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TREM2 Variants in Alzheimer's Disease
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/jamaneurol.2013.579openalex-snapshot
Using Exome Sequencing to Reveal Mutations in TREM2 Presenting as a Frontotemporal Dementia–like Syndrome Without Bone Involvement
inferredcites →https://doi.org/10.1002/eji.200636837openalex-snapshot
Blockade of TREM‐2 exacerbates experimental autoimmune encephalomyelitis
inferredcites →https://doi.org/10.1002/glia.20710openalex-snapshot
TREM2 is upregulated in amyloid plaque‐associated microglia in aged APP23 transgenic mice
inferredcites →https://doi.org/10.1016/j.imbio.2011.07.017openalex-snapshot
Complement receptor 1 (CR1) and Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jneuroim.2006.11.032openalex-snapshot
Essential role of the microglial triggering receptor expressed on myeloid cells-2 (TREM2) for central nervous tissue immune homeostasis
inferredcites →https://doi.org/10.1016/j.neuint.2006.04.002openalex-snapshot
Microglia and the control of autoreactive T cell responses
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2009.03.013openalex-snapshot
Meta-analysis of linkage studies for Alzheimer's disease—A web resource
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2012.07.001openalex-snapshot
Genetic association of CR1 with Alzheimer's disease: A tentative disease mechanism
inferredcites →https://doi.org/10.1016/j.tig.2010.07.004openalex-snapshot
Towards a complete resolution of the genetic architecture of disease
inferredcites →https://doi.org/10.1038/349704a0openalex-snapshot
Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease
inferredcites →https://doi.org/10.1038/35050110openalex-snapshot
Aβ peptide immunization reduces behavioural impairment and plaques in a model of Alzheimer's disease
inferredcites →https://doi.org/10.1038/375754a0openalex-snapshot
Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease
inferredcites →https://doi.org/10.1038/376775a0openalex-snapshot
Familial Alzheimer's disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer's disease type 3 gene
inferredcites →https://doi.org/10.1038/nature07517openalex-snapshot
Accurate whole human genome sequencing using reversible terminator chemistry
inferredcites →https://doi.org/10.1038/ng.439openalex-snapshot
Genome-wide association study identifies variants at CLU and CR1 associated with Alzheimer's disease
inferredcites →https://doi.org/10.1038/ng.440openalex-snapshot
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
inferredcites →https://doi.org/10.1038/ng.801openalex-snapshot
Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease
inferredcites →https://doi.org/10.1038/ng.803openalex-snapshot
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease
inferredcites →https://doi.org/10.1038/ng.806openalex-snapshot
A framework for variation discovery and genotyping using next-generation DNA sequencing data
inferredcites →https://doi.org/10.1038/nmeth0410-248openalex-snapshot
A method and server for predicting damaging missense mutations
inferredcites →https://doi.org/10.1056/nejmc1113592openalex-snapshot
Repeat Expansion inC9ORF72in Alzheimer's Disease
inferredcites →https://doi.org/10.1073/pnas.90.5.1977openalex-snapshot
Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease.
inferredcites →https://doi.org/10.1074/jbc.m100710200openalex-snapshot
Early-onset Amyloid Deposition and Cognitive Deficits in Transgenic Mice Expressing a Double Mutant Form of Amyloid Precursor Protein 695
inferredcites →https://doi.org/10.1086/342259openalex-snapshot
Mutations in Two Genes Encoding Different Subunits of a Receptor Signaling Complex Result in an Identical Disease Phenotype
inferredcites →https://doi.org/10.1093/bioinformatics/btp324openalex-snapshot
Fast and accurate short read alignment with Burrows–Wheeler transform
inferredcites →https://doi.org/10.1093/bioinformatics/btp352openalex-snapshot
The Sequence Alignment/Map format and SAMtools
inferredcites →https://doi.org/10.1093/nar/gkq603openalex-snapshot
ANNOVAR: functional annotation of genetic variants from high-throughput sequencing data
inferredcites →https://doi.org/10.1101/gr.107524.110openalex-snapshot
The Genome Analysis Toolkit: A MapReduce framework for analyzing next-generation DNA sequencing data
inferredcites →https://doi.org/10.1126/science.1215040openalex-snapshot
A Systematic Survey of Loss-of-Function Variants in Human Protein-Coding Genes
inferredcites →https://doi.org/10.1155/2012/983640openalex-snapshot
Microglia, Alzheimer's Disease, and Complement
inferredcites →https://doi.org/10.1212/wnl.53.2.321openalex-snapshot
