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Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory
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1 of the 31 references listed on this page is in the Retraction Watch database as retracted (export 2026-11-04): 10.1038/nature04533 (notice 2024-06-24).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/1531-8249(199912)46:6<860::aid-ana8>3.0.co;2-mopenalex-snapshot
Soluble pool of A? amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease
inferredcites →https://doi.org/10.1002/ana.410300410openalex-snapshot
Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment
inferredcites →https://doi.org/10.1016/0896-6273(92)90248-copenalex-snapshot
Mechanisms underlying induction of homosynaptic long-term depression in area CA1 of the hippocampus
inferredcites →https://doi.org/10.1016/j.neuron.2006.10.035openalex-snapshot
AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss
inferredcites →https://doi.org/10.1016/s0002-9440(10)65184-xopenalex-snapshot
Soluble Amyloid β Peptide Concentration as a Predictor of Synaptic Change in Alzheimer's Disease
inferredcites →https://doi.org/10.1016/s0301-0082(01)00013-2openalex-snapshot
Long-term depression: a cascade of induction and expression mechanisms
inferredcites →https://doi.org/10.1021/bi001048sopenalex-snapshot
The Oligomerization of Amyloid β-Protein Begins Intracellularly in Cells Derived from Human Brain
inferredcites →https://doi.org/10.1038/35050116openalex-snapshot
Aβ peptide vaccination prevents memory loss in an animal model of Alzheimer's disease
inferredcites →https://doi.org/10.1038/416535aopenalex-snapshot
Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo
inferredcites →https://doi.org/10.1038/nature04533openalex-snapshot
RETRACTED ARTICLE: A specific amyloid-β protein assembly in the brain impairs memory
inferredcites →https://doi.org/10.1038/nn1372openalex-snapshot
Natural oligomers of the amyloid-β protein specifically disrupt cognitive function
inferredcites →https://doi.org/10.1038/nn1566openalex-snapshot
Regulation of neuronal morphology and function by the tumor suppressors Tsc1 and Tsc2
inferredcites →https://doi.org/10.1038/sj.emboj.7601953openalex-snapshot
Lipids revert inert Aβ amyloid fibrils to neurotoxic protofibrils that affect learning in mice
inferredcites →https://doi.org/10.1046/j.1471-4159.1995.65062796.xopenalex-snapshot
Memory Consolidation Induces a Transient and Time‐Dependent Increase in the Frequency of Neural Cell Adhesion Molecule Polysialylated Cells in the Adult Rat Hippocampus
inferredcites →https://doi.org/10.1073/pnas.82.12.4245openalex-snapshot
Amyloid plaque core protein in Alzheimer disease and Down syndrome.
inferredcites →https://doi.org/10.1073/pnas.89.10.4363openalex-snapshot
Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade.
inferredcites →https://doi.org/10.1073/pnas.91.25.12243openalex-snapshot
Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.
inferredcites →https://doi.org/10.1073/pnas.95.11.6448openalex-snapshot
Diffusible, nonfibrillar ligands derived from Aβ 1–42 are potent central nervous system neurotoxins
inferredcites →https://doi.org/10.1074/jbc.271.8.4077openalex-snapshot
Water-soluble Aβ(N-40, N-42) Oligomers in Normal and Alzheimer Disease Brains
inferredcites →https://doi.org/10.1111/j.1471-4159.1986.tb08501.xopenalex-snapshot
Isolation of Low‐Molecular‐Weight Proteins from Amyloid Plaque Fibers in Alzheimer's Disease
inferredcites →https://doi.org/10.1111/j.1471-4159.1993.tb09834.xopenalex-snapshot
