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Expression of human beta-amyloid peptide in transgenic Caenorhabditis elegans.

https://doi.org/10.1073/pnas.92.20.9368

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Cerebrospinal fluid inhibits Alzheimer β‐amyloid fibril formation in vitro

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Transgenic mouse brain histopathology resembles early Alzheimer's disease

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Amyloid β protein-induced neuronal cell death: neurotoxic properties of aggregated amyloid β protein

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The transthyretin cDNA sequence is normal in transthyretin‐derived senile systemic amyloidosis

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Theoretical models of the ion channel structure of amyloid beta-protein

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Expression of the human beta-amyloid protein of Alzheimer's disease specifically in the brains of transgenic mice.

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Secondary structure of amyloid beta peptide correlates with neurotoxic activity in vitro.

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Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease

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Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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Transthyretin sequesters amyloid beta protein and prevents amyloid formation.

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Familial amyloidosis

inferredcites →https://doi.org/10.1111/j.1749-6632.1993.tb23042.xopenalex-snapshot

Sequestration of Amyloid β‐Peptidea

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Deposits of Amyloid β Protein in the Central Nervous System of Transgenic Mice

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Apolipoprotein E associates with beta amyloid peptide of Alzheimer's disease to form novel monofibrils. Isoform apoE4 associates more efficiently than apoE3.

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Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state

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Neuroprotective Effects of 2-Substituted 1, 3-Selenazole Amide Derivatives on Amyloid-Beta-Induced Toxicity in a Transgenic Caenorhabditis Elegans Model of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s13258-013-0116-2openalex-snapshot

Tackling neurodegenerative diseases: animal models of Alzheimer’s disease and Parkinson’s disease

inferred← citeshttps://doi.org/10.1007/s13530-016-0261-5openalex-snapshot

Electrolyzed-reduced water mitigates amyloid beta toxicity via DAF-16 in C. elegans

inferred← citeshttps://doi.org/10.1016/0304-3940(96)12334-xopenalex-snapshot

Transthyretin gene in Alzheimer's disease patients

inferred← citeshttps://doi.org/10.1016/b978-0-12-374228-5.00006-8openalex-snapshot

Ginkgo biloba Extract in Prevention of Age-Associated Diseases in Elderly Population

inferred← citeshttps://doi.org/10.1016/b978-0-12-382032-7.10014-1openalex-snapshot

Reproductive and developmental toxicity models in relation to neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/b978-0-12-384878-9.00015-7openalex-snapshot

Watching Worms Whither

inferred← citeshttps://doi.org/10.1016/b978-0-12-384937-3.00093-8openalex-snapshot

Epigallocatechin Gallate (EGCG) from Green Tea (Camellia sinensis) and Other Natural Products Mediate Stress Resistance and Slows Down Aging Processes in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/b978-0-12-384937-3.00118-xopenalex-snapshot

Green Tea (−)-Epigallocatechin-3-Gallate and Amyloid Precursor Protein

inferred← citeshttps://doi.org/10.1016/b978-0-12-394620-1.00012-6openalex-snapshot

Analysis of Aging in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/b978-0-12-405195-9.00033-0openalex-snapshot

Modeling Huntington Disease in Yeast and Invertebrates

inferred← citeshttps://doi.org/10.1016/b978-0-12-415894-8.00025-7openalex-snapshot

Models of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-802810-0.00011-8openalex-snapshot

Simple In Vivo Models of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-809468-6.00040-1openalex-snapshot

Animal Models of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1016/b978-0-12-815238-6.00026-2openalex-snapshot

DHA A Key Supplement for Neurologic Health—An Insight

inferred← citeshttps://doi.org/10.1016/b978-0-12-819132-3.00007-5openalex-snapshot

Detection of amyloid aggregation in living systems

inferred← citeshttps://doi.org/10.1016/b978-0-443-16128-5.00015-3openalex-snapshot

Caenorhabditis elegans models of tauopathy

inferred← citeshttps://doi.org/10.1016/b978-0-443-30008-0.00006-5openalex-snapshot

Simple in vivo models of Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/b978-012088382-0/50018-9openalex-snapshot