APOE -ε4 count predicts age when prevalence of AD increases, then declines
inferredcites →https://doi.org/10.1371/journal.pgen.1000529openalex-snapshot
A Flexible and Accurate Genotype Imputation Method for the Next Generation of Genome-Wide Association Studies
inferredcites →https://doi.org/10.1371/journal.pmed.0040124openalex-snapshot
TREM2-Transduced Myeloid Precursors Mediate Nervous Tissue Debris Clearance and Facilitate Recovery in an Animal Model of Multiple Sclerosis
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3155885openalex-extracted
Loss of Ataxin-1 Elevates BACE1 Expression and Impairs Axonal Targeting in the Cerebrum
inferred← citeshttps://doi.org/10.2139/ssrn.3377366openalex-extracted
Transcriptional Signatures of Progressive Neuropathology in Transgenic Models of Tau and Amyloid Pathology
inferred← citeshttps://doi.org/10.2139/ssrn.4588950openalex-extracted
Yishen Huazhuo Decoction Regulates Microglial Polarization to Reduce Neuroinflammation at the Early Stage of Alzheimer's Disease Through TREM2
inferred← citeshttps://doi.org/10.1001/jamanetworkopen.2019.1350openalex-snapshot
Association of Rare Coding Mutations With Alzheimer Disease and Other Dementias Among Adults of European Ancestry
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.2152openalex-snapshot
Cerebrovascular Effects of Apolipoprotein E
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.275openalex-snapshot
An Inflection Point in Gene Discovery Efforts for Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.3545openalex-snapshot
Evidence of Recessive Alzheimer Disease Loci in a Caribbean Hispanic Data Set
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.3682openalex-snapshot
From Genome-Wide Association Studies to Next-Generation Sequencing
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.453openalex-snapshot
TREM2, Frontotemporal Dementia–Like Disease, Nasu-Hakola Disease, and Alzheimer Dementia: A Chicken and Egg Problem?
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.6030openalex-snapshot
Elevated Serum Pesticide Levels and Risk for Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.6237openalex-snapshot
TREM2Variant p.R47H as a Risk Factor for Sporadic Amyotrophic Lateral Sclerosis
inferred← citeshttps://doi.org/10.1001/jamaneurol.2014.1491openalex-snapshot
Effects of Multiple Genetic Loci on Age at Onset in Late-Onset Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2014.3049openalex-snapshot
Association of Brain DNA Methylation inSORL1,ABCA7,HLA-DRB5,SLC24A4, andBIN1With Pathological Diagnosis of Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2014.4040openalex-snapshot
A Multiancestral Genome-Wide Exome Array Study of Alzheimer Disease, Frontotemporal Dementia, and Progressive Supranuclear Palsy
inferred← citeshttps://doi.org/10.1001/jamaneurol.2015.0979openalex-snapshot
R47H Variant ofTREM2Associated With Alzheimer Disease in a Large Late-Onset Family
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.0150openalex-snapshot
Association Between Genetic Traits for Immune-Mediated Diseases and Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.0301openalex-snapshot
Genomics of Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.0407openalex-snapshot
New Genetic Links Between Alzheimer Disease and Immune-Mediated Diseases Revealed by Overlapping Genome-wide Association Study Hits
inferred← citeshttps://doi.org/10.1001/jamaneurol.2017.0843openalex-snapshot
Parkinson Disease and Autoimmune Disorders—What Can We Learn From Genome-wide Pleiotropy?
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.1066openalex-snapshot
Association of Variants in PINX1 and TREM2 With Late-Onset Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.1456openalex-snapshot
Analysis of Whole-Exome Sequencing Data for Alzheimer Disease Stratified by APOE Genotype
inferred← citeshttps://doi.org/10.1001/jamaneurol.2020.3536openalex-snapshot
Novel Alzheimer Disease Risk Loci and Pathways in African American Individuals Using the African Genome Resources Panel
inferred← citeshttps://doi.org/10.1001/jamaneurol.2024.0991openalex-snapshot
Downstream Biomarker Effects of Gantenerumab or Solanezumab in Dominantly Inherited Alzheimer Disease
inferred← citeshttps://doi.org/10.1002/2211-5463.13300openalex-snapshot
Reduced TREM2 activation in microglia of patients with Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/9780470015902.a0020228.pub2openalex-snapshot
Genetics of A lzheimer's Disease
inferred← citeshttps://doi.org/10.1002/9781118486160.ch8openalex-snapshot
Dementia and Cognitive Impairment
inferred← citeshttps://doi.org/10.1002/9781118540770.ch1openalex-snapshot
Genetic Screens in Neurodegeneration
