On the public citation graph

Regulation of NMDA receptor trafficking by amyloid-β

https://doi.org/10.1038/nn1503
0 signed · 1535 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1001/jama.283.12.1571openalex-snapshot
Correlation Between Elevated Levels of Amyloid β-Peptide in the Brain and Cognitive Decline
inferredcites →https://doi.org/10.1002/ana.410300410openalex-snapshot
Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment
inferredcites →https://doi.org/10.1016/s0006-8993(99)01220-2openalex-snapshot
Impaired phosphorylation of cyclic AMP response element binding protein in the hippocampus of dementia of the Alzheimer type
inferredcites →https://doi.org/10.1016/s0028-3908(03)00308-3openalex-snapshot
Differential binding of the AP-2 adaptor complex and PSD-95 to the C-terminus of the NMDA receptor subunit NR2B regulates surface expression
inferredcites →https://doi.org/10.1016/s0896-6273(00)00089-1openalex-snapshot
Activity-Dependent Induction of Tonic Calcineurin Activity Mediates a Rapid Developmental Downregulation of NMDA Receptor Currents
inferredcites →https://doi.org/10.1016/s0896-6273(00)00129-xopenalex-snapshot
Reinsertion or Degradation of AMPA Receptors Determined by Activity-Dependent Endocytic Sorting
inferredcites →https://doi.org/10.1016/s0896-6273(00)80962-9openalex-snapshot
Activity Regulates the Synaptic Localization of the NMDA Receptor in Hippocampal Neurons
inferredcites →https://doi.org/10.1016/s0896-6273(00)81010-7openalex-snapshot
Ca2+ Influx Regulates BDNF Transcription by a CREB Family Transcription Factor-Dependent Mechanism
inferredcites →https://doi.org/10.1016/s0896-6273(00)81011-9openalex-snapshot
Identification of a Signaling Pathway Involved in Calcium Regulation of BDNF Expression
inferredcites →https://doi.org/10.1016/s0896-6273(02)00633-5openalex-snapshot
Tyrosine Phosphatase STEP Is a Tonic Brake on Induction of Long-Term Potentiation
inferredcites →https://doi.org/10.1016/s0896-6273(03)00124-7openalex-snapshot
APP Processing and Synaptic Function
inferredcites →https://doi.org/10.1038/369233a0openalex-snapshot
Regulation of NMDA receptors by tyrosine kinases and phosphatases
inferredcites →https://doi.org/10.1038/6374openalex-snapshot
Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice
inferredcites →https://doi.org/10.1038/88404openalex-snapshot
A use-dependent tyrosine dephosphorylation of NMDA receptors is independent of ion flux
inferredcites →https://doi.org/10.1038/90498openalex-snapshot
Molecular determinants of NMDA receptor internalization
inferredcites →https://doi.org/10.1038/90520openalex-snapshot
The consolidation of new but not reactivated memory requires hippocampal C/EBPβ
inferredcites →https://doi.org/10.1038/nature01497openalex-snapshot
Glycine binding primes NMDA receptor internalization
inferredcites →https://doi.org/10.1038/nn1013openalex-snapshot
Activity level controls postsynaptic composition and signaling via the ubiquitin-proteasome system
inferredcites →https://doi.org/10.1038/nn746openalex-snapshot
Internalization of ionotropic glutamate receptors in response to mGluR activation
inferredcites →https://doi.org/10.1038/nn989openalex-snapshot
NMDA-mediated activation of the tyrosine phosphatase STEP regulates the duration of ERK signaling
inferredcites →https://doi.org/10.1073/pnas.0408305102openalex-snapshot
Regulation of a protein phosphatase cascade allows convergent dopamine and glutamate signals to activate ERK in the striatum
inferredcites →https://doi.org/10.1074/jbc.275.8.5626openalex-snapshot
β-Amyloid1–42 Binds to α7 Nicotinic Acetylcholine Receptor with High Affinity
inferredcites →https://doi.org/10.1074/jbc.m010450200openalex-snapshot
β-Amyloid-(1–42) Impairs Activity-dependent cAMP-response Element-binding Protein Signaling in Neurons at Concentrations in Which Cell Survival Is Not Compromised
inferredcites →https://doi.org/10.1074/jbc.m209065200openalex-snapshot
Presenilin-1 Regulates Intracellular Trafficking and Cell Surface Delivery of β-Amyloid Precursor Protein
inferredcites →https://doi.org/10.1074/jbc.m210167200openalex-snapshot
Tyrosine Dephosphorylation and Ethanol Inhibition of N-Methyl-d-aspartate Receptor Function
inferredcites →https://doi.org/10.1126/science.1072994openalex-snapshot
The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics
inferredcites →https://doi.org/10.1126/science.1074069openalex-snapshot
Alzheimer's Disease Is a Synaptic Failure
inferredcites →https://doi.org/10.1126/science.285.5433.1569openalex-snapshot
In Vivo Protein Transduction: Delivery of a Biologically Active Protein into the Mouse
inferredcites →https://doi.org/10.1126/science.286.5448.2358openalex-snapshot
Mediation by a CREB Family Transcription Factor of NGF-Dependent Survival of Sympathetic Neurons
inferredcites →https://doi.org/10.1146/annurev.biochem.68.1.821openalex-snapshot
CREB: A Stimulus-Induced Transcription Factor Activated by A Diverse Array of Extracellular Signals
inferredcites →https://doi.org/10.1152/jn.2001.85.2.708openalex-snapshot
Blockade of Long-Term Potentiation by β-Amyloid Peptides in the CA1 Region of the Rat Hippocampus In Vivo
inferredcites →https://doi.org/10.1196/annals.1300.001openalex-snapshot
Synaptic Plasticity and AMPA Receptor Trafficking
inferredcites →https://doi.org/10.1523/jneurosci.0780-04.2004openalex-snapshot
Endocytosis and Degradative Sorting of NMDA Receptors by Conserved Membrane-Proximal Signals
inferredcites →https://doi.org/10.1523/jneurosci.17-19-07351.1997openalex-snapshot
Redistribution and Stabilization of Cell Surface Glutamate Receptors during Synapse Formation
inferredcites →https://doi.org/10.1523/jneurosci.21-04-01327.2001openalex-snapshot
Use-Dependent Effects of Amyloidogenic Fragments of β-Amyloid Precursor Protein on Synaptic Plasticity in Rat HippocampusIn Vivo
inferredcites →https://doi.org/10.1523/jneurosci.21-12-04125.2001openalex-snapshot
Beta-amyloid activates the mitogen-activated protein kinase cascade via hippocampal alpha7 nicotinic acetylcholine receptors: In vitro and in vivo mechanisms related to Alzheimer's disease.
