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Prion protein codon 129 polymorphism in mild cognitive impairment and dementia: the Rotterdam Study

https://doi.org/10.1093/braincomms/fcaa030

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1001/jamaneurol.2013.5847openalex-snapshot

Amyloid-β and Tau

inferredcites →https://doi.org/10.1002/ana.10507openalex-snapshot

PRNP Val129 homozygosity increases risk for early‐onset Alzheimer's disease

inferredcites →https://doi.org/10.1002/ana.10658openalex-snapshot

Early cognitive decline is associated with prion protein codon 129 polymorphism

inferredcites →https://doi.org/10.1002/ana.21583openalex-snapshot

Variant Creutzfeldt–Jakob disease in France and the United Kingdom: Evidence for the same agent strain

inferredcites →https://doi.org/10.1007/s00401-009-0585-1openalex-snapshot

Incidence and spectrum of sporadic Creutzfeldt–Jakob disease variants with mixed phenotype and co-occurrence of PrPSc types: an updated classification

inferredcites →https://doi.org/10.1007/s00401-012-1002-8openalex-snapshot

Consensus classification of human prion disease histotypes allows reliable identification of molecular subtypes: an inter-rater study among surveillance centres in Europe and USA

inferredcites →https://doi.org/10.1007/s10654-015-0105-7openalex-snapshot

The Rotterdam Scan Study: design update 2016 and main findings

inferredcites →https://doi.org/10.1007/s10654-017-0321-4openalex-snapshot

The Rotterdam Study: 2018 update on objectives, design and main results

inferredcites →https://doi.org/10.1016/b978-0-444-63945-5.00016-7openalex-snapshot

Prion-like mechanisms in Alzheimer disease

inferredcites →https://doi.org/10.1016/j.jns.2013.01.020openalex-snapshot

The association between the methionine/valine (M/V) polymorphism (rs1799990) in the PRNP gene and the risk of Alzheimer disease: An update by meta-analysis

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2005.05.025openalex-snapshot

Is M129V of PRNP gene associated with Alzheimer's disease? A case-control study and a meta-analysis

inferredcites →https://doi.org/10.1016/s0140-6736(99)01342-2openalex-snapshot

Codon 129 prion protein genotype and sporadic Creutzfeldt-Jakob disease

inferredcites →https://doi.org/10.1038/ng.3679openalex-snapshot

Reference-based phasing using the Haplotype Reference Consortium panel

inferredcites →https://doi.org/10.1038/s41588-019-0358-2openalex-snapshot

Genetic meta-analysis of diagnosed Alzheimer’s disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing

inferredcites →https://doi.org/10.1038/sj.mp.4001327openalex-snapshot

M129V variation in the prion protein may influence cognitive performance

inferredcites →https://doi.org/10.1038/srep38435openalex-snapshot

Mutational analysis of PRNP in Alzheimer’s disease and frontotemporal dementia in China

inferredcites →https://doi.org/10.1056/nejmc1610003openalex-snapshot

Variant Creutzfeldt–Jakob Disease in a Patient with Heterozygosity at PRNP Codon 129

inferredcites →https://doi.org/10.1111/j.1468-1331.2007.01786.xopenalex-snapshot

Polymorphisms at codons 129 and 219 of the prion protein gene (PRNP) are not associated with sporadic Alzheimer's disease in the Korean population

inferredcites →https://doi.org/10.1111/j.1468-1331.2007.02021.xopenalex-snapshot

Codon 129 polymorphism of prion protein gene in sporadic Alzheimer’s disease

inferredcites →https://doi.org/10.1126/science.6801762openalex-snapshot

Novel Proteinaceous Infectious Particles Cause Scrapie

inferredcites →https://doi.org/10.1136/heartjnl-2014-307419openalex-snapshot

ABCB1 gene variants, digoxin and risk of sudden cardiac death in a general population

inferredcites →https://doi.org/10.1155/2010/196538openalex-snapshot

Lack of Association betweenPRNPM129V Polymorphism and Multiple Sclerosis, Mild Cognitive Impairment, Alcoholism and Schizophrenia in a Korean Population

inferredcites →https://doi.org/10.1186/s12916-015-0377-5openalex-snapshot

The potential for prevention of dementia across two decades: the prospective, population-based Rotterdam Study

inferredcites →https://doi.org/10.1186/s40478-019-0706-6openalex-snapshot

The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

inferredcites →https://doi.org/10.3233/dma-2010-0711openalex-snapshot

Lack of association between PRNP M129V polymorphism and multiple sclerosis, mild cognitive impairment, alcoholism and schizophrenia in a Korean population.

inferredcites →https://doi.org/10.3233/jad-132558openalex-snapshot

Determinants, MRI Correlates, and Prognosis of Mild Cognitive Impairment: The Rotterdam Study

inferredcites →https://doi.org/10.4161/pri.23903openalex-snapshot

The prion protein M129V polymorphism

inferredcites →https://doi.org/10.4161/pri.3.4.9980openalex-snapshot

Prion protein and Alzheimer disease

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4820084openalex-extracted

Unraveling the Structural Facets of Prp127-132 Polymorphic Variants Using Combined Molecular Dynamics/Markov Modeling

inferred← citeshttps://doi.org/10.1007/s00415-026-13862-6openalex-snapshot

Sporadic Creutzfeldt–Jakob disease: a multidimensional review from epidemiology to treatment and prognosis

inferred← citeshttps://doi.org/10.1016/j.heliyon.2023.e13974openalex-snapshot

Preventive or promotive effects of PRNP polymorphic heterozygosity on the onset of prion disease

inferred← citeshttps://doi.org/10.1016/j.jmgm.2024.108927openalex-snapshot

Structural characterization of codon 129 polymorphism in prion peptide segments (PrP127-132) using the Markov State Models

inferred← citeshttps://doi.org/10.1186/s12877-022-03280-4openalex-snapshot

A clinicopathological study of selected cognitive impairment cases in Lothian, Scotland: enhanced CJD surveillance in the 65 + population group

inferred← citeshttps://doi.org/10.3390/biomedicines12081725openalex-snapshot

Perspectives on CRISPR Genome Editing to Prevent Prion Diseases in High-Risk Individuals

inferred← citeshttps://doi.org/10.3390/molecules27061776openalex-snapshot

Amyloid Cross-Seeding: Mechanism, Implication, and Inhibition

inferred← citeshttps://doi.org/10.53846/goediss-9625openalex-snapshot

Optimization of the RT-QuIC in prion disease diagnostic

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