Morphological and Biochemical Analyses of Amyloid Plaque Core Proteins Purified from Alzheimer Disease Brain Tissue
inferredcites →https://doi.org/10.1111/j.1471-4159.2006.04418.xopenalex-snapshot
Temporal change in gene expression in the rat dentate gyrus following passive avoidance learning
inferredcites →https://doi.org/10.1152/jn.00939.2004openalex-snapshot
β-Amyloid Peptides Impair PKC-Dependent Functions of Metabotropic Glutamate Receptors in Prefrontal Cortical Neurons
inferredcites →https://doi.org/10.1159/000337230openalex-snapshot
Amyloid Oligomers Exacerbate Tau Pathology in a Mouse Model of Tauopathy
inferredcites →https://doi.org/10.1523/jneurosci.14-09-05325.1994openalex-snapshot
Changes in paired-pulse facilitation suggest presynaptic involvement in long-term potentiation
inferredcites →https://doi.org/10.1523/jneurosci.1879-05.2005openalex-snapshot
Dendritic Spine Abnormalities in Amyloid Precursor Protein Transgenic Mice Demonstrated by Gene Transfer and Intravital Multiphoton Microscopy
inferredcites →https://doi.org/10.1523/jneurosci.20-11-04050.2000openalex-snapshot
High-Level Neuronal Expression of Aβ1–42in Wild-Type Human Amyloid Protein Precursor Transgenic Mice: Synaptotoxicity without Plaque Formation
inferredcites →https://doi.org/10.1523/jneurosci.3501-06.2007openalex-snapshot
Aβ Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease
inferredcites →https://doi.org/10.1523/jneurosci.4391-04.2005openalex-snapshot
Certain Inhibitors of Synthetic Amyloid β-Peptide (Aβ) Fibrillogenesis Block Oligomerization of Natural Aβ and Thereby Rescue Long-Term Potentiation
inferredcites →https://doi.org/10.1523/jneurosci.4970-06.2007openalex-snapshot
Natural Oligomers of the Alzheimer Amyloid-β Protein Induce Reversible Synapse Loss by Modulating an NMDA-Type Glutamate Receptor-Dependent Signaling Pathway
inferredcites →https://doi.org/10.1523/jneurosci.5161-07.2008openalex-snapshot
Amyloid β Protein Dimer-Containing Human CSF Disrupts Synaptic Plasticity: Prevention by Systemic Passive Immunization
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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inferred← citeshttps://doi.org/10.5281/zenodo.8515openalex-extracted
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inferred← citeshttps://doi.org/10.1001/archneurol.2008.565openalex-snapshot
A Specific Enzyme-Linked Immunosorbent Assay for Measuring β-Amyloid Protein Oligomers in Human Plasma and Brain Tissue of Patients With Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneurol.2009.223openalex-snapshot
Cellular Prion Protein Mediates the Toxicity of β-Amyloid Oligomers
inferred← citeshttps://doi.org/10.1001/archneurol.2009.279openalex-snapshot
Absence of Pittsburgh Compound B Detection of Cerebral Amyloid β in a Patient With Clinical, Cognitive, and Cerebrospinal Fluid Markers of Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneurol.2012.90openalex-snapshot
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inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.0834openalex-snapshot
Association of Cortical β-Amyloid Protein in the Absence of Insoluble Deposits With Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.2050openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/14651858.cd008782openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/14651858.cd008782.pub2openalex-snapshot
Plasma and Cerebro Spinal Fluid Amyloid Beta 42, 40 and the 42:40 ratio for diagnosis of incipient Dementia in a population with objective cognitive decline.