inferredcites →https://doi.org/10.1523/jneurosci.22-12-05001.2002openalex-snapshot
α7 Nicotinic Acetylcholine Receptors Occur at Postsynaptic Densities of AMPA Receptor-Positive and -Negative Excitatory Synapses in Rat Sensory Cortex
inferredcites →https://doi.org/10.1523/jneurosci.23-31-10093.2003openalex-snapshot
Neuroprotection by Nicotine in Mouse Primary Cortical Cultures Involves Activation of Calcineurin and L-Type Calcium Channel Inactivation
inferredcites →https://doi.org/10.1523/jneurosci.5167-03.2004openalex-snapshot
Oligomerization of Alzheimer's β-Amyloid within Processes and Synapses of Cultured Neurons and Brain

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.5281/zenodo.8515openalex-extracted
Stimulating Neuroprotective and Regenerative Mechanisms in Alzheimer Disease
inferred← citeshttps://doi.org/10.1002/0471142301.ns0116s42openalex-snapshot
Biochemical Fractionation of Brain Tissue for Studies of Receptor Distribution and Trafficking
inferred← citeshttps://doi.org/10.1002/2211-5463.12773openalex-snapshot
Mapping pathogenic processes contributing to neurodegeneration in Drosophila models of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/9780470669600.ch50openalex-snapshot
Neurochemistry of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/9781118395318.ch30openalex-snapshot
Leptin and Cognitive Function
inferred← citeshttps://doi.org/10.1002/9781118553480.ch07openalex-snapshot
Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/9783527628346.ch16openalex-snapshot
Stress and Neurodegeneration: Adding Insult to Injury?
inferred← citeshttps://doi.org/10.1002/9783527634323.ch9openalex-snapshot
Impaired Regulation of Glutamate Receptor Channels and Signaling Molecules by β‐Amyloid in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/9783527654185.ch7openalex-snapshot
The Oligomer Species: Mechanistics and Biochemistry
inferred← citeshttps://doi.org/10.1002/adtp.202000225openalex-snapshot
Naphthalene Monoimide Derivative Ameliorates Amyloid Burden and Cognitive Decline in a Transgenic Mouse Model of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/alz.12302openalex-snapshot
Preclinical and clinical biomarker studies of CT1812: A novel approach to Alzheimer's disease modification
inferred← citeshttps://doi.org/10.1002/ana.24235openalex-snapshot
Myeloid microvesicles in cerebrospinal fluid are associated with myelin damage and neuronal loss in mild cognitive impairment and A lzheimer disease
inferred← citeshttps://doi.org/10.1002/ange.200702399openalex-snapshot
Cleavage Agents for Soluble Oligomers of Amyloid β Peptides
inferred← citeshttps://doi.org/10.1002/ange.200802808openalex-snapshot
Die Alzheimer‐Demenz: von der Pathologie zu therapeutischen Ansätzen
inferred← citeshttps://doi.org/10.1002/anie.200702399openalex-snapshot
Cleavage Agents for Soluble Oligomers of Amyloid β Peptides
inferred← citeshttps://doi.org/10.1002/anie.200802808openalex-snapshot
Alzheimer's Disease: From Pathology to Therapeutic Approaches
inferred← citeshttps://doi.org/10.1002/bab.2739openalex-snapshot
trans‐Ferulic Acid Antagonizes the Anti‐Inflammatory Activity of Etoricoxib: Possible Interaction of COX‐1 and NOS
inferred← citeshttps://doi.org/10.1002/bies.201100193openalex-snapshot
Towards Alzheimer's root cause: ECSIT as an integrating hub between oxidative stress, inflammation and mitochondrial dysfunction
inferred← citeshttps://doi.org/10.1002/bies.201500004openalex-snapshot
Neuronal hyperactivity – A key defect in Alzheimer's disease?
