On the public citation graph
Adult mouse astrocytes degrade amyloid-β in vitro and in situ
0 signed · 898 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/(sici)1097-4547(19960815)45:4<382::aid-jnr7>3.0.co;2-5openalex-snapshot
Mouse astrocytes respond to the chemokines MCP-1 and KC, but reverse transcriptase-polymerase chain reaction does not detect mRNA for the KC or new MCP-1 receptor
inferredcites →https://doi.org/10.1002/(sici)1097-4547(19970801)49:3<342::aid-jnr9>3.0.co;2-copenalex-snapshot
Establishment of human adult astrocyte cultures derived from postmortem multiple sclerosis and control brain and spinal cord regions: immunophenotypical and functional characterization
inferredcites →https://doi.org/10.1002/glia.10148openalex-snapshot
Scavenger receptors in neurobiology and neuropathology: Their role on microglia and other cells of the nervous system
inferredcites →https://doi.org/10.1002/glia.1052openalex-snapshot
In vitro evaluation of reactive astrocyte migration, a component of tissue remodeling in glaucomatous optic nerve head
inferredcites →https://doi.org/10.1002/glia.1128openalex-snapshot
Expression of β‐chemokines and chemokine receptors in human fetal astrocyte and microglial co‐cultures: Potential role of chemokines in the developing CNS
inferredcites →https://doi.org/10.1006/exnr.1997.6738openalex-snapshot
Astrocytes Regulate Microglial Phagocytosis of Senile Plaque Cores of Alzheimer's Disease
inferredcites →https://doi.org/10.1006/exnr.1999.7096openalex-snapshot
β-Amyloid Immunoreactivity in Astrocytes in Alzheimer's Disease Brain Biopsies: An Electron Microscope Study
inferredcites →https://doi.org/10.1007/s004010000199openalex-snapshot
Microglial cells are the driving force in fibrillar plaque formation, whereas astrocytes are a leading factor in plaque degradation
inferredcites →https://doi.org/10.1007/s004010000242openalex-snapshot
Amyloid β-protein (Aβ)-containing astrocytes are located preferentially near N-terminal-truncated Aβ deposits in the human entorhinal cortex
inferredcites →https://doi.org/10.1007/s004010050790openalex-snapshot
Diffuse plaques associated with astroglial amyloid β protein, possibly showing a disappearing stage of senile plaques
inferredcites →https://doi.org/10.1016/0165-5728(95)00064-9openalex-snapshot
Production and function of monocyte chemoattractant protein-1 and other β-chemokines in murine glial cells
inferredcites →https://doi.org/10.1016/0197-4580(95)00055-jopenalex-snapshot
Amyloid β protein (Aβ) removal by neuroglial cells in culture
inferredcites →https://doi.org/10.1016/s0002-9440(10)64354-4openalex-snapshot
CD36, a Class B Scavenger Receptor, Is Expressed on Microglia in Alzheimer's Disease Brains and Can Mediate Production of Reactive Oxygen Species in Response to β-Amyloid Fibrils
inferredcites →https://doi.org/10.1016/s0165-5728(01)00239-9openalex-snapshot
Scavenger receptor class B type I (SR-BI) mediates adhesion of neonatal murine microglia to fibrillar β-amyloid
inferredcites →https://doi.org/10.1016/s0165-5728(98)00226-4openalex-snapshot
A central role for astrocytes in the inflammatory response to β-amyloid; chemokines, cytokines and reactive oxygen species are produced
inferredcites →https://doi.org/10.1016/s0306-4522(99)00538-2openalex-snapshot
Microglia and astrocytes in the adult rat brain: comparative immunocytochemical analysis demonstrates the efficacy of lipocortin 1 immunoreactivity
inferredcites →https://doi.org/10.1016/s0736-5748(00)00012-5openalex-snapshot
Induction of cytokines in glial cells surrounding cortical β‐amyloid plaques in transgenic Tg2576 mice with Alzheimer pathology
inferredcites →https://doi.org/10.1016/s0896-6273(00)80187-7openalex-snapshot
Microglial Cells Internalize Aggregates of the Alzheimer's Disease Amyloid β-Protein Via a Scavenger Receptor
inferredcites →https://doi.org/10.1016/s0896-6273(02)00794-8openalex-snapshot
Inflammation in Neurodegenerative Disease—A Double-Edged Sword
inferredcites →https://doi.org/10.1021/bi00479a028openalex-snapshot
.alpha.1.beta.1 Integrin heterodimer functions as a dual laminin/collagen receptor in neural cells
inferredcites →https://doi.org/10.1038/78682openalex-snapshot
Peripherally administered antibodies against amyloid β-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease
inferredcites →https://doi.org/10.1038/87945openalex-snapshot
TGF-β1 promotes microglial amyloid-β clearance and reduces plaque burden in transgenic mice
inferredcites →https://doi.org/10.1111/j.1440-1819.1997.tb02375.xopenalex-snapshot
Identification of monocyte chemoattractant protein‐1 in senile plaques and reactive microglia of Alzheimer's disease