inferred← citeshttps://doi.org/10.1002/14651858.cd008782.pub3openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/14651858.cd008782.pub4openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/1873-3468.12633openalex-snapshot
Structure‐dependent effects of amyloid‐β on long‐term memory in Lymnaea stagnalis
inferred← citeshttps://doi.org/10.1002/1873-3468.13919openalex-snapshot
Internalisation and toxicity of amyloid‐β 1‐42 are influenced by its conformation and assembly state rather than size
inferred← citeshttps://doi.org/10.1002/2211-5463.12056openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/9780470572702.ch24openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/9783527654185.ch13openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/adma.202314354openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/advs.202523791openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/agt2.333openalex-snapshot
Protein amyloid aggregate: Structure and function
inferred← citeshttps://doi.org/10.1002/agt2.463openalex-snapshot
Light‐controlled molecular tweezers capture specific amyloid oligomers
inferred← citeshttps://doi.org/10.1002/alz.12302openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/alz.12345openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/alz.12457openalex-snapshot
An ultra‐sensitive immunoassay detects and quantifies soluble Aβ oligomers in human plasma
inferred← citeshttps://doi.org/10.1002/alz.13720openalex-snapshot
Tracing synaptic loss in Alzheimer's brain with SV2A PET‐tracer UCB‐J
inferred← citeshttps://doi.org/10.1002/alz.14342openalex-snapshot
The case for regulatory approval of amyloid‐lowering immunotherapies in Alzheimer's disease based on clearcut biomarker evidence
inferred← citeshttps://doi.org/10.1002/alz.14385openalex-snapshot
Elevated locus coeruleus metabolism provides resilience against cognitive decline in preclinical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70194openalex-snapshot
Inhibition of hippocampal mossy fiber plasticity and episodic memory by human Aβ oligomers is prevented by enhancing cAMP signaling in Alzheimer's mice
inferred← citeshttps://doi.org/10.1002/alz.70273openalex-snapshot
Experimental modeling of Alzheimer's disease: Translational lessons from cross‐taxon analyses
inferred← citeshttps://doi.org/10.1002/alz.70337openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/alz.70592openalex-snapshot
Decoding amyloid beta clearance systems at inner blood–retina barrier using three‐dimensional ex vivo retinal imaging in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70748openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/alz.71570openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/ame2.12313openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/ana.21843openalex-snapshot
APOE predicts amyloid‐beta but not tau Alzheimer pathology in cognitively normal aging
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Endogenous amyloid‐β is necessary for hippocampal synaptic plasticity and memory
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inferred← citeshttps://doi.org/10.1002/ana.25513openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/ange.201305372openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/ange.202002574openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.200802808openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.200803908openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.201004437openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.201005093openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.201006335openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.201305372openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.201603178openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.202002574openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.202113845openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/anie.202215785openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/chem.202501099openalex-snapshot
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A lzheimer's Disease Biomarkers: More Than Molecular Diagnostics
inferred← citeshttps://doi.org/10.1002/ddr.22102openalex-snapshot
Mesenchymal stem cell‐derived rapid drug screening system for Alzheimer's disease for the identification of novel drugs
inferred← citeshttps://doi.org/10.1002/ejoc.201700010openalex-snapshot
Synthesis and Characterization of Hairpin Mimics that Modulate the Early Oligomerization and Fibrillization of Amyloid β‐Peptide
inferred← citeshttps://doi.org/10.1002/elps.201400271openalex-snapshot
An improved capillary electrophoresis method for in vitro monitoring of the challenging early steps of Aβ1–42 peptide oligomerization: Application to anti‐Alzheimer's drug discovery
inferred← citeshttps://doi.org/10.1002/emmm.201000082openalex-snapshot
Prion protein and Aβ‐related synaptic toxicity impairment
inferred← citeshttps://doi.org/10.1002/emmm.201201923openalex-snapshot
Reducing HDAC6 ameliorates cognitive deficits in a mouse model for Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.22331openalex-snapshot
Complement component C3 and complement receptor type 3 contribute to the phagocytosis and clearance of fibrillar Aβ by microglia
inferred← citeshttps://doi.org/10.1002/glia.24007openalex-snapshot
Haploinsufficiency of microglial MyD88 ameliorates Alzheimer's pathology and vascular disorders in APP / PS1 ‐transgenic mice
inferred← citeshttps://doi.org/10.1002/gps.3879openalex-snapshot
Neurodegeneration, β‐amyloid and mood disorders: state of the art and future perspectives
inferred← citeshttps://doi.org/10.1002/hipo.20861openalex-snapshot
Layer‐specific alterations to CA1 dendritic spines in a mouse model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hipo.23002openalex-snapshot
Chronic ghrelin administration restores hippocampal long‐term potentiation and ameliorates memory impairment in rat model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ijch.201600097openalex-snapshot
Flexible N‐Termini of Amyloid β‐Protein Oligomers: A Link between Structure and Activity?
