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TNF defined as a therapeutic target for rheumatoid arthritis and other autoimmune diseases
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/1529-0131(199807)41:7<1258::aid-art17>3.0.co;2-1openalex-snapshot
Modulation of angiogenic vascular endothelial growth factor by tumor necrosis factor ? and interleukin-1 in rheumatoid arthritis
inferredcites →https://doi.org/10.1002/1529-0131(199809)41:9<1552::aid-art5>3.0.co;2-wopenalex-snapshot
Therapeutic efficacy of multiple intravenous infusions of anti-tumor necrosis factor ? monoclonal antibody combined with low-dose weekly methotrexate in rheumatoid arthritis
inferredcites →https://doi.org/10.1002/1529-0131(200001)43:1<38::aid-anr6>3.0.co;2-lopenalex-snapshot
Reduction of chemokine levels and leukocyte traffic to joints by tumor necrosis factor α blockade in patients with rheumatoid arthritis
inferredcites →https://doi.org/10.1002/art.10697openalex-snapshot
Adalimumab, a fully human anti–tumor necrosis factor α monoclonal antibody, for the treatment of rheumatoid arthritis in patients taking concomitant methotrexate: The ARMADA trial
inferredcites →https://doi.org/10.1002/art.1780340908openalex-snapshot
Localization of Tumor Necrosis Factor α in Synovial Tissues and at the Cartilage–Pannus Junction in Patients With Rheumatoid Arthritis
inferredcites →https://doi.org/10.1002/art.1780361206openalex-snapshot
Treatment of rheumatoid arthritis with chimeric monoclonal antibodies to tumor necrosis factor α
inferredcites →https://doi.org/10.1002/art.1780380202openalex-snapshot
Inhibition of the production and effects of interleukins‐1 and tumor necrosis factor α in rheumatoid arthritis
inferredcites →https://doi.org/10.1002/eji.1830211039openalex-snapshot
Expression of granulocyte‐macrophage colony‐stimulating factor in rheumatoid arthritis: Regulation by tumor necrosis factor‐α
inferredcites →https://doi.org/10.1002/j.1460-2075.1987.tb02598.xopenalex-snapshot
Structure and expression of human B cell stimulatory factor‐2 (BSF‐2/IL‐6) gene.
inferredcites →https://doi.org/10.1002/j.1460-2075.1991.tb04978.xopenalex-snapshot
Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis.
inferredcites →https://doi.org/10.1016/0016-5085(95)90277-5openalex-snapshot
Treatment of Crohn's disease with anti-tumor necrosis factor chimeric monoclonal antibody (cA2)
inferredcites →https://doi.org/10.1016/s0140-6736(02)07718-8openalex-snapshot
Adverse reactions to TNF-α inhibitors in rheumatoid arthritis
inferredcites →https://doi.org/10.1016/s0140-6736(81)91107-7openalex-snapshot
RHEUMATOID ARTHRITIS: A DISEASE OF T-LYMPHOCYTE/MACROPHAGE IMMUNOREGULATION
inferredcites →https://doi.org/10.1016/s0140-6736(83)90629-3openalex-snapshot
ROLE OF ABERRANT HLA-DR EXPRESSION AND ANTIGEN PRESENTATION IN INDUCTION OF ENDOCRINE AUTOIMMUNITY
inferredcites →https://doi.org/10.1016/s0140-6736(89)90430-3openalex-snapshot
INHIBITORY EFFECT OF TNF$alpha; ANTIBODIES ON SYNOVIAL CELL INTERLEUKIN-1 PRODUCTION IN RHEUMATOID ARTHRITIS
inferredcites →https://doi.org/10.1016/s0140-6736(94)90628-9openalex-snapshot
Randomised double-blind comparison of chimeric monoclonal antibody to tumour necrosis factor α (cA2) versus placebo in rheumatoid arthritis
inferredcites →https://doi.org/10.1016/s0140-6736(94)90632-7openalex-snapshot
Repeated therapy with monoclonal antibody to tumour necrosis factor α (cA2) in patients with rheumatoid arthritis
inferredcites →https://doi.org/10.1038/302305a0openalex-snapshot
Structure and expression of a cloned cDNA for human interleukin-2
inferredcites →https://doi.org/10.1038/312724a0openalex-snapshot
Human tumour necrosis factor: precursor structure, expression and homology to lymphotoxin
inferredcites →https://doi.org/10.1038/330662a0openalex-snapshot
Anti-cachectin/TNF monoclonal antibodies prevent septic shock during lethal bacteraemia
inferredcites →https://doi.org/10.1056/nejm199707173370301openalex-snapshot
Treatment of Rheumatoid Arthritis with a Recombinant Human Tumor Necrosis Factor Receptor (p75)–Fc Fusion Protein
inferredcites →https://doi.org/10.1056/nejm200011303432202openalex-snapshot
Infliximab and Methotrexate in the Treatment of Rheumatoid Arthritis
inferredcites →https://doi.org/10.1073/pnas.79.11.3632openalex-snapshot
Evidence in support of a self-perpetuating HLA-DR-dependent delayed-type cell reaction in rheumatoid arthritis.
