Associations and prognostic interactions between circulating levels of hepcidin, ferritin and inflammatory cytokines in primary myelofibrosis
inferred← citeshttps://doi.org/10.1002/ajh.23649openalex-snapshot
Erythropoiesis‐driven regulation of hepcidin in human red cell disorders is better reflected through concentrations of soluble transferrin receptor rather than growth differentiation factor 15
inferred← citeshttps://doi.org/10.1002/ajh.24417openalex-snapshot
Hematopoietic deletion of transferrin receptor 2 in mice leads to a block in erythroid differentiation during iron‐deficient anemia
inferred← citeshttps://doi.org/10.1002/ajh.24530openalex-snapshot
2017 Clinical trials update in new treatments of β‐thalassemia
inferred← citeshttps://doi.org/10.1002/ajh.25909openalex-snapshot
Gene therapies for transfusion dependent β‐thalassemia: Current status and critical criteria for success
inferred← citeshttps://doi.org/10.1002/ajh.26152openalex-snapshot
Marginally reduced maternal hepatic and splenic ferroportin under severe nutritional iron deficiency in pregnancy maintains systemic iron supply
inferred← citeshttps://doi.org/10.1002/ajh.26905openalex-snapshot
Inflammatory and senescence‐associated mediators affect the persistence of humoral response to COVID ‐19 mRNA vaccination in transfusion‐dependent beta‐thalassemic patients
inferred← citeshttps://doi.org/10.1002/biof.1275openalex-snapshot
Liver iron is a major regulator of hepcidin gene expression via BMP/SMAD pathway in a rat model of chronic renal failure under treatment with high r H u EPO doses
inferred← citeshttps://doi.org/10.1002/cam4.502openalex-snapshot
The role of growth differentiation factor 15 in the pathogenesis of primary myelofibrosis
inferred← citeshttps://doi.org/10.1002/cam4.974openalex-snapshot
High pretransplant hepcidin levels are associated with poor overall survival and delayed platelet engraftment after allogeneic hematopoietic stem cell transplantation
inferred← citeshttps://doi.org/10.1002/cbf.3238openalex-snapshot
Strain‐ and time‐dependent alterations in hepatic iron metabolism in a murine model of nonalcoholic steatohepatitis
inferred← citeshttps://doi.org/10.1002/cncr.31550openalex-snapshot
Iron overload in patients with myelodysplastic syndromes: An updated overview
inferred← citeshttps://doi.org/10.1002/hem3.78openalex-snapshot
Interplay between α‐thalassemia and β‐hemoglobinopathies: Translating genotype–phenotype relationships into therapies
inferred← citeshttps://doi.org/10.1002/hep.22507openalex-snapshot
Hereditary hemochromatosis in the post- HFE era
inferred← citeshttps://doi.org/10.1002/hep.26550openalex-snapshot
Liver iron modulates hepcidin expression during chronically elevated erythropoiesis in mice
inferred← citeshttps://doi.org/10.1002/hep.29499openalex-snapshot
Cystathionine β‐synthase is required for body iron homeostasis
inferred← citeshttps://doi.org/10.1002/iub.1629openalex-snapshot
Systemic iron homeostasis and erythropoiesis
inferred← citeshttps://doi.org/10.1002/j.2040-4603.2013.tb00494.xopenalex-snapshot
Iron Homeostasis in the Liver
inferred← citeshttps://doi.org/10.1002/jcb.26715openalex-snapshot
The α1‐adrenergic receptor is involved in hepcidin upregulation induced by adrenaline and norepinephrine via the STAT3 pathway
inferred← citeshttps://doi.org/10.1002/jcp.22196openalex-snapshot