inferred← citeshttps://doi.org/10.1002/cbic.200700351openalex-snapshot
Ligand‐Induced Flocculation of Neurotoxic Fibrillar Aβ(1–42) by Noncovalent Crosslinking
inferred← citeshttps://doi.org/10.1002/cmdc.201900299openalex-snapshot
Allosteric Modulators of Potential Targets Related to Alzheimer's Disease: a Review
inferred← citeshttps://doi.org/10.1002/cne.21176openalex-snapshot
Spatial relationship between synapse loss and β‐amyloid deposition in Tg2576 mice
inferred← citeshttps://doi.org/10.1002/cne.23306openalex-snapshot
In vivo characterization of a bigenic fluorescent mouse model of Alzheimer's disease with neurodegeneration
inferred← citeshttps://doi.org/10.1002/cne.23336openalex-snapshot
In vivo characterization of a bigenic fluorescent mouse model of Alzheimer's disease with neurodegeneration
inferred← citeshttps://doi.org/10.1002/cne.23536openalex-snapshot
Early fear memory defects are associated with altered synaptic plasticity and molecular architecture in the TgCRND8 Alzheimer's disease mouse model
inferred← citeshttps://doi.org/10.1002/ddr.20046openalex-snapshot
Alzheimer's disease, estrogens, and clinical trials: a case study in drug development for complex disorders
inferred← citeshttps://doi.org/10.1002/dneu.20778openalex-snapshot
Toward a comprehensive neurobiology of IGF‐I
inferred← citeshttps://doi.org/10.1002/elps.200900647openalex-snapshot
Profiling of mouse synaptosome proteome and phosphoproteome by IEF
inferred← citeshttps://doi.org/10.1002/emmm.201000082openalex-snapshot
Prion protein and Aβ‐related synaptic toxicity impairment
inferred← citeshttps://doi.org/10.1002/epi4.12718openalex-snapshot
In the fast lane: Receptor trafficking during status epilepticus
inferred← citeshttps://doi.org/10.1002/glia.22548openalex-snapshot
β-amyloid and ATP-induced diffusional trapping of astrocyte and neuronal metabotropic glutamate type-5 receptors
inferred← citeshttps://doi.org/10.1002/gps.3879openalex-snapshot
Neurodegeneration, β‐amyloid and mood disorders: state of the art and future perspectives
inferred← citeshttps://doi.org/10.1002/ijch.201600103openalex-snapshot
Oligomer Formation and Cross‐Seeding: The New Frontier
inferred← citeshttps://doi.org/10.1002/iub.308openalex-snapshot
Role of the SNK‐SPAR pathway in the development of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/iub.91openalex-snapshot
Calcium ions in neuronal degeneration
inferred← citeshttps://doi.org/10.1002/jcp.24284openalex-snapshot
Amyloid toxicity and platelet‐activating factor signaling
inferred← citeshttps://doi.org/10.1002/jcp.30646openalex-snapshot
The role of trimethylamine‐N‐Oxide in the development of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/jnr.20952openalex-snapshot
Absence of synaptophysin near cortical neurons containing oligomer Aβ in Alzheimer's disease brain
inferred← citeshttps://doi.org/10.1002/jnr.21998openalex-snapshot
NMDA receptor–dependent signaling pathways that underlie amyloid β‐protein disruption of LTP in the hippocampus
inferred← citeshttps://doi.org/10.1002/jnr.22227openalex-snapshot
Decreased levels of PSD95 and two associated proteins and increased levels of BCl 2 and caspase 3 in hippocampus from subjects with amnestic mild cognitive impairment: Insights into their potential roles for loss of synapses and memory, accumulation of Aβ, and neurodegeneration in a prodromal…
inferred← citeshttps://doi.org/10.1002/jnr.22278openalex-snapshot
The galanin receptor 2/3 agonist Gal2‐11 protects the SN56 cells against β‐amyloid25–35 toxicity
inferred← citeshttps://doi.org/10.1002/jnr.22445openalex-snapshot
Amyloid‐β oligomers impair fear conditioned memory in a calcineurin‐dependent fashion in mice
inferred← citeshttps://doi.org/10.1002/jnr.23263openalex-snapshot
Tyrosine phosphatase STEP61 negatively regulates amyloid β‐mediated ERK/CREB signaling pathways via α7 nicotinic acetylcholine receptors