inferredcites →https://doi.org/10.1073/pnas.89.20.9784openalex-snapshot
Anti-tumor necrosis factor ameliorates joint disease in murine collagen-induced arthritis.
inferredcites →https://doi.org/10.1073/pnas.91.7.2762openalex-snapshot
Synergy between anti-CD4 and anti-tumor necrosis factor in the amelioration of established collagen-induced arthritis.
inferredcites →https://doi.org/10.1084/jem.170.5.1627openalex-snapshot
Antibodies to cachectin/tumor necrosis factor reduce interleukin 1 beta and interleukin 6 appearance during lethal bacteremia.
inferredcites →https://doi.org/10.1146/annurev.immunol.14.1.397openalex-snapshot
ROLE OF CYTOKINES IN RHEUMATOID ARTHRITIS
inferredcites →https://doi.org/10.1146/annurev.immunol.19.1.163openalex-snapshot
Anti-TNFα Therapy of Rheumatoid Arthritis: What Have We Learned?
inferredcites →https://doi.org/10.4049/jimmunol.163.3.1521openalex-snapshot
Regulation of Cytokines, Cytokine Inhibitors, and Acute-Phase Proteins Following Anti-TNF-α Therapy in Rheumatoid Arthritis
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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The CD95/CD95L signaling pathway: A role in carcinogenesis
inferred← citeshttps://doi.org/10.1016/j.bbi.2005.03.015openalex-snapshot
The cholinergic anti-inflammatory pathway
inferred← citeshttps://doi.org/10.1016/j.bbi.2015.08.005openalex-snapshot
Circulating tumour necrosis factor is highly correlated with brainstem serotonin transporter availability in humans
inferred← citeshttps://doi.org/10.1016/j.bbi.2015.08.022openalex-snapshot
TNF-α from hippocampal microglia induces working memory deficits by acute stress in mice
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.06.018openalex-snapshot
The tumor necrosis factor-α AU-rich element inhibits the stable association of the 40S ribosomal subunit with RNA transcripts
inferred← citeshttps://doi.org/10.1016/j.bbrc.2009.08.052openalex-snapshot
Novel protein engineering strategy for creating highly receptor-selective mutant TNFs
inferred← citeshttps://doi.org/10.1016/j.bbrc.2015.07.094openalex-snapshot
Selective inhibition of TNFR1 reduces osteoclast numbers and is differentiated from anti-TNF in a LPS-driven model of inflammatory bone loss
inferred← citeshttps://doi.org/10.1016/j.bbrc.2017.03.106openalex-snapshot
Activation of PPARγ inhibits pro-inflammatory cytokines production by upregulation of miR-124 in vitro and in vivo
inferred← citeshttps://doi.org/10.1016/j.bbrc.2019.12.131openalex-snapshot
PTUPB ameliorates high-fat diet-induced non-alcoholic fatty liver disease via inhibiting NLRP3 inflammasome activation in mice
inferred← citeshttps://doi.org/10.1016/j.bbrc.2020.04.073openalex-snapshot
Development and application of an antibody that binds to interleukin-1β of various mammalian species for the treatment of inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.bcp.2004.12.008openalex-snapshot
The role of adenosine A2A and A2B receptors in the regulation of TNF-α production by human monocytes
inferred← citeshttps://doi.org/10.1016/j.bcp.2011.07.088openalex-snapshot
Island biogeography effects on microbial evolution may contribute to Crohn's disease
inferred← citeshttps://doi.org/10.1016/j.berh.2006.05.005openalex-snapshot
Treating very early rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.berh.2021.101740openalex-snapshot
Rheumatoid arthritis: Evolving recognition of a common disease
inferred← citeshttps://doi.org/10.1016/j.berh.2024.101974openalex-snapshot
Pharmacogenetics of therapies in rheumatoid arthritis: An update
inferred← citeshttps://doi.org/10.1016/j.biocel.2006.09.009openalex-snapshot
Duchenne muscular dystrophy: Focus on pharmaceutical and nutritional interventions
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2009.08.041openalex-snapshot
The treatment of established murine collagen-induced arthritis with a TNFR1-selective antagonistic mutant TNF
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2011.04.018openalex-snapshot
