On the public citation graph

Suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, ameliorates motor deficits in a mouse model of Huntington's disease

0 signed · 842 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/9780470751077openalex-snapshot

Handbook of Laboratory Animal Management and Welfare

inferredcites →https://doi.org/10.1002/ana.10094openalex-snapshot

Environmental enrichment slows disease progression in R6/2 Huntington's disease mice

inferredcites →https://doi.org/10.1002/sim.4780090710openalex-snapshot

More powerful procedures for multiple significance testing

inferredcites →https://doi.org/10.1006/nbdi.2001.0438openalex-snapshot

Inhibition of Polyglutamine Aggregation in R6/2 HD Brain Slices—Complex Dose–Response Profiles

inferredcites →https://doi.org/10.1016/s0092-8674(00)80513-9openalex-snapshot

Formation of Neuronal Intranuclear Inclusions Underlies the Neurological Dysfunction in Mice Transgenic for the HD Mutation

inferredcites →https://doi.org/10.1016/s0092-8674(00)81369-0openalex-snapshot

Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic Mice

inferredcites →https://doi.org/10.1021/cr970025popenalex-snapshot

Cyclodextrin Drug Carrier Systems

inferredcites →https://doi.org/10.1038/35099568openalex-snapshot

Histone deacetylase inhibitors arrest polyglutamine-dependent neurodegeneration in Drosophila

inferredcites →https://doi.org/10.1038/35106079openalex-snapshot

Histone deacetylases and cancer: causes and therapies

inferredcites →https://doi.org/10.1038/77528openalex-snapshot

Minocycline inhibits caspase-1 and caspase-3 expression and delays mortality in a transgenic mouse model of Huntington disease

inferredcites →https://doi.org/10.1038/79139openalex-snapshot

Expanded polyglutamine stretches interact with TAFII130, interfering with CREB-dependent transcription

inferredcites →https://doi.org/10.1038/ng0297-197openalex-snapshot

Instability of highly expanded CAG repeats in mice transgenic for the Huntington's disease mutation

inferredcites →https://doi.org/10.1038/nm0202-143openalex-snapshot

Prolonged survival and decreased abnormal movements in transgenic model of Huntington disease, with administration of the transglutaminase inhibitor cystamine

inferredcites →https://doi.org/10.1073/pnas.100110097openalex-snapshot

The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription

inferredcites →https://doi.org/10.1073/pnas.180316197openalex-snapshot

Histone deacetylase inhibitor selectively induces p21 WAF1 expression and gene-associated histone acetylation

inferredcites →https://doi.org/10.1073/pnas.191498198openalex-snapshot

Altered transcription in yeast expressing expanded polyglutamine

inferredcites →https://doi.org/10.1073/pnas.261400698openalex-snapshot

Histone deacetylase inhibitors reduce polyglutamine toxicity

inferredcites →https://doi.org/10.1073/pnas.95.11.6480openalex-snapshot

Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human Huntington disease gene

inferredcites →https://doi.org/10.1073/pnas.95.6.3003openalex-snapshot

A class of hybrid polar inducers of transformed cell differentiation inhibits histone deacetylases

inferredcites →https://doi.org/10.1073/pnas.96.20.11404openalex-snapshot

Insoluble detergent-resistant aggregates form between pathological and nonpathological lengths of polyglutamine in mammalian cells

inferredcites →https://doi.org/10.1093/hmg/10.21.2425openalex-snapshot

Centrosome disorganization in fibroblast cultures derived from R6/2 Huntington's disease (HD) transgenic mice and HD patients

inferredcites →https://doi.org/10.1093/hmg/11.17.1911openalex-snapshot

Dysregulation of gene expression in the R6/2 model of polyglutamine disease: parallel changes in muscle and brain

inferredcites →https://doi.org/10.1093/hmg/9.9.1259openalex-snapshot

Decreased expression of striatal signaling genes in a mouse model of Huntington's disease

inferredcites →https://doi.org/10.1093/jnci/92.15.1210openalex-snapshot

Histone Deacetylase Inhibitors: Inducers of Differentiation or Apoptosis of Transformed Cells

inferredcites →https://doi.org/10.1126/science.1056784openalex-snapshot

Interference by Huntingtin and Atrophin-1 with CBP-Mediated Transcription Leading to Cellular Toxicity

inferredcites →https://doi.org/10.1126/science.1072613openalex-snapshot

Sp1 and TAFII130 Transcriptional Activity Disrupted in Early Huntington's Disease

inferredcites →https://doi.org/10.1128/mcb.22.5.1277-1287.2002openalex-snapshot

Interaction of Huntington Disease Protein with Transcriptional Activator Sp1

inferredcites →https://doi.org/10.1523/jneurosci.20-12-04389.2000openalex-snapshot

Neuroprotective Effects of Creatine in a Transgenic Mouse Model of Huntington's Disease

inferredcites →https://doi.org/10.1523/jneurosci.22-05-01592.2002openalex-snapshot