inferred← citeshttps://doi.org/10.1002/9781119356752.ch15openalex-snapshot
The Role of Genetics in Alzheimer's Disease and Parkinson's Disease
inferred← citeshttps://doi.org/10.1002/acn3.369openalex-snapshot
Genomics implicates adaptive and innate immunity in Alzheimer's and Parkinson's diseases
inferred← citeshttps://doi.org/10.1002/acn3.537openalex-snapshot
Whole genome sequencing of Caribbean Hispanic families with late‐onset Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/acn3.730openalex-snapshot
Anti‐PrPC antibody rescues cognition and synapses in transgenic alzheimer mice
inferred← citeshttps://doi.org/10.1002/adhm.202304180openalex-snapshot
Nanomaterials as Microglia Modulators in the Treatment of Central Nervous System Disorders
inferred← citeshttps://doi.org/10.1002/adhm.202500498openalex-snapshot
Biomaterials in Alzheimer's Disease: An Anti‐Neuroinflammatory Perspective
inferred← citeshttps://doi.org/10.1002/advs.202400064openalex-snapshot
Microglia Gravitate toward Amyloid Plaques Surrounded by Externalized Phosphatidylserine via TREM2
inferred← citeshttps://doi.org/10.1002/advs.202407812openalex-snapshot
A Novel RAGE Modulator Induces Soluble RAGE to Reduce BACE1 Expression in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/advs.202417733openalex-snapshot
Microglial MS4A4A Protects against Epileptic Seizures in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/ajmg.b.32201openalex-snapshot
Epigenetic studies in Alzheimer's disease: Current findings, caveats, and considerations for future studies
inferred← citeshttps://doi.org/10.1002/ajmg.b.32493openalex-snapshot
Mutations modifying sporadic Alzheimer's disease age of onset
inferred← citeshttps://doi.org/10.1002/ajmg.b.32499openalex-snapshot
Genomic variants, genes, and pathways of Alzheimer's disease: An overview
inferred← citeshttps://doi.org/10.1002/alz.043897openalex-snapshot
Integration of Alzheimer’s disease genetics and myeloid genomics reveals novel disease risk mechanisms
inferred← citeshttps://doi.org/10.1002/alz.044901openalex-snapshot
How understudied populations leverage our understanding of Alzheimer’s disease genetics
inferred← citeshttps://doi.org/10.1002/alz.075938openalex-snapshot
Amyloid β Induces Lipid Droplet‐Mediated Microglial Dysfunction in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1002/alz.12011openalex-snapshot
Genetic screening of a large series of North American sporadic and familial frontotemporal dementia cases
inferred← citeshttps://doi.org/10.1002/alz.12088openalex-snapshot
GABAergic dysfunction in excitatory and inhibitory (E/I) imbalance drives the pathogenesis of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12119openalex-snapshot
Exploratory analysis of mtDNA haplogroups in two Alzheimer's longitudinal cohorts
inferred← citeshttps://doi.org/10.1002/alz.12153openalex-snapshot
The APOE ε4 exerts differential effects on familial and other subtypes of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12194openalex-snapshot
Functional insights from biophysical study of TREM2 interactions with apoE and Aβ 1‐42
inferred← citeshttps://doi.org/10.1002/alz.12240openalex-snapshot
Harnessing the paradoxical phenotypes of APOE ɛ2 and APOE ɛ4 to identify genetic modifiers in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12256openalex-snapshot
Reactive or transgenic increase in microglial TYROBP reveals a TREM2‐independent TYROBP–APOE link in wild‐type and Alzheimer's‐related mice
inferred← citeshttps://doi.org/10.1002/alz.12319openalex-snapshot
Whole‐genome sequencing reveals new Alzheimer's disease–associated rare variants in loci related to synaptic function and neuronal development
inferred← citeshttps://doi.org/10.1002/alz.12389openalex-snapshot
Diversity of transcriptomic microglial phenotypes in aging and Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12481openalex-snapshot
Engagement of vascular early response genes typifies mild cognitive impairment
inferred← citeshttps://doi.org/10.1002/alz.12577openalex-snapshot
The P522R protective variant of PLCG2 promotes the expression of antigen presentation genes by human microglia in an Alzheimer's disease mouse model
inferred← citeshttps://doi.org/10.1002/alz.13427openalex-snapshot
Artificial intelligence for dementia genetics and omics
inferred← citeshttps://doi.org/10.1002/alz.13502openalex-snapshot
Alzheimer's disease‐induced phagocytic microglia express a specific profile of coding and non‐coding RNAs
inferred← citeshttps://doi.org/10.1002/alz.13705openalex-snapshot
Key variants via the Alzheimer's Disease Sequencing Project whole genome sequence data
inferred← citeshttps://doi.org/10.1002/alz.13858openalex-snapshot