inferred← citeshttps://doi.org/10.1002/jnr.70046openalex-snapshot
Alzheimer's Disease and Frontotemporal Dementia: A Review of Pathophysiology and Therapeutic Approaches
inferred← citeshttps://doi.org/10.1002/mds.26624openalex-snapshot
Oligomeropathies and pathogenesis of Alzheimer and Parkinson's diseases
inferred← citeshttps://doi.org/10.1002/med.20248openalex-snapshot
Recent advances in the multitarget‐directed ligands approach for the treatment of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/prot.21232openalex-snapshot
Molecular dynamics studies of hexamers of amyloid‐β peptide (16–35) and its mutants: Influence of charge states on amyloid formation
inferred← citeshttps://doi.org/10.1002/sita.200600098openalex-snapshot
Yin and Yang in BMP signaling: Impact on the pathology of diseases and potential for tissue regeneration
inferred← citeshttps://doi.org/10.1002/smll.202506572openalex-snapshot
Multimodal Therapeutic Approach Using Donor‐Engineered AIE Photosensitizer to Combat Multifaceted Aspects of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/stem.296openalex-snapshot
Neurons Generated from APP/APLP1/APLP2 Triple Knockout Embryonic Stem Cells Behave Normally in Vitro and in Vivo: Lack of Evidence for a Cell Autonomous Role of the Amyloid Precursor Protein in Neuronal Differentiation
inferred← citeshttps://doi.org/10.1002/syn.20608openalex-snapshot
Knockout of STriatal enriched protein tyrosine phosphatase in mice results in increased ERK1/2 phosphorylation
inferred← citeshttps://doi.org/10.1002/syn.21770openalex-snapshot
Unilateral injection of Aβ25–35in the hippocampus reduces the number of dendritic spines in hyperglycemic rats
inferred← citeshttps://doi.org/10.1002/syn.21915openalex-snapshot
Calcium channel blockade attenuates abnormal synaptic transmission in the dentate gyrus elicited by entorhinal amyloidopathy
inferred← citeshttps://doi.org/10.1002/wsbm.1262openalex-snapshot
Apolipoprotein E receptor pathways in Alzheimer disease
inferred← citeshttps://doi.org/10.1007/7355_2010_11openalex-snapshot
Glutamate and Neurodegenerative Disease
inferred← citeshttps://doi.org/10.1007/978-0-387-35135-3_4openalex-snapshot
β-secretase: Physiological Role and Target Validation
inferred← citeshttps://doi.org/10.1007/978-0-387-35135-3_9openalex-snapshot
Beta Amyloid and Excitatory Synapses
inferred← citeshttps://doi.org/10.1007/978-0-387-71522-3_6openalex-snapshot
The Rationale for Glutamatergic Therapy in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/978-0-387-73023-3_8openalex-snapshot
Glutamate Receptors and Neurological Disorders
inferred← citeshttps://doi.org/10.1007/978-0-387-76761-1_2openalex-snapshot
Apolipoprotein E Receptor 2 and Very-Low-Density Lipoprotein Receptor: An Overview
inferred← citeshttps://doi.org/10.1007/978-0-387-77232-5_12openalex-snapshot
Molecular Properties and Cell Biology of the NMDA Receptor
inferred← citeshttps://doi.org/10.1007/978-0-387-77232-5_8openalex-snapshot
Dendritic Organelles for Postsynaptic Trafficking
inferred← citeshttps://doi.org/10.1007/978-0-387-87995-6_2openalex-snapshot
Pore-Forming Neurotoxin-Like Mechanism for Aβ Oligomer-Induced Synaptic Failure
inferred← citeshttps://doi.org/10.1007/978-1-0716-2589-7_19openalex-snapshot
Measurement of Silent Synapses
inferred← citeshttps://doi.org/10.1007/978-1-4020-6191-2_19openalex-snapshot
Role of Calcium in the Pathogenesis of Alzheimer’s Disease and Transgenic Models
inferred← citeshttps://doi.org/10.1007/978-1-4419-0461-4_630openalex-snapshot
Striatal-Enriched Protein Tyrosine Phosphatase (STEP)
inferred← citeshttps://doi.org/10.1007/978-1-4419-6382-6_10openalex-snapshot
Protein Phosphatases in the Brain: Regulation, Function and Disease
inferred← citeshttps://doi.org/10.1007/978-1-4419-7061-9_14openalex-snapshot
Synapses and Alzheimers’s Disease: Effect of Immunotherapy?
