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ALS-causing SOD1 mutants generate vascular changes prior to motor neuron degeneration
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/(sici)1098-1136(199807)23:3<249::aid-glia7>3.0.co;2-#openalex-snapshot
Relationship of microglial and astrocytic activation to disease onset and progression in a transgenic model of familial ALS
inferredcites →https://doi.org/10.1002/glia.20210openalex-snapshot
Microglia as potential contributors to motor neuron injury in amyotrophic lateral sclerosis
inferredcites →https://doi.org/10.1016/j.brainres.2007.04.044openalex-snapshot
Ultrastructure of blood–brain barrier and blood–spinal cord barrier in SOD1 mice modeling ALS
inferredcites →https://doi.org/10.1016/j.neuron.2006.09.018openalex-snapshot
ALS: A Disease of Motor Neurons and Their Nonneuronal Neighbors
inferredcites →https://doi.org/10.1016/j.tins.2005.02.001openalex-snapshot
Neurovascular mechanisms of Alzheimer's neurodegeneration
inferredcites →https://doi.org/10.1038/88842openalex-snapshot
Deletion of the hypoxia-response element in the vascular endothelial growth factor promoter causes motor neuron degeneration
inferredcites →https://doi.org/10.1038/ng1211openalex-snapshot
VEGF is a modifier of amyotrophic lateral sclerosis in mice and humans and protects motoneurons against ischemic death
inferredcites →https://doi.org/10.1038/nn1653openalex-snapshot
Selective vulnerability and pruning of phasic motoneuron axons in motoneuron disease alleviated by CNTF
inferredcites →https://doi.org/10.1073/pnas.0702230104openalex-snapshot
Toxicity from different SOD1 mutants dysregulates the complement system and the neuronal regenerative response in ALS motor neurons
inferredcites →https://doi.org/10.1073/pnas.95.26.15763openalex-snapshot
Caspase-1 is activated in neural cells and tissue with amyotrophic lateral sclerosis-associated mutations in copper-zinc superoxide dismutase
inferredcites →https://doi.org/10.1089/neu.1998.15.645openalex-snapshot
Toxic Effect of Hemoglobin on Spinal Cord Neurons in Culture
inferredcites →https://doi.org/10.1126/science.1123511openalex-snapshot
Onset and Progression in Inherited ALS Determined by Motor Neurons and Microglia
inferredcites →https://doi.org/10.1126/science.8209258openalex-snapshot
Motor Neuron Degeneration in Mice that Express a Human Cu,Zn Superoxide Dismutase Mutation
inferredcites →https://doi.org/10.1212/01.wnl.0000203120.85850.54openalex-snapshot
Axonal damage markers in cerebrospinal fluid are increased in ALS
inferredcites →https://doi.org/10.1371/journal.pone.0001205openalex-snapshot
Evidence of Compromised Blood-Spinal Cord Barrier in Early and Late Symptomatic SOD1 Mice Modeling ALS
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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inferred← citeshttps://doi.org/10.1016/j.mehy.2015.09.028openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.mehy.2016.01.006openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.nbd.2009.10.017openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.nbd.2009.12.005openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.nbd.2012.03.040openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.nbd.2014.12.002openalex-snapshot
Molecular classification of amyotrophic lateral sclerosis by unsupervised clustering of gene expression in motor cortex
inferred← citeshttps://doi.org/10.1016/j.nbd.2015.02.017openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.nbd.2015.05.009openalex-snapshot
Iron accumulation promotes TACE-mediated TNF-α secretion and neurodegeneration in a mouse model of ALS
inferred← citeshttps://doi.org/10.1016/j.nbd.2017.05.008openalex-snapshot
Brain barriers in health and disease
inferred← citeshttps://doi.org/10.1016/j.nbd.2021.105315openalex-snapshot
ALS-causing SOD1 mutants regulate occludin phosphorylation/ubiquitination and endocytic trafficking via the ITCH/Eps15/Rab5 axis
inferred← citeshttps://doi.org/10.1016/j.nbd.2025.106987openalex-snapshot
TIMP1 inhibits Rac1-mediated ROS production to ameliorate blood–spinal cord barrier disruption in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1016/j.neubiorev.2021.06.027openalex-snapshot
Interplay between immunity and amyotrophic lateral sclerosis: Clinical impact
inferred← citeshttps://doi.org/10.1016/j.neulet.2015.05.005openalex-snapshot
Alterations in the blood–spinal cord barrier in TDP-43 conditional knockout mice
inferred← citeshttps://doi.org/10.1016/j.neulet.2016.09.030openalex-snapshot
Transplantation of bone marrow mononuclear cells prolongs survival, delays disease onset and progression and mitigates neuronal loss in pre-symptomatic, but not symptomatic ALS mice
inferred← citeshttps://doi.org/10.1016/j.neulet.2019.134523openalex-snapshot