Divergent molecular mechanisms underlying the pleiotropic functions of macrophage inhibitory cytokine‐1 in cancer
inferred← citeshttps://doi.org/10.1002/jor.25323openalex-snapshot
The role of disrupted iron homeostasis in the development and progression of arthropathy
inferred← citeshttps://doi.org/10.1002/pbc.24373openalex-snapshot
GDF15‐mediated upregulation of ferroportin plays a key role in the development of hyperferritinemia in children with hemophagocytic lymphohistiocytosis
inferred← citeshttps://doi.org/10.1002/prca.201600086openalex-snapshot
Analysis of protein profiling studies of β‐thalassemia/Hb E disease
inferred← citeshttps://doi.org/10.1007/978-1-59745-462-9_1openalex-snapshot
The Cellular Physiology of Iron
inferred← citeshttps://doi.org/10.1007/978-1-59745-462-9_2openalex-snapshot
Regulation of Iron Absorption and Distribution
inferred← citeshttps://doi.org/10.1007/978-1-59745-462-9_3openalex-snapshot
The Role of Hepcidin in Iron Homeostasis
inferred← citeshttps://doi.org/10.1007/978-1-60327-485-2_1openalex-snapshot
Proteins of Iron Homeostasis
inferred← citeshttps://doi.org/10.1007/978-1-60327-485-2_11openalex-snapshot
Iron and the Reticuloendothelial System
inferred← citeshttps://doi.org/10.1007/978-1-60327-485-2_16openalex-snapshot
Disorders of Red Cell Production and the Iron-Loading Anemias
inferred← citeshttps://doi.org/10.1007/978-1-60327-485-2_28openalex-snapshot
Clinical Use of Iron Chelators
inferred← citeshttps://doi.org/10.1007/978-1-60327-485-2_9openalex-snapshot
Molecular Regulation of Systemic Iron Metabolism
inferred← citeshttps://doi.org/10.1007/978-1-60761-857-7_23openalex-snapshot
Oxygen-Dependent Regulation of Erythropoiesis
inferred← citeshttps://doi.org/10.1007/978-3-0348-0620-6_54-1openalex-snapshot
TGF-b Superfamily Cytokine MIC-1/GDF15 in Health and Inflammatory Diseases
inferred← citeshttps://doi.org/10.1007/978-3-0348-0620-6_54-2openalex-snapshot
TGF beta Superfamily Cytokine MIC-1/GDF15 in Health and Inflammatory Diseases
inferred← citeshttps://doi.org/10.1007/978-3-642-18081-1_7openalex-snapshot
Iron Deficiency in Critically III Patients: Highlighting the Role of Hepcidin
inferred← citeshttps://doi.org/10.1007/978-3-7643-8550-7_54openalex-snapshot
TGF beta Superfamily Cytokine MIC-1/GDF15 in Health and Inflammatory Diseases
inferred← citeshttps://doi.org/10.1007/978-94-007-0440-4_11openalex-snapshot
Iron and Copper Metabolism in the Myelodysplastic Syndromes
inferred← citeshttps://doi.org/10.1007/978-94-007-0440-4_12openalex-snapshot
Pathogenesis and Management of Iron Overload in MDS
inferred← citeshttps://doi.org/10.1007/978-981-13-3672-0_4openalex-snapshot
Recapitulating Hematopoietic Development in a Dish
inferred← citeshttps://doi.org/10.1007/978-981-96-1305-2_11openalex-snapshot
The Fight for Iron: A Central Theme in Host-Pathogen Interactions
inferred← citeshttps://doi.org/10.1007/s00005-010-0095-9openalex-snapshot
Iron and Immunity: Immunological Consequences of Iron Deficiency and Overload
inferred← citeshttps://doi.org/10.1007/s00109-011-0751-5openalex-snapshot
Growth differentiation factor 15 in patients with congenital dyserythropoietic anaemia (CDA) type II
inferred← citeshttps://doi.org/10.1007/s00216-011-5318-3openalex-snapshot
Functionalized gold nanoparticles for ultrasensitive DNA detection
inferred← citeshttps://doi.org/10.1007/s00277-009-0879-3openalex-snapshot