inferred← citeshttps://doi.org/10.1007/978-1-4419-9663-3_30openalex-snapshot
Copper and Alzheimer Disease: The Good, the Bad and the Ugly
inferred← citeshttps://doi.org/10.1007/978-1-4614-4756-6_37openalex-snapshot
Nonselective Cation Channels and Links to Hippocampal Ischemia, Aging, and Dementia
inferred← citeshttps://doi.org/10.1007/978-1-4614-6438-9_630-1openalex-snapshot
Striatal-Enriched Protein-Tyrosine Phosphatase (STEP)
inferred← citeshttps://doi.org/10.1007/978-1-4939-3274-0_19openalex-snapshot
Measurement of Silent Synapses
inferred← citeshttps://doi.org/10.1007/978-1-4939-3560-4_3openalex-snapshot
Immunotherapy Against N-Truncated Amyloid-β Oligomers
inferred← citeshttps://doi.org/10.1007/978-1-4939-3768-4_9openalex-snapshot
α7-Nicotinic Acetylcholine Receptors: New Therapeutic Avenues in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-8739-9_14openalex-snapshot
Profiling of MicroRNA and Protein from Purified Synaptoneurosomes in a Neurodegenerative Disease Model
inferred← citeshttps://doi.org/10.1007/978-1-4939-9944-6_4openalex-snapshot
A Novel Preclinical Rat Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-60761-898-0_17openalex-snapshot
Aβ Infusion and Related Models of Alzheimer Dementia
inferred← citeshttps://doi.org/10.1007/978-1-60761-898-0_21openalex-snapshot
Presenilin-Based Transgenic Models of Alzheimer’s Dementia
inferred← citeshttps://doi.org/10.1007/978-1-60761-898-0_24openalex-snapshot
The 3xTg-AD Mouse Model: Reproducing and Modulating Plaque and Tangle Pathology
inferred← citeshttps://doi.org/10.1007/978-1-62703-562-0_13openalex-snapshot
Protein Tyrosine Phosphatases: Structure, Function, and Implication in Human Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-12457-1_39openalex-snapshot
Calcium Signaling During Brain Aging and Its Influence on the Hippocampal Synaptic Plasticity
inferred← citeshttps://doi.org/10.1007/978-3-030-38266-7_8openalex-snapshot
The Ubiquitin System in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-42667-5_10openalex-snapshot
The Challenge of Antidepressant Therapeutics in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-49844-3_11openalex-snapshot
Alzheimer’s Disease and Protein Kinases
inferred← citeshttps://doi.org/10.1007/978-3-031-68550-7_15openalex-snapshot
Perturbations to Stimulus-Dependent Gene Activity Patterns in Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1007/978-3-031-68550-7_4openalex-snapshot
Gene Regulation by Nuclear Calcium and Its Antagonism by NMDAR/TRPM4 Signaling
inferred← citeshttps://doi.org/10.1007/978-3-319-16522-6_12openalex-snapshot
Assessing Cognitive Improvement in People with Down Syndrome: Important Considerations for Drug-Efficacy Trials
inferred← citeshttps://doi.org/10.1007/978-3-319-67199-4_630openalex-snapshot
Striatal-Enriched Protein-Tyrosine Phosphatase (STEP)
inferred← citeshttps://doi.org/10.1007/978-3-540-35109-2_17openalex-snapshot
Conditional Animal Models for the Study of Lipid Metabolism and Lipid Disorders
inferred← citeshttps://doi.org/10.1007/978-3-540-45702-2_11openalex-snapshot
Molecules that Disrupt Memory Circuits in Alzheimer’s Disease: The Attack on Synapses by Aβ Oligomers (ADDLs)
inferred← citeshttps://doi.org/10.1007/978-3-540-76330-7_5openalex-snapshot
Impact of Beta Amyloid on Excitatory Synaptic Transmission and Plasticity
inferred← citeshttps://doi.org/10.1007/978-3-540-76330-7_7openalex-snapshot
Multiple Levels of Synaptic Regulation by NMDA-type Glutamate Receptor in Normal and Disease States
inferred← citeshttps://doi.org/10.1007/978-3-540-76330-7_8openalex-snapshot
Soluble Oligomers of the Amyloid β-Protein: Impair Synaptic Plasticity and Behavior
inferred← citeshttps://doi.org/10.1007/978-3-540-76330-7_9openalex-snapshot
Why Alzheimer’s is a Disease of Memory: Synaptic Targeting by Pathogenic Aβ Oligomers (ADDLs)
inferred← citeshttps://doi.org/10.1007/978-3-642-21649-7_12openalex-snapshot
Taking STEPs Forward to Understand Fragile X Syndrome
inferred← citeshttps://doi.org/10.1007/978-3-642-22144-6_178openalex-snapshot
Prevention of Brain Disorders by Nicotine
inferred← citeshttps://doi.org/10.1007/978-3-642-30574-0_52openalex-snapshot
Integrating Data for Modeling Biological Complexity
inferred← citeshttps://doi.org/10.1007/978-3-642-36202-6_178-1openalex-snapshot
Nicotine: A Possible Neuroprotector in Brain Disorders
inferred← citeshttps://doi.org/10.1007/978-3-7091-0932-8_25openalex-snapshot
Synaptic Dysfunction in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-4-431-56550-5_12openalex-snapshot
Drebrin in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-94-007-2774-8_4openalex-snapshot
Biological Targeting and Activity of Pre-fibrillar Aβ Assemblies
inferred← citeshttps://doi.org/10.1007/978-94-007-2774-8_5openalex-snapshot
The Role of Aβ and Tau Oligomers in the Pathogenesis of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-94-024-1088-4_7openalex-snapshot
TRPC Channels and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-10-8854-4_31openalex-snapshot
Influence of β-Amyloid Plaques on the Local Network Activity in the APP/PS1 Mouse Model of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-13-0529-0_3openalex-snapshot