A cross‐sectional study of α‐synuclein seed amplification assay in Alzheimer's disease neuroimaging initiative: Prevalence and associations with Alzheimer's disease biomarkers and cognitive function
inferred← citeshttps://doi.org/10.1002/alz.13862openalex-snapshot
CSF glial biomarkers are associated with cognition in individuals at risk of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.13921openalex-snapshot
Primate cerebrospinal fluid CHI3L1 reflects brain TREM2 agonism
inferred← citeshttps://doi.org/10.1002/alz.14065openalex-snapshot
Osteopontin: A novel marker of pre‐symptomatic sporadic Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.14124openalex-snapshot
Multimerization of TREM2 is impaired by Alzheimer's disease–associated variants
inferred← citeshttps://doi.org/10.1002/alz.14214openalex-snapshot
Transethnic analysis identifies SORL1 variants and haplotypes protective against Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70120openalex-snapshot
Synergistic reduction in interfacial flexibility of TREM2 R47H and ApoE4 may underlie AD pathology
inferred← citeshttps://doi.org/10.1002/alz.70220openalex-snapshot
Genetic and environmental risk factors for dementia in African adults: A systematic review
inferred← citeshttps://doi.org/10.1002/alz.70458openalex-snapshot
The Alzheimer's disease‐associated complement receptor 1 variant confers risk by impacting glial phagocytosis
inferred← citeshttps://doi.org/10.1002/alz.70580openalex-snapshot
Ms4a4a deficiency ameliorates plaque pathology in a mouse model of amyloid accumulation
inferred← citeshttps://doi.org/10.1002/alz.70586openalex-snapshot
The TREM2 H157Y variant is associated with more severe neurodegeneration in Alzheimer's disease and altered immune‐related processes
inferred← citeshttps://doi.org/10.1002/alz.70729openalex-snapshot
TREM2‐apoE3 interactions and Alzheimer's disease: Molecular and structural insights and effects of TREM2 R47H and apoE4 variants
inferred← citeshttps://doi.org/10.1002/alz.70896openalex-snapshot
Genetic variation in the Nr1d1 transcription factor binding site shapes metabolism‐related protein networks associated with cognitive resilience in an Alzheimer's disease mouse reference panel
inferred← citeshttps://doi.org/10.1002/alz.70911openalex-snapshot
Neuroinflammatory mechanisms linking high‐fat diets to Alzheimer's disease vulnerability: Beyond the amyloid hypothesis
inferred← citeshttps://doi.org/10.1002/alz.70978openalex-snapshot
Genotype–phenotype interaction in Alzheimer's disease immune activation
inferred← citeshttps://doi.org/10.1002/alz.71185openalex-snapshot
Spatial heterogeneity in microglia‐complement crosstalk: Implications for synaptic pruning in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.71452openalex-snapshot
Abi3 S212F Alzheimer's disease variant alters plaque structure and disrupts microglia
inferred← citeshttps://doi.org/10.1002/alz.71688openalex-snapshot
Proteomic signatures of protected APOE ε4 carriers reveal causal pathways associated with delayed Alzheimer's disease onset
inferred← citeshttps://doi.org/10.1002/ame2.12267openalex-snapshot
Potential anti neuro‐inflammatory effect of BioCen‐128 in animal models of dementia
inferred← citeshttps://doi.org/10.1002/ana.24001openalex-snapshot
The therapeutics of Alzheimer's disease: Where we stand and where we are heading
inferred← citeshttps://doi.org/10.1002/ana.24337openalex-snapshot
A TREM 1 variant alters the accumulation of Alzheimer‐related amyloid pathology
inferred← citeshttps://doi.org/10.1002/ana.24394openalex-snapshot
F yn inhibition rescues established memory and synapse loss in A lzheimer mice
inferred← citeshttps://doi.org/10.1002/ana.24466openalex-snapshot
Rare coding mutations identified by sequencing of A lzheimer disease genome‐wide association studies loci
inferred← citeshttps://doi.org/10.1002/ana.25385openalex-snapshot
Serum Soluble Triggering Receptor Expressed on Myeloid Cells 2 as a Biomarker for Incident Dementia: The Hisayama Study
inferred← citeshttps://doi.org/10.1002/ana.25543openalex-snapshot
Interleukin 9 alterations linked to alzheimer disease in african americans
inferred← citeshttps://doi.org/10.1002/ana.26032openalex-snapshot
Heritability Enrichment Implicates Microglia in Parkinson's Disease Pathogenesis
inferred← citeshttps://doi.org/10.1002/ana.78170openalex-snapshot
Understanding Neurodegenerative Diseases From the −Omics Perspective: Lessons Learnt
inferred← citeshttps://doi.org/10.1002/bkcs.12126openalex-snapshot
Phosphonamidate Compounds for Butyrylcholinesterase Selective Inhibitors
inferred← citeshttps://doi.org/10.1002/brb3.1573openalex-snapshot