Absence of cardiac siderosis despite hepatic iron overload in Italian patients with thalassemia intermedia: an MRI T2* study
inferred← citeshttps://doi.org/10.1007/s00277-010-1091-1openalex-snapshot
Iron overload in MDS—pathophysiology, diagnosis, and complications
inferred← citeshttps://doi.org/10.1007/s00277-011-1385-yopenalex-snapshot
A useful relationship between the presence of extramedullary erythropoeisis and the level of the soluble form of the transferrin receptor in a large cohort of adult patients with thalassemia intermedia: a prospective study
inferred← citeshttps://doi.org/10.1007/s00277-014-2113-1openalex-snapshot
Association of growth differentiation factor 15 (GDF15) polymorphisms with serum GDF15 and ferritin levels in β-thalassemia
inferred← citeshttps://doi.org/10.1007/s00277-020-03931-9openalex-snapshot
Evaluation of regulatory T cells frequency and FoxP3/GDF-15 gene expression in β-thalassemia major patients with and without alloantibody; correlation with serum ferritin and folate levels
inferred← citeshttps://doi.org/10.1007/s00277-021-04697-4openalex-snapshot
Pregnancy outcome in women with transfused beta-thalassemia in France
inferred← citeshttps://doi.org/10.1007/s00277-026-06774-yopenalex-snapshot
Diagnostic reassessment in myeloproliferative neoplasms: the value of functional iron parameters and JAK2 allelic burden
inferred← citeshttps://doi.org/10.1007/s00392-021-01944-6openalex-snapshot
Growth differentiation factor 15 predicts poor prognosis in patients with heart failure and reduced ejection fraction and anemia: results from RED-HF
inferred← citeshttps://doi.org/10.1007/s00404-023-07032-6openalex-snapshot
Molecular insights into placental iron transfer mechanisms and maternofetal regulation
inferred← citeshttps://doi.org/10.1007/s00421-011-2097-7openalex-snapshot
Early effects of erythropoietin on serum hepcidin and serum iron bioavailability in healthy volunteers
inferred← citeshttps://doi.org/10.1007/s00421-014-2829-6openalex-snapshot
Influence of post-exercise hypoxic exposure on hepcidin response in athletes
inferred← citeshttps://doi.org/10.1007/s00421-014-2938-2openalex-snapshot
Acute hypoxic exercise does not alter post-exercise iron metabolism in moderately trained endurance athletes
inferred← citeshttps://doi.org/10.1007/s00439-010-0852-1openalex-snapshot
Factors influencing disease phenotype and penetrance in HFE haemochromatosis
inferred← citeshttps://doi.org/10.1007/s10354-016-0505-7openalex-snapshot
Iron deficiency or anemia of inflammation?
inferred← citeshttps://doi.org/10.1007/s10534-008-9187-yopenalex-snapshot
Interacting signals in the control of hepcidin expression
inferred← citeshttps://doi.org/10.1007/s10534-012-9520-3openalex-snapshot
Known and potential roles of transferrin in iron biology
inferred← citeshttps://doi.org/10.1007/s10534-012-9586-yopenalex-snapshot
Human apo-lactoferrin as a physiological mimetic of hypoxia stabilizes hypoxia-inducible factor-1 alpha
inferred← citeshttps://doi.org/10.1007/s10534-015-9833-0openalex-snapshot
The use of hypotransferrinemic mice in studies of iron biology
inferred← citeshttps://doi.org/10.1007/s10545-017-0017-7openalex-snapshot
Disturbed iron metabolism in erythropoietic protoporphyria and association of GDF15 and gender with disease severity
inferred← citeshttps://doi.org/10.1007/s10555-011-9301-4openalex-snapshot
COX inhibitors directly alter gene expression: role in cancer prevention?