inferredcites →https://doi.org/10.1523/jneurosci.20-11-04050.2000openalex-snapshot
High-Level Neuronal Expression of Aβ1–42in Wild-Type Human Amyloid Protein Precursor Transgenic Mice: Synaptotoxicity without Plaque Formation
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.5281/zenodo.8515openalex-extracted
Stimulating Neuroprotective and Regenerative Mechanisms in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archopht.122.8.1224openalex-snapshot
Unusual Phenotype of an Individual With the R124C Mutation in the TGFBI Gene
inferred← citeshttps://doi.org/10.1002/9781118406557.ch6openalex-snapshot
Neuroinflammation in Alzheimer's, Parkinson's and Huntington's Diseases
inferred← citeshttps://doi.org/10.1002/acn3.79openalex-snapshot
Phosphodiesterase III inhibitor promotes drainage of cerebrovascular β‐amyloid
inferred← citeshttps://doi.org/10.1002/advs.202510270openalex-snapshot
Uric Acid Functions as an Endogenous Modulator of Microglial Function and Amyloid Clearance in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/alz.13063openalex-snapshot
FDA‐approved carbonic anhydrase inhibitors reduce amyloid β pathology and improve cognition, by ameliorating cerebrovascular health and glial fitness
inferred← citeshttps://doi.org/10.1002/alz.13450openalex-snapshot
Astrocyte biomarkers GFAP and YKL‐40 mediate early Alzheimer's disease progression
inferred← citeshttps://doi.org/10.1002/alz.14084openalex-snapshot
Correlations between plasma markers and brain Aβ deposition across the AD continuum: Evidence from SILCODE
inferred← citeshttps://doi.org/10.1002/alz.14152openalex-snapshot
CT1812 biomarker signature from a meta‐analysis of CSF proteomic findings from two Phase 2 clinical trials in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70472openalex-snapshot
Integrating neuroinflammation biomarkers into the ATN(X) framework: Advances in Alzheimer's pathogenesis, diagnosis, and insights from non‐human primate models
inferred← citeshttps://doi.org/10.1002/ana.22191openalex-snapshot
γ‐Secretase component presenilin is important for microglia β‐amyloid clearance
inferred← citeshttps://doi.org/10.1002/ar.21018openalex-snapshot
Alzheimer's Mechanisms in Ischemic Brain Degeneration
inferred← citeshttps://doi.org/10.1002/biof.37openalex-snapshot
Greasing the wheels of Aβ clearance in Alzheimer's Disease: The role of lipids and apolipoprotein E
inferred← citeshttps://doi.org/10.1002/brb3.2736openalex-snapshot
Molecular and histological correlates of cognitive decline across age in male C57BL/6J mice
inferred← citeshttps://doi.org/10.1002/glia.20005openalex-snapshot
Astrocyte modulation of in vitro β‐amyloid neurotoxicity
inferred← citeshttps://doi.org/10.1002/glia.20171openalex-snapshot
Pigment epithelium-derived factor induces pro-inflammatory genes in neonatal astrocytes through activation of NF-?B and CREB
inferred← citeshttps://doi.org/10.1002/glia.20599openalex-snapshot
Transplanted astrocytes internalize deposited β‐amyloid peptides in a transgenic mouse model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.20726openalex-snapshot
Minocycline reduces engraftment and activation of bone marrow‐derived cells but sustains their phagocytic activity in a mouse model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.20822openalex-snapshot
Binding and uptake of Aβ1‐42 by primary human astrocytes in vitro
inferred← citeshttps://doi.org/10.1002/glia.21004openalex-snapshot
Astrocytic Aβ1‐42 uptake is determined by Aβ‐aggregation state and the presence of amyloid‐associated proteins
inferred← citeshttps://doi.org/10.1002/glia.21025openalex-snapshot
NOV/CCN3 upregulates CCL2 and CXCL1 expression in astrocytes through β1 and β5 integrins
inferred← citeshttps://doi.org/10.1002/glia.21212openalex-snapshot
Multiple cellular and molecular mechanisms Are involved in human Aβ clearance by transplanted adult astrocytes
inferred← citeshttps://doi.org/10.1002/glia.22347openalex-snapshot
Upregulation of astrocytes protein phosphatase‐2A stimulates astrocytes migration via inhibiting p38 MAPK in tg2576 mice
inferred← citeshttps://doi.org/10.1002/glia.22548openalex-snapshot
β-amyloid and ATP-induced diffusional trapping of astrocyte and neuronal metabotropic glutamate type-5 receptors
inferred← citeshttps://doi.org/10.1002/glia.22784openalex-snapshot
Fatty acid‐binding protein 7 regulates function of caveolae in astrocytes through expression of caveolin‐1
inferred← citeshttps://doi.org/10.1002/glia.22800openalex-snapshot
