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Unravelling neurogenetic networks implicated in developmental language disorders

https://doi.org/10.1042/bst0371263
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Current directions in research on autism
inferredcites →https://doi.org/10.1002/ajmg.a.31110openalex-snapshot
Speech and language impairment and oromotor dyspraxia due to deletion of 7q31 that involves FOXP2
inferredcites →https://doi.org/10.1002/ajmg.a.31632openalex-snapshot
Deletion of 7q31.1 supports involvement of FOXP2 in language impairment: Clinical report and review
inferredcites →https://doi.org/10.1002/dvg.20305openalex-snapshot
Generation of mice with a conditional Foxp2 null allele
inferredcites →https://doi.org/10.1006/dbio.2002.0780openalex-snapshot
Forkhead Transcription Factors: Key Players in Development and Metabolism
inferredcites →https://doi.org/10.1007/s00439-002-0768-5openalex-snapshot
FOXP2: novel exons, splice variants, and CAG repeat length stability
inferredcites →https://doi.org/10.1016/j.ajhg.2007.09.005openalex-snapshot
Linkage, Association, and Gene-Expression Analyses Identify CNTNAP2 as an Autism-Susceptibility Gene
inferredcites →https://doi.org/10.1016/j.bbrc.2006.12.130openalex-snapshot
Intracellular distribution of a speech/language disorder associated FOXP2 mutant
inferredcites →https://doi.org/10.1016/j.biocel.2004.02.003openalex-snapshot
Microtubule disruption inhibits autophagosome-lysosome fusion: implications for studying the roles of aggresomes in polyglutamine diseases
inferredcites →https://doi.org/10.1016/j.cub.2008.01.060openalex-snapshot
Impaired Synaptic Plasticity and Motor Learning in Mice with a Point Mutation Implicated in Human Speech Deficits
inferredcites →https://doi.org/10.1016/j.gene.2008.01.015openalex-snapshot
Multiple transcription start sites for FOXP2 with varying cellular specificities
inferredcites →https://doi.org/10.1016/j.str.2005.10.005openalex-snapshot
Structure of the Forkhead Domain of FOXP2 Bound to DNA
inferredcites →https://doi.org/10.1016/j.tics.2005.09.002openalex-snapshot
Genetic influences on language impairment and phonological short-term memory
inferredcites →https://doi.org/10.1016/j.tics.2006.04.003openalex-snapshot
Genes, cognition and dyslexia: learning to read the genome
inferredcites →https://doi.org/10.1016/j.tig.2009.03.002openalex-snapshot
FOXP2 as a molecular window into speech and language
inferredcites →https://doi.org/10.1016/s0168-9525(03)00111-2openalex-snapshot
Fox's in development and disease
inferredcites →https://doi.org/10.1016/s0962-8924(00)01852-3openalex-snapshot
Aggresomes, inclusion bodies and protein aggregation
inferredcites →https://doi.org/10.1038/35097076openalex-snapshot
A forkhead-domain gene is mutated in a severe speech and language disorder
inferredcites →https://doi.org/10.1038/84781openalex-snapshot
The putative forkhead transcription factor FOXL2 is mutated in blepharophimosis/ptosis/epicanthus inversus syndrome
inferredcites →https://doi.org/10.1038/ejhg.2009.43openalex-snapshot
Assessing the impact of FOXP1 mutations on developmental verbal dyspraxia
inferredcites →https://doi.org/10.1038/embor.2008.130openalex-snapshot
Many forkheads in the road to regulation
inferredcites →https://doi.org/10.1038/ng0298-168openalex-snapshot
Localisation of a gene implicated in a severe speech and language disorder
inferredcites →https://doi.org/10.1038/ng890openalex-snapshot
Regulation of insulin action and pancreatic β-cell function by mutated alleles of the gene encoding forkhead transcription factor Foxo1
inferredcites →https://doi.org/10.1038/nn1138openalex-snapshot
Language fMRI abnormalities associated with FOXP2 gene mutation
inferredcites →https://doi.org/10.1038/nrg1747openalex-snapshot
The eloquent ape: genes, brains and the evolution of language
inferredcites →https://doi.org/10.1038/nrg2346openalex-snapshot
Advances in autism genetics: on the threshold of a new neurobiology
inferredcites →https://doi.org/10.1038/nrn936openalex-snapshot
Developmental dyslexia: genetic dissection of a complex cognitive trait
inferredcites →https://doi.org/10.1044/1092-4388(2006/038)openalex-snapshot
Speech, Prosody, and Voice Characteristics of a Mother and Daughter With a 7;13 Translocation AffectingFOXP2
inferredcites →https://doi.org/10.1056/nejmoa052773openalex-snapshot
Recessive Symptomatic Focal Epilepsy and Mutant Contactin-Associated Protein-like 2
inferredcites →https://doi.org/10.1056/nejmoa0802828openalex-snapshot
A Functional Genetic Link between Distinct Developmental Language Disorders
inferredcites →https://doi.org/10.1073/pnas.0511197103openalex-snapshot
Voltage-gated ion channels in the axon initial segment of human cortical pyramidal cells and their relationship with chandelier cells
inferredcites →https://doi.org/10.1074/jbc.m207174200openalex-snapshot
Multiple Domains Define the Expression and Regulatory Properties of Foxp1 Forkhead Transcriptional Repressors
inferredcites →https://doi.org/10.1074/jbc.m310994200openalex-snapshot
Aggresomes Formed by α-Synuclein and Synphilin-1 Are Cytoprotective
inferredcites →https://doi.org/10.1086/303011openalex-snapshot
The SPCH1 Region on Human 7q31: Genomic Characterization of the Critical Interval and Localization of Translocations Associated with Speech and Language Disorder
inferredcites →https://doi.org/10.1086/338649openalex-snapshot
A Genomewide Scan Identifies Two Novel Loci Involved in Specific Language Impairment**Members of the consortium are listed in the Appendix.