C9ORF72 protein function and immune dysregulation in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1016/j.neulet.2021.135911openalex-snapshot
Mini-Review: Induced pluripotent stem cells and the search for new cell-specific ALS therapeutic targets
inferred← citeshttps://doi.org/10.1016/j.neures.2014.01.007openalex-snapshot
Pericyte function in the physiological central nervous system
inferred← citeshttps://doi.org/10.1016/j.neures.2017.11.001openalex-snapshot
Dissociation of blood-brain barrier disruption and disease manifestation in an aquaporin-4-deficient mouse model of amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1016/j.neuro.2009.07.005openalex-snapshot
Protracted exposure to supraphysiological levels of corticosterone does not cause neuronal loss or damage and protects against kainic acid-induced neurotoxicity in the hippocampus of C57BL/6J mice
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2012.09.009openalex-snapshot
Spumiform capillary basement membrane swelling: a new type of microvascular degeneration in senescent hamster
inferred← citeshttps://doi.org/10.1016/j.neurol.2009.01.038openalex-snapshot
Actualités dans la recherche fondamentale et clinique sur la sclérose latérale amyotrophique
inferred← citeshttps://doi.org/10.1016/j.neurol.2009.05.018openalex-snapshot
Mécanismes de dérégulation de la réponse à l’hypoxie dans la sclérose latérale amyotrophique
inferred← citeshttps://doi.org/10.1016/j.neurol.2022.11.010openalex-snapshot
Retinal vessels as a window on amyotrophic lateral sclerosis pathophysiology: A systematic review
inferred← citeshttps://doi.org/10.1016/j.neuron.2010.09.009openalex-snapshot
NG2+ CNS Glial Progenitors Remain Committed to the Oligodendrocyte Lineage in Postnatal Life and following Neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.neuron.2010.09.043openalex-snapshot
Pericytes Control Key Neurovascular Functions and Neuronal Phenotype in the Adult Brain and during Brain Aging
inferred← citeshttps://doi.org/10.1016/j.neuron.2010.10.024openalex-snapshot
Pericytes: Blood-Brain Barrier Safeguards against Neurodegeneration?
inferred← citeshttps://doi.org/10.1016/j.neuron.2011.06.031openalex-snapshot
Selective Neuronal Vulnerability in Neurodegenerative Diseases: from Stressor Thresholds to Degeneration
inferred← citeshttps://doi.org/10.1016/j.neuron.2013.12.034openalex-snapshot
Reactive Gliosis and the Multicellular Response to CNS Damage and Disease
inferred← citeshttps://doi.org/10.1016/j.neuron.2017.07.030openalex-snapshot
The Neurovascular Unit Coming of Age: A Journey through Neurovascular Coupling in Health and Disease
inferred← citeshttps://doi.org/10.1016/j.neuron.2025.09.017openalex-snapshot
Neurovascular dynamics in the spinal cord from development to pathophysiology
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2018.02.022openalex-snapshot
Pleiotropic activity of systemically delivered angiogenin in the SOD1G93A mouse model
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2015.09.027openalex-snapshot
Adverse effects of a SOD1-peptide immunotherapy on SOD1G93A mouse slow model of amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1016/j.nmd.2011.10.021openalex-snapshot
Density, calibre and ramification of muscle capillaries are altered in a mouse model of severe spinal muscular atrophy
inferred← citeshttps://doi.org/10.1016/j.pain.2012.09.008openalex-snapshot
Resident glial cell activation in response to perispinal inflammation leads to acute changes in nociceptive sensitivity: Implications for the generation of neuropathic pain
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2023.105361openalex-snapshot
Correlates of Zonulin and Claudin-5, markers of intestinal and brain endothelial permeability, in Parkinson's Disease: A pilot study
inferred← citeshttps://doi.org/10.1016/j.pharmr.2025.100101openalex-snapshot
Edaravone: Advances on cytoprotective effects, pharmacological properties, and mechanisms of action
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2010.12.004openalex-snapshot
Targeting the neurovascular unit for treatment of neurological disorders
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2011.06.002openalex-snapshot
ALS: Focus on purinergic signalling
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2016.07.016openalex-snapshot
Pharmacological targeting of the PDGF-CC signaling pathway for blood–brain barrier restoration in neurological disorders
inferred← citeshttps://doi.org/10.1016/j.phrs.2019.104299openalex-snapshot
Spermine oxidase: A promising therapeutic target for neurodegeneration in diabetic retinopathy
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.105226openalex-snapshot
Repairing blood-CNS barriers: Future therapeutic approaches for neuropsychiatric disorders
inferred← citeshttps://doi.org/10.1016/j.prostaglandins.2009.04.003openalex-snapshot
Emerging roles of PGE2 receptors in models of neurological disease