GFAP and vimentin deficiency alters gene expression in astrocytes and microglia in wild‐type mice and changes the transcriptional response of reactive glia in mouse model for A lzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.22926openalex-snapshot
Regulation of astrocyte pathology by fluoxetine prevents the deterioration of Alzheimer phenotypes in an APP/PS 1 mouse model
inferred← citeshttps://doi.org/10.1002/glia.22974openalex-snapshot
Simvastatin and atorvastatin facilitates amyloid β‐protein degradation in extracellular spaces by increasing neprilysin secretion from astrocytes through activation of MAPK/E rk1/2 pathways
inferred← citeshttps://doi.org/10.1002/glia.23270openalex-snapshot
Phagocytic clearance of presynaptic dystrophies by reactive astrocytes in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.23460openalex-snapshot
Microglia have a more extensive and divergent response to interferon‐α compared with astrocytes
inferred← citeshttps://doi.org/10.1002/glia.23571openalex-snapshot
Neuroinflammation in the central nervous system: Symphony of glial cells
inferred← citeshttps://doi.org/10.1002/glia.23759openalex-snapshot
Ablation of reactive astrocytes exacerbates disease pathology in a model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.23845openalex-snapshot
Morphological and molecular alterations of reactive astrocytes without proliferation in cerebral cortex of an APP / PS1 transgenic mouse model and Alzheimer's patients
inferred← citeshttps://doi.org/10.1002/glia.23943openalex-snapshot
Amyloid‐β impairs the phagocytosis of dystrophic synapses by astrocytes in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.23952openalex-snapshot
Roles for H+/K+‐ ATPase and zinc transporter 3 in cAMP ‐mediated lysosomal acidification in bafilomycin A1 ‐treated astrocytes
inferred← citeshttps://doi.org/10.1002/glia.24312openalex-snapshot
Altered glial expression of the cannabinoid 1 receptor in the subiculum of a mouse model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.24539openalex-snapshot
Astrocytic uptake of posttranslationally modified amyloid‐β leads to endolysosomal system disruption and induction of pro‐inflammatory signaling
inferred← citeshttps://doi.org/10.1002/glia.70114openalex-snapshot
LRP4+ Astrocytes: A Unique Subpopulation Crucial for Blood Vessel Maintenance and Function in the Somatosensory Cortex of Normal and 5xFAD Mice
inferred← citeshttps://doi.org/10.1002/ibra.12026openalex-snapshot
Research progress of the CXCR4 mechanism in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/jcp.26462openalex-snapshot
HIV‐1 increases extracellular amyloid‐beta levels through neprilysin regulation in primary cultures of human astrocytes
inferred← citeshttps://doi.org/10.1002/jcp.30646openalex-snapshot
The role of trimethylamine‐N‐Oxide in the development of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/jnr.20234openalex-snapshot
Lovastatin modulation of microglial activation via suppression of functional CD40 expression
inferred← citeshttps://doi.org/10.1002/jnr.20340openalex-snapshot
High mobility group box protein‐1 inhibits microglial Aβ clearance and enhances Aβ neurotoxicity
inferred← citeshttps://doi.org/10.1002/jnr.20984openalex-snapshot
Amyloid β up‐regulates brain‐derived neurotrophic factor production from astrocytes: Rescue from amyloid β‐related neuritic degeneration
inferred← citeshttps://doi.org/10.1002/jnr.20992openalex-snapshot
Possible role of scavenger receptor SRCL in the clearance of amyloid‐βin Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/jnr.21252openalex-snapshot
Differential regulation of BACE1 promoter activity by nuclear factor‐κB in neurons and glia upon exposure to β‐amyloid peptides
inferred← citeshttps://doi.org/10.1002/jnr.23082openalex-snapshot
Transforming growth factor‐β stimulates β amyloid uptake by microglia through Smad3‐dependent mechanisms
inferred← citeshttps://doi.org/10.1007/978-0-387-35135-3_11openalex-snapshot
Role of A β Transport and Clearance in the Pathogenesis and Treatment of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-0-387-70830-0_14openalex-snapshot
Neuroinflammation and Mitochondrial Dysfunction in Alzheimer's and Prion's Diseases
inferred← citeshttps://doi.org/10.1007/978-0-387-71522-3_11openalex-snapshot
Nonsteroidal Anti-inflammatory Drugs (NSAIDs) and Derived Aβ42-Lowering Molecules for Treatment and Prevention of Alzheimer's Disease (AD)
inferred← citeshttps://doi.org/10.1007/978-0-387-87995-6_12openalex-snapshot
Participation of Glial Cells in the Pathogenesis of AD: A Different View on Neuroinflammation
inferred← citeshttps://doi.org/10.1007/978-1-0716-1712-0_3openalex-snapshot
Activation of Astrocytes in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/978-1-4614-4729-0_2openalex-snapshot