inferredcites →https://doi.org/10.1086/430841openalex-snapshot
Identification of FOXP2 Truncation as a Novel Cause of Developmental Speech and Language Deficits
inferredcites →https://doi.org/10.1086/508902openalex-snapshot
Absence of a Paternally Inherited FOXP2 Gene in Developmental Verbal Dyspraxia
inferredcites →https://doi.org/10.1086/522237openalex-snapshot
Identification of the Transcriptional Targets of FOXP2, a Gene Linked to Speech and Language, in Developing Human Brain
inferredcites →https://doi.org/10.1086/522238openalex-snapshot
High-Throughput Analysis of Promoter Occupancy Reveals Direct Neural Targets of FOXP2, a Gene Mutated in Speech and Language Disorders
inferredcites →https://doi.org/10.1093/brain/awf057openalex-snapshot
MRI analysis of an inherited speech and language disorder: structural brain abnormalities
inferredcites →https://doi.org/10.1093/brain/awf058openalex-snapshot
Behavioural analysis of an inherited speech and language disorder: comparison with acquired aphasia
inferredcites →https://doi.org/10.1093/brain/awg247openalex-snapshot
FOXP2 expression during brain development coincides with adult sites of pathology in a severe speech and language disorder
inferredcites →https://doi.org/10.1093/hmg/ddg324openalex-snapshot
Structural and functional analyses of disease-causing missense mutations in the forkhead domain of FOXC1
inferredcites →https://doi.org/10.1093/hmg/ddl392openalex-snapshot
Functional genetic analysis of mutations implicated in a human speech and language disorder
inferredcites →https://doi.org/10.1097/mcd.0b013e3282f17688openalex-snapshot
De-novo balanced translocation between 7q31 and 10p14 in a girl with central precocious puberty, moderate mental retardation, and severe speech impairment
inferredcites →https://doi.org/10.1109/tnb.2004.842463openalex-snapshot
Voltage-Gated Ion Channels
inferredcites →https://doi.org/10.1111/j.1469-8749.1990.tb16948.xopenalex-snapshot
An extended Family with a Dominantly Inherited Speech Disorder
inferredcites →https://doi.org/10.1128/mcb.24.2.809-822.2004openalex-snapshot
Transcriptional and DNA Binding Activity of the Foxp1/2/4 Family Is Modulated by Heterotypic and Homotypic Protein Interactions
inferredcites →https://doi.org/10.1146/annurev.neuro.26.041002.131144openalex-snapshot
DECIPHERING THE GENETIC BASIS OF SPEECH AND LANGUAGE DISORDERS
inferredcites →https://doi.org/10.1159/000101771openalex-snapshot
Molecular Windows into Speech and Language Disorders
inferredcites →https://doi.org/10.4049/jimmunol.177.5.3133openalex-snapshot
Analysis of FOXP3 Reveals Multiple Domains Required for Its Function as a Transcriptional Repressor

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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inferred← citeshttps://doi.org/10.1002/ajmg.a.34354openalex-snapshot
Phenotype of FOXP2 haploinsufficiency in a mother and son
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An Informatics Approach to Integrating Genetic and Neurological Data in Speech and Language Neuroscience
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Genes expressed in Atoh1 neuronal lineages arising from the r1/isthmus rhombic lip
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Foxp2 Mutations Impair Auditory-Motor Association Learning
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