inferred← citeshttps://doi.org/10.1002/iub.308openalex-snapshot
Role of the SNK‐SPAR pathway in the development of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/j.2055-2335.2012.tb00133.xopenalex-snapshot
Drug Treatments in Development for Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/jbio.201800359openalex-snapshot
Photobiomodulation preconditioning prevents cognitive impairment in a neonatal rat model of hypoxia‐ischemia
inferred← citeshttps://doi.org/10.1002/jbt.70903openalex-snapshot
Escin Attenuates Amyloid Beta 1‐42‐Induced Oxidative Stress, Apoptosis, and Neuroinflammation in Neuron‐Like SH‐SY5Y Cells
inferred← citeshttps://doi.org/10.1002/jcb.26129openalex-snapshot
Dysregulated Metabolism of the Amyloid‐β Protein and Therapeutic Approaches in Alzheimer Disease
inferred← citeshttps://doi.org/10.1002/jccs.70264openalex-snapshot
Enhanced Seeding Efficacy of Reverse Micelle‐Propagated Aβ 40 Oligomers Arises From Local Conformational Dynamics
inferred← citeshttps://doi.org/10.1002/jcph.249openalex-snapshot
Safety, tolerability, pharmacokinetics, and pharmacodynamics of the novel γ-secretase modulator, E2212, in healthy human subjects
inferred← citeshttps://doi.org/10.1002/jnr.21998openalex-snapshot
NMDA receptor–dependent signaling pathways that underlie amyloid β‐protein disruption of LTP in the hippocampus
inferred← citeshttps://doi.org/10.1002/jnr.22145openalex-snapshot
The small heat shock protein Hsp27 protects cortical neurons against the toxic effects of β‐amyloid peptide
inferred← citeshttps://doi.org/10.1002/jnr.22445openalex-snapshot
Amyloid‐β oligomers impair fear conditioned memory in a calcineurin‐dependent fashion in mice
inferred← citeshttps://doi.org/10.1002/jnr.22615openalex-snapshot
Dissociation of β-amyloid from lipoprotein in cerebrospinal fluid from Alzheimer's disease accelerates β-amyloid-42 assembly
inferred← citeshttps://doi.org/10.1002/jnr.22775openalex-snapshot
Huperzine a alleviates synaptic deficits and modulates amyloidogenic and nonamyloidogenic pathways in APPswe/PS1dE9 transgenic mice
inferred← citeshttps://doi.org/10.1002/jnr.23256openalex-snapshot
Effects of antiparkinsonian agents on β-amyloid and α-synuclein oligomer formation in vitro
inferred← citeshttps://doi.org/10.1002/jnr.23510openalex-snapshot
Small Aβ1–42 oligomer‐induced membrane depolarization of neuronal and microglial cells: Role of N‐methyl‐D‐aspartate receptors
inferred← citeshttps://doi.org/10.1002/jnr.24075openalex-snapshot
Astrocyte dysfunction in Alzheimer disease
inferred← citeshttps://doi.org/10.1002/jnr.24561openalex-snapshot
PPARα serves as a new receptor of aspirin for neuroprotection
inferred← citeshttps://doi.org/10.1002/mco2.70156openalex-snapshot
DEAD‐Box Helicase 6 Blockade in Brain‐Derived Aβ Oligomers From Alzheimer's Disease Patients Attenuates Neurotoxicity
inferred← citeshttps://doi.org/10.1002/med.21440openalex-snapshot
Bridging Type 2 Diabetes and Alzheimer's Disease: Assembling the Puzzle Pieces in the Quest for the Molecules With Therapeutic and Preventive Potential
inferred← citeshttps://doi.org/10.1002/mma.70029openalex-snapshot
The Kinetics and Mechanism of Inhibitor Drugs in the Treatment of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/mnfr.202100626openalex-snapshot
Green Tea Suppresses Amyloid β Levels and Alleviates Cognitive Impairment by Inhibiting APP Cleavage and Preventing Neurotoxicity in 5XFAD Mice
inferred← citeshttps://doi.org/10.1002/mnfr.202101032openalex-snapshot
Bioavailability and Health Impact of Ingested Amyloid‐like Protein Fibrils and their Link with Inflammatory Status: A Need for More Research?