Astrocyte–Neuron Communications
inferred← citeshttps://doi.org/10.1007/978-1-4614-4815-0_9openalex-snapshot
Extracellular Amyloid-β Protein Dynamics in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4614-8317-5_14openalex-snapshot
Ongoing Studies of Deimination in Neurodegenerative Diseases Using the F95 Antibody
inferred← citeshttps://doi.org/10.1007/978-1-4614-9123-1_22openalex-snapshot
The Interplay Between White Matter, Mitochondria, and Neuroprotection
inferred← citeshttps://doi.org/10.1007/978-1-4939-0974-2_12openalex-snapshot
Neurodegeneration and Neuroglia: Emphasis on Astroglia in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-1071-7_4openalex-snapshot
Inflammation and the Pathophysiology of Astrocytes in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/978-1-60761-898-0_7openalex-snapshot
Pathological Validation of Animal Models of Dementia
inferred← citeshttps://doi.org/10.1007/978-1-60761-938-3_3openalex-snapshot
Morphology and Properties of Astrocytes
inferred← citeshttps://doi.org/10.1007/978-1-84628-440-3_15openalex-snapshot
Immunotherapeutic Approaches to Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-33946-3_8openalex-snapshot
Neuroprotective Immunity for Neurodegenerative and Neuroinfectious Diseases
inferred← citeshttps://doi.org/10.1007/978-3-031-41188-5_2openalex-snapshot
Pathophysiological Mechanisms of Brain Disorders
inferred← citeshttps://doi.org/10.1007/978-3-031-68237-7_11openalex-snapshot
Autophagic and Lysosomal Disorders
inferred← citeshttps://doi.org/10.1007/978-3-319-07320-0_15openalex-snapshot
The Chemokine (C-C Motif) Ligand 2 in Neuroinflammation and Neurodegeneration
inferred← citeshttps://doi.org/10.1007/978-3-319-23246-1_7openalex-snapshot
The Role of Aging in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-319-45096-4_13openalex-snapshot
Glutamine Synthetase: Role in Neurological Disorders
inferred← citeshttps://doi.org/10.1007/978-3-319-60733-7_7openalex-snapshot
Can Astrocytes Be a Target for Precision Medicine?
inferred← citeshttps://doi.org/10.1007/978-94-024-1088-4_7openalex-snapshot
TRPC Channels and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-13-9913-8_11openalex-snapshot
Astroglia in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-16-8313-8_9openalex-snapshot
Role of Reactive Astrocytes in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-19-7376-5_14openalex-snapshot
Molecular Imaging of Neuroinflammation in Alzheimer’s Disease and Mild Cognitive Impairment
inferred← citeshttps://doi.org/10.1007/bf03033906openalex-snapshot
Glial cell dysregulation: a new perspective on Alzheimer disease
inferred← citeshttps://doi.org/10.1007/s00011-025-02016-5openalex-snapshot
APOE4 impairs autophagy and Aβ clearance by microglial cells
inferred← citeshttps://doi.org/10.1007/s00018-019-03040-5openalex-snapshot
Cerebrospinal fluid biomarkers for understanding multiple aspects of Alzheimer’s disease pathogenesis
inferred← citeshttps://doi.org/10.1007/s00221-017-4928-1openalex-snapshot
Astrocytes and synaptic plasticity in health and disease
inferred← citeshttps://doi.org/10.1007/s00259-014-2859-7openalex-snapshot
Astrocytosis measured by 11C-deprenyl PET correlates with decrease in gray matter density in the parahippocampus of prodromal Alzheimer’s patients
inferred← citeshttps://doi.org/10.1007/s00259-015-3047-0openalex-snapshot
Astrocytosis precedes amyloid plaque deposition in Alzheimer APPswe transgenic mouse brain: a correlative positron emission tomography and in vitro imaging study
inferred← citeshttps://doi.org/10.1007/s00259-018-4217-7openalex-snapshot
Longitudinal association between astrocyte function and glucose metabolism in autosomal dominant Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-019-04462-wopenalex-snapshot
Prospects and challenges of imaging neuroinflammation beyond TSPO in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00401-004-0824-4openalex-snapshot
Glial reactions and the clearance of amyloid β protein in the brains of patients with hereditary cerebral hemorrhage with amyloidosis-Dutch type
inferred← citeshttps://doi.org/10.1007/s00401-007-0284-8openalex-snapshot
Mechanisms of amyloid plaque pathogenesis
inferred← citeshttps://doi.org/10.1007/s00401-008-0366-2openalex-snapshot
Cerebral amyloid angiopathy and its relationship to Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00401-009-0513-4openalex-snapshot
Receptor for advanced glycation end products is upregulated in optic neuropathy of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00401-009-0619-8openalex-snapshot
Astrocytes: biology and pathology