C. elegans Models of Parkinson Disease

inferred← citeshttps://doi.org/10.1016/b978-012088387-5/50019-4openalex-snapshot

A Critical Evaluation of Nonmammalian Models for Aging Research

inferred← citeshttps://doi.org/10.1016/bs.mcb.2024.06.001openalex-snapshot

Evaluating amyloid-beta aggregation and toxicity in transgenic Caenorhabditis elegans models of Alzheimer’s disease

inferred← citeshttps://doi.org/10.1016/j.abb.2004.12.020openalex-snapshot

Small glutamine-rich tetratricopeptide repeat-containing protein is composed of three structural units with distinct functions

inferred← citeshttps://doi.org/10.1016/j.abb.2008.12.006openalex-snapshot

The sea urchin embryo: A model to study Alzheimer’s beta amyloid induced toxicity

inferred← citeshttps://doi.org/10.1016/j.addr.2013.12.001openalex-snapshot

C. elegans in high-throughput drug discovery

inferred← citeshttps://doi.org/10.1016/j.addr.2025.115655openalex-snapshot

Caenorhabditis elegans as an emerging high throughput chronotherapeutic drug screening platform for human neurodegenerative disorders

inferred← citeshttps://doi.org/10.1016/j.archger.2024.105377openalex-snapshot

Cynaroside extends lifespan and improves the neurondegeneration diseases via insulin/IGF-1 signaling pathway in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.archger.2024.105644openalex-snapshot

Berberine extends healthspan and delays neurodegenerative diseases in Caenorhabditis elegans through ROS-dependent PMK-1/SKN-1 activation

inferred← citeshttps://doi.org/10.1016/j.arr.2012.06.006openalex-snapshot

Pharmacological lifespan extension of invertebrates

inferred← citeshttps://doi.org/10.1016/j.atc.2006.02.002openalex-snapshot

Interactions of Volatile Anesthetics with Neurodegenerative-Disease-Associated Proteins

inferred← citeshttps://doi.org/10.1016/j.autrev.2017.03.001openalex-snapshot

Animal models in idiopathic inflammatory myopathies: How to overcome a translational roadblock?

inferred← citeshttps://doi.org/10.1016/j.bbadis.2006.12.001openalex-snapshot

Alzheimer disease: Amyloidogenesis, the presenilins and animal models

inferred← citeshttps://doi.org/10.1016/j.bbadis.2010.09.012openalex-snapshot

Zebrafish as a tool in Alzheimer's disease research

inferred← citeshttps://doi.org/10.1016/j.bbadis.2013.09.011openalex-snapshot

Transthyretin suppresses the toxicity of oligomers formed by misfolded proteins in vitro

inferred← citeshttps://doi.org/10.1016/j.bbadis.2014.01.015openalex-snapshot

Genetic screens in Caenorhabditis elegans models for neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/j.bbadis.2021.166203openalex-snapshot

Human amyloid beta and α-synuclein co-expression in neurons impair behavior and recapitulate features for Lewy body dementia in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.bbagen.2016.07.013openalex-snapshot

Identification and functional characterization of a putative IDE, C28F5.4 (ceIDE-1), in Caenorhabditis elegans: Implications for Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/j.bbagen.2018.10.008openalex-snapshot

Methacycline displays a strong efficacy in reducing toxicity in a SCA3 Caenorhabditis elegans model

inferred← citeshttps://doi.org/10.1016/j.bbrc.2008.05.132openalex-snapshot

Cytoplasmic expression of mouse prion protein causes severe toxicity in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.biopha.2018.08.037openalex-snapshot

Sesamin and sesamolin reduce amyloid-β toxicity in a transgenic Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.bpc.2024.107180openalex-snapshot

Islet amyloid polypeptide tagged with green fluorescent protein localises to mitochondria and forms filamentous aggregates in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.brainres.2009.08.002openalex-snapshot

Effects of non-steroidal anti-inflammatory drugs on Aβ deposition in Aβ1–42 transgenic C. elegans

inferred← citeshttps://doi.org/10.1016/j.cbpa.2022.111166openalex-snapshot

Invited review: Unearthing the mechanisms of age-related neurodegenerative disease using Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.cell.2009.11.014openalex-snapshot

Reduced IGF-1 Signaling Delays Age-Associated Proteotoxicity in Mice

inferred← citeshttps://doi.org/10.1016/j.cell.2010.07.020openalex-snapshot

Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity

inferred← citeshttps://doi.org/10.1016/j.cell.2014.01.061openalex-snapshot

Hexosamine Pathway Metabolites Enhance Protein Quality Control and Prolong Life

inferred← citeshttps://doi.org/10.1016/j.cell.2017.05.036openalex-snapshot

Microbial Genetic Composition Tunes Host Longevity

inferred← citeshttps://doi.org/10.1016/j.celrep.2014.09.042openalex-snapshot

A Chaperome Subnetwork Safeguards Proteostasis in Aging and Neurodegenerative Disease

inferred← citeshttps://doi.org/10.1016/j.celrep.2014.11.028openalex-snapshot

Differential Regulation of the Heat Shock Factor 1 and DAF-16 by Neuronal nhl-1 in the Nematode C. elegans