inferred← citeshttps://doi.org/10.1002/mrm.24665openalex-snapshot
Use of Magnetization Transfer Contrast MRI to Detect Early Molecular Pathology in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/msj.20156openalex-snapshot
Vaccination as a Therapeutic Approach to Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/nep3.70037openalex-snapshot
Depletion of microglial compensation in glial network: Disease‐associated response dynamics in the revised amyloid hypothesis
inferred← citeshttps://doi.org/10.1002/path.4491openalex-snapshot
Effect of active Aβ immunotherapy on neurons in human Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/path.5056openalex-snapshot
The diabetes drug liraglutide reverses cognitive impairment in mice and attenuates insulin receptor and synaptic pathology in a non‐human primate model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/pmic.201200192openalex-snapshot
A fast and reproducible method for albumin isolation and depletion from serum and cerebrospinal fluid
inferred← citeshttps://doi.org/10.1002/pmic.201800059openalex-snapshot
Formation of Heterotypic Amyloids: α‐Synuclein in Co‐Aggregation
inferred← citeshttps://doi.org/10.1002/pnp.12008openalex-snapshot
The Role of Hormone Replacement Therapy (HRT) for the Prevention of Alzheimer's Disease in Postmenopausal Women
inferred← citeshttps://doi.org/10.1002/pro.2479openalex-snapshot
Structural evolution and membrane interactions of Alzheimer's amyloid‐beta peptide oligomers: New knowledge from single‐molecule fluorescence studies
inferred← citeshttps://doi.org/10.1002/pro.33openalex-snapshot
Discovery of amyloid‐beta aggregation inhibitors using an engineered assay for intracellular protein folding and solubility
inferred← citeshttps://doi.org/10.1002/pro.3778openalex-snapshot
β‐amyloid model core peptides: Effects of hydrophobes and disulfides
inferred← citeshttps://doi.org/10.1002/pro.3906openalex-snapshot
Progress toward Alzheimer's disease treatment: Leveraging the Achilles' heel of Aβ oligomers?
inferred← citeshttps://doi.org/10.1002/pro.4283openalex-snapshot
Molecular insights into the primary nucleation of polymorphic amyloid β dimers in DOPC lipid bilayer membrane
inferred← citeshttps://doi.org/10.1002/pro.4639openalex-snapshot
Structural polymorphism and cytotoxicity of brain‐derived β‐amyloid extracts
inferred← citeshttps://doi.org/10.1002/pro.4728openalex-snapshot
An ankyrin repeat chaperone targets toxic oligomers during amyloidogenesis
inferred← citeshttps://doi.org/10.1002/pro.70519openalex-snapshot
Toxic mechanisms of amyloid oligomers and therapeutic strategies
inferred← citeshttps://doi.org/10.1002/pro.70754openalex-snapshot
Multimerization of a rationally designed nanobody for enhanced avidity toward Aβ42 oligomers
inferred← citeshttps://doi.org/10.1002/pro.87openalex-snapshot
9,10‐Anthraquinone hinders β‐aggregation: How does a small molecule interfere with Aβ‐peptide amyloid fibrillation?
inferred← citeshttps://doi.org/10.1002/prot.22406openalex-snapshot
Interpeptide interactions induce helix to strand structural transition in Aβ peptides
inferred← citeshttps://doi.org/10.1002/prot.22763openalex-snapshot
A molecular dynamics study of the early stages of amyloid‐β(1–42) oligomerization: The role of lipid membranes
inferred← citeshttps://doi.org/10.1002/prot.22861openalex-snapshot
Asymmetric amyloid fibril elongation: A new perspective on a symmetric world
inferred← citeshttps://doi.org/10.1002/prot.24312openalex-snapshot
Structural studies of the tethered N-terminus of the Alzheimer's disease amyloid-β peptide
inferred← citeshttps://doi.org/10.1002/prot.24921openalex-snapshot
Sucrose prevents protein fibrillation through compaction of the tertiary structure but hardly affects the secondary structure
inferred← citeshttps://doi.org/10.1002/prot.24995openalex-snapshot
A2T and A2V Aβ peptides exhibit different aggregation kinetics, primary nucleation, morphology, structure, and LTP inhibition
inferred← citeshttps://doi.org/10.1002/prot.25972openalex-snapshot