inferred← citeshttps://doi.org/10.1007/s10753-023-01872-9openalex-snapshot
Low Serum Hepcidin Levels in Patients with Ulcerative Colitis – Implications for Treatment of Co-existent Iron-Deficiency Anemia
inferred← citeshttps://doi.org/10.1007/s11239-014-1073-6openalex-snapshot
Management of pulmonary arterial hypertension associated to thalassemia: when pulmonary endarterectomy is the best therapeutical option? A case report
inferred← citeshttps://doi.org/10.1007/s11255-014-0696-zopenalex-snapshot
Effects of additional iron doses on hepcidin-25 level in hemodialysis patients without evident iron deficiency
inferred← citeshttps://doi.org/10.1007/s11255-016-1278-zopenalex-snapshot
GDF-15, iron, and inflammation in early chronic kidney disease among elderly patients
inferred← citeshttps://doi.org/10.1007/s11739-010-0387-4openalex-snapshot
Recent advances in iron metabolism and related disorders
inferred← citeshttps://doi.org/10.1007/s11834-010-0139-9openalex-snapshot
Érythropoïèse et métabolisme du fer: interactions et applications biomédicales
inferred← citeshttps://doi.org/10.1007/s11899-011-0084-zopenalex-snapshot
The Relevance of Iron Overload and the Appropriateness of Iron Chelation Therapy for Patients with Myelodysplastic Syndromes: A Dialogue and Debate
inferred← citeshttps://doi.org/10.1007/s12072-009-9144-zopenalex-snapshot
The global burden of iron overload
inferred← citeshttps://doi.org/10.1007/s12185-010-0743-1openalex-snapshot
Iron and erythropoiesis: a dual relationship
inferred← citeshttps://doi.org/10.1007/s12185-011-0875-yopenalex-snapshot
Need for early recognition and therapeutic guidelines of congenital sideroblastic anaemia
inferred← citeshttps://doi.org/10.1007/s12185-012-1217-4openalex-snapshot
Erythropoietin stimulation decreases hepcidin expression through hematopoietic activity on bone marrow cells in mice
inferred← citeshttps://doi.org/10.1007/s12185-012-1223-6openalex-snapshot
Acidic milieu augments the expression of hepcidin, the central regulator of iron homeostasis
inferred← citeshttps://doi.org/10.1007/s12185-013-1476-8openalex-snapshot
Hepcidin production in response to iron is controlled by monocyte-derived humoral factors
inferred← citeshttps://doi.org/10.1007/s12185-014-1626-7openalex-snapshot
Hepcidin and GDF15 in anemia of multiple myeloma
inferred← citeshttps://doi.org/10.1007/s12185-017-2231-3openalex-snapshot
Hepcidin suppression in β-thalassemia is associated with the down-regulation of atonal homolog 8
inferred← citeshttps://doi.org/10.1007/s12185-017-2365-3openalex-snapshot
The mechanisms of systemic iron homeostasis and etiology, diagnosis, and treatment of hereditary hemochromatosis
inferred← citeshttps://doi.org/10.1007/s12185-017-2368-0openalex-snapshot
Iron metabolism in erythroid cells and patients with congenital sideroblastic anemia
inferred← citeshttps://doi.org/10.1007/s12185-018-2411-9openalex-snapshot
β-Thalassemia intermedia: a comprehensive overview and novel approaches
inferred← citeshttps://doi.org/10.1007/s12185-025-04035-8openalex-snapshot
Growth differentiation factor-15 as a non-invasive biomarker of liver fibrosis in sickle cell disease
inferred← citeshttps://doi.org/10.1007/s12254-010-0179-6openalex-snapshot
Iron chelation therapy in myelodysplastic syndromes: evidence-based rationale and comparison of guidelines
inferred← citeshttps://doi.org/10.1007/s12288-015-0551-0openalex-snapshot
Can Serum Gdf-15 be Associated with Functional Iron Deficiency in Hemodialysis Patients?