inferred← citeshttps://doi.org/10.1016/j.stemcr.2018.02.012openalex-snapshot
Human iPSC-Derived Endothelial Cells and Microengineered Organ-Chip Enhance Neuronal Development
inferred← citeshttps://doi.org/10.1016/j.tins.2011.01.005openalex-snapshot
Cytoprotective protein C pathways and implications for stroke and neurological disorders
inferred← citeshttps://doi.org/10.1016/j.tins.2012.05.001openalex-snapshot
Unique biology of gliomas: challenges and opportunities
inferred← citeshttps://doi.org/10.1017/cbo9780511980053.013openalex-snapshot
Amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1017/s1462399411001918openalex-snapshot
Cerebral microvascular endothelium and the pathogenesis of neurodegenerative diseases
inferred← citeshttps://doi.org/10.1021/acsptsci.5c00153openalex-snapshot
Crossing Barriers: Advancements in Macromolecular Therapeutics for Neurodegenerative Diseases and Strategies to Overcome the Blood–Brain Barrier
inferred← citeshttps://doi.org/10.1021/cn500256eopenalex-snapshot
Brain Tissue Responses to Neural Implants Impact Signal Sensitivity and Intervention Strategies
inferred← citeshttps://doi.org/10.1021/mp3004786openalex-snapshot
Vascular Physiology and Protein Disposition in a Preclinical Model of Neurodegeneration
inferred← citeshttps://doi.org/10.1038/jcbfm.2011.155openalex-snapshot
Early and Progressive Impairment of Spinal Blood Flow—Glucose Metabolism Coupling in Motor Neuron Degeneration of ALS Model Mice
inferred← citeshttps://doi.org/10.1038/jcbfm.2011.197openalex-snapshot
Neurological Diseases in Relation to the Blood–Brain Barrier
inferred← citeshttps://doi.org/10.1038/jcbfm.2012.113openalex-snapshot
Blood–Spinal Cord Barrier Pericyte Reductions Contribute to Increased Capillary Permeability
inferred← citeshttps://doi.org/10.1038/jcbfm.2014.19openalex-snapshot
T 2 -Weighted MRI Detects Presymptomatic Pathology in the SOD1 Mouse Model of ALS
inferred← citeshttps://doi.org/10.1038/jcbfm.2015.57openalex-snapshot
Expression of the ALS-Causing Variant hSOD1 G93A Leads to an Impaired Integrity and Altered Regulation of Claudin-5 Expression in an in Vitro Blood–Spinal Cord Barrier Model
inferred← citeshttps://doi.org/10.1038/mt.2013.211openalex-snapshot
Therapeutic AAV9-mediated Suppression of Mutant SOD1 Slows Disease Progression and Extends Survival in Models of Inherited ALS
inferred← citeshttps://doi.org/10.1038/nature13324openalex-snapshot
Mfsd2a is critical for the formation and function of the blood–brain barrier
inferred← citeshttps://doi.org/10.1038/nm.2226openalex-snapshot
Degeneration and repair in central nervous system disease
inferred← citeshttps://doi.org/10.1038/nm.3407openalex-snapshot
Development, maintenance and disruption of the blood-brain barrier
inferred← citeshttps://doi.org/10.1038/nm1210-1370openalex-snapshot
Neurodegeneration and the neurovascular unit
inferred← citeshttps://doi.org/10.1038/nn.2297openalex-snapshot
A role for motoneuron subtype–selective ER stress in disease manifestations of FALS mice
inferred← citeshttps://doi.org/10.1038/nn.2946openalex-snapshot
Central nervous system pericytes in health and disease
inferred← citeshttps://doi.org/10.1038/nn.2947openalex-snapshot
Cerebrovascular disorders: molecular insights and therapeutic opportunities
inferred← citeshttps://doi.org/10.1038/nn.4288openalex-snapshot
Pericytes of the neurovascular unit: key functions and signaling pathways
inferred← citeshttps://doi.org/10.1038/nrn3114openalex-snapshot
Neurovascular pathways to neurodegeneration in Alzheimer's disease and other disorders
inferred← citeshttps://doi.org/10.1038/nrn3430openalex-snapshot
The changing scene of amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1038/nrneurol.2017.188openalex-snapshot
Blood–brain barrier breakdown in Alzheimer disease and other neurodegenerative disorders
inferred← citeshttps://doi.org/10.1038/s41380-022-01511-zopenalex-snapshot
The blood-brain barrier in aging and neurodegeneration
inferred← citeshttps://doi.org/10.1038/s41419-019-2104-1openalex-snapshot
A role for spinal cord hypoxia in neurodegeneration
inferred← citeshttps://doi.org/10.1038/s41582-024-00991-7openalex-snapshot
Brain–body mechanisms contribute to sexual dimorphism in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1038/s41591-021-01295-9openalex-snapshot
Altered perivascular fibroblast activity precedes ALS disease onset
inferred← citeshttps://doi.org/10.1038/s41598-017-00993-0openalex-snapshot
Endothelial and Astrocytic Support by Human Bone Marrow Stem Cell Grafts into Symptomatic ALS Mice towards Blood-Spinal Cord Barrier Repair
inferred← citeshttps://doi.org/10.1038/s41598-018-29845-1openalex-snapshot
Decreased signalling of EphA4 improves functional performance and motor neuron survival in the SOD1G93A ALS mouse model
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