A TREML2 missense variant influences specific hippocampal subfield volumes in cognitively normal elderly subjects
inferred← citeshttps://doi.org/10.1002/cbic.202400224openalex-snapshot
Role of Amyloidogenic and Non‐Amyloidogenic Protein Spaces in Neurodegenerative Diseases and their Mitigation Using Theranostic Agents
inferred← citeshttps://doi.org/10.1002/cbic.202500515openalex-snapshot
A Drug Screening Targeting Upregulation of Syk Gene Identifies Neochlorogenic Acid and L‐Arabinitol Capable of Inducing the M2 Phenotype of Microglia in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/cne.24484openalex-snapshot
Microglia changes associated to Alzheimer's disease pathology in aged chimpanzees
inferred← citeshttps://doi.org/10.1002/cpns.81openalex-snapshot
Overview of Transgenic Mouse Models for Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/cpt.195openalex-snapshot
Promising Targets for the Treatment of Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1002/ctd2.70140openalex-snapshot
Physiology, pathology and therapeutic potential of microglial triggering receptor expressed on myeloid cells 2 signalling: A narrative update
inferred← citeshttps://doi.org/10.1002/ctm2.70863openalex-snapshot
Context‐dependent TREM2 signalling in myeloid cells: Molecular mechanisms and immunotherapeutic divergence in neurodegeneration and cancer
inferred← citeshttps://doi.org/10.1002/dad2.12074openalex-snapshot
Genetic and polygenic risk score analysis for Alzheimer's disease in the Chinese population
inferred← citeshttps://doi.org/10.1002/dad2.12128openalex-snapshot
A high cerebrospinal fluid soluble TREM2 level is associated with slow clinical progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/dad2.12155openalex-snapshot
Insight into the genetic etiology of Alzheimer's disease: A comprehensive review of the role of rare variants
inferred← citeshttps://doi.org/10.1002/dad2.12255openalex-snapshot
Large‐scale sequencing studies expand the known genetic architecture of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/dneu.22777openalex-snapshot
Understanding microglial diversity and implications for neuronal function in health and disease
inferred← citeshttps://doi.org/10.1002/dneu.22814openalex-snapshot
Microglia regulate synaptic development and plasticity
inferred← citeshttps://doi.org/10.1002/dneu.22864openalex-snapshot
Microglial TREM2 in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1002/dvdy.1openalex-snapshot
Developmental roles of microglia: A window into mechanisms of disease
inferred← citeshttps://doi.org/10.1002/efd2.145openalex-snapshot
The potential use of natural killer cells to treat Alzheimer's disease: Can nutrition play a role?
inferred← citeshttps://doi.org/10.1002/eji.202250266openalex-snapshot
The innate immune response in tauopathies
inferred← citeshttps://doi.org/10.1002/eji.70250openalex-snapshot
Active Vaccination Strategies for Alzheimer's Disease: An Expanded Review
inferred← citeshttps://doi.org/10.1002/embj.201387637openalex-snapshot
Epigenetic memory: the Lamarckian brain
inferred← citeshttps://doi.org/10.1002/gepi.22093openalex-snapshot
Using imputed genotype data in the joint score tests for genetic association and gene–environment interactions in case‐control studies
inferred← citeshttps://doi.org/10.1002/glia.22802openalex-snapshot
A DAP12‐Dependent signal promotes pro‐inflammatory polarization in microglia following nerve injury and exacerbates degeneration of injured neurons
inferred← citeshttps://doi.org/10.1002/glia.22866openalex-snapshot
Next generation transcriptomics and genomics elucidate biological complexity of microglia in health and disease
inferred← citeshttps://doi.org/10.1002/glia.22988openalex-snapshot
Targeting microglia for the treatment of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/glia.23024openalex-snapshot
Inhomogeneous distribution of Iba‐1 characterizes microglial pathology in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.23074openalex-snapshot
Microglia contributes to plaque growth by cell death due to uptake of amyloid β in the brain of Alzheimer's disease mouse model
inferred← citeshttps://doi.org/10.1002/glia.23204openalex-snapshot
Siglec‐H is a microglia‐specific marker that discriminates microglia from CNS‐associated macrophages and CNS‐infiltrating monocytes
inferred← citeshttps://doi.org/10.1002/glia.23563openalex-snapshot
TREM2 triggers microglial density and age‐related neuronal loss
inferred← citeshttps://doi.org/10.1002/glia.23664openalex-snapshot
TREM2 is required for microglial instruction of astrocytic synaptic engulfment in neurodevelopment
inferred← citeshttps://doi.org/10.1002/glia.23781openalex-snapshot
Human iPSC‐derived microglia: A growing toolset to study the brain's innate immune cells