inferred← citeshttps://doi.org/10.1007/s00401-014-1375-yopenalex-snapshot
Neuropathology and biochemistry of Aβ and its aggregates in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00401-015-1513-1openalex-snapshot
Astrocytes: a central element in neurological diseases
inferred← citeshttps://doi.org/10.1007/s00429-015-1007-xopenalex-snapshot
Histamine induces the production of matrix metalloproteinase-9 in human astrocytic cultures via H1-receptor subtype
inferred← citeshttps://doi.org/10.1007/s00702-010-0433-4openalex-snapshot
The role of microglia in amyloid clearance from the AD brain
inferred← citeshttps://doi.org/10.1007/s00702-010-0438-zopenalex-snapshot
Neuroinflammatory processes in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00775-007-0317-8openalex-snapshot
13C heteronuclear NMR studies of the interaction of cultured neurons and astrocytes and aluminum blockade of the preferential release of citrate from astrocytes
inferred← citeshttps://doi.org/10.1007/s10522-009-9217-3openalex-snapshot
The difference in gliosis induced by β-amyloid and Tau treatments in astrocyte cultures derived from senescence accelerated and normal mouse strains
inferred← citeshttps://doi.org/10.1007/s10571-006-9127-8openalex-snapshot
Aβ Upregulates and Colocalizes with LGI3 in Cultured Rat Astrocytes
inferred← citeshttps://doi.org/10.1007/s10571-014-0101-6openalex-snapshot
Microglial Aβ Receptors in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s11011-022-00902-zopenalex-snapshot
A kinetic scheme to examine the role of glial cells in the pathogenesis of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s11064-005-6962-9openalex-snapshot
Basal Forebrain Cholinergic Dysfunction in Alzheimer’s Disease – Interrelationship with β-amyloid, Inflammation and Neurotrophin Signaling
inferred← citeshttps://doi.org/10.1007/s11064-006-9128-5openalex-snapshot
Glycogen Synthase Kinase-3 (GSK3): Inflammation, Diseases, and Therapeutics
inferred← citeshttps://doi.org/10.1007/s11064-012-0866-2openalex-snapshot
Ginsennoside Rd Attenuates Cognitive Dysfunction in a Rat Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s11064-012-0868-0openalex-snapshot
Primary Cultures of Astrocytes: Their Value in Understanding Astrocytes in Health and Disease
inferred← citeshttps://doi.org/10.1007/s11064-017-2237-5openalex-snapshot
Oxabicycloheptene Sulfonate Protects Against β-Amyloid-induced Toxicity by Activation of PI3K/Akt and ERK Signaling Pathways Via GPER1 in C6 Cells
inferred← citeshttps://doi.org/10.1007/s11064-022-03699-6openalex-snapshot
Aß Pathology and Neuron–Glia Interactions: A Synaptocentric View
inferred← citeshttps://doi.org/10.1007/s11064-022-03714-wopenalex-snapshot
The Emerging Role of Astrocytic Autophagy in Central Nervous System Disorders
inferred← citeshttps://doi.org/10.1007/s11302-025-10066-xopenalex-snapshot
Extracellular ATP regulates phagocytic activity, mitochondrial respiration, and cytokine secretion of human astrocytic cells
inferred← citeshttps://doi.org/10.1007/s11357-023-00916-0openalex-snapshot
FABP7 drives an inflammatory response in human astrocytes and is upregulated in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s11481-012-9427-3openalex-snapshot
Amyloid-β-Induced Astrocytic Phagocytosis is Mediated by CD36, CD47 and RAGE
inferred← citeshttps://doi.org/10.1007/s11481-013-9439-7openalex-snapshot
Glia Maturation Factor Induces Interleukin-33 Release from Astrocytes: Implications for Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/s11481-017-9768-zopenalex-snapshot
Extracellular Vesicle Biology in Alzheimer’s Disease and Related Tauopathy
inferred← citeshttps://doi.org/10.1007/s11910-022-01235-1openalex-snapshot
Vascular Considerations for Amyloid Immunotherapy
inferred← citeshttps://doi.org/10.1007/s12031-014-0465-6openalex-snapshot
Haptoglobin Modulates Beta-Amyloid Uptake by U-87 MG Astrocyte Cell Line
inferred← citeshttps://doi.org/10.1007/s12031-016-0757-0openalex-snapshot
Modulation of Corpus Striatal Neurochemistry by Astrocytes and Vasoactive Intestinal Peptide (VIP) in Parkinsonian Rats
inferred← citeshttps://doi.org/10.1007/s12031-018-1085-3openalex-snapshot
Periodic Variation of AAK1 in an Aβ1–42-Induced Mouse Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12031-019-01357-1openalex-snapshot
Friends Turn Foe—Astrocytes Contribute to Neuronal Damage in NeuroAIDS
inferred← citeshttps://doi.org/10.1007/s12031-022-02026-6openalex-snapshot
Ketone Body Rescued Seizure Behavior of LRP1 Deficiency in Drosophila by Modulating Glutamate Transport
inferred← citeshttps://doi.org/10.1007/s12035-008-8043-yopenalex-snapshot