Alzheimer’s Disease Model System Using Drosophila
inferred← citeshttps://doi.org/10.1007/978-981-15-1792-1_30openalex-snapshot
Effects of Exercise on Long-Term Potentiation in Neuropsychiatric Disorders
inferred← citeshttps://doi.org/10.1007/978-981-96-0259-9_4openalex-snapshot
Pathophysiology and Underlying Mechanisms: Pathways to Alzheimer’s Plaque Formation
inferred← citeshttps://doi.org/10.1007/bf02982588openalex-snapshot
The amyloid cascade: The old and the new
inferred← citeshttps://doi.org/10.1007/s00018-008-8271-3openalex-snapshot
Understanding BACE1: essential protease for amyloid-β production in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00018-011-0750-2openalex-snapshot
Modes of Aβ toxicity in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00018-014-1706-0openalex-snapshot
Amyloid beta receptors responsible for neurotoxicity and cellular defects in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00018-015-2057-1openalex-snapshot
Inhibition of the tyrosine phosphatase STEP61 restores BDNF expression and reverses motor and cognitive deficits in phencyclidine-treated mice
inferred← citeshttps://doi.org/10.1007/s00018-018-2825-9openalex-snapshot
Impact of late-onset Alzheimer’s genetic risk factors on beta-amyloid endocytic production
inferred← citeshttps://doi.org/10.1007/s00018-020-03464-4openalex-snapshot
Misfolded amyloid-β-42 impairs the endosomal–lysosomal pathway
inferred← citeshttps://doi.org/10.1007/s00018-023-05028-8openalex-snapshot
STIM2 regulates NMDA receptor endocytosis that is induced by short-term NMDA receptor overactivation in cortical neurons
inferred← citeshttps://doi.org/10.1007/s00018-024-05357-2openalex-snapshot
VEGFD signaling balances stability and activity-dependent structural plasticity of dendrites
inferred← citeshttps://doi.org/10.1007/s00018-025-05691-zopenalex-snapshot
Early involvement of D-serine in β-amyloid-dependent pathophysiology
inferred← citeshttps://doi.org/10.1007/s00213-014-3526-1openalex-snapshot
Green tea extract enhances parieto-frontal connectivity during working memory processing
inferred← citeshttps://doi.org/10.1007/s00213-017-4775-6openalex-snapshot
Xanthoceraside modulates NR2B-containing NMDA receptors at synapses and rescues learning-memory deficits in APP/PS1 transgenic mice
inferred← citeshttps://doi.org/10.1007/s00221-020-06028-xopenalex-snapshot
STEP inhibition prevents Aβ-mediated damage in dendritic complexity and spine density in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00232-025-00341-8openalex-snapshot
Role of Ion Channels in Alzheimer’s Disease Pathophysiology
inferred← citeshttps://doi.org/10.1007/s00249-007-0237-0openalex-snapshot
How is protein aggregation in amyloidogenic diseases modulated by biological membranes?
inferred← citeshttps://doi.org/10.1007/s00401-010-0679-9openalex-snapshot
Intraneuronal β-amyloid accumulation and synapse pathology in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00401-010-0787-6openalex-snapshot
In vivo multiphoton imaging reveals gradual growth of newborn amyloid plaques over weeks
inferred← citeshttps://doi.org/10.1007/s00401-014-1287-xopenalex-snapshot
Focusing the amyloid cascade hypothesis on N-truncated Abeta peptides as drug targets against Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00401-014-1371-2openalex-snapshot
Tau aggregation and its interplay with amyloid-β
inferred← citeshttps://doi.org/10.1007/s00401-015-1499-8openalex-snapshot
RCAN1 overexpression promotes age-dependent mitochondrial dysregulation related to neurodegeneration in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00424-009-0736-1openalex-snapshot
Calcium hypothesis of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00429-013-0518-6openalex-snapshot
Spine pruning in 5xFAD mice starts on basal dendrites of layer 5 pyramidal neurons
inferred← citeshttps://doi.org/10.1007/s00702-013-1125-7openalex-snapshot
Potential involvement of GRIN2B encoding the NMDA receptor subunit NR2B in the spectrum of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00775-009-0600-yopenalex-snapshot
Copper in the brain and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s10059-011-0077-8openalex-snapshot
Pathway-Specific Alteration of Synaptic Plasticity in Tg2576 Mice
inferred← citeshttps://doi.org/10.1007/s10059-013-0113-yopenalex-snapshot
NMDA Receptor as a Newly Identified Member of the Metabotropic Glutamate Receptor Family: Clinical Implications for Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/s10072-016-2546-5openalex-snapshot
Dysfunction of NMDA receptors in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s10571-007-9235-0openalex-snapshot
Carnosine Protects Against Aβ42-induced Neurotoxicity in Differentiated Rat PC12 Cells
inferred← citeshttps://doi.org/10.1007/s10571-011-9691-4openalex-snapshot
Aβ Oligomer-Induced Synapse Degeneration in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s10571-011-9751-9openalex-snapshot
Donepezil in a Narrow Concentration Range Augments Control and Impaired by Beta-Amyloid Peptide Hippocampal LTP in NMDAR-Independent Manner
inferred← citeshttps://doi.org/10.1007/s10571-014-0051-zopenalex-snapshot