inferred← citeshttps://doi.org/10.1016/j.celrep.2018.05.093openalex-snapshot

A PQM-1-Mediated Response Triggers Transcellular Chaperone Signaling and Regulates Organismal Proteostasis

inferred← citeshttps://doi.org/10.1016/j.celrep.2019.06.057openalex-snapshot

XBP-1 Remodels Lipid Metabolism to Extend Longevity

inferred← citeshttps://doi.org/10.1016/j.celrep.2021.109753openalex-snapshot

Vitamin B12 impacts amyloid beta-induced proteotoxicity by regulating the methionine/S-adenosylmethionine cycle

inferred← citeshttps://doi.org/10.1016/j.celrep.2022.110350openalex-snapshot

Neuropeptide signaling and SKN-1 orchestrate differential responses of the proteostasis network to dissimilar proteotoxic insults

inferred← citeshttps://doi.org/10.1016/j.chemosphere.2020.128594openalex-snapshot

Early-life and chronic exposure to di(2-ethylhexyl) phthalate enhances amyloid-β toxicity associated with an autophagy-related gene in Caenorhabditis elegans Alzheimer’s disease models

inferred← citeshttps://doi.org/10.1016/j.cmet.2012.12.013openalex-snapshot

Direct and Indirect Gene Regulation by a Life-Extending FOXO Protein in C. elegans: Roles for GATA Factors and Lipid Gene Regulators

inferred← citeshttps://doi.org/10.1016/j.colsurfb.2026.115464openalex-snapshot

Biomimetic red blood cell membrane–coated cerium metal–organic framework for multi-target synergistic therapy of Alzheimer’s disease

inferred← citeshttps://doi.org/10.1016/j.copbio.2019.12.011openalex-snapshot

Caenorhabditis elegans as a model system for human diseases

inferred← citeshttps://doi.org/10.1016/j.crfs.2022.05.001openalex-snapshot

Caenorhabditis elegans as an in vivo model for food bioactives: A review

inferred← citeshttps://doi.org/10.1016/j.cub.2019.06.031openalex-snapshot

Neuronal XBP-1 Activates Intestinal Lysosomes to Improve Proteostasis in C. elegans

inferred← citeshttps://doi.org/10.1016/j.ddmod.2018.11.001openalex-snapshot

The nematode Caenorhabditis elegans as a model for aging research

inferred← citeshttps://doi.org/10.1016/j.ddtec.2012.02.002openalex-snapshot

Alzheimer's disease drug discovery: in vivo screening using Caenorhabditis elegans as a model for β-amyloid peptide-induced toxicity

inferred← citeshttps://doi.org/10.1016/j.ecoenv.2026.120406openalex-snapshot

Time-dependent shift from antagonism to synergism in mixture toxicity mediated by GFP-modified Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.ejmcr.2022.100044openalex-snapshot

Experimental modelling of Alzheimer's disease for therapeutic screening

inferred← citeshttps://doi.org/10.1016/j.ejmech.2024.116684openalex-snapshot

Natural products targeting amyloid-β oligomer neurotoxicity in Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/j.ejpb.2022.10.008openalex-snapshot

Improving physicochemical properties and pharmacological activities of ternary co-amorphous systems

inferred← citeshttps://doi.org/10.1016/j.ejphar.2017.09.017openalex-snapshot

Lower vertebrate and invertebrate models of Alzheimer's disease – A review

inferred← citeshttps://doi.org/10.1016/j.ejphar.2017.11.051openalex-snapshot

Caenorhabditis elegans as a model system for target identification and drug screening against neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/j.ejphar.2022.174829openalex-snapshot

Cannabidiol protects against Alzheimer's disease in C. elegans via ROS scavenging activity of its phenolic hydroxyl groups

inferred← citeshttps://doi.org/10.1016/j.ejphar.2022.174887openalex-snapshot

Ginsenoside Rf inhibits human tau proteotoxicity and causes specific LncRNA, miRNA and mRNA expression changes in Caenorhabditis elegans model of tauopathy

inferred← citeshttps://doi.org/10.1016/j.envint.2022.107482openalex-snapshot

Decabromodiphenyl ethane induces locomotion neurotoxicity and potential Alzheimer’s disease risks through intensifying amyloid-beta deposition by inhibiting transthyretin/transthyretin-like proteins

inferred← citeshttps://doi.org/10.1016/j.exger.2006.06.059openalex-snapshot

C. elegans models of age-associated neurodegenerative diseases: Lessons from transgenic worm models of Alzheimer’s disease

inferred← citeshttps://doi.org/10.1016/j.exger.2009.02.010openalex-snapshot

Reserpine ameliorates Aβ toxicity in the Alzheimer’s disease model in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2010.08.032openalex-snapshot