Therapeutic Effects of Coenzyme Q10and Remacemide in Transgenic Mouse Models of Huntington's Disease

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.5281/zenodo.886877openalex-extracted

Lysine (K)-Deacetylase Inhibitors: The Real Next Step To Neuropsychiatric And Neurodegenerative Disorders?

inferred← citeshttps://doi.org/10.1002/0470022574.ch5openalex-snapshot

Huntington's Disease and Polyglutamine Expansion Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1002/9780470027318.a9365openalex-snapshot

Histone Deacetylases ( HDACs ): Function, Mechanism, & Inhibition

inferred← citeshttps://doi.org/10.1002/9780470508169.ch35openalex-snapshot

Histone Deacetylase Inhibitors

inferred← citeshttps://doi.org/10.1002/9780470572702.ch40openalex-snapshot

Therapeutic Prospects for Polyglutamine Disease

inferred← citeshttps://doi.org/10.1002/9780470917060.ch13openalex-snapshot

A Neurodevelopmental Origin for Pakinson's Disease: A Link to the Fetal Basis for Adult Disease Hypothesis

inferred← citeshttps://doi.org/10.1002/9781118349045.ch4openalex-snapshot

Chromatin at the Intersection of Disease and Therapy

inferred← citeshttps://doi.org/10.1002/9781118540770.ch14openalex-snapshot

Epigenetics in Brain Development and Disease

inferred← citeshttps://doi.org/10.1002/9781118553480.ch08openalex-snapshot

Huntington's Disease and Polyglutamine Expansion Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1002/9781118661895.ch21openalex-snapshot

Huntington's Disease and Other Choreas

inferred← citeshttps://doi.org/10.1002/9783527619375.ch12openalex-snapshot

The Bicyclic Depsipeptide Family of Histone Deacetylase Inhibitors

inferred← citeshttps://doi.org/10.1002/acn3.147openalex-snapshot

The promise and perils of HDAC inhibitors in neurodegeneration

inferred← citeshttps://doi.org/10.1002/advs.202301367openalex-snapshot

The Lysine Demethylase KDM7A Regulates Immediate Early Genes in Neurons

inferred← citeshttps://doi.org/10.1002/ajmg.a.38494openalex-snapshot

Therapy development in Huntington disease: From current strategies to emerging opportunities

inferred← citeshttps://doi.org/10.1002/ange.200703198openalex-snapshot

Fluorous‐Based Small‐Molecule Microarrays for the Discovery of Histone Deacetylase Inhibitors

inferred← citeshttps://doi.org/10.1002/anie.200703198openalex-snapshot

Fluorous‐Based Small‐Molecule Microarrays for the Discovery of Histone Deacetylase Inhibitors

inferred← citeshttps://doi.org/10.1002/bies.20029openalex-snapshot

Can flies help humans treat neurodegenerative diseases?

inferred← citeshttps://doi.org/10.1002/biot.201300560openalex-snapshot

Stem cell therapy and cellular engineering for treatment of neuronal dysfunction in Huntington's disease

inferred← citeshttps://doi.org/10.1002/bkcs.11366openalex-snapshot

Identification of Novel Human HDAC8 Inhibitors by Pharmacophore‐based Virtual Screening and Density Functional Theory Approaches

inferred← citeshttps://doi.org/10.1002/cne.22241openalex-snapshot

Regional and subcellular distribution of HDAC4 in mouse brain

inferred← citeshttps://doi.org/10.1002/dmrr.559openalex-snapshot

Role of histone and transcription factor acetylation in diabetes pathogenesis

inferred← citeshttps://doi.org/10.1002/dneu.20637openalex-snapshot

HDAC4 inhibits cell‐cycle progression and protects neurons from cell death

inferred← citeshttps://doi.org/10.1002/embj.201387637openalex-snapshot

Epigenetic memory: the Lamarckian brain

inferred← citeshttps://doi.org/10.1002/glia.20098openalex-snapshot

Glial and neuronal expression of polyglutamine proteins induce behavioral changes and aggregate formation in Drosophila

inferred← citeshttps://doi.org/10.1002/ibd.22934openalex-snapshot

Epigenetics: Concepts and relevance to IBD pathogenesis

inferred← citeshttps://doi.org/10.1002/ijch.202000021openalex-snapshot

Challenges in RNA Regulation in Huntington's Disease: Insights from Computational Studies

inferred← citeshttps://doi.org/10.1002/jcp.24362openalex-snapshot

Sorafenib and HDAC inhibitors synergize with TRAIL to kill tumor cells

inferred← citeshttps://doi.org/10.1002/jcp.24871openalex-snapshot

HDAC4 Degradation Mediates HDAC Inhibition‐Induced Protective Effects Against Hypoxia/Reoxygenation Injury

inferred← citeshttps://doi.org/10.1002/jcp.28622openalex-snapshot

Involvement of aberrant regulation of epigenetic mechanisms in the pathogenesis of Parkinson's disease and epigenetic‐based therapies

inferred← citeshttps://doi.org/10.1002/mds.21632openalex-snapshot

Sodium phenylbutyrate in Huntington's disease: A dose‐finding study

inferred← citeshttps://doi.org/10.1002/mds.26007openalex-snapshot

Targets for future clinical trials in Huntington's disease: What's in the pipeline?