Simvastatin Treatment Enhances NMDAR-Mediated Synaptic Transmission by Upregulating the Surface Distribution of the GluN2B Subunit
inferred← citeshttps://doi.org/10.1007/s10571-023-01364-2openalex-snapshot
Uncovering the Significance of STEP61 in Alzheimer’s Disease: Structure, Substrates, and Interactome
inferred← citeshttps://doi.org/10.1007/s10571-025-01559-9openalex-snapshot
Let-7 Family as a Mediator of Exercise on Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s11011-019-00516-yopenalex-snapshot
Mechanisms of action of amyloid-beta and its precursor protein in neuronal cell death
inferred← citeshttps://doi.org/10.1007/s11064-007-9469-8openalex-snapshot
Delineating the Mechanism of Alzheimer’s Disease Aβ Peptide Neurotoxicity
inferred← citeshttps://doi.org/10.1007/s11064-007-9497-4openalex-snapshot
The Place of Choline Acetyltransferase Activity Measurement in the “Cholinergic Hypothesis” of Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/s11064-008-9731-8openalex-snapshot
Chronic NMDA Administration Increases Neuroinflammatory Markers in Rat Frontal Cortex: Cross-Talk Between Excitotoxicity and Neuroinflammation
inferred← citeshttps://doi.org/10.1007/s11064-009-9960-5openalex-snapshot
Dysregulation of Calcium Homeostasis in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s11064-010-0371-4openalex-snapshot
Calcium Signalling and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s11064-016-1930-0openalex-snapshot
Ginsenoside Rd Protects Against Cerebral Ischemia–Reperfusion Injury Via Decreasing the Expression of the NMDA Receptor 2B Subunit and its Phosphorylated Product
inferred← citeshttps://doi.org/10.1007/s11064-022-03649-2openalex-snapshot
Shati/Nat8l Overexpression Improves Cognitive Decline by Upregulating Neuronal Trophic Factor in Alzheimer’s Disease Model Mice
inferred← citeshttps://doi.org/10.1007/s11064-022-03699-6openalex-snapshot
Aß Pathology and Neuron–Glia Interactions: A Synaptocentric View
inferred← citeshttps://doi.org/10.1007/s11357-016-9913-2openalex-snapshot
Higher levels of phosphorylated Y1472 on GluN2B subunits in the frontal cortex of aged mice are associated with good spatial reference memory, but not cognitive flexibility
inferred← citeshttps://doi.org/10.1007/s11357-026-02492-5openalex-snapshot
Calcium dysregulation in neurovascular function in Alzheimer’s disease: mechanisms, therapeutic targets
inferred← citeshttps://doi.org/10.1007/s11434-014-0529-6openalex-snapshot
The neuroprotective effect of L-Theanine and its inhibition on nicotine dependence
inferred← citeshttps://doi.org/10.1007/s11515-016-1425-zopenalex-snapshot
Neuroprotective strategies for NMDAR-mediated excitotoxicity in Huntington’s Disease
inferred← citeshttps://doi.org/10.1007/s11906-022-01209-1openalex-snapshot
WNT/β-catenin Pathway: a Possible Link Between Hypertension and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12013-008-9033-4openalex-snapshot
Disordered Proteins: Biological Membranes as Two-Dimensional Aggregation Matrices
inferred← citeshttps://doi.org/10.1007/s12017-007-8009-7openalex-snapshot
NR1 Knockdown Reveals CA1 Injuryduring a Developmental Period of High Seizure Susceptibility Despite Reduced Seizure Activity
inferred← citeshttps://doi.org/10.1007/s12017-008-8052-zopenalex-snapshot
Botanical Phenolics and Brain Health
inferred← citeshttps://doi.org/10.1007/s12017-009-8091-0openalex-snapshot
Alzheimer’s Disease Amyloid β-Protein and Synaptic Function
inferred← citeshttps://doi.org/10.1007/s12017-009-8097-7openalex-snapshot
Synaptic Depression and Aberrant Excitatory Network Activity in Alzheimer’s Disease: Two Faces of the Same Coin?
inferred← citeshttps://doi.org/10.1007/s12017-010-8141-7openalex-snapshot
APP Transgenic Mice: Their Use and Limitations
inferred← citeshttps://doi.org/10.1007/s12017-013-8257-7openalex-snapshot
SUMO and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12017-015-8355-9openalex-snapshot
Neuronal Network Oscillations in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/s12031-009-9237-0openalex-snapshot
Nicotinic Receptors, Amyloid-β, and Synaptic Failure in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12031-021-01918-3openalex-snapshot
Aplysia Neurons as a Model of Alzheimer’s Disease: Shared Genes and Differential Expression
inferred← citeshttps://doi.org/10.1007/s12035-008-8018-zopenalex-snapshot
Dendritic Spine Loss and Synaptic Alterations in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-011-8177-1openalex-snapshot
Mechanisms of Neural and Behavioral Dysfunction in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-012-8251-3openalex-snapshot
Aβ Toxicity in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/s12035-012-8253-1openalex-snapshot
Isoprenoids and Related Pharmacological Interventions: Potential Application in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-012-8261-1openalex-snapshot
Integrating Cytosolic Phospholipase A2 with Oxidative/Nitrosative Signaling Pathways in Neurons: A Novel Therapeutic Strategy for AD
inferred← citeshttps://doi.org/10.1007/s12035-012-8324-3openalex-snapshot
Targeting Synaptic Dysfunction in Alzheimer’s Disease Therapy
inferred← citeshttps://doi.org/10.1007/s12035-013-8627-zopenalex-snapshot