Dimerization of Aβ40 inside dipalmitoylphosphatidylcholine bilayer and its effect on bilayer integrity: Atomistic simulation at three temperatures
inferred← citeshttps://doi.org/10.1002/psc.2474openalex-snapshot
Critique of the use of fluorescence‐based reporters in Escherichia coli as a screening tool for the identification of peptide inhibitors of Aβ42 aggregation
inferred← citeshttps://doi.org/10.1002/smll.201701828openalex-snapshot
Detection of β‐Amyloid by Sialic Acid Coated Bovine Serum Albumin Magnetic Nanoparticles in a Mouse Model of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/smtd.202400058openalex-snapshot
Characterizing Prion‐Like Protein Aggregation: Emerging Nanopore‐Based Approaches
inferred← citeshttps://doi.org/10.1002/syn.21642openalex-snapshot
Glycogen synthase kinase‐3β (GSK3β) expression in a mouse model of Alzheimer's disease: A light and electron microscopy study
inferred← citeshttps://doi.org/10.1002/syn.21915openalex-snapshot
Calcium channel blockade attenuates abnormal synaptic transmission in the dentate gyrus elicited by entorhinal amyloidopathy
inferred← citeshttps://doi.org/10.1002/syn.22025openalex-snapshot
Characterization of a 3xTg‐AD mouse model of Alzheimer's disease with the senescence accelerated mouse prone 8 (SAMP8) background
inferred← citeshttps://doi.org/10.1002/tcr.201200016openalex-snapshot
Design, Synthesis, and Evaluation of Bioactive Small Molecules
inferred← citeshttps://doi.org/10.1002/trc2.12087openalex-snapshot
Aβ oligomer induced cognitive impairment and evaluation of ACU193‐MNS‐based MRI in rabbit
inferred← citeshttps://doi.org/10.1002/trc2.70069openalex-snapshot
Understanding the impact of amyloid beta targeted therapies on biomarkers and clinical endpoints in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/trc2.70184openalex-snapshot
Relationship between efficacy and preferential targeting of soluble Aβ aggregates
inferred← citeshttps://doi.org/10.1002/wcms.1359openalex-snapshot
Revisiting the earliest signatures of amyloidogenesis: Roadmaps emerging from computational modeling and experiment
inferred← citeshttps://doi.org/10.1002/wcs.62openalex-snapshot
Long‐term potentiation (LTP) of human sensory‐evoked potentials
inferred← citeshttps://doi.org/10.1002/wnan.102openalex-snapshot
Nanoimaging for protein misfolding diseases
inferred← citeshttps://doi.org/10.1002/wsbm.1262openalex-snapshot
Apolipoprotein E receptor pathways in Alzheimer disease
inferred← citeshttps://doi.org/10.1007/7355_2016_14openalex-snapshot
Targets and Strategies Toward the Development of Alzheimer Therapeutics
inferred← citeshttps://doi.org/10.1007/7355_2026_186openalex-snapshot
Drug Discovery for Alzheimer’s Disease: Current Status
inferred← citeshttps://doi.org/10.1007/7515_2020_28openalex-snapshot
Overview of Multifaceted Role and Significance of Heat Shock Proteins During Inflammation, Apoptosis and Other Diseases
inferred← citeshttps://doi.org/10.1007/7854_2024_565openalex-snapshot
Hippocampal Synaptic Plasticity: Integrating Memory and Anxiety Impairments in the Early Stages of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-0716-1546-1_10openalex-snapshot
All-Atom Molecular Dynamics Simulation Methods for the Aggregation of Protein and Peptides: Replica Exchange/Permutation and Nonequilibrium Simulations
inferred← citeshttps://doi.org/10.1007/978-1-0716-1843-1_18openalex-snapshot
Analyzing Morphological Properties of Early-Stage Toxic Amyloid β Oligomers by Atomic Force Microscopy
inferred← citeshttps://doi.org/10.1007/978-1-0716-2597-2_2openalex-snapshot
Preparation and Investigation of Crucial Oligomers in the Early Stages of Aβ40 and Aβ42 Aggregation
inferred← citeshttps://doi.org/10.1007/978-1-0716-2597-2_6openalex-snapshot
Time-Resolved In Situ AFM Measurement of Growth Rates of Aβ40 Fibrils
inferred← citeshttps://doi.org/10.1007/978-1-0716-2655-9_16openalex-snapshot
Aβ and Tau Prions Causing Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4419-7061-9_14openalex-snapshot
Synapses and Alzheimers’s Disease: Effect of Immunotherapy?
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