inferred← citeshttps://doi.org/10.1002/med.21425openalex-snapshot

Discovery of Therapeutic Approaches for Polyglutamine Diseases: A Summary of Recent Efforts

inferred← citeshttps://doi.org/10.1002/mrdd.20083openalex-snapshot

Mental retardation genes in drosophila: New approaches to understanding and treating developmental brain disorders

inferred← citeshttps://doi.org/10.1002/pbc.24541openalex-snapshot

A pediatric phase 1 trial of vorinostat and temozolomide in relapsed or refractory primary brain or spinal cord tumors: A children's oncology group phase 1 consortium study

inferred← citeshttps://doi.org/10.1002/prca.200800018openalex-snapshot

Age‐dependent gene expression profile and protein expression in a transgenic rat model of Huntington's disease

inferred← citeshttps://doi.org/10.1007/0-387-27421-9_31openalex-snapshot

The Androgen Receptor and Spinal and Bulbar Muscular Atrophy

inferred← citeshttps://doi.org/10.1007/1-4020-5466-1_12openalex-snapshot

Chromatin Acetylation Status in the Manifestation of Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1007/3-540-29623-9_0770openalex-snapshot

Polyglutamine Disease, the Emerging Role of Transcription Interference

inferred← citeshttps://doi.org/10.1007/3-540-33336-3_12openalex-snapshot

Current Issues and Therapeutic Prospects

inferred← citeshttps://doi.org/10.1007/3-540-33336-3_9openalex-snapshot

Polyglutamine Diseases

inferred← citeshttps://doi.org/10.1007/7355_2010_10openalex-snapshot

The Role of Histone Deacetylases in Neurodegenerative Diseases and Small-Molecule Inhibitors as a Potential Therapeutic Approach

inferred← citeshttps://doi.org/10.1007/7854_2013_250openalex-snapshot

Therapeutic Strategies for Huntington’s Disease

inferred← citeshttps://doi.org/10.1007/978-0-387-30377-2_11openalex-snapshot

Huntington's Disease: Unraveling the Pathophysiological Cascade Behind a Singular Gene Defect

inferred← citeshttps://doi.org/10.1007/978-0-387-30379-6_12openalex-snapshot

Polyglutamine Diseases

inferred← citeshttps://doi.org/10.1007/978-0-387-36534-3_23openalex-snapshot

Mechanistic Insights into the Polyglutamine Ataxias

inferred← citeshttps://doi.org/10.1007/978-0-387-36534-3_24openalex-snapshot

Molecular Pathogenesis of the Polyglutamine Disease: Spinal and Bulbar Muscular Atrophy

inferred← citeshttps://doi.org/10.1007/978-0-387-40049-5_6openalex-snapshot

HATs and HDACs

inferred← citeshttps://doi.org/10.1007/978-0-387-70830-0_18openalex-snapshot

Polyglutamine Expansion Diseases – the Case of Machado-Joseph Disease

inferred← citeshttps://doi.org/10.1007/978-0-387-73609-9_7openalex-snapshot

CREB-Dependent Transcription and Synaptic Plasticity

inferred← citeshttps://doi.org/10.1007/978-1-4419-5638-5_11openalex-snapshot

Creating a Pro-survival and Anti-inflammatory Phenotype by Modulation of Acetylation in Models of Hemorrhagic and Septic Shock

inferred← citeshttps://doi.org/10.1007/978-1-4419-6448-9_5openalex-snapshot

Huntington’s Disease

inferred← citeshttps://doi.org/10.1007/978-1-4471-6455-5_6openalex-snapshot

Huntington’s Disease: Molecular Pathogenesis and New Therapeutic Perspectives

inferred← citeshttps://doi.org/10.1007/978-1-4939-1655-9_24openalex-snapshot

Latent HIV-1 Infection of Resting CD4+ T cells: Testing Approaches to Overcome HIV Latency