inferred← citeshttps://doi.org/10.1002/glia.23804openalex-snapshot
Increased oxidative stress, hyperphosphorylation of tau, and dystrophic microglia in the hippocampus of aged Tupaia belangeri
inferred← citeshttps://doi.org/10.1002/glia.23953openalex-snapshot
A reporter cell system for the triggering receptor expressed on myeloid cells 2 reveals differential effects of disease‐associated variants on receptor signaling and activation by antibodies against the stalk region
inferred← citeshttps://doi.org/10.1002/glia.24047openalex-snapshot
Regulation of microglia population dynamics throughout development, health, and disease
inferred← citeshttps://doi.org/10.1002/glia.24077openalex-snapshot
Differential interaction with TREM2 modulates microglial uptake of modified Aβ species
inferred← citeshttps://doi.org/10.1002/glia.24144openalex-snapshot
MAC2 is a long‐lasting marker of peripheral cell infiltrates into the mouse CNS after bone marrow transplantation and coronavirus infection
inferred← citeshttps://doi.org/10.1002/glia.24145openalex-snapshot
Phagocytic astrocytes: Emerging from the shadows of microglia
inferred← citeshttps://doi.org/10.1002/glia.24318openalex-snapshot
Alzheimer's disease‐associated R47H TREM2 increases, but wild‐type TREM2 decreases, microglial phagocytosis of synaptosomes and neuronal loss
inferred← citeshttps://doi.org/10.1002/glia.24355openalex-snapshot
Complement receptor 1 is expressed on brain cells and in the human brain
inferred← citeshttps://doi.org/10.1002/glia.24458openalex-snapshot
Plaque vicinity as a hotspot of microglial turnover in a mouse model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.24461openalex-snapshot
Endothelial senescence alleviates cognitive impairment in a mouse model of Alzheimer 's disease
inferred← citeshttps://doi.org/10.1002/glia.24465openalex-snapshot
How brain ‘cleaners’ fail: Mechanisms and therapeutic value of microglial phagocytosis in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/glia.24485openalex-snapshot
iPSC ‐derived microglia carrying the TREM2 R47H /+ mutation are proinflammatory and promote synapse loss
inferred← citeshttps://doi.org/10.1002/glia.24574openalex-snapshot
The role of microglial TREM2 in development: A path toward neurodegeneration?
inferred← citeshttps://doi.org/10.1002/glia.24652openalex-snapshot
Multi Layered Omics Approaches Reveal Glia Specific Alterations in Alzheimer's Disease: A Systematic Review and Future Prospects
inferred← citeshttps://doi.org/10.1002/gps.5260openalex-snapshot
Systematic review of genetic association studies in people with Lewy body dementia
inferred← citeshttps://doi.org/10.1002/hbm.24782openalex-snapshot
Applications of amyloid, tau, and neuroinflammation PET imaging to Alzheimer's disease and mild cognitive impairment
inferred← citeshttps://doi.org/10.1002/hsr2.71631openalex-snapshot
Post‐Covid Alzheimer and Its Remediation via PROTACs Therapy: A Comprehensive Review
inferred← citeshttps://doi.org/10.1002/jbmr.4572openalex-snapshot
Triggering Receptor Expressed on Myeloid Cells 2 (TREM2) R47H Variant Causes Distinct Age- and Sex-Dependent Musculoskeletal Alterations in Mice
inferred← citeshttps://doi.org/10.1002/jcb.25089openalex-snapshot
Lyar Is a New Ligand for Retinal Pigment Epithelial Phagocytosis
inferred← citeshttps://doi.org/10.1002/jcp.25225openalex-snapshot
Common and Rare Genetic Variants Associated With Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/jcp.30829openalex-snapshot
An overview on microglial origin, distribution, and phenotype in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/jlb.2hi0720-463rropenalex-snapshot
Novel TREM2 splicing isoform that lacks the V-set immunoglobulin domain is abundant in the human brain
inferred← citeshttps://doi.org/10.1002/jlb.3mr1021-531ropenalex-snapshot
Microglial activation in Alzheimer's disease: The role of flavonoids and microRNAs
inferred← citeshttps://doi.org/10.1002/jlb.3ru0820-536ropenalex-snapshot
Functional consequences of a close encounter between microglia and brain-infiltrating monocytes during CNS pathology and repair
inferred← citeshttps://doi.org/10.1002/jlb.mr0818-319ropenalex-snapshot
Microglia in Alzheimer’s disease: A target for immunotherapy
inferred← citeshttps://doi.org/10.1002/jms.5008openalex-snapshot
Spatial neurolipidomics—MALDI mass spectrometry imaging of lipids in brain pathologies
inferred← citeshttps://doi.org/10.1002/jnr.24064openalex-snapshot
The vicious circle of hypometabolism in neurodegenerative diseases: Ways and mechanisms of metabolic correction
inferred← citeshttps://doi.org/10.1002/mco2.70789openalex-snapshot