inferred← citeshttps://doi.org/10.1007/s12288-020-01315-7openalex-snapshot
Prognostic Impact of Serum Growth Differentiation Factor 15 Level in Acute Myeloid Leukemia Patients
inferred← citeshttps://doi.org/10.1007/s12288-022-01533-1openalex-snapshot
The Effect of Long-Term Iron Chelator Therapy on Serum Levels of Hepcidin and Ferritin in Patients with Thalassemia Major and Intermediate
inferred← citeshttps://doi.org/10.1007/s12308-019-00363-xopenalex-snapshot
Association between serum levels of hepcidin and ferritin in patients with thalassemia major and intermedia, the role of iron chelator
inferred← citeshttps://doi.org/10.1007/s40265-020-01341-9openalex-snapshot
Beta Thalassemia: New Therapeutic Options Beyond Transfusion and Iron Chelation
inferred← citeshttps://doi.org/10.1007/s40618-025-02636-yopenalex-snapshot
GDF15: An emerging disease target and biomarker of metabolic diseases
inferred← citeshttps://doi.org/10.1007/s40620-021-01020-2openalex-snapshot
Urinary Growth Differentiation Factor-15 (GDF15) levels as a biomarker of adverse outcomes and biopsy findings in chronic kidney disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-373870-7.00002-8openalex-snapshot
Erythropoiesis: The Roles of Erythropoietin and Iron
inferred← citeshttps://doi.org/10.1016/b978-0-12-382026-6.00071-3openalex-snapshot
Molecular Mechanisms of Intestinal Iron Transport
inferred← citeshttps://doi.org/10.1016/b978-0-12-407681-5.00003-9openalex-snapshot
Protein Antioxidants in Thalassemia
inferred← citeshttps://doi.org/10.1016/b978-0-12-800773-0.00028-8openalex-snapshot
Molecular Effects of Alcohol on Iron Metabolism
inferred← citeshttps://doi.org/10.1016/b978-0-12-801238-3.11557-0openalex-snapshot
Crosstalk Between Iron and Bone Metabolism: Evidence from Patients and Mouse Models of β-Thalassemia
inferred← citeshttps://doi.org/10.1016/b978-0-12-801816-3.00037-6openalex-snapshot
Iron Metabolism in Aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-802168-2.00015-4openalex-snapshot
Hepcidin and the Hormonal Control of Iron Homeostasis
inferred← citeshttps://doi.org/10.1016/b978-0-12-809712-0.00004-6openalex-snapshot
Alphavirus Nonstructural Proteases and Their Inhibitors
inferred← citeshttps://doi.org/10.1016/b978-0-12-809954-4.00060-8openalex-snapshot
Mechanisms and Regulation of Intestinal Iron Transport
inferred← citeshttps://doi.org/10.1016/b978-0-323-35762-3.00040-8openalex-snapshot
Thalassemia Syndromes
inferred← citeshttps://doi.org/10.1016/b978-0-7020-3147-2.00011-0openalex-snapshot
Iron deficiency anemia, anemia of chronic disorders and iron overload
inferred← citeshttps://doi.org/10.1016/b978-0-7020-3398-8.00005-2openalex-snapshot
Disorders of iron overload
inferred← citeshttps://doi.org/10.1016/b978-0-7020-3408-4.00009-6openalex-snapshot
Iron deficiency anaemia and iron overload
inferred← citeshttps://doi.org/10.1016/b978-0-7020-6696-2.00009-6openalex-snapshot
Iron Deficiency Anaemia and Iron Overload
inferred← citeshttps://doi.org/10.1016/b978-0-7020-6697-9.00004-2openalex-snapshot
Disorders of Iron Overload
inferred← citeshttps://doi.org/10.1016/b978-0-7020-8228-3.00004-1openalex-snapshot
Disorders of Iron Overload
inferred← citeshttps://doi.org/10.1016/b978-1-4160-6164-9.00032-9openalex-snapshot
Hemoglobin, Iron, and Bilirubin
inferred← citeshttps://doi.org/10.1016/b978-1-4160-6193-9.10056-9openalex-snapshot
Hematologic Aspects of Kidney Disease
inferred← citeshttps://doi.org/10.1016/b978-1-4557-5414-4.00011-5openalex-snapshot
10.1016/b978-1-4557-5414-4.00011-5