Targeted Activation of Astrocytes: A Potential Neuroprotective Strategy
inferred← citeshttps://doi.org/10.1007/s12035-013-8439-1openalex-snapshot
Sporadic Alzheimer’s Disease Begins as Episodes of Brain Ischemia and Ischemically Dysregulated Alzheimer’s Disease Genes
inferred← citeshttps://doi.org/10.1007/s12035-013-8539-yopenalex-snapshot
Intracerebroventricular Streptozotocin Exacerbates Alzheimer-Like Changes of 3xTg-AD Mice
inferred← citeshttps://doi.org/10.1007/s12035-015-9427-4openalex-snapshot
Astrocytes Surviving Severe Stress Can Still Protect Neighboring Neurons from Proteotoxic Injury
inferred← citeshttps://doi.org/10.1007/s12035-016-0193-8openalex-snapshot
Relationship Between Obesity, Alzheimer’s Disease, and Parkinson’s Disease: an Astrocentric View
inferred← citeshttps://doi.org/10.1007/s12035-017-0467-9openalex-snapshot
NLRP3 Inflammasome Inhibitor Ameliorates Amyloid Pathology in a Mouse Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-017-0762-5openalex-snapshot
SVCT2 Expression and Function in Reactive Astrocytes Is a Common Event in Different Brain Pathologies
inferred← citeshttps://doi.org/10.1007/s12035-020-01982-7openalex-snapshot
Silencing of LRP1 Exacerbates Inflammatory Response Via TLR4/NF-κB/MAPKs Signaling Pathways in APP/PS1 Transgenic Mice
inferred← citeshttps://doi.org/10.1007/s12079-014-0243-9openalex-snapshot
Heat shock proteins in neurodegenerative disorders and aging
inferred← citeshttps://doi.org/10.1007/s12264-018-0262-2openalex-snapshot
Early Activation of Astrocytes does not Affect Amyloid Plaque Load in an Animal Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12264-021-00690-zopenalex-snapshot
A Novel Phagocytic Role of Astrocytes in Activity-dependent Elimination of Mature Excitatory Synapses
inferred← citeshttps://doi.org/10.1007/s12264-025-01572-4openalex-snapshot
A Question of Origins: Non-neuronal Sources of Amyloid-β
inferred← citeshttps://doi.org/10.1007/s12272-010-1006-7openalex-snapshot
Inflammation and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s12551-012-0091-xopenalex-snapshot
Computational modeling of the relationship between amyloid and disease
inferred← citeshttps://doi.org/10.1007/s12640-013-9453-9openalex-snapshot
Recent Discoveries on the Functions of Astrocytes in the Copper Homeostasis of the Brain: A Brief Update
inferred← citeshttps://doi.org/10.1007/s13205-025-04468-2openalex-snapshot
Implicating neuroinflammation in hippocampus, prefrontal cortex and amygdala with cognitive deficit: a narrative review
inferred← citeshttps://doi.org/10.1007/s13206-025-00228-9openalex-snapshot
Piezo1 Activation Promotes Glial Anti-inflammation and Tau Dephosphorylation in an Alzheimer’s Disease Model
inferred← citeshttps://doi.org/10.1007/s13273-026-00625-7openalex-snapshot
Acetate-induced reactive astrocytes enhance uptake of the toxic molecule amyloid-β with upregulation of MEGF10 and MERTK
inferred← citeshttps://doi.org/10.1007/s13670-011-0004-zopenalex-snapshot
Animal Models of Alzheimer’s Disease: Utilization of Transgenic Alzheimer’s Disease Models in Studies of Amyloid Beta Clearance
inferred← citeshttps://doi.org/10.1007/s13670-012-0008-3openalex-snapshot
Erratum to: Animal Models of Alzheimer’s Disease: Utilization of Transgenic Alzheimer’s Disease Models in Studies of Amyloid Beta Clearance
inferred← citeshttps://doi.org/10.1007/s40263-017-0483-3openalex-snapshot
Targeting Neuroinflammation to Treat Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s40778-016-0049-1openalex-snapshot
Human Stem Cell-Derived Astrocytes: Specification and Relevance for Neurological Disorders
inferred← citeshttps://doi.org/10.1016/b978-0-12-394317-0.00011-xopenalex-snapshot
Clusterin in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-802173-6.00010-1openalex-snapshot
Alzheimer’s Disease Therapeutics Targeting Apolipoprotein E
inferred← citeshttps://doi.org/10.1016/b978-0-12-804256-4.00008-5openalex-snapshot
Consequences of Intracellular Amyloid in Vascular System
inferred← citeshttps://doi.org/10.1016/b978-0-12-805088-0.00014-1openalex-snapshot
Noradrenaline, Astroglia, and Neuroinflammation
inferred← citeshttps://doi.org/10.1016/b978-0-12-809813-4.00005-3openalex-snapshot
Targeting Astrocytes With Viral Gene Therapy for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-809937-7.00006-9openalex-snapshot
Contribution of Neuroinflammation in the Pathogenesis of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-811304-2.00014-6openalex-snapshot
Non-cell Autonomous Degeneration
inferred← citeshttps://doi.org/10.1016/b978-0-12-815854-8.00013-6openalex-snapshot