Countering neurodegeneration by reducing the activity of the insulin/IGF signaling pathway: Current knowledge and future prospects

inferred← citeshttps://doi.org/10.1016/j.exger.2011.12.006openalex-snapshot

Reserpine modulates neurotransmitter release to extend lifespan and alleviate age-dependent Aβ proteotoxicity in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2017.07.015openalex-snapshot

The anti-aging and anti-oxidation effects of tea water extract in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2018.04.021openalex-snapshot

5-Methoxyindole-2-carboxylic acid (MICA) suppresses Aβ-mediated pathology in C. elegans

inferred← citeshttps://doi.org/10.1016/j.expneurol.2013.09.024openalex-snapshot

Modeling neurodegenerative diseases in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.fct.2019.110962openalex-snapshot

Phytol loaded PLGA nanoparticles regulate the expression of Alzheimer's related genes and neuronal apoptosis against amyloid-β induced toxicity in Neuro-2a cells and transgenic Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.fct.2020.111808openalex-snapshot

Ferulic acid delayed amyloid β-induced pathological symptoms by autophagy pathway via a fasting-like effect in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.febslet.2008.02.034openalex-snapshot

Transthyretin binding to A‐Beta peptide – Impact on A‐Beta fibrillogenesis and toxicity

inferred← citeshttps://doi.org/10.1016/j.fitote.2020.104536openalex-snapshot

Cadinane-type sesquiterpenes from the resinous exudates of Commiphora myrrha and their anti-Alzheimer's disease bioactivities

inferred← citeshttps://doi.org/10.1016/j.fitote.2026.107377openalex-snapshot

Novel oligomeric caffeoyl derivatives and acylated flavone glycosides from Satureja nervosa Desf.: Chemical characterisation, structure-activity relationships, and multi-target bioactivity in Caenorhabditis elegans disease models

inferred← citeshttps://doi.org/10.1016/j.foodres.2022.111696openalex-snapshot

Black mulberry (Morus nigra) fruit extract alleviated AD-Like symptoms induced by toxic Aβ protein in transgenic Caenorhabditis elegans via insulin DAF-16 signaling pathway

inferred← citeshttps://doi.org/10.1016/j.foodres.2025.116916openalex-snapshot

Discovery of dietary beneficial compounds for healthy aging: From worm to human

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2007.05.037openalex-snapshot

Roles of amyloid β-peptide-associated oxidative stress and brain protein modifications in the pathogenesis of Alzheimer's disease and mild cognitive impairment

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2010.10.707openalex-snapshot

β-Amyloid peptide increases levels of iron content and oxidative stress in human cell and Caenorhabditis elegans models of Alzheimer disease

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2014.03.003openalex-snapshot

The mitochondria-targeted antioxidant MitoQ extends lifespan and improves healthspan of a transgenic Caenorhabditis elegans model of Alzheimer disease

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2022.09.030openalex-snapshot

Tanshinone IIA loaded chitosan nanoparticles decrease toxicity of β-amyloid peptide in a Caenorhabditis elegans model of Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2024.09.034openalex-snapshot

DMT1 knockout abolishes ferroptosis induced mitochondrial dysfunction in C. elegans amyloid β proteotoxicity

inferred← citeshttps://doi.org/10.1016/j.gendis.2020.08.004openalex-snapshot

Suppression of a core metabolic enzyme dihydrolipoamide dehydrogenase (dld) protects against amyloid beta toxicity in C. elegans model of Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/j.heliyon.2024.e34963openalex-snapshot

The prion-like effect and prion-like protein targeting strategy in amyotrophic lateral sclerosis

inferred← citeshttps://doi.org/10.1016/j.idairyj.2024.105921openalex-snapshot

Functional assessment of a whey protein hydrolysate: In vivo responses in lipid regulation, oxidant defense, and neural protection in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2018.03.035openalex-snapshot

Neuroprotective effects of Coptis chinensis Franch polysaccharide on amyloid-beta (Aβ)-induced toxicity in a transgenic Caenorhabditis elegans model of Alzheimer's disease (AD)

inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2022.04.124openalex-snapshot

Chondroitin sulfate E alleviates β-amyloid toxicity in transgenic Caenorhabditis elegans by inhibiting its aggregation

inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2025.140758openalex-snapshot

Lycium ruthenicum Murray exosome-like nanovesicles alleviated Alzheimer's disease–like symptoms induced by Aβ protein in transgenic Caenorhabditis elegans through the DAF-16 pathway

inferred← citeshttps://doi.org/10.1016/j.indcrop.2022.115798openalex-snapshot

Amaryllidaceae alkaloids from Lycoris suppress amyloid β-induced neurodegeneration in transgenic Caenorhabditis elegans CL2355

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