inferred← citeshttps://doi.org/10.1016/bs.vh.2017.01.001openalex-snapshot
Mutual Cross Talk Between Iron Homeostasis and Erythropoiesis
inferred← citeshttps://doi.org/10.1016/bs.vh.2019.01.002openalex-snapshot
Hepcidin-ferroportin axis in health and disease
inferred← citeshttps://doi.org/10.1016/bs.vh.2019.01.003openalex-snapshot
Signaling pathways regulating hepcidin
inferred← citeshttps://doi.org/10.1016/bs.vh.2019.01.004openalex-snapshot
Hepcidin and the BMP-SMAD pathway: An unexpected liaison
inferred← citeshttps://doi.org/10.1016/bs.vh.2019.01.005openalex-snapshot
Regulators of hepcidin expression
inferred← citeshttps://doi.org/10.1016/bs.vh.2019.01.008openalex-snapshot
The role of heparin, heparanase and heparan sulfates in hepcidin regulation
inferred← citeshttps://doi.org/10.1016/bs.vh.2019.01.012openalex-snapshot
Hepcidin in chronic kidney disease anemia
inferred← citeshttps://doi.org/10.1016/bs.vh.2023.03.001openalex-snapshot
Hepcidin and its multiple partners: Complex regulation of iron metabolism in health and disease
inferred← citeshttps://doi.org/10.1016/bs.vh.2024.10.003openalex-snapshot
Hepcidin: A multifaceted hormone in iron homeostasis and tumor biology
inferred← citeshttps://doi.org/10.1016/j.abb.2025.110724openalex-snapshot
NAG-1/GDF15 modulates hepcidin expression through STAT3 and SMAD pathways
inferred← citeshttps://doi.org/10.1016/j.advms.2024.02.003openalex-snapshot
The association between growth differentiation factor 15 and presence and severity of coronary atherosclerosis
inferred← citeshttps://doi.org/10.1016/j.amjms.2023.05.002openalex-snapshot
The association of growth differentiation factor-15 levels and osteoporosis in patients with thalassemia
inferred← citeshttps://doi.org/10.1016/j.ando.2024.101681openalex-snapshot
Key data from the 2024 European Thyroid Association annual meeting: “Thyroid and pregnancy”
inferred← citeshttps://doi.org/10.1016/j.apjtm.2016.07.035openalex-snapshot
Iron dysregulation in beta-thalassemia
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.03.011openalex-snapshot
An overview of molecular basis of iron metabolism regulation and the associated pathologies
inferred← citeshttps://doi.org/10.1016/j.bbadis.2020.165798openalex-snapshot
Functional roles of GDF15 in modulating microenvironment to promote carcinogenesis
inferred← citeshttps://doi.org/10.1016/j.bbagen.2008.08.006openalex-snapshot
Iron metabolism in the anemia of chronic disease
inferred← citeshttps://doi.org/10.1016/j.bbagen.2010.08.005openalex-snapshot
Hepcidin expression by human monocytes in response to adhesion and pro-inflammatory cytokines
inferred← citeshttps://doi.org/10.1016/j.bbagen.2011.07.015openalex-snapshot
Hereditary hemochromatosis and transferrin receptor 2
inferred← citeshttps://doi.org/10.1016/j.bbagen.2011.08.002openalex-snapshot
The long history of iron in the Universe and in health and disease
inferred← citeshttps://doi.org/10.1016/j.bbagen.2011.10.013openalex-snapshot
Regulation of iron transport and the role of transferrin
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2012.01.014openalex-snapshot
Hepcidin and iron homeostasis
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2024.119800openalex-snapshot
Erythropoietin enhances iron bioavailability in HepG2 cells by downregulating hepcidin through mTOR, C/EBPα and HIF-1α
inferred← citeshttps://doi.org/10.1016/j.bbrc.2020.03.079openalex-snapshot
GDF15 knockdown promotes erastin-induced ferroptosis by decreasing SLC7A11 expression
inferred← citeshttps://doi.org/10.1016/j.bcmd.2007.10.002openalex-snapshot