The 5×FAD mouse model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-819410-2.00022-9openalex-snapshot
Synaptic dysfunction in early phases of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-820122-0.00001-7openalex-snapshot
Role of caspases, apoptosis and additional factors in pathology of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-821565-4.00012-2openalex-snapshot
Neurodegenerative diseases
inferred← citeshttps://doi.org/10.1016/b978-0-12-824010-6.00006-xopenalex-snapshot
Development of early biomarkers of Alzheimer's disease: A precision medicine perspective
inferred← citeshttps://doi.org/10.1016/b978-0-323-85764-2.00006-5openalex-snapshot
Human induced pluripotent stem cell– based modeling of Alzheimer’s disease, a glial perspective
inferred← citeshttps://doi.org/10.1016/b978-0-323-91704-9.00006-9openalex-snapshot
Neuro–glia communication and glycolysis
inferred← citeshttps://doi.org/10.1016/b978-008045046-9.00125-xopenalex-snapshot
Glial Cells: Astrocytes and Oligodendrocytes during Normal Brain Aging
inferred← citeshttps://doi.org/10.1016/bs.apcsb.2017.02.001openalex-snapshot
Neuroinflammation in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1016/j.ajpath.2010.11.057openalex-snapshot
Genetic Ablation of Apolipoprotein A-IV Accelerates Alzheimer's Disease Pathogenesis in a Mouse Model
inferred← citeshttps://doi.org/10.1016/j.ajpath.2010.12.051openalex-snapshot
Phagocytic Clearance in Neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.arr.2014.07.004openalex-snapshot
When astrocytes become harmful: Functional and inflammatory responses that contribute to Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.arr.2016.11.003openalex-snapshot
Disordered APP metabolism and neurovasculature in trauma and aging: Combined risks for chronic neurodegenerative disorders
inferred← citeshttps://doi.org/10.1016/j.arr.2020.101039openalex-snapshot
Astrocyte mitochondria: Central players and potential therapeutic targets for neurodegenerative diseases and injury
inferred← citeshttps://doi.org/10.1016/j.arr.2023.101865openalex-snapshot
The hippocampus associated GABAergic neural network impairment in early-stage of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.arr.2023.101871openalex-snapshot
A novel regulator in Alzheimer's disease progression: The astrocyte-derived extracellular vesicles
inferred← citeshttps://doi.org/10.1016/j.arr.2024.102481openalex-snapshot
Pathogenesis, diagnostics, and therapeutics for Alzheimer's disease: Breaking the memory barrier
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103275openalex-snapshot
The human retina in Alzheimer’s disease: Pathology, mechanisms, and biomarkers
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103280openalex-snapshot
Is amyloid beta peptide a driver of inflammaging?
inferred← citeshttps://doi.org/10.1016/j.autrev.2018.09.009openalex-snapshot
The impact of self-replicating proteins on inflammation, autoimmunity and neurodegeneration—An untraveled path
inferred← citeshttps://doi.org/10.1016/j.bbadis.2006.12.001openalex-snapshot
Alzheimer disease: Amyloidogenesis, the presenilins and animal models
inferred← citeshttps://doi.org/10.1016/j.bbadis.2012.07.003openalex-snapshot
Inhibition of amyloid-beta peptide aggregation rescues the autophagic deficits in the TgCRND8 mouse model of Alzheimer disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.10.014openalex-snapshot
Regulation of cerebrospinal fluid (CSF) flow in neurodegenerative, neurovascular and neuroinflammatory disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.11.010openalex-snapshot
Vascular mTOR-dependent mechanisms linking the control of aging to Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.11.014openalex-snapshot
Reactive gliosis in the pathogenesis of CNS diseases
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2014.05.014openalex-snapshot
Cytokines and chemokines: At the crossroads of cell signalling and inflammatory disease
inferred← citeshttps://doi.org/10.1016/j.bbi.2018.10.004openalex-snapshot
Restoring microglial and astroglial homeostasis using DNA immunization in a Down Syndrome mouse model
inferred← citeshttps://doi.org/10.1016/j.bbi.2020.03.014openalex-snapshot
TIMP-1: A key cytokine released from activated astrocytes protects neurons and ameliorates cognitive behaviours in a rodent model of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.bbi.2020.10.007openalex-snapshot
Role of astroglial toll-like receptors (TLRs) in central nervous system infections, injury and neurodegenerative diseases
inferred← citeshttps://doi.org/10.1016/j.bbi.2021.11.021openalex-snapshot