Fine tuning of receptor-selectivity for tumor necrosis factor-α using a phage display system with one-step competitive panning
inferred← citeshttps://doi.org/10.1016/j.biopha.2010.01.007openalex-snapshot
Therapeutic efficacy of experimental rheumatoid arthritis with low-dose methotrexate by increasing partially CD4+CD25+Treg cells and inducing Th1 to Th2 shift in both cells and cytokines
inferred← citeshttps://doi.org/10.1016/j.bios.2014.11.018openalex-snapshot
Approaching near real-time biosensing: Microfluidic microsphere based biosensor for real-time analyte detection
inferred← citeshttps://doi.org/10.1016/j.bmc.2009.12.008openalex-snapshot
Discovery of 3-(4-bromophenyl)-6-nitrobenzo[1.3.2]dithiazolium ylide 1,1-dioxide as a novel dual cyclooxygenase/5-lipoxygenase inhibitor that also inhibits tumor necrosis factor-α production
inferred← citeshttps://doi.org/10.1016/j.bmc.2017.05.061openalex-snapshot
Aligator: A computational tool for optimizing total chemical synthesis of large proteins
inferred← citeshttps://doi.org/10.1016/j.bmcl.2009.10.138openalex-snapshot
Novel 3-aminopyrazole inhibitors of MK-2 discovered by scaffold hopping strategy
inferred← citeshttps://doi.org/10.1016/j.cej.2022.140399openalex-snapshot
Targeted therapy for autoimmune diseases based on multifunctional frame nucleic acid system: Blocking TNF-α-NF-κB signaling and mediating macrophage polarization
inferred← citeshttps://doi.org/10.1016/j.cell.2006.02.016openalex-snapshot
Innate Immunity Gone Awry: Linking Microbial Infections to Chronic Inflammation and Cancer
inferred← citeshttps://doi.org/10.1016/j.cell.2008.03.036openalex-snapshot
TNF-α Induces Two Distinct Caspase-8 Activation Pathways
inferred← citeshttps://doi.org/10.1016/j.cellsig.2015.07.007openalex-snapshot
The mutual regulation between miR-214 and A2AR signaling plays an important role in inflammatory response
inferred← citeshttps://doi.org/10.1016/j.chempr.2016.07.007openalex-snapshot
Immunomodulatory Effects of Coated Gold Nanoparticles in LPS-Stimulated In Vitro and In Vivo Murine Model Systems
inferred← citeshttps://doi.org/10.1016/j.clim.2008.08.015openalex-snapshot
Gene-specific control of the TLR-induced inflammatory response
inferred← citeshttps://doi.org/10.1016/j.clim.2011.03.005openalex-snapshot
Tumor necrosis factor alpha (TNF-α) autoregulates its expression and induces adhesion molecule expression in asthma
inferred← citeshttps://doi.org/10.1016/j.compbiolchem.2023.107823openalex-snapshot
Computational Analyses of the Interactome between TNF and TNFR Superfamilies
inferred← citeshttps://doi.org/10.1016/j.copbio.2004.09.005openalex-snapshot
Tumour necrosis factor α as a therapeutic target for immune-mediated inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.coph.2004.04.003openalex-snapshot
Interleukin-15: a new cytokine target for the treatment of inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.coph.2004.05.003openalex-snapshot
New approaches to therapeutic immunomodulation for immune-mediated inflammatory disorders
inferred← citeshttps://doi.org/10.1016/j.cpcardiol.2021.100853openalex-snapshot
Triggers of Cardiovascular Diseases in Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.1016/j.csbj.2019.12.016openalex-snapshot
A computational model for understanding the oligomerization mechanisms of TNF receptor superfamily
inferred← citeshttps://doi.org/10.1016/j.cyto.2005.11.004openalex-snapshot
Hepatic steatosis in the absence of tumor necrosis factor in mice
inferred← citeshttps://doi.org/10.1016/j.dcmed.2021.09.003openalex-snapshot
In silico prediction of phytoconstituents from Ehretia laevis targeting TNF-α in arthritis
inferred← citeshttps://doi.org/10.1016/j.ddmec.2004.10.006openalex-snapshot
Rheumatoid arthritis: pathogenic mechanisms and therapeutic targets
inferred← citeshttps://doi.org/10.1016/j.ddmec.2006.06.009openalex-snapshot