inferred← citeshttps://doi.org/10.1007/978-1-4939-2528-5_30openalex-snapshot

Novel Targets for Drug Treatment in Psychiatry

inferred← citeshttps://doi.org/10.1007/978-1-4939-7825-0_5openalex-snapshot

Nonmammalian Models of Huntington’s Disease

inferred← citeshttps://doi.org/10.1007/978-1-59259-852-6_23openalex-snapshot

Dopamine and Glutamate in Huntington’s Disease

inferred← citeshttps://doi.org/10.1007/978-1-59259-963-9_116openalex-snapshot

Trinucleotide Repeat Disorders

inferred← citeshttps://doi.org/10.1007/978-1-62703-411-1openalex-snapshot

Trinucleotide Repeat Protocols

inferred← citeshttps://doi.org/10.1007/978-3-030-76320-6_9openalex-snapshot

Neurological Disorders: Biochemistry of Drug Resistance and Future Challenges

inferred← citeshttps://doi.org/10.1007/978-3-031-24930-3_4openalex-snapshot

Translatable Models of Brain and Cognitive Reserve

inferred← citeshttps://doi.org/10.1007/978-3-031-75980-2_4openalex-snapshot

The Promise of Epigenetic Editing for Treating Brain Disorders

inferred← citeshttps://doi.org/10.1007/978-3-319-05392-9_33openalex-snapshot

Animal Models of Neurological Disorders

inferred← citeshttps://doi.org/10.1007/978-3-319-24951-3_8openalex-snapshot

Epigenetic Post transcriptional Mutation in Neuro-Oncology

inferred← citeshttps://doi.org/10.1007/978-3-319-52067-4_4openalex-snapshot

Translatable Models of Brain and Cognitive Reserve

inferred← citeshttps://doi.org/10.1007/978-3-540-70995-4_6openalex-snapshot

Psychotropic and Neurotropic Activity

inferred← citeshttps://doi.org/10.1007/978-3-642-17426-1_12openalex-snapshot

Histone Deacetylase Inhibitors: A Novel Therapeutic Approach for Cognitive Disorders

inferred← citeshttps://doi.org/10.1007/978-3-642-27728-3_33-1openalex-snapshot

Animal Models of Neurological Disorders

inferred← citeshttps://doi.org/10.1007/978-3-642-27728-3_33-2openalex-snapshot

Animal Models of Neurological Disorders

inferred← citeshttps://doi.org/10.1007/978-4-431-55763-0_23openalex-snapshot

Therapeutics for Polyglutamine Diseases Through Protein Degradation Pathway: Targeting the Nucleus

inferred← citeshttps://doi.org/10.1007/978-981-10-5981-0openalex-snapshot

Animal Models of Neurological Disorders

inferred← citeshttps://doi.org/10.1007/978-981-13-0529-0_5openalex-snapshot

Repeat Expansion Disease Models

inferred← citeshttps://doi.org/10.1007/978-981-13-8019-8_6openalex-snapshot

HDAC Inhibitors as Novel Therapeutic Option Against Therapeutically Challenging Neurological Disorders

inferred← citeshttps://doi.org/10.1007/978-981-97-4288-2_2openalex-snapshot

Mechanism Underlying Major Neurodegenerative Disorders

inferred← citeshttps://doi.org/10.1007/978-981-99-2657-2_10openalex-snapshot

Epigenetic Therapy for Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/bf02686117openalex-snapshot

Distribution of histone deacetylases 1–11 in the rat brain

inferred← citeshttps://doi.org/10.1007/bf03033279openalex-snapshot

Gene-environment interplay in neurogenesis and neurodegeneration

inferred← citeshttps://doi.org/10.1007/bf03206573openalex-snapshot

The paradigm of Huntington’s disease: Therapeutic opportunities in neurodegeneration

inferred← citeshttps://doi.org/10.1007/s00018-006-6242-0openalex-snapshot

Huntington’s disease: from huntingtin function and dysfunction to therapeutic strategies

inferred← citeshttps://doi.org/10.1007/s00018-007-7035-9openalex-snapshot

Histone deacetylases: Focus on the nervous system

inferred← citeshttps://doi.org/10.1007/s00018-013-1296-2openalex-snapshot

The role of genetics in the establishment and maintenance of the epigenome

inferred← citeshttps://doi.org/10.1007/s00018-018-2854-4openalex-snapshot

Mechanisms of protein toxicity in neurodegenerative diseases

inferred← citeshttps://doi.org/10.1007/s00018-022-04280-8openalex-snapshot

Modifier pathways in polyglutamine (PolyQ) diseases: from genetic screens to drug targets

inferred← citeshttps://doi.org/10.1007/s00109-004-0530-7openalex-snapshot

Spinal and bulbar muscular atrophy: ligand-dependent pathogenesis and therapeutic perspectives

inferred← citeshttps://doi.org/10.1007/s00109-004-0632-2openalex-snapshot

A novel therapeutic strategy for polyglutamine diseases by stabilizing aggregation-prone proteins with small molecules

inferred← citeshttps://doi.org/10.1007/s00280-007-0622-5openalex-snapshot

Plasma and cerebrospinal fluid pharmacokinetics of the histone deacetylase inhibitor, belinostat (PXD101), in non-human primates

inferred← citeshttps://doi.org/10.1007/s00280-010-1302-4openalex-snapshot

The histone deacetylase inhibitor vorinostat induces calreticulin exposure in childhood brain tumour cells in vitro

inferred← citeshttps://doi.org/10.1007/s00401-006-0082-8openalex-snapshot

Ex vivo therapy of malignant melanomas transplanted into organotypic brain slice cultures using inhibitors of histone deacetylases

inferred← citeshttps://doi.org/10.1007/s00401-017-1732-8openalex-snapshot

Remodeling of heterochromatin structure slows neuropathological progression and prolongs survival in an animal model of Huntington’s disease