Beyond the Known and Established Neurodegenerative Effects: Roles of APOE Across a Wide Spectrum of Pathophysiological Condition
inferred← citeshttps://doi.org/10.1002/mds.27718openalex-snapshot
The A1 astrocyte paradigm: New avenues for pharmacological intervention in neurodegeneration
inferred← citeshttps://doi.org/10.1002/mds.27911openalex-snapshot
Association Between Toll‐Like Receptor 4 (TLR4) and Triggering Receptor Expressed on Myeloid Cells 2 (TREM2) Genetic Variants and Clinical Progression of Huntington's Disease
inferred← citeshttps://doi.org/10.1002/mds.27992openalex-snapshot
Microglia‐Related Gene Triggering Receptor Expressed in Myeloid Cells 2 (TREM2) Is Upregulated in the Substantia Nigra of Progressive Supranuclear Palsy
inferred← citeshttps://doi.org/10.1002/mds.28089openalex-snapshot
Dysregulated Brain Cholesterol Metabolism Is Linked to Neuroinflammation in Huntington's Disease
inferred← citeshttps://doi.org/10.1002/mgg3.2476openalex-snapshot
Homozygous TREM2 c.549del; p.( Leu184Serfs *5) variant causing Nasu‐Hakola disease in three siblings in a consanguineous Iraqi family: Case report and review of literature
inferred← citeshttps://doi.org/10.1002/mnfr.70168openalex-snapshot
Exploring the Impact of a High‐Fat Diet on Brain Homeostasis: A Comprehensive Analysis of the Absence of Inflammation
inferred← citeshttps://doi.org/10.1002/pdi3.70024openalex-snapshot
TREM2 Downregulation Disrupts Microglial Function and Synaptic Pruning Through RA/RARα Signaling: Mechanisms Underlying Autism‐Like Behaviors
inferred← citeshttps://doi.org/10.1002/pmic.202200183openalex-snapshot
Advances in proteomic phenotyping of microglia in neurodegeneration
inferred← citeshttps://doi.org/10.1002/psp4.13095openalex-snapshot
Microglial roles in Alzheimer's disease: An agent‐based model to elucidate microglial spatiotemporal response to beta‐amyloid
inferred← citeshttps://doi.org/10.1002/syn.22161openalex-snapshot
Triggering receptor expressed on myeloid cells 2 activation downregulates toll‐like receptor 4 expression and ameliorates cognitive impairment in the Aβ1‐42‐induced Alzheimer's disease mouse model
inferred← citeshttps://doi.org/10.1002/syn.22196openalex-snapshot
Mitochondria‐associated endoplasmic reticulum membranes: At the crossroad between familiar and sporadic Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/trc2.12070openalex-snapshot
Toward refining Alzheimer's disease into overlapping subgroups
inferred← citeshttps://doi.org/10.1002/trc2.12110openalex-snapshot
Model organism development and evaluation for late‐onset Alzheimer's disease: MODEL‐AD
inferred← citeshttps://doi.org/10.1002/trc2.12211openalex-snapshot
GenoRisk: A polygenic risk score for Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/trc2.12462openalex-snapshot
The power of representation: Statistical analysis of diversity in US Alzheimer's disease genetics data
inferred← citeshttps://doi.org/10.1002/trc2.70037openalex-snapshot
Data‐driven discovery of associations between prescribed drugs and dementia risk: A systematic review
inferred← citeshttps://doi.org/10.1002/wsbm.1310openalex-snapshot
Integrative biomarker discovery in neurodegenerative diseases
inferred← citeshttps://doi.org/10.1002/wsbm.1591openalex-snapshot
Beyond the classical amyloid hypothesis in Alzheimer's disease: Molecular insights into current concepts of pathogenesis, therapeutic targets, and study models
inferred← citeshttps://doi.org/10.1007/978-1-0716-2265-0_13openalex-snapshot
Systems Biology to Address Unmet Medical Needs in Neurological Disorders
inferred← citeshttps://doi.org/10.1007/978-1-0716-4200-9_5openalex-snapshot
Biomarkers Unveiling the Interplay of Mind, Nervous System, and Immunity
inferred← citeshttps://doi.org/10.1007/978-1-4471-6380-0_3openalex-snapshot
Autosomal Dominant Alzheimer’s Disease: Underlying Causes
inferred← citeshttps://doi.org/10.1007/978-1-4471-6380-0_4openalex-snapshot
Genetic Risk Factors for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4614-3209-8_27openalex-snapshot
Microglia in the Aging Retina
inferred← citeshttps://doi.org/10.1007/978-1-4614-7309-1_1openalex-snapshot
The Genetics of Alzheimer’s Disease: Introduction and Perspective for the Future
inferred← citeshttps://doi.org/10.1007/978-1-4614-7309-1_12openalex-snapshot
Other Genes Implicated in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4614-9123-1_14openalex-snapshot
Degenerative Brain Diseases and White Matter Injury
inferred← citeshttps://doi.org/10.1007/978-1-4939-1429-6_18openalex-snapshot
Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1007/978-1-4939-1670-2_7openalex-snapshot
Can Microbes Play a Role in the Pathogenesis of Alzheimer Disease?