ICAM-1 protects neurons against Amyloid-β and improves cognitive behaviors in 5xFAD mice by inhibiting NF-κB
inferred← citeshttps://doi.org/10.1016/j.bbr.2013.09.024openalex-snapshot
Voluntary exercise counteracts Aβ25-35-induced memory impairment in mice
inferred← citeshttps://doi.org/10.1016/j.bbr.2016.06.001openalex-snapshot
Infliximab ameliorates AD-associated object recognition memory impairment
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.07.117openalex-snapshot
Lib, transcriptionally induced in senile plaque-associated astrocytes, promotes glial migration through extracellular matrix
inferred← citeshttps://doi.org/10.1016/j.bbrc.2009.12.114openalex-snapshot
Ischemia-activated microglia induces neuronal injury via activation of gp91phox NADPH oxidase
inferred← citeshttps://doi.org/10.1016/j.bbrc.2024.150418openalex-snapshot
Role of astrocytes in Alzheimer's disease pathogenesis and the impact of exercise-induced remodeling
inferred← citeshttps://doi.org/10.1016/j.bcp.2012.04.009openalex-snapshot
G protein-coupled receptor signalling in astrocytes in health and disease: A focus on metabotropic glutamate receptors
inferred← citeshttps://doi.org/10.1016/j.biocel.2004.07.009openalex-snapshot
The role of inflammation in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.biocel.2015.04.013openalex-snapshot
Mitogen-activated protein kinase signaling pathways promote low-density lipoprotein receptor-related protein 1-mediated internalization of beta-amyloid protein in primary cortical neurons
inferred← citeshttps://doi.org/10.1016/j.biochi.2020.02.003openalex-snapshot
Cyclical amyloid beta-astrocyte activity induces oxidative stress in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.biopha.2026.119473openalex-snapshot
Glyceraldehyde-induced metabolic defects in cortical astrocytes: implication for Alzheimer's disease pathogenesis and therapy
inferred← citeshttps://doi.org/10.1016/j.brainres.2004.06.090openalex-snapshot
Accumulation of aluminum by primary cultured astrocytes from aluminum amino acid complex and its apoptotic effect
inferred← citeshttps://doi.org/10.1016/j.brainres.2004.07.084openalex-snapshot
Microglial reactivity to β-amyloid is modulated by astrocytes and proinflammatory factors
inferred← citeshttps://doi.org/10.1016/j.brainres.2005.06.018openalex-snapshot
Astrocytes react to oligemia in the forebrain induced by chronic bilateral common carotid artery occlusion in rats
inferred← citeshttps://doi.org/10.1016/j.brainres.2006.05.081openalex-snapshot
Cholesterol deficiency perturbs actin signaling and glutamate homeostasis in hippocampal astrocytes
inferred← citeshttps://doi.org/10.1016/j.brainres.2006.06.041openalex-snapshot
Effects of apoE isoforms on beta-amyloid-induced matrix metalloproteinase-9 in rat astrocytes
inferred← citeshttps://doi.org/10.1016/j.brainres.2006.06.050openalex-snapshot
A novel membrane protein, encoded by the gene covering KIAA0233, is transcriptionally induced in senile plaque-associated astrocytes
inferred← citeshttps://doi.org/10.1016/j.brainres.2008.12.082openalex-snapshot
Up-regulation of astrocyte metabotropic glutamate receptor 5 by amyloid-β peptide
inferred← citeshttps://doi.org/10.1016/j.brainres.2009.05.098openalex-snapshot
The activation of cannabinoid CB2 receptors stimulates in situ and in vitro beta-amyloid removal by human macrophages
inferred← citeshttps://doi.org/10.1016/j.brainres.2009.12.039openalex-snapshot
Apolipoprotein E protects cultured pericytes and astrocytes from D-Aβ1–40-mediated cell death
inferred← citeshttps://doi.org/10.1016/j.brainres.2011.02.078openalex-snapshot
Regulation of matrix metalloproteinase 2 by oligomeric amyloid β protein
inferred← citeshttps://doi.org/10.1016/j.brainres.2011.10.020openalex-snapshot
Genistein inhibits aggregation of exogenous amyloid-beta1–40 and alleviates astrogliosis in the hippocampus of rats
inferred← citeshttps://doi.org/10.1016/j.brainres.2011.12.062openalex-snapshot
Mitsugumin 29 is transcriptionally induced in senile plaque-associated astrocytes
inferred← citeshttps://doi.org/10.1016/j.brainres.2016.06.010openalex-snapshot
β-amyloid increases neurocan expression through regulating Sox9 in astrocytes: A potential relationship between Sox9 and chondroitin sulfate proteoglycans in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.brainresbull.2009.07.012openalex-snapshot
Astrocytic dysfunction: Insights on the role in neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.brainresbull.2016.03.017openalex-snapshot
Heterogeneous astrocytes: Active players in CNS
showing the first 200 of 874 · page through all 874
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.