inferred← citeshttps://doi.org/10.1007/s00439-013-1271-xopenalex-snapshot

Genetic syndromes caused by mutations in epigenetic genes

inferred← citeshttps://doi.org/10.1007/s00702-004-0201-4openalex-snapshot

Huntington?s disease: pathomechanism and therapeutic perspectives

inferred← citeshttps://doi.org/10.1007/s00702-010-0430-7openalex-snapshot

Neuroinflammation in Huntington’s disease

inferred← citeshttps://doi.org/10.1007/s10048-003-0169-5openalex-snapshot

Genetic and environmental factors in the pathogenesis of Huntington’s disease

inferred← citeshttps://doi.org/10.1007/s10142-011-0246-zopenalex-snapshot

Emerging roles of epigenetic mechanisms in Parkinson’s disease

inferred← citeshttps://doi.org/10.1007/s10517-011-1201-7openalex-snapshot

Time Course of Histone Deacetylase 1 and Acetylated H3 and H4 Histones in the Brain of Rats Treated with Ladasten

inferred← citeshttps://doi.org/10.1007/s10571-020-00979-zopenalex-snapshot

Epigenetic Changes and Its Intervention in Age-Related Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1007/s10571-021-01133-zopenalex-snapshot

The Essential Role of Epigenetic Modifications in Neurodegenerative Diseases with Dyskinesia

inferred← citeshttps://doi.org/10.1007/s11033-021-06787-yopenalex-snapshot

Zebrafish an experimental model of Huntington’s disease: molecular aspects, therapeutic targets and current challenges

inferred← citeshttps://doi.org/10.1007/s11060-006-9142-0openalex-snapshot

Suberoylanilide hydroxamic acid is effective in preclinical studies of medulloblastoma

inferred← citeshttps://doi.org/10.1007/s11060-007-9337-zopenalex-snapshot

Continuous intracranial administration of suberoylanilide hydroxamic acid (SAHA) inhibits tumor growth in an orthotopic glioma model

inferred← citeshttps://doi.org/10.1007/s11060-011-0604-7openalex-snapshot

Vorinostat modulates cell cycle regulatory proteins in glioma cells and human glioma slice cultures

inferred← citeshttps://doi.org/10.1007/s11060-014-1433-2openalex-snapshot

Vorinostat as a radiosensitizer for brain metastasis: a phase I clinical trial

inferred← citeshttps://doi.org/10.1007/s11064-021-03367-1openalex-snapshot

Treatment with Histone Deacetylase Inhibitor Attenuates Peripheral Inflammation-Induced Cognitive Dysfunction and Microglial Activation: The Effect of SAHA as a Peripheral HDAC Inhibitor

inferred← citeshttps://doi.org/10.1007/s11910-003-0028-7openalex-snapshot

Invertebrate models of neurologic disease: Insights into pathogenesis and therapy

inferred← citeshttps://doi.org/10.1007/s11910-006-0019-6openalex-snapshot

Update on huntington’s disease

inferred← citeshttps://doi.org/10.1007/s12017-021-08660-4openalex-snapshot

Repurposing Vorinostat for the Treatment of Disorders Affecting Brain

inferred← citeshttps://doi.org/10.1007/s12035-009-8067-yopenalex-snapshot

Focal Nature of Neurological Disorders Necessitates Isotype-Selective Histone Deacetylase (HDAC) Inhibitors

inferred← citeshttps://doi.org/10.1007/s12035-012-8316-3openalex-snapshot

Mouse Models of Polyglutamine Diseases in Therapeutic Approaches: Review and Data Table. Part II

inferred← citeshttps://doi.org/10.1007/s12035-014-9058-1openalex-snapshot

c-Fos Protects Neurons Through a Noncanonical Mechanism Involving HDAC3 Interaction: Identification of a 21-Amino Acid Fragment with Neuroprotective Activity

inferred← citeshttps://doi.org/10.1007/s12038-008-0047-2openalex-snapshot

Non-coding RNAs in polyglutamine disorders: friend or foe?

inferred← citeshttps://doi.org/10.1007/s12041-010-0072-4openalex-snapshot

Modifiers and mechanisms of multi-system polyglutamine neurodegenerative disorders: lessons from fly models

inferred← citeshttps://doi.org/10.1007/s12192-023-01395-0openalex-snapshot

Recombinant cellular model system for human muscle-type nicotinic acetylcholine receptor α12β1δε

inferred← citeshttps://doi.org/10.1007/s12311-009-0144-2openalex-snapshot

Cellular and Molecular Pathways Triggering Neurodegeneration in the Spinocerebellar Ataxias

inferred← citeshttps://doi.org/10.1007/s12975-015-0421-yopenalex-snapshot

The Histone Deacetylase Inhibitor Suberoylanilide Hydroxamic Acid (SAHA) Confers Acute Neuroprotection After Intracerebral Hemorrhage in Mice