The role of immune cells and cytokines in the pathogenesis of rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.drudis.2008.10.003openalex-snapshot
Drug discovery: new models for industry–academic partnerships
inferred← citeshttps://doi.org/10.1016/j.drudis.2012.05.007openalex-snapshot
Drug discovery in pharmaceutical industry: productivity challenges and trends
inferred← citeshttps://doi.org/10.1016/j.ejps.2014.09.012openalex-snapshot
Melittin–glutathione S-transferase fusion protein exhibits anti-inflammatory properties and minimal toxicity
inferred← citeshttps://doi.org/10.1016/j.ekir.2025.10.018openalex-snapshot
Infliximab for Grade III or IV Immune Checkpoint Inhibitor Nephritis Clinical and Translational Evidence
inferred← citeshttps://doi.org/10.1016/j.exer.2006.05.005openalex-snapshot
Possibility of inducing anterior chamber-associated immune deviation by TGF-β2 treatment of monocytes isolated from Behcet's patients
inferred← citeshttps://doi.org/10.1016/j.expneurol.2012.10.007openalex-snapshot
Astrocytes: Conductors of the Alzheimer disease neuroinflammatory symphony
inferred← citeshttps://doi.org/10.1016/j.febslet.2011.04.065openalex-snapshot
Progranulin: A promising therapeutic target for rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2021.12.004openalex-snapshot
Neuroinflammation-mediated mitochondrial dysregulation involved in postoperative cognitive dysfunction
inferred← citeshttps://doi.org/10.1016/j.heliyon.2024.e38074openalex-snapshot
Therapeutic potential of Leea asiatica: Chemical isolation and validation of ethnomedicinal claims through in vitro and in silico assessment of antioxidant and anti-inflammatory properties
inferred← citeshttps://doi.org/10.1016/j.iac.2017.01.002openalex-snapshot
Immune Mechanisms and Novel Targets in Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.1016/j.ijpharm.2022.121939openalex-snapshot
Hyaluronic acid coated teriflunomide (A771726) loaded lipid carriers for the oral management of rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.ijpharm.2024.124449openalex-snapshot
Melatonin hyalurosomes in collagen thermosensitive gel as a potential repurposing approach for rheumatoid arthritis management via the intra-articular route
inferred← citeshttps://doi.org/10.1016/j.imlet.2016.04.007openalex-snapshot
T-cell immune adaptor SKAP1 regulates the induction of collagen-induced arthritis in mice
inferred← citeshttps://doi.org/10.1016/j.immuni.2008.03.002openalex-snapshot
Cytokine Signaling Modules in Inflammatory Responses
inferred← citeshttps://doi.org/10.1016/j.intimp.2004.04.007openalex-snapshot
Lipopolysaccharide-induced paw edema model for detection of cytokine modulating anti-inflammatory agents
inferred← citeshttps://doi.org/10.1016/j.intimp.2006.12.001openalex-snapshot
The role of adenosine A2 receptors in the regulation of TNF-α production and PGE2 release in mouse peritoneal macrophages
inferred← citeshttps://doi.org/10.1016/j.intimp.2011.12.014openalex-snapshot
Ginsenoside Rd attenuates the inflammatory response via modulating p38 and JNK signaling pathways in rats with TNBS-induced relapsing colitis
inferred← citeshttps://doi.org/10.1016/j.intimp.2013.10.001openalex-snapshot
(E)-3-(3,4-Dimethoxyphenyl)-1-(5-hydroxy-2,2-dimethyl-2H-chromen-6-yl)prop-2-en-1-one ameliorates the collagen-arthritis via blocking ERK/JNK and NF-κB signaling pathway
inferred← citeshttps://doi.org/10.1016/j.intimp.2015.07.036openalex-snapshot
Anti-TNF-α monoclonal antibody reverses psoriasis through dual inhibition of inflammation and angiogenesis
inferred← citeshttps://doi.org/10.1016/j.jaci.2006.01.039openalex-snapshot
Understanding the pathophysiology of severe asthma to generate new therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/j.jaci.2021.05.030openalex-snapshot
Anti-TNF treatment corrects IFN-γ–dependent proinflammatory signatures in Blau syndrome patient–derived macrophages
inferred← citeshttps://doi.org/10.1016/j.jaut.2005.09.006openalex-snapshot
Anti-TNF therapy: Where have we got to in 2005?