inferred← citeshttps://doi.org/10.1007/978-1-4939-2247-5_1openalex-snapshot
An Overview of Methods Used in Neurogenomics and Their Applications
inferred← citeshttps://doi.org/10.1007/978-1-4939-2627-5_17openalex-snapshot
Next Generation Sequencing in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-2627-5_18openalex-snapshot
Pooled-DNA Sequencing for Elucidating New Genomic Risk Factors, Rare Variants Underlying Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-2627-5_2openalex-snapshot
Application of Systems Theory in Longitudinal Studies on the Origin and Progression of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-2627-5_25openalex-snapshot
AlzPathway, an Updated Map of Curated Signaling Pathways: Towards Deciphering Alzheimer’s Disease Pathogenesis
inferred← citeshttps://doi.org/10.1007/978-1-4939-2627-5_29openalex-snapshot
Network-Based Analysis for Uncovering Mechanisms Underlying Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-3661-8_18openalex-snapshot
Better Utilization of Mouse Models of Neurodegenerative Diseases in Preclinical Studies: From the Bench to the Clinic
inferred← citeshttps://doi.org/10.1007/978-1-4939-3777-6_6openalex-snapshot
Neurodegenerative Diseases and Dementia
inferred← citeshttps://doi.org/10.1007/978-1-4939-7674-4_11openalex-snapshot
A Selected Reaction Monitoring Protocol for the Measurement of sTREM2 in Cerebrospinal Fluid
inferred← citeshttps://doi.org/10.1007/978-1-4939-9658-2_21openalex-snapshot
In Vivo Phagocytosis Analysis of Amyloid Beta
inferred← citeshttps://doi.org/10.1007/978-1-4939-9658-2_5openalex-snapshot
Microglia in Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1007/978-3-030-05542-4_5openalex-snapshot
Genetic Risk Factors for Alzheimer Disease: Emerging Roles of Microglia in Disease Pathomechanisms
inferred← citeshttps://doi.org/10.1007/978-3-030-14792-1_5openalex-snapshot
DNA Methylation in Neuronal Development and Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-17935-9_11openalex-snapshot
Mathematical Modeling and Docking of Medicinal Plants and Synthetic Drugs to Determine Their Effects on Abnormal Expression of Cholinesterase and Acetyl Cholinesterase Proteins in Alzheimer
inferred← citeshttps://doi.org/10.1007/978-3-030-38266-7_8openalex-snapshot
The Ubiquitin System in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-51140-1_9openalex-snapshot
Fluid Biomarkers of Frontotemporal Lobar Degeneration
inferred← citeshttps://doi.org/10.1007/978-3-030-57267-9_20openalex-snapshot
Mild Cognitive Impairment and Early Dementia
inferred← citeshttps://doi.org/10.1007/978-3-030-71519-9_180-1openalex-snapshot
Microglial Cell Dysregulation in the Aged Brain and Neurodegeneration
inferred← citeshttps://doi.org/10.1007/978-3-031-15080-7_180openalex-snapshot
Microglial Cell Dysregulation in the Aged Brain and Neurodegeneration
inferred← citeshttps://doi.org/10.1007/978-3-031-31570-1_14openalex-snapshot
Alzheimer’s Research Gains Momentum and Spreads Out
inferred← citeshttps://doi.org/10.1007/978-3-031-31570-1_16openalex-snapshot
Beyond APP, PSEN1, PSEN2, and APOE: What Else Does the Genome Tell Us?
inferred← citeshttps://doi.org/10.1007/978-3-031-31978-5_30openalex-snapshot
Pathological Roles of INPP5D in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-55529-9_21openalex-snapshot
Aging Microglia and Their Impact in the Nervous System
inferred← citeshttps://doi.org/10.1007/978-3-031-55529-9_35openalex-snapshot
Emerging Microglial Therapies and Targets in Clinical Trial
inferred← citeshttps://doi.org/10.1007/978-3-031-55529-9_7openalex-snapshot
Calcium Signalling in Microglia
inferred← citeshttps://doi.org/10.1007/978-3-031-68237-7_3openalex-snapshot
Immune Sensors, Regulators, and Effectors of Brain Health
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