inferred← citeshttps://doi.org/10.1007/s13148-011-0025-7openalex-snapshot

Targeting Huntington’s disease through histone deacetylases

inferred← citeshttps://doi.org/10.1007/s13311-012-0112-2openalex-snapshot

Huntington's Disease and the Striatal Medium Spiny Neuron: Cell-Autonomous and Non-Cell-Autonomous Mechanisms of Disease

inferred← citeshttps://doi.org/10.1007/s13311-013-0195-4openalex-snapshot

Experimental Models for Identifying Modifiers of Polyglutamine-Induced Aggregation and Neurodegeneration

inferred← citeshttps://doi.org/10.1007/s13311-013-0206-5openalex-snapshot

Epigenetic Mechanisms of Neurodegeneration in Huntington's Disease

inferred← citeshttps://doi.org/10.1007/s13311-013-0217-2openalex-snapshot

Treatment of Niemann–Pick Type C Disease by Histone Deacetylase Inhibitors

inferred← citeshttps://doi.org/10.1007/s13311-013-0219-0openalex-snapshot

Epigenetics of Alzheimer's Disease and Frontotemporal Dementia

inferred← citeshttps://doi.org/10.1007/s13353-016-0367-0openalex-snapshot

Relative efficacy of nicotinamide treatment of a mouse model of infantile Niemann-Pick C1 disease

inferred← citeshttps://doi.org/10.1007/s13365-014-0287-xopenalex-snapshot

HIV-1 target cells in the CNS

inferred← citeshttps://doi.org/10.1016/b978-0-12-373861-5.00018-7openalex-snapshot

Huntington Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-374028-1.00024-5openalex-snapshot

Parkinson's Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-374767-9.00030-5openalex-snapshot

Chromatin Remodeling

inferred← citeshttps://doi.org/10.1016/b978-0-12-381328-2.00015-8openalex-snapshot

Pathogenic Mechanisms in Huntington's Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-381328-2.00016-xopenalex-snapshot

Experimental Models of HD and Reflection on Therapeutic Strategies

inferred← citeshttps://doi.org/10.1016/b978-0-12-385883-2.00010-2openalex-snapshot

Huntington Disease and the Huntingtin Protein

inferred← citeshttps://doi.org/10.1016/b978-0-12-391494-1.00008-2openalex-snapshot

HDAC Inhibitors as Novel Therapeutics in Aging and Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-407198-8.00006-0openalex-snapshot

PAK1 in Alzheimer’s and Huntington’s Diseases

inferred← citeshttps://doi.org/10.1016/b978-0-12-800226-1.00004-6openalex-snapshot

Epigenetics and cognitive disorders—translational aspects

inferred← citeshttps://doi.org/10.1016/b978-0-12-800681-8.00002-5openalex-snapshot

The Functional Genome

inferred← citeshttps://doi.org/10.1016/b978-0-12-800684-9.00017-4openalex-snapshot

The functional genome: epigenetics and epigenomics

inferred← citeshttps://doi.org/10.1016/b978-0-12-801893-4.00002-xopenalex-snapshot

Mechanisms underlying neurodegeneration in Huntington disease: applications to novel disease-modifying therapies

inferred← citeshttps://doi.org/10.1016/b978-0-12-801899-6.00026-7openalex-snapshot

Chromatin Landscape and Epigenetic Signatures in Neurological Disorders

inferred← citeshttps://doi.org/10.1016/b978-0-12-802206-1.00032-5openalex-snapshot

Epigenetics in Neuropathologies of the Basal Ganglia

inferred← citeshttps://doi.org/10.1016/b978-0-12-803239-8.00003-xopenalex-snapshot

Histone Deacetylases as Therapeutic Targets in Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1016/b978-0-12-803685-3.00016-1openalex-snapshot

Spinal Muscular Atrophy Therapeutics Development

inferred← citeshttps://doi.org/10.1016/b978-0-12-805420-8.00007-xopenalex-snapshot

The Role of Histone Deacetylase Inhibition in the Accumulation and Stability of Disease-Related Proteins

inferred← citeshttps://doi.org/10.1016/b978-0-12-811069-0.00006-9openalex-snapshot

Huntington’s Chorea

inferred← citeshttps://doi.org/10.1016/b978-0-12-813796-3.00004-3openalex-snapshot

Epigenetic mechanisms in Huntington's disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-815762-6.00002-0openalex-snapshot

Physiology of cellular demise: Apoptosis, necrosis, and autophagy

inferred← citeshttps://doi.org/10.1016/b978-0-12-817819-5.00008-5openalex-snapshot

Chromatin, histones, and histone modifications in health and disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-823931-5.00014-1openalex-snapshot

Epigenetic regulations in inflammatory diseases

inferred← citeshttps://doi.org/10.1016/b978-0-323-31071-0.00013-5openalex-snapshot

Huntington disease

inferred← citeshttps://doi.org/10.1016/b978-0-323-95539-3.00009-0openalex-snapshot

Detection of genetic and epigenetic markers in neurodegenerative disorders

inferred← citeshttps://doi.org/10.1016/b978-0-443-07941-2.50018-8openalex-snapshot