inferred← citeshttps://doi.org/10.1016/j.jbspin.2007.08.005openalex-snapshot
The paradox of infection and inflammation: Friends or foes? The dilemma of biological usage in high endemic tuberculous areas
inferred← citeshttps://doi.org/10.1016/j.jbspin.2020.02.009openalex-snapshot
Important determinants of the patient choice between TNF- vs. non-TNF Biologic disease-modifying anti-rheumatic drugs (DMARDs) for active rheumatoid arthritis (RA)
inferred← citeshttps://doi.org/10.1016/j.jconrel.2008.04.021openalex-snapshot
Synthesis and characterization of a thermally-responsive tumor necrosis factor antagonist
inferred← citeshttps://doi.org/10.1016/j.jconrel.2014.04.027openalex-snapshot
Theranostic nanoparticles for future personalized medicine
inferred← citeshttps://doi.org/10.1016/j.jconrel.2020.11.041openalex-snapshot
RNA interference-mediated suppression of TNF-α converting enzyme as an alternative anti-TNF-α therapy for rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.jdent.2026.106630openalex-snapshot
Periodontal inflammation indices in association to systemic diseases
inferred← citeshttps://doi.org/10.1016/j.jep.2020.112938openalex-snapshot
Dereplication and quantification of the ethanol extract of Miconia albicans (Melastomaceae) by HPLC-DAD-ESI-/MS/MS, and assessment of its anti-hyperalgesic and anti-inflammatory profiles in a mice arthritis-like model: Evidence for involvement of TNF-α, IL-1β and IL-6
inferred← citeshttps://doi.org/10.1016/j.jff.2026.107195openalex-snapshot
Compositional characterization and immunomodulatory efficacy of konjac-derived manno-oligosaccharides in cyclophosphamide-induced immunosuppressed mice
inferred← citeshttps://doi.org/10.1016/j.jim.2008.02.019openalex-snapshot
Simple and highly sensitive assay system for TNFR2-mediated soluble- and transmembrane-TNF activity
inferred← citeshttps://doi.org/10.1016/j.jmb.2008.11.053openalex-snapshot
Structure–Function Relationship of Tumor Necrosis Factor (TNF) and Its Receptor Interaction Based on 3D Structural Analysis of a Fully Active TNFR1-Selective TNF Mutant
inferred← citeshttps://doi.org/10.1016/j.jmgm.2020.107774openalex-snapshot
Computational engineering the binding affinity of Adalimumab monoclonal antibody for designing potential biosimilar candidate
inferred← citeshttps://doi.org/10.1016/j.joca.2012.05.003openalex-snapshot
Enhanced COMP catabolism detected in serum of patients with arthritis and animal disease models through a novel capture ELISA
inferred← citeshttps://doi.org/10.1016/j.jphotobiol.2017.01.018openalex-snapshot
Characterization of the therapeutic properties of Chinese herbal materials by measuring delayed luminescence and dendritic cell-based immunomodulatory response
inferred← citeshttps://doi.org/10.1016/j.jsb.2012.02.002openalex-snapshot
Enriching the human apoptosis pathway by predicting the structures of protein–protein complexes
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.122247openalex-snapshot
Increased contractile activity and dilation of popliteal lymphatic vessels in the TNF-α-overexpressing TNFΔARE/+ arthritic mouse
inferred← citeshttps://doi.org/10.1016/j.medj.2020.11.005openalex-snapshot
The Potential for Repurposing Anti-TNF as a Therapy for the Treatment of COVID-19
inferred← citeshttps://doi.org/10.1016/j.molimm.2013.08.011openalex-snapshot
Human cathelicidin LL-37-derived peptide IG-19 confers protection in a murine model of collagen-induced arthritis
inferred← citeshttps://doi.org/10.1016/j.molstruc.2023.137165openalex-snapshot
Synthesis, X-ray crystallographic, Hirshfeld, DFT study and interactions with TNFα receptors of chiral bisimines and diamines holding thiophene spacers
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