Protein Prenylation and Synaptic Plasticity: Implications for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-014-9053-6openalex-snapshot
Mechanism of Oxidative Stress and Synapse Dysfunction in the Pathogenesis of Alzheimer’s Disease: Understanding the Therapeutics Strategies
inferred← citeshttps://doi.org/10.1007/s12035-015-9143-0openalex-snapshot
The Essential Role of Soluble Aβ Oligomers in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-015-9335-7openalex-snapshot
BDNF Induces Striatal-Enriched Protein Tyrosine Phosphatase 61 Degradation Through the Proteasome
inferred← citeshttps://doi.org/10.1007/s12035-015-9459-9openalex-snapshot
The Role of Reelin Signaling in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12035-016-0082-1openalex-snapshot
Guanosine Prevents Anhedonic-Like Behavior and Impairment in Hippocampal Glutamate Transport Following Amyloid-β1–40 Administration in Mice
inferred← citeshttps://doi.org/10.1007/s12035-016-0222-7openalex-snapshot
Increased Src Family Kinase Activity Disrupts Excitatory Synaptic Transmission and Impairs Remote Fear Memory in Forebrain Shp2-Deficient Mice
inferred← citeshttps://doi.org/10.1007/s12035-016-9745-1openalex-snapshot
Posttranslational Modifications Regulate the Postsynaptic Localization of PSD-95
inferred← citeshttps://doi.org/10.1007/s12035-017-0555-xopenalex-snapshot
Synaptic NMDA Receptor Activation Induces Ubiquitination and Degradation of STEP61
inferred← citeshttps://doi.org/10.1007/s12035-018-0980-5openalex-snapshot
Altered Intracellular Calcium Homeostasis Underlying Enhanced Glutamatergic Transmission in Striatal-Enriched Tyrosine Phosphatase (STEP) Knockout Mice
inferred← citeshttps://doi.org/10.1007/s12035-019-01707-5openalex-snapshot
Avenanthramide-C Restores Impaired Plasticity and Cognition in Alzheimer’s Disease Model Mice
inferred← citeshttps://doi.org/10.1007/s12035-023-03487-5openalex-snapshot
Role of Non-Receptor-Type Tyrosine Phosphatases in Brain-Related Diseases
inferred← citeshttps://doi.org/10.1007/s12035-023-03671-7openalex-snapshot
Current Insights of Nanocarrier-Mediated Gene Therapeutics to Treat Potential Impairment of Amyloid Beta Protein and Tau Protein in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12264-013-1383-2openalex-snapshot
Dysregulation of synaptic and extrasynaptic N-methyl-D-aspartate receptors induced by amyloid-β
inferred← citeshttps://doi.org/10.1007/s12264-014-1504-6openalex-snapshot
Contribution of NMDA receptors to dorsolateral prefrontal cortical networks in primates
inferred← citeshttps://doi.org/10.1007/s12264-021-00716-6openalex-snapshot
Illuminating Neural Circuits in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12640-013-9407-2openalex-snapshot
Neurotoxic Saboteurs: Straws that Break the Hippo’s (Hippocampus) Back Drive Cognitive Impairment and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12640-013-9440-1openalex-snapshot
1MeTIQ Provides Protection Against Aβ-Induced Reduction of Surface Expression of Synaptic Proteins and Inhibits H2O2-Induced Oxidative Stress in Primary Hippocampal Neurons
inferred← citeshttps://doi.org/10.1007/s12640-019-00053-7openalex-snapshot
Creatine as a Neuroprotector: an Actor that Can Play Many Parts
inferred← citeshttps://doi.org/10.1007/s12640-019-00116-9openalex-snapshot
β-Amyloid Peptide: the Cell Compartment Multi-faceted Interaction in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s13167-021-00261-2openalex-snapshot
Integrative genomic analysis of PPP3R1 in Alzheimer’s disease: a potential biomarker for predictive, preventive, and personalized medical approach
inferred← citeshttps://doi.org/10.1007/s13311-019-00782-9openalex-snapshot
Cell-Autonomous and Non-cell-Autonomous Pathogenic Mechanisms in Huntington's Disease: Insights from In Vitro and In Vivo Models
inferred← citeshttps://doi.org/10.1007/s13311-023-01356-6openalex-snapshot
Ferulic Acid Improves Synaptic Plasticity and Cognitive Impairments by Alleviating the PP2B/DARPP-32/PP1 Axis-Mediated STEP Increase and Aβ Burden in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/s13311-023-01405-0openalex-snapshot
Anti-Amyloid Immunotherapies for Alzheimer's Disease: A 2023 Clinical Update
inferred← citeshttps://doi.org/10.1007/s13365-021-01012-9openalex-snapshot
How an increase in the copy number of HSV-1 during latency can cause Alzheimer’s disease: the viral and cellular dynamics according to the microcompetition model
inferred← citeshttps://doi.org/10.1007/s40263-013-0089-3openalex-snapshot
Prospects for Improving Brain Function in Individuals with Down Syndrome
inferred← citeshttps://doi.org/10.1016/b978-0-12-386986-9.00011-9openalex-snapshot
Alzheimer's Disease and Amyloid: Culprit or Coincidence?
inferred← citeshttps://doi.org/10.1016/b978-0-12-394816-8.00009-xopenalex-snapshot
Striatal-Enriched Protein Tyrosine Phosphatase in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-396456-4.00012-2openalex-snapshot
Protein Phosphatases and Alzheimer's Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-405941-2.00009-2openalex-snapshot
From Signaling Pathways to Behavior
showing the first 200 of 1496 · page through all 1496
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.