Huntington Disease

inferred← citeshttps://doi.org/10.1016/b978-0-443-18516-8.00016-8openalex-snapshot

Epigenetics and cognitive disorders—translational aspects

inferred← citeshttps://doi.org/10.1016/b978-0-443-29898-1.00001-0openalex-snapshot

Application of brain organoids in Huntington's disease

inferred← citeshttps://doi.org/10.1016/b978-0-444-51892-7.00046-2openalex-snapshot

Treatment and management issues in ataxic diseases

inferred← citeshttps://doi.org/10.1016/b978-0-444-52002-9.00030-9openalex-snapshot

Huntington's disease

inferred← citeshttps://doi.org/10.1016/b978-0-444-52014-2.00003-3openalex-snapshot

Molecular biology of Huntington's disease

inferred← citeshttps://doi.org/10.1016/b978-0-444-52014-2.00006-9openalex-snapshot

Spinocerebellar degenerations

inferred← citeshttps://doi.org/10.1016/b978-012088382-0/50026-8openalex-snapshot

Clinical and Pathological Characteristics of Huntington Disease

inferred← citeshttps://doi.org/10.1016/b978-012088382-0/50029-3openalex-snapshot

Drosophila Models of Huntington Disease

inferred← citeshttps://doi.org/10.1016/b978-012088382-0/50056-6openalex-snapshot

SCA7 Mouse Models

inferred← citeshttps://doi.org/10.1016/b978-012369437-9/50024-4openalex-snapshot

Trafficking Defects in Huntington's Disease

inferred← citeshttps://doi.org/10.1016/b978-012369462-1/50014-4openalex-snapshot

Clinical Features and Molecular Biology of Kennedy's Disease

inferred← citeshttps://doi.org/10.1016/b978-012369462-1/50015-6openalex-snapshot

Molecular Pathogenesis and Therapeutic Targets in Huntington's Disease

inferred← citeshttps://doi.org/10.1016/b978-012369462-1/50027-2openalex-snapshot

Pathogenesis of Spinocerebellar Ataxia Type 7: New Insights from Mouse Models and Ataxin-7 Function

inferred← citeshttps://doi.org/10.1016/b978-012369462-1/50028-4openalex-snapshot

Spinocerebellar Ataxia Type 7: Clinical Features to Cellular Pathogenesis

inferred← citeshttps://doi.org/10.1016/b978-012369462-1/50038-7openalex-snapshot

Drosophila Models of Polyglutamine Disorders

inferred← citeshttps://doi.org/10.1016/b978-012370509-9.00043-7openalex-snapshot

Epigenetics – Chromatin Structure and Rett Syndrome

inferred← citeshttps://doi.org/10.1016/b978-1-4377-2369-4.00014-7openalex-snapshot

Huntington disease

inferred← citeshttps://doi.org/10.1016/bs.acc.2020.09.004openalex-snapshot

Histone deacetylase in neuropathology

inferred← citeshttps://doi.org/10.1016/bs.pmbts.2023.02.011openalex-snapshot

Histone deacetylase (HDACs) inhibitors: Clinical applications

inferred← citeshttps://doi.org/10.1016/j.ab.2010.01.010openalex-snapshot

Identification of phenylbutyrate-generated metabolites in Huntington disease patients using parallel liquid chromatography/electrochemical array/mass spectrometry and off-line tandem mass spectrometry

inferred← citeshttps://doi.org/10.1016/j.abb.2011.04.002openalex-snapshot

Sir2 is induced by oxidative stress in a yeast model of Huntington disease and its activation reduces protein aggregation

inferred← citeshttps://doi.org/10.1016/j.apsb.2020.01.015openalex-snapshot

Gene therapy for neurodegenerative disorders: advances, insights and prospects

inferred← citeshttps://doi.org/10.1016/j.arr.2020.101069openalex-snapshot

Discovery of new epigenomics-based biomarkers and the early diagnosis of neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/j.bbadis.2009.07.012openalex-snapshot

Mitochondria in Huntington's disease

inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.10.026openalex-snapshot

Signal transduction and epigenetic mechanisms in the control of microglia activation during neuroinflammation

inferred← citeshttps://doi.org/10.1016/j.bbadis.2016.04.002openalex-snapshot

Loss of striatal 90-kDa ribosomal S6 kinase (Rsk) is a key factor for motor, synaptic and transcription dysfunction in Huntington's disease

inferred← citeshttps://doi.org/10.1016/j.bbadis.2017.11.004openalex-snapshot

Epigenetic regulation of astrocyte function in neuroinflammation and neurodegeneration

inferred← citeshttps://doi.org/10.1016/j.bbagrm.2010.05.008openalex-snapshot

Histone deacetylase inhibitors: A chemical genetics approach to understanding cellular functions

inferred← citeshttps://doi.org/10.1016/j.bbagrm.2010.08.012openalex-snapshot

Tuning acetylation levels with HAT activators: Therapeutic strategy in neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/j.bbalip.2019.04.002openalex-snapshot