Regulation of hepcidin: Insights from biochemical analyses on human serum samples
inferred← citeshttps://doi.org/10.1016/j.bcmd.2010.02.012openalex-snapshot
Effect of erythropoietin on hepcidin expression in hemojuvelin-mutant mice
inferred← citeshttps://doi.org/10.1016/j.bcmd.2011.07.005openalex-snapshot
Levels of growth differentiation factor-15 are high and correlate with clinical severity in transfusion-independent patients with β thalassemia intermedia
inferred← citeshttps://doi.org/10.1016/j.bcmd.2012.02.001openalex-snapshot
Hepcidin is correlated to soluble hemojuvelin but not to increased GDF15 during pregnancy
inferred← citeshttps://doi.org/10.1016/j.bcmd.2014.06.006openalex-snapshot
Circulating factors are involved in hypoxia-induced hepcidin suppression
inferred← citeshttps://doi.org/10.1016/j.bcmd.2014.07.003openalex-snapshot
Growth differentiation factor-15 in young sickle cell disease patients: Relation to hemolysis, iron overload and vascular complications
inferred← citeshttps://doi.org/10.1016/j.bcmd.2015.06.001openalex-snapshot
Growth differentiation factor-15 in children and adolescents with thalassemia intermedia: Relation to subclinical atherosclerosis and pulmonary vasculopathy
inferred← citeshttps://doi.org/10.1016/j.bcmd.2015.07.010openalex-snapshot
Erythropoietic drive is the strongest predictor of hepcidin level in adults with sickle cell disease
inferred← citeshttps://doi.org/10.1016/j.bcmd.2016.02.005openalex-snapshot
EPO-dependent induction of erythroferrone drives hepcidin suppression and systematic iron absorption under phenylhydrazine-induced hemolytic anemia
inferred← citeshttps://doi.org/10.1016/j.bcmd.2016.04.008openalex-snapshot
Recombinant human erythropoietin-induced erythropoiesis regulates hepcidin expression over iron status in the rat
inferred← citeshttps://doi.org/10.1016/j.bcmd.2019.04.011openalex-snapshot
The Growth Differentiation Factor-15 (GDF-15) levels are increased in patients with compound heterozygous sickle cell and beta-thalassemia (HbS/βthal), correlate with markers of hemolysis, iron burden, coagulation, endothelial dysfunction and pulmonary hypertension
inferred← citeshttps://doi.org/10.1016/j.bcmd.2019.04.014openalex-snapshot
EPO-R+ myelodysplastic cells with ring sideroblasts produce high erythroferrone levels to reduce hepcidin expression in hepatic cells
inferred← citeshttps://doi.org/10.1016/j.bcmd.2023.102727openalex-snapshot
Impact of maternal iron deficiency anemia on fetal iron status and placental iron transporters in human pregnancy
inferred← citeshttps://doi.org/10.1016/j.beha.2013.09.009openalex-snapshot
When is iron overload deleterious, and when and how should iron chelation therapy be administered in myelodysplastic syndromes?
inferred← citeshttps://doi.org/10.1016/j.berh.2011.10.014openalex-snapshot
Haemochromatosis: The bone and the joint
inferred← citeshttps://doi.org/10.1016/j.biochi.2009.06.012openalex-snapshot
Hepcidin, the iron watcher
inferred← citeshttps://doi.org/10.1016/j.biopha.2021.112548openalex-snapshot
Impaired bone marrow microenvironment and stem cells in transfusion-dependent beta-thalassemia
inferred← citeshttps://doi.org/10.1016/j.blre.2008.03.001openalex-snapshot
Current approach to hemochromatosis
inferred← citeshttps://doi.org/10.1016/j.blre.2012.12.003openalex-snapshot
Regulation of erythropoiesis by hypoxia-inducible factors
inferred← citeshttps://doi.org/10.1016/j.blre.2017.10.001openalex-snapshot
What can we learn from ineffective erythropoiesis in thalassemia?
inferred← citeshttps://doi.org/10.1016/j.blre.2019.100617openalex-snapshot
Too much iron: A masked foe for leukemias
inferred← citeshttps://doi.org/10.1016/j.blre.2021.100830openalex-snapshot
Tackling the unknowns in understanding and management of hospital acquired anemia
inferred← citeshttps://doi.org/10.1016/j.bone.2018.10.005openalex-snapshot
Lowering iron level protects against bone loss in focally irradiated and contralateral femurs through distinct mechanisms
inferred← citeshttps://doi.org/10.1016/j.bone.2020.115495openalex-snapshot
Bone morphogenic proteins in iron homeostasis
inferred← citeshttps://doi.org/10.1016/j.bpa.2012.11.002openalex-snapshot
Pathophysiology of perioperative anaemia
inferred← citeshttps://doi.org/10.1016/j.cca.2013.10.015openalex-snapshot
Regulation of hepcidin through GDF-15 in cancer-related anemia
inferred← citeshttps://doi.org/10.1016/j.cca.2021.10.032openalex-snapshot
Iron disorders and hepcidin
inferred← citeshttps://doi.org/10.1016/j.cca.2026.121179openalex-snapshot
Erythroferrone in β-thalassemia: Systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1016/j.cell.2010.06.028openalex-snapshot
Two to Tango: Regulation of Mammalian Iron Metabolism
inferred← citeshttps://doi.org/10.1016/j.cell.2016.12.034openalex-snapshot
A Red Carpet for Iron Metabolism
inferred← citeshttps://doi.org/10.1016/j.cell.2019.07.033openalex-snapshot
GDF15 Is an Inflammation-Induced Central Mediator of Tissue Tolerance
inferred← citeshttps://doi.org/10.1016/j.cell.2024.10.045openalex-snapshot
Atlas of the plasma proteome in health and disease in 53,026 adults
inferred← citeshttps://doi.org/10.1016/j.clgc.2026.102683openalex-snapshot
Growth-Differentiation-Factor 15 (GDF15) and Hepcidin as Prognostic Biomarkers for Abiraterone-Treated Metastatic Castration-Resistant Prostate Cancer
inferred← citeshttps://doi.org/10.1016/j.clinre.2013.11.003openalex-snapshot
Non-HFE hemochromatosis: Pathophysiological and diagnostic aspects
inferred← citeshttps://doi.org/10.1016/j.clml.2011.04.003openalex-snapshot
Allogeneic Stem Cell Transplantation for Myelodysplastic Syndromes: Critical for Cure?
inferred← citeshttps://doi.org/10.1016/j.cmet.2008.03.008openalex-snapshot
Iron Homeostasis: Fitting the Puzzle Pieces Together
inferred← citeshttps://doi.org/10.1016/j.cnd.2012.01.001openalex-snapshot
Fer et inflammation, mariage impossible ?
inferred← citeshttps://doi.org/10.1016/j.crbiot.2020.10.001openalex-snapshot
Comparative analysis of high altitude hypoxia induced erythropoiesis and iron homeostasis in Indian and Kyrgyz lowlander males
inferred← citeshttps://doi.org/10.1016/j.critrevonc.2014.01.006openalex-snapshot
Iron overload and chelation therapy in myelodysplastic syndromes
inferred← citeshttps://doi.org/10.1016/j.critrevonc.2015.02.006openalex-snapshot
Update on iron metabolism and molecular perspective of common genetic and acquired disorder, hemochromatosis
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