The complexity of a monogenic neurodegenerative disease: More than two decades of therapeutic driven research into Niemann-Pick type C disease

inferred← citeshttps://doi.org/10.1016/j.bbamcr.2019.03.006openalex-snapshot

Role of HDACs in cardiac electropathology: Therapeutic implications for atrial fibrillation

inferred← citeshttps://doi.org/10.1016/j.bbr.2008.01.021openalex-snapshot

Epigenetic codes in cognition and behaviour

inferred← citeshttps://doi.org/10.1016/j.bbrc.2003.11.086openalex-snapshot

General transcriptional repression by polyglutamine disease proteins is not directly linked to the presence of inclusion bodies

inferred← citeshttps://doi.org/10.1016/j.bbrc.2008.12.109openalex-snapshot

Identification of novel targets for PGC-1α and histone deacetylase inhibitors in neuroblastoma cells

inferred← citeshttps://doi.org/10.1016/j.bbrc.2010.02.054openalex-snapshot

Histone deacetylase inhibitors de-repress tyrosine hydroxylase expression in the olfactory bulb and rostral migratory stream

inferred← citeshttps://doi.org/10.1016/j.bbrc.2022.11.073openalex-snapshot

Suberoylanilide hydroxamic acid (SAHA) attenuates memory impairment in the offspring of rats exposed to sevoflurane anesthesia

inferred← citeshttps://doi.org/10.1016/j.bcp.2004.05.035openalex-snapshot

Targeting CREB-binding protein (CBP) loss of function as a therapeutic strategy in neurological disorders

inferred← citeshttps://doi.org/10.1016/j.bcp.2022.115295openalex-snapshot

Targeting epigenetics as a promising therapeutic strategy for treatment of neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/j.biocel.2008.08.009openalex-snapshot

Genetic and epigenetic defects in mental retardation

inferred← citeshttps://doi.org/10.1016/j.bioorg.2025.109343openalex-snapshot

Rationally engineered cyclodextrins: smart shuttles for neurological drug targeting and BBB modulation

inferred← citeshttps://doi.org/10.1016/j.biopsych.2005.10.021openalex-snapshot

Cellular Plasticity Cascades: Targets for the Development of Novel Therapeutics for Bipolar Disorder

inferred← citeshttps://doi.org/10.1016/j.biopsych.2006.06.036openalex-snapshot

Antidepressant-Like Effects of the Histone Deacetylase Inhibitor, Sodium Butyrate, in the Mouse

inferred← citeshttps://doi.org/10.1016/j.bmc.2009.05.042openalex-snapshot

Molecular modifications on carboxylic acid derivatives as potent histone deacetylase inhibitors: Activity and docking studies

inferred← citeshttps://doi.org/10.1016/j.bmcl.2012.05.084openalex-snapshot

Photoinduced DNA cleavage by anthracene based hydroxamic acids

inferred← citeshttps://doi.org/10.1016/j.brainres.2008.11.092openalex-snapshot

p300 expression is induced by oxygen deficiency and protects neuron cells from damage

inferred← citeshttps://doi.org/10.1016/j.brainres.2012.01.038openalex-snapshot

Epigenetic programming of neurodegenerative diseases by an adverse environment

inferred← citeshttps://doi.org/10.1016/j.brainres.2012.04.013openalex-snapshot

NF‐κB and epigenetic mechanisms as integrative regulators of brain resilience to anoxic stress

inferred← citeshttps://doi.org/10.1016/j.brainres.2013.02.012openalex-snapshot

Histone deacetylase class II and acetylated core histone immunohistochemistry in human brains with Huntington’s disease

inferred← citeshttps://doi.org/10.1016/j.brainres.2015.07.030openalex-snapshot

Metabolism and epigenetics in the nervous system: Creating cellular fitness and resistance to neuronal death in neurological conditions via modulation of oxygen-, iron-, and 2-oxoglutarate-dependent dioxygenases

inferred← citeshttps://doi.org/10.1016/j.brainresbull.2011.07.021openalex-snapshot

Gene expression and behaviour in mouse models of HD

inferred← citeshttps://doi.org/10.1016/j.brainresbull.2011.10.001openalex-snapshot

Longitudinal analysis of gene expression and behaviour in the HdhQ150 mouse model of Huntington's disease

inferred← citeshttps://doi.org/10.1016/j.brainresbull.2011.10.002openalex-snapshot

Comparative analysis of pathology and behavioural phenotypes in mouse models of Huntington's disease

inferred← citeshttps://doi.org/10.1016/j.brainresbull.2021.04.016openalex-snapshot

NLRP3 inflammasome inhibition by histone acetylation ameliorates sevoflurane-induced cognitive impairment in aged mice by activating the autophagy pathway

inferred← citeshttps://doi.org/10.1016/j.brainresrev.2008.12.001openalex-snapshot

The R6 lines of transgenic mice: A model for screening new therapies for Huntington's disease

showing the first 200 of 813

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.